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You searched for +publisher:"University of Alabama – Birmingham" +contributor:("Lopez, Richard D.<br>"). Showing records 1 – 6 of 6 total matches.

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1. Kojima, Kyoko, Ph.D. Mouse modeling of pancreatic ductal adenocarcinoma (PDAC): search for early diagnostic markers and therapeutic targets.

Degree: PhD, 2009, University of Alabama – Birmingham

PDAC is a highly malignant neoplasm that carries a very poor prognosis. PDAC development is a multistage transformation process that involves multiple genetic alterations that… (more)

Subjects/Keywords: Carcinoma in Situ  – genetics<; br>; Genes, ras<; br>; Pancreatic Neoplasms  – diagnosis<; br>; Pancreatic Neoplasms  – genetics<; br>; Proteomics<; br>; Smad4 Protein  – genetics<; br>; Tumor Markers, Biological  – analysis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kojima, Kyoko, P. D. (2009). Mouse modeling of pancreatic ductal adenocarcinoma (PDAC): search for early diagnostic markers and therapeutic targets. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,396

Chicago Manual of Style (16th Edition):

Kojima, Kyoko, Ph D. “Mouse modeling of pancreatic ductal adenocarcinoma (PDAC): search for early diagnostic markers and therapeutic targets.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,396.

MLA Handbook (7th Edition):

Kojima, Kyoko, Ph D. “Mouse modeling of pancreatic ductal adenocarcinoma (PDAC): search for early diagnostic markers and therapeutic targets.” 2009. Web. 29 Mar 2020.

Vancouver:

Kojima, Kyoko PD. Mouse modeling of pancreatic ductal adenocarcinoma (PDAC): search for early diagnostic markers and therapeutic targets. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,396.

Council of Science Editors:

Kojima, Kyoko PD. Mouse modeling of pancreatic ductal adenocarcinoma (PDAC): search for early diagnostic markers and therapeutic targets. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,396

2. Hensel, Jonathan Adam. The role of LL-37 in prostate cancer and its potential as a therapeutic target.

Degree: 2011, University of Alabama – Birmingham

LL-37 is the only cathelicidin-derived anti-microbial peptide in humans and has been shown to stimulate proliferation, angiogenesis, cellular migration and inhibit apoptosis, in addition to… (more)

Subjects/Keywords: Antimicrobial Cationic Peptides  – metabolism<; br>; Cathelicidins<; br>; Molecular Targeted Therapy  – methods<; br>; Neoplasms, Hormone-Dependent<; br>; Prostatic Neoplasms  – genetics<; br>; Prostatic Neoplasms  – metabolism<; br>; Prostatic Neoplasms  – pathology

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APA (6th Edition):

Hensel, J. A. (2011). The role of LL-37 in prostate cancer and its potential as a therapeutic target. (Thesis). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,875

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hensel, Jonathan Adam. “The role of LL-37 in prostate cancer and its potential as a therapeutic target.” 2011. Thesis, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,875.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hensel, Jonathan Adam. “The role of LL-37 in prostate cancer and its potential as a therapeutic target.” 2011. Web. 29 Mar 2020.

Vancouver:

Hensel JA. The role of LL-37 in prostate cancer and its potential as a therapeutic target. [Internet] [Thesis]. University of Alabama – Birmingham; 2011. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,875.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hensel JA. The role of LL-37 in prostate cancer and its potential as a therapeutic target. [Thesis]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,875

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

3. Warram, Jason M. (Jason Morgan). Strategies for combined screening and imaging of breast cancer.

Degree: PhD, 2011, University of Alabama – Birmingham

Successful treatment of breast cancer directly correlates with tumor stage and grade at diagnosis. Early diagnosis leads to a five-year relative survival rate of 96.8%… (more)

Subjects/Keywords: Adenoviridae  – genetics<; br>; Breast Neoplasms  – diagnosis<; br>; Gene Therapy<; br>; Mass Screening  – methods<; br>; Microbubbles<; br>; Neoplasm Metastasis<; br>; Promoter Regions, Genetic  – genetics<; br>; Vascular Endothelial Growth Factor Receptor-2  – immunology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Warram, J. M. (. M. (2011). Strategies for combined screening and imaging of breast cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,881

Chicago Manual of Style (16th Edition):

Warram, Jason M (Jason Morgan). “Strategies for combined screening and imaging of breast cancer.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,881.

MLA Handbook (7th Edition):

Warram, Jason M (Jason Morgan). “Strategies for combined screening and imaging of breast cancer.” 2011. Web. 29 Mar 2020.

Vancouver:

Warram JM(M. Strategies for combined screening and imaging of breast cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,881.

Council of Science Editors:

Warram JM(M. Strategies for combined screening and imaging of breast cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,881

4. Nick, Heidi Jean. Differential contributions of c-Kit activating mutations to promotion of AML1-ETO associated neoplasia.

Degree: PhD, 2011, University of Alabama – Birmingham

The t(8;21) translocation, which generates an AML1-ETO fusion protein (also known as RUNX1-ETO), is one of the most frequent cytogenetic abnormalities in acute myeloid leukemia… (more)

Subjects/Keywords: Chromosomes, Human, Pair 21<; br>; Chromosomes, Human, Pair 8<; br>; DNA Mutational Analysis<; br>; Leukemia, Myeloid, Acute  – genetics<; br>; Proto-Oncogene Proteins c-kit  – genetics<; br>; Translocation, Genetic

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APA (6th Edition):

Nick, H. J. (2011). Differential contributions of c-Kit activating mutations to promotion of AML1-ETO associated neoplasia. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,883

Chicago Manual of Style (16th Edition):

Nick, Heidi Jean. “Differential contributions of c-Kit activating mutations to promotion of AML1-ETO associated neoplasia.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,883.

MLA Handbook (7th Edition):

Nick, Heidi Jean. “Differential contributions of c-Kit activating mutations to promotion of AML1-ETO associated neoplasia.” 2011. Web. 29 Mar 2020.

Vancouver:

Nick HJ. Differential contributions of c-Kit activating mutations to promotion of AML1-ETO associated neoplasia. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,883.

Council of Science Editors:

Nick HJ. Differential contributions of c-Kit activating mutations to promotion of AML1-ETO associated neoplasia. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,883

5. Beck, Benjamin H. (Benjamin Hester). Immunotherapy of cancer employing γδ-T cells: a study examining their utility and feasibility.

Degree: PhD, 2009, University of Alabama – Birmingham

Unlike antigen-specific αβ-T cells, γδ-T cells can recognize and lyse cancerous cells rapidly upon encounter in a manner that does not require the recognition of… (more)

Subjects/Keywords: Adenocarcinoma  – therapy<; br>; Breast Neoplasms  – pathology<; br>; Immunotherapy, Adoptive<; br>; Mammary Neoplasms, Experimental  – therapy<; br>; Receptors, Antigen, T-Cell, gamma-delta  – immunology T-Lymphocyte Subsets  – transplantation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Beck, B. H. (. H. (2009). Immunotherapy of cancer employing γδ-T cells: a study examining their utility and feasibility. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1087

Chicago Manual of Style (16th Edition):

Beck, Benjamin H (Benjamin Hester). “Immunotherapy of cancer employing γδ-T cells: a study examining their utility and feasibility.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1087.

MLA Handbook (7th Edition):

Beck, Benjamin H (Benjamin Hester). “Immunotherapy of cancer employing γδ-T cells: a study examining their utility and feasibility.” 2009. Web. 29 Mar 2020.

Vancouver:

Beck BH(H. Immunotherapy of cancer employing γδ-T cells: a study examining their utility and feasibility. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1087.

Council of Science Editors:

Beck BH(H. Immunotherapy of cancer employing γδ-T cells: a study examining their utility and feasibility. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1087

6. Bodenstine, Thomas Morgan. Molecular Mechanisms Of Breast Cancer Metastasis: Gap Junction Intercellular Communication And The Bone Microenvironment.

Degree: PhD, 2010, University of Alabama – Birmingham

Metastatic disease accounts for the overwhelming majority of cancer related deaths. More specifically, breast cancer remains one of the leading causes of death in women… (more)

Subjects/Keywords: Atherosclerosis – etiology.<; br>; Cardiovascular Diseases – etiology<; br>; Disease Susceptibility<; br>; DNA, Mitochondrial – genetics<; br>; Mice<; br>; Mitochondria – physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Bodenstine, T. M. (2010). Molecular Mechanisms Of Breast Cancer Metastasis: Gap Junction Intercellular Communication And The Bone Microenvironment. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1386

Chicago Manual of Style (16th Edition):

Bodenstine, Thomas Morgan. “Molecular Mechanisms Of Breast Cancer Metastasis: Gap Junction Intercellular Communication And The Bone Microenvironment.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed March 29, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1386.

MLA Handbook (7th Edition):

Bodenstine, Thomas Morgan. “Molecular Mechanisms Of Breast Cancer Metastasis: Gap Junction Intercellular Communication And The Bone Microenvironment.” 2010. Web. 29 Mar 2020.

Vancouver:

Bodenstine TM. Molecular Mechanisms Of Breast Cancer Metastasis: Gap Junction Intercellular Communication And The Bone Microenvironment. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Mar 29]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1386.

Council of Science Editors:

Bodenstine TM. Molecular Mechanisms Of Breast Cancer Metastasis: Gap Junction Intercellular Communication And The Bone Microenvironment. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1386

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