Advanced search options

Advanced Search Options 🞨

Browse by author name (“Author name starts with…”).

Find ETDs with:

in
/  
in
/  
in
/  
in

Written in Published in Earliest date Latest date

Sorted by

Results per page:

Sorted by: relevance · author · university · dateNew search

You searched for +publisher:"University of Alabama – Birmingham" +contributor:("Lin, Fang-Tsyr"). Showing records 1 – 13 of 13 total matches.

Search Limiters

Last 2 Years | English Only

No search limiters apply to these results.

▼ Search Limiters

1. Harms, Paul William. Modulation of cell signaling by Tomoregulins in embryogenesis and cancer.

Degree: PhD, 2006, University of Alabama – Birmingham

Growth factor signals often regulate similar cellular processes both during embryogenesis and in adult homeostasis. Stringent control of these signals ensures proper embryonic development and… (more)

Subjects/Keywords: Homeodomain Proteins <; br>; Membrane Proteins  – metabolism <; br>; Neoplasm Proteins  – metabolism <; br>; Proteins <; br>; Signal Transduction  – physiology <; br>; Transcription Factors <; br>; Xenopus Proteins

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Harms, P. W. (2006). Modulation of cell signaling by Tomoregulins in embryogenesis and cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,369

Chicago Manual of Style (16th Edition):

Harms, Paul William. “Modulation of cell signaling by Tomoregulins in embryogenesis and cancer.” 2006. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,369.

MLA Handbook (7th Edition):

Harms, Paul William. “Modulation of cell signaling by Tomoregulins in embryogenesis and cancer.” 2006. Web. 19 Sep 2019.

Vancouver:

Harms PW. Modulation of cell signaling by Tomoregulins in embryogenesis and cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2006. [cited 2019 Sep 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,369.

Council of Science Editors:

Harms PW. Modulation of cell signaling by Tomoregulins in embryogenesis and cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2006. Available from: http://contentdm.mhsl.uab.edu/u?/etd,369

2. Qian, Yingjuan, Ph.D. The role of DEC1 in P53-dependent cellular senescence.

Degree: PhD, 2008, University of Alabama – Birmingham

The p53 tumor suppressor is the most commonly mutated gene in human cancers. As a transcription factor, p53 exerts its tumor suppressor function through the… (more)

Subjects/Keywords: Basic Helix-Loop-Helix Transcription Factors  – genetics<; br>; Basic Helix-Loop-Helix Transcription Factors  – physiology<; br>; Cell Aging  – physiology<; br>; DNA Damage<; br>; Tumor Suppressor Protein p53  – physiology<; br>; Tumor Suppressor Proteins  – genetics

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Qian, Yingjuan, P. D. (2008). The role of DEC1 in P53-dependent cellular senescence. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,462

Chicago Manual of Style (16th Edition):

Qian, Yingjuan, Ph D. “The role of DEC1 in P53-dependent cellular senescence.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,462.

MLA Handbook (7th Edition):

Qian, Yingjuan, Ph D. “The role of DEC1 in P53-dependent cellular senescence.” 2008. Web. 19 Sep 2019.

Vancouver:

Qian, Yingjuan PD. The role of DEC1 in P53-dependent cellular senescence. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Sep 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,462.

Council of Science Editors:

Qian, Yingjuan PD. The role of DEC1 in P53-dependent cellular senescence. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,462

3. Cowan, Jon Walter. Proteolysis and the growth hormone receptor: identification and characterization of GHR as a [gamma]-secretase substrate.

Degree: PhD, 2007, University of Alabama – Birmingham

Growth hormone (GH) is a powerful promoter of postnatal longitudinal bone growth in mammals. GH signaling is mediated exclusively by the GH receptor (GHR), and… (more)

Subjects/Keywords: Acetylcysteine  – analogs & derivatives<; br>; ADAM Proteins<; br>; Growth Hormone  – metabolism<; br>; Membrane Proteins  – chemistry<; br>; Protein-Tyrosine Kinases  – pharmacology<; br>; Receptors, Somatotropin  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cowan, J. W. (2007). Proteolysis and the growth hormone receptor: identification and characterization of GHR as a [gamma]-secretase substrate. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,513

Chicago Manual of Style (16th Edition):

Cowan, Jon Walter. “Proteolysis and the growth hormone receptor: identification and characterization of GHR as a [gamma]-secretase substrate.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,513.

MLA Handbook (7th Edition):

Cowan, Jon Walter. “Proteolysis and the growth hormone receptor: identification and characterization of GHR as a [gamma]-secretase substrate.” 2007. Web. 19 Sep 2019.

Vancouver:

Cowan JW. Proteolysis and the growth hormone receptor: identification and characterization of GHR as a [gamma]-secretase substrate. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Sep 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,513.

Council of Science Editors:

Cowan JW. Proteolysis and the growth hormone receptor: identification and characterization of GHR as a [gamma]-secretase substrate. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,513

4. Zhao, Xueyan. Regulation of human MMP-9 gene expression by transcriptional coactivators and interferon beta.

Degree: PhD, 2008, University of Alabama – Birmingham

Matrix metalloprotinases are zinc-dependent endopeptidases with broad substrates from extracellular matrix proteins to bioactive molecules. Physiologically, they regulate tissue remodeling and immune responses. However, in… (more)

Subjects/Keywords: Gene Expression Regulation, Enzymologic<; br>; Interferon-beta  – pharmacology<; br>; Interferon-Stimulated Gene Factor 3  – metabolism<; br>; Interferon-beta/pharmacology<; br>; Matrix Metalloproteinase 9  – genetics<; br>; Matrix Metalloproteinase 9  – metabolism<; br>; Transcription Factors  – metabolism<; br>; Transcriptional Activation  – genetics

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhao, X. (2008). Regulation of human MMP-9 gene expression by transcriptional coactivators and interferon beta. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,542

Chicago Manual of Style (16th Edition):

Zhao, Xueyan. “Regulation of human MMP-9 gene expression by transcriptional coactivators and interferon beta.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,542.

MLA Handbook (7th Edition):

Zhao, Xueyan. “Regulation of human MMP-9 gene expression by transcriptional coactivators and interferon beta.” 2008. Web. 19 Sep 2019.

Vancouver:

Zhao X. Regulation of human MMP-9 gene expression by transcriptional coactivators and interferon beta. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Sep 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,542.

Council of Science Editors:

Zhao X. Regulation of human MMP-9 gene expression by transcriptional coactivators and interferon beta. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,542

5. Yan, Ming. The structural and functional studies of yeast nucleotide exchange factor Sil1P and its complex with Bip.

Degree: PhD, 2010, University of Alabama – Birmingham

Sil1 is an Endoplasmic Reticulum (ER) localized protein. SIL1 was initially identified as a UPR-regulated gene. Later studies show Sil1 functions as the nucleotide exchange… (more)

Subjects/Keywords: Fungal Proteins  – chemistry<; br>; HSP70 Heat-Shock Proteins  – chemistry<; br>; Membrane Transport Proteins  – chemistry<; br>; Saccharomyces cerevisiae  – metabolism<; br>; Saccharomyces cerevisiae Proteins  – chemistry

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yan, M. (2010). The structural and functional studies of yeast nucleotide exchange factor Sil1P and its complex with Bip. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1115

Chicago Manual of Style (16th Edition):

Yan, Ming. “The structural and functional studies of yeast nucleotide exchange factor Sil1P and its complex with Bip.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1115.

MLA Handbook (7th Edition):

Yan, Ming. “The structural and functional studies of yeast nucleotide exchange factor Sil1P and its complex with Bip.” 2010. Web. 19 Sep 2019.

Vancouver:

Yan M. The structural and functional studies of yeast nucleotide exchange factor Sil1P and its complex with Bip. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Sep 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1115.

Council of Science Editors:

Yan M. The structural and functional studies of yeast nucleotide exchange factor Sil1P and its complex with Bip. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1115

6. Zheng, Ying. A CK2-dependent mechanism for activation of the JAK-STAT signaling pathway: implications for cancer biology.

Degree: PhD, 2010, University of Alabama – Birmingham

Janus Kinase (JAK)-Signal Transducer and Activator of Transcription (STAT) signaling is involved in regulation of cell survival, proliferation and differentiation. JAK tyrosine kinases can be… (more)

Subjects/Keywords: Casein Kinase II  – metabolism<; br>; Hematologic Neoplasms  – metabolism<; br>; JNK Mitogen-Activated Protein Kinases  – metabolism<; br>; Polycythemia Vera  – metabolism<; br>; STAT Transcription Factors  – metabolism<; br>; Signal Transduction  – physiology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zheng, Y. (2010). A CK2-dependent mechanism for activation of the JAK-STAT signaling pathway: implications for cancer biology. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1168

Chicago Manual of Style (16th Edition):

Zheng, Ying. “A CK2-dependent mechanism for activation of the JAK-STAT signaling pathway: implications for cancer biology.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1168.

MLA Handbook (7th Edition):

Zheng, Ying. “A CK2-dependent mechanism for activation of the JAK-STAT signaling pathway: implications for cancer biology.” 2010. Web. 19 Sep 2019.

Vancouver:

Zheng Y. A CK2-dependent mechanism for activation of the JAK-STAT signaling pathway: implications for cancer biology. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Sep 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1168.

Council of Science Editors:

Zheng Y. A CK2-dependent mechanism for activation of the JAK-STAT signaling pathway: implications for cancer biology. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1168

7. Champattanachai, Voraratt. Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia.

Degree: PhD, 2008, University of Alabama – Birmingham

Increased levels of protein O-linked-N-acetylglucosamine (O-GlcNAc) have been correlated with increased cell survival following stress. Therefore the goal of this study was to determine whether… (more)

Subjects/Keywords: Acetylglucosamine  – metabolism <; br>; Glucosamine  – pharmacology <; br>; Glycoproteins  – metabolism <; br>; Mitochondria  – metabolism <; br>; Myocardial Reperfusion Injury  – metabolism <; br>; Myocardial Reperfusion Injury  – pathology <; br>; Myocytes, Cardiac  – metabolism <; br>; Myocytes, Cardiac  – pathology

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Champattanachai, V. (2008). Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,194

Chicago Manual of Style (16th Edition):

Champattanachai, Voraratt. “Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,194.

MLA Handbook (7th Edition):

Champattanachai, Voraratt. “Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia.” 2008. Web. 19 Sep 2019.

Vancouver:

Champattanachai V. Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Sep 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,194.

Council of Science Editors:

Champattanachai V. Effects of hexosamine biosynthesis on an in vitro model of cardiac ischemia. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,194

8. Lee, Sun Jung. Transcriptional regulation of CD40 and class II MHC molecules in macrophages and microglia by statins.

Degree: PhD, 2008, University of Alabama – Birmingham

Microglia/macrophages are the main intrinsic immune effector cells and the first cell types that respond to a variety of CNS injures. They have important functions… (more)

Subjects/Keywords: Antigens, CD40  – antagonists & inhibitors <; br>; Antigens, CD40  – genetics <; br>; Genes, MHC Class II <; br>; Histocompatibility Antigens Class II <; br>; Hydroxymethylglutaryl-CoA Reductase Inhibitors  – pharmacology  – Macrophages  – Microglia

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lee, S. J. (2008). Transcriptional regulation of CD40 and class II MHC molecules in macrophages and microglia by statins. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,205

Chicago Manual of Style (16th Edition):

Lee, Sun Jung. “Transcriptional regulation of CD40 and class II MHC molecules in macrophages and microglia by statins.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,205.

MLA Handbook (7th Edition):

Lee, Sun Jung. “Transcriptional regulation of CD40 and class II MHC molecules in macrophages and microglia by statins.” 2008. Web. 19 Sep 2019.

Vancouver:

Lee SJ. Transcriptional regulation of CD40 and class II MHC molecules in macrophages and microglia by statins. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Sep 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,205.

Council of Science Editors:

Lee SJ. Transcriptional regulation of CD40 and class II MHC molecules in macrophages and microglia by statins. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,205

9. Yu, Jei-Hwa. MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1.

Degree: PhD, 2008, University of Alabama – Birmingham

Papillomaviruses (PV) are prevalent pathogens that infect human or animal squamous epithelia. Its genome is a double strand circular DNA of approximately 7.9 kb. It… (more)

Subjects/Keywords: DNA Helicases  – metabolism <; br>; DNA-Binding Proteins  – metabolism <; br>; Human papillomavirus 11  – physiology <; br>; Mitogen-Activated Protein Kinases  – metabolism <; br>; Nuclear Localization Signals  – metabolism <; br>; Replication Origin <; br>; Viral Proteins  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yu, J. (2008). MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,213

Chicago Manual of Style (16th Edition):

Yu, Jei-Hwa. “MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,213.

MLA Handbook (7th Edition):

Yu, Jei-Hwa. “MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1.” 2008. Web. 19 Sep 2019.

Vancouver:

Yu J. MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Sep 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,213.

Council of Science Editors:

Yu J. MAPKs regulate nuclear import of human papillomavirus type 11 replicative helicase E1. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,213

10. Lai, Yun-Ju. Role of TRIP6 in LPA-induced cell migration.

Degree: PhD, 2007, University of Alabama – Birmingham

The LIM domain-containing Thyroid Receptor-Interacting Protein 6 (TRIP6) is a zyxin family member that has been implicated in actin dynamics and cell motility. In this… (more)

Subjects/Keywords: Adaptor Proteins, Signal Transducing<; br>; Carrier Proteins  – physiology<; br>; Cell Movement<; br>; Feedback, Biochemical<; br>; Lysophospholipids<; br>; Protein-Tyrosine Kinases  – physiology<; br>; Receptors, G-Protein-Coupled  – metabolism<; br>; Transcription Factors

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lai, Y. (2007). Role of TRIP6 in LPA-induced cell migration. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,453

Chicago Manual of Style (16th Edition):

Lai, Yun-Ju. “Role of TRIP6 in LPA-induced cell migration.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,453.

MLA Handbook (7th Edition):

Lai, Yun-Ju. “Role of TRIP6 in LPA-induced cell migration.” 2007. Web. 19 Sep 2019.

Vancouver:

Lai Y. Role of TRIP6 in LPA-induced cell migration. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Sep 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,453.

Council of Science Editors:

Lai Y. Role of TRIP6 in LPA-induced cell migration. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,453

11. Lee, Seung-Ah. Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism.

Degree: PhD, 2008, University of Alabama – Birmingham

Retinol dehydrogenase 12 (RDH12) is a member of the microsomal short-chain dehydrogenase/reductase superfamily of proteins that is highly expressed in photorecep-tor cells. Mutations in RDH12… (more)

Subjects/Keywords: Alcohol Oxidoreductases  – metabolism<; br>; Genetic Diseases, Inborn  – enzymology<; br>; Lipid Peroxidation<; br>; Mutation, Missense<; br>; Photoreceptor Cells  – enzymology<; br>; Retinal Diseases  – enzymology<; br>; Retinaldehyde  – metabolism<; br>; Retinoids  – metabolism<; br>; Tretinoin  – metabolism

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lee, S. (2008). Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,814

Chicago Manual of Style (16th Edition):

Lee, Seung-Ah. “Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,814.

MLA Handbook (7th Edition):

Lee, Seung-Ah. “Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism.” 2008. Web. 19 Sep 2019.

Vancouver:

Lee S. Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2019 Sep 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,814.

Council of Science Editors:

Lee S. Function of human retinol dehydrogenase 12 (RDH12) in retinoid metabolism. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,814

12. Almonte, Antoine Gabriel. The role of protease-activated reseptor-1 in synaptic plasticity and memory.

Degree: PhD, 2011, University of Alabama – Birmingham

Protease-activated receptor-1 (PAR1) is an unusual G-protein coupled receptor (GPCR) that is activated through proteolytic cleavage by extracellular serine proteases. While previous work has shown… (more)

Subjects/Keywords: Association Learning  – physiology<; br>; Avoidance Learning  – physiology<; br>; Conditioning, Classical  – physiology<; br>; Hippocampus<; br>; Long-Term Potentiation<; br>; Receptor, PAR-1  – metabolism<; br>; Retention (Psychology)  – physiology<; br>; Serine Proteases

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Almonte, A. G. (2011). The role of protease-activated reseptor-1 in synaptic plasticity and memory. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,944

Chicago Manual of Style (16th Edition):

Almonte, Antoine Gabriel. “The role of protease-activated reseptor-1 in synaptic plasticity and memory.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,944.

MLA Handbook (7th Edition):

Almonte, Antoine Gabriel. “The role of protease-activated reseptor-1 in synaptic plasticity and memory.” 2011. Web. 19 Sep 2019.

Vancouver:

Almonte AG. The role of protease-activated reseptor-1 in synaptic plasticity and memory. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Sep 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,944.

Council of Science Editors:

Almonte AG. The role of protease-activated reseptor-1 in synaptic plasticity and memory. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,944

13. Ho, Shiuh-Rong. O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress.

Degree: PhD, 2010, University of Alabama – Birmingham

O-GlcNAcylation is an abundant and dynamic post-translational modification on serine and threonine residues of nuclear and cytoplasmic proteins. O-GlcNAc Transferase (OGT) and Nuclear Cytoplasmic O-GlcNAcase… (more)

Subjects/Keywords: Acetylglucosamine  – metabolism<; br>; Gene Expression Regulation<; br>; Oxidative Stress  – genetics<; br>; Transcription Factors  – metabolism<; br>; Transcription, Genetic

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ho, S. (2010). O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1162

Chicago Manual of Style (16th Edition):

Ho, Shiuh-Rong. “O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed September 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1162.

MLA Handbook (7th Edition):

Ho, Shiuh-Rong. “O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress.” 2010. Web. 19 Sep 2019.

Vancouver:

Ho S. O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Sep 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1162.

Council of Science Editors:

Ho S. O-GLCNAc transferase modulates JNK1 and FOXO4 transcription factor to resist acute oxidative stress. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1162

.