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You searched for +publisher:"University of Alabama – Birmingham" +contributor:("Darley-Usmar, Victor<br>"). Showing records 1 – 6 of 6 total matches.

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1. Isbell, T. Scott (Timothy Scott). Nitrite conversion to nitric oxide : biological mechaisms and therapeutic implications.

Degree: PhD, 2007, University of Alabama – Birmingham

Nitrite is a ubiquitous anion that until recently was thought to be an inert oxidation byproduct of nitric oxide, a critical signaling molecule required for… (more)

Subjects/Keywords: Cell Hypoxia<; br>; Nitric Oxide  – metabolism<; br>; Nitrites  – pharmacology<; br>; Reperfusion Injury  – physiopathology<; br>; Reperfusion Injury  – prevention & control<; br>; Sodium Nitrite  – pharmacology<; br>; Vasodilation

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APA (6th Edition):

Isbell, T. S. (. S. (2007). Nitrite conversion to nitric oxide : biological mechaisms and therapeutic implications. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,597

Chicago Manual of Style (16th Edition):

Isbell, T Scott (Timothy Scott). “Nitrite conversion to nitric oxide : biological mechaisms and therapeutic implications.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,597.

MLA Handbook (7th Edition):

Isbell, T Scott (Timothy Scott). “Nitrite conversion to nitric oxide : biological mechaisms and therapeutic implications.” 2007. Web. 19 Oct 2019.

Vancouver:

Isbell TS(S. Nitrite conversion to nitric oxide : biological mechaisms and therapeutic implications. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2019 Oct 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,597.

Council of Science Editors:

Isbell TS(S. Nitrite conversion to nitric oxide : biological mechaisms and therapeutic implications. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,597

2. Diers, Anne R. Regulation of redox signaling by lipid electrophiles in breast cancer.

Degree: PhD, 2009, University of Alabama – Birmingham

A number of steps in breast cancer progression and metastasis are regulated by redox signaling pathways. Electrophilic lipids such as 15-deoxy-[delta]12,14-prostaglandin J2 (15d-PGJ2) are mediators… (more)

Subjects/Keywords: Antineoplastic Agents  – pharmacology<; br>; Apoptosis  – drug effects<; br>; Breast Neoplasms<; br>; Mitochondria  – drug effects<; br>; Mitochondria  – metabolism<; br>; Prostaglandin D2  – analogs & derivatives

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APA (6th Edition):

Diers, A. R. (2009). Regulation of redox signaling by lipid electrophiles in breast cancer. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,908

Chicago Manual of Style (16th Edition):

Diers, Anne R. “Regulation of redox signaling by lipid electrophiles in breast cancer.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,908.

MLA Handbook (7th Edition):

Diers, Anne R. “Regulation of redox signaling by lipid electrophiles in breast cancer.” 2009. Web. 19 Oct 2019.

Vancouver:

Diers AR. Regulation of redox signaling by lipid electrophiles in breast cancer. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,908.

Council of Science Editors:

Diers AR. Regulation of redox signaling by lipid electrophiles in breast cancer. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,908

3. Vitturi, Dario A. Red blood cell and hemoglobin dependent modulation of reactive species metabolism: implications for vascular homeostasis.

Degree: PhD, 2010, University of Alabama – Birmingham

The recent discovery that red blood cells might mediate hypoxic blood flow together with accumulating evidence that suggests a role for the modulation of vascular… (more)

Subjects/Keywords: Anoxia  – blood<; br>; Antihypertensive Agents<; br>; Blood Substitutes<; br>; Erythrocytes  – metabolism<; br>; Hemoglobins  – metabolism<; br>; Hypertension  – drug therapy<; br>; Nitrites  – blood<; br>; Oxygen  – blood<; br>; Oxyhemoglobins  – metabolism

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APA (6th Edition):

Vitturi, D. A. (2010). Red blood cell and hemoglobin dependent modulation of reactive species metabolism: implications for vascular homeostasis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1004

Chicago Manual of Style (16th Edition):

Vitturi, Dario A. “Red blood cell and hemoglobin dependent modulation of reactive species metabolism: implications for vascular homeostasis.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1004.

MLA Handbook (7th Edition):

Vitturi, Dario A. “Red blood cell and hemoglobin dependent modulation of reactive species metabolism: implications for vascular homeostasis.” 2010. Web. 19 Oct 2019.

Vancouver:

Vitturi DA. Red blood cell and hemoglobin dependent modulation of reactive species metabolism: implications for vascular homeostasis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2019 Oct 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1004.

Council of Science Editors:

Vitturi DA. Red blood cell and hemoglobin dependent modulation of reactive species metabolism: implications for vascular homeostasis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1004

4. Harrison, Corey (Corey Michael). The role of mitochondrial oxidative stress in cardiovascular disease development.

Degree: PhD, 2011, University of Alabama – Birmingham

An estimated 70 million Americans have one or more types of cardiovascular disease (CVD). Although the sequence of events leading to CVD are still controversial,… (more)

Subjects/Keywords: Atherosclerosis  – metabolism<; br>; Atherosclerosis  – chemically induced<; br>; Mitochondria  – metabolism<; br>; Oxidative Stress  – physiology<; br>; Superoxide Dismutase  – metabolism<; br>; Tobacco Smoke Pollution  – adverse effects

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APA (6th Edition):

Harrison, C. (. M. (2011). The role of mitochondrial oxidative stress in cardiovascular disease development. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1043

Chicago Manual of Style (16th Edition):

Harrison, Corey (Corey Michael). “The role of mitochondrial oxidative stress in cardiovascular disease development.” 2011. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1043.

MLA Handbook (7th Edition):

Harrison, Corey (Corey Michael). “The role of mitochondrial oxidative stress in cardiovascular disease development.” 2011. Web. 19 Oct 2019.

Vancouver:

Harrison C(M. The role of mitochondrial oxidative stress in cardiovascular disease development. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2011. [cited 2019 Oct 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1043.

Council of Science Editors:

Harrison C(M. The role of mitochondrial oxidative stress in cardiovascular disease development. [Doctoral Dissertation]. University of Alabama – Birmingham; 2011. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1043

5. Byon, Chang Hyun. Oxidative stress-stimulated vascular calcification.

Degree: PhD, 2009, University of Alabama – Birmingham

Oxidative stress plays a critical role in pathogenesis of atherosclerosis. However, the effect of oxidative stress-induced molecular signaling in development of vascular calcification, a feature… (more)

Subjects/Keywords: Calcinosis  – metabolism<; br>; Core Binding Factor Alpha 1 Subunit  – metabolism<; br>; Hydrogen Peroxide  – metabolism<; br>; Muscle, smooth, Vascular  – metabolism<; br>; Oxidative Stress<; br>; Proto-Oncogene Proteins c-akt  – metabolism<; br>; Signal Transduction

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APA (6th Edition):

Byon, C. H. (2009). Oxidative stress-stimulated vascular calcification. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,662

Chicago Manual of Style (16th Edition):

Byon, Chang Hyun. “Oxidative stress-stimulated vascular calcification.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,662.

MLA Handbook (7th Edition):

Byon, Chang Hyun. “Oxidative stress-stimulated vascular calcification.” 2009. Web. 19 Oct 2019.

Vancouver:

Byon CH. Oxidative stress-stimulated vascular calcification. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2019 Oct 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,662.

Council of Science Editors:

Byon CH. Oxidative stress-stimulated vascular calcification. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,662

6. Stein, Asaf. The Hypometabolic Actions Of Hydrogen Sulfide In Mammalian Systems.

Degree: PhD, 2012, University of Alabama – Birmingham

In recent years, hydrogen sulfide (H2S) has been identified as a ubiquitous cell signaling molecule. In addition to its diverse physiological roles, H2S has emerged… (more)

Subjects/Keywords: Anoxia – metabolism. Hydrogen Sulfide – administration & dosage. Myocardium – metabolism Oxygen – metabolism Signal Transduction – drug effects Thiosulfate Sulfurtransferase.

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Stein, A. (2012). The Hypometabolic Actions Of Hydrogen Sulfide In Mammalian Systems. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1641

Chicago Manual of Style (16th Edition):

Stein, Asaf. “The Hypometabolic Actions Of Hydrogen Sulfide In Mammalian Systems.” 2012. Doctoral Dissertation, University of Alabama – Birmingham. Accessed October 19, 2019. http://contentdm.mhsl.uab.edu/u?/etd,1641.

MLA Handbook (7th Edition):

Stein, Asaf. “The Hypometabolic Actions Of Hydrogen Sulfide In Mammalian Systems.” 2012. Web. 19 Oct 2019.

Vancouver:

Stein A. The Hypometabolic Actions Of Hydrogen Sulfide In Mammalian Systems. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2012. [cited 2019 Oct 19]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1641.

Council of Science Editors:

Stein A. The Hypometabolic Actions Of Hydrogen Sulfide In Mammalian Systems. [Doctoral Dissertation]. University of Alabama – Birmingham; 2012. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1641

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