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You searched for +publisher:"University of Alabama – Birmingham" +contributor:("Chang, Chenbei<br>"). Showing records 1 – 20 of 20 total matches.

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1. Helton, Eric Scott. A role for p63 in the regulation of cell cycle progression and cell death.

Degree: PhD, 2007, University of Alabama – Birmingham

p63 is a member of the p53 family of transcription factors that is a critical regulator of epithelial development. Studies have shown that p63 does… (more)

Subjects/Keywords: Cell Cycle  – physiology <; br>; Cell Death  – physiology <; br>; Transcription Factors <; br>; Tumor Suppressor Protein p53  – physiology

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APA (6th Edition):

Helton, E. S. (2007). A role for p63 in the regulation of cell cycle progression and cell death. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,279

Chicago Manual of Style (16th Edition):

Helton, Eric Scott. “A role for p63 in the regulation of cell cycle progression and cell death.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 07, 2020. http://contentdm.mhsl.uab.edu/u?/etd,279.

MLA Handbook (7th Edition):

Helton, Eric Scott. “A role for p63 in the regulation of cell cycle progression and cell death.” 2007. Web. 07 Apr 2020.

Vancouver:

Helton ES. A role for p63 in the regulation of cell cycle progression and cell death. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Apr 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,279.

Council of Science Editors:

Helton ES. A role for p63 in the regulation of cell cycle progression and cell death. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,279

2. Yu, Wanfeng. Importance of tRNALys,3 structure and use in gag translation for primer selection required for replication of human immunodeficiency virus type I.

Degree: PhD, 2007, University of Alabama – Birmingham

The features of tRNALys,3 that dictate why human immunodeficiency virus exclu-sively selects this tRNA as the primer for initiation of reverse transcription is unknown. The… (more)

Subjects/Keywords: HIV-1  – genetics <; br>; HIV-1  – physiology <; br>; Mutation  – genetics <; br>; RNA, Transfer, Amino Acyl  – genetics <; br>; Virus Replication  – genetics

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APA (6th Edition):

Yu, W. (2007). Importance of tRNALys,3 structure and use in gag translation for primer selection required for replication of human immunodeficiency virus type I. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,306

Chicago Manual of Style (16th Edition):

Yu, Wanfeng. “Importance of tRNALys,3 structure and use in gag translation for primer selection required for replication of human immunodeficiency virus type I.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 07, 2020. http://contentdm.mhsl.uab.edu/u?/etd,306.

MLA Handbook (7th Edition):

Yu, Wanfeng. “Importance of tRNALys,3 structure and use in gag translation for primer selection required for replication of human immunodeficiency virus type I.” 2007. Web. 07 Apr 2020.

Vancouver:

Yu W. Importance of tRNALys,3 structure and use in gag translation for primer selection required for replication of human immunodeficiency virus type I. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Apr 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,306.

Council of Science Editors:

Yu W. Importance of tRNALys,3 structure and use in gag translation for primer selection required for replication of human immunodeficiency virus type I. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,306

3. Roarty, Kevin Patrick. The role of TGF-[beta] and Wnt5a in mammary gland development and tumorigenesis.

Degree: PhD, 2008, University of Alabama – Birmingham

Breast cancer is the second most common cancer worldwide behind lung cancer, affecting women of all ages, races, ethnicities, and socioeconomic strata. Concerted efforts have… (more)

Subjects/Keywords: Gene Expression Regulation, Developmental <; br>; Mammary Glands, Animal  – growth & development <; br>; Mammary Glands, Animal  – metabolism <; br>; Transforming Growth Factor beta1  – pharmacology <; br>; Wnt Proteins  – metabolism

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APA (6th Edition):

Roarty, K. P. (2008). The role of TGF-[beta] and Wnt5a in mammary gland development and tumorigenesis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,337

Chicago Manual of Style (16th Edition):

Roarty, Kevin Patrick. “The role of TGF-[beta] and Wnt5a in mammary gland development and tumorigenesis.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 07, 2020. http://contentdm.mhsl.uab.edu/u?/etd,337.

MLA Handbook (7th Edition):

Roarty, Kevin Patrick. “The role of TGF-[beta] and Wnt5a in mammary gland development and tumorigenesis.” 2008. Web. 07 Apr 2020.

Vancouver:

Roarty KP. The role of TGF-[beta] and Wnt5a in mammary gland development and tumorigenesis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Apr 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,337.

Council of Science Editors:

Roarty KP. The role of TGF-[beta] and Wnt5a in mammary gland development and tumorigenesis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,337

4. Seo, Hwa-Seon. The role of TGF[beta] signaling in skeletal development.

Degree: PhD, 2008, University of Alabama – Birmingham

Each skeletal element is essential for body movement along with the joints, muscles, tendons and ligaments. They also protect internal organs and serve as a… (more)

Subjects/Keywords: Bone and Bones  – abnormalities <; br>; Homeodomain Proteins  – genetics <; br>; Joints  – abnormalities <; br>; Mesoderm  – embryology <; br>; Protein-Serine-Threonine Kinases  – Metabolism <; br>; Receptors, Transforming Growth Factor beta  – metabolism <; br>; Transforming Growth Factor beta1  – physiology

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APA (6th Edition):

Seo, H. (2008). The role of TGF[beta] signaling in skeletal development. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,338

Chicago Manual of Style (16th Edition):

Seo, Hwa-Seon. “The role of TGF[beta] signaling in skeletal development.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 07, 2020. http://contentdm.mhsl.uab.edu/u?/etd,338.

MLA Handbook (7th Edition):

Seo, Hwa-Seon. “The role of TGF[beta] signaling in skeletal development.” 2008. Web. 07 Apr 2020.

Vancouver:

Seo H. The role of TGF[beta] signaling in skeletal development. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Apr 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,338.

Council of Science Editors:

Seo H. The role of TGF[beta] signaling in skeletal development. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,338

5. Deng, Luqin. Determinants of growth hormone receptor downregulation.

Degree: PhD, 2008, University of Alabama – Birmingham

Growth hormone (GH), a 22 kD polypeptide primarily produced in the anterior pituitary gland, is a key regulator of postnatal growth and affects carbohydrate, protein… (more)

Subjects/Keywords: Down-Regulation  – drug effects<; br>; Human Growth Hormone  – pharmacology<; br>; Janus Kinase 2  – chemistry<; br>; Janus Kinase 2  – genetics<; br>; Janus Kinase 2  – metabolism<; br>; Receptors, Somatotropin  – genetics<; br>; Receptors, Somatotropin  – metabolism

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APA (6th Edition):

Deng, L. (2008). Determinants of growth hormone receptor downregulation. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,389

Chicago Manual of Style (16th Edition):

Deng, Luqin. “Determinants of growth hormone receptor downregulation.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 07, 2020. http://contentdm.mhsl.uab.edu/u?/etd,389.

MLA Handbook (7th Edition):

Deng, Luqin. “Determinants of growth hormone receptor downregulation.” 2008. Web. 07 Apr 2020.

Vancouver:

Deng L. Determinants of growth hormone receptor downregulation. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Apr 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,389.

Council of Science Editors:

Deng L. Determinants of growth hormone receptor downregulation. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,389

6. Josyula, Ratnakar. Structural studies of yeast mitochondrial peripheral membrane protein TIM44.

Degree: PhD, 2009, University of Alabama – Birmingham

Tim44 is a peripheral membrane protein and a component of the TIM23 translocon on the matrix side. It is well established that Tim44 tethers the… (more)

Subjects/Keywords: Intracellular Membranes  – chemistry<; br>; Membrane Transport Proteins  – chemistry<; br>; Mitochondrial Membrane Transport Proteins  – chemistry<; br>; Mitochondrial Membranes  – chemistry<; br>; Models, Molecular<; br>; Saccharomyces cerevisiae Proteins  – chemistry

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APA (6th Edition):

Josyula, R. (2009). Structural studies of yeast mitochondrial peripheral membrane protein TIM44. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,395

Chicago Manual of Style (16th Edition):

Josyula, Ratnakar. “Structural studies of yeast mitochondrial peripheral membrane protein TIM44.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 07, 2020. http://contentdm.mhsl.uab.edu/u?/etd,395.

MLA Handbook (7th Edition):

Josyula, Ratnakar. “Structural studies of yeast mitochondrial peripheral membrane protein TIM44.” 2009. Web. 07 Apr 2020.

Vancouver:

Josyula R. Structural studies of yeast mitochondrial peripheral membrane protein TIM44. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Apr 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,395.

Council of Science Editors:

Josyula R. Structural studies of yeast mitochondrial peripheral membrane protein TIM44. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,395

7. Qian, Yingjuan, Ph.D. The role of DEC1 in P53-dependent cellular senescence.

Degree: PhD, 2008, University of Alabama – Birmingham

The p53 tumor suppressor is the most commonly mutated gene in human cancers. As a transcription factor, p53 exerts its tumor suppressor function through the… (more)

Subjects/Keywords: Basic Helix-Loop-Helix Transcription Factors  – genetics<; br>; Basic Helix-Loop-Helix Transcription Factors  – physiology<; br>; Cell Aging  – physiology<; br>; DNA Damage<; br>; Tumor Suppressor Protein p53  – physiology<; br>; Tumor Suppressor Proteins  – genetics

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APA (6th Edition):

Qian, Yingjuan, P. D. (2008). The role of DEC1 in P53-dependent cellular senescence. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,462

Chicago Manual of Style (16th Edition):

Qian, Yingjuan, Ph D. “The role of DEC1 in P53-dependent cellular senescence.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 07, 2020. http://contentdm.mhsl.uab.edu/u?/etd,462.

MLA Handbook (7th Edition):

Qian, Yingjuan, Ph D. “The role of DEC1 in P53-dependent cellular senescence.” 2008. Web. 07 Apr 2020.

Vancouver:

Qian, Yingjuan PD. The role of DEC1 in P53-dependent cellular senescence. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Apr 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,462.

Council of Science Editors:

Qian, Yingjuan PD. The role of DEC1 in P53-dependent cellular senescence. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,462

8. Wu, Yunkun. X-ray crystal structures of yeast heat shock proteins and mitochondrial outer membrane translocon member Tom70p.

Degree: PhD, 2007, University of Alabama – Birmingham

The molecular chaperones have special functions in mitochondrial protein translocation. For some mitochondrial preproteins, the protein targeting from the cytosolic ribosome to the surface of… (more)

Subjects/Keywords: Adenosine Triphosphatases  – metabolism<; br>; HSP70 Heat-Shock Proteins  – metabolism<; br>; Heat-Shock Proteins  – chemistry<; br>; Heat-Shock Proteins  – metabolism<; br>; Membrane Proteins  – chemistry<; br>; Peptide Fragments  – chemistry<; br>; Peptide Fragments  – metabolism<; br>; Saccharomyces cerevisiae  – chemistry<; br>; Saccharomyces cerevisiae Proteins  – chemistry<; br>; Saccharomyces cerevisiae Proteins  – metabolism

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APA (6th Edition):

Wu, Y. (2007). X-ray crystal structures of yeast heat shock proteins and mitochondrial outer membrane translocon member Tom70p. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,541

Chicago Manual of Style (16th Edition):

Wu, Yunkun. “X-ray crystal structures of yeast heat shock proteins and mitochondrial outer membrane translocon member Tom70p.” 2007. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 07, 2020. http://contentdm.mhsl.uab.edu/u?/etd,541.

MLA Handbook (7th Edition):

Wu, Yunkun. “X-ray crystal structures of yeast heat shock proteins and mitochondrial outer membrane translocon member Tom70p.” 2007. Web. 07 Apr 2020.

Vancouver:

Wu Y. X-ray crystal structures of yeast heat shock proteins and mitochondrial outer membrane translocon member Tom70p. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2007. [cited 2020 Apr 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,541.

Council of Science Editors:

Wu Y. X-ray crystal structures of yeast heat shock proteins and mitochondrial outer membrane translocon member Tom70p. [Doctoral Dissertation]. University of Alabama – Birmingham; 2007. Available from: http://contentdm.mhsl.uab.edu/u?/etd,541

9. Chang, Chia-Wei. Polycistronic lentiviral vector for hit and run reprogramming of mouse and human somatic cells to induced pluripotent stem cell.

Degree: PhD, 2009, University of Alabama – Birmingham

Embryonic stem (ES) cells are pluripotent and, therefore, can differentiate into most if not all somatic cell types. Because of this characteristic, ES cells have… (more)

Subjects/Keywords: Fibroblasts  – cytology<; br>; Genetic Vectors  – genetics<; br>; Lentivirus  – genetics<; br>; Nuclear Reprogramming  – genetics<; br>; Pluripotent Stem Cells  – cytology<; br>; Skin  – cytology

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APA (6th Edition):

Chang, C. (2009). Polycistronic lentiviral vector for hit and run reprogramming of mouse and human somatic cells to induced pluripotent stem cell. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,561

Chicago Manual of Style (16th Edition):

Chang, Chia-Wei. “Polycistronic lentiviral vector for hit and run reprogramming of mouse and human somatic cells to induced pluripotent stem cell.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 07, 2020. http://contentdm.mhsl.uab.edu/u?/etd,561.

MLA Handbook (7th Edition):

Chang, Chia-Wei. “Polycistronic lentiviral vector for hit and run reprogramming of mouse and human somatic cells to induced pluripotent stem cell.” 2009. Web. 07 Apr 2020.

Vancouver:

Chang C. Polycistronic lentiviral vector for hit and run reprogramming of mouse and human somatic cells to induced pluripotent stem cell. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Apr 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,561.

Council of Science Editors:

Chang C. Polycistronic lentiviral vector for hit and run reprogramming of mouse and human somatic cells to induced pluripotent stem cell. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,561

10. Li, Xin. Mechanism of the cross talk between growth hormone receptor and epidermal growth factor receptor.

Degree: PhD, 2008, University of Alabama – Birmingham

EGF receptor (EGFR) is a receptor tyrosine kinase, mediating cell growth from ectodermal and mesodermal origin. The overexpression and aberrant function of EGFR are involved… (more)

Subjects/Keywords: Growth Hormone  – physiology<; br>; Protein-Tyrosine Kinases  – metabolism<; br>; Receptor, Epidermal Growth Factor  – metabolism<; br>; Receptor Cross-Talk  – physiology

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APA (6th Edition):

Li, X. (2008). Mechanism of the cross talk between growth hormone receptor and epidermal growth factor receptor. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,605

Chicago Manual of Style (16th Edition):

Li, Xin. “Mechanism of the cross talk between growth hormone receptor and epidermal growth factor receptor.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 07, 2020. http://contentdm.mhsl.uab.edu/u?/etd,605.

MLA Handbook (7th Edition):

Li, Xin. “Mechanism of the cross talk between growth hormone receptor and epidermal growth factor receptor.” 2008. Web. 07 Apr 2020.

Vancouver:

Li X. Mechanism of the cross talk between growth hormone receptor and epidermal growth factor receptor. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Apr 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,605.

Council of Science Editors:

Li X. Mechanism of the cross talk between growth hormone receptor and epidermal growth factor receptor. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,605

11. Saxena, Ritu. Mechanisms and countermeasures of microgravity-induced bone loss: in vitro and in vivo model systems.

Degree: PhD, 2009, University of Alabama – Birmingham

Rapid bone loss occurs during prolonged periods of weightlessness experienced by astronauts during spaceflights which leads to osteopenia and increased fracture risk upon return to… (more)

Subjects/Keywords: Bone Resorption<; br>; Cell Differentiation<; br>; Osteoblasts  – cytology<; br>; Osteoclasts  – cytology<; br>; Parathyroid Hormone<; br>; Weightlessness

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APA (6th Edition):

Saxena, R. (2009). Mechanisms and countermeasures of microgravity-induced bone loss: in vitro and in vivo model systems. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,689

Chicago Manual of Style (16th Edition):

Saxena, Ritu. “Mechanisms and countermeasures of microgravity-induced bone loss: in vitro and in vivo model systems.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 07, 2020. http://contentdm.mhsl.uab.edu/u?/etd,689.

MLA Handbook (7th Edition):

Saxena, Ritu. “Mechanisms and countermeasures of microgravity-induced bone loss: in vitro and in vivo model systems.” 2009. Web. 07 Apr 2020.

Vancouver:

Saxena R. Mechanisms and countermeasures of microgravity-induced bone loss: in vitro and in vivo model systems. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Apr 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,689.

Council of Science Editors:

Saxena R. Mechanisms and countermeasures of microgravity-induced bone loss: in vitro and in vivo model systems. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,689

12. Kim, Nam Chul. Identification and characterization of downstream target genes of the BMP signaling pathway.

Degree: PhD, 2010, University of Alabama – Birmingham

During nervous system development, neurons proliferate, differentiate, project their axons to their targets and make synapses with them. At the neuromuscular junction of Drosophila, these… (more)

Subjects/Keywords: Bone Morphogenetic Proteins  – metabolism<; br>; Central Nervous System<; br>; Drosophila melanogaster<; br>; Gene Expression Regulation, Developmental<; br>; Microarray Analysis<; br>; Signal Transduction  – physiology

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APA (6th Edition):

Kim, N. C. (2010). Identification and characterization of downstream target genes of the BMP signaling pathway. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,919

Chicago Manual of Style (16th Edition):

Kim, Nam Chul. “Identification and characterization of downstream target genes of the BMP signaling pathway.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 07, 2020. http://contentdm.mhsl.uab.edu/u?/etd,919.

MLA Handbook (7th Edition):

Kim, Nam Chul. “Identification and characterization of downstream target genes of the BMP signaling pathway.” 2010. Web. 07 Apr 2020.

Vancouver:

Kim NC. Identification and characterization of downstream target genes of the BMP signaling pathway. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Apr 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,919.

Council of Science Editors:

Kim NC. Identification and characterization of downstream target genes of the BMP signaling pathway. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,919

13. Cui, Wenjun. Structural and mechanistic studies on ER UPR sensor PERK and mitochondrial translocon element TIM44.

Degree: PhD, 2010, University of Alabama – Birmingham

The unfolded protein response is one mechanism utilized by endoplasmic reticulum (ER) to maintain the homeostasis between ER protein folding machinery and ER proteins. UPR… (more)

Subjects/Keywords: Mitochondrial Membrane Transport Proteins  – chemistry<; br>; Mitochondrial Membrane Transport Proteins  – metabolism<; br>; Mitochondrial Membranes  – metabolism<; br>; Saccharomyces cerevisiae  – metabolism<; br>; Saccharomyces cerevisiae Proteins  – chemistry<; br>; Saccharomyces cerevisiae Proteins  – metabolism<; br>; Unfolded Protein Response

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APA (6th Edition):

Cui, W. (2010). Structural and mechanistic studies on ER UPR sensor PERK and mitochondrial translocon element TIM44. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1088

Chicago Manual of Style (16th Edition):

Cui, Wenjun. “Structural and mechanistic studies on ER UPR sensor PERK and mitochondrial translocon element TIM44.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 07, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1088.

MLA Handbook (7th Edition):

Cui, Wenjun. “Structural and mechanistic studies on ER UPR sensor PERK and mitochondrial translocon element TIM44.” 2010. Web. 07 Apr 2020.

Vancouver:

Cui W. Structural and mechanistic studies on ER UPR sensor PERK and mitochondrial translocon element TIM44. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Apr 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1088.

Council of Science Editors:

Cui W. Structural and mechanistic studies on ER UPR sensor PERK and mitochondrial translocon element TIM44. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1088

14. Joo, Heui Yun. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.

Degree: PhD, 2009, University of Alabama – Birmingham

Posttranslational modifications of histones regulate important chromatin and cellular functions. Among them, ubiquitination of histone H2A is correlated to transcriptional repression, such as HOX gene… (more)

Subjects/Keywords: Chromatin  – physiology<; br>; Endopeptidases  – metabolism<; br>; Histones  – metabolism<; br>; Ubiquitin Thiolesterase  – metabolism<; br>; Xenopus Proteins  – metabolism<; br>; Xenopus laevis  – embryology

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APA (6th Edition):

Joo, H. Y. (2009). Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1101

Chicago Manual of Style (16th Edition):

Joo, Heui Yun. “Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 07, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1101.

MLA Handbook (7th Edition):

Joo, Heui Yun. “Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions.” 2009. Web. 07 Apr 2020.

Vancouver:

Joo HY. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Apr 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1101.

Council of Science Editors:

Joo HY. Understanding the regulatory mechanisms of UBP-M and H2A deubiquitination in chromatin and cellular functions. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1101

15. Yan, Ming. The structural and functional studies of yeast nucleotide exchange factor Sil1P and its complex with Bip.

Degree: PhD, 2010, University of Alabama – Birmingham

Sil1 is an Endoplasmic Reticulum (ER) localized protein. SIL1 was initially identified as a UPR-regulated gene. Later studies show Sil1 functions as the nucleotide exchange… (more)

Subjects/Keywords: Fungal Proteins  – chemistry<; br>; HSP70 Heat-Shock Proteins  – chemistry<; br>; Membrane Transport Proteins  – chemistry<; br>; Saccharomyces cerevisiae  – metabolism<; br>; Saccharomyces cerevisiae Proteins  – chemistry

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yan, M. (2010). The structural and functional studies of yeast nucleotide exchange factor Sil1P and its complex with Bip. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1115

Chicago Manual of Style (16th Edition):

Yan, Ming. “The structural and functional studies of yeast nucleotide exchange factor Sil1P and its complex with Bip.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 07, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1115.

MLA Handbook (7th Edition):

Yan, Ming. “The structural and functional studies of yeast nucleotide exchange factor Sil1P and its complex with Bip.” 2010. Web. 07 Apr 2020.

Vancouver:

Yan M. The structural and functional studies of yeast nucleotide exchange factor Sil1P and its complex with Bip. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Apr 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1115.

Council of Science Editors:

Yan M. The structural and functional studies of yeast nucleotide exchange factor Sil1P and its complex with Bip. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1115

16. Moore, Lakisha Dionne. Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis.

Degree: PhD, 2008, University of Alabama – Birmingham

Overexpresssion of transforming growth factor (TGF)-[beta] has been implicated in promoting immune suppression, tumor angiogenesis, tumor cell migration, and invasion in many cancers including carcinoma… (more)

Subjects/Keywords: Breast Neoplasms  – genetics <; br>; Neoplasm Metastasis <; br>; RNA Interference <; br>; Transforming Growth Factor beta1  – genetics <; br>; Transforming Growth Factor beta1  – metabolism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Moore, L. D. (2008). Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,233

Chicago Manual of Style (16th Edition):

Moore, Lakisha Dionne. “Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis.” 2008. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 07, 2020. http://contentdm.mhsl.uab.edu/u?/etd,233.

MLA Handbook (7th Edition):

Moore, Lakisha Dionne. “Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis.” 2008. Web. 07 Apr 2020.

Vancouver:

Moore LD. Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2008. [cited 2020 Apr 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,233.

Council of Science Editors:

Moore LD. Modulation of Transforming Growth Factor (TGF)-[beta]1 and its implications in breast cancer metastasis. [Doctoral Dissertation]. University of Alabama – Birmingham; 2008. Available from: http://contentdm.mhsl.uab.edu/u?/etd,233

17. Akhtar, Lisa Nowoslawski. The role of SOCS proteins in HIV immune evasion.

Degree: PhD, 2010, University of Alabama – Birmingham

Upon human immunodeficiency virus (HIV) infection the host mounts a robust and multifaceted immune response. To achieve successful replication, HIV must possess a powerful arsenal… (more)

Subjects/Keywords: Central Nervous System  – immunology<; br>; Cytokines  – immunology<; br>; HIV Infections  – immunology<; br>; Suppressor of Cytokine Signaling Proteins  – metabolism

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Akhtar, L. N. (2010). The role of SOCS proteins in HIV immune evasion. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,640

Chicago Manual of Style (16th Edition):

Akhtar, Lisa Nowoslawski. “The role of SOCS proteins in HIV immune evasion.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 07, 2020. http://contentdm.mhsl.uab.edu/u?/etd,640.

MLA Handbook (7th Edition):

Akhtar, Lisa Nowoslawski. “The role of SOCS proteins in HIV immune evasion.” 2010. Web. 07 Apr 2020.

Vancouver:

Akhtar LN. The role of SOCS proteins in HIV immune evasion. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Apr 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,640.

Council of Science Editors:

Akhtar LN. The role of SOCS proteins in HIV immune evasion. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,640

18. Byon, Chang Hyun. Oxidative stress-stimulated vascular calcification.

Degree: PhD, 2009, University of Alabama – Birmingham

Oxidative stress plays a critical role in pathogenesis of atherosclerosis. However, the effect of oxidative stress-induced molecular signaling in development of vascular calcification, a feature… (more)

Subjects/Keywords: Calcinosis  – metabolism<; br>; Core Binding Factor Alpha 1 Subunit  – metabolism<; br>; Hydrogen Peroxide  – metabolism<; br>; Muscle, smooth, Vascular  – metabolism<; br>; Oxidative Stress<; br>; Proto-Oncogene Proteins c-akt  – metabolism<; br>; Signal Transduction

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Byon, C. H. (2009). Oxidative stress-stimulated vascular calcification. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,662

Chicago Manual of Style (16th Edition):

Byon, Chang Hyun. “Oxidative stress-stimulated vascular calcification.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 07, 2020. http://contentdm.mhsl.uab.edu/u?/etd,662.

MLA Handbook (7th Edition):

Byon, Chang Hyun. “Oxidative stress-stimulated vascular calcification.” 2009. Web. 07 Apr 2020.

Vancouver:

Byon CH. Oxidative stress-stimulated vascular calcification. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Apr 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,662.

Council of Science Editors:

Byon CH. Oxidative stress-stimulated vascular calcification. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,662

19. Atkinson, George P. (George Prescott). The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma.

Degree: PhD, 2009, University of Alabama – Birmingham

Glioblastoma (GBM) is an incurable tumor of the central nervous system (CNS). Over the past 50 years, little progress has made in improving the quality… (more)

Subjects/Keywords: Glioblastoma  – pathology<; br>; Neoplasms<; br>; NF-kappa B  – metabolism<; br>; Peptidylprolyl Isomerase<; br>; STAT3 Transcription Factor

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APA (6th Edition):

Atkinson, G. P. (. P. (2009). The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,812

Chicago Manual of Style (16th Edition):

Atkinson, George P (George Prescott). “The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma.” 2009. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 07, 2020. http://contentdm.mhsl.uab.edu/u?/etd,812.

MLA Handbook (7th Edition):

Atkinson, George P (George Prescott). “The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma.” 2009. Web. 07 Apr 2020.

Vancouver:

Atkinson GP(P. The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2009. [cited 2020 Apr 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,812.

Council of Science Editors:

Atkinson GP(P. The peptidyl-prolyl isomerase Pin1 promotes NF-[kappa]B and STAT3 signaling in glioblastoma. [Doctoral Dissertation]. University of Alabama – Birmingham; 2009. Available from: http://contentdm.mhsl.uab.edu/u?/etd,812

20. Yang, Youfeng. The Role Of Map Kinase Cascade In Msp Signaling Response.

Degree: PhD, 2010, University of Alabama – Birmingham

The MSP domain is an evolutionarily conserved immunoglobulin-like structure of about 120 amino acids (Miller et al., 2001). A P56S missense mutation in the MSP… (more)

Subjects/Keywords: Caenorhabditis elegans – metabolism.<; br>; Helminth Proteins – physiology.<; br>; MAP Kinase Signaling System – physiology.<; br>; Mitogen-Activated Protein Kinases – metabolism.<; br>; Oocytes – physiology<; br>; Reactive Oxygen Species – metabolism<; br>; Signal Transduction – physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yang, Y. (2010). The Role Of Map Kinase Cascade In Msp Signaling Response. (Doctoral Dissertation). University of Alabama – Birmingham. Retrieved from http://contentdm.mhsl.uab.edu/u?/etd,1399

Chicago Manual of Style (16th Edition):

Yang, Youfeng. “The Role Of Map Kinase Cascade In Msp Signaling Response.” 2010. Doctoral Dissertation, University of Alabama – Birmingham. Accessed April 07, 2020. http://contentdm.mhsl.uab.edu/u?/etd,1399.

MLA Handbook (7th Edition):

Yang, Youfeng. “The Role Of Map Kinase Cascade In Msp Signaling Response.” 2010. Web. 07 Apr 2020.

Vancouver:

Yang Y. The Role Of Map Kinase Cascade In Msp Signaling Response. [Internet] [Doctoral dissertation]. University of Alabama – Birmingham; 2010. [cited 2020 Apr 07]. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1399.

Council of Science Editors:

Yang Y. The Role Of Map Kinase Cascade In Msp Signaling Response. [Doctoral Dissertation]. University of Alabama – Birmingham; 2010. Available from: http://contentdm.mhsl.uab.edu/u?/etd,1399

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