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You searched for +publisher:"Università degli Studi di Milano" +contributor:("LOCATI, MASSIMO"). Showing records 1 – 30 of 62 total matches.

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1. T.A. Renzi. ROLE OF MICRORNA IN THE REGULATION OF TLR SIGNALING PATHWAY.

Degree: 2015, Università degli Studi di Milano

 Toll-like receptors (TLRs) play key roles in detecting pathogens and initiating inflammatory responses via the activation of specific signaling pathways. The TLRs activity must be… (more)

Subjects/Keywords: microRNA; TLR; LPS; miR-125a; miR-99; let-7e; miR-146b; IL-10; TGFβ; THP-1; IL-10 knockout; anti-inflammatory; endotoxin tolerance; Settore MED/04 - Patologia Generale; Settore BIO/11 - Biologia Molecolare

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Renzi, T. (2015). ROLE OF MICRORNA IN THE REGULATION OF TLR SIGNALING PATHWAY. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/254528

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Renzi, T.A.. “ROLE OF MICRORNA IN THE REGULATION OF TLR SIGNALING PATHWAY.” 2015. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/254528.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Renzi, T.A.. “ROLE OF MICRORNA IN THE REGULATION OF TLR SIGNALING PATHWAY.” 2015. Web. 16 Jan 2021.

Vancouver:

Renzi T. ROLE OF MICRORNA IN THE REGULATION OF TLR SIGNALING PATHWAY. [Internet] [Thesis]. Università degli Studi di Milano; 2015. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/254528.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Renzi T. ROLE OF MICRORNA IN THE REGULATION OF TLR SIGNALING PATHWAY. [Thesis]. Università degli Studi di Milano; 2015. Available from: http://hdl.handle.net/2434/254528

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

2. C.B. Chighizola. THE PATHOGENIC ROLE OF IMMUNE COMPLEXES CONTAINING SCLERODERMA-SPECIFIC AUTOANTIBODIES.

Degree: 2015, Università degli Studi di Milano

 AIM OF THE STUDY Systemic sclerosis (SSc) is a chronic autoimmune condition characterized by excessive tissue fibrosis, microvascular alterations and immune dysfunction with the production… (more)

Subjects/Keywords: Settore MED/16 - Reumatologia

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APA (6th Edition):

Chighizola, C. (2015). THE PATHOGENIC ROLE OF IMMUNE COMPLEXES CONTAINING SCLERODERMA-SPECIFIC AUTOANTIBODIES. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/259694

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chighizola, C.B.. “THE PATHOGENIC ROLE OF IMMUNE COMPLEXES CONTAINING SCLERODERMA-SPECIFIC AUTOANTIBODIES.” 2015. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/259694.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chighizola, C.B.. “THE PATHOGENIC ROLE OF IMMUNE COMPLEXES CONTAINING SCLERODERMA-SPECIFIC AUTOANTIBODIES.” 2015. Web. 16 Jan 2021.

Vancouver:

Chighizola C. THE PATHOGENIC ROLE OF IMMUNE COMPLEXES CONTAINING SCLERODERMA-SPECIFIC AUTOANTIBODIES. [Internet] [Thesis]. Università degli Studi di Milano; 2015. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/259694.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chighizola C. THE PATHOGENIC ROLE OF IMMUNE COMPLEXES CONTAINING SCLERODERMA-SPECIFIC AUTOANTIBODIES. [Thesis]. Università degli Studi di Milano; 2015. Available from: http://hdl.handle.net/2434/259694

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

3. F. Tomay. REGULATION AND FUNCTION OF THE TETRASPANIN-LIKE MOLECULE MS4A4A IN ALTERNATIVELY ACTIVATED AND TUMOR-ASSOCIATED MACROPHAGES.

Degree: 2015, Università degli Studi di Milano

 Macrophages are a heterogeneous population of the innate immune system operating at the boundary between health and disease. They represent the most plastic elements of… (more)

Subjects/Keywords: Macrophages; Tumor Associated Macrophages (TAM); Membrane Spanning 4-domains subfamily A (MS4A); Tetraspanins; Lipid rafts; Hodgkin Lymphoma; monoclonal antibodies; Dectin1; beta-glucans; Settore MED/04 - Patologia Generale

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APA (6th Edition):

Tomay, F. (2015). REGULATION AND FUNCTION OF THE TETRASPANIN-LIKE MOLECULE MS4A4A IN ALTERNATIVELY ACTIVATED AND TUMOR-ASSOCIATED MACROPHAGES. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/248877

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tomay, F.. “REGULATION AND FUNCTION OF THE TETRASPANIN-LIKE MOLECULE MS4A4A IN ALTERNATIVELY ACTIVATED AND TUMOR-ASSOCIATED MACROPHAGES.” 2015. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/248877.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tomay, F.. “REGULATION AND FUNCTION OF THE TETRASPANIN-LIKE MOLECULE MS4A4A IN ALTERNATIVELY ACTIVATED AND TUMOR-ASSOCIATED MACROPHAGES.” 2015. Web. 16 Jan 2021.

Vancouver:

Tomay F. REGULATION AND FUNCTION OF THE TETRASPANIN-LIKE MOLECULE MS4A4A IN ALTERNATIVELY ACTIVATED AND TUMOR-ASSOCIATED MACROPHAGES. [Internet] [Thesis]. Università degli Studi di Milano; 2015. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/248877.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tomay F. REGULATION AND FUNCTION OF THE TETRASPANIN-LIKE MOLECULE MS4A4A IN ALTERNATIVELY ACTIVATED AND TUMOR-ASSOCIATED MACROPHAGES. [Thesis]. Università degli Studi di Milano; 2015. Available from: http://hdl.handle.net/2434/248877

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

4. S. Gentile. THE PROTECTIVE ROLE OF PTX3 IN MOUSE SKIN CARCINOGENESIS.

Degree: 2015, Università degli Studi di Milano

 Abstract The implication of the innate immune system on inflammatory carcinogenesis is a central topic in tumor biology. The humoral pattern recognition molecule PTX3 plays… (more)

Subjects/Keywords: PTX3; DMBA/TPA-induced skin cancer; cancer-related inflammation; Settore MED/04 - Patologia Generale

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APA (6th Edition):

Gentile, S. (2015). THE PROTECTIVE ROLE OF PTX3 IN MOUSE SKIN CARCINOGENESIS. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/251045

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gentile, S.. “THE PROTECTIVE ROLE OF PTX3 IN MOUSE SKIN CARCINOGENESIS.” 2015. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/251045.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gentile, S.. “THE PROTECTIVE ROLE OF PTX3 IN MOUSE SKIN CARCINOGENESIS.” 2015. Web. 16 Jan 2021.

Vancouver:

Gentile S. THE PROTECTIVE ROLE OF PTX3 IN MOUSE SKIN CARCINOGENESIS. [Internet] [Thesis]. Università degli Studi di Milano; 2015. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/251045.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gentile S. THE PROTECTIVE ROLE OF PTX3 IN MOUSE SKIN CARCINOGENESIS. [Thesis]. Università degli Studi di Milano; 2015. Available from: http://hdl.handle.net/2434/251045

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

5. N.G. Sukubo. NO CODING RNAs IN MACROPHAGE POLARIZATION: THE RELEVANCE OF THE "JUNK" RNA.

Degree: 2016, Università degli Studi di Milano

 Abstract Form the central dogma of the biology formulated by Crick in 1958, to the detection of “junk RNA”, a new prospective has been open.… (more)

Subjects/Keywords: macrophages; miRNAs; miR-135b; polarization; long non-coding RNA; MSU; Polycomb 2; gouty arthritis; IL-1β pathway; Settore MED/04 - Patologia Generale

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APA (6th Edition):

Sukubo, N. (2016). NO CODING RNAs IN MACROPHAGE POLARIZATION: THE RELEVANCE OF THE "JUNK" RNA. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/365870

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sukubo, N.G.. “NO CODING RNAs IN MACROPHAGE POLARIZATION: THE RELEVANCE OF THE "JUNK" RNA.” 2016. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/365870.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sukubo, N.G.. “NO CODING RNAs IN MACROPHAGE POLARIZATION: THE RELEVANCE OF THE "JUNK" RNA.” 2016. Web. 16 Jan 2021.

Vancouver:

Sukubo N. NO CODING RNAs IN MACROPHAGE POLARIZATION: THE RELEVANCE OF THE "JUNK" RNA. [Internet] [Thesis]. Università degli Studi di Milano; 2016. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/365870.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sukubo N. NO CODING RNAs IN MACROPHAGE POLARIZATION: THE RELEVANCE OF THE "JUNK" RNA. [Thesis]. Università degli Studi di Milano; 2016. Available from: http://hdl.handle.net/2434/365870

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

6. S. GONZALVO FEO. CCRL2, A PUTATIVE CHEMOTACTIC RECEPTOR, PARTICIPATES IN THE LUNG DENDRITIC CELL TRAFFICKING.

Degree: 2010, Università degli Studi di Milano

 CCRL2 (chemokine CC motif receptor-like 2) is an orphan heptahelic serpentine receptor that is rapidly induced in dendritic cells (DC) during maturation. CCRL2 shares the… (more)

Subjects/Keywords: CCRL2; chemerin; ChemR23; dendritic cell; asthma; airway hyperresponsiveness; endothelial cell; dendritic cell traffic; Settore MED/04 - Patologia Generale

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APA (6th Edition):

FEO, S. G. (2010). CCRL2, A PUTATIVE CHEMOTACTIC RECEPTOR, PARTICIPATES IN THE LUNG DENDRITIC CELL TRAFFICKING. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/150132

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

FEO, S. GONZALVO. “CCRL2, A PUTATIVE CHEMOTACTIC RECEPTOR, PARTICIPATES IN THE LUNG DENDRITIC CELL TRAFFICKING.” 2010. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/150132.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

FEO, S. GONZALVO. “CCRL2, A PUTATIVE CHEMOTACTIC RECEPTOR, PARTICIPATES IN THE LUNG DENDRITIC CELL TRAFFICKING.” 2010. Web. 16 Jan 2021.

Vancouver:

FEO SG. CCRL2, A PUTATIVE CHEMOTACTIC RECEPTOR, PARTICIPATES IN THE LUNG DENDRITIC CELL TRAFFICKING. [Internet] [Thesis]. Università degli Studi di Milano; 2010. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/150132.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

FEO SG. CCRL2, A PUTATIVE CHEMOTACTIC RECEPTOR, PARTICIPATES IN THE LUNG DENDRITIC CELL TRAFFICKING. [Thesis]. Università degli Studi di Milano; 2010. Available from: http://hdl.handle.net/2434/150132

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

7. F. Cavaciocchi. T CELL SUBTYPES IN MANAGEMENT OF OSTEOPOROSIS WITH BISPHOSPHONATES AND THE AUTOIMMUNE REACTION TO THE COLLAGEN IN RHEUMATOID ARTHRITIS.

Degree: 2015, Università degli Studi di Milano

 T lymphocytes with different T cell receptors are at the crossroad of inflammation and autoimmunity. We investigated the role of γδTCR and αβTCR T lymphocytes… (more)

Subjects/Keywords: Osteoporosis; zoledronic acid; γδTCR T lymphocytes; rheumatoid arthritis; human collagen epitopes; citrullinations; carbamylation; Settore MED/16 - Reumatologia; Settore MED/04 - Patologia Generale

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APA (6th Edition):

Cavaciocchi, F. (2015). T CELL SUBTYPES IN MANAGEMENT OF OSTEOPOROSIS WITH BISPHOSPHONATES AND THE AUTOIMMUNE REACTION TO THE COLLAGEN IN RHEUMATOID ARTHRITIS. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/251417

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cavaciocchi, F.. “T CELL SUBTYPES IN MANAGEMENT OF OSTEOPOROSIS WITH BISPHOSPHONATES AND THE AUTOIMMUNE REACTION TO THE COLLAGEN IN RHEUMATOID ARTHRITIS.” 2015. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/251417.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cavaciocchi, F.. “T CELL SUBTYPES IN MANAGEMENT OF OSTEOPOROSIS WITH BISPHOSPHONATES AND THE AUTOIMMUNE REACTION TO THE COLLAGEN IN RHEUMATOID ARTHRITIS.” 2015. Web. 16 Jan 2021.

Vancouver:

Cavaciocchi F. T CELL SUBTYPES IN MANAGEMENT OF OSTEOPOROSIS WITH BISPHOSPHONATES AND THE AUTOIMMUNE REACTION TO THE COLLAGEN IN RHEUMATOID ARTHRITIS. [Internet] [Thesis]. Università degli Studi di Milano; 2015. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/251417.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cavaciocchi F. T CELL SUBTYPES IN MANAGEMENT OF OSTEOPOROSIS WITH BISPHOSPHONATES AND THE AUTOIMMUNE REACTION TO THE COLLAGEN IN RHEUMATOID ARTHRITIS. [Thesis]. Università degli Studi di Milano; 2015. Available from: http://hdl.handle.net/2434/251417

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. S. Muggeo. A STEP-BY-STEP PROCESS TO GENERATE FUNCTIONAL OSTEOCLASTS FROM SITE SPECIFIC GENE-CORRECTED INDUCED PLURIPOTENT STEM CELLS: AN AUTOLOGOUS CELL THERAPY APPROACH TO TREAT AUTOSOMAL RECESSIVE OSTEOPETROSIS.

Degree: 2015, Università degli Studi di Milano

 Autosomal recessive osteopetrosis (ARO) is a human severe inherited disorder leading to increased bone mass, decreased bone strength with risk of multiple fractures and progressive… (more)

Subjects/Keywords: induced pluripotent stem cells; hematopoiesis; osteopetrosis; Settore MED/03 - Genetica Medica; Settore MED/04 - Patologia Generale; Settore MED/05 - Patologia Clinica

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APA (6th Edition):

Muggeo, S. (2015). A STEP-BY-STEP PROCESS TO GENERATE FUNCTIONAL OSTEOCLASTS FROM SITE SPECIFIC GENE-CORRECTED INDUCED PLURIPOTENT STEM CELLS: AN AUTOLOGOUS CELL THERAPY APPROACH TO TREAT AUTOSOMAL RECESSIVE OSTEOPETROSIS. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/252730

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Muggeo, S.. “A STEP-BY-STEP PROCESS TO GENERATE FUNCTIONAL OSTEOCLASTS FROM SITE SPECIFIC GENE-CORRECTED INDUCED PLURIPOTENT STEM CELLS: AN AUTOLOGOUS CELL THERAPY APPROACH TO TREAT AUTOSOMAL RECESSIVE OSTEOPETROSIS.” 2015. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/252730.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Muggeo, S.. “A STEP-BY-STEP PROCESS TO GENERATE FUNCTIONAL OSTEOCLASTS FROM SITE SPECIFIC GENE-CORRECTED INDUCED PLURIPOTENT STEM CELLS: AN AUTOLOGOUS CELL THERAPY APPROACH TO TREAT AUTOSOMAL RECESSIVE OSTEOPETROSIS.” 2015. Web. 16 Jan 2021.

Vancouver:

Muggeo S. A STEP-BY-STEP PROCESS TO GENERATE FUNCTIONAL OSTEOCLASTS FROM SITE SPECIFIC GENE-CORRECTED INDUCED PLURIPOTENT STEM CELLS: AN AUTOLOGOUS CELL THERAPY APPROACH TO TREAT AUTOSOMAL RECESSIVE OSTEOPETROSIS. [Internet] [Thesis]. Università degli Studi di Milano; 2015. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/252730.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Muggeo S. A STEP-BY-STEP PROCESS TO GENERATE FUNCTIONAL OSTEOCLASTS FROM SITE SPECIFIC GENE-CORRECTED INDUCED PLURIPOTENT STEM CELLS: AN AUTOLOGOUS CELL THERAPY APPROACH TO TREAT AUTOSOMAL RECESSIVE OSTEOPETROSIS. [Thesis]. Università degli Studi di Milano; 2015. Available from: http://hdl.handle.net/2434/252730

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

9. O. Bonavita. DOWN-REGULATION OF ATYPICAL CHEMOKINE RECEPTOR ACKR2/D6 EXPRESSION BY HEMATOPOIETIC PROGENITORS PROMOTES MYELOID CELL MOBILIZATION AND DIFFERENTIATION.

Degree: 2017, Università degli Studi di Milano

 Chemokines and chemokine receptors are key mediators of inflammation and important regulators of leukocyte migration in homeostatic conditions as well as during infection and cancer.… (more)

Subjects/Keywords: Settore MED/04 - Patologia Generale

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APA (6th Edition):

Bonavita, O. (2017). DOWN-REGULATION OF ATYPICAL CHEMOKINE RECEPTOR ACKR2/D6 EXPRESSION BY HEMATOPOIETIC PROGENITORS PROMOTES MYELOID CELL MOBILIZATION AND DIFFERENTIATION. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/488818

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bonavita, O.. “DOWN-REGULATION OF ATYPICAL CHEMOKINE RECEPTOR ACKR2/D6 EXPRESSION BY HEMATOPOIETIC PROGENITORS PROMOTES MYELOID CELL MOBILIZATION AND DIFFERENTIATION.” 2017. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/488818.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bonavita, O.. “DOWN-REGULATION OF ATYPICAL CHEMOKINE RECEPTOR ACKR2/D6 EXPRESSION BY HEMATOPOIETIC PROGENITORS PROMOTES MYELOID CELL MOBILIZATION AND DIFFERENTIATION.” 2017. Web. 16 Jan 2021.

Vancouver:

Bonavita O. DOWN-REGULATION OF ATYPICAL CHEMOKINE RECEPTOR ACKR2/D6 EXPRESSION BY HEMATOPOIETIC PROGENITORS PROMOTES MYELOID CELL MOBILIZATION AND DIFFERENTIATION. [Internet] [Thesis]. Università degli Studi di Milano; 2017. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/488818.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bonavita O. DOWN-REGULATION OF ATYPICAL CHEMOKINE RECEPTOR ACKR2/D6 EXPRESSION BY HEMATOPOIETIC PROGENITORS PROMOTES MYELOID CELL MOBILIZATION AND DIFFERENTIATION. [Thesis]. Università degli Studi di Milano; 2017. Available from: http://hdl.handle.net/2434/488818

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

10. G. Marelli. ROLE OF INTESTINAL CX3CR1+ MACROPHAGES IN THE DEVELOPMENT OF COLITIS-ASSOCIATED CANCER: FOCUS ON THE RELEVANCE OF THE MICROBIOME AND HEME-OXYGENASE-1.

Degree: 2016, Università degli Studi di Milano

 Gut represents a complex landscape in which commensal bacteria, harmless antigens and food proteins are strictly in contact with the immune system that has to… (more)

Subjects/Keywords: Macrophages; microbiota; mucosal immunology; colorectal cancer; inflammation; colon; cx3cr1; Settore MED/04 - Patologia Generale

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APA (6th Edition):

Marelli, G. (2016). ROLE OF INTESTINAL CX3CR1+ MACROPHAGES IN THE DEVELOPMENT OF COLITIS-ASSOCIATED CANCER: FOCUS ON THE RELEVANCE OF THE MICROBIOME AND HEME-OXYGENASE-1. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/365306

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Marelli, G.. “ROLE OF INTESTINAL CX3CR1+ MACROPHAGES IN THE DEVELOPMENT OF COLITIS-ASSOCIATED CANCER: FOCUS ON THE RELEVANCE OF THE MICROBIOME AND HEME-OXYGENASE-1.” 2016. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/365306.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Marelli, G.. “ROLE OF INTESTINAL CX3CR1+ MACROPHAGES IN THE DEVELOPMENT OF COLITIS-ASSOCIATED CANCER: FOCUS ON THE RELEVANCE OF THE MICROBIOME AND HEME-OXYGENASE-1.” 2016. Web. 16 Jan 2021.

Vancouver:

Marelli G. ROLE OF INTESTINAL CX3CR1+ MACROPHAGES IN THE DEVELOPMENT OF COLITIS-ASSOCIATED CANCER: FOCUS ON THE RELEVANCE OF THE MICROBIOME AND HEME-OXYGENASE-1. [Internet] [Thesis]. Università degli Studi di Milano; 2016. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/365306.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Marelli G. ROLE OF INTESTINAL CX3CR1+ MACROPHAGES IN THE DEVELOPMENT OF COLITIS-ASSOCIATED CANCER: FOCUS ON THE RELEVANCE OF THE MICROBIOME AND HEME-OXYGENASE-1. [Thesis]. Università degli Studi di Milano; 2016. Available from: http://hdl.handle.net/2434/365306

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

11. A. Vacchini. ANALYSIS OF BIASED SIGNALING IN THE CHEMOKINE SYSTEM.

Degree: 2016, Università degli Studi di Milano

 Chemokines constitute a family of almost 50 small secreted cytokines, recognized by about 20 different 7TM spanning G protein coupled receptors (GPCRs), that activating pertussis… (more)

Subjects/Keywords: Chemokine; Atypical Chemokine Receptor; Biased Signaling; Settore MED/04 - Patologia Generale

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APA (6th Edition):

Vacchini, A. (2016). ANALYSIS OF BIASED SIGNALING IN THE CHEMOKINE SYSTEM. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/365864

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Vacchini, A.. “ANALYSIS OF BIASED SIGNALING IN THE CHEMOKINE SYSTEM.” 2016. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/365864.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Vacchini, A.. “ANALYSIS OF BIASED SIGNALING IN THE CHEMOKINE SYSTEM.” 2016. Web. 16 Jan 2021.

Vancouver:

Vacchini A. ANALYSIS OF BIASED SIGNALING IN THE CHEMOKINE SYSTEM. [Internet] [Thesis]. Università degli Studi di Milano; 2016. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/365864.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Vacchini A. ANALYSIS OF BIASED SIGNALING IN THE CHEMOKINE SYSTEM. [Thesis]. Università degli Studi di Milano; 2016. Available from: http://hdl.handle.net/2434/365864

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

12. L. Drufuca. ROLE OF MICRORNA IN MACROPHAGE POLARIZED ACTIVATION.

Degree: 2018, Università degli Studi di Milano

 Macrophages are cells from the innate branch of immune system with central roles both under physiologic and pathologic conditions. Such complex behaviour relies on extreme… (more)

Subjects/Keywords: Settore BIO/13 - Biologia Applicata

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APA (6th Edition):

Drufuca, L. (2018). ROLE OF MICRORNA IN MACROPHAGE POLARIZED ACTIVATION. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/565701

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Drufuca, L.. “ROLE OF MICRORNA IN MACROPHAGE POLARIZED ACTIVATION.” 2018. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/565701.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Drufuca, L.. “ROLE OF MICRORNA IN MACROPHAGE POLARIZED ACTIVATION.” 2018. Web. 16 Jan 2021.

Vancouver:

Drufuca L. ROLE OF MICRORNA IN MACROPHAGE POLARIZED ACTIVATION. [Internet] [Thesis]. Università degli Studi di Milano; 2018. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/565701.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Drufuca L. ROLE OF MICRORNA IN MACROPHAGE POLARIZED ACTIVATION. [Thesis]. Università degli Studi di Milano; 2018. Available from: http://hdl.handle.net/2434/565701

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

13. F. Roma. RUOLO DEI MICRORNA NEL DIFFERENZIAMENTO MIOGENICO NORMALE E PATOLOGICO.

Degree: 2015, Università degli Studi di Milano

 riassunto Facioscapulohumeral muscular dystrophy (FSHD) is an autosomal dominant disease, is the third most common myopathy, with an incidence of 1 in 15.000 in the… (more)

Subjects/Keywords: Settore BIO/13 - Biologia Applicata

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APA (6th Edition):

Roma, F. (2015). RUOLO DEI MICRORNA NEL DIFFERENZIAMENTO MIOGENICO NORMALE E PATOLOGICO. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/252093

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Roma, F.. “RUOLO DEI MICRORNA NEL DIFFERENZIAMENTO MIOGENICO NORMALE E PATOLOGICO.” 2015. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/252093.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Roma, F.. “RUOLO DEI MICRORNA NEL DIFFERENZIAMENTO MIOGENICO NORMALE E PATOLOGICO.” 2015. Web. 16 Jan 2021.

Vancouver:

Roma F. RUOLO DEI MICRORNA NEL DIFFERENZIAMENTO MIOGENICO NORMALE E PATOLOGICO. [Internet] [Thesis]. Università degli Studi di Milano; 2015. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/252093.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Roma F. RUOLO DEI MICRORNA NEL DIFFERENZIAMENTO MIOGENICO NORMALE E PATOLOGICO. [Thesis]. Università degli Studi di Milano; 2015. Available from: http://hdl.handle.net/2434/252093

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

14. R. Gualtierotti. ACTIVITY OF MONOMERIC SECOND MITOCHONDRIAL ACTIVATOR OF CASPASES (SMAC) MIMETIC COMPOUNDS ON HUMAN FIBROBLAST-LIKE SYNOVIOCYTES FROM RHEUMATOID ARTHRITIS PATIENTS.

Degree: 2016, Università degli Studi di Milano

 Rheumatoid arthritis (RA) is a chronic autoimmune disease characterised by systemic and intrarticular inflammation. Fibroblast-like synoviocytes (FLSs) in RA have an activated phenotype with increased… (more)

Subjects/Keywords: Settore MED/04 - Patologia Generale; Settore MED/16 - Reumatologia; Settore MED/09 - Medicina Interna

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APA (6th Edition):

Gualtierotti, R. (2016). ACTIVITY OF MONOMERIC SECOND MITOCHONDRIAL ACTIVATOR OF CASPASES (SMAC) MIMETIC COMPOUNDS ON HUMAN FIBROBLAST-LIKE SYNOVIOCYTES FROM RHEUMATOID ARTHRITIS PATIENTS. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/366559

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gualtierotti, R.. “ACTIVITY OF MONOMERIC SECOND MITOCHONDRIAL ACTIVATOR OF CASPASES (SMAC) MIMETIC COMPOUNDS ON HUMAN FIBROBLAST-LIKE SYNOVIOCYTES FROM RHEUMATOID ARTHRITIS PATIENTS.” 2016. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/366559.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gualtierotti, R.. “ACTIVITY OF MONOMERIC SECOND MITOCHONDRIAL ACTIVATOR OF CASPASES (SMAC) MIMETIC COMPOUNDS ON HUMAN FIBROBLAST-LIKE SYNOVIOCYTES FROM RHEUMATOID ARTHRITIS PATIENTS.” 2016. Web. 16 Jan 2021.

Vancouver:

Gualtierotti R. ACTIVITY OF MONOMERIC SECOND MITOCHONDRIAL ACTIVATOR OF CASPASES (SMAC) MIMETIC COMPOUNDS ON HUMAN FIBROBLAST-LIKE SYNOVIOCYTES FROM RHEUMATOID ARTHRITIS PATIENTS. [Internet] [Thesis]. Università degli Studi di Milano; 2016. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/366559.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gualtierotti R. ACTIVITY OF MONOMERIC SECOND MITOCHONDRIAL ACTIVATOR OF CASPASES (SMAC) MIMETIC COMPOUNDS ON HUMAN FIBROBLAST-LIKE SYNOVIOCYTES FROM RHEUMATOID ARTHRITIS PATIENTS. [Thesis]. Università degli Studi di Milano; 2016. Available from: http://hdl.handle.net/2434/366559

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

15. E. Toffolo. POST-TRANSCRIPTIONAL AND POST-TRANSLATIONAL REGULATION OF LSD1 IN MAMMALIAN BRAIN.

Degree: 2015, Università degli Studi di Milano

 Epigenetic mechanisms play important roles in brain development, orchestrating proliferation, differentiation, and morphogenesis. Lysine-Specific Demethylase 1 (LSD1 also known as KDM1A and AOF2) is a… (more)

Subjects/Keywords: EPIGENETICS; LSD1; NEURONAL MATURATION; Settore BIO/13 - Biologia Applicata

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APA (6th Edition):

Toffolo, E. (2015). POST-TRANSCRIPTIONAL AND POST-TRANSLATIONAL REGULATION OF LSD1 IN MAMMALIAN BRAIN. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/286318

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Toffolo, E.. “POST-TRANSCRIPTIONAL AND POST-TRANSLATIONAL REGULATION OF LSD1 IN MAMMALIAN BRAIN.” 2015. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/286318.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Toffolo, E.. “POST-TRANSCRIPTIONAL AND POST-TRANSLATIONAL REGULATION OF LSD1 IN MAMMALIAN BRAIN.” 2015. Web. 16 Jan 2021.

Vancouver:

Toffolo E. POST-TRANSCRIPTIONAL AND POST-TRANSLATIONAL REGULATION OF LSD1 IN MAMMALIAN BRAIN. [Internet] [Thesis]. Università degli Studi di Milano; 2015. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/286318.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Toffolo E. POST-TRANSCRIPTIONAL AND POST-TRANSLATIONAL REGULATION OF LSD1 IN MAMMALIAN BRAIN. [Thesis]. Università degli Studi di Milano; 2015. Available from: http://hdl.handle.net/2434/286318

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

16. M. Rubino. EPIGENETIC MODIFICATIONS ABOLISH THE EXPRESSION OF THE LONG PENTRAXIN PTX3 IN HUMAN TUMORS.

Degree: 2015, Università degli Studi di Milano

 PTX3 is a fluid-phase pattern recognition receptor that participates in innate immunity and inflammation by modulating complement activation, leukocyte recruitment, extracellular matrix deposition and angiogenesis.… (more)

Subjects/Keywords: PTX3; cancer; epigenetics modifications; enhancers; miRNAs; DNA methylation; histone modifications; Settore MED/04 - Patologia Generale

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APA (6th Edition):

Rubino, M. (2015). EPIGENETIC MODIFICATIONS ABOLISH THE EXPRESSION OF THE LONG PENTRAXIN PTX3 IN HUMAN TUMORS. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/254345

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rubino, M.. “EPIGENETIC MODIFICATIONS ABOLISH THE EXPRESSION OF THE LONG PENTRAXIN PTX3 IN HUMAN TUMORS.” 2015. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/254345.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rubino, M.. “EPIGENETIC MODIFICATIONS ABOLISH THE EXPRESSION OF THE LONG PENTRAXIN PTX3 IN HUMAN TUMORS.” 2015. Web. 16 Jan 2021.

Vancouver:

Rubino M. EPIGENETIC MODIFICATIONS ABOLISH THE EXPRESSION OF THE LONG PENTRAXIN PTX3 IN HUMAN TUMORS. [Internet] [Thesis]. Università degli Studi di Milano; 2015. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/254345.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rubino M. EPIGENETIC MODIFICATIONS ABOLISH THE EXPRESSION OF THE LONG PENTRAXIN PTX3 IN HUMAN TUMORS. [Thesis]. Università degli Studi di Milano; 2015. Available from: http://hdl.handle.net/2434/254345

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. M.C. Filomena. THE ROLE OF THE SARCOMERIC PROTEIN MYOPALLADIN IN SKELETAL AND CARDIAC MUSCLE STRUCTURE, FUNCTION AND DISEASE.

Degree: 2016, Università degli Studi di Milano

 Myopalladin (MYPN) is a striated muscle-specific sarcomeric protein belonging to the small palladin/myopalladin/myotilin family of actin-associated immunoglobulin-containing proteins [1]. Within the sarcomeric Z-line, MYPN binds… (more)

Subjects/Keywords: Settore MED/11 - Malattie dell'Apparato Cardiovascolare

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APA (6th Edition):

Filomena, M. (2016). THE ROLE OF THE SARCOMERIC PROTEIN MYOPALLADIN IN SKELETAL AND CARDIAC MUSCLE STRUCTURE, FUNCTION AND DISEASE. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/365482

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Filomena, M.C.. “THE ROLE OF THE SARCOMERIC PROTEIN MYOPALLADIN IN SKELETAL AND CARDIAC MUSCLE STRUCTURE, FUNCTION AND DISEASE.” 2016. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/365482.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Filomena, M.C.. “THE ROLE OF THE SARCOMERIC PROTEIN MYOPALLADIN IN SKELETAL AND CARDIAC MUSCLE STRUCTURE, FUNCTION AND DISEASE.” 2016. Web. 16 Jan 2021.

Vancouver:

Filomena M. THE ROLE OF THE SARCOMERIC PROTEIN MYOPALLADIN IN SKELETAL AND CARDIAC MUSCLE STRUCTURE, FUNCTION AND DISEASE. [Internet] [Thesis]. Università degli Studi di Milano; 2016. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/365482.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Filomena M. THE ROLE OF THE SARCOMERIC PROTEIN MYOPALLADIN IN SKELETAL AND CARDIAC MUSCLE STRUCTURE, FUNCTION AND DISEASE. [Thesis]. Università degli Studi di Milano; 2016. Available from: http://hdl.handle.net/2434/365482

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

18. G. DI CARO. ADAPTIVE AND NATIVE IMMUNE CELLS AS PROGNOSTIC AND PREDICTIVE BIOMARKERS ALONG COLORECTAL CANCER PROGRESSION.

Degree: 2013, Università degli Studi di Milano

 Inflammation and cells of the innate immune system are known to contribute to tumour initiation and progression. Differently, the adaptive immune response controls growth and… (more)

Subjects/Keywords: colorectal cancer; immune infiltration; chemotherapy; Settore MED/03 - Genetica Medica; Settore MED/04 - Patologia Generale; Settore MED/05 - Patologia Clinica

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APA (6th Edition):

CARO, G. D. (2013). ADAPTIVE AND NATIVE IMMUNE CELLS AS PROGNOSTIC AND PREDICTIVE BIOMARKERS ALONG COLORECTAL CANCER PROGRESSION. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/218989

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

CARO, G. DI. “ADAPTIVE AND NATIVE IMMUNE CELLS AS PROGNOSTIC AND PREDICTIVE BIOMARKERS ALONG COLORECTAL CANCER PROGRESSION.” 2013. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/218989.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

CARO, G. DI. “ADAPTIVE AND NATIVE IMMUNE CELLS AS PROGNOSTIC AND PREDICTIVE BIOMARKERS ALONG COLORECTAL CANCER PROGRESSION.” 2013. Web. 16 Jan 2021.

Vancouver:

CARO GD. ADAPTIVE AND NATIVE IMMUNE CELLS AS PROGNOSTIC AND PREDICTIVE BIOMARKERS ALONG COLORECTAL CANCER PROGRESSION. [Internet] [Thesis]. Università degli Studi di Milano; 2013. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/218989.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

CARO GD. ADAPTIVE AND NATIVE IMMUNE CELLS AS PROGNOSTIC AND PREDICTIVE BIOMARKERS ALONG COLORECTAL CANCER PROGRESSION. [Thesis]. Università degli Studi di Milano; 2013. Available from: http://hdl.handle.net/2434/218989

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

19. C. Cancellieri. BETA-ARRESTIN DEPENDENT REGULATION OF CYTOSKELETON DYNAMICS AND SIGNALLING OF CHEMOKINE RECEPTOR ACKR2.

Degree: 2014, Università degli Studi di Milano

 Chemokines promote leukocyte migration through the activation of dedicated G-protein coupled receptors. Beyond conventional chemokine receptors, which directly induce cell migration through heterotrimeric Gαi-mediated signalling… (more)

Subjects/Keywords: chemokine; atypical chemokine receptor; sytoskeleton; actin; microtubules; myosin; beta-arrestin; Settore MED/04 - Patologia Generale

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APA (6th Edition):

Cancellieri, C. (2014). BETA-ARRESTIN DEPENDENT REGULATION OF CYTOSKELETON DYNAMICS AND SIGNALLING OF CHEMOKINE RECEPTOR ACKR2. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/229565

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cancellieri, C.. “BETA-ARRESTIN DEPENDENT REGULATION OF CYTOSKELETON DYNAMICS AND SIGNALLING OF CHEMOKINE RECEPTOR ACKR2.” 2014. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/229565.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cancellieri, C.. “BETA-ARRESTIN DEPENDENT REGULATION OF CYTOSKELETON DYNAMICS AND SIGNALLING OF CHEMOKINE RECEPTOR ACKR2.” 2014. Web. 16 Jan 2021.

Vancouver:

Cancellieri C. BETA-ARRESTIN DEPENDENT REGULATION OF CYTOSKELETON DYNAMICS AND SIGNALLING OF CHEMOKINE RECEPTOR ACKR2. [Internet] [Thesis]. Università degli Studi di Milano; 2014. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/229565.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cancellieri C. BETA-ARRESTIN DEPENDENT REGULATION OF CYTOSKELETON DYNAMICS AND SIGNALLING OF CHEMOKINE RECEPTOR ACKR2. [Thesis]. Università degli Studi di Milano; 2014. Available from: http://hdl.handle.net/2434/229565

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

20. L. Petti. ROLE OF SPHINGOSINE-1-PHOSPATE PATHWAY IN INTESTINAL EPITHELIAL CELLS AND ITS INVOLVEMENT IN INTESTINAL TUMORIGENESIS.

Degree: 2017, Università degli Studi di Milano

 Title: Role of Sphingosine-1-phosphate pathway in intestinal epithelial cells and its involvement in intestinal tumorigenesis. Sphingosine-1-phosphate (S1P) is a bioactive sphingolipid metabolite, involved in several… (more)

Subjects/Keywords: Settore MED/04 - Patologia Generale

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APA (6th Edition):

Petti, L. (2017). ROLE OF SPHINGOSINE-1-PHOSPATE PATHWAY IN INTESTINAL EPITHELIAL CELLS AND ITS INVOLVEMENT IN INTESTINAL TUMORIGENESIS. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/487921

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Petti, L.. “ROLE OF SPHINGOSINE-1-PHOSPATE PATHWAY IN INTESTINAL EPITHELIAL CELLS AND ITS INVOLVEMENT IN INTESTINAL TUMORIGENESIS.” 2017. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/487921.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Petti, L.. “ROLE OF SPHINGOSINE-1-PHOSPATE PATHWAY IN INTESTINAL EPITHELIAL CELLS AND ITS INVOLVEMENT IN INTESTINAL TUMORIGENESIS.” 2017. Web. 16 Jan 2021.

Vancouver:

Petti L. ROLE OF SPHINGOSINE-1-PHOSPATE PATHWAY IN INTESTINAL EPITHELIAL CELLS AND ITS INVOLVEMENT IN INTESTINAL TUMORIGENESIS. [Internet] [Thesis]. Università degli Studi di Milano; 2017. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/487921.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Petti L. ROLE OF SPHINGOSINE-1-PHOSPATE PATHWAY IN INTESTINAL EPITHELIAL CELLS AND ITS INVOLVEMENT IN INTESTINAL TUMORIGENESIS. [Thesis]. Università degli Studi di Milano; 2017. Available from: http://hdl.handle.net/2434/487921

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. L. Mori. REGULATORY ROLE OF MICRORNAS ON INFLAMMATORY SIGNALING PATHWAYS.

Degree: 2012, Università degli Studi di Milano

 Innate immunity is the primary defense mechanism that recognizes and respond to invading infectious microbes or their components, known as pathogen-associated molecular patterns (PAMPs), or… (more)

Subjects/Keywords: microRNA; post-transcriptional regulation; inflammation; monocyte; neutrophil; Settore MED/04 - Patologia Generale

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APA (6th Edition):

Mori, L. (2012). REGULATORY ROLE OF MICRORNAS ON INFLAMMATORY SIGNALING PATHWAYS. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/168371

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mori, L.. “REGULATORY ROLE OF MICRORNAS ON INFLAMMATORY SIGNALING PATHWAYS.” 2012. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/168371.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mori, L.. “REGULATORY ROLE OF MICRORNAS ON INFLAMMATORY SIGNALING PATHWAYS.” 2012. Web. 16 Jan 2021.

Vancouver:

Mori L. REGULATORY ROLE OF MICRORNAS ON INFLAMMATORY SIGNALING PATHWAYS. [Internet] [Thesis]. Università degli Studi di Milano; 2012. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/168371.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mori L. REGULATORY ROLE OF MICRORNAS ON INFLAMMATORY SIGNALING PATHWAYS. [Thesis]. Università degli Studi di Milano; 2012. Available from: http://hdl.handle.net/2434/168371

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

22. I. Mattiola. CROSS-TALK BETWEEN HUMAN NK CELLS AND MACROPHAGES: INFLUENCE OF THE TUMOR MICRO-ENVIRONMENT.

Degree: 2014, Università degli Studi di Milano

 Natural killer (NK) cells are important effectors of innate immune responses providing cellular immunity against tumor-transformed and virally-infected cells. The existence of cross-talks between NK… (more)

Subjects/Keywords: NK cells; Macrophages; Cross-talk; Cancer; Settore MED/04 - Patologia Generale

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APA (6th Edition):

Mattiola, I. (2014). CROSS-TALK BETWEEN HUMAN NK CELLS AND MACROPHAGES: INFLUENCE OF THE TUMOR MICRO-ENVIRONMENT. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/229562

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mattiola, I.. “CROSS-TALK BETWEEN HUMAN NK CELLS AND MACROPHAGES: INFLUENCE OF THE TUMOR MICRO-ENVIRONMENT.” 2014. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/229562.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mattiola, I.. “CROSS-TALK BETWEEN HUMAN NK CELLS AND MACROPHAGES: INFLUENCE OF THE TUMOR MICRO-ENVIRONMENT.” 2014. Web. 16 Jan 2021.

Vancouver:

Mattiola I. CROSS-TALK BETWEEN HUMAN NK CELLS AND MACROPHAGES: INFLUENCE OF THE TUMOR MICRO-ENVIRONMENT. [Internet] [Thesis]. Università degli Studi di Milano; 2014. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/229562.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mattiola I. CROSS-TALK BETWEEN HUMAN NK CELLS AND MACROPHAGES: INFLUENCE OF THE TUMOR MICRO-ENVIRONMENT. [Thesis]. Università degli Studi di Milano; 2014. Available from: http://hdl.handle.net/2434/229562

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. I. Ferrari. THE ROLE OF THE IRSP53-LIN7 COMPLEX IN CELL MEMBRANE DYNAMICS.

Degree: 2015, Università degli Studi di Milano

 The insulin receptor substrate protein of 53 kDa is a critical factor in determining neuronal polarisation, as it initiates membrane protrusions to form filopodia and… (more)

Subjects/Keywords: IRSp53; LIN7; IRSp53-LIN7 complex; N2A cells; NSC34 cells; filopodia; neurites; tip complex; mitochondrial morphology; mitochondrial fission; mitochondria pre-constriction sites; Settore BIO/13 - Biologia Applicata

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APA (6th Edition):

Ferrari, I. (2015). THE ROLE OF THE IRSP53-LIN7 COMPLEX IN CELL MEMBRANE DYNAMICS. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/250654

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ferrari, I.. “THE ROLE OF THE IRSP53-LIN7 COMPLEX IN CELL MEMBRANE DYNAMICS.” 2015. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/250654.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ferrari, I.. “THE ROLE OF THE IRSP53-LIN7 COMPLEX IN CELL MEMBRANE DYNAMICS.” 2015. Web. 16 Jan 2021.

Vancouver:

Ferrari I. THE ROLE OF THE IRSP53-LIN7 COMPLEX IN CELL MEMBRANE DYNAMICS. [Internet] [Thesis]. Università degli Studi di Milano; 2015. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/250654.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ferrari I. THE ROLE OF THE IRSP53-LIN7 COMPLEX IN CELL MEMBRANE DYNAMICS. [Thesis]. Università degli Studi di Milano; 2015. Available from: http://hdl.handle.net/2434/250654

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. C.L. Politano. TRANSCRIPTOME ANALYSIS OF TUMOR INFILTRATING T REGULATORY CELLS UNVEILS SPECIFIC CODING AND NON CODING GENE SIGNATURE.

Degree: 2019, Università degli Studi di Milano

 CD4+ Regulatory T cells (Treg cells) are a specialized subpopulation of T cells that act to suppress immune response, thereby maintaining homeostasis and self-tolerance. These… (more)

Subjects/Keywords: Settore BIO/11 - Biologia Molecolare

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Politano, C. (2019). TRANSCRIPTOME ANALYSIS OF TUMOR INFILTRATING T REGULATORY CELLS UNVEILS SPECIFIC CODING AND NON CODING GENE SIGNATURE. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/613244

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Politano, C.L.. “TRANSCRIPTOME ANALYSIS OF TUMOR INFILTRATING T REGULATORY CELLS UNVEILS SPECIFIC CODING AND NON CODING GENE SIGNATURE.” 2019. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/613244.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Politano, C.L.. “TRANSCRIPTOME ANALYSIS OF TUMOR INFILTRATING T REGULATORY CELLS UNVEILS SPECIFIC CODING AND NON CODING GENE SIGNATURE.” 2019. Web. 16 Jan 2021.

Vancouver:

Politano C. TRANSCRIPTOME ANALYSIS OF TUMOR INFILTRATING T REGULATORY CELLS UNVEILS SPECIFIC CODING AND NON CODING GENE SIGNATURE. [Internet] [Thesis]. Università degli Studi di Milano; 2019. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/613244.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Politano C. TRANSCRIPTOME ANALYSIS OF TUMOR INFILTRATING T REGULATORY CELLS UNVEILS SPECIFIC CODING AND NON CODING GENE SIGNATURE. [Thesis]. Università degli Studi di Milano; 2019. Available from: http://hdl.handle.net/2434/613244

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

25. M. Fazzari. NOVEL APPROACHES OF ¿PERSONALISED MEDICINE¿ AS PROOF-OF-PRINCIPLE FOR CDKL5-RELATED PATHOLOGIES.

Degree: 2018, Università degli Studi di Milano

 Alterations of CDKL5 give rise to several forms of neurological disorders generally characterised by epileptic encephalopathy, severe developmental delay, hypotonia and RTT-like features. To date… (more)

Subjects/Keywords: Settore BIO/11 - Biologia Molecolare

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Fazzari, M. (2018). NOVEL APPROACHES OF ¿PERSONALISED MEDICINE¿ AS PROOF-OF-PRINCIPLE FOR CDKL5-RELATED PATHOLOGIES. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/548108

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fazzari, M.. “NOVEL APPROACHES OF ¿PERSONALISED MEDICINE¿ AS PROOF-OF-PRINCIPLE FOR CDKL5-RELATED PATHOLOGIES.” 2018. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/548108.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fazzari, M.. “NOVEL APPROACHES OF ¿PERSONALISED MEDICINE¿ AS PROOF-OF-PRINCIPLE FOR CDKL5-RELATED PATHOLOGIES.” 2018. Web. 16 Jan 2021.

Vancouver:

Fazzari M. NOVEL APPROACHES OF ¿PERSONALISED MEDICINE¿ AS PROOF-OF-PRINCIPLE FOR CDKL5-RELATED PATHOLOGIES. [Internet] [Thesis]. Università degli Studi di Milano; 2018. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/548108.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fazzari M. NOVEL APPROACHES OF ¿PERSONALISED MEDICINE¿ AS PROOF-OF-PRINCIPLE FOR CDKL5-RELATED PATHOLOGIES. [Thesis]. Università degli Studi di Milano; 2018. Available from: http://hdl.handle.net/2434/548108

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. L. Brioschi. SPHINGOLIPID SIGNALING AND DISEASE: THE KEY ROLE OF CERAMIDE TRAFFIC IN CELL FATE REGULATION.

Degree: 2016, Università degli Studi di Milano

 Cancer and diabetes are among the most common diseases in western societies. Sphingolipids, a class of lipids ubiquitously present in eukaryotic membranes, play a key… (more)

Subjects/Keywords: SPHINGOLIPID, CELL FATE, CERAMIDE, TRAFFIC, GLIOBLASTOMA, TYPE 2 DIABETES, SPHINGOSINE 1-PHOSPHATE; Settore BIO/10 - Biochimica

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Brioschi, L. (2016). SPHINGOLIPID SIGNALING AND DISEASE: THE KEY ROLE OF CERAMIDE TRAFFIC IN CELL FATE REGULATION. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/362927

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Brioschi, L.. “SPHINGOLIPID SIGNALING AND DISEASE: THE KEY ROLE OF CERAMIDE TRAFFIC IN CELL FATE REGULATION.” 2016. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/362927.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Brioschi, L.. “SPHINGOLIPID SIGNALING AND DISEASE: THE KEY ROLE OF CERAMIDE TRAFFIC IN CELL FATE REGULATION.” 2016. Web. 16 Jan 2021.

Vancouver:

Brioschi L. SPHINGOLIPID SIGNALING AND DISEASE: THE KEY ROLE OF CERAMIDE TRAFFIC IN CELL FATE REGULATION. [Internet] [Thesis]. Università degli Studi di Milano; 2016. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/362927.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Brioschi L. SPHINGOLIPID SIGNALING AND DISEASE: THE KEY ROLE OF CERAMIDE TRAFFIC IN CELL FATE REGULATION. [Thesis]. Università degli Studi di Milano; 2016. Available from: http://hdl.handle.net/2434/362927

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. A. Cinque. PLEIOTROPIC EFFECTS OF PERIFOSINE ON GLIOBLASTOMA CELLS SURVIVAL: ALTERED MEMBRANE LIPID METABOLISM AND CELL SIGNALING.

Degree: 2016, Università degli Studi di Milano

 Glioblastoma multiforme (GBM) is the most frequent and aggressive malignant tumor of the central nervous system in adults. Despite decades of experimentation to improve the… (more)

Subjects/Keywords: Settore BIO/10 - Biochimica

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cinque, A. (2016). PLEIOTROPIC EFFECTS OF PERIFOSINE ON GLIOBLASTOMA CELLS SURVIVAL: ALTERED MEMBRANE LIPID METABOLISM AND CELL SIGNALING. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/364862

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cinque, A.. “PLEIOTROPIC EFFECTS OF PERIFOSINE ON GLIOBLASTOMA CELLS SURVIVAL: ALTERED MEMBRANE LIPID METABOLISM AND CELL SIGNALING.” 2016. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/364862.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cinque, A.. “PLEIOTROPIC EFFECTS OF PERIFOSINE ON GLIOBLASTOMA CELLS SURVIVAL: ALTERED MEMBRANE LIPID METABOLISM AND CELL SIGNALING.” 2016. Web. 16 Jan 2021.

Vancouver:

Cinque A. PLEIOTROPIC EFFECTS OF PERIFOSINE ON GLIOBLASTOMA CELLS SURVIVAL: ALTERED MEMBRANE LIPID METABOLISM AND CELL SIGNALING. [Internet] [Thesis]. Università degli Studi di Milano; 2016. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/364862.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cinque A. PLEIOTROPIC EFFECTS OF PERIFOSINE ON GLIOBLASTOMA CELLS SURVIVAL: ALTERED MEMBRANE LIPID METABOLISM AND CELL SIGNALING. [Thesis]. Università degli Studi di Milano; 2016. Available from: http://hdl.handle.net/2434/364862

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. M. Liguori. BIOLOGICAL ROLE OF OSTEOACTIVIN, A PROTEIN EXPRESSED BY TUMOR-CONDITIONED MACROPHAGES, IN THE PROCESSES OF TUMOR GROWTH AND TISSUE REPAIR.

Degree: 2017, Università degli Studi di Milano

 Tumor-Associated Macrophages (TAM) are key orchestrators of the tumor microenvironment, directly affecting neoplastic cell growth, neo-angiogenesis, extracellular matrix remodeling and immunosuppression. In a gene profiling… (more)

Subjects/Keywords: Settore MED/04 - Patologia Generale; Settore MED/06 - Oncologia Medica

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Liguori, M. (2017). BIOLOGICAL ROLE OF OSTEOACTIVIN, A PROTEIN EXPRESSED BY TUMOR-CONDITIONED MACROPHAGES, IN THE PROCESSES OF TUMOR GROWTH AND TISSUE REPAIR. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/481318

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liguori, M.. “BIOLOGICAL ROLE OF OSTEOACTIVIN, A PROTEIN EXPRESSED BY TUMOR-CONDITIONED MACROPHAGES, IN THE PROCESSES OF TUMOR GROWTH AND TISSUE REPAIR.” 2017. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/481318.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liguori, M.. “BIOLOGICAL ROLE OF OSTEOACTIVIN, A PROTEIN EXPRESSED BY TUMOR-CONDITIONED MACROPHAGES, IN THE PROCESSES OF TUMOR GROWTH AND TISSUE REPAIR.” 2017. Web. 16 Jan 2021.

Vancouver:

Liguori M. BIOLOGICAL ROLE OF OSTEOACTIVIN, A PROTEIN EXPRESSED BY TUMOR-CONDITIONED MACROPHAGES, IN THE PROCESSES OF TUMOR GROWTH AND TISSUE REPAIR. [Internet] [Thesis]. Università degli Studi di Milano; 2017. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/481318.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liguori M. BIOLOGICAL ROLE OF OSTEOACTIVIN, A PROTEIN EXPRESSED BY TUMOR-CONDITIONED MACROPHAGES, IN THE PROCESSES OF TUMOR GROWTH AND TISSUE REPAIR. [Thesis]. Università degli Studi di Milano; 2017. Available from: http://hdl.handle.net/2434/481318

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. M. Massara. THE NEUTROPHIL ANTI-TUMORAL RESPONSE IN CANCER: ROLE OF ACKR2 AND CHEMOKINE RECEPTORS.

Degree: 2018, Università degli Studi di Milano

 Chemokines are key mediators of inflammation and are involved in both extrinsic and intrinsic pathway of cancer. Their main function is to induce leukocyte migration… (more)

Subjects/Keywords: Settore MED/04 - Patologia Generale

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Massara, M. (2018). THE NEUTROPHIL ANTI-TUMORAL RESPONSE IN CANCER: ROLE OF ACKR2 AND CHEMOKINE RECEPTORS. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/528533

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Massara, M.. “THE NEUTROPHIL ANTI-TUMORAL RESPONSE IN CANCER: ROLE OF ACKR2 AND CHEMOKINE RECEPTORS.” 2018. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/528533.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Massara, M.. “THE NEUTROPHIL ANTI-TUMORAL RESPONSE IN CANCER: ROLE OF ACKR2 AND CHEMOKINE RECEPTORS.” 2018. Web. 16 Jan 2021.

Vancouver:

Massara M. THE NEUTROPHIL ANTI-TUMORAL RESPONSE IN CANCER: ROLE OF ACKR2 AND CHEMOKINE RECEPTORS. [Internet] [Thesis]. Università degli Studi di Milano; 2018. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/528533.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Massara M. THE NEUTROPHIL ANTI-TUMORAL RESPONSE IN CANCER: ROLE OF ACKR2 AND CHEMOKINE RECEPTORS. [Thesis]. Università degli Studi di Milano; 2018. Available from: http://hdl.handle.net/2434/528533

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. M. Manzoni. MOLECULAR HETEROGENEITY OF MULTIPLE MYELOMA: THE BIOLOGICAL AND CLINICAL RELEVANCE OF NOVEL GENE MUTATIONS BY NEXT GENERATION SEQUENCING.

Degree: 2018, Università degli Studi di Milano

 Il mieloma multiplo (MM) è una proliferazione maligna fatale delle plasmacellule (PC) secernenti anticorpi del midollo osseo (BM), caratterizzata da un ampio spettro clinico e… (more)

Subjects/Keywords: MM; NGS; mutations; cfDNA; Settore MED/15 - Malattie del Sangue

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Manzoni, M. (2018). MOLECULAR HETEROGENEITY OF MULTIPLE MYELOMA: THE BIOLOGICAL AND CLINICAL RELEVANCE OF NOVEL GENE MUTATIONS BY NEXT GENERATION SEQUENCING. (Thesis). Università degli Studi di Milano. Retrieved from http://hdl.handle.net/2434/546254

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Manzoni, M.. “MOLECULAR HETEROGENEITY OF MULTIPLE MYELOMA: THE BIOLOGICAL AND CLINICAL RELEVANCE OF NOVEL GENE MUTATIONS BY NEXT GENERATION SEQUENCING.” 2018. Thesis, Università degli Studi di Milano. Accessed January 16, 2021. http://hdl.handle.net/2434/546254.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Manzoni, M.. “MOLECULAR HETEROGENEITY OF MULTIPLE MYELOMA: THE BIOLOGICAL AND CLINICAL RELEVANCE OF NOVEL GENE MUTATIONS BY NEXT GENERATION SEQUENCING.” 2018. Web. 16 Jan 2021.

Vancouver:

Manzoni M. MOLECULAR HETEROGENEITY OF MULTIPLE MYELOMA: THE BIOLOGICAL AND CLINICAL RELEVANCE OF NOVEL GENE MUTATIONS BY NEXT GENERATION SEQUENCING. [Internet] [Thesis]. Università degli Studi di Milano; 2018. [cited 2021 Jan 16]. Available from: http://hdl.handle.net/2434/546254.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Manzoni M. MOLECULAR HETEROGENEITY OF MULTIPLE MYELOMA: THE BIOLOGICAL AND CLINICAL RELEVANCE OF NOVEL GENE MUTATIONS BY NEXT GENERATION SEQUENCING. [Thesis]. Università degli Studi di Milano; 2018. Available from: http://hdl.handle.net/2434/546254

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2] [3]

.