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You searched for +publisher:"Texas A&M University" +contributor:("Winzer-Serhan, Ursula H"). Showing records 1 – 4 of 4 total matches.

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1. Cheng, Yifeng. The Role of Dopamine D1 and D2 Receptor-Expressing Striatal Neurons in the Corticostriatal Circuitry of Alcohol Use Disorder.

Degree: PhD, Medical Sciences, 2018, Texas A&M University

 Alcohol use disorder (AUD) changes glutamatergic and -aminobutyric acidergic (GABAergic) neurotransmission in many neuronal populations. The dorsomedial part of the striatum (DMS) and its critical… (more)

Subjects/Keywords: alcohol; striatum

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cheng, Y. (2018). The Role of Dopamine D1 and D2 Receptor-Expressing Striatal Neurons in the Corticostriatal Circuitry of Alcohol Use Disorder. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/174358

Chicago Manual of Style (16th Edition):

Cheng, Yifeng. “The Role of Dopamine D1 and D2 Receptor-Expressing Striatal Neurons in the Corticostriatal Circuitry of Alcohol Use Disorder.” 2018. Doctoral Dissertation, Texas A&M University. Accessed May 10, 2021. http://hdl.handle.net/1969.1/174358.

MLA Handbook (7th Edition):

Cheng, Yifeng. “The Role of Dopamine D1 and D2 Receptor-Expressing Striatal Neurons in the Corticostriatal Circuitry of Alcohol Use Disorder.” 2018. Web. 10 May 2021.

Vancouver:

Cheng Y. The Role of Dopamine D1 and D2 Receptor-Expressing Striatal Neurons in the Corticostriatal Circuitry of Alcohol Use Disorder. [Internet] [Doctoral dissertation]. Texas A&M University; 2018. [cited 2021 May 10]. Available from: http://hdl.handle.net/1969.1/174358.

Council of Science Editors:

Cheng Y. The Role of Dopamine D1 and D2 Receptor-Expressing Striatal Neurons in the Corticostriatal Circuitry of Alcohol Use Disorder. [Doctoral Dissertation]. Texas A&M University; 2018. Available from: http://hdl.handle.net/1969.1/174358


Texas A&M University

2. Nigussie, Fikru. Analysis of Hippocampal Cell Proliferation, Survival, and Neuronal Morphology in P/Q-Type Voltage-Gated Calcium Channel Mutant Mice.

Degree: PhD, Biomedical Sciences, 2013, Texas A&M University

 Tottering and leaner mutant mice carry mutations in the pore-forming subunit (1A) of P/Q-type (CaV 2.1) voltage-gated calcium ion (Ca2+) channels that result in reduced… (more)

Subjects/Keywords: Tottering; Leaner; P/Q-type calcium channel; hippocampus

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APA (6th Edition):

Nigussie, F. (2013). Analysis of Hippocampal Cell Proliferation, Survival, and Neuronal Morphology in P/Q-Type Voltage-Gated Calcium Channel Mutant Mice. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/149268

Chicago Manual of Style (16th Edition):

Nigussie, Fikru. “Analysis of Hippocampal Cell Proliferation, Survival, and Neuronal Morphology in P/Q-Type Voltage-Gated Calcium Channel Mutant Mice.” 2013. Doctoral Dissertation, Texas A&M University. Accessed May 10, 2021. http://hdl.handle.net/1969.1/149268.

MLA Handbook (7th Edition):

Nigussie, Fikru. “Analysis of Hippocampal Cell Proliferation, Survival, and Neuronal Morphology in P/Q-Type Voltage-Gated Calcium Channel Mutant Mice.” 2013. Web. 10 May 2021.

Vancouver:

Nigussie F. Analysis of Hippocampal Cell Proliferation, Survival, and Neuronal Morphology in P/Q-Type Voltage-Gated Calcium Channel Mutant Mice. [Internet] [Doctoral dissertation]. Texas A&M University; 2013. [cited 2021 May 10]. Available from: http://hdl.handle.net/1969.1/149268.

Council of Science Editors:

Nigussie F. Analysis of Hippocampal Cell Proliferation, Survival, and Neuronal Morphology in P/Q-Type Voltage-Gated Calcium Channel Mutant Mice. [Doctoral Dissertation]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/149268


Texas A&M University

3. Huang, Pei-San. Effects of Chronic Nicotine Exposure and Lack of High Affinity Nicotinic Receptors on Cortico-Hippocampal Areas in the Aging Mouse Brain.

Degree: PhD, Biomedical Sciences, 2012, Texas A&M University

 Nicotine, the major psychoactive ingredient of tobacco smoke, underlies numerous effects by activating neuronal nicotinic acetylcholine receptors. Both in vitro and in vivo studies suggest… (more)

Subjects/Keywords: nicotine; neuroprotection; aging; nAChRs; neurodegeneration; caloric restriction

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Huang, P. (2012). Effects of Chronic Nicotine Exposure and Lack of High Affinity Nicotinic Receptors on Cortico-Hippocampal Areas in the Aging Mouse Brain. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2012-05-11067

Chicago Manual of Style (16th Edition):

Huang, Pei-San. “Effects of Chronic Nicotine Exposure and Lack of High Affinity Nicotinic Receptors on Cortico-Hippocampal Areas in the Aging Mouse Brain.” 2012. Doctoral Dissertation, Texas A&M University. Accessed May 10, 2021. http://hdl.handle.net/1969.1/ETD-TAMU-2012-05-11067.

MLA Handbook (7th Edition):

Huang, Pei-San. “Effects of Chronic Nicotine Exposure and Lack of High Affinity Nicotinic Receptors on Cortico-Hippocampal Areas in the Aging Mouse Brain.” 2012. Web. 10 May 2021.

Vancouver:

Huang P. Effects of Chronic Nicotine Exposure and Lack of High Affinity Nicotinic Receptors on Cortico-Hippocampal Areas in the Aging Mouse Brain. [Internet] [Doctoral dissertation]. Texas A&M University; 2012. [cited 2021 May 10]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2012-05-11067.

Council of Science Editors:

Huang P. Effects of Chronic Nicotine Exposure and Lack of High Affinity Nicotinic Receptors on Cortico-Hippocampal Areas in the Aging Mouse Brain. [Doctoral Dissertation]. Texas A&M University; 2012. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2012-05-11067


Texas A&M University

4. Chuang, Shu-Hui. Cell-Specific and Phosphorylation-Dependent Actions of Neurosteroids at Extrasynaptic GABA-A Receptors in the Brain.

Degree: PhD, Medical Sciences, 2019, Texas A&M University

 Epilepsy is a chronic neurological disorder characterized by unpredicted recurrent seizures. Dysfunction of GABA-A receptors (GABA-ARs) in the brain plays a key role in the… (more)

Subjects/Keywords: Neurosteroids; Ganaxolone; GABA-A receptors; Tonic inhibition; Hippocampus; Dentate gyrus granule cells; Seizures; Epilepsy; Zinc; Isobolographic analysis; Tiagabine; Midazolam

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chuang, S. (2019). Cell-Specific and Phosphorylation-Dependent Actions of Neurosteroids at Extrasynaptic GABA-A Receptors in the Brain. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/183836

Chicago Manual of Style (16th Edition):

Chuang, Shu-Hui. “Cell-Specific and Phosphorylation-Dependent Actions of Neurosteroids at Extrasynaptic GABA-A Receptors in the Brain.” 2019. Doctoral Dissertation, Texas A&M University. Accessed May 10, 2021. http://hdl.handle.net/1969.1/183836.

MLA Handbook (7th Edition):

Chuang, Shu-Hui. “Cell-Specific and Phosphorylation-Dependent Actions of Neurosteroids at Extrasynaptic GABA-A Receptors in the Brain.” 2019. Web. 10 May 2021.

Vancouver:

Chuang S. Cell-Specific and Phosphorylation-Dependent Actions of Neurosteroids at Extrasynaptic GABA-A Receptors in the Brain. [Internet] [Doctoral dissertation]. Texas A&M University; 2019. [cited 2021 May 10]. Available from: http://hdl.handle.net/1969.1/183836.

Council of Science Editors:

Chuang S. Cell-Specific and Phosphorylation-Dependent Actions of Neurosteroids at Extrasynaptic GABA-A Receptors in the Brain. [Doctoral Dissertation]. Texas A&M University; 2019. Available from: http://hdl.handle.net/1969.1/183836

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