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You searched for +publisher:"Texas A&M University" +contributor:("Welsh, C. Jane"). Showing records 1 – 25 of 25 total matches.

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Texas A&M University

1. Zriba, Slim. Towards the Development of an Improved Vaccine for Swine Against Brucellosis.

Degree: MS, Biomedical Sciences, 2017, Texas A&M University

 Swine brucellosis is a zoonotic disease with variable incidence rates in domestic and feral populations. It is a serious public health issue due to the… (more)

Subjects/Keywords: Abortion; brucellosis; vaccination; iELISA; Western blot; Rose Bengal Test; DIVA; ΔvjbR

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APA (6th Edition):

Zriba, S. (2017). Towards the Development of an Improved Vaccine for Swine Against Brucellosis. (Masters Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/161643

Chicago Manual of Style (16th Edition):

Zriba, Slim. “Towards the Development of an Improved Vaccine for Swine Against Brucellosis.” 2017. Masters Thesis, Texas A&M University. Accessed April 17, 2021. http://hdl.handle.net/1969.1/161643.

MLA Handbook (7th Edition):

Zriba, Slim. “Towards the Development of an Improved Vaccine for Swine Against Brucellosis.” 2017. Web. 17 Apr 2021.

Vancouver:

Zriba S. Towards the Development of an Improved Vaccine for Swine Against Brucellosis. [Internet] [Masters thesis]. Texas A&M University; 2017. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1969.1/161643.

Council of Science Editors:

Zriba S. Towards the Development of an Improved Vaccine for Swine Against Brucellosis. [Masters Thesis]. Texas A&M University; 2017. Available from: http://hdl.handle.net/1969.1/161643


Texas A&M University

2. Bhattarai, Bikash. Economic Consequences Associated with Johne’s Disease in Cow-Calf Operations.

Degree: PhD, Biomedical Sciences, 2013, Texas A&M University

 Johne’s disease (JD) in cattle is a disease of economic importance caused by Mycobacterium avium subspecies paratuberculosis (MAP). Studies were conducted to estimate the losses… (more)

Subjects/Keywords: Mycobacterium avium subspecies paratuberculosis; Johne's Disease; Beef, Cow-calf

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APA (6th Edition):

Bhattarai, B. (2013). Economic Consequences Associated with Johne’s Disease in Cow-Calf Operations. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/151288

Chicago Manual of Style (16th Edition):

Bhattarai, Bikash. “Economic Consequences Associated with Johne’s Disease in Cow-Calf Operations.” 2013. Doctoral Dissertation, Texas A&M University. Accessed April 17, 2021. http://hdl.handle.net/1969.1/151288.

MLA Handbook (7th Edition):

Bhattarai, Bikash. “Economic Consequences Associated with Johne’s Disease in Cow-Calf Operations.” 2013. Web. 17 Apr 2021.

Vancouver:

Bhattarai B. Economic Consequences Associated with Johne’s Disease in Cow-Calf Operations. [Internet] [Doctoral dissertation]. Texas A&M University; 2013. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1969.1/151288.

Council of Science Editors:

Bhattarai B. Economic Consequences Associated with Johne’s Disease in Cow-Calf Operations. [Doctoral Dissertation]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/151288

3. Hodo, Carolyn Louise. The Ecology of Trypanosoma cruzi and Its Mammalian Hosts in Texas, USA.

Degree: PhD, Veterinary Pathobiology, 2017, Texas A&M University

 Trypanosoma cruzi, agent of Chagas disease, is a zoonotic vector-borne protozoan that infects mammalian hosts throughout the Americas. Spillover from sylvatic cycles occurs when triatomine… (more)

Subjects/Keywords: Trypanosoma cruzi; Chagas; disease ecology; epidemiology; reservoir; vector-borne disease

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APA (6th Edition):

Hodo, C. L. (2017). The Ecology of Trypanosoma cruzi and Its Mammalian Hosts in Texas, USA. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/165907

Chicago Manual of Style (16th Edition):

Hodo, Carolyn Louise. “The Ecology of Trypanosoma cruzi and Its Mammalian Hosts in Texas, USA.” 2017. Doctoral Dissertation, Texas A&M University. Accessed April 17, 2021. http://hdl.handle.net/1969.1/165907.

MLA Handbook (7th Edition):

Hodo, Carolyn Louise. “The Ecology of Trypanosoma cruzi and Its Mammalian Hosts in Texas, USA.” 2017. Web. 17 Apr 2021.

Vancouver:

Hodo CL. The Ecology of Trypanosoma cruzi and Its Mammalian Hosts in Texas, USA. [Internet] [Doctoral dissertation]. Texas A&M University; 2017. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1969.1/165907.

Council of Science Editors:

Hodo CL. The Ecology of Trypanosoma cruzi and Its Mammalian Hosts in Texas, USA. [Doctoral Dissertation]. Texas A&M University; 2017. Available from: http://hdl.handle.net/1969.1/165907


Texas A&M University

4. Robinson, Abraham. Development of a Biomarker and Clay Based Remediation Strategy for Populations at Risk for Fumonisin Toxicosis.

Degree: PhD, Toxicology, 2012, Texas A&M University

 Fumonisin B1 is the most prevalent congener of the fumonisin mycotoxins produced by Fusarium verticilliodies and is considered by many to be the most toxic.… (more)

Subjects/Keywords: Fumonisin; mycotoxin; FB1; Ghana; NovaSil; Enterosorbent

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APA (6th Edition):

Robinson, A. (2012). Development of a Biomarker and Clay Based Remediation Strategy for Populations at Risk for Fumonisin Toxicosis. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2010-12-8814

Chicago Manual of Style (16th Edition):

Robinson, Abraham. “Development of a Biomarker and Clay Based Remediation Strategy for Populations at Risk for Fumonisin Toxicosis.” 2012. Doctoral Dissertation, Texas A&M University. Accessed April 17, 2021. http://hdl.handle.net/1969.1/ETD-TAMU-2010-12-8814.

MLA Handbook (7th Edition):

Robinson, Abraham. “Development of a Biomarker and Clay Based Remediation Strategy for Populations at Risk for Fumonisin Toxicosis.” 2012. Web. 17 Apr 2021.

Vancouver:

Robinson A. Development of a Biomarker and Clay Based Remediation Strategy for Populations at Risk for Fumonisin Toxicosis. [Internet] [Doctoral dissertation]. Texas A&M University; 2012. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2010-12-8814.

Council of Science Editors:

Robinson A. Development of a Biomarker and Clay Based Remediation Strategy for Populations at Risk for Fumonisin Toxicosis. [Doctoral Dissertation]. Texas A&M University; 2012. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2010-12-8814


Texas A&M University

5. Bijalwan, Megha. Pathogenesis of Theiler’s Murine Encephalomyelitis Virus (TMEV) in an Experimental Model of Epilepsy.

Degree: PhD, Biomedical Sciences, 2017, Texas A&M University

 Epilepsy is a complex neurological disease composed of two or more unprovoked seizures that occur due to aberrant neuronal hyperexcitability. Detailed analysis of the mechanisms… (more)

Subjects/Keywords: Epilepsy; immunology; virus; neurological disease

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APA (6th Edition):

Bijalwan, M. (2017). Pathogenesis of Theiler’s Murine Encephalomyelitis Virus (TMEV) in an Experimental Model of Epilepsy. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/173064

Chicago Manual of Style (16th Edition):

Bijalwan, Megha. “Pathogenesis of Theiler’s Murine Encephalomyelitis Virus (TMEV) in an Experimental Model of Epilepsy.” 2017. Doctoral Dissertation, Texas A&M University. Accessed April 17, 2021. http://hdl.handle.net/1969.1/173064.

MLA Handbook (7th Edition):

Bijalwan, Megha. “Pathogenesis of Theiler’s Murine Encephalomyelitis Virus (TMEV) in an Experimental Model of Epilepsy.” 2017. Web. 17 Apr 2021.

Vancouver:

Bijalwan M. Pathogenesis of Theiler’s Murine Encephalomyelitis Virus (TMEV) in an Experimental Model of Epilepsy. [Internet] [Doctoral dissertation]. Texas A&M University; 2017. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1969.1/173064.

Council of Science Editors:

Bijalwan M. Pathogenesis of Theiler’s Murine Encephalomyelitis Virus (TMEV) in an Experimental Model of Epilepsy. [Doctoral Dissertation]. Texas A&M University; 2017. Available from: http://hdl.handle.net/1969.1/173064


Texas A&M University

6. Roltsch, Emily 1983-. S100A1 and S100B: Novel Drug Targets for Alzheimer's Disease Therapy.

Degree: PhD, Veterinary Microbiology, 2011, Texas A&M University

 Numerous factors/gene products, including S100A1 and S100B, have been implicated in the onset and progression of Alzheimer's disease (AD). However, deciphering S100A1/S100B's role in AD… (more)

Subjects/Keywords: Alzheimer's disease; plaque load; immunotherapy; inflammation; microRNA; S100B; S100A1

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APA (6th Edition):

Roltsch, E. 1. (2011). S100A1 and S100B: Novel Drug Targets for Alzheimer's Disease Therapy. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/174180

Chicago Manual of Style (16th Edition):

Roltsch, Emily 1983-. “S100A1 and S100B: Novel Drug Targets for Alzheimer's Disease Therapy.” 2011. Doctoral Dissertation, Texas A&M University. Accessed April 17, 2021. http://hdl.handle.net/1969.1/174180.

MLA Handbook (7th Edition):

Roltsch, Emily 1983-. “S100A1 and S100B: Novel Drug Targets for Alzheimer's Disease Therapy.” 2011. Web. 17 Apr 2021.

Vancouver:

Roltsch E1. S100A1 and S100B: Novel Drug Targets for Alzheimer's Disease Therapy. [Internet] [Doctoral dissertation]. Texas A&M University; 2011. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1969.1/174180.

Council of Science Editors:

Roltsch E1. S100A1 and S100B: Novel Drug Targets for Alzheimer's Disease Therapy. [Doctoral Dissertation]. Texas A&M University; 2011. Available from: http://hdl.handle.net/1969.1/174180


Texas A&M University

7. Nigussie, Fikru. Analysis of Hippocampal Cell Proliferation, Survival, and Neuronal Morphology in P/Q-Type Voltage-Gated Calcium Channel Mutant Mice.

Degree: PhD, Biomedical Sciences, 2013, Texas A&M University

 Tottering and leaner mutant mice carry mutations in the pore-forming subunit (1A) of P/Q-type (CaV 2.1) voltage-gated calcium ion (Ca2+) channels that result in reduced… (more)

Subjects/Keywords: Tottering; Leaner; P/Q-type calcium channel; hippocampus

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APA (6th Edition):

Nigussie, F. (2013). Analysis of Hippocampal Cell Proliferation, Survival, and Neuronal Morphology in P/Q-Type Voltage-Gated Calcium Channel Mutant Mice. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/149268

Chicago Manual of Style (16th Edition):

Nigussie, Fikru. “Analysis of Hippocampal Cell Proliferation, Survival, and Neuronal Morphology in P/Q-Type Voltage-Gated Calcium Channel Mutant Mice.” 2013. Doctoral Dissertation, Texas A&M University. Accessed April 17, 2021. http://hdl.handle.net/1969.1/149268.

MLA Handbook (7th Edition):

Nigussie, Fikru. “Analysis of Hippocampal Cell Proliferation, Survival, and Neuronal Morphology in P/Q-Type Voltage-Gated Calcium Channel Mutant Mice.” 2013. Web. 17 Apr 2021.

Vancouver:

Nigussie F. Analysis of Hippocampal Cell Proliferation, Survival, and Neuronal Morphology in P/Q-Type Voltage-Gated Calcium Channel Mutant Mice. [Internet] [Doctoral dissertation]. Texas A&M University; 2013. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1969.1/149268.

Council of Science Editors:

Nigussie F. Analysis of Hippocampal Cell Proliferation, Survival, and Neuronal Morphology in P/Q-Type Voltage-Gated Calcium Channel Mutant Mice. [Doctoral Dissertation]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/149268


Texas A&M University

8. Huang, Pei-San. Effects of Chronic Nicotine Exposure and Lack of High Affinity Nicotinic Receptors on Cortico-Hippocampal Areas in the Aging Mouse Brain.

Degree: PhD, Biomedical Sciences, 2012, Texas A&M University

 Nicotine, the major psychoactive ingredient of tobacco smoke, underlies numerous effects by activating neuronal nicotinic acetylcholine receptors. Both in vitro and in vivo studies suggest… (more)

Subjects/Keywords: nicotine; neuroprotection; aging; nAChRs; neurodegeneration; caloric restriction

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APA (6th Edition):

Huang, P. (2012). Effects of Chronic Nicotine Exposure and Lack of High Affinity Nicotinic Receptors on Cortico-Hippocampal Areas in the Aging Mouse Brain. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2012-05-11067

Chicago Manual of Style (16th Edition):

Huang, Pei-San. “Effects of Chronic Nicotine Exposure and Lack of High Affinity Nicotinic Receptors on Cortico-Hippocampal Areas in the Aging Mouse Brain.” 2012. Doctoral Dissertation, Texas A&M University. Accessed April 17, 2021. http://hdl.handle.net/1969.1/ETD-TAMU-2012-05-11067.

MLA Handbook (7th Edition):

Huang, Pei-San. “Effects of Chronic Nicotine Exposure and Lack of High Affinity Nicotinic Receptors on Cortico-Hippocampal Areas in the Aging Mouse Brain.” 2012. Web. 17 Apr 2021.

Vancouver:

Huang P. Effects of Chronic Nicotine Exposure and Lack of High Affinity Nicotinic Receptors on Cortico-Hippocampal Areas in the Aging Mouse Brain. [Internet] [Doctoral dissertation]. Texas A&M University; 2012. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2012-05-11067.

Council of Science Editors:

Huang P. Effects of Chronic Nicotine Exposure and Lack of High Affinity Nicotinic Receptors on Cortico-Hippocampal Areas in the Aging Mouse Brain. [Doctoral Dissertation]. Texas A&M University; 2012. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2012-05-11067


Texas A&M University

9. Benjamin, Jade Marie. Understanding the Imprinting Mechanism of UBE3A for Therapeutic Intervention.

Degree: PhD, Genetics, 2017, Texas A&M University

 Human chromosome 15q11-q13 contains a cluster of imprinted genes that are associated with a number of neurological disorders that exhibit non-Mendelian patterns of inheritance, such… (more)

Subjects/Keywords: Angelman syndrome; 15q11-q13; neurodevelopmental disorders; bioinformatics; HTS

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APA (6th Edition):

Benjamin, J. M. (2017). Understanding the Imprinting Mechanism of UBE3A for Therapeutic Intervention. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/187202

Chicago Manual of Style (16th Edition):

Benjamin, Jade Marie. “Understanding the Imprinting Mechanism of UBE3A for Therapeutic Intervention.” 2017. Doctoral Dissertation, Texas A&M University. Accessed April 17, 2021. http://hdl.handle.net/1969.1/187202.

MLA Handbook (7th Edition):

Benjamin, Jade Marie. “Understanding the Imprinting Mechanism of UBE3A for Therapeutic Intervention.” 2017. Web. 17 Apr 2021.

Vancouver:

Benjamin JM. Understanding the Imprinting Mechanism of UBE3A for Therapeutic Intervention. [Internet] [Doctoral dissertation]. Texas A&M University; 2017. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1969.1/187202.

Council of Science Editors:

Benjamin JM. Understanding the Imprinting Mechanism of UBE3A for Therapeutic Intervention. [Doctoral Dissertation]. Texas A&M University; 2017. Available from: http://hdl.handle.net/1969.1/187202

10. Raymond, Elizabeth S. Effects of the Developmental Teratogens Ethanol and Nicotine on Long Noncoding RNA Expression in Mouse Fetal Neural Stem Cells of the Cortex.

Degree: MS, Neuroscience, 2016, Texas A&M University

 Research on long noncoding RNAs (lncRNAs) has increased significantly over the past few decades. However, investigation of lncRNA continues to lag behind that of other… (more)

Subjects/Keywords: long noncoding RNA; mouse stem cells; Ethanol; Nicotine

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APA (6th Edition):

Raymond, E. S. (2016). Effects of the Developmental Teratogens Ethanol and Nicotine on Long Noncoding RNA Expression in Mouse Fetal Neural Stem Cells of the Cortex. (Masters Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/158037

Chicago Manual of Style (16th Edition):

Raymond, Elizabeth S. “Effects of the Developmental Teratogens Ethanol and Nicotine on Long Noncoding RNA Expression in Mouse Fetal Neural Stem Cells of the Cortex.” 2016. Masters Thesis, Texas A&M University. Accessed April 17, 2021. http://hdl.handle.net/1969.1/158037.

MLA Handbook (7th Edition):

Raymond, Elizabeth S. “Effects of the Developmental Teratogens Ethanol and Nicotine on Long Noncoding RNA Expression in Mouse Fetal Neural Stem Cells of the Cortex.” 2016. Web. 17 Apr 2021.

Vancouver:

Raymond ES. Effects of the Developmental Teratogens Ethanol and Nicotine on Long Noncoding RNA Expression in Mouse Fetal Neural Stem Cells of the Cortex. [Internet] [Masters thesis]. Texas A&M University; 2016. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1969.1/158037.

Council of Science Editors:

Raymond ES. Effects of the Developmental Teratogens Ethanol and Nicotine on Long Noncoding RNA Expression in Mouse Fetal Neural Stem Cells of the Cortex. [Masters Thesis]. Texas A&M University; 2016. Available from: http://hdl.handle.net/1969.1/158037

11. Vichaya, Elisabeth Good. The impact of glial inhibition on the spinal instrumental learning paradigm.

Degree: MS, Psychology, 2009, Texas A&M University

 Although neural plasticity has traditionally been studied within the brain, evidence indicates that the spinal cord is quite plastic as well. Spinal neurons can even… (more)

Subjects/Keywords: glia; plasticity; spinal instrumental learning; fluorocitrate; minocycline

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APA (6th Edition):

Vichaya, E. G. (2009). The impact of glial inhibition on the spinal instrumental learning paradigm. (Masters Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-1606

Chicago Manual of Style (16th Edition):

Vichaya, Elisabeth Good. “The impact of glial inhibition on the spinal instrumental learning paradigm.” 2009. Masters Thesis, Texas A&M University. Accessed April 17, 2021. http://hdl.handle.net/1969.1/ETD-TAMU-1606.

MLA Handbook (7th Edition):

Vichaya, Elisabeth Good. “The impact of glial inhibition on the spinal instrumental learning paradigm.” 2009. Web. 17 Apr 2021.

Vancouver:

Vichaya EG. The impact of glial inhibition on the spinal instrumental learning paradigm. [Internet] [Masters thesis]. Texas A&M University; 2009. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-1606.

Council of Science Editors:

Vichaya EG. The impact of glial inhibition on the spinal instrumental learning paradigm. [Masters Thesis]. Texas A&M University; 2009. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-1606

12. Lu, Hsueh-Chung. The Role of Myeloid STAT3 in Experimental Autoimmune Encephalomyelitis and Postnatal Brain Development.

Degree: PhD, Neuroscience, 2017, Texas A&M University

 Multiple sclerosis (MS) is a debilitating disease characterized by demyelination and neurodegeneration in the CNS. Both activated microglia and infiltrating myeloid cells are thought to… (more)

Subjects/Keywords: STAT3; Multiple Sclerosis; Experimental Autoimmune Encephalomyelitis; Demyelination

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APA (6th Edition):

Lu, H. (2017). The Role of Myeloid STAT3 in Experimental Autoimmune Encephalomyelitis and Postnatal Brain Development. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/165986

Chicago Manual of Style (16th Edition):

Lu, Hsueh-Chung. “The Role of Myeloid STAT3 in Experimental Autoimmune Encephalomyelitis and Postnatal Brain Development.” 2017. Doctoral Dissertation, Texas A&M University. Accessed April 17, 2021. http://hdl.handle.net/1969.1/165986.

MLA Handbook (7th Edition):

Lu, Hsueh-Chung. “The Role of Myeloid STAT3 in Experimental Autoimmune Encephalomyelitis and Postnatal Brain Development.” 2017. Web. 17 Apr 2021.

Vancouver:

Lu H. The Role of Myeloid STAT3 in Experimental Autoimmune Encephalomyelitis and Postnatal Brain Development. [Internet] [Doctoral dissertation]. Texas A&M University; 2017. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1969.1/165986.

Council of Science Editors:

Lu H. The Role of Myeloid STAT3 in Experimental Autoimmune Encephalomyelitis and Postnatal Brain Development. [Doctoral Dissertation]. Texas A&M University; 2017. Available from: http://hdl.handle.net/1969.1/165986

13. Vichaya, Elisabeth Good. The Impact of Social Stress on Central Nervous System Inflammation and T Cell Response to Theiler’s Virus Infection.

Degree: PhD, Psychology, 2012, Texas A&M University

 A growing body of evidence suggests that social stress contributes to the pathogenesis of neurodegenerative diseases, such as multiple sclerosis (MS). For example, prior research… (more)

Subjects/Keywords: multiple sclerosis; Theiler's Virus; TMEV; social stress; IL-6; neuroinflammation; T cells

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APA (6th Edition):

Vichaya, E. G. (2012). The Impact of Social Stress on Central Nervous System Inflammation and T Cell Response to Theiler’s Virus Infection. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2011-05-9139

Chicago Manual of Style (16th Edition):

Vichaya, Elisabeth Good. “The Impact of Social Stress on Central Nervous System Inflammation and T Cell Response to Theiler’s Virus Infection.” 2012. Doctoral Dissertation, Texas A&M University. Accessed April 17, 2021. http://hdl.handle.net/1969.1/ETD-TAMU-2011-05-9139.

MLA Handbook (7th Edition):

Vichaya, Elisabeth Good. “The Impact of Social Stress on Central Nervous System Inflammation and T Cell Response to Theiler’s Virus Infection.” 2012. Web. 17 Apr 2021.

Vancouver:

Vichaya EG. The Impact of Social Stress on Central Nervous System Inflammation and T Cell Response to Theiler’s Virus Infection. [Internet] [Doctoral dissertation]. Texas A&M University; 2012. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2011-05-9139.

Council of Science Editors:

Vichaya EG. The Impact of Social Stress on Central Nervous System Inflammation and T Cell Response to Theiler’s Virus Infection. [Doctoral Dissertation]. Texas A&M University; 2012. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2011-05-9139

14. Wright, Rachel. Characterization of RPGR Variants and Their Role in Inherited Retinal Degeneration.

Degree: PhD, Genetics, 2012, Texas A&M University

 Retinitis Pigmentosa (RP) refers to a group of inherited retinal dystrophies resulting from progressive photoreceptor degeneration and accumulation of intra-retinal pigment-like deposits. X-linked forms of… (more)

Subjects/Keywords: RPGR; Retinitis Pigmentosa; RP; Usher Syndrome; Retinal Degeneration

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APA (6th Edition):

Wright, R. (2012). Characterization of RPGR Variants and Their Role in Inherited Retinal Degeneration. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2011-08-9804

Chicago Manual of Style (16th Edition):

Wright, Rachel. “Characterization of RPGR Variants and Their Role in Inherited Retinal Degeneration.” 2012. Doctoral Dissertation, Texas A&M University. Accessed April 17, 2021. http://hdl.handle.net/1969.1/ETD-TAMU-2011-08-9804.

MLA Handbook (7th Edition):

Wright, Rachel. “Characterization of RPGR Variants and Their Role in Inherited Retinal Degeneration.” 2012. Web. 17 Apr 2021.

Vancouver:

Wright R. Characterization of RPGR Variants and Their Role in Inherited Retinal Degeneration. [Internet] [Doctoral dissertation]. Texas A&M University; 2012. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2011-08-9804.

Council of Science Editors:

Wright R. Characterization of RPGR Variants and Their Role in Inherited Retinal Degeneration. [Doctoral Dissertation]. Texas A&M University; 2012. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2011-08-9804

15. Bawa, Bhupinder. Neurodegeneration in cerebellar granule cells of p/q type voltage gated calcium channel mutant leaner mice.

Degree: PhD, Veterinary Anatomy, 2009, Texas A&M University

 Mutations of the α1A subunit of CaV 2.1 voltage gated calcium (VGCC) channels are responsible for several inherited disorders affecting humans, including familial hemiplegic migraine,… (more)

Subjects/Keywords: Neurodegeneration; cerebellum; Leaner; calcium channels

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APA (6th Edition):

Bawa, B. (2009). Neurodegeneration in cerebellar granule cells of p/q type voltage gated calcium channel mutant leaner mice. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2609

Chicago Manual of Style (16th Edition):

Bawa, Bhupinder. “Neurodegeneration in cerebellar granule cells of p/q type voltage gated calcium channel mutant leaner mice.” 2009. Doctoral Dissertation, Texas A&M University. Accessed April 17, 2021. http://hdl.handle.net/1969.1/ETD-TAMU-2609.

MLA Handbook (7th Edition):

Bawa, Bhupinder. “Neurodegeneration in cerebellar granule cells of p/q type voltage gated calcium channel mutant leaner mice.” 2009. Web. 17 Apr 2021.

Vancouver:

Bawa B. Neurodegeneration in cerebellar granule cells of p/q type voltage gated calcium channel mutant leaner mice. [Internet] [Doctoral dissertation]. Texas A&M University; 2009. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2609.

Council of Science Editors:

Bawa B. Neurodegeneration in cerebellar granule cells of p/q type voltage gated calcium channel mutant leaner mice. [Doctoral Dissertation]. Texas A&M University; 2009. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2609


Texas A&M University

16. Sieve, Amy Nicole. The effects of psychological stress on an animal model of multiple sclerosis, Theiler's virus induced demyelination.

Degree: PhD, Psychology, 2005, Texas A&M University

 Multiple Sclerosis (MS) is the most common demyelinating condition of the central nervous system (CNS), resulting in paralysis and death. The etiology of MS is… (more)

Subjects/Keywords: stress; Multiple Sclerosis; sex differences; Theiler's Virus; demyelination; autoimmunity

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APA (6th Edition):

Sieve, A. N. (2005). The effects of psychological stress on an animal model of multiple sclerosis, Theiler's virus induced demyelination. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/1558

Chicago Manual of Style (16th Edition):

Sieve, Amy Nicole. “The effects of psychological stress on an animal model of multiple sclerosis, Theiler's virus induced demyelination.” 2005. Doctoral Dissertation, Texas A&M University. Accessed April 17, 2021. http://hdl.handle.net/1969.1/1558.

MLA Handbook (7th Edition):

Sieve, Amy Nicole. “The effects of psychological stress on an animal model of multiple sclerosis, Theiler's virus induced demyelination.” 2005. Web. 17 Apr 2021.

Vancouver:

Sieve AN. The effects of psychological stress on an animal model of multiple sclerosis, Theiler's virus induced demyelination. [Internet] [Doctoral dissertation]. Texas A&M University; 2005. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1969.1/1558.

Council of Science Editors:

Sieve AN. The effects of psychological stress on an animal model of multiple sclerosis, Theiler's virus induced demyelination. [Doctoral Dissertation]. Texas A&M University; 2005. Available from: http://hdl.handle.net/1969.1/1558


Texas A&M University

17. Steelman, Andrew Jonathan. The Effects of Chronic Restraint Stress on Innate and Adaptive Immune Responses to Acute Theiler?s Murine Encephalomyelitis Virus Infection ? An Animal Model of Human Multiple Sclerosis.

Degree: PhD, Biomedical Sciences, 2009, Texas A&M University

 Multiple sclerosis (MS) is an immune-mediated prevalent chronic demyelinating and neurodegenerative disease of the central nervous system that begins with an abrupt onset during early… (more)

Subjects/Keywords: Theiler's virus; Multiple Sclerosis; Restraint stress; T-cell; B-cell; Immune response

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APA (6th Edition):

Steelman, A. J. (2009). The Effects of Chronic Restraint Stress on Innate and Adaptive Immune Responses to Acute Theiler?s Murine Encephalomyelitis Virus Infection ? An Animal Model of Human Multiple Sclerosis. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2661

Chicago Manual of Style (16th Edition):

Steelman, Andrew Jonathan. “The Effects of Chronic Restraint Stress on Innate and Adaptive Immune Responses to Acute Theiler?s Murine Encephalomyelitis Virus Infection ? An Animal Model of Human Multiple Sclerosis.” 2009. Doctoral Dissertation, Texas A&M University. Accessed April 17, 2021. http://hdl.handle.net/1969.1/ETD-TAMU-2661.

MLA Handbook (7th Edition):

Steelman, Andrew Jonathan. “The Effects of Chronic Restraint Stress on Innate and Adaptive Immune Responses to Acute Theiler?s Murine Encephalomyelitis Virus Infection ? An Animal Model of Human Multiple Sclerosis.” 2009. Web. 17 Apr 2021.

Vancouver:

Steelman AJ. The Effects of Chronic Restraint Stress on Innate and Adaptive Immune Responses to Acute Theiler?s Murine Encephalomyelitis Virus Infection ? An Animal Model of Human Multiple Sclerosis. [Internet] [Doctoral dissertation]. Texas A&M University; 2009. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2661.

Council of Science Editors:

Steelman AJ. The Effects of Chronic Restraint Stress on Innate and Adaptive Immune Responses to Acute Theiler?s Murine Encephalomyelitis Virus Infection ? An Animal Model of Human Multiple Sclerosis. [Doctoral Dissertation]. Texas A&M University; 2009. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2661


Texas A&M University

18. Arenas Gamboa, Angela Maria. Evaluation of microencapsulation as an improved vaccination strategy against brucellosis.

Degree: PhD, Veterinary Microbiology, 2009, Texas A&M University

 Brucellosis is an important zoonotic disease of nearly worldwide distribution. Despite the availability of live vaccine strains for bovine (S19, RB51) and small ruminants (Rev… (more)

Subjects/Keywords: Brucella; Microencapsulation

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APA (6th Edition):

Arenas Gamboa, A. M. (2009). Evaluation of microencapsulation as an improved vaccination strategy against brucellosis. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-1384

Chicago Manual of Style (16th Edition):

Arenas Gamboa, Angela Maria. “Evaluation of microencapsulation as an improved vaccination strategy against brucellosis.” 2009. Doctoral Dissertation, Texas A&M University. Accessed April 17, 2021. http://hdl.handle.net/1969.1/ETD-TAMU-1384.

MLA Handbook (7th Edition):

Arenas Gamboa, Angela Maria. “Evaluation of microencapsulation as an improved vaccination strategy against brucellosis.” 2009. Web. 17 Apr 2021.

Vancouver:

Arenas Gamboa AM. Evaluation of microencapsulation as an improved vaccination strategy against brucellosis. [Internet] [Doctoral dissertation]. Texas A&M University; 2009. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-1384.

Council of Science Editors:

Arenas Gamboa AM. Evaluation of microencapsulation as an improved vaccination strategy against brucellosis. [Doctoral Dissertation]. Texas A&M University; 2009. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-1384


Texas A&M University

19. Ferguson, Adam Richard. Neuropathic pain and the inhibition of learning within the spinal cord.

Degree: PhD, Psychology, 2004, Texas A&M University

 Prior work from our laboratory has shown that the spinal cord is capable of supporting a simple form of instrumental (response-outcome) learning. In a typical… (more)

Subjects/Keywords: spinal learning; instrumental learning; formalin; inflammation; central sensitization; plasticity; glutamate; neuropathic pain

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APA (6th Edition):

Ferguson, A. R. (2004). Neuropathic pain and the inhibition of learning within the spinal cord. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/350

Chicago Manual of Style (16th Edition):

Ferguson, Adam Richard. “Neuropathic pain and the inhibition of learning within the spinal cord.” 2004. Doctoral Dissertation, Texas A&M University. Accessed April 17, 2021. http://hdl.handle.net/1969.1/350.

MLA Handbook (7th Edition):

Ferguson, Adam Richard. “Neuropathic pain and the inhibition of learning within the spinal cord.” 2004. Web. 17 Apr 2021.

Vancouver:

Ferguson AR. Neuropathic pain and the inhibition of learning within the spinal cord. [Internet] [Doctoral dissertation]. Texas A&M University; 2004. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1969.1/350.

Council of Science Editors:

Ferguson AR. Neuropathic pain and the inhibition of learning within the spinal cord. [Doctoral Dissertation]. Texas A&M University; 2004. Available from: http://hdl.handle.net/1969.1/350


Texas A&M University

20. Lim, Wah-Seng. Differential cytokine mRNA expression induced by binding of virulent and avirulent molecularly cloned equine infectious anemia viruses to equine macrophages.

Degree: PhD, Veterinary Microbiology, 2004, Texas A&M University

 Equine infectious anemia virus (EIAV) causes rapid development of acute disease followed by recurring episodes of fever, thrombocytopenia and viremia, termed chronic EIA. Most infected… (more)

Subjects/Keywords: equine infectious anemia viruse; macrophage; cytokine; virulence

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APA (6th Edition):

Lim, W. (2004). Differential cytokine mRNA expression induced by binding of virulent and avirulent molecularly cloned equine infectious anemia viruses to equine macrophages. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/1225

Chicago Manual of Style (16th Edition):

Lim, Wah-Seng. “Differential cytokine mRNA expression induced by binding of virulent and avirulent molecularly cloned equine infectious anemia viruses to equine macrophages.” 2004. Doctoral Dissertation, Texas A&M University. Accessed April 17, 2021. http://hdl.handle.net/1969.1/1225.

MLA Handbook (7th Edition):

Lim, Wah-Seng. “Differential cytokine mRNA expression induced by binding of virulent and avirulent molecularly cloned equine infectious anemia viruses to equine macrophages.” 2004. Web. 17 Apr 2021.

Vancouver:

Lim W. Differential cytokine mRNA expression induced by binding of virulent and avirulent molecularly cloned equine infectious anemia viruses to equine macrophages. [Internet] [Doctoral dissertation]. Texas A&M University; 2004. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1969.1/1225.

Council of Science Editors:

Lim W. Differential cytokine mRNA expression induced by binding of virulent and avirulent molecularly cloned equine infectious anemia viruses to equine macrophages. [Doctoral Dissertation]. Texas A&M University; 2004. Available from: http://hdl.handle.net/1969.1/1225


Texas A&M University

21. Ly, Lan H. Immunosuppressive dietary n-3 polyunsaturated fatty acids differentially modulate costimulatory regulation of murine CD4+ T-cell function.

Degree: PhD, Nutrition, 2005, Texas A&M University

 Consumption of fish oils (FO) enriched with the n-3 polyunsaturated fatty acids (PUFA), docosahexaenoic acid (DHA) and eicosapentaenoic acid (EPA), is beneficial to a variety… (more)

Subjects/Keywords: Mouse; Diet; T-lymphocytes; Immunity

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APA (6th Edition):

Ly, L. H. (2005). Immunosuppressive dietary n-3 polyunsaturated fatty acids differentially modulate costimulatory regulation of murine CD4+ T-cell function. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/1535

Chicago Manual of Style (16th Edition):

Ly, Lan H. “Immunosuppressive dietary n-3 polyunsaturated fatty acids differentially modulate costimulatory regulation of murine CD4+ T-cell function.” 2005. Doctoral Dissertation, Texas A&M University. Accessed April 17, 2021. http://hdl.handle.net/1969.1/1535.

MLA Handbook (7th Edition):

Ly, Lan H. “Immunosuppressive dietary n-3 polyunsaturated fatty acids differentially modulate costimulatory regulation of murine CD4+ T-cell function.” 2005. Web. 17 Apr 2021.

Vancouver:

Ly LH. Immunosuppressive dietary n-3 polyunsaturated fatty acids differentially modulate costimulatory regulation of murine CD4+ T-cell function. [Internet] [Doctoral dissertation]. Texas A&M University; 2005. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1969.1/1535.

Council of Science Editors:

Ly LH. Immunosuppressive dietary n-3 polyunsaturated fatty acids differentially modulate costimulatory regulation of murine CD4+ T-cell function. [Doctoral Dissertation]. Texas A&M University; 2005. Available from: http://hdl.handle.net/1969.1/1535


Texas A&M University

22. Gull, Tamara Brownsey. In vivo infection biology of contagious bovine pleuropneumonia.

Degree: PhD, Veterinary Microbiology, 2009, Texas A&M University

 Contagious Bovine Pleuropneumonia (CBPP), caused by Mycoplasma mycoides mycoides small colony (MmmSC), is a devastating respiratory disease of cattle in Africa, Asia and the Middle… (more)

Subjects/Keywords: Contagious Bovine Pleuropneumonia; Mycoplasma mycoides mycoides small colony; cattle; infectious disease; foreign animal disease

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APA (6th Edition):

Gull, T. B. (2009). In vivo infection biology of contagious bovine pleuropneumonia. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2611

Chicago Manual of Style (16th Edition):

Gull, Tamara Brownsey. “In vivo infection biology of contagious bovine pleuropneumonia.” 2009. Doctoral Dissertation, Texas A&M University. Accessed April 17, 2021. http://hdl.handle.net/1969.1/ETD-TAMU-2611.

MLA Handbook (7th Edition):

Gull, Tamara Brownsey. “In vivo infection biology of contagious bovine pleuropneumonia.” 2009. Web. 17 Apr 2021.

Vancouver:

Gull TB. In vivo infection biology of contagious bovine pleuropneumonia. [Internet] [Doctoral dissertation]. Texas A&M University; 2009. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2611.

Council of Science Editors:

Gull TB. In vivo infection biology of contagious bovine pleuropneumonia. [Doctoral Dissertation]. Texas A&M University; 2009. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2611


Texas A&M University

23. Frank-Cannon, Tamy Catherine. Cerebellar Purkinje cell death in the P/Q -type voltage-gated calcium ion channel mutant mouse, leaner.

Degree: PhD, Veterinary Anatomy, 2006, Texas A&M University

 Mutations of the á1A subunit of P/Q-type voltage-gated calcium channels are responsible for several inherited disorders affecting humans, including familial hemiplegic migraine, episodic ataxia type… (more)

Subjects/Keywords: cerebellum; Purkinje cell; cell death; neuron; leaner mouse

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APA (6th Edition):

Frank-Cannon, T. C. (2006). Cerebellar Purkinje cell death in the P/Q -type voltage-gated calcium ion channel mutant mouse, leaner. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/3321

Chicago Manual of Style (16th Edition):

Frank-Cannon, Tamy Catherine. “Cerebellar Purkinje cell death in the P/Q -type voltage-gated calcium ion channel mutant mouse, leaner.” 2006. Doctoral Dissertation, Texas A&M University. Accessed April 17, 2021. http://hdl.handle.net/1969.1/3321.

MLA Handbook (7th Edition):

Frank-Cannon, Tamy Catherine. “Cerebellar Purkinje cell death in the P/Q -type voltage-gated calcium ion channel mutant mouse, leaner.” 2006. Web. 17 Apr 2021.

Vancouver:

Frank-Cannon TC. Cerebellar Purkinje cell death in the P/Q -type voltage-gated calcium ion channel mutant mouse, leaner. [Internet] [Doctoral dissertation]. Texas A&M University; 2006. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1969.1/3321.

Council of Science Editors:

Frank-Cannon TC. Cerebellar Purkinje cell death in the P/Q -type voltage-gated calcium ion channel mutant mouse, leaner. [Doctoral Dissertation]. Texas A&M University; 2006. Available from: http://hdl.handle.net/1969.1/3321


Texas A&M University

24. Mi, Wentao. The effect of stress on the neuropathogenesis of Theiler's virus-induced demyelination as an animal model of multiple sclerosis.

Degree: PhD, Genetics, 2006, Texas A&M University

 Stressful life events have been associated with the onset and/or exacerbation of multiple sclerosis (MS). To investigate the effects of stress on the pathogenesis of… (more)

Subjects/Keywords: stress; Theiler's virus; demyelination; multiple sclerosis

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APA (6th Edition):

Mi, W. (2006). The effect of stress on the neuropathogenesis of Theiler's virus-induced demyelination as an animal model of multiple sclerosis. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/4255

Chicago Manual of Style (16th Edition):

Mi, Wentao. “The effect of stress on the neuropathogenesis of Theiler's virus-induced demyelination as an animal model of multiple sclerosis.” 2006. Doctoral Dissertation, Texas A&M University. Accessed April 17, 2021. http://hdl.handle.net/1969.1/4255.

MLA Handbook (7th Edition):

Mi, Wentao. “The effect of stress on the neuropathogenesis of Theiler's virus-induced demyelination as an animal model of multiple sclerosis.” 2006. Web. 17 Apr 2021.

Vancouver:

Mi W. The effect of stress on the neuropathogenesis of Theiler's virus-induced demyelination as an animal model of multiple sclerosis. [Internet] [Doctoral dissertation]. Texas A&M University; 2006. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1969.1/4255.

Council of Science Editors:

Mi W. The effect of stress on the neuropathogenesis of Theiler's virus-induced demyelination as an animal model of multiple sclerosis. [Doctoral Dissertation]. Texas A&M University; 2006. Available from: http://hdl.handle.net/1969.1/4255


Texas A&M University

25. Johnson, Robin Ranee. Social stress exacerbations on acute Theiler's virus infection: a role for Interleukin-6.

Degree: PhD, Psychology, 2006, Texas A&M University

 Neurodegenerative diseases, such as multiple sclerosis (MS), are adversely affected by both stress and inflammation. Theiler's murine encephalomyelitis virus infection is an excellent animal model… (more)

Subjects/Keywords: social stress; TMEV; cytokines; motor impairment

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APA (6th Edition):

Johnson, R. R. (2006). Social stress exacerbations on acute Theiler's virus infection: a role for Interleukin-6. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/4362

Chicago Manual of Style (16th Edition):

Johnson, Robin Ranee. “Social stress exacerbations on acute Theiler's virus infection: a role for Interleukin-6.” 2006. Doctoral Dissertation, Texas A&M University. Accessed April 17, 2021. http://hdl.handle.net/1969.1/4362.

MLA Handbook (7th Edition):

Johnson, Robin Ranee. “Social stress exacerbations on acute Theiler's virus infection: a role for Interleukin-6.” 2006. Web. 17 Apr 2021.

Vancouver:

Johnson RR. Social stress exacerbations on acute Theiler's virus infection: a role for Interleukin-6. [Internet] [Doctoral dissertation]. Texas A&M University; 2006. [cited 2021 Apr 17]. Available from: http://hdl.handle.net/1969.1/4362.

Council of Science Editors:

Johnson RR. Social stress exacerbations on acute Theiler's virus infection: a role for Interleukin-6. [Doctoral Dissertation]. Texas A&M University; 2006. Available from: http://hdl.handle.net/1969.1/4362

.