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You searched for +publisher:"Texas A&M University" +contributor:("Safe, Stephen"). Showing records 1 – 30 of 73 total matches.

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Texas A&M University

1. Kanameni, Srikanth. Transcriptional Profiling of Polarized Macrophages using RNA-Sequencing.

Degree: MS, Biomedical Sciences, 2015, Texas A&M University

 Adipose tissue macrophages (ATMs) are pivotal regulators for adipose tissue function, specifically contributing to the homeostasis of the adipose niche. Significantly increased ATMs and their… (more)

Subjects/Keywords: Macrophages; RNA-Sequencing

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APA (6th Edition):

Kanameni, S. (2015). Transcriptional Profiling of Polarized Macrophages using RNA-Sequencing. (Masters Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/155022

Chicago Manual of Style (16th Edition):

Kanameni, Srikanth. “Transcriptional Profiling of Polarized Macrophages using RNA-Sequencing.” 2015. Masters Thesis, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/155022.

MLA Handbook (7th Edition):

Kanameni, Srikanth. “Transcriptional Profiling of Polarized Macrophages using RNA-Sequencing.” 2015. Web. 26 Oct 2020.

Vancouver:

Kanameni S. Transcriptional Profiling of Polarized Macrophages using RNA-Sequencing. [Internet] [Masters thesis]. Texas A&M University; 2015. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/155022.

Council of Science Editors:

Kanameni S. Transcriptional Profiling of Polarized Macrophages using RNA-Sequencing. [Masters Thesis]. Texas A&M University; 2015. Available from: http://hdl.handle.net/1969.1/155022


Texas A&M University

2. Morin, Andrew. Characterization of Cas9-Mediated microRNA Knockout.

Degree: MS, Biomedical Sciences, 2015, Texas A&M University

 MicroRNAs are short regulatory RNAs that primarily operate at the posttranscriptional level, acting as part of the RISC complex to destabilize mRNA prior to, or… (more)

Subjects/Keywords: CRISPR/Cas9; microRNA

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APA (6th Edition):

Morin, A. (2015). Characterization of Cas9-Mediated microRNA Knockout. (Masters Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/156200

Chicago Manual of Style (16th Edition):

Morin, Andrew. “Characterization of Cas9-Mediated microRNA Knockout.” 2015. Masters Thesis, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/156200.

MLA Handbook (7th Edition):

Morin, Andrew. “Characterization of Cas9-Mediated microRNA Knockout.” 2015. Web. 26 Oct 2020.

Vancouver:

Morin A. Characterization of Cas9-Mediated microRNA Knockout. [Internet] [Masters thesis]. Texas A&M University; 2015. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/156200.

Council of Science Editors:

Morin A. Characterization of Cas9-Mediated microRNA Knockout. [Masters Thesis]. Texas A&M University; 2015. Available from: http://hdl.handle.net/1969.1/156200


Texas A&M University

3. Banerjee, Nivedita. Polyphenol-induced Anti-inflammatory and Cytotoxic Activities in Breast and Colon Cancer: Potential Role of miRNA's in Cell Survival and Inflammation.

Degree: PhD, Toxicology, 2013, Texas A&M University

 Cancer is a major cause of death worldwide. Hence, there is a great need to develop novel therapeutic agents for the prevention and treatment of… (more)

Subjects/Keywords: Polyphenol; Polyphenolics; Cancer; miRNA; cytotoxicity

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APA (6th Edition):

Banerjee, N. (2013). Polyphenol-induced Anti-inflammatory and Cytotoxic Activities in Breast and Colon Cancer: Potential Role of miRNA's in Cell Survival and Inflammation. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/151661

Chicago Manual of Style (16th Edition):

Banerjee, Nivedita. “Polyphenol-induced Anti-inflammatory and Cytotoxic Activities in Breast and Colon Cancer: Potential Role of miRNA's in Cell Survival and Inflammation.” 2013. Doctoral Dissertation, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/151661.

MLA Handbook (7th Edition):

Banerjee, Nivedita. “Polyphenol-induced Anti-inflammatory and Cytotoxic Activities in Breast and Colon Cancer: Potential Role of miRNA's in Cell Survival and Inflammation.” 2013. Web. 26 Oct 2020.

Vancouver:

Banerjee N. Polyphenol-induced Anti-inflammatory and Cytotoxic Activities in Breast and Colon Cancer: Potential Role of miRNA's in Cell Survival and Inflammation. [Internet] [Doctoral dissertation]. Texas A&M University; 2013. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/151661.

Council of Science Editors:

Banerjee N. Polyphenol-induced Anti-inflammatory and Cytotoxic Activities in Breast and Colon Cancer: Potential Role of miRNA's in Cell Survival and Inflammation. [Doctoral Dissertation]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/151661


Texas A&M University

4. Gumuser, Esra D. Receptor Ubiquitination Regulates Il-5rα Function.

Degree: MS, Medical Sciences, 2016, Texas A&M University

 Eosinophils are multifunctional leukocytes implicated in the pathogenesis of inflammatory processes including hypereosinophilic syndrome, eosinophilic esophagitis, and allergic asthma. Due to its role in the… (more)

Subjects/Keywords: Eosinophils; inflammation; cytokine receptors; IL-5; IL-5Rα; βc; ubiquitination; JAK kinases

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APA (6th Edition):

Gumuser, E. D. (2016). Receptor Ubiquitination Regulates Il-5rα Function. (Masters Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/156973

Chicago Manual of Style (16th Edition):

Gumuser, Esra D. “Receptor Ubiquitination Regulates Il-5rα Function.” 2016. Masters Thesis, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/156973.

MLA Handbook (7th Edition):

Gumuser, Esra D. “Receptor Ubiquitination Regulates Il-5rα Function.” 2016. Web. 26 Oct 2020.

Vancouver:

Gumuser ED. Receptor Ubiquitination Regulates Il-5rα Function. [Internet] [Masters thesis]. Texas A&M University; 2016. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/156973.

Council of Science Editors:

Gumuser ED. Receptor Ubiquitination Regulates Il-5rα Function. [Masters Thesis]. Texas A&M University; 2016. Available from: http://hdl.handle.net/1969.1/156973


Texas A&M University

5. Eagleton, Navada Lorraine. Novel Functions for the Pregnane X Receptor include Regulation of mRNA Turnover and Involvement in Colon Cancer Progression.

Degree: MS, Toxicology, 2011, Texas A&M University

 To understand the mechanisms of transcriptional regulation of PXR, we performed yeast two-hybrid screenings to search for PXR-interacting proteins in a human liver cDNA library… (more)

Subjects/Keywords: pregnane X receptor; PXR; CNOT2; colon cancer

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APA (6th Edition):

Eagleton, N. L. (2011). Novel Functions for the Pregnane X Receptor include Regulation of mRNA Turnover and Involvement in Colon Cancer Progression. (Masters Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2010-08-8029

Chicago Manual of Style (16th Edition):

Eagleton, Navada Lorraine. “Novel Functions for the Pregnane X Receptor include Regulation of mRNA Turnover and Involvement in Colon Cancer Progression.” 2011. Masters Thesis, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/ETD-TAMU-2010-08-8029.

MLA Handbook (7th Edition):

Eagleton, Navada Lorraine. “Novel Functions for the Pregnane X Receptor include Regulation of mRNA Turnover and Involvement in Colon Cancer Progression.” 2011. Web. 26 Oct 2020.

Vancouver:

Eagleton NL. Novel Functions for the Pregnane X Receptor include Regulation of mRNA Turnover and Involvement in Colon Cancer Progression. [Internet] [Masters thesis]. Texas A&M University; 2011. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2010-08-8029.

Council of Science Editors:

Eagleton NL. Novel Functions for the Pregnane X Receptor include Regulation of mRNA Turnover and Involvement in Colon Cancer Progression. [Masters Thesis]. Texas A&M University; 2011. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2010-08-8029


Texas A&M University

6. Berg, James Michael. A Big Response to a “Small” Problem: Identifying the Oxidative Potential of Nanomaterials and the Physicochemical Characteristics That Play a Role.

Degree: PhD, Toxicology, 2012, Texas A&M University

 Nanotechnology as a science is emerging rapidly. As materials are synthesized and utilized at the nanometer size scale, concerns of potential health and safety effects… (more)

Subjects/Keywords: Nanotoxicology; Oxidative Stress; Nanoparticle; Mixtures Nanotoxicology; Zeta Potential; NRF2

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APA (6th Edition):

Berg, J. M. (2012). A Big Response to a “Small” Problem: Identifying the Oxidative Potential of Nanomaterials and the Physicochemical Characteristics That Play a Role. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2011-12-10234

Chicago Manual of Style (16th Edition):

Berg, James Michael. “A Big Response to a “Small” Problem: Identifying the Oxidative Potential of Nanomaterials and the Physicochemical Characteristics That Play a Role.” 2012. Doctoral Dissertation, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/ETD-TAMU-2011-12-10234.

MLA Handbook (7th Edition):

Berg, James Michael. “A Big Response to a “Small” Problem: Identifying the Oxidative Potential of Nanomaterials and the Physicochemical Characteristics That Play a Role.” 2012. Web. 26 Oct 2020.

Vancouver:

Berg JM. A Big Response to a “Small” Problem: Identifying the Oxidative Potential of Nanomaterials and the Physicochemical Characteristics That Play a Role. [Internet] [Doctoral dissertation]. Texas A&M University; 2012. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2011-12-10234.

Council of Science Editors:

Berg JM. A Big Response to a “Small” Problem: Identifying the Oxidative Potential of Nanomaterials and the Physicochemical Characteristics That Play a Role. [Doctoral Dissertation]. Texas A&M University; 2012. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2011-12-10234


Texas A&M University

7. Wang, Haiqing. Preconception Paternal Alcohol Exposure Disrupts Placental Gene Expression and Alters Chromatin Boundary Elements.

Degree: MS, Biomedical Sciences, 2017, Texas A&M University

 Fetal alcohol spectrum disorders (FASD) are a spectrum of pathophysiological consequences that include structural, neurological and behavior disorders that arise following prenatal alcohol exposure (PAE).… (more)

Subjects/Keywords: paternal; FASD; CTCF

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APA (6th Edition):

Wang, H. (2017). Preconception Paternal Alcohol Exposure Disrupts Placental Gene Expression and Alters Chromatin Boundary Elements. (Masters Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/161417

Chicago Manual of Style (16th Edition):

Wang, Haiqing. “Preconception Paternal Alcohol Exposure Disrupts Placental Gene Expression and Alters Chromatin Boundary Elements.” 2017. Masters Thesis, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/161417.

MLA Handbook (7th Edition):

Wang, Haiqing. “Preconception Paternal Alcohol Exposure Disrupts Placental Gene Expression and Alters Chromatin Boundary Elements.” 2017. Web. 26 Oct 2020.

Vancouver:

Wang H. Preconception Paternal Alcohol Exposure Disrupts Placental Gene Expression and Alters Chromatin Boundary Elements. [Internet] [Masters thesis]. Texas A&M University; 2017. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/161417.

Council of Science Editors:

Wang H. Preconception Paternal Alcohol Exposure Disrupts Placental Gene Expression and Alters Chromatin Boundary Elements. [Masters Thesis]. Texas A&M University; 2017. Available from: http://hdl.handle.net/1969.1/161417


Texas A&M University

8. Al Muhisen, Hadil Mahdi J. Sex Differences in Fetal Neural Stem Cells’ Response to Ethanol.

Degree: MS, Toxicology, 2018, Texas A&M University

 Fetal Alcohol Spectrum Disorders (FASDs) can result from prenatal exposure to alcohol. Alcohol is a known teratogen and in utero exposure is the leading non-genetic… (more)

Subjects/Keywords: Fetal Alcohol Spectrum Disorders; FASD; Sex Differences; Neural Stem Cells; Intellectual Disability; Prenatal Alcohol Exposure; PAE

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APA (6th Edition):

Al Muhisen, H. M. J. (2018). Sex Differences in Fetal Neural Stem Cells’ Response to Ethanol. (Masters Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/174164

Chicago Manual of Style (16th Edition):

Al Muhisen, Hadil Mahdi J. “Sex Differences in Fetal Neural Stem Cells’ Response to Ethanol.” 2018. Masters Thesis, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/174164.

MLA Handbook (7th Edition):

Al Muhisen, Hadil Mahdi J. “Sex Differences in Fetal Neural Stem Cells’ Response to Ethanol.” 2018. Web. 26 Oct 2020.

Vancouver:

Al Muhisen HMJ. Sex Differences in Fetal Neural Stem Cells’ Response to Ethanol. [Internet] [Masters thesis]. Texas A&M University; 2018. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/174164.

Council of Science Editors:

Al Muhisen HMJ. Sex Differences in Fetal Neural Stem Cells’ Response to Ethanol. [Masters Thesis]. Texas A&M University; 2018. Available from: http://hdl.handle.net/1969.1/174164


Texas A&M University

9. Mitchell, Nicole Jean. Human Exposure to Foodborne Toxins in Ghana: Intervention Strategy for Reduction of Aflatoxin and Fumonisin Bioavailability.

Degree: PhD, Toxicology, 2013, Texas A&M University

 International health has typically focused on remediation of infectious diseases in developing countries. However, recent reports from the International Agency for Research on Cancer (IARC)… (more)

Subjects/Keywords: Aflatoxin; Fumonisin; Bioavailability; NovaSil clay; Enterosorption

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APA (6th Edition):

Mitchell, N. J. (2013). Human Exposure to Foodborne Toxins in Ghana: Intervention Strategy for Reduction of Aflatoxin and Fumonisin Bioavailability. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/151665

Chicago Manual of Style (16th Edition):

Mitchell, Nicole Jean. “Human Exposure to Foodborne Toxins in Ghana: Intervention Strategy for Reduction of Aflatoxin and Fumonisin Bioavailability.” 2013. Doctoral Dissertation, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/151665.

MLA Handbook (7th Edition):

Mitchell, Nicole Jean. “Human Exposure to Foodborne Toxins in Ghana: Intervention Strategy for Reduction of Aflatoxin and Fumonisin Bioavailability.” 2013. Web. 26 Oct 2020.

Vancouver:

Mitchell NJ. Human Exposure to Foodborne Toxins in Ghana: Intervention Strategy for Reduction of Aflatoxin and Fumonisin Bioavailability. [Internet] [Doctoral dissertation]. Texas A&M University; 2013. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/151665.

Council of Science Editors:

Mitchell NJ. Human Exposure to Foodborne Toxins in Ghana: Intervention Strategy for Reduction of Aflatoxin and Fumonisin Bioavailability. [Doctoral Dissertation]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/151665


Texas A&M University

10. Nemec, Matthew James. Polyphenolics from Mango (Mangifera Indica L.) Suppress Breast Cancer Ductal Carcinoma In Situ Proliferation Both In Vitro And In Vivo: Potential Role of the IGFR-1-AKT-AMPK-mTOR-Signaling Axis.

Degree: PhD, Toxicology, 2016, Texas A&M University

 More than 25% of all newly diagnosed breast cancer cases are ductal carcinoma in situ (DCIS), the most commonly diagnosed form of non-invasive breast cancer.… (more)

Subjects/Keywords: AMPK; mTOR; polyphenols; DCIS; breast cancer; mangos; pyrogallol

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APA (6th Edition):

Nemec, M. J. (2016). Polyphenolics from Mango (Mangifera Indica L.) Suppress Breast Cancer Ductal Carcinoma In Situ Proliferation Both In Vitro And In Vivo: Potential Role of the IGFR-1-AKT-AMPK-mTOR-Signaling Axis. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/158043

Chicago Manual of Style (16th Edition):

Nemec, Matthew James. “Polyphenolics from Mango (Mangifera Indica L.) Suppress Breast Cancer Ductal Carcinoma In Situ Proliferation Both In Vitro And In Vivo: Potential Role of the IGFR-1-AKT-AMPK-mTOR-Signaling Axis.” 2016. Doctoral Dissertation, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/158043.

MLA Handbook (7th Edition):

Nemec, Matthew James. “Polyphenolics from Mango (Mangifera Indica L.) Suppress Breast Cancer Ductal Carcinoma In Situ Proliferation Both In Vitro And In Vivo: Potential Role of the IGFR-1-AKT-AMPK-mTOR-Signaling Axis.” 2016. Web. 26 Oct 2020.

Vancouver:

Nemec MJ. Polyphenolics from Mango (Mangifera Indica L.) Suppress Breast Cancer Ductal Carcinoma In Situ Proliferation Both In Vitro And In Vivo: Potential Role of the IGFR-1-AKT-AMPK-mTOR-Signaling Axis. [Internet] [Doctoral dissertation]. Texas A&M University; 2016. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/158043.

Council of Science Editors:

Nemec MJ. Polyphenolics from Mango (Mangifera Indica L.) Suppress Breast Cancer Ductal Carcinoma In Situ Proliferation Both In Vitro And In Vivo: Potential Role of the IGFR-1-AKT-AMPK-mTOR-Signaling Axis. [Doctoral Dissertation]. Texas A&M University; 2016. Available from: http://hdl.handle.net/1969.1/158043


Texas A&M University

11. Elmore, Sarah Elizabeth. Aflatoxin and Fumonisin Exposure: International Survey and Enterosorption Mitigation Strategy in Humans.

Degree: PhD, Toxicology, 2016, Texas A&M University

 Aflatoxins (AFs) are toxic metabolites produced by Aspergillus flavus and A. parasiticus. Fumonisins (FBs) are also toxic products of fungi, specifically Fusarium verticilloides and F.… (more)

Subjects/Keywords: Aflatoxin; Fumonisin; Calcium Montmorillonite Clay; ACCS100; UPSN; Enterosorption; Clay Mitigation Therapy; Biomarkers Of Exposure

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APA (6th Edition):

Elmore, S. E. (2016). Aflatoxin and Fumonisin Exposure: International Survey and Enterosorption Mitigation Strategy in Humans. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/157053

Chicago Manual of Style (16th Edition):

Elmore, Sarah Elizabeth. “Aflatoxin and Fumonisin Exposure: International Survey and Enterosorption Mitigation Strategy in Humans.” 2016. Doctoral Dissertation, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/157053.

MLA Handbook (7th Edition):

Elmore, Sarah Elizabeth. “Aflatoxin and Fumonisin Exposure: International Survey and Enterosorption Mitigation Strategy in Humans.” 2016. Web. 26 Oct 2020.

Vancouver:

Elmore SE. Aflatoxin and Fumonisin Exposure: International Survey and Enterosorption Mitigation Strategy in Humans. [Internet] [Doctoral dissertation]. Texas A&M University; 2016. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/157053.

Council of Science Editors:

Elmore SE. Aflatoxin and Fumonisin Exposure: International Survey and Enterosorption Mitigation Strategy in Humans. [Doctoral Dissertation]. Texas A&M University; 2016. Available from: http://hdl.handle.net/1969.1/157053


Texas A&M University

12. Xie, Ying. Transcriptional Regulation of Pregnane X Receptor by Protein Arginine Methyltransferase.

Degree: PhD, Toxicology, 2011, Texas A&M University

 Pregnane X receptor (PXR) is a ligand-dependent transcription factor that plays an important role in xenobiotic/drug metabolism. The ligand-receptor interaction transcriptionally activates phase I and… (more)

Subjects/Keywords: PXR; PRMT1; epigenetics

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APA (6th Edition):

Xie, Y. (2011). Transcriptional Regulation of Pregnane X Receptor by Protein Arginine Methyltransferase. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2010-05-7978

Chicago Manual of Style (16th Edition):

Xie, Ying. “Transcriptional Regulation of Pregnane X Receptor by Protein Arginine Methyltransferase.” 2011. Doctoral Dissertation, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/ETD-TAMU-2010-05-7978.

MLA Handbook (7th Edition):

Xie, Ying. “Transcriptional Regulation of Pregnane X Receptor by Protein Arginine Methyltransferase.” 2011. Web. 26 Oct 2020.

Vancouver:

Xie Y. Transcriptional Regulation of Pregnane X Receptor by Protein Arginine Methyltransferase. [Internet] [Doctoral dissertation]. Texas A&M University; 2011. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2010-05-7978.

Council of Science Editors:

Xie Y. Transcriptional Regulation of Pregnane X Receptor by Protein Arginine Methyltransferase. [Doctoral Dissertation]. Texas A&M University; 2011. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2010-05-7978


Texas A&M University

13. Li, Xi. Mechanism of Action of Sulindac and Diindolylmethane Analogs as Anticancer Agents.

Degree: PhD, Medical Sciences, 2014, Texas A&M University

 Cancer accounts for one in eight deaths worldwide and one in four deaths in the United States. Chemotherapy is the most common treatment option for… (more)

Subjects/Keywords: Cancer; Colon Cancer; Pancreatic Cancer; Sulindac; NSAID; Di-indolylmethane; C-DIM; Sp; Specificity Protein; Nuclear Receptor; NR4A; NR4A1; NR4A2; Nurr1; TR3; Nur77; Transactivation

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APA (6th Edition):

Li, X. (2014). Mechanism of Action of Sulindac and Diindolylmethane Analogs as Anticancer Agents. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/158901

Chicago Manual of Style (16th Edition):

Li, Xi. “Mechanism of Action of Sulindac and Diindolylmethane Analogs as Anticancer Agents.” 2014. Doctoral Dissertation, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/158901.

MLA Handbook (7th Edition):

Li, Xi. “Mechanism of Action of Sulindac and Diindolylmethane Analogs as Anticancer Agents.” 2014. Web. 26 Oct 2020.

Vancouver:

Li X. Mechanism of Action of Sulindac and Diindolylmethane Analogs as Anticancer Agents. [Internet] [Doctoral dissertation]. Texas A&M University; 2014. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/158901.

Council of Science Editors:

Li X. Mechanism of Action of Sulindac and Diindolylmethane Analogs as Anticancer Agents. [Doctoral Dissertation]. Texas A&M University; 2014. Available from: http://hdl.handle.net/1969.1/158901


Texas A&M University

14. Hedrick, Erik Duane. Diindolylmethane Analogs as Novel NR4A1 Antagonists and as a Novel Class of Anticancer Agents and Sp Transcription Factors as Nononcogene Addiction Genes That Are Targets of ROS Inducing Agents.

Degree: PhD, Toxicology, 2016, Texas A&M University

 The orphan nuclear receptor 4A1 (NR4A1) and specificity protein (Sp) transcription factors (TFs) are both overexpressed in the majority of solid tumors. Our laboratory has… (more)

Subjects/Keywords: NR4A1; Sp transcription factors

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APA (6th Edition):

Hedrick, E. D. (2016). Diindolylmethane Analogs as Novel NR4A1 Antagonists and as a Novel Class of Anticancer Agents and Sp Transcription Factors as Nononcogene Addiction Genes That Are Targets of ROS Inducing Agents. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/159116

Chicago Manual of Style (16th Edition):

Hedrick, Erik Duane. “Diindolylmethane Analogs as Novel NR4A1 Antagonists and as a Novel Class of Anticancer Agents and Sp Transcription Factors as Nononcogene Addiction Genes That Are Targets of ROS Inducing Agents.” 2016. Doctoral Dissertation, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/159116.

MLA Handbook (7th Edition):

Hedrick, Erik Duane. “Diindolylmethane Analogs as Novel NR4A1 Antagonists and as a Novel Class of Anticancer Agents and Sp Transcription Factors as Nononcogene Addiction Genes That Are Targets of ROS Inducing Agents.” 2016. Web. 26 Oct 2020.

Vancouver:

Hedrick ED. Diindolylmethane Analogs as Novel NR4A1 Antagonists and as a Novel Class of Anticancer Agents and Sp Transcription Factors as Nononcogene Addiction Genes That Are Targets of ROS Inducing Agents. [Internet] [Doctoral dissertation]. Texas A&M University; 2016. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/159116.

Council of Science Editors:

Hedrick ED. Diindolylmethane Analogs as Novel NR4A1 Antagonists and as a Novel Class of Anticancer Agents and Sp Transcription Factors as Nononcogene Addiction Genes That Are Targets of ROS Inducing Agents. [Doctoral Dissertation]. Texas A&M University; 2016. Available from: http://hdl.handle.net/1969.1/159116

15. Cheng, Yating. The Role of Long Noncoding RNAs in Cancer and Microbiota-derived Aryl Hydrocarbon Receptor Ligands.

Degree: PhD, Toxicology, 2016, Texas A&M University

 LncRNAs are a group of non-coding RNAs containing >200 nucleotides and these RNAs have no significant protein coding potential. In the past 10-15 years the… (more)

Subjects/Keywords: Long noncoding RNAs; Pancreatic cancer; Gut microbiota, AhR

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APA (6th Edition):

Cheng, Y. (2016). The Role of Long Noncoding RNAs in Cancer and Microbiota-derived Aryl Hydrocarbon Receptor Ligands. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/159030

Chicago Manual of Style (16th Edition):

Cheng, Yating. “The Role of Long Noncoding RNAs in Cancer and Microbiota-derived Aryl Hydrocarbon Receptor Ligands.” 2016. Doctoral Dissertation, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/159030.

MLA Handbook (7th Edition):

Cheng, Yating. “The Role of Long Noncoding RNAs in Cancer and Microbiota-derived Aryl Hydrocarbon Receptor Ligands.” 2016. Web. 26 Oct 2020.

Vancouver:

Cheng Y. The Role of Long Noncoding RNAs in Cancer and Microbiota-derived Aryl Hydrocarbon Receptor Ligands. [Internet] [Doctoral dissertation]. Texas A&M University; 2016. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/159030.

Council of Science Editors:

Cheng Y. The Role of Long Noncoding RNAs in Cancer and Microbiota-derived Aryl Hydrocarbon Receptor Ligands. [Doctoral Dissertation]. Texas A&M University; 2016. Available from: http://hdl.handle.net/1969.1/159030


Texas A&M University

16. Ramachandran Nair Vasanthakum, Vijayalekshmi. Mechanisms of Action of Metformin as an Anti-cancer Agent.

Degree: PhD, Toxicology, 2014, Texas A&M University

 Cancer is the second leading cause of death worldwide and epidemiological studies suggest the association of diabetes mellitus with an enhanced risk for multiple cancers.… (more)

Subjects/Keywords: Specificity protein transcription factors; IGF-1R; EGFR; mTOR

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APA (6th Edition):

Ramachandran Nair Vasanthakum, V. (2014). Mechanisms of Action of Metformin as an Anti-cancer Agent. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/153440

Chicago Manual of Style (16th Edition):

Ramachandran Nair Vasanthakum, Vijayalekshmi. “Mechanisms of Action of Metformin as an Anti-cancer Agent.” 2014. Doctoral Dissertation, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/153440.

MLA Handbook (7th Edition):

Ramachandran Nair Vasanthakum, Vijayalekshmi. “Mechanisms of Action of Metformin as an Anti-cancer Agent.” 2014. Web. 26 Oct 2020.

Vancouver:

Ramachandran Nair Vasanthakum V. Mechanisms of Action of Metformin as an Anti-cancer Agent. [Internet] [Doctoral dissertation]. Texas A&M University; 2014. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/153440.

Council of Science Editors:

Ramachandran Nair Vasanthakum V. Mechanisms of Action of Metformin as an Anti-cancer Agent. [Doctoral Dissertation]. Texas A&M University; 2014. Available from: http://hdl.handle.net/1969.1/153440


Texas A&M University

17. Hou, Tim Yu-Tien. n-3 Polyunsaturated Fatty Acids Alter Mouse CD4+ T Cell Activation by Modifying the Lipid Bilayer Properties.

Degree: PhD, Biochemistry, 2014, Texas A&M University

 Epidemiological and clinical studies have shown that very long chain n-3 polyunsaturated fatty acids (n-3 PUFA) such as eicosapentaenoic acid (EPA) and docosahexaenoic acid (DHA)… (more)

Subjects/Keywords: n-3 polyunsaturated fatty acids; CD4+ T cell; plasma membrane; lipid rafts; phosphatidylinositol-(4,5)-bisphosphate; actin remodeling; palmitoylation

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APA (6th Edition):

Hou, T. Y. (2014). n-3 Polyunsaturated Fatty Acids Alter Mouse CD4+ T Cell Activation by Modifying the Lipid Bilayer Properties. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/154130

Chicago Manual of Style (16th Edition):

Hou, Tim Yu-Tien. “n-3 Polyunsaturated Fatty Acids Alter Mouse CD4+ T Cell Activation by Modifying the Lipid Bilayer Properties.” 2014. Doctoral Dissertation, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/154130.

MLA Handbook (7th Edition):

Hou, Tim Yu-Tien. “n-3 Polyunsaturated Fatty Acids Alter Mouse CD4+ T Cell Activation by Modifying the Lipid Bilayer Properties.” 2014. Web. 26 Oct 2020.

Vancouver:

Hou TY. n-3 Polyunsaturated Fatty Acids Alter Mouse CD4+ T Cell Activation by Modifying the Lipid Bilayer Properties. [Internet] [Doctoral dissertation]. Texas A&M University; 2014. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/154130.

Council of Science Editors:

Hou TY. n-3 Polyunsaturated Fatty Acids Alter Mouse CD4+ T Cell Activation by Modifying the Lipid Bilayer Properties. [Doctoral Dissertation]. Texas A&M University; 2014. Available from: http://hdl.handle.net/1969.1/154130


Texas A&M University

18. Ying, Wei. Regulation of Obesity-Induced Adipose Tissue Inflammation and Insulin Resistance through Modulation of Immune Cell Function.

Degree: PhD, Animal Science, 2015, Texas A&M University

 The recruitment of immune cells into adipose tissue is a hallmark of obesity and a key event contributing to the development of adipose tissue inflammation… (more)

Subjects/Keywords: immune cell function; adipose tissue inflammation

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APA (6th Edition):

Ying, W. (2015). Regulation of Obesity-Induced Adipose Tissue Inflammation and Insulin Resistance through Modulation of Immune Cell Function. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/155008

Chicago Manual of Style (16th Edition):

Ying, Wei. “Regulation of Obesity-Induced Adipose Tissue Inflammation and Insulin Resistance through Modulation of Immune Cell Function.” 2015. Doctoral Dissertation, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/155008.

MLA Handbook (7th Edition):

Ying, Wei. “Regulation of Obesity-Induced Adipose Tissue Inflammation and Insulin Resistance through Modulation of Immune Cell Function.” 2015. Web. 26 Oct 2020.

Vancouver:

Ying W. Regulation of Obesity-Induced Adipose Tissue Inflammation and Insulin Resistance through Modulation of Immune Cell Function. [Internet] [Doctoral dissertation]. Texas A&M University; 2015. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/155008.

Council of Science Editors:

Ying W. Regulation of Obesity-Induced Adipose Tissue Inflammation and Insulin Resistance through Modulation of Immune Cell Function. [Doctoral Dissertation]. Texas A&M University; 2015. Available from: http://hdl.handle.net/1969.1/155008


Texas A&M University

19. Scribner, Kelly C. Regulation of Mammary cell Differentiation and Metabolism by Singleminded-2s.

Degree: PhD, Toxicology, 2013, Texas A&M University

 Ductal carcinoma in situ (DCIS) has been shown to be a precursor to invasive ductal cancer (IDC). Though the progression of DCIS to IDC is… (more)

Subjects/Keywords: Breast Cancer; Warburg Effect; Metabolism; Singleminded-2s; Mammary gland

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APA (6th Edition):

Scribner, K. C. (2013). Regulation of Mammary cell Differentiation and Metabolism by Singleminded-2s. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/151007

Chicago Manual of Style (16th Edition):

Scribner, Kelly C. “Regulation of Mammary cell Differentiation and Metabolism by Singleminded-2s.” 2013. Doctoral Dissertation, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/151007.

MLA Handbook (7th Edition):

Scribner, Kelly C. “Regulation of Mammary cell Differentiation and Metabolism by Singleminded-2s.” 2013. Web. 26 Oct 2020.

Vancouver:

Scribner KC. Regulation of Mammary cell Differentiation and Metabolism by Singleminded-2s. [Internet] [Doctoral dissertation]. Texas A&M University; 2013. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/151007.

Council of Science Editors:

Scribner KC. Regulation of Mammary cell Differentiation and Metabolism by Singleminded-2s. [Doctoral Dissertation]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/151007


Texas A&M University

20. Sreevalsan, Sandeep. Molecular Mechanisms of Cannabinoids as Anti-cancer Agents.

Degree: PhD, Toxicology, 2013, Texas A&M University

 Cancer is a growing health concern world-wide and is the second most common cause of death after heart diseases. Current treatment strategies such as surgery,… (more)

Subjects/Keywords: Cancer; Cannabinoids

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APA (6th Edition):

Sreevalsan, S. (2013). Molecular Mechanisms of Cannabinoids as Anti-cancer Agents. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/151011

Chicago Manual of Style (16th Edition):

Sreevalsan, Sandeep. “Molecular Mechanisms of Cannabinoids as Anti-cancer Agents.” 2013. Doctoral Dissertation, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/151011.

MLA Handbook (7th Edition):

Sreevalsan, Sandeep. “Molecular Mechanisms of Cannabinoids as Anti-cancer Agents.” 2013. Web. 26 Oct 2020.

Vancouver:

Sreevalsan S. Molecular Mechanisms of Cannabinoids as Anti-cancer Agents. [Internet] [Doctoral dissertation]. Texas A&M University; 2013. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/151011.

Council of Science Editors:

Sreevalsan S. Molecular Mechanisms of Cannabinoids as Anti-cancer Agents. [Doctoral Dissertation]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/151011


Texas A&M University

21. Chang, Cheng-An Richard. The Epigenetic Effects of Preconception Paternal Alcohol Exposure on Adult Health and Disease.

Degree: PhD, Biomedical Sciences, 2019, Texas A&M University

 Alcohol is a notorious teratogen and a major driver of both mental and physical defects. Recently, alcohol has been discovered to exert intergenerational effects on… (more)

Subjects/Keywords: Epigenetics

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APA (6th Edition):

Chang, C. R. (2019). The Epigenetic Effects of Preconception Paternal Alcohol Exposure on Adult Health and Disease. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/188715

Chicago Manual of Style (16th Edition):

Chang, Cheng-An Richard. “The Epigenetic Effects of Preconception Paternal Alcohol Exposure on Adult Health and Disease.” 2019. Doctoral Dissertation, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/188715.

MLA Handbook (7th Edition):

Chang, Cheng-An Richard. “The Epigenetic Effects of Preconception Paternal Alcohol Exposure on Adult Health and Disease.” 2019. Web. 26 Oct 2020.

Vancouver:

Chang CR. The Epigenetic Effects of Preconception Paternal Alcohol Exposure on Adult Health and Disease. [Internet] [Doctoral dissertation]. Texas A&M University; 2019. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/188715.

Council of Science Editors:

Chang CR. The Epigenetic Effects of Preconception Paternal Alcohol Exposure on Adult Health and Disease. [Doctoral Dissertation]. Texas A&M University; 2019. Available from: http://hdl.handle.net/1969.1/188715


Texas A&M University

22. Hieke, Anne-Sophie Charlotte. Investigating the Inactivation, Physiological Characteristics and Transcriptomic Responses of Bacteria Exposed to Ionizing Radiation.

Degree: PhD, Toxicology, 2015, Texas A&M University

 Ionizing radiation is used for many different applications that require a reduction in microbial bioburden. Yet, the scientific literature remains unsettled when it comes to… (more)

Subjects/Keywords: ionizing radiation; bacteria; inactivation kinetics; transcriptomics

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APA (6th Edition):

Hieke, A. C. (2015). Investigating the Inactivation, Physiological Characteristics and Transcriptomic Responses of Bacteria Exposed to Ionizing Radiation. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/187477

Chicago Manual of Style (16th Edition):

Hieke, Anne-Sophie Charlotte. “Investigating the Inactivation, Physiological Characteristics and Transcriptomic Responses of Bacteria Exposed to Ionizing Radiation.” 2015. Doctoral Dissertation, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/187477.

MLA Handbook (7th Edition):

Hieke, Anne-Sophie Charlotte. “Investigating the Inactivation, Physiological Characteristics and Transcriptomic Responses of Bacteria Exposed to Ionizing Radiation.” 2015. Web. 26 Oct 2020.

Vancouver:

Hieke AC. Investigating the Inactivation, Physiological Characteristics and Transcriptomic Responses of Bacteria Exposed to Ionizing Radiation. [Internet] [Doctoral dissertation]. Texas A&M University; 2015. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/187477.

Council of Science Editors:

Hieke AC. Investigating the Inactivation, Physiological Characteristics and Transcriptomic Responses of Bacteria Exposed to Ionizing Radiation. [Doctoral Dissertation]. Texas A&M University; 2015. Available from: http://hdl.handle.net/1969.1/187477

23. Banerjee, Nivedita. Systematic Approach to Compare the Inflammatory Response of Liver Cell Culture Systems Exposed to Silver, Copper, and Nickel Nanoparticles.

Degree: MS, Toxicology, 2011, Texas A&M University

 Although nano-sized metal colloids are used in industrial and medicinal applications, little is known about the potential liver toxicity of these materials after occupational or… (more)

Subjects/Keywords: Nanoparticles; Inflammation; Cytokines; Cellular Uptake; Cytotoxicity; Co-culture

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APA (6th Edition):

Banerjee, N. (2011). Systematic Approach to Compare the Inflammatory Response of Liver Cell Culture Systems Exposed to Silver, Copper, and Nickel Nanoparticles. (Masters Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2010-08-8414

Chicago Manual of Style (16th Edition):

Banerjee, Nivedita. “Systematic Approach to Compare the Inflammatory Response of Liver Cell Culture Systems Exposed to Silver, Copper, and Nickel Nanoparticles.” 2011. Masters Thesis, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/ETD-TAMU-2010-08-8414.

MLA Handbook (7th Edition):

Banerjee, Nivedita. “Systematic Approach to Compare the Inflammatory Response of Liver Cell Culture Systems Exposed to Silver, Copper, and Nickel Nanoparticles.” 2011. Web. 26 Oct 2020.

Vancouver:

Banerjee N. Systematic Approach to Compare the Inflammatory Response of Liver Cell Culture Systems Exposed to Silver, Copper, and Nickel Nanoparticles. [Internet] [Masters thesis]. Texas A&M University; 2011. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2010-08-8414.

Council of Science Editors:

Banerjee N. Systematic Approach to Compare the Inflammatory Response of Liver Cell Culture Systems Exposed to Silver, Copper, and Nickel Nanoparticles. [Masters Thesis]. Texas A&M University; 2011. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2010-08-8414

24. Guthrie, Aaron S 1987-. Phenethyl Isothiocyanate (PEITC) Decreases Specficity Protein (SP) Tanscription Factors through an ROS-dependent Mechanism.

Degree: MS, Biochemistry, 2012, Texas A&M University

 Isothiocyanates (ITCs) are phytochemicals highly expressed in cruciferous vegetables and these compounds are associated with the decreased incidence of cancers in populations consuming high levels… (more)

Subjects/Keywords: Sp transcription factors; Sp dependent genes; ROS; PEITC

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APA (6th Edition):

Guthrie, A. S. 1. (2012). Phenethyl Isothiocyanate (PEITC) Decreases Specficity Protein (SP) Tanscription Factors through an ROS-dependent Mechanism. (Masters Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/148086

Chicago Manual of Style (16th Edition):

Guthrie, Aaron S 1987-. “Phenethyl Isothiocyanate (PEITC) Decreases Specficity Protein (SP) Tanscription Factors through an ROS-dependent Mechanism.” 2012. Masters Thesis, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/148086.

MLA Handbook (7th Edition):

Guthrie, Aaron S 1987-. “Phenethyl Isothiocyanate (PEITC) Decreases Specficity Protein (SP) Tanscription Factors through an ROS-dependent Mechanism.” 2012. Web. 26 Oct 2020.

Vancouver:

Guthrie AS1. Phenethyl Isothiocyanate (PEITC) Decreases Specficity Protein (SP) Tanscription Factors through an ROS-dependent Mechanism. [Internet] [Masters thesis]. Texas A&M University; 2012. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/148086.

Council of Science Editors:

Guthrie AS1. Phenethyl Isothiocyanate (PEITC) Decreases Specficity Protein (SP) Tanscription Factors through an ROS-dependent Mechanism. [Masters Thesis]. Texas A&M University; 2012. Available from: http://hdl.handle.net/1969.1/148086

25. Meng, Cong. microRNA-223 Regulates Macrophage Polarization and Diet-induced Insulin Resistance.

Degree: MS, Biomedical Sciences, 2013, Texas A&M University

 Macrophage activation plays a crucial role in regulating adipose tissue inflammation and is a major contributor to the pathogenesis of obesity-associated cardiovascular diseases. On various… (more)

Subjects/Keywords: microRNAs; macrophages; adipose tissue inflammation

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APA (6th Edition):

Meng, C. (2013). microRNA-223 Regulates Macrophage Polarization and Diet-induced Insulin Resistance. (Masters Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/149464

Chicago Manual of Style (16th Edition):

Meng, Cong. “microRNA-223 Regulates Macrophage Polarization and Diet-induced Insulin Resistance.” 2013. Masters Thesis, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/149464.

MLA Handbook (7th Edition):

Meng, Cong. “microRNA-223 Regulates Macrophage Polarization and Diet-induced Insulin Resistance.” 2013. Web. 26 Oct 2020.

Vancouver:

Meng C. microRNA-223 Regulates Macrophage Polarization and Diet-induced Insulin Resistance. [Internet] [Masters thesis]. Texas A&M University; 2013. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/149464.

Council of Science Editors:

Meng C. microRNA-223 Regulates Macrophage Polarization and Diet-induced Insulin Resistance. [Masters Thesis]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/149464

26. Ferguson, Brent Wade. Regulation of PERLECAN and 2OST Expression in Prostate Cancer Progression by Stress-activated Transcription Factors.

Degree: PhD, Biochemistry, 2012, Texas A&M University

 Heparan sulfate proteoglycans modulate many of the growth factor pathways that drive prostate cancer progression. Prior to being secreted into the extracellular matrix, the covalently… (more)

Subjects/Keywords: Perlecan; 2OST; hypoxia; ROS

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APA (6th Edition):

Ferguson, B. W. (2012). Regulation of PERLECAN and 2OST Expression in Prostate Cancer Progression by Stress-activated Transcription Factors. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2010-12-8871

Chicago Manual of Style (16th Edition):

Ferguson, Brent Wade. “Regulation of PERLECAN and 2OST Expression in Prostate Cancer Progression by Stress-activated Transcription Factors.” 2012. Doctoral Dissertation, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/ETD-TAMU-2010-12-8871.

MLA Handbook (7th Edition):

Ferguson, Brent Wade. “Regulation of PERLECAN and 2OST Expression in Prostate Cancer Progression by Stress-activated Transcription Factors.” 2012. Web. 26 Oct 2020.

Vancouver:

Ferguson BW. Regulation of PERLECAN and 2OST Expression in Prostate Cancer Progression by Stress-activated Transcription Factors. [Internet] [Doctoral dissertation]. Texas A&M University; 2012. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2010-12-8871.

Council of Science Editors:

Ferguson BW. Regulation of PERLECAN and 2OST Expression in Prostate Cancer Progression by Stress-activated Transcription Factors. [Doctoral Dissertation]. Texas A&M University; 2012. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2010-12-8871

27. Lacey, Alexandra Dianne. NR4A1 Antagonists Target the Oncogenic PAX3-FOXO1A Gene and Induce Interleukin -24 in Rhabdomyosarcoma (RMS).

Degree: PhD, Toxicology, 2017, Texas A&M University

 Rhabdomyosarcoma (RMS) is a pediatric cancer for which common therapeutics include cytotoxic chemotherapeutics that result in adverse health outcomes later in adulthood. Previous studies have… (more)

Subjects/Keywords: Rhabdomyosarcoma; Pediatric cancer; orphan nuclear receptor; NR4A1; PAX3-FOXO1A; Interleukin-24

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APA (6th Edition):

Lacey, A. D. (2017). NR4A1 Antagonists Target the Oncogenic PAX3-FOXO1A Gene and Induce Interleukin -24 in Rhabdomyosarcoma (RMS). (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/166039

Chicago Manual of Style (16th Edition):

Lacey, Alexandra Dianne. “NR4A1 Antagonists Target the Oncogenic PAX3-FOXO1A Gene and Induce Interleukin -24 in Rhabdomyosarcoma (RMS).” 2017. Doctoral Dissertation, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/166039.

MLA Handbook (7th Edition):

Lacey, Alexandra Dianne. “NR4A1 Antagonists Target the Oncogenic PAX3-FOXO1A Gene and Induce Interleukin -24 in Rhabdomyosarcoma (RMS).” 2017. Web. 26 Oct 2020.

Vancouver:

Lacey AD. NR4A1 Antagonists Target the Oncogenic PAX3-FOXO1A Gene and Induce Interleukin -24 in Rhabdomyosarcoma (RMS). [Internet] [Doctoral dissertation]. Texas A&M University; 2017. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/166039.

Council of Science Editors:

Lacey AD. NR4A1 Antagonists Target the Oncogenic PAX3-FOXO1A Gene and Induce Interleukin -24 in Rhabdomyosarcoma (RMS). [Doctoral Dissertation]. Texas A&M University; 2017. Available from: http://hdl.handle.net/1969.1/166039

28. Liu, Xinyi. Mechanistic Investigation of Tolfenamic Acid, Betulinic Acid, and Aspirin in Anti-Cancer Therapy.

Degree: PhD, Biochemistry, 2012, Texas A&M University

 Tolfenamic acid (TA), betulinic acid (BA) and acetylsalicylic acid (aspirin) are anticancer drugs, and Sp1, Sp3, Sp4 transcription factors and growth factor 2 (EGFR2, HER2… (more)

Subjects/Keywords: Sp protein; ErbB2; tolfenamic acid; betulinic acid; aspirin

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APA (6th Edition):

Liu, X. (2012). Mechanistic Investigation of Tolfenamic Acid, Betulinic Acid, and Aspirin in Anti-Cancer Therapy. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2011-08-9741

Chicago Manual of Style (16th Edition):

Liu, Xinyi. “Mechanistic Investigation of Tolfenamic Acid, Betulinic Acid, and Aspirin in Anti-Cancer Therapy.” 2012. Doctoral Dissertation, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/ETD-TAMU-2011-08-9741.

MLA Handbook (7th Edition):

Liu, Xinyi. “Mechanistic Investigation of Tolfenamic Acid, Betulinic Acid, and Aspirin in Anti-Cancer Therapy.” 2012. Web. 26 Oct 2020.

Vancouver:

Liu X. Mechanistic Investigation of Tolfenamic Acid, Betulinic Acid, and Aspirin in Anti-Cancer Therapy. [Internet] [Doctoral dissertation]. Texas A&M University; 2012. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2011-08-9741.

Council of Science Editors:

Liu X. Mechanistic Investigation of Tolfenamic Acid, Betulinic Acid, and Aspirin in Anti-Cancer Therapy. [Doctoral Dissertation]. Texas A&M University; 2012. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2011-08-9741

29. Amini-Vaughan, Zhaleh Jacqueline. Harnessing Eosinophils as a Potential Tumoricidal Immunotherapy.

Degree: MS, Medical Sciences, 2016, Texas A&M University

 Eosinophils have numerous functions for defense against pathogens, despite being only 1-5% of total circulating blood leukocytes. Most well known for their actions contributing to… (more)

Subjects/Keywords: eosinophil; cancer; immunotherapy

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APA (6th Edition):

Amini-Vaughan, Z. J. (2016). Harnessing Eosinophils as a Potential Tumoricidal Immunotherapy. (Masters Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/157005

Chicago Manual of Style (16th Edition):

Amini-Vaughan, Zhaleh Jacqueline. “Harnessing Eosinophils as a Potential Tumoricidal Immunotherapy.” 2016. Masters Thesis, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/157005.

MLA Handbook (7th Edition):

Amini-Vaughan, Zhaleh Jacqueline. “Harnessing Eosinophils as a Potential Tumoricidal Immunotherapy.” 2016. Web. 26 Oct 2020.

Vancouver:

Amini-Vaughan ZJ. Harnessing Eosinophils as a Potential Tumoricidal Immunotherapy. [Internet] [Masters thesis]. Texas A&M University; 2016. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/157005.

Council of Science Editors:

Amini-Vaughan ZJ. Harnessing Eosinophils as a Potential Tumoricidal Immunotherapy. [Masters Thesis]. Texas A&M University; 2016. Available from: http://hdl.handle.net/1969.1/157005

30. Tang, Su. Mycobacterium Tuberculosis Methylcitrate Cycle Regulation and Study of Non-Covalent Natural Product Inhibitors of Proteasome.

Degree: PhD, Biochemistry, 2018, Texas A&M University

 The methylcitrate cycle and glyoxylate bypass are two critical pathways of the central carbon metabolism of the human pathogen Mycobacterium tuberculosis (Mtb). Transcription of the… (more)

Subjects/Keywords: Mycobacterium tuberculosis; methylcitrate cycle; PrpR; RamB; proteasome inhibitor; natural products; Auxarthron umbrinum

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tang, S. (2018). Mycobacterium Tuberculosis Methylcitrate Cycle Regulation and Study of Non-Covalent Natural Product Inhibitors of Proteasome. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/174301

Chicago Manual of Style (16th Edition):

Tang, Su. “Mycobacterium Tuberculosis Methylcitrate Cycle Regulation and Study of Non-Covalent Natural Product Inhibitors of Proteasome.” 2018. Doctoral Dissertation, Texas A&M University. Accessed October 26, 2020. http://hdl.handle.net/1969.1/174301.

MLA Handbook (7th Edition):

Tang, Su. “Mycobacterium Tuberculosis Methylcitrate Cycle Regulation and Study of Non-Covalent Natural Product Inhibitors of Proteasome.” 2018. Web. 26 Oct 2020.

Vancouver:

Tang S. Mycobacterium Tuberculosis Methylcitrate Cycle Regulation and Study of Non-Covalent Natural Product Inhibitors of Proteasome. [Internet] [Doctoral dissertation]. Texas A&M University; 2018. [cited 2020 Oct 26]. Available from: http://hdl.handle.net/1969.1/174301.

Council of Science Editors:

Tang S. Mycobacterium Tuberculosis Methylcitrate Cycle Regulation and Study of Non-Covalent Natural Product Inhibitors of Proteasome. [Doctoral Dissertation]. Texas A&M University; 2018. Available from: http://hdl.handle.net/1969.1/174301

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