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You searched for +publisher:"Texas A&M University" +contributor:("Safe, Stephen H"). Showing records 1 – 30 of 41 total matches.

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Texas A&M University

1. Gumuser, Esra D. Receptor Ubiquitination Regulates Il-5rα Function.

Degree: MS, Medical Sciences, 2016, Texas A&M University

 Eosinophils are multifunctional leukocytes implicated in the pathogenesis of inflammatory processes including hypereosinophilic syndrome, eosinophilic esophagitis, and allergic asthma. Due to its role in the… (more)

Subjects/Keywords: Eosinophils; inflammation; cytokine receptors; IL-5; IL-5Rα; βc; ubiquitination; JAK kinases

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APA (6th Edition):

Gumuser, E. D. (2016). Receptor Ubiquitination Regulates Il-5rα Function. (Masters Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/156973

Chicago Manual of Style (16th Edition):

Gumuser, Esra D. “Receptor Ubiquitination Regulates Il-5rα Function.” 2016. Masters Thesis, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/156973.

MLA Handbook (7th Edition):

Gumuser, Esra D. “Receptor Ubiquitination Regulates Il-5rα Function.” 2016. Web. 28 Nov 2020.

Vancouver:

Gumuser ED. Receptor Ubiquitination Regulates Il-5rα Function. [Internet] [Masters thesis]. Texas A&M University; 2016. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/156973.

Council of Science Editors:

Gumuser ED. Receptor Ubiquitination Regulates Il-5rα Function. [Masters Thesis]. Texas A&M University; 2016. Available from: http://hdl.handle.net/1969.1/156973


Texas A&M University

2. Mitchell, Nicole Jean. Human Exposure to Foodborne Toxins in Ghana: Intervention Strategy for Reduction of Aflatoxin and Fumonisin Bioavailability.

Degree: PhD, Toxicology, 2013, Texas A&M University

 International health has typically focused on remediation of infectious diseases in developing countries. However, recent reports from the International Agency for Research on Cancer (IARC)… (more)

Subjects/Keywords: Aflatoxin; Fumonisin; Bioavailability; NovaSil clay; Enterosorption

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APA (6th Edition):

Mitchell, N. J. (2013). Human Exposure to Foodborne Toxins in Ghana: Intervention Strategy for Reduction of Aflatoxin and Fumonisin Bioavailability. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/151665

Chicago Manual of Style (16th Edition):

Mitchell, Nicole Jean. “Human Exposure to Foodborne Toxins in Ghana: Intervention Strategy for Reduction of Aflatoxin and Fumonisin Bioavailability.” 2013. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/151665.

MLA Handbook (7th Edition):

Mitchell, Nicole Jean. “Human Exposure to Foodborne Toxins in Ghana: Intervention Strategy for Reduction of Aflatoxin and Fumonisin Bioavailability.” 2013. Web. 28 Nov 2020.

Vancouver:

Mitchell NJ. Human Exposure to Foodborne Toxins in Ghana: Intervention Strategy for Reduction of Aflatoxin and Fumonisin Bioavailability. [Internet] [Doctoral dissertation]. Texas A&M University; 2013. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/151665.

Council of Science Editors:

Mitchell NJ. Human Exposure to Foodborne Toxins in Ghana: Intervention Strategy for Reduction of Aflatoxin and Fumonisin Bioavailability. [Doctoral Dissertation]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/151665


Texas A&M University

3. Elmore, Sarah Elizabeth. Aflatoxin and Fumonisin Exposure: International Survey and Enterosorption Mitigation Strategy in Humans.

Degree: PhD, Toxicology, 2016, Texas A&M University

 Aflatoxins (AFs) are toxic metabolites produced by Aspergillus flavus and A. parasiticus. Fumonisins (FBs) are also toxic products of fungi, specifically Fusarium verticilloides and F.… (more)

Subjects/Keywords: Aflatoxin; Fumonisin; Calcium Montmorillonite Clay; ACCS100; UPSN; Enterosorption; Clay Mitigation Therapy; Biomarkers Of Exposure

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APA (6th Edition):

Elmore, S. E. (2016). Aflatoxin and Fumonisin Exposure: International Survey and Enterosorption Mitigation Strategy in Humans. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/157053

Chicago Manual of Style (16th Edition):

Elmore, Sarah Elizabeth. “Aflatoxin and Fumonisin Exposure: International Survey and Enterosorption Mitigation Strategy in Humans.” 2016. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/157053.

MLA Handbook (7th Edition):

Elmore, Sarah Elizabeth. “Aflatoxin and Fumonisin Exposure: International Survey and Enterosorption Mitigation Strategy in Humans.” 2016. Web. 28 Nov 2020.

Vancouver:

Elmore SE. Aflatoxin and Fumonisin Exposure: International Survey and Enterosorption Mitigation Strategy in Humans. [Internet] [Doctoral dissertation]. Texas A&M University; 2016. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/157053.

Council of Science Editors:

Elmore SE. Aflatoxin and Fumonisin Exposure: International Survey and Enterosorption Mitigation Strategy in Humans. [Doctoral Dissertation]. Texas A&M University; 2016. Available from: http://hdl.handle.net/1969.1/157053


Texas A&M University

4. Li, Xi. Mechanism of Action of Sulindac and Diindolylmethane Analogs as Anticancer Agents.

Degree: PhD, Medical Sciences, 2014, Texas A&M University

 Cancer accounts for one in eight deaths worldwide and one in four deaths in the United States. Chemotherapy is the most common treatment option for… (more)

Subjects/Keywords: Cancer; Colon Cancer; Pancreatic Cancer; Sulindac; NSAID; Di-indolylmethane; C-DIM; Sp; Specificity Protein; Nuclear Receptor; NR4A; NR4A1; NR4A2; Nurr1; TR3; Nur77; Transactivation

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APA (6th Edition):

Li, X. (2014). Mechanism of Action of Sulindac and Diindolylmethane Analogs as Anticancer Agents. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/158901

Chicago Manual of Style (16th Edition):

Li, Xi. “Mechanism of Action of Sulindac and Diindolylmethane Analogs as Anticancer Agents.” 2014. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/158901.

MLA Handbook (7th Edition):

Li, Xi. “Mechanism of Action of Sulindac and Diindolylmethane Analogs as Anticancer Agents.” 2014. Web. 28 Nov 2020.

Vancouver:

Li X. Mechanism of Action of Sulindac and Diindolylmethane Analogs as Anticancer Agents. [Internet] [Doctoral dissertation]. Texas A&M University; 2014. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/158901.

Council of Science Editors:

Li X. Mechanism of Action of Sulindac and Diindolylmethane Analogs as Anticancer Agents. [Doctoral Dissertation]. Texas A&M University; 2014. Available from: http://hdl.handle.net/1969.1/158901


Texas A&M University

5. Hedrick, Erik Duane. Diindolylmethane Analogs as Novel NR4A1 Antagonists and as a Novel Class of Anticancer Agents and Sp Transcription Factors as Nononcogene Addiction Genes That Are Targets of ROS Inducing Agents.

Degree: PhD, Toxicology, 2016, Texas A&M University

 The orphan nuclear receptor 4A1 (NR4A1) and specificity protein (Sp) transcription factors (TFs) are both overexpressed in the majority of solid tumors. Our laboratory has… (more)

Subjects/Keywords: NR4A1; Sp transcription factors

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APA (6th Edition):

Hedrick, E. D. (2016). Diindolylmethane Analogs as Novel NR4A1 Antagonists and as a Novel Class of Anticancer Agents and Sp Transcription Factors as Nononcogene Addiction Genes That Are Targets of ROS Inducing Agents. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/159116

Chicago Manual of Style (16th Edition):

Hedrick, Erik Duane. “Diindolylmethane Analogs as Novel NR4A1 Antagonists and as a Novel Class of Anticancer Agents and Sp Transcription Factors as Nononcogene Addiction Genes That Are Targets of ROS Inducing Agents.” 2016. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/159116.

MLA Handbook (7th Edition):

Hedrick, Erik Duane. “Diindolylmethane Analogs as Novel NR4A1 Antagonists and as a Novel Class of Anticancer Agents and Sp Transcription Factors as Nononcogene Addiction Genes That Are Targets of ROS Inducing Agents.” 2016. Web. 28 Nov 2020.

Vancouver:

Hedrick ED. Diindolylmethane Analogs as Novel NR4A1 Antagonists and as a Novel Class of Anticancer Agents and Sp Transcription Factors as Nononcogene Addiction Genes That Are Targets of ROS Inducing Agents. [Internet] [Doctoral dissertation]. Texas A&M University; 2016. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/159116.

Council of Science Editors:

Hedrick ED. Diindolylmethane Analogs as Novel NR4A1 Antagonists and as a Novel Class of Anticancer Agents and Sp Transcription Factors as Nononcogene Addiction Genes That Are Targets of ROS Inducing Agents. [Doctoral Dissertation]. Texas A&M University; 2016. Available from: http://hdl.handle.net/1969.1/159116

6. Cheng, Yating. The Role of Long Noncoding RNAs in Cancer and Microbiota-derived Aryl Hydrocarbon Receptor Ligands.

Degree: PhD, Toxicology, 2016, Texas A&M University

 LncRNAs are a group of non-coding RNAs containing >200 nucleotides and these RNAs have no significant protein coding potential. In the past 10-15 years the… (more)

Subjects/Keywords: Long noncoding RNAs; Pancreatic cancer; Gut microbiota, AhR

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APA (6th Edition):

Cheng, Y. (2016). The Role of Long Noncoding RNAs in Cancer and Microbiota-derived Aryl Hydrocarbon Receptor Ligands. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/159030

Chicago Manual of Style (16th Edition):

Cheng, Yating. “The Role of Long Noncoding RNAs in Cancer and Microbiota-derived Aryl Hydrocarbon Receptor Ligands.” 2016. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/159030.

MLA Handbook (7th Edition):

Cheng, Yating. “The Role of Long Noncoding RNAs in Cancer and Microbiota-derived Aryl Hydrocarbon Receptor Ligands.” 2016. Web. 28 Nov 2020.

Vancouver:

Cheng Y. The Role of Long Noncoding RNAs in Cancer and Microbiota-derived Aryl Hydrocarbon Receptor Ligands. [Internet] [Doctoral dissertation]. Texas A&M University; 2016. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/159030.

Council of Science Editors:

Cheng Y. The Role of Long Noncoding RNAs in Cancer and Microbiota-derived Aryl Hydrocarbon Receptor Ligands. [Doctoral Dissertation]. Texas A&M University; 2016. Available from: http://hdl.handle.net/1969.1/159030


Texas A&M University

7. Ramachandran Nair Vasanthakum, Vijayalekshmi. Mechanisms of Action of Metformin as an Anti-cancer Agent.

Degree: PhD, Toxicology, 2014, Texas A&M University

 Cancer is the second leading cause of death worldwide and epidemiological studies suggest the association of diabetes mellitus with an enhanced risk for multiple cancers.… (more)

Subjects/Keywords: Specificity protein transcription factors; IGF-1R; EGFR; mTOR

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APA (6th Edition):

Ramachandran Nair Vasanthakum, V. (2014). Mechanisms of Action of Metformin as an Anti-cancer Agent. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/153440

Chicago Manual of Style (16th Edition):

Ramachandran Nair Vasanthakum, Vijayalekshmi. “Mechanisms of Action of Metformin as an Anti-cancer Agent.” 2014. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/153440.

MLA Handbook (7th Edition):

Ramachandran Nair Vasanthakum, Vijayalekshmi. “Mechanisms of Action of Metformin as an Anti-cancer Agent.” 2014. Web. 28 Nov 2020.

Vancouver:

Ramachandran Nair Vasanthakum V. Mechanisms of Action of Metformin as an Anti-cancer Agent. [Internet] [Doctoral dissertation]. Texas A&M University; 2014. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/153440.

Council of Science Editors:

Ramachandran Nair Vasanthakum V. Mechanisms of Action of Metformin as an Anti-cancer Agent. [Doctoral Dissertation]. Texas A&M University; 2014. Available from: http://hdl.handle.net/1969.1/153440


Texas A&M University

8. Hou, Tim Yu-Tien. n-3 Polyunsaturated Fatty Acids Alter Mouse CD4+ T Cell Activation by Modifying the Lipid Bilayer Properties.

Degree: PhD, Biochemistry, 2014, Texas A&M University

 Epidemiological and clinical studies have shown that very long chain n-3 polyunsaturated fatty acids (n-3 PUFA) such as eicosapentaenoic acid (EPA) and docosahexaenoic acid (DHA)… (more)

Subjects/Keywords: n-3 polyunsaturated fatty acids; CD4+ T cell; plasma membrane; lipid rafts; phosphatidylinositol-(4,5)-bisphosphate; actin remodeling; palmitoylation

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APA (6th Edition):

Hou, T. Y. (2014). n-3 Polyunsaturated Fatty Acids Alter Mouse CD4+ T Cell Activation by Modifying the Lipid Bilayer Properties. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/154130

Chicago Manual of Style (16th Edition):

Hou, Tim Yu-Tien. “n-3 Polyunsaturated Fatty Acids Alter Mouse CD4+ T Cell Activation by Modifying the Lipid Bilayer Properties.” 2014. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/154130.

MLA Handbook (7th Edition):

Hou, Tim Yu-Tien. “n-3 Polyunsaturated Fatty Acids Alter Mouse CD4+ T Cell Activation by Modifying the Lipid Bilayer Properties.” 2014. Web. 28 Nov 2020.

Vancouver:

Hou TY. n-3 Polyunsaturated Fatty Acids Alter Mouse CD4+ T Cell Activation by Modifying the Lipid Bilayer Properties. [Internet] [Doctoral dissertation]. Texas A&M University; 2014. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/154130.

Council of Science Editors:

Hou TY. n-3 Polyunsaturated Fatty Acids Alter Mouse CD4+ T Cell Activation by Modifying the Lipid Bilayer Properties. [Doctoral Dissertation]. Texas A&M University; 2014. Available from: http://hdl.handle.net/1969.1/154130


Texas A&M University

9. Sreevalsan, Sandeep. Molecular Mechanisms of Cannabinoids as Anti-cancer Agents.

Degree: PhD, Toxicology, 2013, Texas A&M University

 Cancer is a growing health concern world-wide and is the second most common cause of death after heart diseases. Current treatment strategies such as surgery,… (more)

Subjects/Keywords: Cancer; Cannabinoids

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APA (6th Edition):

Sreevalsan, S. (2013). Molecular Mechanisms of Cannabinoids as Anti-cancer Agents. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/151011

Chicago Manual of Style (16th Edition):

Sreevalsan, Sandeep. “Molecular Mechanisms of Cannabinoids as Anti-cancer Agents.” 2013. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/151011.

MLA Handbook (7th Edition):

Sreevalsan, Sandeep. “Molecular Mechanisms of Cannabinoids as Anti-cancer Agents.” 2013. Web. 28 Nov 2020.

Vancouver:

Sreevalsan S. Molecular Mechanisms of Cannabinoids as Anti-cancer Agents. [Internet] [Doctoral dissertation]. Texas A&M University; 2013. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/151011.

Council of Science Editors:

Sreevalsan S. Molecular Mechanisms of Cannabinoids as Anti-cancer Agents. [Doctoral Dissertation]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/151011


Texas A&M University

10. Chang, Cheng-An Richard. The Epigenetic Effects of Preconception Paternal Alcohol Exposure on Adult Health and Disease.

Degree: PhD, Biomedical Sciences, 2019, Texas A&M University

 Alcohol is a notorious teratogen and a major driver of both mental and physical defects. Recently, alcohol has been discovered to exert intergenerational effects on… (more)

Subjects/Keywords: Epigenetics

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APA (6th Edition):

Chang, C. R. (2019). The Epigenetic Effects of Preconception Paternal Alcohol Exposure on Adult Health and Disease. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/188715

Chicago Manual of Style (16th Edition):

Chang, Cheng-An Richard. “The Epigenetic Effects of Preconception Paternal Alcohol Exposure on Adult Health and Disease.” 2019. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/188715.

MLA Handbook (7th Edition):

Chang, Cheng-An Richard. “The Epigenetic Effects of Preconception Paternal Alcohol Exposure on Adult Health and Disease.” 2019. Web. 28 Nov 2020.

Vancouver:

Chang CR. The Epigenetic Effects of Preconception Paternal Alcohol Exposure on Adult Health and Disease. [Internet] [Doctoral dissertation]. Texas A&M University; 2019. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/188715.

Council of Science Editors:

Chang CR. The Epigenetic Effects of Preconception Paternal Alcohol Exposure on Adult Health and Disease. [Doctoral Dissertation]. Texas A&M University; 2019. Available from: http://hdl.handle.net/1969.1/188715

11. Meng, Cong. microRNA-223 Regulates Macrophage Polarization and Diet-induced Insulin Resistance.

Degree: MS, Biomedical Sciences, 2013, Texas A&M University

 Macrophage activation plays a crucial role in regulating adipose tissue inflammation and is a major contributor to the pathogenesis of obesity-associated cardiovascular diseases. On various… (more)

Subjects/Keywords: microRNAs; macrophages; adipose tissue inflammation

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APA (6th Edition):

Meng, C. (2013). microRNA-223 Regulates Macrophage Polarization and Diet-induced Insulin Resistance. (Masters Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/149464

Chicago Manual of Style (16th Edition):

Meng, Cong. “microRNA-223 Regulates Macrophage Polarization and Diet-induced Insulin Resistance.” 2013. Masters Thesis, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/149464.

MLA Handbook (7th Edition):

Meng, Cong. “microRNA-223 Regulates Macrophage Polarization and Diet-induced Insulin Resistance.” 2013. Web. 28 Nov 2020.

Vancouver:

Meng C. microRNA-223 Regulates Macrophage Polarization and Diet-induced Insulin Resistance. [Internet] [Masters thesis]. Texas A&M University; 2013. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/149464.

Council of Science Editors:

Meng C. microRNA-223 Regulates Macrophage Polarization and Diet-induced Insulin Resistance. [Masters Thesis]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/149464

12. Amini-Vaughan, Zhaleh Jacqueline. Harnessing Eosinophils as a Potential Tumoricidal Immunotherapy.

Degree: MS, Medical Sciences, 2016, Texas A&M University

 Eosinophils have numerous functions for defense against pathogens, despite being only 1-5% of total circulating blood leukocytes. Most well known for their actions contributing to… (more)

Subjects/Keywords: eosinophil; cancer; immunotherapy

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APA (6th Edition):

Amini-Vaughan, Z. J. (2016). Harnessing Eosinophils as a Potential Tumoricidal Immunotherapy. (Masters Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/157005

Chicago Manual of Style (16th Edition):

Amini-Vaughan, Zhaleh Jacqueline. “Harnessing Eosinophils as a Potential Tumoricidal Immunotherapy.” 2016. Masters Thesis, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/157005.

MLA Handbook (7th Edition):

Amini-Vaughan, Zhaleh Jacqueline. “Harnessing Eosinophils as a Potential Tumoricidal Immunotherapy.” 2016. Web. 28 Nov 2020.

Vancouver:

Amini-Vaughan ZJ. Harnessing Eosinophils as a Potential Tumoricidal Immunotherapy. [Internet] [Masters thesis]. Texas A&M University; 2016. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/157005.

Council of Science Editors:

Amini-Vaughan ZJ. Harnessing Eosinophils as a Potential Tumoricidal Immunotherapy. [Masters Thesis]. Texas A&M University; 2016. Available from: http://hdl.handle.net/1969.1/157005

13. Walker, Kelcey Manae Becker. Inhibitory actions of Ah receptor agonists and indole-containing compounds in breast cancer cell lines and mouse models.

Degree: PhD, Toxicology, 2005, Texas A&M University

 The aryl hydrocarbon receptor (AhR) binds synthetic and chemoprotective phytochemicals, and research in this laboratory has developed selective AhR modulators (SAhRMs) for treatment of breast… (more)

Subjects/Keywords: estrogen receptor; aryl hydrocarbon receptor; ErbB2; TCDD; MMTV-c-neu; SAhRM; uterus; ERKO; AhRKO; BT-474; MDA-MB-453

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APA (6th Edition):

Walker, K. M. B. (2005). Inhibitory actions of Ah receptor agonists and indole-containing compounds in breast cancer cell lines and mouse models. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/2433

Chicago Manual of Style (16th Edition):

Walker, Kelcey Manae Becker. “Inhibitory actions of Ah receptor agonists and indole-containing compounds in breast cancer cell lines and mouse models.” 2005. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/2433.

MLA Handbook (7th Edition):

Walker, Kelcey Manae Becker. “Inhibitory actions of Ah receptor agonists and indole-containing compounds in breast cancer cell lines and mouse models.” 2005. Web. 28 Nov 2020.

Vancouver:

Walker KMB. Inhibitory actions of Ah receptor agonists and indole-containing compounds in breast cancer cell lines and mouse models. [Internet] [Doctoral dissertation]. Texas A&M University; 2005. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/2433.

Council of Science Editors:

Walker KMB. Inhibitory actions of Ah receptor agonists and indole-containing compounds in breast cancer cell lines and mouse models. [Doctoral Dissertation]. Texas A&M University; 2005. Available from: http://hdl.handle.net/1969.1/2433

14. Jutooru, Indira Devi. New Mechanism Based Anticancer Drugs for Treatment of Pancreatic and Bladder Cancers.

Degree: PhD, Toxicology, 2011, Texas A&M University

 Methyl 2-cyano-3,11-dioxo-18b-olean-1,12-dien-30-oate (CDODA-Me) is a synthetic triterpenoid that inhibits growth of Panc1 and Panc28 pancreatic cancer cell lines and activates peroxisome proliferator-activated receptor B (PPARB)-dependent… (more)

Subjects/Keywords: Pancreatic and bladder cancer; CDODA-Me; Curcumin; Arsenic trioxide; CDDO-Me; Celastrol; Sp transcription factors

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APA (6th Edition):

Jutooru, I. D. (2011). New Mechanism Based Anticancer Drugs for Treatment of Pancreatic and Bladder Cancers. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2010-05-7860

Chicago Manual of Style (16th Edition):

Jutooru, Indira Devi. “New Mechanism Based Anticancer Drugs for Treatment of Pancreatic and Bladder Cancers.” 2011. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/ETD-TAMU-2010-05-7860.

MLA Handbook (7th Edition):

Jutooru, Indira Devi. “New Mechanism Based Anticancer Drugs for Treatment of Pancreatic and Bladder Cancers.” 2011. Web. 28 Nov 2020.

Vancouver:

Jutooru ID. New Mechanism Based Anticancer Drugs for Treatment of Pancreatic and Bladder Cancers. [Internet] [Doctoral dissertation]. Texas A&M University; 2011. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2010-05-7860.

Council of Science Editors:

Jutooru ID. New Mechanism Based Anticancer Drugs for Treatment of Pancreatic and Bladder Cancers. [Doctoral Dissertation]. Texas A&M University; 2011. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2010-05-7860

15. Johnson, Natalie Malek. Biomarkers of Exposure to Foodborne and Environmental Carcinogens: Enterosorbent Intervention in a High Risk Population.

Degree: PhD, Toxicology, 2011, Texas A&M University

 The need to assess human exposures to foodborne and environmental carcinogens, particularly in populations at high risk for cancer and disease, has led to the… (more)

Subjects/Keywords: Aflatoxin; enterosorbent; NovaSil clay; biomarkers; micronutrients; minerals; polycyclic aromatic hydrocarbons; food safety; environment; hepatocellular carcinoma; hepatitis C virus

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APA (6th Edition):

Johnson, N. M. (2011). Biomarkers of Exposure to Foodborne and Environmental Carcinogens: Enterosorbent Intervention in a High Risk Population. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2010-08-8342

Chicago Manual of Style (16th Edition):

Johnson, Natalie Malek. “Biomarkers of Exposure to Foodborne and Environmental Carcinogens: Enterosorbent Intervention in a High Risk Population.” 2011. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/ETD-TAMU-2010-08-8342.

MLA Handbook (7th Edition):

Johnson, Natalie Malek. “Biomarkers of Exposure to Foodborne and Environmental Carcinogens: Enterosorbent Intervention in a High Risk Population.” 2011. Web. 28 Nov 2020.

Vancouver:

Johnson NM. Biomarkers of Exposure to Foodborne and Environmental Carcinogens: Enterosorbent Intervention in a High Risk Population. [Internet] [Doctoral dissertation]. Texas A&M University; 2011. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2010-08-8342.

Council of Science Editors:

Johnson NM. Biomarkers of Exposure to Foodborne and Environmental Carcinogens: Enterosorbent Intervention in a High Risk Population. [Doctoral Dissertation]. Texas A&M University; 2011. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2010-08-8342

16. Chadalapaka, Gayathri. Mechanism Based Anticancer Drugs that Degrade Sp Transcription Factors.

Degree: PhD, Toxicology, 2013, Texas A&M University

 Curcumin is the active component of tumeric, and this polyphenolic compound has been extensively investigated as an anticancer drug that modulates multiple pathways and genes.… (more)

Subjects/Keywords: cancer; Bladder cancer; Pancreatic cancer Sp proteins; natural products

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APA (6th Edition):

Chadalapaka, G. (2013). Mechanism Based Anticancer Drugs that Degrade Sp Transcription Factors. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/148439

Chicago Manual of Style (16th Edition):

Chadalapaka, Gayathri. “Mechanism Based Anticancer Drugs that Degrade Sp Transcription Factors.” 2013. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/148439.

MLA Handbook (7th Edition):

Chadalapaka, Gayathri. “Mechanism Based Anticancer Drugs that Degrade Sp Transcription Factors.” 2013. Web. 28 Nov 2020.

Vancouver:

Chadalapaka G. Mechanism Based Anticancer Drugs that Degrade Sp Transcription Factors. [Internet] [Doctoral dissertation]. Texas A&M University; 2013. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/148439.

Council of Science Editors:

Chadalapaka G. Mechanism Based Anticancer Drugs that Degrade Sp Transcription Factors. [Doctoral Dissertation]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/148439

17. Cho, Sunggook Gook. KISS1 and Its G Protein-Coupled Receptor (GPR54) in Cancer Progression and Metastasis.

Degree: PhD, Genetics, 2012, Texas A&M University

 Activation of G-protein coupled receptor 54 (GPR54) signaling generated by kisspeptins (endogenous GPR54 ligands encoded by KISS1 gene) has been known to regulate puberty and… (more)

Subjects/Keywords: KISS1; GPR54; Cancer

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APA (6th Edition):

Cho, S. G. (2012). KISS1 and Its G Protein-Coupled Receptor (GPR54) in Cancer Progression and Metastasis. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2010-12-8892

Chicago Manual of Style (16th Edition):

Cho, Sunggook Gook. “KISS1 and Its G Protein-Coupled Receptor (GPR54) in Cancer Progression and Metastasis.” 2012. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/ETD-TAMU-2010-12-8892.

MLA Handbook (7th Edition):

Cho, Sunggook Gook. “KISS1 and Its G Protein-Coupled Receptor (GPR54) in Cancer Progression and Metastasis.” 2012. Web. 28 Nov 2020.

Vancouver:

Cho SG. KISS1 and Its G Protein-Coupled Receptor (GPR54) in Cancer Progression and Metastasis. [Internet] [Doctoral dissertation]. Texas A&M University; 2012. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2010-12-8892.

Council of Science Editors:

Cho SG. KISS1 and Its G Protein-Coupled Receptor (GPR54) in Cancer Progression and Metastasis. [Doctoral Dissertation]. Texas A&M University; 2012. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2010-12-8892

18. Reddivari, Lavanya. Influence of genetic variability on specialty potato functional components and their effect on prostate cancer cell lines.

Degree: PhD, Horticulture, 2009, Texas A&M University

 The influence of genotype (selection), location, and year on antioxidant activity (AOA), total phenolics (TP), total carotenoids (TC), phenolic and carotenoid composition was studied using… (more)

Subjects/Keywords: Potato; anthocyanins; phenolic acids; prostate cancer cells; caspase-dependent; caspase-independent; apoptosis

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APA (6th Edition):

Reddivari, L. (2009). Influence of genetic variability on specialty potato functional components and their effect on prostate cancer cell lines. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-1330

Chicago Manual of Style (16th Edition):

Reddivari, Lavanya. “Influence of genetic variability on specialty potato functional components and their effect on prostate cancer cell lines.” 2009. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/ETD-TAMU-1330.

MLA Handbook (7th Edition):

Reddivari, Lavanya. “Influence of genetic variability on specialty potato functional components and their effect on prostate cancer cell lines.” 2009. Web. 28 Nov 2020.

Vancouver:

Reddivari L. Influence of genetic variability on specialty potato functional components and their effect on prostate cancer cell lines. [Internet] [Doctoral dissertation]. Texas A&M University; 2009. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-1330.

Council of Science Editors:

Reddivari L. Influence of genetic variability on specialty potato functional components and their effect on prostate cancer cell lines. [Doctoral Dissertation]. Texas A&M University; 2009. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-1330

19. Naufal, Ziad Sami. Biomarkers of exposure to complex environmental mixtures.

Degree: PhD, Toxicology, 2009, Texas A&M University

 Maternal exposure to genotoxic chemicals may produce a variety of adverse birth outcomes. Depending on the dose and duration of exposure, adverse birth outcomes can… (more)

Subjects/Keywords: PAHs; Biomarkers

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APA (6th Edition):

Naufal, Z. S. (2009). Biomarkers of exposure to complex environmental mixtures. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-3284

Chicago Manual of Style (16th Edition):

Naufal, Ziad Sami. “Biomarkers of exposure to complex environmental mixtures.” 2009. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/ETD-TAMU-3284.

MLA Handbook (7th Edition):

Naufal, Ziad Sami. “Biomarkers of exposure to complex environmental mixtures.” 2009. Web. 28 Nov 2020.

Vancouver:

Naufal ZS. Biomarkers of exposure to complex environmental mixtures. [Internet] [Doctoral dissertation]. Texas A&M University; 2009. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-3284.

Council of Science Editors:

Naufal ZS. Biomarkers of exposure to complex environmental mixtures. [Doctoral Dissertation]. Texas A&M University; 2009. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-3284

20. Kim, KyoungHyun. Domain analysis for estrogen receptor/Sp1-mediated transactivation and detection of estrogen receptor/Sp1 protein interactions in living cells.

Degree: PhD, Toxicology, 2005, Texas A&M University

 Estrogen Receptor ? (ER?)/Sp1 activation of GC-rich gene promoters in breast cancer cells is dependent, in part, on the activation function 1 (AF1) of ER?.… (more)

Subjects/Keywords: Estrogen receptor; Sp1; antiestrogens

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APA (6th Edition):

Kim, K. (2005). Domain analysis for estrogen receptor/Sp1-mediated transactivation and detection of estrogen receptor/Sp1 protein interactions in living cells. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/2666

Chicago Manual of Style (16th Edition):

Kim, KyoungHyun. “Domain analysis for estrogen receptor/Sp1-mediated transactivation and detection of estrogen receptor/Sp1 protein interactions in living cells.” 2005. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/2666.

MLA Handbook (7th Edition):

Kim, KyoungHyun. “Domain analysis for estrogen receptor/Sp1-mediated transactivation and detection of estrogen receptor/Sp1 protein interactions in living cells.” 2005. Web. 28 Nov 2020.

Vancouver:

Kim K. Domain analysis for estrogen receptor/Sp1-mediated transactivation and detection of estrogen receptor/Sp1 protein interactions in living cells. [Internet] [Doctoral dissertation]. Texas A&M University; 2005. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/2666.

Council of Science Editors:

Kim K. Domain analysis for estrogen receptor/Sp1-mediated transactivation and detection of estrogen receptor/Sp1 protein interactions in living cells. [Doctoral Dissertation]. Texas A&M University; 2005. Available from: http://hdl.handle.net/1969.1/2666

21. Khan, Shaheen Munawar Ali. Mechanisms of hormonal regulation of CAD gene expression and inhibition by Aryl hydrocarbon receptor agonist in human breast cancer cells.

Degree: PhD, Genetics, 2007, Texas A&M University

 The CAD gene is trifunctional and expresses carbamoylphosphate synthetase/aspartate carbamyltransferase/dihydroorotase, which are required for pyrimidine biosynthesis. CAD gene activities are induced in MCF-7 human breast… (more)

Subjects/Keywords: CAD; Breast Cancer

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APA (6th Edition):

Khan, S. M. A. (2007). Mechanisms of hormonal regulation of CAD gene expression and inhibition by Aryl hydrocarbon receptor agonist in human breast cancer cells. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/4935

Chicago Manual of Style (16th Edition):

Khan, Shaheen Munawar Ali. “Mechanisms of hormonal regulation of CAD gene expression and inhibition by Aryl hydrocarbon receptor agonist in human breast cancer cells.” 2007. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/4935.

MLA Handbook (7th Edition):

Khan, Shaheen Munawar Ali. “Mechanisms of hormonal regulation of CAD gene expression and inhibition by Aryl hydrocarbon receptor agonist in human breast cancer cells.” 2007. Web. 28 Nov 2020.

Vancouver:

Khan SMA. Mechanisms of hormonal regulation of CAD gene expression and inhibition by Aryl hydrocarbon receptor agonist in human breast cancer cells. [Internet] [Doctoral dissertation]. Texas A&M University; 2007. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/4935.

Council of Science Editors:

Khan SMA. Mechanisms of hormonal regulation of CAD gene expression and inhibition by Aryl hydrocarbon receptor agonist in human breast cancer cells. [Doctoral Dissertation]. Texas A&M University; 2007. Available from: http://hdl.handle.net/1969.1/4935

22. Kwak, Hyeong-il. The Role of single minded 2 short in mammary gland development and breast cancer.

Degree: PhD, Toxicology, 2009, Texas A&M University

 Single minded 2 (Sim2) is a member of the basic helix-loop-helix Per-ARNT-Sim (Period-Arylhydrocarbon Nuclear Translocator-Single minded) family. Human SIM2 is involved in the etiology of… (more)

Subjects/Keywords: sim2s; breast cancer; MMP; E-cad; Tumor Suppressor; EMT

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APA (6th Edition):

Kwak, H. (2009). The Role of single minded 2 short in mammary gland development and breast cancer. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-1206

Chicago Manual of Style (16th Edition):

Kwak, Hyeong-il. “The Role of single minded 2 short in mammary gland development and breast cancer.” 2009. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/ETD-TAMU-1206.

MLA Handbook (7th Edition):

Kwak, Hyeong-il. “The Role of single minded 2 short in mammary gland development and breast cancer.” 2009. Web. 28 Nov 2020.

Vancouver:

Kwak H. The Role of single minded 2 short in mammary gland development and breast cancer. [Internet] [Doctoral dissertation]. Texas A&M University; 2009. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-1206.

Council of Science Editors:

Kwak H. The Role of single minded 2 short in mammary gland development and breast cancer. [Doctoral Dissertation]. Texas A&M University; 2009. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-1206

23. Chen, Chien-Cheng. Mechanisms of transcriptional activation of estrogen responsive genes in breast cancer cells.

Degree: PhD, Toxicology, 2009, Texas A&M University

 Estrogen receptor (ER) acts as a ligand-activated transcription factor that regulates the expression of genes. The genomic mechanisms of ER action include ligand-induced dimerization of… (more)

Subjects/Keywords: Estrogen; Transcription

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APA (6th Edition):

Chen, C. (2009). Mechanisms of transcriptional activation of estrogen responsive genes in breast cancer cells. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-1869

Chicago Manual of Style (16th Edition):

Chen, Chien-Cheng. “Mechanisms of transcriptional activation of estrogen responsive genes in breast cancer cells.” 2009. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/ETD-TAMU-1869.

MLA Handbook (7th Edition):

Chen, Chien-Cheng. “Mechanisms of transcriptional activation of estrogen responsive genes in breast cancer cells.” 2009. Web. 28 Nov 2020.

Vancouver:

Chen C. Mechanisms of transcriptional activation of estrogen responsive genes in breast cancer cells. [Internet] [Doctoral dissertation]. Texas A&M University; 2009. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-1869.

Council of Science Editors:

Chen C. Mechanisms of transcriptional activation of estrogen responsive genes in breast cancer cells. [Doctoral Dissertation]. Texas A&M University; 2009. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-1869

24. Kim, Myung Hee 1982-. Factors Affecting Biodefluorination of Fluorotelomer Alcohols (FTOHs): Degradative Microorganisms, Transformation Metabolites and Pathways, and Effects of Co-substrates.

Degree: PhD, Civil Engineering, 2012, Texas A&M University

 Fluorotelomer alcohols (FTOHs, F(CF2)nCH2CH2OH) are emerging contaminants in the environment. Biodegradation of 6:2 and 8:2 FTOHs has been intensively studied using soils and activated sludge.… (more)

Subjects/Keywords: Real-time-t-RFLP; Catabolic gene; FTOH-degrading consortia; Fluoroacetate-degrading bacteria; Alkane-degrading bacteria; Biodegradation pathways; Biodefluorination; Fluorotelomer Alcohols (FTOHs)

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APA (6th Edition):

Kim, M. H. 1. (2012). Factors Affecting Biodefluorination of Fluorotelomer Alcohols (FTOHs): Degradative Microorganisms, Transformation Metabolites and Pathways, and Effects of Co-substrates. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/148234

Chicago Manual of Style (16th Edition):

Kim, Myung Hee 1982-. “Factors Affecting Biodefluorination of Fluorotelomer Alcohols (FTOHs): Degradative Microorganisms, Transformation Metabolites and Pathways, and Effects of Co-substrates.” 2012. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/148234.

MLA Handbook (7th Edition):

Kim, Myung Hee 1982-. “Factors Affecting Biodefluorination of Fluorotelomer Alcohols (FTOHs): Degradative Microorganisms, Transformation Metabolites and Pathways, and Effects of Co-substrates.” 2012. Web. 28 Nov 2020.

Vancouver:

Kim MH1. Factors Affecting Biodefluorination of Fluorotelomer Alcohols (FTOHs): Degradative Microorganisms, Transformation Metabolites and Pathways, and Effects of Co-substrates. [Internet] [Doctoral dissertation]. Texas A&M University; 2012. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/148234.

Council of Science Editors:

Kim MH1. Factors Affecting Biodefluorination of Fluorotelomer Alcohols (FTOHs): Degradative Microorganisms, Transformation Metabolites and Pathways, and Effects of Co-substrates. [Doctoral Dissertation]. Texas A&M University; 2012. Available from: http://hdl.handle.net/1969.1/148234

25. Abdel Rahim, Ma'en Ahmad. Gene silencing in cancer cells using siRNA : genetic and functional studies.

Degree: PhD, Toxicology, 2004, Texas A&M University

 Sequence-specific small interfering RNA (siRNA) duplexes can be used for gene silencing in mammalian cells and as mechanistic probes for determining gene function. Transfection of… (more)

Subjects/Keywords: RNAi; siRNA; Sp proteins; TCDD; AhR; ARNT; VEGF; breast cancer; pancreatic cancer.

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APA (6th Edition):

Abdel Rahim, M. A. (2004). Gene silencing in cancer cells using siRNA : genetic and functional studies. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/218

Chicago Manual of Style (16th Edition):

Abdel Rahim, Ma'en Ahmad. “Gene silencing in cancer cells using siRNA : genetic and functional studies.” 2004. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/218.

MLA Handbook (7th Edition):

Abdel Rahim, Ma'en Ahmad. “Gene silencing in cancer cells using siRNA : genetic and functional studies.” 2004. Web. 28 Nov 2020.

Vancouver:

Abdel Rahim MA. Gene silencing in cancer cells using siRNA : genetic and functional studies. [Internet] [Doctoral dissertation]. Texas A&M University; 2004. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/218.

Council of Science Editors:

Abdel Rahim MA. Gene silencing in cancer cells using siRNA : genetic and functional studies. [Doctoral Dissertation]. Texas A&M University; 2004. Available from: http://hdl.handle.net/1969.1/218

26. Hosako, Hiromi. The role of p53 in normal development and teratogen-induced apoptosis and birth defects in mouse embryos.

Degree: PhD, Toxicology, 2009, Texas A&M University

 In the studies described in this dissertation, we investigated the roles of p53 in normal development, teratogen-induced apoptosis, and birth defects. In the first study,… (more)

Subjects/Keywords: p53; Apoptosis; Birth Defects; Teratogens

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APA (6th Edition):

Hosako, H. (2009). The role of p53 in normal development and teratogen-induced apoptosis and birth defects in mouse embryos. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-3251

Chicago Manual of Style (16th Edition):

Hosako, Hiromi. “The role of p53 in normal development and teratogen-induced apoptosis and birth defects in mouse embryos.” 2009. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/ETD-TAMU-3251.

MLA Handbook (7th Edition):

Hosako, Hiromi. “The role of p53 in normal development and teratogen-induced apoptosis and birth defects in mouse embryos.” 2009. Web. 28 Nov 2020.

Vancouver:

Hosako H. The role of p53 in normal development and teratogen-induced apoptosis and birth defects in mouse embryos. [Internet] [Doctoral dissertation]. Texas A&M University; 2009. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-3251.

Council of Science Editors:

Hosako H. The role of p53 in normal development and teratogen-induced apoptosis and birth defects in mouse embryos. [Doctoral Dissertation]. Texas A&M University; 2009. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-3251


Texas A&M University

27. Higgins, Kelly Jean. Regulation of vascular endothelial growth factor receptor-2 in pancreatic and breast cancer cells by Sp proteins.

Degree: PhD, Biochemistry, 2007, Texas A&M University

 Vascular endothelial growth factor receptor-2 (VEGFR2) is a key angiogenic factor, and angiogenesis is an important physiological process associated with neovascularization, growth, and metastasis of… (more)

Subjects/Keywords: mechanisms of gene transcription; angiogenesis in cancer

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APA (6th Edition):

Higgins, K. J. (2007). Regulation of vascular endothelial growth factor receptor-2 in pancreatic and breast cancer cells by Sp proteins. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/6010

Chicago Manual of Style (16th Edition):

Higgins, Kelly Jean. “Regulation of vascular endothelial growth factor receptor-2 in pancreatic and breast cancer cells by Sp proteins.” 2007. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/6010.

MLA Handbook (7th Edition):

Higgins, Kelly Jean. “Regulation of vascular endothelial growth factor receptor-2 in pancreatic and breast cancer cells by Sp proteins.” 2007. Web. 28 Nov 2020.

Vancouver:

Higgins KJ. Regulation of vascular endothelial growth factor receptor-2 in pancreatic and breast cancer cells by Sp proteins. [Internet] [Doctoral dissertation]. Texas A&M University; 2007. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/6010.

Council of Science Editors:

Higgins KJ. Regulation of vascular endothelial growth factor receptor-2 in pancreatic and breast cancer cells by Sp proteins. [Doctoral Dissertation]. Texas A&M University; 2007. Available from: http://hdl.handle.net/1969.1/6010


Texas A&M University

28. Zhang, Shu. Role of estrogen receptor alpha (ER alpha) insulin-like growth factor (IGF)-I-induced responses in MCF-7 breast cancer cells.

Degree: PhD, Toxicology, 2009, Texas A&M University

 Insulin-like growth factor-I (IGF-I) is a mitogenic polypeptide that induces proliferation and activation of kinase pathways in MCF-7 breast cancer cells. The role of estrogen… (more)

Subjects/Keywords: IGF-I; breast cancer

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APA (6th Edition):

Zhang, S. (2009). Role of estrogen receptor alpha (ER alpha) insulin-like growth factor (IGF)-I-induced responses in MCF-7 breast cancer cells. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2603

Chicago Manual of Style (16th Edition):

Zhang, Shu. “Role of estrogen receptor alpha (ER alpha) insulin-like growth factor (IGF)-I-induced responses in MCF-7 breast cancer cells.” 2009. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/ETD-TAMU-2603.

MLA Handbook (7th Edition):

Zhang, Shu. “Role of estrogen receptor alpha (ER alpha) insulin-like growth factor (IGF)-I-induced responses in MCF-7 breast cancer cells.” 2009. Web. 28 Nov 2020.

Vancouver:

Zhang S. Role of estrogen receptor alpha (ER alpha) insulin-like growth factor (IGF)-I-induced responses in MCF-7 breast cancer cells. [Internet] [Doctoral dissertation]. Texas A&M University; 2009. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2603.

Council of Science Editors:

Zhang S. Role of estrogen receptor alpha (ER alpha) insulin-like growth factor (IGF)-I-induced responses in MCF-7 breast cancer cells. [Doctoral Dissertation]. Texas A&M University; 2009. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2603


Texas A&M University

29. Lee, Wan-Ru. Mechanisms of hormonal activation of Cdc25A and coactivation of estrogen receptor alpha by protein inhibitor of activated STAT3 (PIAS3).

Degree: PhD, Toxicology, 2009, Texas A&M University

 The estrogen receptor (ER) is a ligand-activated transcription factor that regulates gene expression. The classical mechanisms of nuclear ER action include ligand-induced dimerization of ER… (more)

Subjects/Keywords: Estrogen receptor; Cdc25A; PIAS3

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APA (6th Edition):

Lee, W. (2009). Mechanisms of hormonal activation of Cdc25A and coactivation of estrogen receptor alpha by protein inhibitor of activated STAT3 (PIAS3). (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-1207

Chicago Manual of Style (16th Edition):

Lee, Wan-Ru. “Mechanisms of hormonal activation of Cdc25A and coactivation of estrogen receptor alpha by protein inhibitor of activated STAT3 (PIAS3).” 2009. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/ETD-TAMU-1207.

MLA Handbook (7th Edition):

Lee, Wan-Ru. “Mechanisms of hormonal activation of Cdc25A and coactivation of estrogen receptor alpha by protein inhibitor of activated STAT3 (PIAS3).” 2009. Web. 28 Nov 2020.

Vancouver:

Lee W. Mechanisms of hormonal activation of Cdc25A and coactivation of estrogen receptor alpha by protein inhibitor of activated STAT3 (PIAS3). [Internet] [Doctoral dissertation]. Texas A&M University; 2009. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-1207.

Council of Science Editors:

Lee W. Mechanisms of hormonal activation of Cdc25A and coactivation of estrogen receptor alpha by protein inhibitor of activated STAT3 (PIAS3). [Doctoral Dissertation]. Texas A&M University; 2009. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-1207


Texas A&M University

30. Hill, Denise Suzanne. Investigations into arsenate-induced neural tube defects in a mouse model.

Degree: PhD, Toxicology, 2009, Texas A&M University

 Neural tube defects (NTDs) are malformations affecting about 2.6/1000 births worldwide, and 1/1000 in the United States. Their etiology remains unknown, and is likely due… (more)

Subjects/Keywords: arsenic; teratogen; arsenate; NTD; environmental; neural tube defect

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APA (6th Edition):

Hill, D. S. (2009). Investigations into arsenate-induced neural tube defects in a mouse model. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-3274

Chicago Manual of Style (16th Edition):

Hill, Denise Suzanne. “Investigations into arsenate-induced neural tube defects in a mouse model.” 2009. Doctoral Dissertation, Texas A&M University. Accessed November 28, 2020. http://hdl.handle.net/1969.1/ETD-TAMU-3274.

MLA Handbook (7th Edition):

Hill, Denise Suzanne. “Investigations into arsenate-induced neural tube defects in a mouse model.” 2009. Web. 28 Nov 2020.

Vancouver:

Hill DS. Investigations into arsenate-induced neural tube defects in a mouse model. [Internet] [Doctoral dissertation]. Texas A&M University; 2009. [cited 2020 Nov 28]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-3274.

Council of Science Editors:

Hill DS. Investigations into arsenate-induced neural tube defects in a mouse model. [Doctoral Dissertation]. Texas A&M University; 2009. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-3274

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