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You searched for +publisher:"Texas A&M University" +contributor:("Miranda, Rajesh"). Showing records 1 – 30 of 47 total matches.

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Texas A&M University

1. Kanameni, Srikanth. Transcriptional Profiling of Polarized Macrophages using RNA-Sequencing.

Degree: 2015, Texas A&M University

 Adipose tissue macrophages (ATMs) are pivotal regulators for adipose tissue function, specifically contributing to the homeostasis of the adipose niche. Significantly increased ATMs and their… (more)

Subjects/Keywords: Macrophages; RNA-Sequencing

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kanameni, S. (2015). Transcriptional Profiling of Polarized Macrophages using RNA-Sequencing. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/155022

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kanameni, Srikanth. “Transcriptional Profiling of Polarized Macrophages using RNA-Sequencing.” 2015. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/155022.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kanameni, Srikanth. “Transcriptional Profiling of Polarized Macrophages using RNA-Sequencing.” 2015. Web. 20 Apr 2019.

Vancouver:

Kanameni S. Transcriptional Profiling of Polarized Macrophages using RNA-Sequencing. [Internet] [Thesis]. Texas A&M University; 2015. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/155022.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kanameni S. Transcriptional Profiling of Polarized Macrophages using RNA-Sequencing. [Thesis]. Texas A&M University; 2015. Available from: http://hdl.handle.net/1969.1/155022

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

2. Chronister, Sarah Katherine. Preconception Chronic Male Alcohol Exposure Induces FAS Like Growth Restriction.

Degree: MS, Biomedical Sciences, 2016, Texas A&M University

 Epidemiologic studies demonstrate that alcohol is the most prevalent teratogen to which humans are exposed. In the US, it is estimated that at least one… (more)

Subjects/Keywords: FAS; Paternal Exposure to Alcohol

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APA (6th Edition):

Chronister, S. K. (2016). Preconception Chronic Male Alcohol Exposure Induces FAS Like Growth Restriction. (Masters Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/158134

Chicago Manual of Style (16th Edition):

Chronister, Sarah Katherine. “Preconception Chronic Male Alcohol Exposure Induces FAS Like Growth Restriction.” 2016. Masters Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/158134.

MLA Handbook (7th Edition):

Chronister, Sarah Katherine. “Preconception Chronic Male Alcohol Exposure Induces FAS Like Growth Restriction.” 2016. Web. 20 Apr 2019.

Vancouver:

Chronister SK. Preconception Chronic Male Alcohol Exposure Induces FAS Like Growth Restriction. [Internet] [Masters thesis]. Texas A&M University; 2016. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/158134.

Council of Science Editors:

Chronister SK. Preconception Chronic Male Alcohol Exposure Induces FAS Like Growth Restriction. [Masters Thesis]. Texas A&M University; 2016. Available from: http://hdl.handle.net/1969.1/158134


Texas A&M University

3. Gillis, David Christopher. The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain.

Degree: 2015, Texas A&M University

 Inflammation in the developing central nervous system (CNS) can contribute to numerous issues in the fully developed adult. Uncovering pathways responsible for the inflammation is… (more)

Subjects/Keywords: RIP3; neuroinflammation; LPS; programmed cell death

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APA (6th Edition):

Gillis, D. C. (2015). The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/155480

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Gillis, David Christopher. “The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain.” 2015. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/155480.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Gillis, David Christopher. “The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain.” 2015. Web. 20 Apr 2019.

Vancouver:

Gillis DC. The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain. [Internet] [Thesis]. Texas A&M University; 2015. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/155480.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Gillis DC. The Role of RIP3 in Neuroinflammation in the Postnatal Mouse Brain. [Thesis]. Texas A&M University; 2015. Available from: http://hdl.handle.net/1969.1/155480

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

4. Wang, Haiqing. Preconception Paternal Alcohol Exposure Disrupts Placental Gene Expression and Alters Chromatin Boundary Elements.

Degree: 2017, Texas A&M University

 Fetal alcohol spectrum disorders (FASD) are a spectrum of pathophysiological consequences that include structural, neurological and behavior disorders that arise following prenatal alcohol exposure (PAE).… (more)

Subjects/Keywords: paternal; FASD; CTCF

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APA (6th Edition):

Wang, H. (2017). Preconception Paternal Alcohol Exposure Disrupts Placental Gene Expression and Alters Chromatin Boundary Elements. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/161417

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wang, Haiqing. “Preconception Paternal Alcohol Exposure Disrupts Placental Gene Expression and Alters Chromatin Boundary Elements.” 2017. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/161417.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wang, Haiqing. “Preconception Paternal Alcohol Exposure Disrupts Placental Gene Expression and Alters Chromatin Boundary Elements.” 2017. Web. 20 Apr 2019.

Vancouver:

Wang H. Preconception Paternal Alcohol Exposure Disrupts Placental Gene Expression and Alters Chromatin Boundary Elements. [Internet] [Thesis]. Texas A&M University; 2017. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/161417.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang H. Preconception Paternal Alcohol Exposure Disrupts Placental Gene Expression and Alters Chromatin Boundary Elements. [Thesis]. Texas A&M University; 2017. Available from: http://hdl.handle.net/1969.1/161417

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

5. Huie, John Russell. The role of BDNF in spinal learning.

Degree: 2009, Texas A&M University

 Previous research in our laboratory has shown that the spinal cord is capable of a simple form of instrumental learning. Spinally transected rats that receive… (more)

Subjects/Keywords: Spinal Learning; BDNF

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APA (6th Edition):

Huie, J. R. (2009). The role of BDNF in spinal learning. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2824

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Huie, John Russell. “The role of BDNF in spinal learning.” 2009. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/ETD-TAMU-2824.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Huie, John Russell. “The role of BDNF in spinal learning.” 2009. Web. 20 Apr 2019.

Vancouver:

Huie JR. The role of BDNF in spinal learning. [Internet] [Thesis]. Texas A&M University; 2009. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2824.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huie JR. The role of BDNF in spinal learning. [Thesis]. Texas A&M University; 2009. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2824

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

6. Hoy, Kevin Corcoran. AMPA-receptor mediated plasticity within the rat spinal cord.

Degree: 2009, Texas A&M University

 Previous research from our laboratory has demonstrated that the spinal cord is capable of a simple form of instrumental learning. In this instrumental learning paradigm,… (more)

Subjects/Keywords: Learning

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APA (6th Edition):

Hoy, K. C. (2009). AMPA-receptor mediated plasticity within the rat spinal cord. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-3056

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hoy, Kevin Corcoran. “AMPA-receptor mediated plasticity within the rat spinal cord.” 2009. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/ETD-TAMU-3056.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hoy, Kevin Corcoran. “AMPA-receptor mediated plasticity within the rat spinal cord.” 2009. Web. 20 Apr 2019.

Vancouver:

Hoy KC. AMPA-receptor mediated plasticity within the rat spinal cord. [Internet] [Thesis]. Texas A&M University; 2009. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-3056.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hoy KC. AMPA-receptor mediated plasticity within the rat spinal cord. [Thesis]. Texas A&M University; 2009. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-3056

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

7. Puga, Denise Alejandra. Pain Processing in the Isolated Spinal Cord: Adaptive Nociceptive Modifications.

Degree: 2012, Texas A&M University

 We utilize a simple instrumental (response-outcome) learning task to measure spinal plasticity in the isolated spinal cord. Peripheral uncontrollable nociceptive input has been shown to… (more)

Subjects/Keywords: Spinal cord; plasticity; spinal learning; antinociception

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APA (6th Edition):

Puga, D. A. (2012). Pain Processing in the Isolated Spinal Cord: Adaptive Nociceptive Modifications. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2011-05-9149

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Puga, Denise Alejandra. “Pain Processing in the Isolated Spinal Cord: Adaptive Nociceptive Modifications.” 2012. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/ETD-TAMU-2011-05-9149.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Puga, Denise Alejandra. “Pain Processing in the Isolated Spinal Cord: Adaptive Nociceptive Modifications.” 2012. Web. 20 Apr 2019.

Vancouver:

Puga DA. Pain Processing in the Isolated Spinal Cord: Adaptive Nociceptive Modifications. [Internet] [Thesis]. Texas A&M University; 2012. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2011-05-9149.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Puga DA. Pain Processing in the Isolated Spinal Cord: Adaptive Nociceptive Modifications. [Thesis]. Texas A&M University; 2012. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2011-05-9149

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

8. Strickland, Eric. MicroRNA Dysregulation Following Spinal Cord Contusion: Implications for Neural Plasticity and Neuropathic Pain.

Degree: 2013, Texas A&M University

 Spinal cord injury (SCI) results in a number of devastating consequences, including loss of motor function, paralysis, and neuropathic pain. Concomitant peripheral tissue injury below… (more)

Subjects/Keywords: Spinal cord injury; miRNAs; Neuropathic pain; morphine; dysregulation; neuronal plasticity

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APA (6th Edition):

Strickland, E. (2013). MicroRNA Dysregulation Following Spinal Cord Contusion: Implications for Neural Plasticity and Neuropathic Pain. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/151055

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Strickland, Eric. “MicroRNA Dysregulation Following Spinal Cord Contusion: Implications for Neural Plasticity and Neuropathic Pain.” 2013. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/151055.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Strickland, Eric. “MicroRNA Dysregulation Following Spinal Cord Contusion: Implications for Neural Plasticity and Neuropathic Pain.” 2013. Web. 20 Apr 2019.

Vancouver:

Strickland E. MicroRNA Dysregulation Following Spinal Cord Contusion: Implications for Neural Plasticity and Neuropathic Pain. [Internet] [Thesis]. Texas A&M University; 2013. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/151055.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Strickland E. MicroRNA Dysregulation Following Spinal Cord Contusion: Implications for Neural Plasticity and Neuropathic Pain. [Thesis]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/151055

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

9. Sawant, Onkar Balkrishna. Effects of Maternal L-glutamine Supplementation on Fetus to Mitigate Teratogenic Effects of Alcohol.

Degree: 2013, Texas A&M University

 Women who drink alcohol during pregnancy are at high risk of giving birth to children with physical, behavioral or cognitive developmental problems called Fetal Alcohol… (more)

Subjects/Keywords: FASD; Teratology; Glutamine; Alcohol; Blood-flow

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APA (6th Edition):

Sawant, O. B. (2013). Effects of Maternal L-glutamine Supplementation on Fetus to Mitigate Teratogenic Effects of Alcohol. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/151882

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Sawant, Onkar Balkrishna. “Effects of Maternal L-glutamine Supplementation on Fetus to Mitigate Teratogenic Effects of Alcohol.” 2013. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/151882.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Sawant, Onkar Balkrishna. “Effects of Maternal L-glutamine Supplementation on Fetus to Mitigate Teratogenic Effects of Alcohol.” 2013. Web. 20 Apr 2019.

Vancouver:

Sawant OB. Effects of Maternal L-glutamine Supplementation on Fetus to Mitigate Teratogenic Effects of Alcohol. [Internet] [Thesis]. Texas A&M University; 2013. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/151882.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sawant OB. Effects of Maternal L-glutamine Supplementation on Fetus to Mitigate Teratogenic Effects of Alcohol. [Thesis]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/151882

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

10. Tsai, Pai Chi. miR-153 and miR-335, Ethanol Sensitive MicroRNAs, Control NSC/NPC Maturation during Fetal Brain Development.

Degree: 2014, Texas A&M University

 Maternal alcohol consumption during pregnancy, especially during the first and second trimester, can cause the wide range of severe birth defects classified as fetal alcohol… (more)

Subjects/Keywords: miR-153; miR-335

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APA (6th Edition):

Tsai, P. C. (2014). miR-153 and miR-335, Ethanol Sensitive MicroRNAs, Control NSC/NPC Maturation during Fetal Brain Development. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/152634

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tsai, Pai Chi. “miR-153 and miR-335, Ethanol Sensitive MicroRNAs, Control NSC/NPC Maturation during Fetal Brain Development.” 2014. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/152634.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tsai, Pai Chi. “miR-153 and miR-335, Ethanol Sensitive MicroRNAs, Control NSC/NPC Maturation during Fetal Brain Development.” 2014. Web. 20 Apr 2019.

Vancouver:

Tsai PC. miR-153 and miR-335, Ethanol Sensitive MicroRNAs, Control NSC/NPC Maturation during Fetal Brain Development. [Internet] [Thesis]. Texas A&M University; 2014. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/152634.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tsai PC. miR-153 and miR-335, Ethanol Sensitive MicroRNAs, Control NSC/NPC Maturation during Fetal Brain Development. [Thesis]. Texas A&M University; 2014. Available from: http://hdl.handle.net/1969.1/152634

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

11. Zhi, Hui. Deciphering Novel Functions of Borrelia Burgdorferi Surface Proteins and Their Potential Roles in Borrelial Pathogenesis.

Degree: PhD, Medical Sciences, 2016, Texas A&M University

 Lyme disease is a significant and re-emerging disease that impacts the public health, particularly in endemic regions. The causative agent of Lyme disease, Borrelia burgdorferi,… (more)

Subjects/Keywords: borrelia burgdorferi; collagen; lyme disease; complement system; infection; immune response; classical pathway; C1; BBK32; BBA33

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APA (6th Edition):

Zhi, H. (2016). Deciphering Novel Functions of Borrelia Burgdorferi Surface Proteins and Their Potential Roles in Borrelial Pathogenesis. (Doctoral Dissertation). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/174250

Chicago Manual of Style (16th Edition):

Zhi, Hui. “Deciphering Novel Functions of Borrelia Burgdorferi Surface Proteins and Their Potential Roles in Borrelial Pathogenesis.” 2016. Doctoral Dissertation, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/174250.

MLA Handbook (7th Edition):

Zhi, Hui. “Deciphering Novel Functions of Borrelia Burgdorferi Surface Proteins and Their Potential Roles in Borrelial Pathogenesis.” 2016. Web. 20 Apr 2019.

Vancouver:

Zhi H. Deciphering Novel Functions of Borrelia Burgdorferi Surface Proteins and Their Potential Roles in Borrelial Pathogenesis. [Internet] [Doctoral dissertation]. Texas A&M University; 2016. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/174250.

Council of Science Editors:

Zhi H. Deciphering Novel Functions of Borrelia Burgdorferi Surface Proteins and Their Potential Roles in Borrelial Pathogenesis. [Doctoral Dissertation]. Texas A&M University; 2016. Available from: http://hdl.handle.net/1969.1/174250


Texas A&M University

12. Aceves, Miriam. Morphine Undermines Recovery Following Spinal Cord Injury: Evaluation of Opioid Receptors and Cellular Mechanisms.

Degree: 2017, Texas A&M University

 Opioids are amongst the most effective and widely prescribed medications for the treatment of pain following spinal cord injury (SCI). Spinally-injured patients receive opioids shortly… (more)

Subjects/Keywords: spinal cord injury; morphine; opioid receptors; contusion; pain

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APA (6th Edition):

Aceves, M. (2017). Morphine Undermines Recovery Following Spinal Cord Injury: Evaluation of Opioid Receptors and Cellular Mechanisms. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/161487

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Aceves, Miriam. “Morphine Undermines Recovery Following Spinal Cord Injury: Evaluation of Opioid Receptors and Cellular Mechanisms.” 2017. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/161487.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Aceves, Miriam. “Morphine Undermines Recovery Following Spinal Cord Injury: Evaluation of Opioid Receptors and Cellular Mechanisms.” 2017. Web. 20 Apr 2019.

Vancouver:

Aceves M. Morphine Undermines Recovery Following Spinal Cord Injury: Evaluation of Opioid Receptors and Cellular Mechanisms. [Internet] [Thesis]. Texas A&M University; 2017. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/161487.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Aceves M. Morphine Undermines Recovery Following Spinal Cord Injury: Evaluation of Opioid Receptors and Cellular Mechanisms. [Thesis]. Texas A&M University; 2017. Available from: http://hdl.handle.net/1969.1/161487

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

13. Ko, Eunhwa. Exploring Key Orientations of Small Molecules to Disrupt Protein-protein Interactions.

Degree: 2012, Texas A&M University

 Protein-protein interactions (PPIs) are attractive targets because of their therapeutic potential. One approach to design small molecules that can disrupt the PPIs is to use… (more)

Subjects/Keywords: Explores Key Orientation; Universal Peptidomimetics; minimalist mimics; protein-protein interactions; TrkC; targeted drug delivery; triazole-based beta-turn mimics; bivalent mimics

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APA (6th Edition):

Ko, E. (2012). Exploring Key Orientations of Small Molecules to Disrupt Protein-protein Interactions. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2012-05-10961

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ko, Eunhwa. “Exploring Key Orientations of Small Molecules to Disrupt Protein-protein Interactions.” 2012. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/ETD-TAMU-2012-05-10961.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ko, Eunhwa. “Exploring Key Orientations of Small Molecules to Disrupt Protein-protein Interactions.” 2012. Web. 20 Apr 2019.

Vancouver:

Ko E. Exploring Key Orientations of Small Molecules to Disrupt Protein-protein Interactions. [Internet] [Thesis]. Texas A&M University; 2012. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2012-05-10961.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ko E. Exploring Key Orientations of Small Molecules to Disrupt Protein-protein Interactions. [Thesis]. Texas A&M University; 2012. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2012-05-10961

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

14. Shah, Manasvi Shailesh 1984-. Modulation of Intestinal Micrornas by a Chemoprotective Diet.

Degree: 2012, Texas A&M University

 We have hypothesized that dietary modulation of intestinal miRNA expression may contribute to the chemoprotective effects of nutritional bioactives (fish oil and pectin). Using a… (more)

Subjects/Keywords: chemoprevention; omega-3 PUFA; microRNAs; colon cancer

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APA (6th Edition):

Shah, M. S. 1. (2012). Modulation of Intestinal Micrornas by a Chemoprotective Diet. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/148169

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shah, Manasvi Shailesh 1984-. “Modulation of Intestinal Micrornas by a Chemoprotective Diet.” 2012. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/148169.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shah, Manasvi Shailesh 1984-. “Modulation of Intestinal Micrornas by a Chemoprotective Diet.” 2012. Web. 20 Apr 2019.

Vancouver:

Shah MS1. Modulation of Intestinal Micrornas by a Chemoprotective Diet. [Internet] [Thesis]. Texas A&M University; 2012. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/148169.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shah MS1. Modulation of Intestinal Micrornas by a Chemoprotective Diet. [Thesis]. Texas A&M University; 2012. Available from: http://hdl.handle.net/1969.1/148169

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

15. Shende, Vikram Ravindra 1982-. MicroRNAs Function as Cis- and Trans- Acting Modulators of Clock Gene Expression in SCN and Peripheral Circadian Oscillators.

Degree: 2012, Texas A&M University

 The circadian system in mammals is arranged as a hierarchical network of oscillators, with the master pacemaker of circadian rhythms located in the suprachiasmatic nuclei… (more)

Subjects/Keywords: miRNA regulation of intercellular communication; miRNA; Circadian temporal synchronization; Circadian

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APA (6th Edition):

Shende, V. R. 1. (2012). MicroRNAs Function as Cis- and Trans- Acting Modulators of Clock Gene Expression in SCN and Peripheral Circadian Oscillators. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/148183

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shende, Vikram Ravindra 1982-. “MicroRNAs Function as Cis- and Trans- Acting Modulators of Clock Gene Expression in SCN and Peripheral Circadian Oscillators.” 2012. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/148183.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shende, Vikram Ravindra 1982-. “MicroRNAs Function as Cis- and Trans- Acting Modulators of Clock Gene Expression in SCN and Peripheral Circadian Oscillators.” 2012. Web. 20 Apr 2019.

Vancouver:

Shende VR1. MicroRNAs Function as Cis- and Trans- Acting Modulators of Clock Gene Expression in SCN and Peripheral Circadian Oscillators. [Internet] [Thesis]. Texas A&M University; 2012. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/148183.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shende VR1. MicroRNAs Function as Cis- and Trans- Acting Modulators of Clock Gene Expression in SCN and Peripheral Circadian Oscillators. [Thesis]. Texas A&M University; 2012. Available from: http://hdl.handle.net/1969.1/148183

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

16. Woller, Sarah Ann. Characterization of Morphine Self-Administration Following Spinal Cord Injury.

Degree: 2013, Texas A&M University

 Approximately two-thirds of patients will experience pain following spinal cord injury (SCI). This pain can arise as an immediate consequence of SCI, or can develop… (more)

Subjects/Keywords: spinal cord injury; morphine; self-administration

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APA (6th Edition):

Woller, S. A. (2013). Characterization of Morphine Self-Administration Following Spinal Cord Injury. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/151129

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Woller, Sarah Ann. “Characterization of Morphine Self-Administration Following Spinal Cord Injury.” 2013. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/151129.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Woller, Sarah Ann. “Characterization of Morphine Self-Administration Following Spinal Cord Injury.” 2013. Web. 20 Apr 2019.

Vancouver:

Woller SA. Characterization of Morphine Self-Administration Following Spinal Cord Injury. [Internet] [Thesis]. Texas A&M University; 2013. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/151129.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Woller SA. Characterization of Morphine Self-Administration Following Spinal Cord Injury. [Thesis]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/151129

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

17. Lee, Kuan Hsien. Timing in the Absence of Supraspinal Input: Effects of Temporally Regular Stimulation on Spinal Plasticity.

Degree: 2013, Texas A&M University

 Prior work has shown that spinal neurons are capable of discriminating between temporally regular and temporally irregular stimulation. These effects have been observed using an… (more)

Subjects/Keywords: Spinal plasticity; Timing

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APA (6th Edition):

Lee, K. H. (2013). Timing in the Absence of Supraspinal Input: Effects of Temporally Regular Stimulation on Spinal Plasticity. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/151216

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, Kuan Hsien. “Timing in the Absence of Supraspinal Input: Effects of Temporally Regular Stimulation on Spinal Plasticity.” 2013. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/151216.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, Kuan Hsien. “Timing in the Absence of Supraspinal Input: Effects of Temporally Regular Stimulation on Spinal Plasticity.” 2013. Web. 20 Apr 2019.

Vancouver:

Lee KH. Timing in the Absence of Supraspinal Input: Effects of Temporally Regular Stimulation on Spinal Plasticity. [Internet] [Thesis]. Texas A&M University; 2013. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/151216.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee KH. Timing in the Absence of Supraspinal Input: Effects of Temporally Regular Stimulation on Spinal Plasticity. [Thesis]. Texas A&M University; 2013. Available from: http://hdl.handle.net/1969.1/151216

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

18. Maldonado, Sioui. Psychological Well-Being and Spinal Cord Injury Recovery: A Two-Way Street?.

Degree: 2014, Texas A&M University

 Spinal cord injury (SCI) leads to increased anxiety and depression in as many as 60% of patients. Yet despite extensive clinical research focused on understanding… (more)

Subjects/Keywords: spinal cord injury; inflammation; HPA axis; stress; depression; anxiety; cytokines; social enrichment; caregiving

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APA (6th Edition):

Maldonado, S. (2014). Psychological Well-Being and Spinal Cord Injury Recovery: A Two-Way Street?. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/153842

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Maldonado, Sioui. “Psychological Well-Being and Spinal Cord Injury Recovery: A Two-Way Street?.” 2014. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/153842.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Maldonado, Sioui. “Psychological Well-Being and Spinal Cord Injury Recovery: A Two-Way Street?.” 2014. Web. 20 Apr 2019.

Vancouver:

Maldonado S. Psychological Well-Being and Spinal Cord Injury Recovery: A Two-Way Street?. [Internet] [Thesis]. Texas A&M University; 2014. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/153842.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Maldonado S. Psychological Well-Being and Spinal Cord Injury Recovery: A Two-Way Street?. [Thesis]. Texas A&M University; 2014. Available from: http://hdl.handle.net/1969.1/153842

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

19. Hillman, Paul Randolph. Analysis of Genomic Imprinting of UBE3A in Neurons.

Degree: 2015, Texas A&M University

 Angelman syndrome (AS), chromosome 15q11-q13 duplication syndrome (Dup15q), and Prader-Willi syndrome (PWS) are neurodevelopmental disorders associated with dysregulated expression of imprinted genes located within the… (more)

Subjects/Keywords: Angelman syndrome; Imprinting; Genomics; Next generation sequencing; RNA-seq; Neuordevelopment

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APA (6th Edition):

Hillman, P. R. (2015). Analysis of Genomic Imprinting of UBE3A in Neurons. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/155041

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hillman, Paul Randolph. “Analysis of Genomic Imprinting of UBE3A in Neurons.” 2015. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/155041.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hillman, Paul Randolph. “Analysis of Genomic Imprinting of UBE3A in Neurons.” 2015. Web. 20 Apr 2019.

Vancouver:

Hillman PR. Analysis of Genomic Imprinting of UBE3A in Neurons. [Internet] [Thesis]. Texas A&M University; 2015. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/155041.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hillman PR. Analysis of Genomic Imprinting of UBE3A in Neurons. [Thesis]. Texas A&M University; 2015. Available from: http://hdl.handle.net/1969.1/155041

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

20. Hsiao, Hao-Ching. Protein Interactions in Regulation and Assembly.

Degree: 2015, Texas A&M University

 The objectives of this work include: validation of yeast-based assays, investigation of protein-protein interactions in the regulatory role of an intrinsically disordered protein, Ultrabithorax (Ubx),… (more)

Subjects/Keywords: Protein interactions; Ubx

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APA (6th Edition):

Hsiao, H. (2015). Protein Interactions in Regulation and Assembly. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/155045

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hsiao, Hao-Ching. “Protein Interactions in Regulation and Assembly.” 2015. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/155045.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hsiao, Hao-Ching. “Protein Interactions in Regulation and Assembly.” 2015. Web. 20 Apr 2019.

Vancouver:

Hsiao H. Protein Interactions in Regulation and Assembly. [Internet] [Thesis]. Texas A&M University; 2015. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/155045.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hsiao H. Protein Interactions in Regulation and Assembly. [Thesis]. Texas A&M University; 2015. Available from: http://hdl.handle.net/1969.1/155045

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

21. Veazey, Kylee Jordan. An Epigenetic Basis to the Etiology of Fetal Alcohol Spectrum Disorders.

Degree: 2015, Texas A&M University

 Observations from a number of independent laboratories indicate that ethanol has the capacity to act as a powerful epigenetic disruptor and potentially derail the process… (more)

Subjects/Keywords: Epigenetics; FASD; Histone Modifications

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APA (6th Edition):

Veazey, K. J. (2015). An Epigenetic Basis to the Etiology of Fetal Alcohol Spectrum Disorders. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/156462

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Veazey, Kylee Jordan. “An Epigenetic Basis to the Etiology of Fetal Alcohol Spectrum Disorders.” 2015. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/156462.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Veazey, Kylee Jordan. “An Epigenetic Basis to the Etiology of Fetal Alcohol Spectrum Disorders.” 2015. Web. 20 Apr 2019.

Vancouver:

Veazey KJ. An Epigenetic Basis to the Etiology of Fetal Alcohol Spectrum Disorders. [Internet] [Thesis]. Texas A&M University; 2015. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/156462.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Veazey KJ. An Epigenetic Basis to the Etiology of Fetal Alcohol Spectrum Disorders. [Thesis]. Texas A&M University; 2015. Available from: http://hdl.handle.net/1969.1/156462

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Texas A&M University

22. Menger, Gus John, III. Comparison of circadian gene expression among different oscillator models: identification of critical output signals of the SCN pacemaker.

Degree: 2009, Texas A&M University

 Diverse forms of life have evolved 24-hour or circadian timekeeping systems serving to coordinate internal biological events with the daily solar cycle. The generation of… (more)

Subjects/Keywords: Suprachiasmatic nucleus; circadian oscillator

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APA (6th Edition):

Menger, Gus John, I. (2009). Comparison of circadian gene expression among different oscillator models: identification of critical output signals of the SCN pacemaker. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-1244

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Menger, Gus John, III. “Comparison of circadian gene expression among different oscillator models: identification of critical output signals of the SCN pacemaker.” 2009. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/ETD-TAMU-1244.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Menger, Gus John, III. “Comparison of circadian gene expression among different oscillator models: identification of critical output signals of the SCN pacemaker.” 2009. Web. 20 Apr 2019.

Vancouver:

Menger, Gus John I. Comparison of circadian gene expression among different oscillator models: identification of critical output signals of the SCN pacemaker. [Internet] [Thesis]. Texas A&M University; 2009. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-1244.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Menger, Gus John I. Comparison of circadian gene expression among different oscillator models: identification of critical output signals of the SCN pacemaker. [Thesis]. Texas A&M University; 2009. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-1244

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

23. Puga, Denise Alejandra. The opponent consequences of intermittent and continuous stimulation within the rat spinal cord.

Degree: 2009, Texas A&M University

 A substantial body of work exists to suggest that brain and spinal mechanisms react differently to nociceptive information. The current experiments were design to identify… (more)

Subjects/Keywords: spinal cord; thermal reactivity

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APA (6th Edition):

Puga, D. A. (2009). The opponent consequences of intermittent and continuous stimulation within the rat spinal cord. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-1605

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Puga, Denise Alejandra. “The opponent consequences of intermittent and continuous stimulation within the rat spinal cord.” 2009. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/ETD-TAMU-1605.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Puga, Denise Alejandra. “The opponent consequences of intermittent and continuous stimulation within the rat spinal cord.” 2009. Web. 20 Apr 2019.

Vancouver:

Puga DA. The opponent consequences of intermittent and continuous stimulation within the rat spinal cord. [Internet] [Thesis]. Texas A&M University; 2009. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-1605.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Puga DA. The opponent consequences of intermittent and continuous stimulation within the rat spinal cord. [Thesis]. Texas A&M University; 2009. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-1605

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

24. Hoy, Kevin. Contribution of the Peripheral Nervous System to Instrumental Learning and Performance.

Degree: 2012, Texas A&M University

 Previous research has demonstrated that the spinal cord is capable of a simple form of instrumental learning. In this instrumental learning paradigm, rats typically receive… (more)

Subjects/Keywords: Spinal Cord; Peripheral Nervous System; Neuromuscularjunction

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APA (6th Edition):

Hoy, K. (2012). Contribution of the Peripheral Nervous System to Instrumental Learning and Performance. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2011-08-9973

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hoy, Kevin. “Contribution of the Peripheral Nervous System to Instrumental Learning and Performance.” 2012. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/ETD-TAMU-2011-08-9973.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hoy, Kevin. “Contribution of the Peripheral Nervous System to Instrumental Learning and Performance.” 2012. Web. 20 Apr 2019.

Vancouver:

Hoy K. Contribution of the Peripheral Nervous System to Instrumental Learning and Performance. [Internet] [Thesis]. Texas A&M University; 2012. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2011-08-9973.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hoy K. Contribution of the Peripheral Nervous System to Instrumental Learning and Performance. [Thesis]. Texas A&M University; 2012. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2011-08-9973

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

25. Woller, Sarah Ann. Analgesia or Addiction: Implications for Morphine Use After Spinal Cord Injury.

Degree: 2012, Texas A&M University

 Up to 65% of individuals with a spinal cord injury (SCI) experience neuropathic pain, and cite this as one of the most significant consequences of… (more)

Subjects/Keywords: SCI; Morphine; Addiction; Analgesia

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APA (6th Edition):

Woller, S. A. (2012). Analgesia or Addiction: Implications for Morphine Use After Spinal Cord Injury. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/ETD-TAMU-2010-05-7807

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Woller, Sarah Ann. “Analgesia or Addiction: Implications for Morphine Use After Spinal Cord Injury.” 2012. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/ETD-TAMU-2010-05-7807.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Woller, Sarah Ann. “Analgesia or Addiction: Implications for Morphine Use After Spinal Cord Injury.” 2012. Web. 20 Apr 2019.

Vancouver:

Woller SA. Analgesia or Addiction: Implications for Morphine Use After Spinal Cord Injury. [Internet] [Thesis]. Texas A&M University; 2012. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2010-05-7807.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Woller SA. Analgesia or Addiction: Implications for Morphine Use After Spinal Cord Injury. [Thesis]. Texas A&M University; 2012. Available from: http://hdl.handle.net/1969.1/ETD-TAMU-2010-05-7807

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

26. Bopp, Anne Caroline. Shock induces a deficit in the recovery of function after a contusion injury: identifying the relative contributions of the brain and spinal cord.

Degree: 2006, Texas A&M University

 Prior studies have shown that exposure to uncontrollable stimulation can have a variety of adverse consequences on plasticity. For example, as little as 30 min… (more)

Subjects/Keywords: Shock; Recovery

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APA (6th Edition):

Bopp, A. C. (2006). Shock induces a deficit in the recovery of function after a contusion injury: identifying the relative contributions of the brain and spinal cord. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/4169

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bopp, Anne Caroline. “Shock induces a deficit in the recovery of function after a contusion injury: identifying the relative contributions of the brain and spinal cord.” 2006. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/4169.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bopp, Anne Caroline. “Shock induces a deficit in the recovery of function after a contusion injury: identifying the relative contributions of the brain and spinal cord.” 2006. Web. 20 Apr 2019.

Vancouver:

Bopp AC. Shock induces a deficit in the recovery of function after a contusion injury: identifying the relative contributions of the brain and spinal cord. [Internet] [Thesis]. Texas A&M University; 2006. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/4169.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bopp AC. Shock induces a deficit in the recovery of function after a contusion injury: identifying the relative contributions of the brain and spinal cord. [Thesis]. Texas A&M University; 2006. Available from: http://hdl.handle.net/1969.1/4169

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

27. Paez, Ximena. Motor Neuron-Specific Restoration of SMN in Two SMA Mouse Models: Insights into the Role of Motor Neurons in Spinal Muscular Atrophy.

Degree: 2014, Texas A&M University

 Spinal muscular atrophy (SMA) results from ?-motor neuron loss in the spinal cord due to low levels of the survival of motor neuron (SMN) protein,… (more)

Subjects/Keywords: SMA; SMA; mouse models; Neuromuscular Junction

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APA (6th Edition):

Paez, X. (2014). Motor Neuron-Specific Restoration of SMN in Two SMA Mouse Models: Insights into the Role of Motor Neurons in Spinal Muscular Atrophy. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/153270

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Paez, Ximena. “Motor Neuron-Specific Restoration of SMN in Two SMA Mouse Models: Insights into the Role of Motor Neurons in Spinal Muscular Atrophy.” 2014. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/153270.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Paez, Ximena. “Motor Neuron-Specific Restoration of SMN in Two SMA Mouse Models: Insights into the Role of Motor Neurons in Spinal Muscular Atrophy.” 2014. Web. 20 Apr 2019.

Vancouver:

Paez X. Motor Neuron-Specific Restoration of SMN in Two SMA Mouse Models: Insights into the Role of Motor Neurons in Spinal Muscular Atrophy. [Internet] [Thesis]. Texas A&M University; 2014. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/153270.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Paez X. Motor Neuron-Specific Restoration of SMN in Two SMA Mouse Models: Insights into the Role of Motor Neurons in Spinal Muscular Atrophy. [Thesis]. Texas A&M University; 2014. Available from: http://hdl.handle.net/1969.1/153270

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

28. Dave, Jui Mayank. Identification of Scaffold Proteins RACK1 and Hic-5 as Regulators of Endothelial Sprouting Resoponses.

Degree: 2014, Texas A&M University

 Angiogenesis is defined as growth of new blood vessels from pre-existing ones and occurs during normal physiological development as well as pathological conditions like cancer.… (more)

Subjects/Keywords: Endothelial; sprouting; angiogenesis; collagen; sphingosine 1-phosphate; three-dimensional matrix; RACK1; Hic-5; FAK; vimentin; MT1-MMP

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Dave, J. M. (2014). Identification of Scaffold Proteins RACK1 and Hic-5 as Regulators of Endothelial Sprouting Resoponses. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/154023

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Dave, Jui Mayank. “Identification of Scaffold Proteins RACK1 and Hic-5 as Regulators of Endothelial Sprouting Resoponses.” 2014. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/154023.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Dave, Jui Mayank. “Identification of Scaffold Proteins RACK1 and Hic-5 as Regulators of Endothelial Sprouting Resoponses.” 2014. Web. 20 Apr 2019.

Vancouver:

Dave JM. Identification of Scaffold Proteins RACK1 and Hic-5 as Regulators of Endothelial Sprouting Resoponses. [Internet] [Thesis]. Texas A&M University; 2014. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/154023.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Dave JM. Identification of Scaffold Proteins RACK1 and Hic-5 as Regulators of Endothelial Sprouting Resoponses. [Thesis]. Texas A&M University; 2014. Available from: http://hdl.handle.net/1969.1/154023

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

29. Huang, Yung-Jen. Spinal Cord Injury (SCI) Switches How GABA Affects Nociceptive Plasticity.

Degree: 2015, Texas A&M University

 Research has shown that spinal cord injury (SCI) can induce neural hyperexcitability within the spinal cord that facilitates nociceptive reflexes. Nociceptive inputs have been shown… (more)

Subjects/Keywords: GABA; SCI; Nociceptive plasticity

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Huang, Y. (2015). Spinal Cord Injury (SCI) Switches How GABA Affects Nociceptive Plasticity. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/155236

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Huang, Yung-Jen. “Spinal Cord Injury (SCI) Switches How GABA Affects Nociceptive Plasticity.” 2015. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/155236.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Huang, Yung-Jen. “Spinal Cord Injury (SCI) Switches How GABA Affects Nociceptive Plasticity.” 2015. Web. 20 Apr 2019.

Vancouver:

Huang Y. Spinal Cord Injury (SCI) Switches How GABA Affects Nociceptive Plasticity. [Internet] [Thesis]. Texas A&M University; 2015. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/155236.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huang Y. Spinal Cord Injury (SCI) Switches How GABA Affects Nociceptive Plasticity. [Thesis]. Texas A&M University; 2015. Available from: http://hdl.handle.net/1969.1/155236

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

30. Raymond, Elizabeth S. Effects of the Developmental Teratogens Ethanol and Nicotine on Long Noncoding RNA Expression in Mouse Fetal Neural Stem Cells of the Cortex.

Degree: 2016, Texas A&M University

 Research on long noncoding RNAs (lncRNAs) has increased significantly over the past few decades. However, investigation of lncRNA continues to lag behind that of other… (more)

Subjects/Keywords: long noncoding RNA; mouse stem cells; Ethanol; Nicotine

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Raymond, E. S. (2016). Effects of the Developmental Teratogens Ethanol and Nicotine on Long Noncoding RNA Expression in Mouse Fetal Neural Stem Cells of the Cortex. (Thesis). Texas A&M University. Retrieved from http://hdl.handle.net/1969.1/158037

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Raymond, Elizabeth S. “Effects of the Developmental Teratogens Ethanol and Nicotine on Long Noncoding RNA Expression in Mouse Fetal Neural Stem Cells of the Cortex.” 2016. Thesis, Texas A&M University. Accessed April 20, 2019. http://hdl.handle.net/1969.1/158037.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Raymond, Elizabeth S. “Effects of the Developmental Teratogens Ethanol and Nicotine on Long Noncoding RNA Expression in Mouse Fetal Neural Stem Cells of the Cortex.” 2016. Web. 20 Apr 2019.

Vancouver:

Raymond ES. Effects of the Developmental Teratogens Ethanol and Nicotine on Long Noncoding RNA Expression in Mouse Fetal Neural Stem Cells of the Cortex. [Internet] [Thesis]. Texas A&M University; 2016. [cited 2019 Apr 20]. Available from: http://hdl.handle.net/1969.1/158037.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Raymond ES. Effects of the Developmental Teratogens Ethanol and Nicotine on Long Noncoding RNA Expression in Mouse Fetal Neural Stem Cells of the Cortex. [Thesis]. Texas A&M University; 2016. Available from: http://hdl.handle.net/1969.1/158037

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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