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You searched for +publisher:"Temple University" +contributor:("Wolfson, Marla R."). Showing records 1 – 7 of 7 total matches.

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Temple University

1. Sommerville, Laura Jean. The Role of Allograft Inflammatory Factor-1 in Vascular Smooth Muscle Cell Activation and Development of Vascular Proliferative Disease.

Degree: PhD, 2010, Temple University

Molecular and Cellular Physiology

The underlying cause of all vascular proliferative diseases is injury-induced activation of vascular endothelium and vascular smooth muscle cells (VSMC). Activated… (more)

Subjects/Keywords: Biology, Physiology; Allograft Inflammatory Factor-1; Atherosclerosis; Smooth Muscle Cell Activation; Vascular Proliferative Disease

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APA (6th Edition):

Sommerville, L. J. (2010). The Role of Allograft Inflammatory Factor-1 in Vascular Smooth Muscle Cell Activation and Development of Vascular Proliferative Disease. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,76756

Chicago Manual of Style (16th Edition):

Sommerville, Laura Jean. “The Role of Allograft Inflammatory Factor-1 in Vascular Smooth Muscle Cell Activation and Development of Vascular Proliferative Disease.” 2010. Doctoral Dissertation, Temple University. Accessed September 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,76756.

MLA Handbook (7th Edition):

Sommerville, Laura Jean. “The Role of Allograft Inflammatory Factor-1 in Vascular Smooth Muscle Cell Activation and Development of Vascular Proliferative Disease.” 2010. Web. 21 Sep 2019.

Vancouver:

Sommerville LJ. The Role of Allograft Inflammatory Factor-1 in Vascular Smooth Muscle Cell Activation and Development of Vascular Proliferative Disease. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2019 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,76756.

Council of Science Editors:

Sommerville LJ. The Role of Allograft Inflammatory Factor-1 in Vascular Smooth Muscle Cell Activation and Development of Vascular Proliferative Disease. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,76756


Temple University

2. Cuneo, Anthony. THERAPEUTIC MECHANISMS OF INTERLEUKIN-19 FOR VASCULAR PROLIFERATIVE DISEASES.

Degree: PhD, 2012, Temple University

Molecular and Cellular Physiology

Cardiovascular disease is the leading cause of mortality in the western world. The pro-inflammatory and pro-proliferative etiology of vascular proliferative diseases… (more)

Subjects/Keywords: Biology, Molecular; Biology, General; Biology, Cell; Atherosclerosis; Human antigen R (HuR); Interleukin-19 (IL-19); oxidized LDL (ox-LDL); Vascular Proliferative Diseases; Vascular Smooth Muscle Cells (VSMC)

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APA (6th Edition):

Cuneo, A. (2012). THERAPEUTIC MECHANISMS OF INTERLEUKIN-19 FOR VASCULAR PROLIFERATIVE DISEASES. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,78032

Chicago Manual of Style (16th Edition):

Cuneo, Anthony. “THERAPEUTIC MECHANISMS OF INTERLEUKIN-19 FOR VASCULAR PROLIFERATIVE DISEASES.” 2012. Doctoral Dissertation, Temple University. Accessed September 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,78032.

MLA Handbook (7th Edition):

Cuneo, Anthony. “THERAPEUTIC MECHANISMS OF INTERLEUKIN-19 FOR VASCULAR PROLIFERATIVE DISEASES.” 2012. Web. 21 Sep 2019.

Vancouver:

Cuneo A. THERAPEUTIC MECHANISMS OF INTERLEUKIN-19 FOR VASCULAR PROLIFERATIVE DISEASES. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2019 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,78032.

Council of Science Editors:

Cuneo A. THERAPEUTIC MECHANISMS OF INTERLEUKIN-19 FOR VASCULAR PROLIFERATIVE DISEASES. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,78032


Temple University

3. Hubert, Terrence L. Effect of rhCC10 on the Pro/Anti-Inflammatory Profile of the Immature Lung.

Degree: PhD, 2014, Temple University

Physiology

There is a gap in the treatment of preterm infants with respiratory distress syndrome. Despite addressing surfactant insufficiency and mechanical instability, currently available exogenous… (more)

Subjects/Keywords: Physiology;

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APA (6th Edition):

Hubert, T. L. (2014). Effect of rhCC10 on the Pro/Anti-Inflammatory Profile of the Immature Lung. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,240379

Chicago Manual of Style (16th Edition):

Hubert, Terrence L. “Effect of rhCC10 on the Pro/Anti-Inflammatory Profile of the Immature Lung.” 2014. Doctoral Dissertation, Temple University. Accessed September 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,240379.

MLA Handbook (7th Edition):

Hubert, Terrence L. “Effect of rhCC10 on the Pro/Anti-Inflammatory Profile of the Immature Lung.” 2014. Web. 21 Sep 2019.

Vancouver:

Hubert TL. Effect of rhCC10 on the Pro/Anti-Inflammatory Profile of the Immature Lung. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,240379.

Council of Science Editors:

Hubert TL. Effect of rhCC10 on the Pro/Anti-Inflammatory Profile of the Immature Lung. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,240379


Temple University

4. Ronca, Rich Daniel. The Cannabinoid-2 Receptor Agonist O-1966 Reverses Postischemic Learning and Memory Deficits Through Anti-Inflammatory Processes.

Degree: PhD, 2013, Temple University

Pharmacology

Ischemic stroke is the third leading cause of death and the leading cause of morbidity in the United States. Cognitive deficits, specifically with respect… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Ronca, R. D. (2013). The Cannabinoid-2 Receptor Agonist O-1966 Reverses Postischemic Learning and Memory Deficits Through Anti-Inflammatory Processes. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,253154

Chicago Manual of Style (16th Edition):

Ronca, Rich Daniel. “The Cannabinoid-2 Receptor Agonist O-1966 Reverses Postischemic Learning and Memory Deficits Through Anti-Inflammatory Processes.” 2013. Doctoral Dissertation, Temple University. Accessed September 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,253154.

MLA Handbook (7th Edition):

Ronca, Rich Daniel. “The Cannabinoid-2 Receptor Agonist O-1966 Reverses Postischemic Learning and Memory Deficits Through Anti-Inflammatory Processes.” 2013. Web. 21 Sep 2019.

Vancouver:

Ronca RD. The Cannabinoid-2 Receptor Agonist O-1966 Reverses Postischemic Learning and Memory Deficits Through Anti-Inflammatory Processes. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2019 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,253154.

Council of Science Editors:

Ronca RD. The Cannabinoid-2 Receptor Agonist O-1966 Reverses Postischemic Learning and Memory Deficits Through Anti-Inflammatory Processes. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,253154


Temple University

5. Li, Hongbo. SELECTIVE CB2 RECEPTOR ACTIVATION AMELIORATES INFLAMMATION IN CENTRAL NERVOUS SYSTEM BY REDUCING TH17 CELL DIFFERENTIATION AND IMMUNE CELL ACCUMULATION.

Degree: PhD, 2014, Temple University

Physiology

Modulation of the endocannabinoid system by the administration of exogenous agonists and selective antagonists has been shown to have potential to attenuate the contribution… (more)

Subjects/Keywords: Immunology; Physiology;

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APA (6th Edition):

Li, H. (2014). SELECTIVE CB2 RECEPTOR ACTIVATION AMELIORATES INFLAMMATION IN CENTRAL NERVOUS SYSTEM BY REDUCING TH17 CELL DIFFERENTIATION AND IMMUNE CELL ACCUMULATION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,288653

Chicago Manual of Style (16th Edition):

Li, Hongbo. “SELECTIVE CB2 RECEPTOR ACTIVATION AMELIORATES INFLAMMATION IN CENTRAL NERVOUS SYSTEM BY REDUCING TH17 CELL DIFFERENTIATION AND IMMUNE CELL ACCUMULATION.” 2014. Doctoral Dissertation, Temple University. Accessed September 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,288653.

MLA Handbook (7th Edition):

Li, Hongbo. “SELECTIVE CB2 RECEPTOR ACTIVATION AMELIORATES INFLAMMATION IN CENTRAL NERVOUS SYSTEM BY REDUCING TH17 CELL DIFFERENTIATION AND IMMUNE CELL ACCUMULATION.” 2014. Web. 21 Sep 2019.

Vancouver:

Li H. SELECTIVE CB2 RECEPTOR ACTIVATION AMELIORATES INFLAMMATION IN CENTRAL NERVOUS SYSTEM BY REDUCING TH17 CELL DIFFERENTIATION AND IMMUNE CELL ACCUMULATION. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,288653.

Council of Science Editors:

Li H. SELECTIVE CB2 RECEPTOR ACTIVATION AMELIORATES INFLAMMATION IN CENTRAL NERVOUS SYSTEM BY REDUCING TH17 CELL DIFFERENTIATION AND IMMUNE CELL ACCUMULATION. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,288653


Temple University

6. Stabler, Collin Turner. Optimizing Endothelial Repopulation of Decellularized Lung.

Degree: PhD, 2016, Temple University

Bioengineering

Decellularized lung tissue has been recognized as a potential platform to engineer whole lung organs suitable for transplantation or for modeling a variety of… (more)

Subjects/Keywords: Biomedical engineering;

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APA (6th Edition):

Stabler, C. T. (2016). Optimizing Endothelial Repopulation of Decellularized Lung. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,388434

Chicago Manual of Style (16th Edition):

Stabler, Collin Turner. “Optimizing Endothelial Repopulation of Decellularized Lung.” 2016. Doctoral Dissertation, Temple University. Accessed September 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,388434.

MLA Handbook (7th Edition):

Stabler, Collin Turner. “Optimizing Endothelial Repopulation of Decellularized Lung.” 2016. Web. 21 Sep 2019.

Vancouver:

Stabler CT. Optimizing Endothelial Repopulation of Decellularized Lung. [Internet] [Doctoral dissertation]. Temple University; 2016. [cited 2019 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,388434.

Council of Science Editors:

Stabler CT. Optimizing Endothelial Repopulation of Decellularized Lung. [Doctoral Dissertation]. Temple University; 2016. Available from: http://digital.library.temple.edu/u?/p245801coll10,388434


Temple University

7. Malone, Daniel Joseph. PERFLUOROCHEMICAL AUGMENTED INTRATRACHEAL DELIVERY OF ANTIOXIDANT ENZYMES AND GENES TO ATTENUATE OXIDATIVE STRESS-INDUCED LUNG AND RESPIRATORY MUSCLE ALTERATIONS.

Degree: PhD, 2009, Temple University

Physiology

Supraphysiologic concentrations of oxygen are used in the management of critically ill patients across the lifespan. However, hyperoxia (HO) results in alveolar- capillary membrane… (more)

Subjects/Keywords: Biology, Physiology; Health Sciences, Medicine and Surgery; diaphragm; hyperoxia; lung injury

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APA (6th Edition):

Malone, D. J. (2009). PERFLUOROCHEMICAL AUGMENTED INTRATRACHEAL DELIVERY OF ANTIOXIDANT ENZYMES AND GENES TO ATTENUATE OXIDATIVE STRESS-INDUCED LUNG AND RESPIRATORY MUSCLE ALTERATIONS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,24041

Chicago Manual of Style (16th Edition):

Malone, Daniel Joseph. “PERFLUOROCHEMICAL AUGMENTED INTRATRACHEAL DELIVERY OF ANTIOXIDANT ENZYMES AND GENES TO ATTENUATE OXIDATIVE STRESS-INDUCED LUNG AND RESPIRATORY MUSCLE ALTERATIONS.” 2009. Doctoral Dissertation, Temple University. Accessed September 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,24041.

MLA Handbook (7th Edition):

Malone, Daniel Joseph. “PERFLUOROCHEMICAL AUGMENTED INTRATRACHEAL DELIVERY OF ANTIOXIDANT ENZYMES AND GENES TO ATTENUATE OXIDATIVE STRESS-INDUCED LUNG AND RESPIRATORY MUSCLE ALTERATIONS.” 2009. Web. 21 Sep 2019.

Vancouver:

Malone DJ. PERFLUOROCHEMICAL AUGMENTED INTRATRACHEAL DELIVERY OF ANTIOXIDANT ENZYMES AND GENES TO ATTENUATE OXIDATIVE STRESS-INDUCED LUNG AND RESPIRATORY MUSCLE ALTERATIONS. [Internet] [Doctoral dissertation]. Temple University; 2009. [cited 2019 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,24041.

Council of Science Editors:

Malone DJ. PERFLUOROCHEMICAL AUGMENTED INTRATRACHEAL DELIVERY OF ANTIOXIDANT ENZYMES AND GENES TO ATTENUATE OXIDATIVE STRESS-INDUCED LUNG AND RESPIRATORY MUSCLE ALTERATIONS. [Doctoral Dissertation]. Temple University; 2009. Available from: http://digital.library.temple.edu/u?/p245801coll10,24041

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