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You searched for +publisher:"Temple University" +contributor:("Ward, Sara J.;"). Showing records 1 – 8 of 8 total matches.

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Temple University

1. Ronca, Rich Daniel. The Cannabinoid-2 Receptor Agonist O-1966 Reverses Postischemic Learning and Memory Deficits Through Anti-Inflammatory Processes.

Degree: PhD, 2013, Temple University

Pharmacology

Ischemic stroke is the third leading cause of death and the leading cause of morbidity in the United States. Cognitive deficits, specifically with respect… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Ronca, R. D. (2013). The Cannabinoid-2 Receptor Agonist O-1966 Reverses Postischemic Learning and Memory Deficits Through Anti-Inflammatory Processes. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,253154

Chicago Manual of Style (16th Edition):

Ronca, Rich Daniel. “The Cannabinoid-2 Receptor Agonist O-1966 Reverses Postischemic Learning and Memory Deficits Through Anti-Inflammatory Processes.” 2013. Doctoral Dissertation, Temple University. Accessed October 18, 2019. http://digital.library.temple.edu/u?/p245801coll10,253154.

MLA Handbook (7th Edition):

Ronca, Rich Daniel. “The Cannabinoid-2 Receptor Agonist O-1966 Reverses Postischemic Learning and Memory Deficits Through Anti-Inflammatory Processes.” 2013. Web. 18 Oct 2019.

Vancouver:

Ronca RD. The Cannabinoid-2 Receptor Agonist O-1966 Reverses Postischemic Learning and Memory Deficits Through Anti-Inflammatory Processes. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2019 Oct 18]. Available from: http://digital.library.temple.edu/u?/p245801coll10,253154.

Council of Science Editors:

Ronca RD. The Cannabinoid-2 Receptor Agonist O-1966 Reverses Postischemic Learning and Memory Deficits Through Anti-Inflammatory Processes. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,253154


Temple University

2. Reichenbach, Zachary Wilmer. Modulation of the Endogenous Cannabinoid System to Attenuate Inflammation in Central Nervous System Injury.

Degree: PhD, 2015, Temple University

Physiology

In non-pathological states the central nervous system maintains a degree of immunological privilege. When illness or injury occur, this privilege can be lost and… (more)

Subjects/Keywords: Neurosciences; Immunology; Pharmacology;

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APA (6th Edition):

Reichenbach, Z. W. (2015). Modulation of the Endogenous Cannabinoid System to Attenuate Inflammation in Central Nervous System Injury. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,253514

Chicago Manual of Style (16th Edition):

Reichenbach, Zachary Wilmer. “Modulation of the Endogenous Cannabinoid System to Attenuate Inflammation in Central Nervous System Injury.” 2015. Doctoral Dissertation, Temple University. Accessed October 18, 2019. http://digital.library.temple.edu/u?/p245801coll10,253514.

MLA Handbook (7th Edition):

Reichenbach, Zachary Wilmer. “Modulation of the Endogenous Cannabinoid System to Attenuate Inflammation in Central Nervous System Injury.” 2015. Web. 18 Oct 2019.

Vancouver:

Reichenbach ZW. Modulation of the Endogenous Cannabinoid System to Attenuate Inflammation in Central Nervous System Injury. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Oct 18]. Available from: http://digital.library.temple.edu/u?/p245801coll10,253514.

Council of Science Editors:

Reichenbach ZW. Modulation of the Endogenous Cannabinoid System to Attenuate Inflammation in Central Nervous System Injury. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,253514


Temple University

3. Enman, Nicole Marie. Dopamine reward dysfunction and cocaine-seeking in a rat model of PTSD.

Degree: PhD, 2014, Temple University

Pharmacology

Posttraumatic stress disorder (PTSD) co-occurs with substance use disorders at high rates, but the neurobiological basis of this relationship remains largely unknown. Identifying mechanisms… (more)

Subjects/Keywords: Neurosciences; Animal behavior; Pharmacology;

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APA (6th Edition):

Enman, N. M. (2014). Dopamine reward dysfunction and cocaine-seeking in a rat model of PTSD. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,259963

Chicago Manual of Style (16th Edition):

Enman, Nicole Marie. “Dopamine reward dysfunction and cocaine-seeking in a rat model of PTSD.” 2014. Doctoral Dissertation, Temple University. Accessed October 18, 2019. http://digital.library.temple.edu/u?/p245801coll10,259963.

MLA Handbook (7th Edition):

Enman, Nicole Marie. “Dopamine reward dysfunction and cocaine-seeking in a rat model of PTSD.” 2014. Web. 18 Oct 2019.

Vancouver:

Enman NM. Dopamine reward dysfunction and cocaine-seeking in a rat model of PTSD. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Oct 18]. Available from: http://digital.library.temple.edu/u?/p245801coll10,259963.

Council of Science Editors:

Enman NM. Dopamine reward dysfunction and cocaine-seeking in a rat model of PTSD. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,259963


Temple University

4. Li, Hongbo. SELECTIVE CB2 RECEPTOR ACTIVATION AMELIORATES INFLAMMATION IN CENTRAL NERVOUS SYSTEM BY REDUCING TH17 CELL DIFFERENTIATION AND IMMUNE CELL ACCUMULATION.

Degree: PhD, 2014, Temple University

Physiology

Modulation of the endocannabinoid system by the administration of exogenous agonists and selective antagonists has been shown to have potential to attenuate the contribution… (more)

Subjects/Keywords: Immunology; Physiology;

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APA (6th Edition):

Li, H. (2014). SELECTIVE CB2 RECEPTOR ACTIVATION AMELIORATES INFLAMMATION IN CENTRAL NERVOUS SYSTEM BY REDUCING TH17 CELL DIFFERENTIATION AND IMMUNE CELL ACCUMULATION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,288653

Chicago Manual of Style (16th Edition):

Li, Hongbo. “SELECTIVE CB2 RECEPTOR ACTIVATION AMELIORATES INFLAMMATION IN CENTRAL NERVOUS SYSTEM BY REDUCING TH17 CELL DIFFERENTIATION AND IMMUNE CELL ACCUMULATION.” 2014. Doctoral Dissertation, Temple University. Accessed October 18, 2019. http://digital.library.temple.edu/u?/p245801coll10,288653.

MLA Handbook (7th Edition):

Li, Hongbo. “SELECTIVE CB2 RECEPTOR ACTIVATION AMELIORATES INFLAMMATION IN CENTRAL NERVOUS SYSTEM BY REDUCING TH17 CELL DIFFERENTIATION AND IMMUNE CELL ACCUMULATION.” 2014. Web. 18 Oct 2019.

Vancouver:

Li H. SELECTIVE CB2 RECEPTOR ACTIVATION AMELIORATES INFLAMMATION IN CENTRAL NERVOUS SYSTEM BY REDUCING TH17 CELL DIFFERENTIATION AND IMMUNE CELL ACCUMULATION. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Oct 18]. Available from: http://digital.library.temple.edu/u?/p245801coll10,288653.

Council of Science Editors:

Li H. SELECTIVE CB2 RECEPTOR ACTIVATION AMELIORATES INFLAMMATION IN CENTRAL NERVOUS SYSTEM BY REDUCING TH17 CELL DIFFERENTIATION AND IMMUNE CELL ACCUMULATION. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,288653


Temple University

5. Gregg, Ryan Alexander. INVESTIGATIONS INTO THE STEREOCHEMICAL AND GLUTAMATERGIC MECHANISMS OF THE "BATH SALTS" SYNTHETIC CATHINONES MEPHEDRONE AND MDPV IN RATS.

Degree: PhD, 2015, Temple University

Pharmacology

Synthetic cathinones, commonly referred to as “bath salts”, are a subgroup of novel psychoactive substances that have seen a dramatic rise in abuse worldwide… (more)

Subjects/Keywords: Pharmacology; Neurosciences; Medicine;

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APA (6th Edition):

Gregg, R. A. (2015). INVESTIGATIONS INTO THE STEREOCHEMICAL AND GLUTAMATERGIC MECHANISMS OF THE "BATH SALTS" SYNTHETIC CATHINONES MEPHEDRONE AND MDPV IN RATS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,354278

Chicago Manual of Style (16th Edition):

Gregg, Ryan Alexander. “INVESTIGATIONS INTO THE STEREOCHEMICAL AND GLUTAMATERGIC MECHANISMS OF THE "BATH SALTS" SYNTHETIC CATHINONES MEPHEDRONE AND MDPV IN RATS.” 2015. Doctoral Dissertation, Temple University. Accessed October 18, 2019. http://digital.library.temple.edu/u?/p245801coll10,354278.

MLA Handbook (7th Edition):

Gregg, Ryan Alexander. “INVESTIGATIONS INTO THE STEREOCHEMICAL AND GLUTAMATERGIC MECHANISMS OF THE "BATH SALTS" SYNTHETIC CATHINONES MEPHEDRONE AND MDPV IN RATS.” 2015. Web. 18 Oct 2019.

Vancouver:

Gregg RA. INVESTIGATIONS INTO THE STEREOCHEMICAL AND GLUTAMATERGIC MECHANISMS OF THE "BATH SALTS" SYNTHETIC CATHINONES MEPHEDRONE AND MDPV IN RATS. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Oct 18]. Available from: http://digital.library.temple.edu/u?/p245801coll10,354278.

Council of Science Editors:

Gregg RA. INVESTIGATIONS INTO THE STEREOCHEMICAL AND GLUTAMATERGIC MECHANISMS OF THE "BATH SALTS" SYNTHETIC CATHINONES MEPHEDRONE AND MDPV IN RATS. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,354278


Temple University

6. Tashovski, Ivan. BAG6 as a Novel HIV-1 Host Factor.

Degree: PhD, 2016, Temple University

Microbiology and Immunology

The human immunodeficiency virus type-1 (HIV-1) is the major etiological agent of acquired immunodeficiency syndrome (AIDS), the cause of over 30 million… (more)

Subjects/Keywords: Microbiology; Virology; Molecular biology;

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APA (6th Edition):

Tashovski, I. (2016). BAG6 as a Novel HIV-1 Host Factor. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,420479

Chicago Manual of Style (16th Edition):

Tashovski, Ivan. “BAG6 as a Novel HIV-1 Host Factor.” 2016. Doctoral Dissertation, Temple University. Accessed October 18, 2019. http://digital.library.temple.edu/u?/p245801coll10,420479.

MLA Handbook (7th Edition):

Tashovski, Ivan. “BAG6 as a Novel HIV-1 Host Factor.” 2016. Web. 18 Oct 2019.

Vancouver:

Tashovski I. BAG6 as a Novel HIV-1 Host Factor. [Internet] [Doctoral dissertation]. Temple University; 2016. [cited 2019 Oct 18]. Available from: http://digital.library.temple.edu/u?/p245801coll10,420479.

Council of Science Editors:

Tashovski I. BAG6 as a Novel HIV-1 Host Factor. [Doctoral Dissertation]. Temple University; 2016. Available from: http://digital.library.temple.edu/u?/p245801coll10,420479


Temple University

7. Gentile, Taylor Arthur. Investigations into the Role of Orexin (Hypocretin) and Dynorphin in Drug Seeking, Reinforcement, and Withdrawal.

Degree: PhD, 2018, Temple University

Biomedical Sciences

Psychostimulant dependence remains a major health and economic problem, leading to premature death and costing $181 billion annually in health care, crime, and… (more)

Subjects/Keywords: Neurosciences; Behavioral sciences; Pharmacology;

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APA (6th Edition):

Gentile, T. A. (2018). Investigations into the Role of Orexin (Hypocretin) and Dynorphin in Drug Seeking, Reinforcement, and Withdrawal. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,507530

Chicago Manual of Style (16th Edition):

Gentile, Taylor Arthur. “Investigations into the Role of Orexin (Hypocretin) and Dynorphin in Drug Seeking, Reinforcement, and Withdrawal.” 2018. Doctoral Dissertation, Temple University. Accessed October 18, 2019. http://digital.library.temple.edu/u?/p245801coll10,507530.

MLA Handbook (7th Edition):

Gentile, Taylor Arthur. “Investigations into the Role of Orexin (Hypocretin) and Dynorphin in Drug Seeking, Reinforcement, and Withdrawal.” 2018. Web. 18 Oct 2019.

Vancouver:

Gentile TA. Investigations into the Role of Orexin (Hypocretin) and Dynorphin in Drug Seeking, Reinforcement, and Withdrawal. [Internet] [Doctoral dissertation]. Temple University; 2018. [cited 2019 Oct 18]. Available from: http://digital.library.temple.edu/u?/p245801coll10,507530.

Council of Science Editors:

Gentile TA. Investigations into the Role of Orexin (Hypocretin) and Dynorphin in Drug Seeking, Reinforcement, and Withdrawal. [Doctoral Dissertation]. Temple University; 2018. Available from: http://digital.library.temple.edu/u?/p245801coll10,507530


Temple University

8. Saber, Iman A. CHARACTERIZING THE PHARMACOLOGICAL PROFILE OF MEPHEDRONE AND DETERMINING THE ABUSE LIABILITY MECHANISMS.

Degree: PhD, 2017, Temple University

Pharmaceutical Sciences

Illicit drug use has been a growing concern over the past few decades. The rise in use of illegal drugs drove the government… (more)

Subjects/Keywords: Pharmacology; Behavioral sciences; Neurosciences;

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Saber, I. A. (2017). CHARACTERIZING THE PHARMACOLOGICAL PROFILE OF MEPHEDRONE AND DETERMINING THE ABUSE LIABILITY MECHANISMS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,478603

Chicago Manual of Style (16th Edition):

Saber, Iman A. “CHARACTERIZING THE PHARMACOLOGICAL PROFILE OF MEPHEDRONE AND DETERMINING THE ABUSE LIABILITY MECHANISMS.” 2017. Doctoral Dissertation, Temple University. Accessed October 18, 2019. http://digital.library.temple.edu/u?/p245801coll10,478603.

MLA Handbook (7th Edition):

Saber, Iman A. “CHARACTERIZING THE PHARMACOLOGICAL PROFILE OF MEPHEDRONE AND DETERMINING THE ABUSE LIABILITY MECHANISMS.” 2017. Web. 18 Oct 2019.

Vancouver:

Saber IA. CHARACTERIZING THE PHARMACOLOGICAL PROFILE OF MEPHEDRONE AND DETERMINING THE ABUSE LIABILITY MECHANISMS. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2019 Oct 18]. Available from: http://digital.library.temple.edu/u?/p245801coll10,478603.

Council of Science Editors:

Saber IA. CHARACTERIZING THE PHARMACOLOGICAL PROFILE OF MEPHEDRONE AND DETERMINING THE ABUSE LIABILITY MECHANISMS. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,478603

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