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You searched for +publisher:"Temple University" +contributor:("Tsygankov, Alexander;"). Showing records 1 – 30 of 41 total matches.

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Temple University

1. Lee, Hojin. S1_Scrambled_siRNA [Digital file].

Degree: 2008, Temple University

Microbiology and Immunology;

Ph.D.;

c-Cbl functions as a multifunctional adaptor and an E3 ubiquitin protein ligase. Several studies have shown that c-Cbl is involved in… (more)

Subjects/Keywords: Biology, Cell;

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lee, H. (2008). S1_Scrambled_siRNA [Digital file]. (Thesis). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,254229

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, Hojin. “S1_Scrambled_siRNA [Digital file].” 2008. Thesis, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,254229.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, Hojin. “S1_Scrambled_siRNA [Digital file].” 2008. Web. 06 Jun 2020.

Vancouver:

Lee H. S1_Scrambled_siRNA [Digital file]. [Internet] [Thesis]. Temple University; 2008. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,254229.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee H. S1_Scrambled_siRNA [Digital file]. [Thesis]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,254229

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

2. Lee, Hojin. S2_RhoA_siRNA [Digital file].

Degree: 2008, Temple University

Microbiology and Immunology;

Ph.D.;

c-Cbl functions as a multifunctional adaptor and an E3 ubiquitin protein ligase. Several studies have shown that c-Cbl is involved in… (more)

Subjects/Keywords: Biology, Cell;

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APA (6th Edition):

Lee, H. (2008). S2_RhoA_siRNA [Digital file]. (Thesis). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,254230

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, Hojin. “S2_RhoA_siRNA [Digital file].” 2008. Thesis, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,254230.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, Hojin. “S2_RhoA_siRNA [Digital file].” 2008. Web. 06 Jun 2020.

Vancouver:

Lee H. S2_RhoA_siRNA [Digital file]. [Internet] [Thesis]. Temple University; 2008. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,254230.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee H. S2_RhoA_siRNA [Digital file]. [Thesis]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,254230

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

3. Lee, Hojin. S3_Rac1_siRNA [Digital file].

Degree: 2008, Temple University

Microbiology and Immunology;

Ph.D.;

c-Cbl functions as a multifunctional adaptor and an E3 ubiquitin protein ligase. Several studies have shown that c-Cbl is involved in… (more)

Subjects/Keywords: Biology, Cell;

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lee, H. (2008). S3_Rac1_siRNA [Digital file]. (Thesis). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,254231

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lee, Hojin. “S3_Rac1_siRNA [Digital file].” 2008. Thesis, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,254231.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lee, Hojin. “S3_Rac1_siRNA [Digital file].” 2008. Web. 06 Jun 2020.

Vancouver:

Lee H. S3_Rac1_siRNA [Digital file]. [Internet] [Thesis]. Temple University; 2008. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,254231.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lee H. S3_Rac1_siRNA [Digital file]. [Thesis]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,254231

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

4. Lee, Hojin. THE MOLECULAR MECHANISMS OF THE EFFECTS OF C-CBL ON CYTOSKELETON-MEDIATED PHENOMENA.

Degree: PhD, 2008, Temple University

Microbiology and Immunology

c-Cbl functions as a multifunctional adaptor and an E3 ubiquitin protein ligase. Several studies have shown that c-Cbl is involved in cytoskeleton-mediated… (more)

Subjects/Keywords: Biology, Cell; c-Cbl; Cytoskeleton; small GTPases; Signaling; Glioma; Invasion

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lee, H. (2008). THE MOLECULAR MECHANISMS OF THE EFFECTS OF C-CBL ON CYTOSKELETON-MEDIATED PHENOMENA. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,20447

Chicago Manual of Style (16th Edition):

Lee, Hojin. “THE MOLECULAR MECHANISMS OF THE EFFECTS OF C-CBL ON CYTOSKELETON-MEDIATED PHENOMENA.” 2008. Doctoral Dissertation, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,20447.

MLA Handbook (7th Edition):

Lee, Hojin. “THE MOLECULAR MECHANISMS OF THE EFFECTS OF C-CBL ON CYTOSKELETON-MEDIATED PHENOMENA.” 2008. Web. 06 Jun 2020.

Vancouver:

Lee H. THE MOLECULAR MECHANISMS OF THE EFFECTS OF C-CBL ON CYTOSKELETON-MEDIATED PHENOMENA. [Internet] [Doctoral dissertation]. Temple University; 2008. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,20447.

Council of Science Editors:

Lee H. THE MOLECULAR MECHANISMS OF THE EFFECTS OF C-CBL ON CYTOSKELETON-MEDIATED PHENOMENA. [Doctoral Dissertation]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,20447


Temple University

5. Buitrago Murcia, Claudia Lorena. Cbl proteins in platelet functional responses.

Degree: PhD, 2012, Temple University

Physiology

c-Cbl protein functions as an E3 ligase and scaffolding protein, where three residues, Y700, Y731, and Y774, upon phosphorylation, have been shown to initiate… (more)

Subjects/Keywords: Physiology; Cellular biology; Cbl; hemostasis; kinases; platelets; signaling

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APA (6th Edition):

Buitrago Murcia, C. L. (2012). Cbl proteins in platelet functional responses. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,198139

Chicago Manual of Style (16th Edition):

Buitrago Murcia, Claudia Lorena. “Cbl proteins in platelet functional responses.” 2012. Doctoral Dissertation, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,198139.

MLA Handbook (7th Edition):

Buitrago Murcia, Claudia Lorena. “Cbl proteins in platelet functional responses.” 2012. Web. 06 Jun 2020.

Vancouver:

Buitrago Murcia CL. Cbl proteins in platelet functional responses. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,198139.

Council of Science Editors:

Buitrago Murcia CL. Cbl proteins in platelet functional responses. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,198139


Temple University

6. Kocieda, Virginia Polonia. Prostaglandin E2-induced IL-23p19 is regulated by CREB and C/EBP beta in bone marrow derived dendritic cells.

Degree: PhD, 2013, Temple University

Microbiology and Immunology

We reported previously that prostaglandin E2 (PGE2) upregulates IL-23 in vitro in bone marrow-derived dendritic cells (DC), and in vivo in models… (more)

Subjects/Keywords: Immunology; Molecular biology; C/EBP; CREB; dendritic cell; IL-23; PGE2

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Kocieda, V. P. (2013). Prostaglandin E2-induced IL-23p19 is regulated by CREB and C/EBP beta in bone marrow derived dendritic cells. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,214805

Chicago Manual of Style (16th Edition):

Kocieda, Virginia Polonia. “Prostaglandin E2-induced IL-23p19 is regulated by CREB and C/EBP beta in bone marrow derived dendritic cells.” 2013. Doctoral Dissertation, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,214805.

MLA Handbook (7th Edition):

Kocieda, Virginia Polonia. “Prostaglandin E2-induced IL-23p19 is regulated by CREB and C/EBP beta in bone marrow derived dendritic cells.” 2013. Web. 06 Jun 2020.

Vancouver:

Kocieda VP. Prostaglandin E2-induced IL-23p19 is regulated by CREB and C/EBP beta in bone marrow derived dendritic cells. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,214805.

Council of Science Editors:

Kocieda VP. Prostaglandin E2-induced IL-23p19 is regulated by CREB and C/EBP beta in bone marrow derived dendritic cells. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,214805


Temple University

7. Regan, Genevieve Victoria. Activation of sigma G during formation of spores by Bacillus subtilis depends on completion of engulfment, which follows translocation of the chromosome into the prespore.

Degree: PhD, 2012, Temple University

Microbiology and Immunology

The process of sporulation in Bacillus subtilis serves as a paradigm for study of sporulation in all Bacillus and Clostridium species. During… (more)

Subjects/Keywords: Microbiology; Bacillus; sigma G; sporulation; subtilis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Regan, G. V. (2012). Activation of sigma G during formation of spores by Bacillus subtilis depends on completion of engulfment, which follows translocation of the chromosome into the prespore. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,214811

Chicago Manual of Style (16th Edition):

Regan, Genevieve Victoria. “Activation of sigma G during formation of spores by Bacillus subtilis depends on completion of engulfment, which follows translocation of the chromosome into the prespore.” 2012. Doctoral Dissertation, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,214811.

MLA Handbook (7th Edition):

Regan, Genevieve Victoria. “Activation of sigma G during formation of spores by Bacillus subtilis depends on completion of engulfment, which follows translocation of the chromosome into the prespore.” 2012. Web. 06 Jun 2020.

Vancouver:

Regan GV. Activation of sigma G during formation of spores by Bacillus subtilis depends on completion of engulfment, which follows translocation of the chromosome into the prespore. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,214811.

Council of Science Editors:

Regan GV. Activation of sigma G during formation of spores by Bacillus subtilis depends on completion of engulfment, which follows translocation of the chromosome into the prespore. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,214811


Temple University

8. Cutrera, Jason Lewis. Insights into the Structure and Function of PrgW and its Conserved Cysteines.

Degree: PhD, 2014, Temple University

Microbiology and Immunology

Enterococcus faecalis is a Gram-positive bacterial species that is typically a member of the human gastrointestinal tract microbiota. However, E. faecalis is… (more)

Subjects/Keywords: Microbiology;

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cutrera, J. L. (2014). Insights into the Structure and Function of PrgW and its Conserved Cysteines. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,299645

Chicago Manual of Style (16th Edition):

Cutrera, Jason Lewis. “Insights into the Structure and Function of PrgW and its Conserved Cysteines.” 2014. Doctoral Dissertation, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,299645.

MLA Handbook (7th Edition):

Cutrera, Jason Lewis. “Insights into the Structure and Function of PrgW and its Conserved Cysteines.” 2014. Web. 06 Jun 2020.

Vancouver:

Cutrera JL. Insights into the Structure and Function of PrgW and its Conserved Cysteines. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,299645.

Council of Science Editors:

Cutrera JL. Insights into the Structure and Function of PrgW and its Conserved Cysteines. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,299645


Temple University

9. Pokiniewski, Katie Ann. Understanding the cellular mechanism of Adeno-Associated Virus genome stabilization.

Degree: PhD, 2016, Temple University

Microbiology and Immunology

The Adeno-Associated Virus(AAV) is a small, single stranded DNA virus that has been developed as a gene transfer vector. Early clinical trials… (more)

Subjects/Keywords: Virology; Microbiology;

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APA (6th Edition):

Pokiniewski, K. A. (2016). Understanding the cellular mechanism of Adeno-Associated Virus genome stabilization. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,385837

Chicago Manual of Style (16th Edition):

Pokiniewski, Katie Ann. “Understanding the cellular mechanism of Adeno-Associated Virus genome stabilization.” 2016. Doctoral Dissertation, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,385837.

MLA Handbook (7th Edition):

Pokiniewski, Katie Ann. “Understanding the cellular mechanism of Adeno-Associated Virus genome stabilization.” 2016. Web. 06 Jun 2020.

Vancouver:

Pokiniewski KA. Understanding the cellular mechanism of Adeno-Associated Virus genome stabilization. [Internet] [Doctoral dissertation]. Temple University; 2016. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,385837.

Council of Science Editors:

Pokiniewski KA. Understanding the cellular mechanism of Adeno-Associated Virus genome stabilization. [Doctoral Dissertation]. Temple University; 2016. Available from: http://digital.library.temple.edu/u?/p245801coll10,385837


Temple University

10. JAIN,SURBHI. ROLE OF INTERLEUKIN-19 AND ALLOGRAFT INFLAMMATORY FACTOR-1 IN ENDOTHELIAL CELL PROLIFERATION, ACTIVATION, MIGRATION AND ANGIOGENIC POTENTIAL.

Degree: PhD, 2009, Temple University

Microbiology and Immunology

Angiogenesis is an important process in maintaining normal physiology as well as in the pathology of many diseases. Angiogenesis based therapies have… (more)

Subjects/Keywords: Biology, Microbiology; Health Sciences, Immunology; Allograft Inflammatory Factor-1; Angiogenesis; Endothelial Cell; Interleukin-19; Migration; Proliferation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

JAIN,SURBHI. (2009). ROLE OF INTERLEUKIN-19 AND ALLOGRAFT INFLAMMATORY FACTOR-1 IN ENDOTHELIAL CELL PROLIFERATION, ACTIVATION, MIGRATION AND ANGIOGENIC POTENTIAL. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,25799

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

JAIN,SURBHI. “ROLE OF INTERLEUKIN-19 AND ALLOGRAFT INFLAMMATORY FACTOR-1 IN ENDOTHELIAL CELL PROLIFERATION, ACTIVATION, MIGRATION AND ANGIOGENIC POTENTIAL.” 2009. Doctoral Dissertation, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,25799.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

JAIN,SURBHI. “ROLE OF INTERLEUKIN-19 AND ALLOGRAFT INFLAMMATORY FACTOR-1 IN ENDOTHELIAL CELL PROLIFERATION, ACTIVATION, MIGRATION AND ANGIOGENIC POTENTIAL.” 2009. Web. 06 Jun 2020.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

JAIN,SURBHI. ROLE OF INTERLEUKIN-19 AND ALLOGRAFT INFLAMMATORY FACTOR-1 IN ENDOTHELIAL CELL PROLIFERATION, ACTIVATION, MIGRATION AND ANGIOGENIC POTENTIAL. [Internet] [Doctoral dissertation]. Temple University; 2009. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,25799.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

JAIN,SURBHI. ROLE OF INTERLEUKIN-19 AND ALLOGRAFT INFLAMMATORY FACTOR-1 IN ENDOTHELIAL CELL PROLIFERATION, ACTIVATION, MIGRATION AND ANGIOGENIC POTENTIAL. [Doctoral Dissertation]. Temple University; 2009. Available from: http://digital.library.temple.edu/u?/p245801coll10,25799

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


Temple University

11. Zhao, Jianhua. HACE1, a Novel Repressor of RAR Transcriptional Activity.

Degree: PhD, 2009, Temple University

Microbiology and Immunology

The biological activities of retinoic acid (RA) and its synthetic analogues are mediated through nuclear receptors, termed retinoic acid receptors (RARs) and… (more)

Subjects/Keywords: Biology, Molecular; HACE1; RAR; Repressor

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhao, J. (2009). HACE1, a Novel Repressor of RAR Transcriptional Activity. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,27963

Chicago Manual of Style (16th Edition):

Zhao, Jianhua. “HACE1, a Novel Repressor of RAR Transcriptional Activity.” 2009. Doctoral Dissertation, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,27963.

MLA Handbook (7th Edition):

Zhao, Jianhua. “HACE1, a Novel Repressor of RAR Transcriptional Activity.” 2009. Web. 06 Jun 2020.

Vancouver:

Zhao J. HACE1, a Novel Repressor of RAR Transcriptional Activity. [Internet] [Doctoral dissertation]. Temple University; 2009. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,27963.

Council of Science Editors:

Zhao J. HACE1, a Novel Repressor of RAR Transcriptional Activity. [Doctoral Dissertation]. Temple University; 2009. Available from: http://digital.library.temple.edu/u?/p245801coll10,27963


Temple University

12. Thomas, Dafydd Huw. Regulation of Syk activity in GPVI-mediated platelet activation.

Degree: PhD, 2010, Temple University

Pharmacology

Activation of platelets is essential for hemostasis. Following damage to the vascular endothelium collagen is exposed, to which platelets stably adhere. After adhesion on… (more)

Subjects/Keywords: Health Sciences, Pharmacology; Collagen; GPVI; Phosphatase; Platelets; Syk; TULA

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APA (6th Edition):

Thomas, D. H. (2010). Regulation of Syk activity in GPVI-mediated platelet activation. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,92028

Chicago Manual of Style (16th Edition):

Thomas, Dafydd Huw. “Regulation of Syk activity in GPVI-mediated platelet activation.” 2010. Doctoral Dissertation, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,92028.

MLA Handbook (7th Edition):

Thomas, Dafydd Huw. “Regulation of Syk activity in GPVI-mediated platelet activation.” 2010. Web. 06 Jun 2020.

Vancouver:

Thomas DH. Regulation of Syk activity in GPVI-mediated platelet activation. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,92028.

Council of Science Editors:

Thomas DH. Regulation of Syk activity in GPVI-mediated platelet activation. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,92028


Temple University

13. Roberts, Sean Anthony. A GENE THERAPY APPROACH TO THE INHIBITION OF HIV-1 REPLICATION BY RESTORATION OF INNATE ANTIVIRAL DEFENSE PATHWAYS.

Degree: PhD, 2010, Temple University

Microbiology and Immunology

Since it emerged as an infectious agent in 1981, the human immunodeficiency virus type 1 (HIV-1) is continually disseminated and remain fatal… (more)

Subjects/Keywords: Biology, Microbiology; Biology, virology

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APA (6th Edition):

Roberts, S. A. (2010). A GENE THERAPY APPROACH TO THE INHIBITION OF HIV-1 REPLICATION BY RESTORATION OF INNATE ANTIVIRAL DEFENSE PATHWAYS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,99935

Chicago Manual of Style (16th Edition):

Roberts, Sean Anthony. “A GENE THERAPY APPROACH TO THE INHIBITION OF HIV-1 REPLICATION BY RESTORATION OF INNATE ANTIVIRAL DEFENSE PATHWAYS.” 2010. Doctoral Dissertation, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,99935.

MLA Handbook (7th Edition):

Roberts, Sean Anthony. “A GENE THERAPY APPROACH TO THE INHIBITION OF HIV-1 REPLICATION BY RESTORATION OF INNATE ANTIVIRAL DEFENSE PATHWAYS.” 2010. Web. 06 Jun 2020.

Vancouver:

Roberts SA. A GENE THERAPY APPROACH TO THE INHIBITION OF HIV-1 REPLICATION BY RESTORATION OF INNATE ANTIVIRAL DEFENSE PATHWAYS. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,99935.

Council of Science Editors:

Roberts SA. A GENE THERAPY APPROACH TO THE INHIBITION OF HIV-1 REPLICATION BY RESTORATION OF INNATE ANTIVIRAL DEFENSE PATHWAYS. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,99935


Temple University

14. Xenopoulos, Panagiotis. TWIN SPORE FORMATION WITHIN ONE MOTHER CELL BY BACILLUS SUBTILIS.

Degree: PhD, 2011, Temple University

Microbiology and Immunology

Formation of spores by Bacillus subtilis is a primitive system of differentiation that has become a paradigm for studying cell differentiation in… (more)

Subjects/Keywords: Microbiology; Cellular Biology; BACILLUS SUBTILIS; SPORULATION

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APA (6th Edition):

Xenopoulos, P. (2011). TWIN SPORE FORMATION WITHIN ONE MOTHER CELL BY BACILLUS SUBTILIS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,135810

Chicago Manual of Style (16th Edition):

Xenopoulos, Panagiotis. “TWIN SPORE FORMATION WITHIN ONE MOTHER CELL BY BACILLUS SUBTILIS.” 2011. Doctoral Dissertation, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,135810.

MLA Handbook (7th Edition):

Xenopoulos, Panagiotis. “TWIN SPORE FORMATION WITHIN ONE MOTHER CELL BY BACILLUS SUBTILIS.” 2011. Web. 06 Jun 2020.

Vancouver:

Xenopoulos P. TWIN SPORE FORMATION WITHIN ONE MOTHER CELL BY BACILLUS SUBTILIS. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,135810.

Council of Science Editors:

Xenopoulos P. TWIN SPORE FORMATION WITHIN ONE MOTHER CELL BY BACILLUS SUBTILIS. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,135810


Temple University

15. Schiraldi, Michael. Tim-3 and Cell Death in Murine Mercury Induced Autoimmunity.

Degree: PhD, 2011, Temple University

Microbiology and Immunology

There is a role for environmental factors in the pathogenesis of autoimmune diseases in humans and animals. Correlations have been made between… (more)

Subjects/Keywords: Immunology; autoimmunity; mercury; murine

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APA (6th Edition):

Schiraldi, M. (2011). Tim-3 and Cell Death in Murine Mercury Induced Autoimmunity. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,136653

Chicago Manual of Style (16th Edition):

Schiraldi, Michael. “Tim-3 and Cell Death in Murine Mercury Induced Autoimmunity.” 2011. Doctoral Dissertation, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,136653.

MLA Handbook (7th Edition):

Schiraldi, Michael. “Tim-3 and Cell Death in Murine Mercury Induced Autoimmunity.” 2011. Web. 06 Jun 2020.

Vancouver:

Schiraldi M. Tim-3 and Cell Death in Murine Mercury Induced Autoimmunity. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,136653.

Council of Science Editors:

Schiraldi M. Tim-3 and Cell Death in Murine Mercury Induced Autoimmunity. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,136653


Temple University

16. MOTHEY, DEEPA. EFFECT OF MUCIN ON EXPONENTIAL GROWTH, STATIONARY PHASE SURVIVAL AND BIOFILM FORMATION IN STREPTOCOCCUS MUTANS.

Degree: PhD, 2012, Temple University

Microbiology and Immunology

Streptococcus mutans is a member of the dental plaque and is the principal causative agent of dental caries. It can metabolize a… (more)

Subjects/Keywords: Microbiology; biofilm; mucin; stationary phase; streptococcus mutans

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APA (6th Edition):

MOTHEY, D. (2012). EFFECT OF MUCIN ON EXPONENTIAL GROWTH, STATIONARY PHASE SURVIVAL AND BIOFILM FORMATION IN STREPTOCOCCUS MUTANS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,184609

Chicago Manual of Style (16th Edition):

MOTHEY, DEEPA. “EFFECT OF MUCIN ON EXPONENTIAL GROWTH, STATIONARY PHASE SURVIVAL AND BIOFILM FORMATION IN STREPTOCOCCUS MUTANS.” 2012. Doctoral Dissertation, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,184609.

MLA Handbook (7th Edition):

MOTHEY, DEEPA. “EFFECT OF MUCIN ON EXPONENTIAL GROWTH, STATIONARY PHASE SURVIVAL AND BIOFILM FORMATION IN STREPTOCOCCUS MUTANS.” 2012. Web. 06 Jun 2020.

Vancouver:

MOTHEY D. EFFECT OF MUCIN ON EXPONENTIAL GROWTH, STATIONARY PHASE SURVIVAL AND BIOFILM FORMATION IN STREPTOCOCCUS MUTANS. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,184609.

Council of Science Editors:

MOTHEY D. EFFECT OF MUCIN ON EXPONENTIAL GROWTH, STATIONARY PHASE SURVIVAL AND BIOFILM FORMATION IN STREPTOCOCCUS MUTANS. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,184609


Temple University

17. Suriano, April Rose. Distinct Subpopulations in Biofilms of Streptococcus mutans and their Response to Sugar Starvation and Restoration.

Degree: PhD, 2012, Temple University

Microbiology and Immunology

Streptococcus mutans is a secondary colonizer of the dental plaque biofilm and is the primary causative agent of dental caries. Sugar metabolism… (more)

Subjects/Keywords: Microbiology; biofilms; GFP; starvation; Streptococcus mutans; subpopulations; survival

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APA (6th Edition):

Suriano, A. R. (2012). Distinct Subpopulations in Biofilms of Streptococcus mutans and their Response to Sugar Starvation and Restoration. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,187730

Chicago Manual of Style (16th Edition):

Suriano, April Rose. “Distinct Subpopulations in Biofilms of Streptococcus mutans and their Response to Sugar Starvation and Restoration.” 2012. Doctoral Dissertation, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,187730.

MLA Handbook (7th Edition):

Suriano, April Rose. “Distinct Subpopulations in Biofilms of Streptococcus mutans and their Response to Sugar Starvation and Restoration.” 2012. Web. 06 Jun 2020.

Vancouver:

Suriano AR. Distinct Subpopulations in Biofilms of Streptococcus mutans and their Response to Sugar Starvation and Restoration. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,187730.

Council of Science Editors:

Suriano AR. Distinct Subpopulations in Biofilms of Streptococcus mutans and their Response to Sugar Starvation and Restoration. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,187730


Temple University

18. Newman, Tiffanny Nicole. ROLE OF TULA-FAMILY PROTEINS IN T CELL DRIVEN RESPONSES.

Degree: PhD, 2011, Temple University

Microbiology and Immunology

The TULA-family consists of two proteins implicated in cellular regulation. TULA-1 is expressed in T-cells and is involved in apoptosis. TULA-2 is… (more)

Subjects/Keywords: Immunology; autoimmune; inflammatory bowel disease; phosphatase; T cells; TULA-1; TULA-2

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APA (6th Edition):

Newman, T. N. (2011). ROLE OF TULA-FAMILY PROTEINS IN T CELL DRIVEN RESPONSES. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,210733

Chicago Manual of Style (16th Edition):

Newman, Tiffanny Nicole. “ROLE OF TULA-FAMILY PROTEINS IN T CELL DRIVEN RESPONSES.” 2011. Doctoral Dissertation, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,210733.

MLA Handbook (7th Edition):

Newman, Tiffanny Nicole. “ROLE OF TULA-FAMILY PROTEINS IN T CELL DRIVEN RESPONSES.” 2011. Web. 06 Jun 2020.

Vancouver:

Newman TN. ROLE OF TULA-FAMILY PROTEINS IN T CELL DRIVEN RESPONSES. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,210733.

Council of Science Editors:

Newman TN. ROLE OF TULA-FAMILY PROTEINS IN T CELL DRIVEN RESPONSES. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,210733


Temple University

19. Sirisani, Evelyn. The Role of Regulatory Genes in Mediating Growth Arrest by all-trans Retinoic Acid in Ovarian Carcinoma Cell Lines.

Degree: PhD, 2012, Temple University

Microbiology and Immunology

All-trans retinoic acid (atRA) mediated growth inhibition results in the arrest of the cell cycle during the G1 phase in CAOV3 cells… (more)

Subjects/Keywords: Microbiology; Immunology; Molecular biology; all-trans retinoic acid; full length HOXA1; HOXA1; HOXB4; p16INK4a; truncated form HOXA1

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APA (6th Edition):

Sirisani, E. (2012). The Role of Regulatory Genes in Mediating Growth Arrest by all-trans Retinoic Acid in Ovarian Carcinoma Cell Lines. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,210886

Chicago Manual of Style (16th Edition):

Sirisani, Evelyn. “The Role of Regulatory Genes in Mediating Growth Arrest by all-trans Retinoic Acid in Ovarian Carcinoma Cell Lines.” 2012. Doctoral Dissertation, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,210886.

MLA Handbook (7th Edition):

Sirisani, Evelyn. “The Role of Regulatory Genes in Mediating Growth Arrest by all-trans Retinoic Acid in Ovarian Carcinoma Cell Lines.” 2012. Web. 06 Jun 2020.

Vancouver:

Sirisani E. The Role of Regulatory Genes in Mediating Growth Arrest by all-trans Retinoic Acid in Ovarian Carcinoma Cell Lines. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,210886.

Council of Science Editors:

Sirisani E. The Role of Regulatory Genes in Mediating Growth Arrest by all-trans Retinoic Acid in Ovarian Carcinoma Cell Lines. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,210886


Temple University

20. Utter, Bryan David. PHEROMONE-INTERACTING REPLICATION PROTEIN CONTROLS ENTEROCOCCAL CONJUGATIVE PLASMID HOST RANGE AND STABILITY THROUGH DISULFIDE BONDS.

Degree: PhD, 2012, Temple University

Microbiology and Immunology

Enterococci are found in soil, sewage, food, water, and are commensal to the gastrointestinal tracts of mammals, insects, and birds. Enterococci often… (more)

Subjects/Keywords: Microbiology; disulfide bonds; enterococcus faecalis; host range; pCF10; plasmid stability

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APA (6th Edition):

Utter, B. D. (2012). PHEROMONE-INTERACTING REPLICATION PROTEIN CONTROLS ENTEROCOCCAL CONJUGATIVE PLASMID HOST RANGE AND STABILITY THROUGH DISULFIDE BONDS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,211277

Chicago Manual of Style (16th Edition):

Utter, Bryan David. “PHEROMONE-INTERACTING REPLICATION PROTEIN CONTROLS ENTEROCOCCAL CONJUGATIVE PLASMID HOST RANGE AND STABILITY THROUGH DISULFIDE BONDS.” 2012. Doctoral Dissertation, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,211277.

MLA Handbook (7th Edition):

Utter, Bryan David. “PHEROMONE-INTERACTING REPLICATION PROTEIN CONTROLS ENTEROCOCCAL CONJUGATIVE PLASMID HOST RANGE AND STABILITY THROUGH DISULFIDE BONDS.” 2012. Web. 06 Jun 2020.

Vancouver:

Utter BD. PHEROMONE-INTERACTING REPLICATION PROTEIN CONTROLS ENTEROCOCCAL CONJUGATIVE PLASMID HOST RANGE AND STABILITY THROUGH DISULFIDE BONDS. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,211277.

Council of Science Editors:

Utter BD. PHEROMONE-INTERACTING REPLICATION PROTEIN CONTROLS ENTEROCOCCAL CONJUGATIVE PLASMID HOST RANGE AND STABILITY THROUGH DISULFIDE BONDS. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,211277


Temple University

21. Getz, Todd Michael. The Regulation of Phosphorylation Events in Platelets.

Degree: PhD, 2012, Temple University

Physiology

Platelets play a vital role in processes of hemostasis and thrombosis under physiological and pathological conditions. Following vascular damage, platelets will accumulate and stably… (more)

Subjects/Keywords: Physiology; Biology; Biochemistry; Dextran sulfate; Myosin light chain; Phosphorylation; Platelets; signal transduction; Syk kinase

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APA (6th Edition):

Getz, T. M. (2012). The Regulation of Phosphorylation Events in Platelets. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,213114

Chicago Manual of Style (16th Edition):

Getz, Todd Michael. “The Regulation of Phosphorylation Events in Platelets.” 2012. Doctoral Dissertation, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,213114.

MLA Handbook (7th Edition):

Getz, Todd Michael. “The Regulation of Phosphorylation Events in Platelets.” 2012. Web. 06 Jun 2020.

Vancouver:

Getz TM. The Regulation of Phosphorylation Events in Platelets. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,213114.

Council of Science Editors:

Getz TM. The Regulation of Phosphorylation Events in Platelets. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,213114


Temple University

22. Bolton, Elisabeth Spring. Genomic Instability Originates From Leukemia Stem Cells In a Mouse Model of CML-CP.

Degree: PhD, 2013, Temple University

Microbiology and Immunology

In chronic myelogenous leukemia (CML), activation of BCR-ABL, the product of the bcr-abl chimeric gene, leads to constitutive activation of pathways that… (more)

Subjects/Keywords: Biology; Molecular biology; Immunology;

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APA (6th Edition):

Bolton, E. S. (2013). Genomic Instability Originates From Leukemia Stem Cells In a Mouse Model of CML-CP. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,234916

Chicago Manual of Style (16th Edition):

Bolton, Elisabeth Spring. “Genomic Instability Originates From Leukemia Stem Cells In a Mouse Model of CML-CP.” 2013. Doctoral Dissertation, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,234916.

MLA Handbook (7th Edition):

Bolton, Elisabeth Spring. “Genomic Instability Originates From Leukemia Stem Cells In a Mouse Model of CML-CP.” 2013. Web. 06 Jun 2020.

Vancouver:

Bolton ES. Genomic Instability Originates From Leukemia Stem Cells In a Mouse Model of CML-CP. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,234916.

Council of Science Editors:

Bolton ES. Genomic Instability Originates From Leukemia Stem Cells In a Mouse Model of CML-CP. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,234916


Temple University

23. Oppong, Gertrude Odamea. The Role of Bacterial Amyloids In Regulating Gastrointestinal Homeostasis.

Degree: PhD, 2015, Temple University

Microbiology and Immunology

Many bacterial species exist in nature as part of highly structured multicellular communities known as biofilms. Amyloids, proteins with a conserved β-sheet… (more)

Subjects/Keywords: Microbiology;

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APA (6th Edition):

Oppong, G. O. (2015). The Role of Bacterial Amyloids In Regulating Gastrointestinal Homeostasis. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,323094

Chicago Manual of Style (16th Edition):

Oppong, Gertrude Odamea. “The Role of Bacterial Amyloids In Regulating Gastrointestinal Homeostasis.” 2015. Doctoral Dissertation, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,323094.

MLA Handbook (7th Edition):

Oppong, Gertrude Odamea. “The Role of Bacterial Amyloids In Regulating Gastrointestinal Homeostasis.” 2015. Web. 06 Jun 2020.

Vancouver:

Oppong GO. The Role of Bacterial Amyloids In Regulating Gastrointestinal Homeostasis. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,323094.

Council of Science Editors:

Oppong GO. The Role of Bacterial Amyloids In Regulating Gastrointestinal Homeostasis. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,323094


Temple University

24. Chatila, Wissam M. F. MicroRNA Expression in Regulatory T Cells in Chronic Obstructive Pulmonary Disease.

Degree: PhD, 2015, Temple University

Microbiology and Immunology

COPD is characterized by an abnormal regulatory T cell (Treg) response with a shift towards a Th1 and Th17 cell responses. However,… (more)

Subjects/Keywords: Immunology; Cellular biology; Molecular biology;

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APA (6th Edition):

Chatila, W. M. F. (2015). MicroRNA Expression in Regulatory T Cells in Chronic Obstructive Pulmonary Disease. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,333572

Chicago Manual of Style (16th Edition):

Chatila, Wissam M F. “MicroRNA Expression in Regulatory T Cells in Chronic Obstructive Pulmonary Disease.” 2015. Doctoral Dissertation, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,333572.

MLA Handbook (7th Edition):

Chatila, Wissam M F. “MicroRNA Expression in Regulatory T Cells in Chronic Obstructive Pulmonary Disease.” 2015. Web. 06 Jun 2020.

Vancouver:

Chatila WMF. MicroRNA Expression in Regulatory T Cells in Chronic Obstructive Pulmonary Disease. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,333572.

Council of Science Editors:

Chatila WMF. MicroRNA Expression in Regulatory T Cells in Chronic Obstructive Pulmonary Disease. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,333572


Temple University

25. Manne, Bhanu Kanth. CLEC-2 SIGNAL TRANSDUCTION IN PLATELET ACTIVATION.

Degree: PhD, 2015, Temple University

Physiology

Platelets are involved in many processes ranging from fighting microbial infections and triggering inflammation to promoting tumor angiogenesis and metastasis. Nevertheless, the primary physiological… (more)

Subjects/Keywords: Physiology; Cellular biology;

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APA (6th Edition):

Manne, B. K. (2015). CLEC-2 SIGNAL TRANSDUCTION IN PLATELET ACTIVATION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,340495

Chicago Manual of Style (16th Edition):

Manne, Bhanu Kanth. “CLEC-2 SIGNAL TRANSDUCTION IN PLATELET ACTIVATION.” 2015. Doctoral Dissertation, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,340495.

MLA Handbook (7th Edition):

Manne, Bhanu Kanth. “CLEC-2 SIGNAL TRANSDUCTION IN PLATELET ACTIVATION.” 2015. Web. 06 Jun 2020.

Vancouver:

Manne BK. CLEC-2 SIGNAL TRANSDUCTION IN PLATELET ACTIVATION. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,340495.

Council of Science Editors:

Manne BK. CLEC-2 SIGNAL TRANSDUCTION IN PLATELET ACTIVATION. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,340495


Temple University

26. Chang, Jen-Kuan. The Biochemical Basis of The miR-21 Expression by The Mu-Opioid Receptor.

Degree: PhD, 2015, Temple University

Molecular Biology and Genetics

Opioid receptors are members of the superfamily of seven transmembrane G protein-coupled receptors (GPCRs) which share several structural and functional characteristics.… (more)

Subjects/Keywords: Molecular biology; Genetics; Immunology;

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APA (6th Edition):

Chang, J. (2015). The Biochemical Basis of The miR-21 Expression by The Mu-Opioid Receptor. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,352105

Chicago Manual of Style (16th Edition):

Chang, Jen-Kuan. “The Biochemical Basis of The miR-21 Expression by The Mu-Opioid Receptor.” 2015. Doctoral Dissertation, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,352105.

MLA Handbook (7th Edition):

Chang, Jen-Kuan. “The Biochemical Basis of The miR-21 Expression by The Mu-Opioid Receptor.” 2015. Web. 06 Jun 2020.

Vancouver:

Chang J. The Biochemical Basis of The miR-21 Expression by The Mu-Opioid Receptor. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,352105.

Council of Science Editors:

Chang J. The Biochemical Basis of The miR-21 Expression by The Mu-Opioid Receptor. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,352105


Temple University

27. Tashovski, Ivan. BAG6 as a Novel HIV-1 Host Factor.

Degree: PhD, 2016, Temple University

Microbiology and Immunology

The human immunodeficiency virus type-1 (HIV-1) is the major etiological agent of acquired immunodeficiency syndrome (AIDS), the cause of over 30 million… (more)

Subjects/Keywords: Microbiology; Virology; Molecular biology;

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APA (6th Edition):

Tashovski, I. (2016). BAG6 as a Novel HIV-1 Host Factor. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,420479

Chicago Manual of Style (16th Edition):

Tashovski, Ivan. “BAG6 as a Novel HIV-1 Host Factor.” 2016. Doctoral Dissertation, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,420479.

MLA Handbook (7th Edition):

Tashovski, Ivan. “BAG6 as a Novel HIV-1 Host Factor.” 2016. Web. 06 Jun 2020.

Vancouver:

Tashovski I. BAG6 as a Novel HIV-1 Host Factor. [Internet] [Doctoral dissertation]. Temple University; 2016. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,420479.

Council of Science Editors:

Tashovski I. BAG6 as a Novel HIV-1 Host Factor. [Doctoral Dissertation]. Temple University; 2016. Available from: http://digital.library.temple.edu/u?/p245801coll10,420479


Temple University

28. Whitfield, Fatima. Gamma-Delta T-Cells in Patients with Ovarian Carcinoma.

Degree: PhD, 2008, Temple University

Microbiology and Immunology

Ovarian cancer is the fifth most common cause of death from all cancers among women in the Western world and the most… (more)

Subjects/Keywords: Health Sciences, Immunology; Health Sciences, Oncology

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APA (6th Edition):

Whitfield, F. (2008). Gamma-Delta T-Cells in Patients with Ovarian Carcinoma. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,3422

Chicago Manual of Style (16th Edition):

Whitfield, Fatima. “Gamma-Delta T-Cells in Patients with Ovarian Carcinoma.” 2008. Doctoral Dissertation, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,3422.

MLA Handbook (7th Edition):

Whitfield, Fatima. “Gamma-Delta T-Cells in Patients with Ovarian Carcinoma.” 2008. Web. 06 Jun 2020.

Vancouver:

Whitfield F. Gamma-Delta T-Cells in Patients with Ovarian Carcinoma. [Internet] [Doctoral dissertation]. Temple University; 2008. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,3422.

Council of Science Editors:

Whitfield F. Gamma-Delta T-Cells in Patients with Ovarian Carcinoma. [Doctoral Dissertation]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,3422


Temple University

29. Radu, Maria. The role of p27 phosphorylation in mediating atRA sensitivity of ovarian carcinoma cells.

Degree: 2008, Temple University

Microbiology and Immunology

Ph.D.;

All trans retinoic acid (atRA) has been shown to inhibit the growth of CAOV3 ovarian carcinoma cells. This results from arrest… (more)

Subjects/Keywords: Health Sciences, Immunology; Biology, Cell; Health Sciences, Oncology; retinoic acid; p27; phosphorylation; cell cycle; growth arrest

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APA (6th Edition):

Radu, M. (2008). The role of p27 phosphorylation in mediating atRA sensitivity of ovarian carcinoma cells. (Thesis). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,5459

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Radu, Maria. “The role of p27 phosphorylation in mediating atRA sensitivity of ovarian carcinoma cells.” 2008. Thesis, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,5459.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Radu, Maria. “The role of p27 phosphorylation in mediating atRA sensitivity of ovarian carcinoma cells.” 2008. Web. 06 Jun 2020.

Vancouver:

Radu M. The role of p27 phosphorylation in mediating atRA sensitivity of ovarian carcinoma cells. [Internet] [Thesis]. Temple University; 2008. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,5459.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Radu M. The role of p27 phosphorylation in mediating atRA sensitivity of ovarian carcinoma cells. [Thesis]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,5459

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

30. Chawla, Rachna. Role of CDK4 in Development and Cancer.

Degree: PhD, 2008, Temple University

Molecular Biology and Genetics

The mammalian cell cycle is divided into four distinct phases: G1, S, G2, and M. The transition from G1 to S… (more)

Subjects/Keywords: Biology; Molecular

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chawla, R. (2008). Role of CDK4 in Development and Cancer. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,7450

Chicago Manual of Style (16th Edition):

Chawla, Rachna. “Role of CDK4 in Development and Cancer.” 2008. Doctoral Dissertation, Temple University. Accessed June 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,7450.

MLA Handbook (7th Edition):

Chawla, Rachna. “Role of CDK4 in Development and Cancer.” 2008. Web. 06 Jun 2020.

Vancouver:

Chawla R. Role of CDK4 in Development and Cancer. [Internet] [Doctoral dissertation]. Temple University; 2008. [cited 2020 Jun 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,7450.

Council of Science Editors:

Chawla R. Role of CDK4 in Development and Cancer. [Doctoral Dissertation]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,7450

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