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You searched for +publisher:"Temple University" +contributor:("Soprano, Kenneth J."). Showing records 1 – 11 of 11 total matches.

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Temple University

1. Tutton, Stephen Paul. Repression of E2F-4 Mediated Transcription and Growth by Hypophosphorylated pRb2/p130 in Human Prostate Cancer Cells.

Degree: PhD, 2011, Temple University

Biology

A fundamental property of tumor cells is the ability to proliferate in an unscheduled manner, circumventing the cues which would otherwise prevent entry into,… (more)

Subjects/Keywords: Biology

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APA (6th Edition):

Tutton, S. P. (2011). Repression of E2F-4 Mediated Transcription and Growth by Hypophosphorylated pRb2/p130 in Human Prostate Cancer Cells. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,138637

Chicago Manual of Style (16th Edition):

Tutton, Stephen Paul. “Repression of E2F-4 Mediated Transcription and Growth by Hypophosphorylated pRb2/p130 in Human Prostate Cancer Cells.” 2011. Doctoral Dissertation, Temple University. Accessed October 14, 2019. http://digital.library.temple.edu/u?/p245801coll10,138637.

MLA Handbook (7th Edition):

Tutton, Stephen Paul. “Repression of E2F-4 Mediated Transcription and Growth by Hypophosphorylated pRb2/p130 in Human Prostate Cancer Cells.” 2011. Web. 14 Oct 2019.

Vancouver:

Tutton SP. Repression of E2F-4 Mediated Transcription and Growth by Hypophosphorylated pRb2/p130 in Human Prostate Cancer Cells. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2019 Oct 14]. Available from: http://digital.library.temple.edu/u?/p245801coll10,138637.

Council of Science Editors:

Tutton SP. Repression of E2F-4 Mediated Transcription and Growth by Hypophosphorylated pRb2/p130 in Human Prostate Cancer Cells. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,138637


Temple University

2. Zhao, Jianhua. HACE1, a Novel Repressor of RAR Transcriptional Activity.

Degree: PhD, 2009, Temple University

Microbiology and Immunology

The biological activities of retinoic acid (RA) and its synthetic analogues are mediated through nuclear receptors, termed retinoic acid receptors (RARs) and… (more)

Subjects/Keywords: Biology, Molecular; HACE1; RAR; Repressor

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APA (6th Edition):

Zhao, J. (2009). HACE1, a Novel Repressor of RAR Transcriptional Activity. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,27963

Chicago Manual of Style (16th Edition):

Zhao, Jianhua. “HACE1, a Novel Repressor of RAR Transcriptional Activity.” 2009. Doctoral Dissertation, Temple University. Accessed October 14, 2019. http://digital.library.temple.edu/u?/p245801coll10,27963.

MLA Handbook (7th Edition):

Zhao, Jianhua. “HACE1, a Novel Repressor of RAR Transcriptional Activity.” 2009. Web. 14 Oct 2019.

Vancouver:

Zhao J. HACE1, a Novel Repressor of RAR Transcriptional Activity. [Internet] [Doctoral dissertation]. Temple University; 2009. [cited 2019 Oct 14]. Available from: http://digital.library.temple.edu/u?/p245801coll10,27963.

Council of Science Editors:

Zhao J. HACE1, a Novel Repressor of RAR Transcriptional Activity. [Doctoral Dissertation]. Temple University; 2009. Available from: http://digital.library.temple.edu/u?/p245801coll10,27963


Temple University

3. Sirisani, Evelyn. The Role of Regulatory Genes in Mediating Growth Arrest by all-trans Retinoic Acid in Ovarian Carcinoma Cell Lines.

Degree: PhD, 2012, Temple University

Microbiology and Immunology

All-trans retinoic acid (atRA) mediated growth inhibition results in the arrest of the cell cycle during the G1 phase in CAOV3 cells… (more)

Subjects/Keywords: Microbiology; Immunology; Molecular biology; all-trans retinoic acid; full length HOXA1; HOXA1; HOXB4; p16INK4a; truncated form HOXA1

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APA (6th Edition):

Sirisani, E. (2012). The Role of Regulatory Genes in Mediating Growth Arrest by all-trans Retinoic Acid in Ovarian Carcinoma Cell Lines. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,210886

Chicago Manual of Style (16th Edition):

Sirisani, Evelyn. “The Role of Regulatory Genes in Mediating Growth Arrest by all-trans Retinoic Acid in Ovarian Carcinoma Cell Lines.” 2012. Doctoral Dissertation, Temple University. Accessed October 14, 2019. http://digital.library.temple.edu/u?/p245801coll10,210886.

MLA Handbook (7th Edition):

Sirisani, Evelyn. “The Role of Regulatory Genes in Mediating Growth Arrest by all-trans Retinoic Acid in Ovarian Carcinoma Cell Lines.” 2012. Web. 14 Oct 2019.

Vancouver:

Sirisani E. The Role of Regulatory Genes in Mediating Growth Arrest by all-trans Retinoic Acid in Ovarian Carcinoma Cell Lines. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2019 Oct 14]. Available from: http://digital.library.temple.edu/u?/p245801coll10,210886.

Council of Science Editors:

Sirisani E. The Role of Regulatory Genes in Mediating Growth Arrest by all-trans Retinoic Acid in Ovarian Carcinoma Cell Lines. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,210886


Temple University

4. Bolton, Elisabeth Spring. Genomic Instability Originates From Leukemia Stem Cells In a Mouse Model of CML-CP.

Degree: PhD, 2013, Temple University

Microbiology and Immunology

In chronic myelogenous leukemia (CML), activation of BCR-ABL, the product of the bcr-abl chimeric gene, leads to constitutive activation of pathways that… (more)

Subjects/Keywords: Biology; Molecular biology; Immunology;

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APA (6th Edition):

Bolton, E. S. (2013). Genomic Instability Originates From Leukemia Stem Cells In a Mouse Model of CML-CP. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,234916

Chicago Manual of Style (16th Edition):

Bolton, Elisabeth Spring. “Genomic Instability Originates From Leukemia Stem Cells In a Mouse Model of CML-CP.” 2013. Doctoral Dissertation, Temple University. Accessed October 14, 2019. http://digital.library.temple.edu/u?/p245801coll10,234916.

MLA Handbook (7th Edition):

Bolton, Elisabeth Spring. “Genomic Instability Originates From Leukemia Stem Cells In a Mouse Model of CML-CP.” 2013. Web. 14 Oct 2019.

Vancouver:

Bolton ES. Genomic Instability Originates From Leukemia Stem Cells In a Mouse Model of CML-CP. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2019 Oct 14]. Available from: http://digital.library.temple.edu/u?/p245801coll10,234916.

Council of Science Editors:

Bolton ES. Genomic Instability Originates From Leukemia Stem Cells In a Mouse Model of CML-CP. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,234916


Temple University

5. Wang, Fang. The Role of Acinus in Retinoic Acid Signaling Pathway.

Degree: PhD, 2014, Temple University

Biochemistry

Retinoic acid receptor (RAR), a member of the steroid/thyroid hormone nuclear receptor superfamily, functions as a RA-dependent transcription activator bound to the RA response… (more)

Subjects/Keywords: Biochemistry;

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APA (6th Edition):

Wang, F. (2014). The Role of Acinus in Retinoic Acid Signaling Pathway. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,277479

Chicago Manual of Style (16th Edition):

Wang, Fang. “The Role of Acinus in Retinoic Acid Signaling Pathway.” 2014. Doctoral Dissertation, Temple University. Accessed October 14, 2019. http://digital.library.temple.edu/u?/p245801coll10,277479.

MLA Handbook (7th Edition):

Wang, Fang. “The Role of Acinus in Retinoic Acid Signaling Pathway.” 2014. Web. 14 Oct 2019.

Vancouver:

Wang F. The Role of Acinus in Retinoic Acid Signaling Pathway. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Oct 14]. Available from: http://digital.library.temple.edu/u?/p245801coll10,277479.

Council of Science Editors:

Wang F. The Role of Acinus in Retinoic Acid Signaling Pathway. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,277479


Temple University

6. Firrman, Jenni Ann. ENHANCEMENT OF hFVIII ACTIVITY THROUGH LC MODIFICATIONS FOR GENE THERAPY OF HEMOPHILIA A.

Degree: PhD, 2015, Temple University

Microbiology and Immunology

Gene therapy for Hemophilia A (HA) using the recombinant Adeno-associated virus (rAAV) offers an alternative to classic treatment, which consists of FVIII… (more)

Subjects/Keywords: Microbiology; Immunology; Genetics;

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APA (6th Edition):

Firrman, J. A. (2015). ENHANCEMENT OF hFVIII ACTIVITY THROUGH LC MODIFICATIONS FOR GENE THERAPY OF HEMOPHILIA A. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,335863

Chicago Manual of Style (16th Edition):

Firrman, Jenni Ann. “ENHANCEMENT OF hFVIII ACTIVITY THROUGH LC MODIFICATIONS FOR GENE THERAPY OF HEMOPHILIA A.” 2015. Doctoral Dissertation, Temple University. Accessed October 14, 2019. http://digital.library.temple.edu/u?/p245801coll10,335863.

MLA Handbook (7th Edition):

Firrman, Jenni Ann. “ENHANCEMENT OF hFVIII ACTIVITY THROUGH LC MODIFICATIONS FOR GENE THERAPY OF HEMOPHILIA A.” 2015. Web. 14 Oct 2019.

Vancouver:

Firrman JA. ENHANCEMENT OF hFVIII ACTIVITY THROUGH LC MODIFICATIONS FOR GENE THERAPY OF HEMOPHILIA A. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Oct 14]. Available from: http://digital.library.temple.edu/u?/p245801coll10,335863.

Council of Science Editors:

Firrman JA. ENHANCEMENT OF hFVIII ACTIVITY THROUGH LC MODIFICATIONS FOR GENE THERAPY OF HEMOPHILIA A. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,335863


Temple University

7. Radu, Maria. The role of p27 phosphorylation in mediating atRA sensitivity of ovarian carcinoma cells.

Degree: 2008, Temple University

Microbiology and Immunology

Ph.D.;

All trans retinoic acid (atRA) has been shown to inhibit the growth of CAOV3 ovarian carcinoma cells. This results from arrest… (more)

Subjects/Keywords: Health Sciences, Immunology; Biology, Cell; Health Sciences, Oncology; retinoic acid; p27; phosphorylation; cell cycle; growth arrest

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APA (6th Edition):

Radu, M. (2008). The role of p27 phosphorylation in mediating atRA sensitivity of ovarian carcinoma cells. (Thesis). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,5459

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Radu, Maria. “The role of p27 phosphorylation in mediating atRA sensitivity of ovarian carcinoma cells.” 2008. Thesis, Temple University. Accessed October 14, 2019. http://digital.library.temple.edu/u?/p245801coll10,5459.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Radu, Maria. “The role of p27 phosphorylation in mediating atRA sensitivity of ovarian carcinoma cells.” 2008. Web. 14 Oct 2019.

Vancouver:

Radu M. The role of p27 phosphorylation in mediating atRA sensitivity of ovarian carcinoma cells. [Internet] [Thesis]. Temple University; 2008. [cited 2019 Oct 14]. Available from: http://digital.library.temple.edu/u?/p245801coll10,5459.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Radu M. The role of p27 phosphorylation in mediating atRA sensitivity of ovarian carcinoma cells. [Thesis]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,5459

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

8. Hodgdon, Hilary Bridgette. Child Maltreatment and Aggression: The Mediating Role of Moral Disengagement, Emotion Regulation, and Emotional Callousness among Juvenile Offenders.

Degree: PhD, 2009, Temple University

Psychology

Child maltreatment has been linked consistently to the development of aggressive behavior. However, not all maltreated youth later demonstrate increased aggression. The present study… (more)

Subjects/Keywords: Psychology, Clinical; adolescents; aggression; child maltreatment; child welfare; crossover youth; juvenile delinquency

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APA (6th Edition):

Hodgdon, H. B. (2009). Child Maltreatment and Aggression: The Mediating Role of Moral Disengagement, Emotion Regulation, and Emotional Callousness among Juvenile Offenders. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,52200

Chicago Manual of Style (16th Edition):

Hodgdon, Hilary Bridgette. “Child Maltreatment and Aggression: The Mediating Role of Moral Disengagement, Emotion Regulation, and Emotional Callousness among Juvenile Offenders.” 2009. Doctoral Dissertation, Temple University. Accessed October 14, 2019. http://digital.library.temple.edu/u?/p245801coll10,52200.

MLA Handbook (7th Edition):

Hodgdon, Hilary Bridgette. “Child Maltreatment and Aggression: The Mediating Role of Moral Disengagement, Emotion Regulation, and Emotional Callousness among Juvenile Offenders.” 2009. Web. 14 Oct 2019.

Vancouver:

Hodgdon HB. Child Maltreatment and Aggression: The Mediating Role of Moral Disengagement, Emotion Regulation, and Emotional Callousness among Juvenile Offenders. [Internet] [Doctoral dissertation]. Temple University; 2009. [cited 2019 Oct 14]. Available from: http://digital.library.temple.edu/u?/p245801coll10,52200.

Council of Science Editors:

Hodgdon HB. Child Maltreatment and Aggression: The Mediating Role of Moral Disengagement, Emotion Regulation, and Emotional Callousness among Juvenile Offenders. [Doctoral Dissertation]. Temple University; 2009. Available from: http://digital.library.temple.edu/u?/p245801coll10,52200


Temple University

9. Jayadeva, Girish. B55alpha modulates the phosphorylation status of the pRb-related p107 and p130 proteins.

Degree: PhD, 2010, Temple University

Molecular Biology and Genetics

The retinoblastoma family of phosphoproteins consisting of the retinoblastoma protein (pRB) and the two structurally related proteins p130 and p107 play… (more)

Subjects/Keywords: Biology, Molecular; B55; p107; p130; pocket; PP2A; pRb

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APA (6th Edition):

Jayadeva, G. (2010). B55alpha modulates the phosphorylation status of the pRb-related p107 and p130 proteins. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,64785

Chicago Manual of Style (16th Edition):

Jayadeva, Girish. “B55alpha modulates the phosphorylation status of the pRb-related p107 and p130 proteins.” 2010. Doctoral Dissertation, Temple University. Accessed October 14, 2019. http://digital.library.temple.edu/u?/p245801coll10,64785.

MLA Handbook (7th Edition):

Jayadeva, Girish. “B55alpha modulates the phosphorylation status of the pRb-related p107 and p130 proteins.” 2010. Web. 14 Oct 2019.

Vancouver:

Jayadeva G. B55alpha modulates the phosphorylation status of the pRb-related p107 and p130 proteins. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2019 Oct 14]. Available from: http://digital.library.temple.edu/u?/p245801coll10,64785.

Council of Science Editors:

Jayadeva G. B55alpha modulates the phosphorylation status of the pRb-related p107 and p130 proteins. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,64785


Temple University

10. Pickens, Brandy S. THE ROLE OF COUP-TFI DURING RETINOIC ACID INDUCED ENDODERMAL DIFFERENTIATION OF P19 CELLS.

Degree: PhD, 2012, Temple University

Biochemistry

ABSTRACT Retinoic acid (RA) is a positive regulator of P19 EC cell differentiation. Pre-B cell leukemia transcription factors (PBXs) act in conjunction with homeobox… (more)

Subjects/Keywords: Biochemistry; COUP-TFI; Endodermal Differentiation; P19 Cells; Retinoic Acid

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APA (6th Edition):

Pickens, B. S. (2012). THE ROLE OF COUP-TFI DURING RETINOIC ACID INDUCED ENDODERMAL DIFFERENTIATION OF P19 CELLS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,196883

Chicago Manual of Style (16th Edition):

Pickens, Brandy S. “THE ROLE OF COUP-TFI DURING RETINOIC ACID INDUCED ENDODERMAL DIFFERENTIATION OF P19 CELLS.” 2012. Doctoral Dissertation, Temple University. Accessed October 14, 2019. http://digital.library.temple.edu/u?/p245801coll10,196883.

MLA Handbook (7th Edition):

Pickens, Brandy S. “THE ROLE OF COUP-TFI DURING RETINOIC ACID INDUCED ENDODERMAL DIFFERENTIATION OF P19 CELLS.” 2012. Web. 14 Oct 2019.

Vancouver:

Pickens BS. THE ROLE OF COUP-TFI DURING RETINOIC ACID INDUCED ENDODERMAL DIFFERENTIATION OF P19 CELLS. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2019 Oct 14]. Available from: http://digital.library.temple.edu/u?/p245801coll10,196883.

Council of Science Editors:

Pickens BS. THE ROLE OF COUP-TFI DURING RETINOIC ACID INDUCED ENDODERMAL DIFFERENTIATION OF P19 CELLS. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,196883


Temple University

11. Zacharakis, Nikolaos. Identification of putative antigens in Systemic Sclerosis utilizing in vivo clonally expanded T cells.

Degree: PhD, 2014, Temple University

Microbiology and Immunology

Systemic sclerosis (SSc) is a chronic autoimmune disease of the connective tissue. Immune system dysregulation, excessive deposition of collagen and microvascular damage… (more)

Subjects/Keywords: Immunology;

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APA (6th Edition):

Zacharakis, N. (2014). Identification of putative antigens in Systemic Sclerosis utilizing in vivo clonally expanded T cells. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,249827

Chicago Manual of Style (16th Edition):

Zacharakis, Nikolaos. “Identification of putative antigens in Systemic Sclerosis utilizing in vivo clonally expanded T cells.” 2014. Doctoral Dissertation, Temple University. Accessed October 14, 2019. http://digital.library.temple.edu/u?/p245801coll10,249827.

MLA Handbook (7th Edition):

Zacharakis, Nikolaos. “Identification of putative antigens in Systemic Sclerosis utilizing in vivo clonally expanded T cells.” 2014. Web. 14 Oct 2019.

Vancouver:

Zacharakis N. Identification of putative antigens in Systemic Sclerosis utilizing in vivo clonally expanded T cells. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Oct 14]. Available from: http://digital.library.temple.edu/u?/p245801coll10,249827.

Council of Science Editors:

Zacharakis N. Identification of putative antigens in Systemic Sclerosis utilizing in vivo clonally expanded T cells. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,249827

.