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You searched for +publisher:"Temple University" +contributor:("Soprano, Dianne R.;"). Showing records 1 – 27 of 27 total matches.

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Temple University

1. Barton, Maria. Lnc-EPCAM AND Lnc-BHLHE41 AS RNA REGULATORS OF BREAST CANCER AND BREAST CANCER PREVENTION.

Degree: PhD, 2017, Temple University

Biochemistry

The objective of this study was to unveil a novel area of gene regulation in breast cancer and breast cancer prevention through the study… (more)

Subjects/Keywords: Cellular biology; Molecular biology; Biochemistry;

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APA (6th Edition):

Barton, M. (2017). Lnc-EPCAM AND Lnc-BHLHE41 AS RNA REGULATORS OF BREAST CANCER AND BREAST CANCER PREVENTION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,424232

Chicago Manual of Style (16th Edition):

Barton, Maria. “Lnc-EPCAM AND Lnc-BHLHE41 AS RNA REGULATORS OF BREAST CANCER AND BREAST CANCER PREVENTION.” 2017. Doctoral Dissertation, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,424232.

MLA Handbook (7th Edition):

Barton, Maria. “Lnc-EPCAM AND Lnc-BHLHE41 AS RNA REGULATORS OF BREAST CANCER AND BREAST CANCER PREVENTION.” 2017. Web. 06 Aug 2020.

Vancouver:

Barton M. Lnc-EPCAM AND Lnc-BHLHE41 AS RNA REGULATORS OF BREAST CANCER AND BREAST CANCER PREVENTION. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,424232.

Council of Science Editors:

Barton M. Lnc-EPCAM AND Lnc-BHLHE41 AS RNA REGULATORS OF BREAST CANCER AND BREAST CANCER PREVENTION. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,424232


Temple University

2. Teets, Bryan Wilson. SF-1, BUT NOT DAX-1, PREVENTS P19 CELLS FROM DIFFERENTIATING TO EITHER TROMA-1 OR TUJ1 POSITIVE CELLS UPON RA-TREATMENT.

Degree: PhD, 2011, Temple University

Biochemistry

Retinoic acid (RA) is critical for embryonic development and cell differentiation. Previous work in our laboratory has shown that blocking the RA-dependent increase in… (more)

Subjects/Keywords: Biochemistry; adrenal; Differentiation; neuronal; P19; PBX; Vitamin A

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APA (6th Edition):

Teets, B. W. (2011). SF-1, BUT NOT DAX-1, PREVENTS P19 CELLS FROM DIFFERENTIATING TO EITHER TROMA-1 OR TUJ1 POSITIVE CELLS UPON RA-TREATMENT. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,124708

Chicago Manual of Style (16th Edition):

Teets, Bryan Wilson. “SF-1, BUT NOT DAX-1, PREVENTS P19 CELLS FROM DIFFERENTIATING TO EITHER TROMA-1 OR TUJ1 POSITIVE CELLS UPON RA-TREATMENT.” 2011. Doctoral Dissertation, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,124708.

MLA Handbook (7th Edition):

Teets, Bryan Wilson. “SF-1, BUT NOT DAX-1, PREVENTS P19 CELLS FROM DIFFERENTIATING TO EITHER TROMA-1 OR TUJ1 POSITIVE CELLS UPON RA-TREATMENT.” 2011. Web. 06 Aug 2020.

Vancouver:

Teets BW. SF-1, BUT NOT DAX-1, PREVENTS P19 CELLS FROM DIFFERENTIATING TO EITHER TROMA-1 OR TUJ1 POSITIVE CELLS UPON RA-TREATMENT. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,124708.

Council of Science Editors:

Teets BW. SF-1, BUT NOT DAX-1, PREVENTS P19 CELLS FROM DIFFERENTIATING TO EITHER TROMA-1 OR TUJ1 POSITIVE CELLS UPON RA-TREATMENT. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,124708


Temple University

3. Doonan, Patrick John. Mitochondrial Calcium Uptake: LETM1 and MICU1 Are Mitochondrial Proteins That Regulate Mitochondrial Calcium Homeostasis and Cellular Bioenergetics.

Degree: PhD, 2012, Temple University

Biochemistry

Mitochondrial calcium (Ca2+) uptake has been studied for over five decades, with crucial insights into its underlying mechanisms enabled by development of the chemi-osmotic… (more)

Subjects/Keywords: Biochemistry; Molecular biology; Calcium; LETM1; MICU1; Mitochondria; Mitochondria Calcium Uniporter; Uniporter

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APA (6th Edition):

Doonan, P. J. (2012). Mitochondrial Calcium Uptake: LETM1 and MICU1 Are Mitochondrial Proteins That Regulate Mitochondrial Calcium Homeostasis and Cellular Bioenergetics. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,214818

Chicago Manual of Style (16th Edition):

Doonan, Patrick John. “Mitochondrial Calcium Uptake: LETM1 and MICU1 Are Mitochondrial Proteins That Regulate Mitochondrial Calcium Homeostasis and Cellular Bioenergetics.” 2012. Doctoral Dissertation, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,214818.

MLA Handbook (7th Edition):

Doonan, Patrick John. “Mitochondrial Calcium Uptake: LETM1 and MICU1 Are Mitochondrial Proteins That Regulate Mitochondrial Calcium Homeostasis and Cellular Bioenergetics.” 2012. Web. 06 Aug 2020.

Vancouver:

Doonan PJ. Mitochondrial Calcium Uptake: LETM1 and MICU1 Are Mitochondrial Proteins That Regulate Mitochondrial Calcium Homeostasis and Cellular Bioenergetics. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,214818.

Council of Science Editors:

Doonan PJ. Mitochondrial Calcium Uptake: LETM1 and MICU1 Are Mitochondrial Proteins That Regulate Mitochondrial Calcium Homeostasis and Cellular Bioenergetics. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,214818


Temple University

4. Mukherjee, Kaushiki. Role Of Gadd45a In BCR-ABL and NRASD12 Driven Leukemia.

Degree: PhD, 2015, Temple University

Biochemistry

BCR-ABL is known as the most common translocation in the myeloproliferative (MPD) disorder chronic myelogenous leukemia (CML); it is the first leukemia to be… (more)

Subjects/Keywords: Oncology; Molecular biology; Cellular biology;

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APA (6th Edition):

Mukherjee, K. (2015). Role Of Gadd45a In BCR-ABL and NRASD12 Driven Leukemia. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,322334

Chicago Manual of Style (16th Edition):

Mukherjee, Kaushiki. “Role Of Gadd45a In BCR-ABL and NRASD12 Driven Leukemia.” 2015. Doctoral Dissertation, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,322334.

MLA Handbook (7th Edition):

Mukherjee, Kaushiki. “Role Of Gadd45a In BCR-ABL and NRASD12 Driven Leukemia.” 2015. Web. 06 Aug 2020.

Vancouver:

Mukherjee K. Role Of Gadd45a In BCR-ABL and NRASD12 Driven Leukemia. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,322334.

Council of Science Editors:

Mukherjee K. Role Of Gadd45a In BCR-ABL and NRASD12 Driven Leukemia. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,322334


Temple University

5. Zhao, Jianhua. HACE1, a Novel Repressor of RAR Transcriptional Activity.

Degree: PhD, 2009, Temple University

Microbiology and Immunology

The biological activities of retinoic acid (RA) and its synthetic analogues are mediated through nuclear receptors, termed retinoic acid receptors (RARs) and… (more)

Subjects/Keywords: Biology, Molecular; HACE1; RAR; Repressor

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APA (6th Edition):

Zhao, J. (2009). HACE1, a Novel Repressor of RAR Transcriptional Activity. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,27963

Chicago Manual of Style (16th Edition):

Zhao, Jianhua. “HACE1, a Novel Repressor of RAR Transcriptional Activity.” 2009. Doctoral Dissertation, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,27963.

MLA Handbook (7th Edition):

Zhao, Jianhua. “HACE1, a Novel Repressor of RAR Transcriptional Activity.” 2009. Web. 06 Aug 2020.

Vancouver:

Zhao J. HACE1, a Novel Repressor of RAR Transcriptional Activity. [Internet] [Doctoral dissertation]. Temple University; 2009. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,27963.

Council of Science Editors:

Zhao J. HACE1, a Novel Repressor of RAR Transcriptional Activity. [Doctoral Dissertation]. Temple University; 2009. Available from: http://digital.library.temple.edu/u?/p245801coll10,27963


Temple University

6. Potireddy, Santhi. TRANSLATIONAL CONTROL OF MATERNAL mRNA POPULATION IN MOUSE EMBRYOS.

Degree: PhD, 2010, Temple University

Biochemistry

Early mammalian development before the oocyte-to-embryo transition is under 'maternal control' from factors deposited in the cytoplasm during oocyte growth, synthesized independent of de… (more)

Subjects/Keywords: Biology, Molecular; cis-regulatory motif; Embryo; Microarray; Mouse

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APA (6th Edition):

Potireddy, S. (2010). TRANSLATIONAL CONTROL OF MATERNAL mRNA POPULATION IN MOUSE EMBRYOS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,67111

Chicago Manual of Style (16th Edition):

Potireddy, Santhi. “TRANSLATIONAL CONTROL OF MATERNAL mRNA POPULATION IN MOUSE EMBRYOS.” 2010. Doctoral Dissertation, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,67111.

MLA Handbook (7th Edition):

Potireddy, Santhi. “TRANSLATIONAL CONTROL OF MATERNAL mRNA POPULATION IN MOUSE EMBRYOS.” 2010. Web. 06 Aug 2020.

Vancouver:

Potireddy S. TRANSLATIONAL CONTROL OF MATERNAL mRNA POPULATION IN MOUSE EMBRYOS. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,67111.

Council of Science Editors:

Potireddy S. TRANSLATIONAL CONTROL OF MATERNAL mRNA POPULATION IN MOUSE EMBRYOS. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,67111


Temple University

7. Cuneo, Anthony. THERAPEUTIC MECHANISMS OF INTERLEUKIN-19 FOR VASCULAR PROLIFERATIVE DISEASES.

Degree: PhD, 2012, Temple University

Molecular and Cellular Physiology

Cardiovascular disease is the leading cause of mortality in the western world. The pro-inflammatory and pro-proliferative etiology of vascular proliferative diseases… (more)

Subjects/Keywords: Biology, Molecular; Biology, General; Biology, Cell; Atherosclerosis; Human antigen R (HuR); Interleukin-19 (IL-19); oxidized LDL (ox-LDL); Vascular Proliferative Diseases; Vascular Smooth Muscle Cells (VSMC)

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APA (6th Edition):

Cuneo, A. (2012). THERAPEUTIC MECHANISMS OF INTERLEUKIN-19 FOR VASCULAR PROLIFERATIVE DISEASES. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,78032

Chicago Manual of Style (16th Edition):

Cuneo, Anthony. “THERAPEUTIC MECHANISMS OF INTERLEUKIN-19 FOR VASCULAR PROLIFERATIVE DISEASES.” 2012. Doctoral Dissertation, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,78032.

MLA Handbook (7th Edition):

Cuneo, Anthony. “THERAPEUTIC MECHANISMS OF INTERLEUKIN-19 FOR VASCULAR PROLIFERATIVE DISEASES.” 2012. Web. 06 Aug 2020.

Vancouver:

Cuneo A. THERAPEUTIC MECHANISMS OF INTERLEUKIN-19 FOR VASCULAR PROLIFERATIVE DISEASES. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,78032.

Council of Science Editors:

Cuneo A. THERAPEUTIC MECHANISMS OF INTERLEUKIN-19 FOR VASCULAR PROLIFERATIVE DISEASES. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,78032


Temple University

8. Moncada Benavides, Camilo Andres. EFFECT OF NICOTINE ON LUNG S-ADENOSYLMETHIONINE AND PNEUMOCYSTIS PNEUMONIA DEVELOPMENT.

Degree: PhD, 2012, Temple University

Biochemistry

Infection with "Pneumocystis" causes a ≥ 99% depletion of plasma S-adenosylmethionine (AdoMet) levels in both "Pneumocystis" pneumonia (PcP) animal models and patients. AdoMet is… (more)

Subjects/Keywords: Biochemistry; Laser Capture Microdissection; Lung; Nicotine; Pneumocystis; Polyamines; S-Adenosylmethionine

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APA (6th Edition):

Moncada Benavides, C. A. (2012). EFFECT OF NICOTINE ON LUNG S-ADENOSYLMETHIONINE AND PNEUMOCYSTIS PNEUMONIA DEVELOPMENT. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,206623

Chicago Manual of Style (16th Edition):

Moncada Benavides, Camilo Andres. “EFFECT OF NICOTINE ON LUNG S-ADENOSYLMETHIONINE AND PNEUMOCYSTIS PNEUMONIA DEVELOPMENT.” 2012. Doctoral Dissertation, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,206623.

MLA Handbook (7th Edition):

Moncada Benavides, Camilo Andres. “EFFECT OF NICOTINE ON LUNG S-ADENOSYLMETHIONINE AND PNEUMOCYSTIS PNEUMONIA DEVELOPMENT.” 2012. Web. 06 Aug 2020.

Vancouver:

Moncada Benavides CA. EFFECT OF NICOTINE ON LUNG S-ADENOSYLMETHIONINE AND PNEUMOCYSTIS PNEUMONIA DEVELOPMENT. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,206623.

Council of Science Editors:

Moncada Benavides CA. EFFECT OF NICOTINE ON LUNG S-ADENOSYLMETHIONINE AND PNEUMOCYSTIS PNEUMONIA DEVELOPMENT. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,206623


Temple University

9. Sirisani, Evelyn. The Role of Regulatory Genes in Mediating Growth Arrest by all-trans Retinoic Acid in Ovarian Carcinoma Cell Lines.

Degree: PhD, 2012, Temple University

Microbiology and Immunology

All-trans retinoic acid (atRA) mediated growth inhibition results in the arrest of the cell cycle during the G1 phase in CAOV3 cells… (more)

Subjects/Keywords: Microbiology; Immunology; Molecular biology; all-trans retinoic acid; full length HOXA1; HOXA1; HOXB4; p16INK4a; truncated form HOXA1

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APA (6th Edition):

Sirisani, E. (2012). The Role of Regulatory Genes in Mediating Growth Arrest by all-trans Retinoic Acid in Ovarian Carcinoma Cell Lines. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,210886

Chicago Manual of Style (16th Edition):

Sirisani, Evelyn. “The Role of Regulatory Genes in Mediating Growth Arrest by all-trans Retinoic Acid in Ovarian Carcinoma Cell Lines.” 2012. Doctoral Dissertation, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,210886.

MLA Handbook (7th Edition):

Sirisani, Evelyn. “The Role of Regulatory Genes in Mediating Growth Arrest by all-trans Retinoic Acid in Ovarian Carcinoma Cell Lines.” 2012. Web. 06 Aug 2020.

Vancouver:

Sirisani E. The Role of Regulatory Genes in Mediating Growth Arrest by all-trans Retinoic Acid in Ovarian Carcinoma Cell Lines. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,210886.

Council of Science Editors:

Sirisani E. The Role of Regulatory Genes in Mediating Growth Arrest by all-trans Retinoic Acid in Ovarian Carcinoma Cell Lines. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,210886


Temple University

10. Ritchie, Michael. TRANSCRIPTIONAL AND MOLECULAR CONTROL OF CALCIUM SIGNALING.

Degree: PhD, 2012, Temple University

Biochemistry

The extensive relationship between modulation of intracellular Ca2+ content and the control of cell proliferation (Boynton et al. 1974; Whitfield et al. 1979; Berridge… (more)

Subjects/Keywords: Biochemistry; Molecular biology; Calcium Signaling; PMCA; STIM1

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APA (6th Edition):

Ritchie, M. (2012). TRANSCRIPTIONAL AND MOLECULAR CONTROL OF CALCIUM SIGNALING. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,211071

Chicago Manual of Style (16th Edition):

Ritchie, Michael. “TRANSCRIPTIONAL AND MOLECULAR CONTROL OF CALCIUM SIGNALING.” 2012. Doctoral Dissertation, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,211071.

MLA Handbook (7th Edition):

Ritchie, Michael. “TRANSCRIPTIONAL AND MOLECULAR CONTROL OF CALCIUM SIGNALING.” 2012. Web. 06 Aug 2020.

Vancouver:

Ritchie M. TRANSCRIPTIONAL AND MOLECULAR CONTROL OF CALCIUM SIGNALING. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,211071.

Council of Science Editors:

Ritchie M. TRANSCRIPTIONAL AND MOLECULAR CONTROL OF CALCIUM SIGNALING. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,211071


Temple University

11. Lunden, Jason Wesley. The serotonergic dorsal raphe nucleus in opiate dependence and stress-induced relapse.

Degree: PhD, 2013, Temple University

Cell Biology

Opioids are used for the clinical treatment of pain, but can lead to tolerance and addiction. In this project we examined the role… (more)

Subjects/Keywords: Neurosciences; Animal behavior; Cellular biology;

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APA (6th Edition):

Lunden, J. W. (2013). The serotonergic dorsal raphe nucleus in opiate dependence and stress-induced relapse. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,228054

Chicago Manual of Style (16th Edition):

Lunden, Jason Wesley. “The serotonergic dorsal raphe nucleus in opiate dependence and stress-induced relapse.” 2013. Doctoral Dissertation, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,228054.

MLA Handbook (7th Edition):

Lunden, Jason Wesley. “The serotonergic dorsal raphe nucleus in opiate dependence and stress-induced relapse.” 2013. Web. 06 Aug 2020.

Vancouver:

Lunden JW. The serotonergic dorsal raphe nucleus in opiate dependence and stress-induced relapse. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,228054.

Council of Science Editors:

Lunden JW. The serotonergic dorsal raphe nucleus in opiate dependence and stress-induced relapse. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,228054


Temple University

12. Lambi, Alex G. The Intricate Role of Connective Tissue Growth Factor (CTGF/CCN2) in Prenatal Osteogenesis: A Heretofore Oversimplified Dogma of the CCN Field.

Degree: PhD, 2015, Temple University

Cell Biology

Connective tissue growth factor (CTGF/CCN2) is axiomatically necessary for proper skeletal development and function. We need not look further than the studies that… (more)

Subjects/Keywords: Cellular biology; Developmental biology;

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APA (6th Edition):

Lambi, A. G. (2015). The Intricate Role of Connective Tissue Growth Factor (CTGF/CCN2) in Prenatal Osteogenesis: A Heretofore Oversimplified Dogma of the CCN Field. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,233693

Chicago Manual of Style (16th Edition):

Lambi, Alex G. “The Intricate Role of Connective Tissue Growth Factor (CTGF/CCN2) in Prenatal Osteogenesis: A Heretofore Oversimplified Dogma of the CCN Field.” 2015. Doctoral Dissertation, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,233693.

MLA Handbook (7th Edition):

Lambi, Alex G. “The Intricate Role of Connective Tissue Growth Factor (CTGF/CCN2) in Prenatal Osteogenesis: A Heretofore Oversimplified Dogma of the CCN Field.” 2015. Web. 06 Aug 2020.

Vancouver:

Lambi AG. The Intricate Role of Connective Tissue Growth Factor (CTGF/CCN2) in Prenatal Osteogenesis: A Heretofore Oversimplified Dogma of the CCN Field. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,233693.

Council of Science Editors:

Lambi AG. The Intricate Role of Connective Tissue Growth Factor (CTGF/CCN2) in Prenatal Osteogenesis: A Heretofore Oversimplified Dogma of the CCN Field. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,233693


Temple University

13. Korostowski, Lisa. Transcript Regulation within the Kcnq1 Domain.

Degree: PhD, 2012, Temple University

Molecular Biology and Genetics

Epigenetics was a term first coined to understand how cells with the same genetic make up can differentiate into various cell… (more)

Subjects/Keywords: Molecular biology; Genetics;

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APA (6th Edition):

Korostowski, L. (2012). Transcript Regulation within the Kcnq1 Domain. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,235191

Chicago Manual of Style (16th Edition):

Korostowski, Lisa. “Transcript Regulation within the Kcnq1 Domain.” 2012. Doctoral Dissertation, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,235191.

MLA Handbook (7th Edition):

Korostowski, Lisa. “Transcript Regulation within the Kcnq1 Domain.” 2012. Web. 06 Aug 2020.

Vancouver:

Korostowski L. Transcript Regulation within the Kcnq1 Domain. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,235191.

Council of Science Editors:

Korostowski L. Transcript Regulation within the Kcnq1 Domain. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,235191


Temple University

14. Ellison, Stephen Patrick. THE EFFECTS OF INTERLEUKIN-19 ON ATTENUATION OF THE VASCULAR RESPONSE TO INJURY.

Degree: PhD, 2015, Temple University

Physiology

BACKGROUND: Despite aggressive dietary modification, lipid lowering medications, and other medical therapy, vascular proliferative diseases continue to account for 50% of all mortality in… (more)

Subjects/Keywords: Physiology;

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APA (6th Edition):

Ellison, S. P. (2015). THE EFFECTS OF INTERLEUKIN-19 ON ATTENUATION OF THE VASCULAR RESPONSE TO INJURY. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,253270

Chicago Manual of Style (16th Edition):

Ellison, Stephen Patrick. “THE EFFECTS OF INTERLEUKIN-19 ON ATTENUATION OF THE VASCULAR RESPONSE TO INJURY.” 2015. Doctoral Dissertation, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,253270.

MLA Handbook (7th Edition):

Ellison, Stephen Patrick. “THE EFFECTS OF INTERLEUKIN-19 ON ATTENUATION OF THE VASCULAR RESPONSE TO INJURY.” 2015. Web. 06 Aug 2020.

Vancouver:

Ellison SP. THE EFFECTS OF INTERLEUKIN-19 ON ATTENUATION OF THE VASCULAR RESPONSE TO INJURY. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,253270.

Council of Science Editors:

Ellison SP. THE EFFECTS OF INTERLEUKIN-19 ON ATTENUATION OF THE VASCULAR RESPONSE TO INJURY. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,253270


Temple University

15. Romasko, Edward Joseph. Translational Control of Maternal mRNA in Mouse Oocytes.

Degree: PhD, 2014, Temple University

Molecular Biology and Genetics

In contrast to other species, localized maternal mRNAs are not believed to be prominent features of mammalian oocytes. Due to the… (more)

Subjects/Keywords: Molecular biology; Genetics; Developmental biology;

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APA (6th Edition):

Romasko, E. J. (2014). Translational Control of Maternal mRNA in Mouse Oocytes. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,273206

Chicago Manual of Style (16th Edition):

Romasko, Edward Joseph. “Translational Control of Maternal mRNA in Mouse Oocytes.” 2014. Doctoral Dissertation, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,273206.

MLA Handbook (7th Edition):

Romasko, Edward Joseph. “Translational Control of Maternal mRNA in Mouse Oocytes.” 2014. Web. 06 Aug 2020.

Vancouver:

Romasko EJ. Translational Control of Maternal mRNA in Mouse Oocytes. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,273206.

Council of Science Editors:

Romasko EJ. Translational Control of Maternal mRNA in Mouse Oocytes. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,273206


Temple University

16. Rapsinski, Glenn James. Immune Recognition of S. Typhimurium Biofilms via Amyloids and Extracellular DNA.

Degree: PhD, 2016, Temple University

Microbiology and Immunology

Salmonella enterica serovar Typhimurium is an important cause of gastroenteritis in the United States and the developing world. Biofilm growth is an… (more)

Subjects/Keywords: Microbiology; Immunology;

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APA (6th Edition):

Rapsinski, G. J. (2016). Immune Recognition of S. Typhimurium Biofilms via Amyloids and Extracellular DNA. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,273605

Chicago Manual of Style (16th Edition):

Rapsinski, Glenn James. “Immune Recognition of S. Typhimurium Biofilms via Amyloids and Extracellular DNA.” 2016. Doctoral Dissertation, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,273605.

MLA Handbook (7th Edition):

Rapsinski, Glenn James. “Immune Recognition of S. Typhimurium Biofilms via Amyloids and Extracellular DNA.” 2016. Web. 06 Aug 2020.

Vancouver:

Rapsinski GJ. Immune Recognition of S. Typhimurium Biofilms via Amyloids and Extracellular DNA. [Internet] [Doctoral dissertation]. Temple University; 2016. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,273605.

Council of Science Editors:

Rapsinski GJ. Immune Recognition of S. Typhimurium Biofilms via Amyloids and Extracellular DNA. [Doctoral Dissertation]. Temple University; 2016. Available from: http://digital.library.temple.edu/u?/p245801coll10,273605


Temple University

17. Wang, Fang. The Role of Acinus in Retinoic Acid Signaling Pathway.

Degree: PhD, 2014, Temple University

Biochemistry

Retinoic acid receptor (RAR), a member of the steroid/thyroid hormone nuclear receptor superfamily, functions as a RA-dependent transcription activator bound to the RA response… (more)

Subjects/Keywords: Biochemistry;

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APA (6th Edition):

Wang, F. (2014). The Role of Acinus in Retinoic Acid Signaling Pathway. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,277479

Chicago Manual of Style (16th Edition):

Wang, Fang. “The Role of Acinus in Retinoic Acid Signaling Pathway.” 2014. Doctoral Dissertation, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,277479.

MLA Handbook (7th Edition):

Wang, Fang. “The Role of Acinus in Retinoic Acid Signaling Pathway.” 2014. Web. 06 Aug 2020.

Vancouver:

Wang F. The Role of Acinus in Retinoic Acid Signaling Pathway. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,277479.

Council of Science Editors:

Wang F. The Role of Acinus in Retinoic Acid Signaling Pathway. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,277479


Temple University

18. Daswani, Varsha. Fluorescence and aggregation properties of the anti-cancer drug, CA4P, in archaeal liposomes.

Degree: PhD, 2015, Temple University

Biomedical Sciences

Combretastatin A4 phosphate (CA4P) is a potent vascular disrupting agent utilized in the treatment of cancer. The observed rapid vascular shutdown post administration… (more)

Subjects/Keywords: Biochemistry; Biophysics; Chemistry;

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APA (6th Edition):

Daswani, V. (2015). Fluorescence and aggregation properties of the anti-cancer drug, CA4P, in archaeal liposomes. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,334013

Chicago Manual of Style (16th Edition):

Daswani, Varsha. “Fluorescence and aggregation properties of the anti-cancer drug, CA4P, in archaeal liposomes.” 2015. Doctoral Dissertation, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,334013.

MLA Handbook (7th Edition):

Daswani, Varsha. “Fluorescence and aggregation properties of the anti-cancer drug, CA4P, in archaeal liposomes.” 2015. Web. 06 Aug 2020.

Vancouver:

Daswani V. Fluorescence and aggregation properties of the anti-cancer drug, CA4P, in archaeal liposomes. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,334013.

Council of Science Editors:

Daswani V. Fluorescence and aggregation properties of the anti-cancer drug, CA4P, in archaeal liposomes. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,334013


Temple University

19. Srivastava, Isha Narain. The Role of Mammalian Target of Rapamycin (mTOR) in a Mouse Model of Cerebral Palsy.

Degree: PhD, 2017, Temple University

Biomedical Neuroscience

Background and Purpose –The mammalian target of rapamycin (mTOR) pathway has been implicated in cellular responses to hypoxia and inflammation. Cerebral palsy (CP)… (more)

Subjects/Keywords: Neurosciences;

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APA (6th Edition):

Srivastava, I. N. (2017). The Role of Mammalian Target of Rapamycin (mTOR) in a Mouse Model of Cerebral Palsy. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,346472

Chicago Manual of Style (16th Edition):

Srivastava, Isha Narain. “The Role of Mammalian Target of Rapamycin (mTOR) in a Mouse Model of Cerebral Palsy.” 2017. Doctoral Dissertation, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,346472.

MLA Handbook (7th Edition):

Srivastava, Isha Narain. “The Role of Mammalian Target of Rapamycin (mTOR) in a Mouse Model of Cerebral Palsy.” 2017. Web. 06 Aug 2020.

Vancouver:

Srivastava IN. The Role of Mammalian Target of Rapamycin (mTOR) in a Mouse Model of Cerebral Palsy. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,346472.

Council of Science Editors:

Srivastava IN. The Role of Mammalian Target of Rapamycin (mTOR) in a Mouse Model of Cerebral Palsy. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,346472


Temple University

20. Feather, Danielle. GLT-1 dysfunction as an underlying cause of cerebral palsy.

Degree: PhD, 2016, Temple University

Biochemistry

Cerebral Palsy (CP) is an umbrella term that describes abnormal motor movements that is caused by damage in the brain and, depending on the… (more)

Subjects/Keywords: Neurosciences; Biology

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APA (6th Edition):

Feather, D. (2016). GLT-1 dysfunction as an underlying cause of cerebral palsy. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,409354

Chicago Manual of Style (16th Edition):

Feather, Danielle. “GLT-1 dysfunction as an underlying cause of cerebral palsy.” 2016. Doctoral Dissertation, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,409354.

MLA Handbook (7th Edition):

Feather, Danielle. “GLT-1 dysfunction as an underlying cause of cerebral palsy.” 2016. Web. 06 Aug 2020.

Vancouver:

Feather D. GLT-1 dysfunction as an underlying cause of cerebral palsy. [Internet] [Doctoral dissertation]. Temple University; 2016. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,409354.

Council of Science Editors:

Feather D. GLT-1 dysfunction as an underlying cause of cerebral palsy. [Doctoral Dissertation]. Temple University; 2016. Available from: http://digital.library.temple.edu/u?/p245801coll10,409354


Temple University

21. Radu, Maria. The role of p27 phosphorylation in mediating atRA sensitivity of ovarian carcinoma cells.

Degree: 2008, Temple University

Microbiology and Immunology

Ph.D.;

All trans retinoic acid (atRA) has been shown to inhibit the growth of CAOV3 ovarian carcinoma cells. This results from arrest… (more)

Subjects/Keywords: Health Sciences, Immunology; Biology, Cell; Health Sciences, Oncology; retinoic acid; p27; phosphorylation; cell cycle; growth arrest

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APA (6th Edition):

Radu, M. (2008). The role of p27 phosphorylation in mediating atRA sensitivity of ovarian carcinoma cells. (Thesis). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,5459

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Radu, Maria. “The role of p27 phosphorylation in mediating atRA sensitivity of ovarian carcinoma cells.” 2008. Thesis, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,5459.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Radu, Maria. “The role of p27 phosphorylation in mediating atRA sensitivity of ovarian carcinoma cells.” 2008. Web. 06 Aug 2020.

Vancouver:

Radu M. The role of p27 phosphorylation in mediating atRA sensitivity of ovarian carcinoma cells. [Internet] [Thesis]. Temple University; 2008. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,5459.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Radu M. The role of p27 phosphorylation in mediating atRA sensitivity of ovarian carcinoma cells. [Thesis]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,5459

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

22. Collura, Kaitlin Marie. Palmitoylation-Dependent Regulation of the DLK/JNK/cJun and the GP130/JAK/STAT Retrograde Signaling Pathways.

Degree: PhD, 2015, Temple University

Biomedical Sciences

Palmitoylation is the post-translational addition of the 16-carbon fatty acid palmitate to protein cysteine residues. This process is best known for its roles… (more)

Subjects/Keywords: Neurosciences

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APA (6th Edition):

Collura, K. M. (2015). Palmitoylation-Dependent Regulation of the DLK/JNK/cJun and the GP130/JAK/STAT Retrograde Signaling Pathways. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,426710

Chicago Manual of Style (16th Edition):

Collura, Kaitlin Marie. “Palmitoylation-Dependent Regulation of the DLK/JNK/cJun and the GP130/JAK/STAT Retrograde Signaling Pathways.” 2015. Doctoral Dissertation, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,426710.

MLA Handbook (7th Edition):

Collura, Kaitlin Marie. “Palmitoylation-Dependent Regulation of the DLK/JNK/cJun and the GP130/JAK/STAT Retrograde Signaling Pathways.” 2015. Web. 06 Aug 2020.

Vancouver:

Collura KM. Palmitoylation-Dependent Regulation of the DLK/JNK/cJun and the GP130/JAK/STAT Retrograde Signaling Pathways. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,426710.

Council of Science Editors:

Collura KM. Palmitoylation-Dependent Regulation of the DLK/JNK/cJun and the GP130/JAK/STAT Retrograde Signaling Pathways. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,426710


Temple University

23. Srivastava, Isha Narain. The Role of Mammalian Target of Rapamycin (mTOR) in a Mouse Model of Cerebral Palsy.

Degree: PhD, 2017, Temple University

Biomedical Neuroscience

Background and Purpose –The mammalian target of rapamycin (mTOR) pathway has been implicated in cellular responses to hypoxia and inflammation. Cerebral palsy (CP)… (more)

Subjects/Keywords: Neurosciences;

Record DetailsSimilar RecordsGoogle PlusoneFacebookTwitterCiteULikeMendeleyreddit

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Srivastava, I. N. (2017). The Role of Mammalian Target of Rapamycin (mTOR) in a Mouse Model of Cerebral Palsy. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,446570

Chicago Manual of Style (16th Edition):

Srivastava, Isha Narain. “The Role of Mammalian Target of Rapamycin (mTOR) in a Mouse Model of Cerebral Palsy.” 2017. Doctoral Dissertation, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,446570.

MLA Handbook (7th Edition):

Srivastava, Isha Narain. “The Role of Mammalian Target of Rapamycin (mTOR) in a Mouse Model of Cerebral Palsy.” 2017. Web. 06 Aug 2020.

Vancouver:

Srivastava IN. The Role of Mammalian Target of Rapamycin (mTOR) in a Mouse Model of Cerebral Palsy. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,446570.

Council of Science Editors:

Srivastava IN. The Role of Mammalian Target of Rapamycin (mTOR) in a Mouse Model of Cerebral Palsy. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,446570


Temple University

24. Etwebi, Zienab. MYELOPEROXIDASE INDUCES ENDOTHELIAL DYSFUNCTION VIA ACTIVATION OF THE CALCIUM DEPENDENT PROTEASE CALPAIN.

Degree: PhD, 2018, Temple University

Biomedical Sciences

Cardiovascular disease and the associated endothelial dysfunction are characterized by leukocyte activation, decrease endothelial nitric oxide synthase (eNOS) activity, and increased endothelial cell… (more)

Subjects/Keywords: Physiology; Cellular biology;

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APA (6th Edition):

Etwebi, Z. (2018). MYELOPEROXIDASE INDUCES ENDOTHELIAL DYSFUNCTION VIA ACTIVATION OF THE CALCIUM DEPENDENT PROTEASE CALPAIN. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,508536

Chicago Manual of Style (16th Edition):

Etwebi, Zienab. “MYELOPEROXIDASE INDUCES ENDOTHELIAL DYSFUNCTION VIA ACTIVATION OF THE CALCIUM DEPENDENT PROTEASE CALPAIN.” 2018. Doctoral Dissertation, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,508536.

MLA Handbook (7th Edition):

Etwebi, Zienab. “MYELOPEROXIDASE INDUCES ENDOTHELIAL DYSFUNCTION VIA ACTIVATION OF THE CALCIUM DEPENDENT PROTEASE CALPAIN.” 2018. Web. 06 Aug 2020.

Vancouver:

Etwebi Z. MYELOPEROXIDASE INDUCES ENDOTHELIAL DYSFUNCTION VIA ACTIVATION OF THE CALCIUM DEPENDENT PROTEASE CALPAIN. [Internet] [Doctoral dissertation]. Temple University; 2018. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,508536.

Council of Science Editors:

Etwebi Z. MYELOPEROXIDASE INDUCES ENDOTHELIAL DYSFUNCTION VIA ACTIVATION OF THE CALCIUM DEPENDENT PROTEASE CALPAIN. [Doctoral Dissertation]. Temple University; 2018. Available from: http://digital.library.temple.edu/u?/p245801coll10,508536


Temple University

25. Pickens, Brandy S. THE ROLE OF COUP-TFI DURING RETINOIC ACID INDUCED ENDODERMAL DIFFERENTIATION OF P19 CELLS.

Degree: PhD, 2012, Temple University

Biochemistry

ABSTRACT Retinoic acid (RA) is a positive regulator of P19 EC cell differentiation. Pre-B cell leukemia transcription factors (PBXs) act in conjunction with homeobox… (more)

Subjects/Keywords: Biochemistry; COUP-TFI; Endodermal Differentiation; P19 Cells; Retinoic Acid

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APA (6th Edition):

Pickens, B. S. (2012). THE ROLE OF COUP-TFI DURING RETINOIC ACID INDUCED ENDODERMAL DIFFERENTIATION OF P19 CELLS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,196883

Chicago Manual of Style (16th Edition):

Pickens, Brandy S. “THE ROLE OF COUP-TFI DURING RETINOIC ACID INDUCED ENDODERMAL DIFFERENTIATION OF P19 CELLS.” 2012. Doctoral Dissertation, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,196883.

MLA Handbook (7th Edition):

Pickens, Brandy S. “THE ROLE OF COUP-TFI DURING RETINOIC ACID INDUCED ENDODERMAL DIFFERENTIATION OF P19 CELLS.” 2012. Web. 06 Aug 2020.

Vancouver:

Pickens BS. THE ROLE OF COUP-TFI DURING RETINOIC ACID INDUCED ENDODERMAL DIFFERENTIATION OF P19 CELLS. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,196883.

Council of Science Editors:

Pickens BS. THE ROLE OF COUP-TFI DURING RETINOIC ACID INDUCED ENDODERMAL DIFFERENTIATION OF P19 CELLS. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,196883


Temple University

26. Back, Steven. TULA-2: A Novel Protein Tyrosine Phosphatase That Regulates Osteoclast Differentiation and Function.

Degree: PhD, 2014, Temple University

Cell Biology

The human skeleton is a dynamic organ that serves multiple functions to maintain normal physiology and health. It protects vital organs, provides support… (more)

Subjects/Keywords: Cellular biology; Biochemistry; Molecular biology;

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APA (6th Edition):

Back, S. (2014). TULA-2: A Novel Protein Tyrosine Phosphatase That Regulates Osteoclast Differentiation and Function. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,254886

Chicago Manual of Style (16th Edition):

Back, Steven. “TULA-2: A Novel Protein Tyrosine Phosphatase That Regulates Osteoclast Differentiation and Function.” 2014. Doctoral Dissertation, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,254886.

MLA Handbook (7th Edition):

Back, Steven. “TULA-2: A Novel Protein Tyrosine Phosphatase That Regulates Osteoclast Differentiation and Function.” 2014. Web. 06 Aug 2020.

Vancouver:

Back S. TULA-2: A Novel Protein Tyrosine Phosphatase That Regulates Osteoclast Differentiation and Function. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,254886.

Council of Science Editors:

Back S. TULA-2: A Novel Protein Tyrosine Phosphatase That Regulates Osteoclast Differentiation and Function. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,254886


Temple University

27. Reed, Katherine Sullivan. Simultaneous Targeting of PARP1 and RAD52 Triggers Dual Synthetic Lethality in BRCA-Deficient Cancers.

Degree: PhD, 2018, Temple University

Biomedical Sciences

PARP inhibitors (PARPi) have been used to induce synthetic lethality in BRCA-deficient tumors in clinical trials with limited success due to the development… (more)

Subjects/Keywords: Oncology; Cellular biology;

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APA (6th Edition):

Reed, K. S. (2018). Simultaneous Targeting of PARP1 and RAD52 Triggers Dual Synthetic Lethality in BRCA-Deficient Cancers. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,507327

Chicago Manual of Style (16th Edition):

Reed, Katherine Sullivan. “Simultaneous Targeting of PARP1 and RAD52 Triggers Dual Synthetic Lethality in BRCA-Deficient Cancers.” 2018. Doctoral Dissertation, Temple University. Accessed August 06, 2020. http://digital.library.temple.edu/u?/p245801coll10,507327.

MLA Handbook (7th Edition):

Reed, Katherine Sullivan. “Simultaneous Targeting of PARP1 and RAD52 Triggers Dual Synthetic Lethality in BRCA-Deficient Cancers.” 2018. Web. 06 Aug 2020.

Vancouver:

Reed KS. Simultaneous Targeting of PARP1 and RAD52 Triggers Dual Synthetic Lethality in BRCA-Deficient Cancers. [Internet] [Doctoral dissertation]. Temple University; 2018. [cited 2020 Aug 06]. Available from: http://digital.library.temple.edu/u?/p245801coll10,507327.

Council of Science Editors:

Reed KS. Simultaneous Targeting of PARP1 and RAD52 Triggers Dual Synthetic Lethality in BRCA-Deficient Cancers. [Doctoral Dissertation]. Temple University; 2018. Available from: http://digital.library.temple.edu/u?/p245801coll10,507327

.