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You searched for +publisher:"Temple University" +contributor:("Shore, Scott K."). Showing records 1 – 24 of 24 total matches.

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Temple University

1. Lee, Young Shin. MATERNAL DETERMINANTS OF OOCYTE AND EMBRYO QUALITY.

Degree: PhD, 2011, Temple University

Molecular Biology and Genetics

Oocyte quality plays a critical role in establishment of pregnancies, embryo development, implantation and the health of offspring. The oocyte provides… (more)

Subjects/Keywords: Molecular Biology; Genetics; Cumulus cells; Embryo; Microarray; mouse; Oocyte; Rhesus monkey

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APA (6th Edition):

Lee, Y. S. (2011). MATERNAL DETERMINANTS OF OOCYTE AND EMBRYO QUALITY. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,111983

Chicago Manual of Style (16th Edition):

Lee, Young Shin. “MATERNAL DETERMINANTS OF OOCYTE AND EMBRYO QUALITY.” 2011. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,111983.

MLA Handbook (7th Edition):

Lee, Young Shin. “MATERNAL DETERMINANTS OF OOCYTE AND EMBRYO QUALITY.” 2011. Web. 21 Oct 2019.

Vancouver:

Lee YS. MATERNAL DETERMINANTS OF OOCYTE AND EMBRYO QUALITY. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,111983.

Council of Science Editors:

Lee YS. MATERNAL DETERMINANTS OF OOCYTE AND EMBRYO QUALITY. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,111983


Temple University

2. Hoffman, Benjamin. The Genetics of Cancer in Pharmacological Drug Development.

Degree: PhD, 2011, Temple University

Molecular and Cellular Physiology

The field of cancer therapeutic development has been dominated by two research and discovery paradigms, the cytotoxicity-based or phenotype driven strategy… (more)

Subjects/Keywords: Biology; Cancer; Drug Discovery; Therapeutics

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APA (6th Edition):

Hoffman, B. (2011). The Genetics of Cancer in Pharmacological Drug Development. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,212455

Chicago Manual of Style (16th Edition):

Hoffman, Benjamin. “The Genetics of Cancer in Pharmacological Drug Development.” 2011. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,212455.

MLA Handbook (7th Edition):

Hoffman, Benjamin. “The Genetics of Cancer in Pharmacological Drug Development.” 2011. Web. 21 Oct 2019.

Vancouver:

Hoffman B. The Genetics of Cancer in Pharmacological Drug Development. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,212455.

Council of Science Editors:

Hoffman B. The Genetics of Cancer in Pharmacological Drug Development. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,212455


Temple University

3. Beauparlant, Stephen Lewis. Functional characterization of the p97 adaptor protein UBXD1.

Degree: PhD, 2011, Temple University

Molecular Biology and Genetics

p97 is a member of the AAA family of proteins (ATPase Associated with various cellular Activities). It is a highly conserved… (more)

Subjects/Keywords: Molecular biology; Autophagy; ERGIC-53; p97; UBXD1

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APA (6th Edition):

Beauparlant, S. L. (2011). Functional characterization of the p97 adaptor protein UBXD1. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,213118

Chicago Manual of Style (16th Edition):

Beauparlant, Stephen Lewis. “Functional characterization of the p97 adaptor protein UBXD1.” 2011. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,213118.

MLA Handbook (7th Edition):

Beauparlant, Stephen Lewis. “Functional characterization of the p97 adaptor protein UBXD1.” 2011. Web. 21 Oct 2019.

Vancouver:

Beauparlant SL. Functional characterization of the p97 adaptor protein UBXD1. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,213118.

Council of Science Editors:

Beauparlant SL. Functional characterization of the p97 adaptor protein UBXD1. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,213118


Temple University

4. Lee, Young Shin. Supplemental Tables.

Degree: PhD, 2011, Temple University

Molecular Biology and Genetics

Oocyte quality plays a critical role in establishment of pregnancies, embryo development, implantation and the health of offspring. The oocyte provides… (more)

Subjects/Keywords: Molecular Biology; Genetics; Cumulus cells; Embryo; Microarray; mouse; Oocyte; Rhesus monkey

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APA (6th Edition):

Lee, Y. S. (2011). Supplemental Tables. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,215914

Chicago Manual of Style (16th Edition):

Lee, Young Shin. “Supplemental Tables.” 2011. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,215914.

MLA Handbook (7th Edition):

Lee, Young Shin. “Supplemental Tables.” 2011. Web. 21 Oct 2019.

Vancouver:

Lee YS. Supplemental Tables. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,215914.

Council of Science Editors:

Lee YS. Supplemental Tables. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,215914


Temple University

5. Ramkumar, Poornima. Interaction of JLP with PLK1 recruits FoxK1 to form a ternary complex during mitosis.

Degree: PhD, 2015, Temple University

Molecular Biology and Genetics

JLP (JNK associated Leucine zipper protein) is a scaffolding protein that has been shown to interact with and activate the JNK/p38MAPK… (more)

Subjects/Keywords: Molecular biology; Biochemistry; Cellular biology;

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APA (6th Edition):

Ramkumar, P. (2015). Interaction of JLP with PLK1 recruits FoxK1 to form a ternary complex during mitosis. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,323815

Chicago Manual of Style (16th Edition):

Ramkumar, Poornima. “Interaction of JLP with PLK1 recruits FoxK1 to form a ternary complex during mitosis.” 2015. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,323815.

MLA Handbook (7th Edition):

Ramkumar, Poornima. “Interaction of JLP with PLK1 recruits FoxK1 to form a ternary complex during mitosis.” 2015. Web. 21 Oct 2019.

Vancouver:

Ramkumar P. Interaction of JLP with PLK1 recruits FoxK1 to form a ternary complex during mitosis. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,323815.

Council of Science Editors:

Ramkumar P. Interaction of JLP with PLK1 recruits FoxK1 to form a ternary complex during mitosis. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,323815


Temple University

6. Goldsmith, Zachariah G. Oncogenic Signaling Pathways Activated by Lysophosphatidic Acid (LPA) in Ovarian Carcinoma.

Degree: PhD, 2009, Temple University

Molecular Biology and Genetics

Ovarian cancer is currently the most fatal gynecologic cancer and the fifth leading cause of fatal cancer in women overall. As… (more)

Subjects/Keywords: Biology, Molecular; Health Sciences, Oncology; ERK; G alpha 12; JNK; LPA; lysophosphatidic acid; ovarian cancer

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APA (6th Edition):

Goldsmith, Z. G. (2009). Oncogenic Signaling Pathways Activated by Lysophosphatidic Acid (LPA) in Ovarian Carcinoma. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,34336

Chicago Manual of Style (16th Edition):

Goldsmith, Zachariah G. “Oncogenic Signaling Pathways Activated by Lysophosphatidic Acid (LPA) in Ovarian Carcinoma.” 2009. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,34336.

MLA Handbook (7th Edition):

Goldsmith, Zachariah G. “Oncogenic Signaling Pathways Activated by Lysophosphatidic Acid (LPA) in Ovarian Carcinoma.” 2009. Web. 21 Oct 2019.

Vancouver:

Goldsmith ZG. Oncogenic Signaling Pathways Activated by Lysophosphatidic Acid (LPA) in Ovarian Carcinoma. [Internet] [Doctoral dissertation]. Temple University; 2009. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,34336.

Council of Science Editors:

Goldsmith ZG. Oncogenic Signaling Pathways Activated by Lysophosphatidic Acid (LPA) in Ovarian Carcinoma. [Doctoral Dissertation]. Temple University; 2009. Available from: http://digital.library.temple.edu/u?/p245801coll10,34336


Temple University

7. Gardner, Jacob Andrew. GPCR Signaling in the Genesis and Progression of Pancreatic Cancer.

Degree: PhD, 2009, Temple University

Molecular Biology and Genetics

Ductal adenocarcinomas of the pancreas are the 4th most common cause of cancer death. The 1 and 5 year survival rates… (more)

Subjects/Keywords: Biology, Molecular; G alpha 13; GPCR; LPA; lysophosphatidic acid; Migration; Pancreatic Cancer

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APA (6th Edition):

Gardner, J. A. (2009). GPCR Signaling in the Genesis and Progression of Pancreatic Cancer. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,35453

Chicago Manual of Style (16th Edition):

Gardner, Jacob Andrew. “GPCR Signaling in the Genesis and Progression of Pancreatic Cancer.” 2009. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,35453.

MLA Handbook (7th Edition):

Gardner, Jacob Andrew. “GPCR Signaling in the Genesis and Progression of Pancreatic Cancer.” 2009. Web. 21 Oct 2019.

Vancouver:

Gardner JA. GPCR Signaling in the Genesis and Progression of Pancreatic Cancer. [Internet] [Doctoral dissertation]. Temple University; 2009. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,35453.

Council of Science Editors:

Gardner JA. GPCR Signaling in the Genesis and Progression of Pancreatic Cancer. [Doctoral Dissertation]. Temple University; 2009. Available from: http://digital.library.temple.edu/u?/p245801coll10,35453


Temple University

8. Potireddy, Santhi. TRANSLATIONAL CONTROL OF MATERNAL mRNA POPULATION IN MOUSE EMBRYOS.

Degree: PhD, 2010, Temple University

Biochemistry

Early mammalian development before the oocyte-to-embryo transition is under 'maternal control' from factors deposited in the cytoplasm during oocyte growth, synthesized independent of de… (more)

Subjects/Keywords: Biology, Molecular; cis-regulatory motif; Embryo; Microarray; Mouse

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APA (6th Edition):

Potireddy, S. (2010). TRANSLATIONAL CONTROL OF MATERNAL mRNA POPULATION IN MOUSE EMBRYOS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,67111

Chicago Manual of Style (16th Edition):

Potireddy, Santhi. “TRANSLATIONAL CONTROL OF MATERNAL mRNA POPULATION IN MOUSE EMBRYOS.” 2010. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,67111.

MLA Handbook (7th Edition):

Potireddy, Santhi. “TRANSLATIONAL CONTROL OF MATERNAL mRNA POPULATION IN MOUSE EMBRYOS.” 2010. Web. 21 Oct 2019.

Vancouver:

Potireddy S. TRANSLATIONAL CONTROL OF MATERNAL mRNA POPULATION IN MOUSE EMBRYOS. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,67111.

Council of Science Editors:

Potireddy S. TRANSLATIONAL CONTROL OF MATERNAL mRNA POPULATION IN MOUSE EMBRYOS. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,67111


Temple University

9. Mohamed-Hadley, Alisha. THE ROLE OF THE STRESS RESPONSE GENE GADD45A IN MODULATING MYC MEDIATED APOPTOSIS AND DIFFERENTIATION.

Degree: PhD, 2011, Temple University

Molecular Biology and Genetics

The Gadd45 family of proteins is known to play a central role as cellular stress sensors that modulate the response of… (more)

Subjects/Keywords: Molecular Biology; Apoptosis; Cytokine; Gadd45a; Myc; Myeloid cells

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APA (6th Edition):

Mohamed-Hadley, A. (2011). THE ROLE OF THE STRESS RESPONSE GENE GADD45A IN MODULATING MYC MEDIATED APOPTOSIS AND DIFFERENTIATION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,126332

Chicago Manual of Style (16th Edition):

Mohamed-Hadley, Alisha. “THE ROLE OF THE STRESS RESPONSE GENE GADD45A IN MODULATING MYC MEDIATED APOPTOSIS AND DIFFERENTIATION.” 2011. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,126332.

MLA Handbook (7th Edition):

Mohamed-Hadley, Alisha. “THE ROLE OF THE STRESS RESPONSE GENE GADD45A IN MODULATING MYC MEDIATED APOPTOSIS AND DIFFERENTIATION.” 2011. Web. 21 Oct 2019.

Vancouver:

Mohamed-Hadley A. THE ROLE OF THE STRESS RESPONSE GENE GADD45A IN MODULATING MYC MEDIATED APOPTOSIS AND DIFFERENTIATION. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,126332.

Council of Science Editors:

Mohamed-Hadley A. THE ROLE OF THE STRESS RESPONSE GENE GADD45A IN MODULATING MYC MEDIATED APOPTOSIS AND DIFFERENTIATION. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,126332


Temple University

10. Yue, Chanyu. STAT2 in the regulation of tumor growth and antitumor effects of type I interferons in a mouse model of melanoma.

Degree: PhD, 2013, Temple University

Biochemistry

Signal Transducer and Activator of Transcription (STAT) 2 is one of seven members of the STAT family of transcription factors with dual roles in… (more)

Subjects/Keywords: Biochemistry; Oncology;

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APA (6th Edition):

Yue, C. (2013). STAT2 in the regulation of tumor growth and antitumor effects of type I interferons in a mouse model of melanoma. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,233175

Chicago Manual of Style (16th Edition):

Yue, Chanyu. “STAT2 in the regulation of tumor growth and antitumor effects of type I interferons in a mouse model of melanoma.” 2013. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,233175.

MLA Handbook (7th Edition):

Yue, Chanyu. “STAT2 in the regulation of tumor growth and antitumor effects of type I interferons in a mouse model of melanoma.” 2013. Web. 21 Oct 2019.

Vancouver:

Yue C. STAT2 in the regulation of tumor growth and antitumor effects of type I interferons in a mouse model of melanoma. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,233175.

Council of Science Editors:

Yue C. STAT2 in the regulation of tumor growth and antitumor effects of type I interferons in a mouse model of melanoma. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,233175


Temple University

11. Bolton, Elisabeth Spring. Genomic Instability Originates From Leukemia Stem Cells In a Mouse Model of CML-CP.

Degree: PhD, 2013, Temple University

Microbiology and Immunology

In chronic myelogenous leukemia (CML), activation of BCR-ABL, the product of the bcr-abl chimeric gene, leads to constitutive activation of pathways that… (more)

Subjects/Keywords: Biology; Molecular biology; Immunology;

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APA (6th Edition):

Bolton, E. S. (2013). Genomic Instability Originates From Leukemia Stem Cells In a Mouse Model of CML-CP. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,234916

Chicago Manual of Style (16th Edition):

Bolton, Elisabeth Spring. “Genomic Instability Originates From Leukemia Stem Cells In a Mouse Model of CML-CP.” 2013. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,234916.

MLA Handbook (7th Edition):

Bolton, Elisabeth Spring. “Genomic Instability Originates From Leukemia Stem Cells In a Mouse Model of CML-CP.” 2013. Web. 21 Oct 2019.

Vancouver:

Bolton ES. Genomic Instability Originates From Leukemia Stem Cells In a Mouse Model of CML-CP. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,234916.

Council of Science Editors:

Bolton ES. Genomic Instability Originates From Leukemia Stem Cells In a Mouse Model of CML-CP. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,234916


Temple University

12. Korostowski, Lisa. Transcript Regulation within the Kcnq1 Domain.

Degree: PhD, 2012, Temple University

Molecular Biology and Genetics

Epigenetics was a term first coined to understand how cells with the same genetic make up can differentiate into various cell… (more)

Subjects/Keywords: Molecular biology; Genetics;

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APA (6th Edition):

Korostowski, L. (2012). Transcript Regulation within the Kcnq1 Domain. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,235191

Chicago Manual of Style (16th Edition):

Korostowski, Lisa. “Transcript Regulation within the Kcnq1 Domain.” 2012. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,235191.

MLA Handbook (7th Edition):

Korostowski, Lisa. “Transcript Regulation within the Kcnq1 Domain.” 2012. Web. 21 Oct 2019.

Vancouver:

Korostowski L. Transcript Regulation within the Kcnq1 Domain. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,235191.

Council of Science Editors:

Korostowski L. Transcript Regulation within the Kcnq1 Domain. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,235191


Temple University

13. Padgaonkar, Amol. Discovery, Biological and Structural Characterization of ON108600, a Novel Kinase Inhibitor in Triple Negative Breast Cancer.

Degree: PhD, 2014, Temple University

Molecular Biology and Genetics

Selective killing of tumor cells requires the identification of drug targets critical to pathways that drive or support cancer progression. Protein… (more)

Subjects/Keywords: Molecular biology; Cellular biology; Oncology;

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APA (6th Edition):

Padgaonkar, A. (2014). Discovery, Biological and Structural Characterization of ON108600, a Novel Kinase Inhibitor in Triple Negative Breast Cancer. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,269327

Chicago Manual of Style (16th Edition):

Padgaonkar, Amol. “Discovery, Biological and Structural Characterization of ON108600, a Novel Kinase Inhibitor in Triple Negative Breast Cancer.” 2014. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,269327.

MLA Handbook (7th Edition):

Padgaonkar, Amol. “Discovery, Biological and Structural Characterization of ON108600, a Novel Kinase Inhibitor in Triple Negative Breast Cancer.” 2014. Web. 21 Oct 2019.

Vancouver:

Padgaonkar A. Discovery, Biological and Structural Characterization of ON108600, a Novel Kinase Inhibitor in Triple Negative Breast Cancer. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,269327.

Council of Science Editors:

Padgaonkar A. Discovery, Biological and Structural Characterization of ON108600, a Novel Kinase Inhibitor in Triple Negative Breast Cancer. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,269327


Temple University

14. Wang, Fang. The Role of Acinus in Retinoic Acid Signaling Pathway.

Degree: PhD, 2014, Temple University

Biochemistry

Retinoic acid receptor (RAR), a member of the steroid/thyroid hormone nuclear receptor superfamily, functions as a RA-dependent transcription activator bound to the RA response… (more)

Subjects/Keywords: Biochemistry;

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APA (6th Edition):

Wang, F. (2014). The Role of Acinus in Retinoic Acid Signaling Pathway. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,277479

Chicago Manual of Style (16th Edition):

Wang, Fang. “The Role of Acinus in Retinoic Acid Signaling Pathway.” 2014. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,277479.

MLA Handbook (7th Edition):

Wang, Fang. “The Role of Acinus in Retinoic Acid Signaling Pathway.” 2014. Web. 21 Oct 2019.

Vancouver:

Wang F. The Role of Acinus in Retinoic Acid Signaling Pathway. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,277479.

Council of Science Editors:

Wang F. The Role of Acinus in Retinoic Acid Signaling Pathway. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,277479


Temple University

15. Kurimchak, Alison. The B55α/PP2A Holoenzyme in Cell Cycle Exit, Maturation/Differentiation, and Cancer.

Degree: PhD, 2014, Temple University

Molecular Biology and Genetics

The cell cycle is negatively regulated by members of the pocket protein family, which consists of the tumor suppressor pRB and… (more)

Subjects/Keywords: Molecular biology;

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APA (6th Edition):

Kurimchak, A. (2014). The B55α/PP2A Holoenzyme in Cell Cycle Exit, Maturation/Differentiation, and Cancer. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,288430

Chicago Manual of Style (16th Edition):

Kurimchak, Alison. “The B55α/PP2A Holoenzyme in Cell Cycle Exit, Maturation/Differentiation, and Cancer.” 2014. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,288430.

MLA Handbook (7th Edition):

Kurimchak, Alison. “The B55α/PP2A Holoenzyme in Cell Cycle Exit, Maturation/Differentiation, and Cancer.” 2014. Web. 21 Oct 2019.

Vancouver:

Kurimchak A. The B55α/PP2A Holoenzyme in Cell Cycle Exit, Maturation/Differentiation, and Cancer. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,288430.

Council of Science Editors:

Kurimchak A. The B55α/PP2A Holoenzyme in Cell Cycle Exit, Maturation/Differentiation, and Cancer. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,288430


Temple University

16. Zhou, Yu. HCV, Heroin Use, and MicroRNAs.

Degree: PhD, 2014, Temple University

Pathology

Hepatitis C virus (HCV) infection is common among injection drug users (IDUs). There is accumulating evidence that circulating microRNAs (miRNAs) are related to HCV… (more)

Subjects/Keywords: Biology; Microbiology; Immunology;

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APA (6th Edition):

Zhou, Y. (2014). HCV, Heroin Use, and MicroRNAs. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,309425

Chicago Manual of Style (16th Edition):

Zhou, Yu. “HCV, Heroin Use, and MicroRNAs.” 2014. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,309425.

MLA Handbook (7th Edition):

Zhou, Yu. “HCV, Heroin Use, and MicroRNAs.” 2014. Web. 21 Oct 2019.

Vancouver:

Zhou Y. HCV, Heroin Use, and MicroRNAs. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,309425.

Council of Science Editors:

Zhou Y. HCV, Heroin Use, and MicroRNAs. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,309425


Temple University

17. Maifrede, Silvia. EGR-1 TUMOR SUPPRESSOR IN BCR-ABL DRIVEN LEUKEMIA.

Degree: PhD, 2015, Temple University

Molecular Biology and Genetics

Chronic Myelogenous Leukemia (CML) is a hematological disease originated with a chromosomal translocation t(9;22)(q34;q11) in a pluripotent hematopoietic stem cell. CML… (more)

Subjects/Keywords: Molecular biology;

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APA (6th Edition):

Maifrede, S. (2015). EGR-1 TUMOR SUPPRESSOR IN BCR-ABL DRIVEN LEUKEMIA. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,321048

Chicago Manual of Style (16th Edition):

Maifrede, Silvia. “EGR-1 TUMOR SUPPRESSOR IN BCR-ABL DRIVEN LEUKEMIA.” 2015. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,321048.

MLA Handbook (7th Edition):

Maifrede, Silvia. “EGR-1 TUMOR SUPPRESSOR IN BCR-ABL DRIVEN LEUKEMIA.” 2015. Web. 21 Oct 2019.

Vancouver:

Maifrede S. EGR-1 TUMOR SUPPRESSOR IN BCR-ABL DRIVEN LEUKEMIA. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,321048.

Council of Science Editors:

Maifrede S. EGR-1 TUMOR SUPPRESSOR IN BCR-ABL DRIVEN LEUKEMIA. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,321048


Temple University

18. Kotredes, Kevin Patrick. Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression.

Degree: PhD, 2015, Temple University

Molecular Biology and Genetics

The isocitrate dehydrogenase (IDH) family of enzymes is central to cellular metabolism, catalyzing the oxidative decarboxylation of isocitrate to α-ketoglutarate (α-KG)… (more)

Subjects/Keywords: Molecular biology; Genetics; Biochemistry;

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APA (6th Edition):

Kotredes, K. P. (2015). Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,356693

Chicago Manual of Style (16th Edition):

Kotredes, Kevin Patrick. “Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression.” 2015. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,356693.

MLA Handbook (7th Edition):

Kotredes, Kevin Patrick. “Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression.” 2015. Web. 21 Oct 2019.

Vancouver:

Kotredes KP. Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,356693.

Council of Science Editors:

Kotredes KP. Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,356693


Temple University

19. Preston, Kyle J. Macronutrient Activation of Endothelium Dependent Leukocyte Trafficking: Metabolic Implications.

Degree: PhD, 2015, Temple University

Physiology

Obesity and insulin resistance are characterized by elevated pro-inflammatory proteins in the blood and immune cell accumulation in the visceral adipose tissue. Resident leukocytes… (more)

Subjects/Keywords: Physiology;

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APA (6th Edition):

Preston, K. J. (2015). Macronutrient Activation of Endothelium Dependent Leukocyte Trafficking: Metabolic Implications. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,361365

Chicago Manual of Style (16th Edition):

Preston, Kyle J. “Macronutrient Activation of Endothelium Dependent Leukocyte Trafficking: Metabolic Implications.” 2015. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,361365.

MLA Handbook (7th Edition):

Preston, Kyle J. “Macronutrient Activation of Endothelium Dependent Leukocyte Trafficking: Metabolic Implications.” 2015. Web. 21 Oct 2019.

Vancouver:

Preston KJ. Macronutrient Activation of Endothelium Dependent Leukocyte Trafficking: Metabolic Implications. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,361365.

Council of Science Editors:

Preston KJ. Macronutrient Activation of Endothelium Dependent Leukocyte Trafficking: Metabolic Implications. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,361365


Temple University

20. Hughes, Lucinda Jane. Yes-Associated Protein (YAP) and Transcriptional Co-Activator with PDZ Binding Motif (TAZ) Function in Normal Cerebellar Development and Medulloblastoma.

Degree: PhD, 2016, Temple University

Biomedical Sciences

The Hippo signaling pathway was first discovered in Drosophila melanogaster and is involved in organ size control by regulating cell proliferation and apoptosis.… (more)

Subjects/Keywords: Developmental biology; Cellular biology; Neurosciences;

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APA (6th Edition):

Hughes, L. J. (2016). Yes-Associated Protein (YAP) and Transcriptional Co-Activator with PDZ Binding Motif (TAZ) Function in Normal Cerebellar Development and Medulloblastoma. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,412035

Chicago Manual of Style (16th Edition):

Hughes, Lucinda Jane. “Yes-Associated Protein (YAP) and Transcriptional Co-Activator with PDZ Binding Motif (TAZ) Function in Normal Cerebellar Development and Medulloblastoma.” 2016. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,412035.

MLA Handbook (7th Edition):

Hughes, Lucinda Jane. “Yes-Associated Protein (YAP) and Transcriptional Co-Activator with PDZ Binding Motif (TAZ) Function in Normal Cerebellar Development and Medulloblastoma.” 2016. Web. 21 Oct 2019.

Vancouver:

Hughes LJ. Yes-Associated Protein (YAP) and Transcriptional Co-Activator with PDZ Binding Motif (TAZ) Function in Normal Cerebellar Development and Medulloblastoma. [Internet] [Doctoral dissertation]. Temple University; 2016. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,412035.

Council of Science Editors:

Hughes LJ. Yes-Associated Protein (YAP) and Transcriptional Co-Activator with PDZ Binding Motif (TAZ) Function in Normal Cerebellar Development and Medulloblastoma. [Doctoral Dissertation]. Temple University; 2016. Available from: http://digital.library.temple.edu/u?/p245801coll10,412035


Temple University

21. Shikov, Sergei. Structural Determinants for Heparin Binding in Human Coagulation Factor XI.

Degree: PhD, 2008, Temple University

Biochemistry

Coagulation factor XI plays an important role in the consolidation phase of blood coagulation. Previous studies from our laboratory and others have demonstrated that… (more)

Subjects/Keywords: Chemistry, Biochemistry; Biophysics, General; Heparin; blood coagulation; ELISA; SPR

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APA (6th Edition):

Shikov, S. (2008). Structural Determinants for Heparin Binding in Human Coagulation Factor XI. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,13460

Chicago Manual of Style (16th Edition):

Shikov, Sergei. “Structural Determinants for Heparin Binding in Human Coagulation Factor XI.” 2008. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,13460.

MLA Handbook (7th Edition):

Shikov, Sergei. “Structural Determinants for Heparin Binding in Human Coagulation Factor XI.” 2008. Web. 21 Oct 2019.

Vancouver:

Shikov S. Structural Determinants for Heparin Binding in Human Coagulation Factor XI. [Internet] [Doctoral dissertation]. Temple University; 2008. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,13460.

Council of Science Editors:

Shikov S. Structural Determinants for Heparin Binding in Human Coagulation Factor XI. [Doctoral Dissertation]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,13460


Temple University

22. Zumbrun, Steven David. Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b.

Degree: PhD, 2008, Temple University

Molecular Biology and Genetics

The GADD45 family of proteins consists of three small nuclear proteins, GADD45A, GADD45B, and GADD45G, which are implicated in modulating the… (more)

Subjects/Keywords: Biology, Molecular; gadd45b; MMS; post-transcriptional regulation; Sorbitol; stress response; transcriptional regulation

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APA (6th Edition):

Zumbrun, S. D. (2008). Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,23355

Chicago Manual of Style (16th Edition):

Zumbrun, Steven David. “Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b.” 2008. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,23355.

MLA Handbook (7th Edition):

Zumbrun, Steven David. “Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b.” 2008. Web. 21 Oct 2019.

Vancouver:

Zumbrun SD. Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b. [Internet] [Doctoral dissertation]. Temple University; 2008. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,23355.

Council of Science Editors:

Zumbrun SD. Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b. [Doctoral Dissertation]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,23355


Temple University

23. Jayadeva, Girish. B55alpha modulates the phosphorylation status of the pRb-related p107 and p130 proteins.

Degree: PhD, 2010, Temple University

Molecular Biology and Genetics

The retinoblastoma family of phosphoproteins consisting of the retinoblastoma protein (pRB) and the two structurally related proteins p130 and p107 play… (more)

Subjects/Keywords: Biology, Molecular; B55; p107; p130; pocket; PP2A; pRb

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APA (6th Edition):

Jayadeva, G. (2010). B55alpha modulates the phosphorylation status of the pRb-related p107 and p130 proteins. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,64785

Chicago Manual of Style (16th Edition):

Jayadeva, Girish. “B55alpha modulates the phosphorylation status of the pRb-related p107 and p130 proteins.” 2010. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,64785.

MLA Handbook (7th Edition):

Jayadeva, Girish. “B55alpha modulates the phosphorylation status of the pRb-related p107 and p130 proteins.” 2010. Web. 21 Oct 2019.

Vancouver:

Jayadeva G. B55alpha modulates the phosphorylation status of the pRb-related p107 and p130 proteins. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,64785.

Council of Science Editors:

Jayadeva G. B55alpha modulates the phosphorylation status of the pRb-related p107 and p130 proteins. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,64785


Temple University

24. Collura, Kaitlin Marie. Palmitoylation-Dependent Regulation of the DLK/JNK/cJun and the GP130/JAK/STAT Retrograde Signaling Pathways.

Degree: PhD, 2015, Temple University

Biomedical Sciences

Palmitoylation is the post-translational addition of the 16-carbon fatty acid palmitate to protein cysteine residues. This process is best known for its roles… (more)

Subjects/Keywords: Neurosciences

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APA (6th Edition):

Collura, K. M. (2015). Palmitoylation-Dependent Regulation of the DLK/JNK/cJun and the GP130/JAK/STAT Retrograde Signaling Pathways. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,426710

Chicago Manual of Style (16th Edition):

Collura, Kaitlin Marie. “Palmitoylation-Dependent Regulation of the DLK/JNK/cJun and the GP130/JAK/STAT Retrograde Signaling Pathways.” 2015. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,426710.

MLA Handbook (7th Edition):

Collura, Kaitlin Marie. “Palmitoylation-Dependent Regulation of the DLK/JNK/cJun and the GP130/JAK/STAT Retrograde Signaling Pathways.” 2015. Web. 21 Oct 2019.

Vancouver:

Collura KM. Palmitoylation-Dependent Regulation of the DLK/JNK/cJun and the GP130/JAK/STAT Retrograde Signaling Pathways. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,426710.

Council of Science Editors:

Collura KM. Palmitoylation-Dependent Regulation of the DLK/JNK/cJun and the GP130/JAK/STAT Retrograde Signaling Pathways. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,426710

.