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You searched for +publisher:"Temple University" +contributor:("Sanjay, Archana"). Showing records 1 – 6 of 6 total matches.

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Temple University

1. Brennan, Tracy A. Abrogation of Cbl-PI3K Interaction Increases Bone Volume and Osteoblast Proliferation.

Degree: PhD, 2011, Temple University

Cell Biology

Cbl is a multivalent protein that interacts with a number of signaling molecules that affect cell proliferation, migration and apoptosis. Although it is… (more)

Subjects/Keywords: Cellular Biology; Biology; Bone; Cbl; Osteoblasts; PI3K

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Brennan, T. A. (2011). Abrogation of Cbl-PI3K Interaction Increases Bone Volume and Osteoblast Proliferation. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,107475

Chicago Manual of Style (16th Edition):

Brennan, Tracy A. “Abrogation of Cbl-PI3K Interaction Increases Bone Volume and Osteoblast Proliferation.” 2011. Doctoral Dissertation, Temple University. Accessed October 21, 2020. http://digital.library.temple.edu/u?/p245801coll10,107475.

MLA Handbook (7th Edition):

Brennan, Tracy A. “Abrogation of Cbl-PI3K Interaction Increases Bone Volume and Osteoblast Proliferation.” 2011. Web. 21 Oct 2020.

Vancouver:

Brennan TA. Abrogation of Cbl-PI3K Interaction Increases Bone Volume and Osteoblast Proliferation. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2020 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,107475.

Council of Science Editors:

Brennan TA. Abrogation of Cbl-PI3K Interaction Increases Bone Volume and Osteoblast Proliferation. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,107475


Temple University

2. Singh, Maneet. TRANSCRIPTIONAL REGULATION OF OSTEOACTIVIN EXPRESSION BY BMP-2 IN OSTEOBLASTS.

Degree: PhD, 2011, Temple University

Cell Biology

Osteoactivin (OA) is a glycoprotein required for the differentiation of osteoblasts. In osteoblasts, Bone Morphogenetic Protein-2 (BMP-2) activated Smad1 signaling enhances OA expression.… (more)

Subjects/Keywords: Cellular Biology; Molecular Biology; Biology; HOMEDOMAIN PROTEIN; OSTEOACTIVIN; OSTEOBLAST DIFFERENTIATION; RUNX2; SMAD1 AND SMAD4; TRANSCRIPTIONAL REGULATION

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APA (6th Edition):

Singh, M. (2011). TRANSCRIPTIONAL REGULATION OF OSTEOACTIVIN EXPRESSION BY BMP-2 IN OSTEOBLASTS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,135232

Chicago Manual of Style (16th Edition):

Singh, Maneet. “TRANSCRIPTIONAL REGULATION OF OSTEOACTIVIN EXPRESSION BY BMP-2 IN OSTEOBLASTS.” 2011. Doctoral Dissertation, Temple University. Accessed October 21, 2020. http://digital.library.temple.edu/u?/p245801coll10,135232.

MLA Handbook (7th Edition):

Singh, Maneet. “TRANSCRIPTIONAL REGULATION OF OSTEOACTIVIN EXPRESSION BY BMP-2 IN OSTEOBLASTS.” 2011. Web. 21 Oct 2020.

Vancouver:

Singh M. TRANSCRIPTIONAL REGULATION OF OSTEOACTIVIN EXPRESSION BY BMP-2 IN OSTEOBLASTS. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2020 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,135232.

Council of Science Editors:

Singh M. TRANSCRIPTIONAL REGULATION OF OSTEOACTIVIN EXPRESSION BY BMP-2 IN OSTEOBLASTS. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,135232


Temple University

3. Buitrago Murcia, Claudia Lorena. Cbl proteins in platelet functional responses.

Degree: PhD, 2012, Temple University

Physiology

c-Cbl protein functions as an E3 ligase and scaffolding protein, where three residues, Y700, Y731, and Y774, upon phosphorylation, have been shown to initiate… (more)

Subjects/Keywords: Physiology; Cellular biology; Cbl; hemostasis; kinases; platelets; signaling

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APA (6th Edition):

Buitrago Murcia, C. L. (2012). Cbl proteins in platelet functional responses. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,198139

Chicago Manual of Style (16th Edition):

Buitrago Murcia, Claudia Lorena. “Cbl proteins in platelet functional responses.” 2012. Doctoral Dissertation, Temple University. Accessed October 21, 2020. http://digital.library.temple.edu/u?/p245801coll10,198139.

MLA Handbook (7th Edition):

Buitrago Murcia, Claudia Lorena. “Cbl proteins in platelet functional responses.” 2012. Web. 21 Oct 2020.

Vancouver:

Buitrago Murcia CL. Cbl proteins in platelet functional responses. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,198139.

Council of Science Editors:

Buitrago Murcia CL. Cbl proteins in platelet functional responses. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,198139


Temple University

4. Newman, Tiffanny Nicole. ROLE OF TULA-FAMILY PROTEINS IN T CELL DRIVEN RESPONSES.

Degree: PhD, 2011, Temple University

Microbiology and Immunology

The TULA-family consists of two proteins implicated in cellular regulation. TULA-1 is expressed in T-cells and is involved in apoptosis. TULA-2 is… (more)

Subjects/Keywords: Immunology; autoimmune; inflammatory bowel disease; phosphatase; T cells; TULA-1; TULA-2

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APA (6th Edition):

Newman, T. N. (2011). ROLE OF TULA-FAMILY PROTEINS IN T CELL DRIVEN RESPONSES. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,210733

Chicago Manual of Style (16th Edition):

Newman, Tiffanny Nicole. “ROLE OF TULA-FAMILY PROTEINS IN T CELL DRIVEN RESPONSES.” 2011. Doctoral Dissertation, Temple University. Accessed October 21, 2020. http://digital.library.temple.edu/u?/p245801coll10,210733.

MLA Handbook (7th Edition):

Newman, Tiffanny Nicole. “ROLE OF TULA-FAMILY PROTEINS IN T CELL DRIVEN RESPONSES.” 2011. Web. 21 Oct 2020.

Vancouver:

Newman TN. ROLE OF TULA-FAMILY PROTEINS IN T CELL DRIVEN RESPONSES. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2020 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,210733.

Council of Science Editors:

Newman TN. ROLE OF TULA-FAMILY PROTEINS IN T CELL DRIVEN RESPONSES. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,210733


Temple University

5. Back, Steven. TULA-2: A Novel Protein Tyrosine Phosphatase That Regulates Osteoclast Differentiation and Function.

Degree: PhD, 2014, Temple University

Cell Biology

The human skeleton is a dynamic organ that serves multiple functions to maintain normal physiology and health. It protects vital organs, provides support… (more)

Subjects/Keywords: Cellular biology; Biochemistry; Molecular biology;

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Back, S. (2014). TULA-2: A Novel Protein Tyrosine Phosphatase That Regulates Osteoclast Differentiation and Function. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,254886

Chicago Manual of Style (16th Edition):

Back, Steven. “TULA-2: A Novel Protein Tyrosine Phosphatase That Regulates Osteoclast Differentiation and Function.” 2014. Doctoral Dissertation, Temple University. Accessed October 21, 2020. http://digital.library.temple.edu/u?/p245801coll10,254886.

MLA Handbook (7th Edition):

Back, Steven. “TULA-2: A Novel Protein Tyrosine Phosphatase That Regulates Osteoclast Differentiation and Function.” 2014. Web. 21 Oct 2020.

Vancouver:

Back S. TULA-2: A Novel Protein Tyrosine Phosphatase That Regulates Osteoclast Differentiation and Function. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2020 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,254886.

Council of Science Editors:

Back S. TULA-2: A Novel Protein Tyrosine Phosphatase That Regulates Osteoclast Differentiation and Function. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,254886


Temple University

6. Zhang, Xuemei. Src Kinase Signaling Regulates Connective Tissue Growth Factor (CTGF/CCN2) Induction by Transforming Growth Factor-Beta 1 (TGF-b1) in Osteoblasts.

Degree: PhD, 2010, Temple University

Anatomy

Connective tissue growth factor (CTGF/CCN2) is a cysteine rich, extracellular matrix protein that acts as an anabolic growth factor to regulate osteoblast differentiation and… (more)

Subjects/Keywords: Biology; Cell

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhang, X. (2010). Src Kinase Signaling Regulates Connective Tissue Growth Factor (CTGF/CCN2) Induction by Transforming Growth Factor-Beta 1 (TGF-b1) in Osteoblasts. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,63095

Chicago Manual of Style (16th Edition):

Zhang, Xuemei. “Src Kinase Signaling Regulates Connective Tissue Growth Factor (CTGF/CCN2) Induction by Transforming Growth Factor-Beta 1 (TGF-b1) in Osteoblasts.” 2010. Doctoral Dissertation, Temple University. Accessed October 21, 2020. http://digital.library.temple.edu/u?/p245801coll10,63095.

MLA Handbook (7th Edition):

Zhang, Xuemei. “Src Kinase Signaling Regulates Connective Tissue Growth Factor (CTGF/CCN2) Induction by Transforming Growth Factor-Beta 1 (TGF-b1) in Osteoblasts.” 2010. Web. 21 Oct 2020.

Vancouver:

Zhang X. Src Kinase Signaling Regulates Connective Tissue Growth Factor (CTGF/CCN2) Induction by Transforming Growth Factor-Beta 1 (TGF-b1) in Osteoblasts. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,63095.

Council of Science Editors:

Zhang X. Src Kinase Signaling Regulates Connective Tissue Growth Factor (CTGF/CCN2) Induction by Transforming Growth Factor-Beta 1 (TGF-b1) in Osteoblasts. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,63095

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