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You searched for +publisher:"Temple University" +contributor:("Safadi, Fayez F."). Showing records 1 – 12 of 12 total matches.

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Temple University

1. Hanifi, Arash. IMPROVED SPECIFICITY OF MATRIX ASSESSMENT OF NORMAL AND REPAIR CARTILAGE USING INFRARED IMAGING ANALYSIS AND MULTIVARIATE METHODS.

Degree: PhD, 2012, Temple University

Mechanical Engineering

Articular cartilage is a connective tissue that lines the long bones and provides a near frictionless load-bearing surface. Cartilage degeneration, which is associated… (more)

Subjects/Keywords: Biomedical engineering

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APA (6th Edition):

Hanifi, A. (2012). IMPROVED SPECIFICITY OF MATRIX ASSESSMENT OF NORMAL AND REPAIR CARTILAGE USING INFRARED IMAGING ANALYSIS AND MULTIVARIATE METHODS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,194010

Chicago Manual of Style (16th Edition):

Hanifi, Arash. “IMPROVED SPECIFICITY OF MATRIX ASSESSMENT OF NORMAL AND REPAIR CARTILAGE USING INFRARED IMAGING ANALYSIS AND MULTIVARIATE METHODS.” 2012. Doctoral Dissertation, Temple University. Accessed September 21, 2020. http://digital.library.temple.edu/u?/p245801coll10,194010.

MLA Handbook (7th Edition):

Hanifi, Arash. “IMPROVED SPECIFICITY OF MATRIX ASSESSMENT OF NORMAL AND REPAIR CARTILAGE USING INFRARED IMAGING ANALYSIS AND MULTIVARIATE METHODS.” 2012. Web. 21 Sep 2020.

Vancouver:

Hanifi A. IMPROVED SPECIFICITY OF MATRIX ASSESSMENT OF NORMAL AND REPAIR CARTILAGE USING INFRARED IMAGING ANALYSIS AND MULTIVARIATE METHODS. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,194010.

Council of Science Editors:

Hanifi A. IMPROVED SPECIFICITY OF MATRIX ASSESSMENT OF NORMAL AND REPAIR CARTILAGE USING INFRARED IMAGING ANALYSIS AND MULTIVARIATE METHODS. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,194010


Temple University

2. Joshi, Rupali Narayan. IDENTIFICATION OF MECHANISMS OF DELAYED PUBERTY ON BONE STRENGTH DEFICITS DURING DEVELOPMENT.

Degree: PhD, 2010, Temple University

Kinesiology

Osteoporosis which is frequently referred to as a pediatric disease with geriatric consequences (Golden, 2000) can result from a lack of optimal bone accrual… (more)

Subjects/Keywords: Biology, Physiology; BONE STRENGTH; DELAYED PUBERTY; ENERGY RESTRICTION; ESTROGEN DEFICIENCY; GnRH-a; OSTEOPOROSIS

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APA (6th Edition):

Joshi, R. N. (2010). IDENTIFICATION OF MECHANISMS OF DELAYED PUBERTY ON BONE STRENGTH DEFICITS DURING DEVELOPMENT. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,55431

Chicago Manual of Style (16th Edition):

Joshi, Rupali Narayan. “IDENTIFICATION OF MECHANISMS OF DELAYED PUBERTY ON BONE STRENGTH DEFICITS DURING DEVELOPMENT.” 2010. Doctoral Dissertation, Temple University. Accessed September 21, 2020. http://digital.library.temple.edu/u?/p245801coll10,55431.

MLA Handbook (7th Edition):

Joshi, Rupali Narayan. “IDENTIFICATION OF MECHANISMS OF DELAYED PUBERTY ON BONE STRENGTH DEFICITS DURING DEVELOPMENT.” 2010. Web. 21 Sep 2020.

Vancouver:

Joshi RN. IDENTIFICATION OF MECHANISMS OF DELAYED PUBERTY ON BONE STRENGTH DEFICITS DURING DEVELOPMENT. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,55431.

Council of Science Editors:

Joshi RN. IDENTIFICATION OF MECHANISMS OF DELAYED PUBERTY ON BONE STRENGTH DEFICITS DURING DEVELOPMENT. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,55431


Temple University

3. Pallai, Rajash. The CCAAT-box binding transcription factor, nuclear factor-Y (NF-Y) regulates transcription of human aldo-keto reductase 1C1 (AKR1C1) gene.

Degree: PhD, 2010, Temple University

Pathology

Dihydrodiol dehydrogenases are a family of aldo-keto reductases (AKR1Cs) involved in the metabolism of steroid hormones and xenobiotics. Whilst, several phase II drugs as… (more)

Subjects/Keywords: Biology, Molecular; Dihydrodiol dehydrogenase; Hydroxysteroid dehydrogenase; Liver hepatoblastoma; Lung adenocarcinoma; Ovarian carcinoma; Promoter regulation

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APA (6th Edition):

Pallai, R. (2010). The CCAAT-box binding transcription factor, nuclear factor-Y (NF-Y) regulates transcription of human aldo-keto reductase 1C1 (AKR1C1) gene. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,78307

Chicago Manual of Style (16th Edition):

Pallai, Rajash. “The CCAAT-box binding transcription factor, nuclear factor-Y (NF-Y) regulates transcription of human aldo-keto reductase 1C1 (AKR1C1) gene.” 2010. Doctoral Dissertation, Temple University. Accessed September 21, 2020. http://digital.library.temple.edu/u?/p245801coll10,78307.

MLA Handbook (7th Edition):

Pallai, Rajash. “The CCAAT-box binding transcription factor, nuclear factor-Y (NF-Y) regulates transcription of human aldo-keto reductase 1C1 (AKR1C1) gene.” 2010. Web. 21 Sep 2020.

Vancouver:

Pallai R. The CCAAT-box binding transcription factor, nuclear factor-Y (NF-Y) regulates transcription of human aldo-keto reductase 1C1 (AKR1C1) gene. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,78307.

Council of Science Editors:

Pallai R. The CCAAT-box binding transcription factor, nuclear factor-Y (NF-Y) regulates transcription of human aldo-keto reductase 1C1 (AKR1C1) gene. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,78307


Temple University

4. Moore, Andrea Rossi. COX-2 inhibition impaired resolution of chronic inflammation in a murine model of autoimmune arthritis.

Degree: PhD, 2010, Temple University

Microbiology and Immunology

Rheumatoid arthritis (RA) is a chronic disease characterized by cycles of inflammation and resolution. Previously, it was believed that the resolution of… (more)

Subjects/Keywords: Health Sciences, Immunology; autoimmunity; inflammation; lipid mediators; rheumatoid arthritis; rodent

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APA (6th Edition):

Moore, A. R. (2010). COX-2 inhibition impaired resolution of chronic inflammation in a murine model of autoimmune arthritis. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,81890

Chicago Manual of Style (16th Edition):

Moore, Andrea Rossi. “COX-2 inhibition impaired resolution of chronic inflammation in a murine model of autoimmune arthritis.” 2010. Doctoral Dissertation, Temple University. Accessed September 21, 2020. http://digital.library.temple.edu/u?/p245801coll10,81890.

MLA Handbook (7th Edition):

Moore, Andrea Rossi. “COX-2 inhibition impaired resolution of chronic inflammation in a murine model of autoimmune arthritis.” 2010. Web. 21 Sep 2020.

Vancouver:

Moore AR. COX-2 inhibition impaired resolution of chronic inflammation in a murine model of autoimmune arthritis. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,81890.

Council of Science Editors:

Moore AR. COX-2 inhibition impaired resolution of chronic inflammation in a murine model of autoimmune arthritis. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,81890


Temple University

5. Brennan, Tracy A. Abrogation of Cbl-PI3K Interaction Increases Bone Volume and Osteoblast Proliferation.

Degree: PhD, 2011, Temple University

Cell Biology

Cbl is a multivalent protein that interacts with a number of signaling molecules that affect cell proliferation, migration and apoptosis. Although it is… (more)

Subjects/Keywords: Cellular Biology; Biology; Bone; Cbl; Osteoblasts; PI3K

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APA (6th Edition):

Brennan, T. A. (2011). Abrogation of Cbl-PI3K Interaction Increases Bone Volume and Osteoblast Proliferation. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,107475

Chicago Manual of Style (16th Edition):

Brennan, Tracy A. “Abrogation of Cbl-PI3K Interaction Increases Bone Volume and Osteoblast Proliferation.” 2011. Doctoral Dissertation, Temple University. Accessed September 21, 2020. http://digital.library.temple.edu/u?/p245801coll10,107475.

MLA Handbook (7th Edition):

Brennan, Tracy A. “Abrogation of Cbl-PI3K Interaction Increases Bone Volume and Osteoblast Proliferation.” 2011. Web. 21 Sep 2020.

Vancouver:

Brennan TA. Abrogation of Cbl-PI3K Interaction Increases Bone Volume and Osteoblast Proliferation. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2020 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,107475.

Council of Science Editors:

Brennan TA. Abrogation of Cbl-PI3K Interaction Increases Bone Volume and Osteoblast Proliferation. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,107475


Temple University

6. Newman, Tiffanny Nicole. ROLE OF TULA-FAMILY PROTEINS IN T CELL DRIVEN RESPONSES.

Degree: PhD, 2011, Temple University

Microbiology and Immunology

The TULA-family consists of two proteins implicated in cellular regulation. TULA-1 is expressed in T-cells and is involved in apoptosis. TULA-2 is… (more)

Subjects/Keywords: Immunology; autoimmune; inflammatory bowel disease; phosphatase; T cells; TULA-1; TULA-2

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APA (6th Edition):

Newman, T. N. (2011). ROLE OF TULA-FAMILY PROTEINS IN T CELL DRIVEN RESPONSES. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,210733

Chicago Manual of Style (16th Edition):

Newman, Tiffanny Nicole. “ROLE OF TULA-FAMILY PROTEINS IN T CELL DRIVEN RESPONSES.” 2011. Doctoral Dissertation, Temple University. Accessed September 21, 2020. http://digital.library.temple.edu/u?/p245801coll10,210733.

MLA Handbook (7th Edition):

Newman, Tiffanny Nicole. “ROLE OF TULA-FAMILY PROTEINS IN T CELL DRIVEN RESPONSES.” 2011. Web. 21 Sep 2020.

Vancouver:

Newman TN. ROLE OF TULA-FAMILY PROTEINS IN T CELL DRIVEN RESPONSES. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2020 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,210733.

Council of Science Editors:

Newman TN. ROLE OF TULA-FAMILY PROTEINS IN T CELL DRIVEN RESPONSES. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,210733


Temple University

7. Hendesi, Honey. CONNECTIVE TISSUE GROWTH FACTOR (CTGF/CCN2) REGULATES OSTEOBLAST CYTOSKELETAL REORGANIZATION AND MOTILITY AND ENHANCES DIFFERENTIATION VIA BINDING TO INTEGRIN RECEPTORS AND ACTIVATION OF DOWNSTREAM SIGNALINGS.

Degree: PhD, 2014, Temple University

Cell Biology

Connective Tissue Growth Factor (CTGF) is a matricellular protein that has been shown to mediate cell adhesion, and as a consequence, it regulates… (more)

Subjects/Keywords: Cellular biology;

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APA (6th Edition):

Hendesi, H. (2014). CONNECTIVE TISSUE GROWTH FACTOR (CTGF/CCN2) REGULATES OSTEOBLAST CYTOSKELETAL REORGANIZATION AND MOTILITY AND ENHANCES DIFFERENTIATION VIA BINDING TO INTEGRIN RECEPTORS AND ACTIVATION OF DOWNSTREAM SIGNALINGS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,263674

Chicago Manual of Style (16th Edition):

Hendesi, Honey. “CONNECTIVE TISSUE GROWTH FACTOR (CTGF/CCN2) REGULATES OSTEOBLAST CYTOSKELETAL REORGANIZATION AND MOTILITY AND ENHANCES DIFFERENTIATION VIA BINDING TO INTEGRIN RECEPTORS AND ACTIVATION OF DOWNSTREAM SIGNALINGS.” 2014. Doctoral Dissertation, Temple University. Accessed September 21, 2020. http://digital.library.temple.edu/u?/p245801coll10,263674.

MLA Handbook (7th Edition):

Hendesi, Honey. “CONNECTIVE TISSUE GROWTH FACTOR (CTGF/CCN2) REGULATES OSTEOBLAST CYTOSKELETAL REORGANIZATION AND MOTILITY AND ENHANCES DIFFERENTIATION VIA BINDING TO INTEGRIN RECEPTORS AND ACTIVATION OF DOWNSTREAM SIGNALINGS.” 2014. Web. 21 Sep 2020.

Vancouver:

Hendesi H. CONNECTIVE TISSUE GROWTH FACTOR (CTGF/CCN2) REGULATES OSTEOBLAST CYTOSKELETAL REORGANIZATION AND MOTILITY AND ENHANCES DIFFERENTIATION VIA BINDING TO INTEGRIN RECEPTORS AND ACTIVATION OF DOWNSTREAM SIGNALINGS. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2020 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,263674.

Council of Science Editors:

Hendesi H. CONNECTIVE TISSUE GROWTH FACTOR (CTGF/CCN2) REGULATES OSTEOBLAST CYTOSKELETAL REORGANIZATION AND MOTILITY AND ENHANCES DIFFERENTIATION VIA BINDING TO INTEGRIN RECEPTORS AND ACTIVATION OF DOWNSTREAM SIGNALINGS. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,263674


Temple University

8. Frara, Nagat. THE ROLE OF OSTEOACTIVIN IN MUSCULOSKELETAL TISSUES AS A REPAIR AND ANABOLIC FACTOR.

Degree: PhD, 2015, Temple University

Cell Biology

Osteoactivin (OA) is a novel osteogenic and repair factor. It has the ability to regulate cell proliferation, adhesion, differentiation, and synthesis and regulation… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Frara, N. (2015). THE ROLE OF OSTEOACTIVIN IN MUSCULOSKELETAL TISSUES AS A REPAIR AND ANABOLIC FACTOR. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,336629

Chicago Manual of Style (16th Edition):

Frara, Nagat. “THE ROLE OF OSTEOACTIVIN IN MUSCULOSKELETAL TISSUES AS A REPAIR AND ANABOLIC FACTOR.” 2015. Doctoral Dissertation, Temple University. Accessed September 21, 2020. http://digital.library.temple.edu/u?/p245801coll10,336629.

MLA Handbook (7th Edition):

Frara, Nagat. “THE ROLE OF OSTEOACTIVIN IN MUSCULOSKELETAL TISSUES AS A REPAIR AND ANABOLIC FACTOR.” 2015. Web. 21 Sep 2020.

Vancouver:

Frara N. THE ROLE OF OSTEOACTIVIN IN MUSCULOSKELETAL TISSUES AS A REPAIR AND ANABOLIC FACTOR. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,336629.

Council of Science Editors:

Frara N. THE ROLE OF OSTEOACTIVIN IN MUSCULOSKELETAL TISSUES AS A REPAIR AND ANABOLIC FACTOR. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,336629


Temple University

9. Chawla, Rachna. Role of CDK4 in Development and Cancer.

Degree: PhD, 2008, Temple University

Molecular Biology and Genetics

The mammalian cell cycle is divided into four distinct phases: G1, S, G2, and M. The transition from G1 to S… (more)

Subjects/Keywords: Biology; Molecular

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APA (6th Edition):

Chawla, R. (2008). Role of CDK4 in Development and Cancer. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,7450

Chicago Manual of Style (16th Edition):

Chawla, Rachna. “Role of CDK4 in Development and Cancer.” 2008. Doctoral Dissertation, Temple University. Accessed September 21, 2020. http://digital.library.temple.edu/u?/p245801coll10,7450.

MLA Handbook (7th Edition):

Chawla, Rachna. “Role of CDK4 in Development and Cancer.” 2008. Web. 21 Sep 2020.

Vancouver:

Chawla R. Role of CDK4 in Development and Cancer. [Internet] [Doctoral dissertation]. Temple University; 2008. [cited 2020 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,7450.

Council of Science Editors:

Chawla R. Role of CDK4 in Development and Cancer. [Doctoral Dissertation]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,7450


Temple University

10. Zhang, Xuemei. Src Kinase Signaling Regulates Connective Tissue Growth Factor (CTGF/CCN2) Induction by Transforming Growth Factor-Beta 1 (TGF-b1) in Osteoblasts.

Degree: PhD, 2010, Temple University

Anatomy

Connective tissue growth factor (CTGF/CCN2) is a cysteine rich, extracellular matrix protein that acts as an anabolic growth factor to regulate osteoblast differentiation and… (more)

Subjects/Keywords: Biology; Cell

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APA (6th Edition):

Zhang, X. (2010). Src Kinase Signaling Regulates Connective Tissue Growth Factor (CTGF/CCN2) Induction by Transforming Growth Factor-Beta 1 (TGF-b1) in Osteoblasts. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,63095

Chicago Manual of Style (16th Edition):

Zhang, Xuemei. “Src Kinase Signaling Regulates Connective Tissue Growth Factor (CTGF/CCN2) Induction by Transforming Growth Factor-Beta 1 (TGF-b1) in Osteoblasts.” 2010. Doctoral Dissertation, Temple University. Accessed September 21, 2020. http://digital.library.temple.edu/u?/p245801coll10,63095.

MLA Handbook (7th Edition):

Zhang, Xuemei. “Src Kinase Signaling Regulates Connective Tissue Growth Factor (CTGF/CCN2) Induction by Transforming Growth Factor-Beta 1 (TGF-b1) in Osteoblasts.” 2010. Web. 21 Sep 2020.

Vancouver:

Zhang X. Src Kinase Signaling Regulates Connective Tissue Growth Factor (CTGF/CCN2) Induction by Transforming Growth Factor-Beta 1 (TGF-b1) in Osteoblasts. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,63095.

Council of Science Editors:

Zhang X. Src Kinase Signaling Regulates Connective Tissue Growth Factor (CTGF/CCN2) Induction by Transforming Growth Factor-Beta 1 (TGF-b1) in Osteoblasts. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,63095


Temple University

11. Singh, Maneet. TRANSCRIPTIONAL REGULATION OF OSTEOACTIVIN EXPRESSION BY BMP-2 IN OSTEOBLASTS.

Degree: PhD, 2011, Temple University

Cell Biology

Osteoactivin (OA) is a glycoprotein required for the differentiation of osteoblasts. In osteoblasts, Bone Morphogenetic Protein-2 (BMP-2) activated Smad1 signaling enhances OA expression.… (more)

Subjects/Keywords: Cellular Biology; Molecular Biology; Biology; HOMEDOMAIN PROTEIN; OSTEOACTIVIN; OSTEOBLAST DIFFERENTIATION; RUNX2; SMAD1 AND SMAD4; TRANSCRIPTIONAL REGULATION

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APA (6th Edition):

Singh, M. (2011). TRANSCRIPTIONAL REGULATION OF OSTEOACTIVIN EXPRESSION BY BMP-2 IN OSTEOBLASTS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,135232

Chicago Manual of Style (16th Edition):

Singh, Maneet. “TRANSCRIPTIONAL REGULATION OF OSTEOACTIVIN EXPRESSION BY BMP-2 IN OSTEOBLASTS.” 2011. Doctoral Dissertation, Temple University. Accessed September 21, 2020. http://digital.library.temple.edu/u?/p245801coll10,135232.

MLA Handbook (7th Edition):

Singh, Maneet. “TRANSCRIPTIONAL REGULATION OF OSTEOACTIVIN EXPRESSION BY BMP-2 IN OSTEOBLASTS.” 2011. Web. 21 Sep 2020.

Vancouver:

Singh M. TRANSCRIPTIONAL REGULATION OF OSTEOACTIVIN EXPRESSION BY BMP-2 IN OSTEOBLASTS. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2020 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,135232.

Council of Science Editors:

Singh M. TRANSCRIPTIONAL REGULATION OF OSTEOACTIVIN EXPRESSION BY BMP-2 IN OSTEOBLASTS. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,135232


Temple University

12. Belcher, Joyce Yvonne. Bone Cell Autonomous Effects of Osteoactivin In Vivo.

Degree: PhD, 2012, Temple University

Cell Biology

Osteoactivin (OA) is a type I transmembrane glycoprotein initially identified in bone in 2002. The protein is synthesized, processed and heavily glycosylated by… (more)

Subjects/Keywords: Cellular biology; Animal Model; Gpnmb; Osteoactivin; Osteoblasts; Osteoclasts; Osteopetrosis

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APA (6th Edition):

Belcher, J. Y. (2012). Bone Cell Autonomous Effects of Osteoactivin In Vivo. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,183061

Chicago Manual of Style (16th Edition):

Belcher, Joyce Yvonne. “Bone Cell Autonomous Effects of Osteoactivin In Vivo.” 2012. Doctoral Dissertation, Temple University. Accessed September 21, 2020. http://digital.library.temple.edu/u?/p245801coll10,183061.

MLA Handbook (7th Edition):

Belcher, Joyce Yvonne. “Bone Cell Autonomous Effects of Osteoactivin In Vivo.” 2012. Web. 21 Sep 2020.

Vancouver:

Belcher JY. Bone Cell Autonomous Effects of Osteoactivin In Vivo. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,183061.

Council of Science Editors:

Belcher JY. Bone Cell Autonomous Effects of Osteoactivin In Vivo. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,183061

.