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You searched for +publisher:"Temple University" +contributor:("Sabri, Abdelkarim"). Showing records 1 – 15 of 15 total matches.

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Temple University

1. Hooshdaran, Bahman. DUAL INHIBITION OF CATHEPSIN G AND CHYMASE AFTER ISCHEMIA REPERFUSION: THE ROLE OF INFLAMMATORY SERINE PROTEASES IN ISCHEMIA REPERFUSION INJURY.

Degree: PhD, 2017, Temple University

Bioengineering

Acute myocardial infarction (AMI) is a leading cause of morbidity and mortality in the world (4). Restoration of coronary flow to the ischemic myocardium… (more)

Subjects/Keywords: Engineering; Bioengineering;

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APA (6th Edition):

Hooshdaran, B. (2017). DUAL INHIBITION OF CATHEPSIN G AND CHYMASE AFTER ISCHEMIA REPERFUSION: THE ROLE OF INFLAMMATORY SERINE PROTEASES IN ISCHEMIA REPERFUSION INJURY. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,475423

Chicago Manual of Style (16th Edition):

Hooshdaran, Bahman. “DUAL INHIBITION OF CATHEPSIN G AND CHYMASE AFTER ISCHEMIA REPERFUSION: THE ROLE OF INFLAMMATORY SERINE PROTEASES IN ISCHEMIA REPERFUSION INJURY.” 2017. Doctoral Dissertation, Temple University. Accessed August 10, 2020. http://digital.library.temple.edu/u?/p245801coll10,475423.

MLA Handbook (7th Edition):

Hooshdaran, Bahman. “DUAL INHIBITION OF CATHEPSIN G AND CHYMASE AFTER ISCHEMIA REPERFUSION: THE ROLE OF INFLAMMATORY SERINE PROTEASES IN ISCHEMIA REPERFUSION INJURY.” 2017. Web. 10 Aug 2020.

Vancouver:

Hooshdaran B. DUAL INHIBITION OF CATHEPSIN G AND CHYMASE AFTER ISCHEMIA REPERFUSION: THE ROLE OF INFLAMMATORY SERINE PROTEASES IN ISCHEMIA REPERFUSION INJURY. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2020 Aug 10]. Available from: http://digital.library.temple.edu/u?/p245801coll10,475423.

Council of Science Editors:

Hooshdaran B. DUAL INHIBITION OF CATHEPSIN G AND CHYMASE AFTER ISCHEMIA REPERFUSION: THE ROLE OF INFLAMMATORY SERINE PROTEASES IN ISCHEMIA REPERFUSION INJURY. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,475423


Temple University

2. Buitrago Murcia, Claudia Lorena. Cbl proteins in platelet functional responses.

Degree: PhD, 2012, Temple University

Physiology

c-Cbl protein functions as an E3 ligase and scaffolding protein, where three residues, Y700, Y731, and Y774, upon phosphorylation, have been shown to initiate… (more)

Subjects/Keywords: Physiology; Cellular biology; Cbl; hemostasis; kinases; platelets; signaling

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APA (6th Edition):

Buitrago Murcia, C. L. (2012). Cbl proteins in platelet functional responses. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,198139

Chicago Manual of Style (16th Edition):

Buitrago Murcia, Claudia Lorena. “Cbl proteins in platelet functional responses.” 2012. Doctoral Dissertation, Temple University. Accessed August 10, 2020. http://digital.library.temple.edu/u?/p245801coll10,198139.

MLA Handbook (7th Edition):

Buitrago Murcia, Claudia Lorena. “Cbl proteins in platelet functional responses.” 2012. Web. 10 Aug 2020.

Vancouver:

Buitrago Murcia CL. Cbl proteins in platelet functional responses. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Aug 10]. Available from: http://digital.library.temple.edu/u?/p245801coll10,198139.

Council of Science Editors:

Buitrago Murcia CL. Cbl proteins in platelet functional responses. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,198139


Temple University

3. Wang, Fang. DOES CALCIUM INFLUX THROUGH T-TYPE CALCIUM CHANNEL INDUCE CARDIOMYOCYTE PROLIFERATION?.

Degree: PhD, 2012, Temple University

Physiology

Cardiovascular disease remains the number one cause or mortally in the western world. Heart failure is the most rapidly growing cardiovascular disease (Hobbs, 2004;… (more)

Subjects/Keywords: Physiology; Calcium Current; Cardiomyocyte Proliferation; Electrophysiology; Flow Cytometry; Neonatal Mice Ventricular Cardiomyocyte; T-type calcium channel

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APA (6th Edition):

Wang, F. (2012). DOES CALCIUM INFLUX THROUGH T-TYPE CALCIUM CHANNEL INDUCE CARDIOMYOCYTE PROLIFERATION?. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,214814

Chicago Manual of Style (16th Edition):

Wang, Fang. “DOES CALCIUM INFLUX THROUGH T-TYPE CALCIUM CHANNEL INDUCE CARDIOMYOCYTE PROLIFERATION?.” 2012. Doctoral Dissertation, Temple University. Accessed August 10, 2020. http://digital.library.temple.edu/u?/p245801coll10,214814.

MLA Handbook (7th Edition):

Wang, Fang. “DOES CALCIUM INFLUX THROUGH T-TYPE CALCIUM CHANNEL INDUCE CARDIOMYOCYTE PROLIFERATION?.” 2012. Web. 10 Aug 2020.

Vancouver:

Wang F. DOES CALCIUM INFLUX THROUGH T-TYPE CALCIUM CHANNEL INDUCE CARDIOMYOCYTE PROLIFERATION?. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Aug 10]. Available from: http://digital.library.temple.edu/u?/p245801coll10,214814.

Council of Science Editors:

Wang F. DOES CALCIUM INFLUX THROUGH T-TYPE CALCIUM CHANNEL INDUCE CARDIOMYOCYTE PROLIFERATION?. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,214814


Temple University

4. Bhavanasi, Dheeraj. Role of Protein Kinase C delta in regulating platelet functional responses.

Degree: PhD, 2014, Temple University

Physiology

Platelets upon activation change their shape, aggregate and secrete alpha and dense granule contents among which ADP acts as a feedback activator. Different Protein… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Bhavanasi, D. (2014). Role of Protein Kinase C delta in regulating platelet functional responses. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,283099

Chicago Manual of Style (16th Edition):

Bhavanasi, Dheeraj. “Role of Protein Kinase C delta in regulating platelet functional responses.” 2014. Doctoral Dissertation, Temple University. Accessed August 10, 2020. http://digital.library.temple.edu/u?/p245801coll10,283099.

MLA Handbook (7th Edition):

Bhavanasi, Dheeraj. “Role of Protein Kinase C delta in regulating platelet functional responses.” 2014. Web. 10 Aug 2020.

Vancouver:

Bhavanasi D. Role of Protein Kinase C delta in regulating platelet functional responses. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2020 Aug 10]. Available from: http://digital.library.temple.edu/u?/p245801coll10,283099.

Council of Science Editors:

Bhavanasi D. Role of Protein Kinase C delta in regulating platelet functional responses. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,283099


Temple University

5. BADOLIA, RACHIT. MOLECULAR PHYSIOLOGY OF p21-ACTIVATED KINASES.

Degree: PhD, 2015, Temple University

Organ Systems & Translational Medicine

Platelets are involved in many processes ranging from fighting microbial infections and triggering inflammation to promoting tumor angiogenesis and metastasis.… (more)

Subjects/Keywords: Physiology;

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APA (6th Edition):

BADOLIA, R. (2015). MOLECULAR PHYSIOLOGY OF p21-ACTIVATED KINASES. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,368893

Chicago Manual of Style (16th Edition):

BADOLIA, RACHIT. “MOLECULAR PHYSIOLOGY OF p21-ACTIVATED KINASES.” 2015. Doctoral Dissertation, Temple University. Accessed August 10, 2020. http://digital.library.temple.edu/u?/p245801coll10,368893.

MLA Handbook (7th Edition):

BADOLIA, RACHIT. “MOLECULAR PHYSIOLOGY OF p21-ACTIVATED KINASES.” 2015. Web. 10 Aug 2020.

Vancouver:

BADOLIA R. MOLECULAR PHYSIOLOGY OF p21-ACTIVATED KINASES. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Aug 10]. Available from: http://digital.library.temple.edu/u?/p245801coll10,368893.

Council of Science Editors:

BADOLIA R. MOLECULAR PHYSIOLOGY OF p21-ACTIVATED KINASES. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,368893


Temple University

6. Powers, Jeffery. microRNA-21 as a Pro-Fibrotic Mediator in Right Ventricular Failure.

Degree: PhD, 2016, Temple University

Physiology

Historical emphasis on the left ventricle (LV) has left clinicians with a lack of efficacious right ventricle (RV)-specific therapies, and classical pharmacological interventions for… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Powers, J. (2016). microRNA-21 as a Pro-Fibrotic Mediator in Right Ventricular Failure. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,417461

Chicago Manual of Style (16th Edition):

Powers, Jeffery. “microRNA-21 as a Pro-Fibrotic Mediator in Right Ventricular Failure.” 2016. Doctoral Dissertation, Temple University. Accessed August 10, 2020. http://digital.library.temple.edu/u?/p245801coll10,417461.

MLA Handbook (7th Edition):

Powers, Jeffery. “microRNA-21 as a Pro-Fibrotic Mediator in Right Ventricular Failure.” 2016. Web. 10 Aug 2020.

Vancouver:

Powers J. microRNA-21 as a Pro-Fibrotic Mediator in Right Ventricular Failure. [Internet] [Doctoral dissertation]. Temple University; 2016. [cited 2020 Aug 10]. Available from: http://digital.library.temple.edu/u?/p245801coll10,417461.

Council of Science Editors:

Powers J. microRNA-21 as a Pro-Fibrotic Mediator in Right Ventricular Failure. [Doctoral Dissertation]. Temple University; 2016. Available from: http://digital.library.temple.edu/u?/p245801coll10,417461


Temple University

7. Jaleel, Naser. Re-Expression of T-Type Calcium Channels Minimally Affects Cardiac Contractility and Activates Pro-Survival Signaling Pathways in the Myocardium.

Degree: PhD, 2010, Temple University

Physiology

The role of T-type calcium channels (TTCCs) in the heart is unclear. TTCCs are transiently expressed throughout the neonatal heart during a period of… (more)

Subjects/Keywords: Biology, Physiology; Biology, General; Cardiac Electrophysiology; Cardiac Hypertrophy; Excitation Contraction Coupling; L-Type Calcium Channels; T-type Calcium Channels

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APA (6th Edition):

Jaleel, N. (2010). Re-Expression of T-Type Calcium Channels Minimally Affects Cardiac Contractility and Activates Pro-Survival Signaling Pathways in the Myocardium. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,83093

Chicago Manual of Style (16th Edition):

Jaleel, Naser. “Re-Expression of T-Type Calcium Channels Minimally Affects Cardiac Contractility and Activates Pro-Survival Signaling Pathways in the Myocardium.” 2010. Doctoral Dissertation, Temple University. Accessed August 10, 2020. http://digital.library.temple.edu/u?/p245801coll10,83093.

MLA Handbook (7th Edition):

Jaleel, Naser. “Re-Expression of T-Type Calcium Channels Minimally Affects Cardiac Contractility and Activates Pro-Survival Signaling Pathways in the Myocardium.” 2010. Web. 10 Aug 2020.

Vancouver:

Jaleel N. Re-Expression of T-Type Calcium Channels Minimally Affects Cardiac Contractility and Activates Pro-Survival Signaling Pathways in the Myocardium. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Aug 10]. Available from: http://digital.library.temple.edu/u?/p245801coll10,83093.

Council of Science Editors:

Jaleel N. Re-Expression of T-Type Calcium Channels Minimally Affects Cardiac Contractility and Activates Pro-Survival Signaling Pathways in the Myocardium. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,83093


Temple University

8. Zhang, Hongyu. INCREASING MYOCYTE CONTRACTILITY EXACERBATES CARDIAC INJURY AND PUMP DYSFUNCTION AND ABLATION OF PHOSPHORYLATION.

Degree: PhD, 2010, Temple University

Physiology

Myocardial infarction (MI) leads to heart failure (HF) and premature death. The respective roles of myocyte death and depressed myocyte contractility in the induction… (more)

Subjects/Keywords: Biology, Physiology; cardiac function; cell death; heart failure; myocardial infarction; myocyte contractility; ryanodine receptor

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APA (6th Edition):

Zhang, H. (2010). INCREASING MYOCYTE CONTRACTILITY EXACERBATES CARDIAC INJURY AND PUMP DYSFUNCTION AND ABLATION OF PHOSPHORYLATION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,90859

Chicago Manual of Style (16th Edition):

Zhang, Hongyu. “INCREASING MYOCYTE CONTRACTILITY EXACERBATES CARDIAC INJURY AND PUMP DYSFUNCTION AND ABLATION OF PHOSPHORYLATION.” 2010. Doctoral Dissertation, Temple University. Accessed August 10, 2020. http://digital.library.temple.edu/u?/p245801coll10,90859.

MLA Handbook (7th Edition):

Zhang, Hongyu. “INCREASING MYOCYTE CONTRACTILITY EXACERBATES CARDIAC INJURY AND PUMP DYSFUNCTION AND ABLATION OF PHOSPHORYLATION.” 2010. Web. 10 Aug 2020.

Vancouver:

Zhang H. INCREASING MYOCYTE CONTRACTILITY EXACERBATES CARDIAC INJURY AND PUMP DYSFUNCTION AND ABLATION OF PHOSPHORYLATION. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Aug 10]. Available from: http://digital.library.temple.edu/u?/p245801coll10,90859.

Council of Science Editors:

Zhang H. INCREASING MYOCYTE CONTRACTILITY EXACERBATES CARDIAC INJURY AND PUMP DYSFUNCTION AND ABLATION OF PHOSPHORYLATION. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,90859


Temple University

9. Mayanglambam, Azad. Regulation of Protein Kinases (Syk and PKC zeta) in platelets.

Degree: PhD, 2010, Temple University

Physiology

Platelets are crucial components of the hemostatic machinery of the body. When the endothelial continuity is disrupted due to injury or atherosclerotic plaque rupture,… (more)

Subjects/Keywords: Biology, Physiology; Biology, Molecular; Hemostasis; Platelets; Protein Kinase C; Signaling; Syk; Thrombosis

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APA (6th Edition):

Mayanglambam, A. (2010). Regulation of Protein Kinases (Syk and PKC zeta) in platelets. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,91635

Chicago Manual of Style (16th Edition):

Mayanglambam, Azad. “Regulation of Protein Kinases (Syk and PKC zeta) in platelets.” 2010. Doctoral Dissertation, Temple University. Accessed August 10, 2020. http://digital.library.temple.edu/u?/p245801coll10,91635.

MLA Handbook (7th Edition):

Mayanglambam, Azad. “Regulation of Protein Kinases (Syk and PKC zeta) in platelets.” 2010. Web. 10 Aug 2020.

Vancouver:

Mayanglambam A. Regulation of Protein Kinases (Syk and PKC zeta) in platelets. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Aug 10]. Available from: http://digital.library.temple.edu/u?/p245801coll10,91635.

Council of Science Editors:

Mayanglambam A. Regulation of Protein Kinases (Syk and PKC zeta) in platelets. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,91635


Temple University

10. Angert, David W. Repair of the Injured Adult Heart Involves Resident Cardiac Stem Cell Derived New Myocytes.

Degree: PhD, 2011, Temple University

Physiology

The ability of the adult heart to generate new myocytes after injury is not established. Our purpose was to determine if the adult heart… (more)

Subjects/Keywords: Physiology; Cardiac; Catecholamine; Endogenous; Injury; Isoproterenol; Regeneration

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APA (6th Edition):

Angert, D. W. (2011). Repair of the Injured Adult Heart Involves Resident Cardiac Stem Cell Derived New Myocytes. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,125347

Chicago Manual of Style (16th Edition):

Angert, David W. “Repair of the Injured Adult Heart Involves Resident Cardiac Stem Cell Derived New Myocytes.” 2011. Doctoral Dissertation, Temple University. Accessed August 10, 2020. http://digital.library.temple.edu/u?/p245801coll10,125347.

MLA Handbook (7th Edition):

Angert, David W. “Repair of the Injured Adult Heart Involves Resident Cardiac Stem Cell Derived New Myocytes.” 2011. Web. 10 Aug 2020.

Vancouver:

Angert DW. Repair of the Injured Adult Heart Involves Resident Cardiac Stem Cell Derived New Myocytes. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2020 Aug 10]. Available from: http://digital.library.temple.edu/u?/p245801coll10,125347.

Council of Science Editors:

Angert DW. Repair of the Injured Adult Heart Involves Resident Cardiac Stem Cell Derived New Myocytes. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,125347


Temple University

11. Li, Yingxin. THE ROLES OF Cav3.1/a1G T-TYPE CALCIUM CHANNEL IN HEART RATE GENERATION, REGULATION AND CARDIAC ARRHYTHMIAS.

Degree: PhD, 2011, Temple University

Physiology

T-type Ca²+ channels (TTCCs) are expressed in cardiac pacemaker cells and conduction system of mammals. However, the roles of TTCCs in heart rate (HR)… (more)

Subjects/Keywords: Physiology; arrhythmia; Cav3.1/a1G; heart rate

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APA (6th Edition):

Li, Y. (2011). THE ROLES OF Cav3.1/a1G T-TYPE CALCIUM CHANNEL IN HEART RATE GENERATION, REGULATION AND CARDIAC ARRHYTHMIAS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,151806

Chicago Manual of Style (16th Edition):

Li, Yingxin. “THE ROLES OF Cav3.1/a1G T-TYPE CALCIUM CHANNEL IN HEART RATE GENERATION, REGULATION AND CARDIAC ARRHYTHMIAS.” 2011. Doctoral Dissertation, Temple University. Accessed August 10, 2020. http://digital.library.temple.edu/u?/p245801coll10,151806.

MLA Handbook (7th Edition):

Li, Yingxin. “THE ROLES OF Cav3.1/a1G T-TYPE CALCIUM CHANNEL IN HEART RATE GENERATION, REGULATION AND CARDIAC ARRHYTHMIAS.” 2011. Web. 10 Aug 2020.

Vancouver:

Li Y. THE ROLES OF Cav3.1/a1G T-TYPE CALCIUM CHANNEL IN HEART RATE GENERATION, REGULATION AND CARDIAC ARRHYTHMIAS. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2020 Aug 10]. Available from: http://digital.library.temple.edu/u?/p245801coll10,151806.

Council of Science Editors:

Li Y. THE ROLES OF Cav3.1/a1G T-TYPE CALCIUM CHANNEL IN HEART RATE GENERATION, REGULATION AND CARDIAC ARRHYTHMIAS. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,151806


Temple University

12. Cheheltani, Rabee. DEVELOPMENT OF INFRARED SPECTROSCOPIC METHODS FOR ASSESSMENT OF EXTRACELLULAR MATRIX CHANGES IN CARDIOVASCULAR DISEASES.

Degree: PhD, 2014, Temple University

Mechanical Engineering

Extracellular matrix (ECM) is a key component and regulator of many biological tissues. Several cardiovascular pathologies are associated with significant changes in the… (more)

Subjects/Keywords: Biomedical engineering; Mechanical engineering; Analytical chemistry;

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APA (6th Edition):

Cheheltani, R. (2014). DEVELOPMENT OF INFRARED SPECTROSCOPIC METHODS FOR ASSESSMENT OF EXTRACELLULAR MATRIX CHANGES IN CARDIOVASCULAR DISEASES. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,255000

Chicago Manual of Style (16th Edition):

Cheheltani, Rabee. “DEVELOPMENT OF INFRARED SPECTROSCOPIC METHODS FOR ASSESSMENT OF EXTRACELLULAR MATRIX CHANGES IN CARDIOVASCULAR DISEASES.” 2014. Doctoral Dissertation, Temple University. Accessed August 10, 2020. http://digital.library.temple.edu/u?/p245801coll10,255000.

MLA Handbook (7th Edition):

Cheheltani, Rabee. “DEVELOPMENT OF INFRARED SPECTROSCOPIC METHODS FOR ASSESSMENT OF EXTRACELLULAR MATRIX CHANGES IN CARDIOVASCULAR DISEASES.” 2014. Web. 10 Aug 2020.

Vancouver:

Cheheltani R. DEVELOPMENT OF INFRARED SPECTROSCOPIC METHODS FOR ASSESSMENT OF EXTRACELLULAR MATRIX CHANGES IN CARDIOVASCULAR DISEASES. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2020 Aug 10]. Available from: http://digital.library.temple.edu/u?/p245801coll10,255000.

Council of Science Editors:

Cheheltani R. DEVELOPMENT OF INFRARED SPECTROSCOPIC METHODS FOR ASSESSMENT OF EXTRACELLULAR MATRIX CHANGES IN CARDIOVASCULAR DISEASES. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,255000


Temple University

13. Manne, Bhanu Kanth. CLEC-2 SIGNAL TRANSDUCTION IN PLATELET ACTIVATION.

Degree: PhD, 2015, Temple University

Physiology

Platelets are involved in many processes ranging from fighting microbial infections and triggering inflammation to promoting tumor angiogenesis and metastasis. Nevertheless, the primary physiological… (more)

Subjects/Keywords: Physiology; Cellular biology;

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APA (6th Edition):

Manne, B. K. (2015). CLEC-2 SIGNAL TRANSDUCTION IN PLATELET ACTIVATION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,340495

Chicago Manual of Style (16th Edition):

Manne, Bhanu Kanth. “CLEC-2 SIGNAL TRANSDUCTION IN PLATELET ACTIVATION.” 2015. Doctoral Dissertation, Temple University. Accessed August 10, 2020. http://digital.library.temple.edu/u?/p245801coll10,340495.

MLA Handbook (7th Edition):

Manne, Bhanu Kanth. “CLEC-2 SIGNAL TRANSDUCTION IN PLATELET ACTIVATION.” 2015. Web. 10 Aug 2020.

Vancouver:

Manne BK. CLEC-2 SIGNAL TRANSDUCTION IN PLATELET ACTIVATION. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Aug 10]. Available from: http://digital.library.temple.edu/u?/p245801coll10,340495.

Council of Science Editors:

Manne BK. CLEC-2 SIGNAL TRANSDUCTION IN PLATELET ACTIVATION. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,340495


Temple University

14. Sharp III, Thomas E. DRUG AND CELL–BASED THERAPIES TO REDUCE PATHOLOGICAL REMODELING AND CARDIAC DYSFUNCTION AFTER ACUTE MYOCARDIAL INFARCTION.

Degree: PhD, 2017, Temple University

Physiology

Remarkable advances have been made in the treatment of cardiovascular diseases (CVD), however, CVD still accounts for the most deaths in industrialized nations. Ischemic… (more)

Subjects/Keywords: Physiology;

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APA (6th Edition):

Sharp III, T. E. (2017). DRUG AND CELL–BASED THERAPIES TO REDUCE PATHOLOGICAL REMODELING AND CARDIAC DYSFUNCTION AFTER ACUTE MYOCARDIAL INFARCTION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,445275

Chicago Manual of Style (16th Edition):

Sharp III, Thomas E. “DRUG AND CELL–BASED THERAPIES TO REDUCE PATHOLOGICAL REMODELING AND CARDIAC DYSFUNCTION AFTER ACUTE MYOCARDIAL INFARCTION.” 2017. Doctoral Dissertation, Temple University. Accessed August 10, 2020. http://digital.library.temple.edu/u?/p245801coll10,445275.

MLA Handbook (7th Edition):

Sharp III, Thomas E. “DRUG AND CELL–BASED THERAPIES TO REDUCE PATHOLOGICAL REMODELING AND CARDIAC DYSFUNCTION AFTER ACUTE MYOCARDIAL INFARCTION.” 2017. Web. 10 Aug 2020.

Vancouver:

Sharp III TE. DRUG AND CELL–BASED THERAPIES TO REDUCE PATHOLOGICAL REMODELING AND CARDIAC DYSFUNCTION AFTER ACUTE MYOCARDIAL INFARCTION. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2020 Aug 10]. Available from: http://digital.library.temple.edu/u?/p245801coll10,445275.

Council of Science Editors:

Sharp III TE. DRUG AND CELL–BASED THERAPIES TO REDUCE PATHOLOGICAL REMODELING AND CARDIAC DYSFUNCTION AFTER ACUTE MYOCARDIAL INFARCTION. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,445275


Temple University

15. Patel, Akruti. NOVEL REGULATORS OF GPVI-MEDIATED PLATELET ACTIVATION.

Degree: PhD, 2017, Temple University

Biomedical Sciences

Platelets are anucleate cells that are crucial mediators of hemostasis and thrombosis. Under physiological conditions, platelets are maintained in a quiescent state within… (more)

Subjects/Keywords: Physiology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Patel, A. (2017). NOVEL REGULATORS OF GPVI-MEDIATED PLATELET ACTIVATION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,473397

Chicago Manual of Style (16th Edition):

Patel, Akruti. “NOVEL REGULATORS OF GPVI-MEDIATED PLATELET ACTIVATION.” 2017. Doctoral Dissertation, Temple University. Accessed August 10, 2020. http://digital.library.temple.edu/u?/p245801coll10,473397.

MLA Handbook (7th Edition):

Patel, Akruti. “NOVEL REGULATORS OF GPVI-MEDIATED PLATELET ACTIVATION.” 2017. Web. 10 Aug 2020.

Vancouver:

Patel A. NOVEL REGULATORS OF GPVI-MEDIATED PLATELET ACTIVATION. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2020 Aug 10]. Available from: http://digital.library.temple.edu/u?/p245801coll10,473397.

Council of Science Editors:

Patel A. NOVEL REGULATORS OF GPVI-MEDIATED PLATELET ACTIVATION. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,473397

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