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You searched for +publisher:"Temple University" +contributor:("Rogers, Thomas J."). Showing records 1 – 8 of 8 total matches.

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Temple University

1. Adhikary, Sabina. CANNABINOID RECEPTOR 2 AGONIST REDUCES IMMUNE CELL MIGRATION IN NEUROINFLAMMATION VIA INHIBITION OF MATRIX METALLOPROTEINASE-9.

Degree: PhD, 2013, Temple University

Physiology

Several studies have reported that administration of cannabinoid receptor agonists in inflammatory/autoimmune and CNS injury models resulted in significant attenuation of clinical disease. The… (more)

Subjects/Keywords: Immunology; Cannabinoid Receptor 2; Chemokines; Dendritic cell migration; Matrix Metalloproteinase 9; Multiple sclerosis; Spinal cord injury

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APA (6th Edition):

Adhikary, S. (2013). CANNABINOID RECEPTOR 2 AGONIST REDUCES IMMUNE CELL MIGRATION IN NEUROINFLAMMATION VIA INHIBITION OF MATRIX METALLOPROTEINASE-9. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,214799

Chicago Manual of Style (16th Edition):

Adhikary, Sabina. “CANNABINOID RECEPTOR 2 AGONIST REDUCES IMMUNE CELL MIGRATION IN NEUROINFLAMMATION VIA INHIBITION OF MATRIX METALLOPROTEINASE-9.” 2013. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,214799.

MLA Handbook (7th Edition):

Adhikary, Sabina. “CANNABINOID RECEPTOR 2 AGONIST REDUCES IMMUNE CELL MIGRATION IN NEUROINFLAMMATION VIA INHIBITION OF MATRIX METALLOPROTEINASE-9.” 2013. Web. 21 Oct 2019.

Vancouver:

Adhikary S. CANNABINOID RECEPTOR 2 AGONIST REDUCES IMMUNE CELL MIGRATION IN NEUROINFLAMMATION VIA INHIBITION OF MATRIX METALLOPROTEINASE-9. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,214799.

Council of Science Editors:

Adhikary S. CANNABINOID RECEPTOR 2 AGONIST REDUCES IMMUNE CELL MIGRATION IN NEUROINFLAMMATION VIA INHIBITION OF MATRIX METALLOPROTEINASE-9. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,214799


Temple University

2. Robinson, Rebecca Hartzell. Cannabinoid Receptor 2-Selective Ligands as Immunosuppressive Compounds: Utility in Graft Rejection.

Degree: PhD, 2014, Temple University

Microbiology and Immunology

Cannabinoids are known to have anti-inflammatory and immunomodulatory properties. Cannabinoid receptor 2 (CB2) is expressed mainly on leukocytes and is the receptor… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Robinson, R. H. (2014). Cannabinoid Receptor 2-Selective Ligands as Immunosuppressive Compounds: Utility in Graft Rejection. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,246094

Chicago Manual of Style (16th Edition):

Robinson, Rebecca Hartzell. “Cannabinoid Receptor 2-Selective Ligands as Immunosuppressive Compounds: Utility in Graft Rejection.” 2014. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,246094.

MLA Handbook (7th Edition):

Robinson, Rebecca Hartzell. “Cannabinoid Receptor 2-Selective Ligands as Immunosuppressive Compounds: Utility in Graft Rejection.” 2014. Web. 21 Oct 2019.

Vancouver:

Robinson RH. Cannabinoid Receptor 2-Selective Ligands as Immunosuppressive Compounds: Utility in Graft Rejection. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,246094.

Council of Science Editors:

Robinson RH. Cannabinoid Receptor 2-Selective Ligands as Immunosuppressive Compounds: Utility in Graft Rejection. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,246094


Temple University

3. Chatila, Wissam M. F. MicroRNA Expression in Regulatory T Cells in Chronic Obstructive Pulmonary Disease.

Degree: PhD, 2015, Temple University

Microbiology and Immunology

COPD is characterized by an abnormal regulatory T cell (Treg) response with a shift towards a Th1 and Th17 cell responses. However,… (more)

Subjects/Keywords: Immunology; Cellular biology; Molecular biology;

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APA (6th Edition):

Chatila, W. M. F. (2015). MicroRNA Expression in Regulatory T Cells in Chronic Obstructive Pulmonary Disease. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,333572

Chicago Manual of Style (16th Edition):

Chatila, Wissam M F. “MicroRNA Expression in Regulatory T Cells in Chronic Obstructive Pulmonary Disease.” 2015. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,333572.

MLA Handbook (7th Edition):

Chatila, Wissam M F. “MicroRNA Expression in Regulatory T Cells in Chronic Obstructive Pulmonary Disease.” 2015. Web. 21 Oct 2019.

Vancouver:

Chatila WMF. MicroRNA Expression in Regulatory T Cells in Chronic Obstructive Pulmonary Disease. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,333572.

Council of Science Editors:

Chatila WMF. MicroRNA Expression in Regulatory T Cells in Chronic Obstructive Pulmonary Disease. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,333572


Temple University

4. Chang, Jen-Kuan. The Biochemical Basis of The miR-21 Expression by The Mu-Opioid Receptor.

Degree: PhD, 2015, Temple University

Molecular Biology and Genetics

Opioid receptors are members of the superfamily of seven transmembrane G protein-coupled receptors (GPCRs) which share several structural and functional characteristics.… (more)

Subjects/Keywords: Molecular biology; Genetics; Immunology;

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APA (6th Edition):

Chang, J. (2015). The Biochemical Basis of The miR-21 Expression by The Mu-Opioid Receptor. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,352105

Chicago Manual of Style (16th Edition):

Chang, Jen-Kuan. “The Biochemical Basis of The miR-21 Expression by The Mu-Opioid Receptor.” 2015. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,352105.

MLA Handbook (7th Edition):

Chang, Jen-Kuan. “The Biochemical Basis of The miR-21 Expression by The Mu-Opioid Receptor.” 2015. Web. 21 Oct 2019.

Vancouver:

Chang J. The Biochemical Basis of The miR-21 Expression by The Mu-Opioid Receptor. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,352105.

Council of Science Editors:

Chang J. The Biochemical Basis of The miR-21 Expression by The Mu-Opioid Receptor. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,352105


Temple University

5. Kotredes, Kevin Patrick. Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression.

Degree: PhD, 2015, Temple University

Molecular Biology and Genetics

The isocitrate dehydrogenase (IDH) family of enzymes is central to cellular metabolism, catalyzing the oxidative decarboxylation of isocitrate to α-ketoglutarate (α-KG)… (more)

Subjects/Keywords: Molecular biology; Genetics; Biochemistry;

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APA (6th Edition):

Kotredes, K. P. (2015). Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,356693

Chicago Manual of Style (16th Edition):

Kotredes, Kevin Patrick. “Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression.” 2015. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,356693.

MLA Handbook (7th Edition):

Kotredes, Kevin Patrick. “Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression.” 2015. Web. 21 Oct 2019.

Vancouver:

Kotredes KP. Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,356693.

Council of Science Editors:

Kotredes KP. Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,356693


Temple University

6. Happel, Christine. Molecular Basis for Mu-Opioid Regulation of Chemokine Gene Expression.

Degree: PhD, 2009, Temple University

Molecular Biology and Genetics

Opioid receptor modulation of pro-inflammatory cytokine production is vital for host defense and the inflammatory response. Previous results have shown the… (more)

Subjects/Keywords: Biology, Microbiology; Chemokines; NF-kappaB; Opioid; Opioid receptor; PKC

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APA (6th Edition):

Happel, C. (2009). Molecular Basis for Mu-Opioid Regulation of Chemokine Gene Expression. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,28438

Chicago Manual of Style (16th Edition):

Happel, Christine. “Molecular Basis for Mu-Opioid Regulation of Chemokine Gene Expression.” 2009. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,28438.

MLA Handbook (7th Edition):

Happel, Christine. “Molecular Basis for Mu-Opioid Regulation of Chemokine Gene Expression.” 2009. Web. 21 Oct 2019.

Vancouver:

Happel C. Molecular Basis for Mu-Opioid Regulation of Chemokine Gene Expression. [Internet] [Doctoral dissertation]. Temple University; 2009. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,28438.

Council of Science Editors:

Happel C. Molecular Basis for Mu-Opioid Regulation of Chemokine Gene Expression. [Doctoral Dissertation]. Temple University; 2009. Available from: http://digital.library.temple.edu/u?/p245801coll10,28438


Temple University

7. Finley, Matthew James. Molecular Basis for Kappa-Opioid Regulation of Chemokine Receptor Function.

Degree: PhD, 2009, Temple University

Molecular Biology and Genetics

Opioid receptor-mediated regulation of chemokine receptors is vital for the host immune response, development, and neurological function. Previous studies have demonstrated… (more)

Subjects/Keywords: Biology, Molecular; Biology, Genetics; Biology, Neuroscience; Chemokine; Expression; Interferon Regulatory Factor; Opioid, Regulation; STAT3

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APA (6th Edition):

Finley, M. J. (2009). Molecular Basis for Kappa-Opioid Regulation of Chemokine Receptor Function. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,62878

Chicago Manual of Style (16th Edition):

Finley, Matthew James. “Molecular Basis for Kappa-Opioid Regulation of Chemokine Receptor Function.” 2009. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,62878.

MLA Handbook (7th Edition):

Finley, Matthew James. “Molecular Basis for Kappa-Opioid Regulation of Chemokine Receptor Function.” 2009. Web. 21 Oct 2019.

Vancouver:

Finley MJ. Molecular Basis for Kappa-Opioid Regulation of Chemokine Receptor Function. [Internet] [Doctoral dissertation]. Temple University; 2009. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,62878.

Council of Science Editors:

Finley MJ. Molecular Basis for Kappa-Opioid Regulation of Chemokine Receptor Function. [Doctoral Dissertation]. Temple University; 2009. Available from: http://digital.library.temple.edu/u?/p245801coll10,62878


Temple University

8. Shrestha, Jenny. HIV-1 gp120 Mediated Neuronal Deregulation: Unraveling the Molecular Mechanisms Involved.

Degree: PhD, 2016, Temple University

Molecular Biology and Genetics

The advancement in combinatory antiretroviral therapy (cART) has granted people with HIV-1 an improved lifespan by decreasing the likelihood of AIDS-defining… (more)

Subjects/Keywords: Molecular biology

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APA (6th Edition):

Shrestha, J. (2016). HIV-1 gp120 Mediated Neuronal Deregulation: Unraveling the Molecular Mechanisms Involved. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,402666

Chicago Manual of Style (16th Edition):

Shrestha, Jenny. “HIV-1 gp120 Mediated Neuronal Deregulation: Unraveling the Molecular Mechanisms Involved.” 2016. Doctoral Dissertation, Temple University. Accessed October 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,402666.

MLA Handbook (7th Edition):

Shrestha, Jenny. “HIV-1 gp120 Mediated Neuronal Deregulation: Unraveling the Molecular Mechanisms Involved.” 2016. Web. 21 Oct 2019.

Vancouver:

Shrestha J. HIV-1 gp120 Mediated Neuronal Deregulation: Unraveling the Molecular Mechanisms Involved. [Internet] [Doctoral dissertation]. Temple University; 2016. [cited 2019 Oct 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,402666.

Council of Science Editors:

Shrestha J. HIV-1 gp120 Mediated Neuronal Deregulation: Unraveling the Molecular Mechanisms Involved. [Doctoral Dissertation]. Temple University; 2016. Available from: http://digital.library.temple.edu/u?/p245801coll10,402666

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