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You searched for +publisher:"Temple University" +contributor:("Rizzo, Victor;"). Showing records 1 – 22 of 22 total matches.

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Temple University

1. Forrester, Steven James. MITOCHONDRIA FACILITATE VASCULAR INFLAMMATION: THE ROLE OF CANONICAL INFLAMMATORY SIGNALING IN THE REGULATION OF MITOCHONDRIAL MORPHOLOGY.

Degree: PhD, 2017, Temple University

Kinesiology

Vascular inflammation is an underlying cause to numerous diseases and is characterized by classical NF-κB activation and downstream physiological responses including inflammatory gene induction… (more)

Subjects/Keywords: Physiology; Cellular biology; Medicine

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APA (6th Edition):

Forrester, S. J. (2017). MITOCHONDRIA FACILITATE VASCULAR INFLAMMATION: THE ROLE OF CANONICAL INFLAMMATORY SIGNALING IN THE REGULATION OF MITOCHONDRIAL MORPHOLOGY. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,429386

Chicago Manual of Style (16th Edition):

Forrester, Steven James. “MITOCHONDRIA FACILITATE VASCULAR INFLAMMATION: THE ROLE OF CANONICAL INFLAMMATORY SIGNALING IN THE REGULATION OF MITOCHONDRIAL MORPHOLOGY.” 2017. Doctoral Dissertation, Temple University. Accessed October 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,429386.

MLA Handbook (7th Edition):

Forrester, Steven James. “MITOCHONDRIA FACILITATE VASCULAR INFLAMMATION: THE ROLE OF CANONICAL INFLAMMATORY SIGNALING IN THE REGULATION OF MITOCHONDRIAL MORPHOLOGY.” 2017. Web. 22 Oct 2019.

Vancouver:

Forrester SJ. MITOCHONDRIA FACILITATE VASCULAR INFLAMMATION: THE ROLE OF CANONICAL INFLAMMATORY SIGNALING IN THE REGULATION OF MITOCHONDRIAL MORPHOLOGY. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2019 Oct 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,429386.

Council of Science Editors:

Forrester SJ. MITOCHONDRIA FACILITATE VASCULAR INFLAMMATION: THE ROLE OF CANONICAL INFLAMMATORY SIGNALING IN THE REGULATION OF MITOCHONDRIAL MORPHOLOGY. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,429386


Temple University

2. England, Ross N. Cellular Mechanisms of the Anti-Inflammatory Effects of Interleukin-19.

Degree: PhD, 2015, Temple University

Physiology

BACKGROUND: Atherosclerotic vascular disease is a significant medical and socioeconomic problem and contributes to mortality in multiple diseases including myocardial infarction (MI), stroke, renal… (more)

Subjects/Keywords: Physiology; Pathology; Molecular biology;

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APA (6th Edition):

England, R. N. (2015). Cellular Mechanisms of the Anti-Inflammatory Effects of Interleukin-19. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,216875

Chicago Manual of Style (16th Edition):

England, Ross N. “Cellular Mechanisms of the Anti-Inflammatory Effects of Interleukin-19.” 2015. Doctoral Dissertation, Temple University. Accessed October 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,216875.

MLA Handbook (7th Edition):

England, Ross N. “Cellular Mechanisms of the Anti-Inflammatory Effects of Interleukin-19.” 2015. Web. 22 Oct 2019.

Vancouver:

England RN. Cellular Mechanisms of the Anti-Inflammatory Effects of Interleukin-19. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Oct 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,216875.

Council of Science Editors:

England RN. Cellular Mechanisms of the Anti-Inflammatory Effects of Interleukin-19. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,216875


Temple University

3. Singh, Harinder. CAVEOLAE AS SPATIO-TEMPORAL COMPARTMENTS FOR ROS/RNS GENERATION AND NITROXIDATIVE STRESS SIGNALING.

Degree: PhD, 2014, Temple University

Cell Biology

During inflammatory conditions excessive production of reactive oxygen (ROS) and nitrogen species (RNS), peroxynitrite, is implicated in the development of vascular pathologies. Our… (more)

Subjects/Keywords: Biology; Cellular biology

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APA (6th Edition):

Singh, H. (2014). CAVEOLAE AS SPATIO-TEMPORAL COMPARTMENTS FOR ROS/RNS GENERATION AND NITROXIDATIVE STRESS SIGNALING. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,275561

Chicago Manual of Style (16th Edition):

Singh, Harinder. “CAVEOLAE AS SPATIO-TEMPORAL COMPARTMENTS FOR ROS/RNS GENERATION AND NITROXIDATIVE STRESS SIGNALING.” 2014. Doctoral Dissertation, Temple University. Accessed October 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,275561.

MLA Handbook (7th Edition):

Singh, Harinder. “CAVEOLAE AS SPATIO-TEMPORAL COMPARTMENTS FOR ROS/RNS GENERATION AND NITROXIDATIVE STRESS SIGNALING.” 2014. Web. 22 Oct 2019.

Vancouver:

Singh H. CAVEOLAE AS SPATIO-TEMPORAL COMPARTMENTS FOR ROS/RNS GENERATION AND NITROXIDATIVE STRESS SIGNALING. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Oct 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,275561.

Council of Science Editors:

Singh H. CAVEOLAE AS SPATIO-TEMPORAL COMPARTMENTS FOR ROS/RNS GENERATION AND NITROXIDATIVE STRESS SIGNALING. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,275561


Temple University

4. JAIN,SURBHI. ROLE OF INTERLEUKIN-19 AND ALLOGRAFT INFLAMMATORY FACTOR-1 IN ENDOTHELIAL CELL PROLIFERATION, ACTIVATION, MIGRATION AND ANGIOGENIC POTENTIAL.

Degree: PhD, 2009, Temple University

Microbiology and Immunology

Angiogenesis is an important process in maintaining normal physiology as well as in the pathology of many diseases. Angiogenesis based therapies have… (more)

Subjects/Keywords: Biology, Microbiology; Health Sciences, Immunology; Allograft Inflammatory Factor-1; Angiogenesis; Endothelial Cell; Interleukin-19; Migration; Proliferation

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APA (6th Edition):

JAIN,SURBHI. (2009). ROLE OF INTERLEUKIN-19 AND ALLOGRAFT INFLAMMATORY FACTOR-1 IN ENDOTHELIAL CELL PROLIFERATION, ACTIVATION, MIGRATION AND ANGIOGENIC POTENTIAL. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,25799

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

JAIN,SURBHI. “ROLE OF INTERLEUKIN-19 AND ALLOGRAFT INFLAMMATORY FACTOR-1 IN ENDOTHELIAL CELL PROLIFERATION, ACTIVATION, MIGRATION AND ANGIOGENIC POTENTIAL.” 2009. Doctoral Dissertation, Temple University. Accessed October 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,25799.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

JAIN,SURBHI. “ROLE OF INTERLEUKIN-19 AND ALLOGRAFT INFLAMMATORY FACTOR-1 IN ENDOTHELIAL CELL PROLIFERATION, ACTIVATION, MIGRATION AND ANGIOGENIC POTENTIAL.” 2009. Web. 22 Oct 2019.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

JAIN,SURBHI. ROLE OF INTERLEUKIN-19 AND ALLOGRAFT INFLAMMATORY FACTOR-1 IN ENDOTHELIAL CELL PROLIFERATION, ACTIVATION, MIGRATION AND ANGIOGENIC POTENTIAL. [Internet] [Doctoral dissertation]. Temple University; 2009. [cited 2019 Oct 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,25799.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

JAIN,SURBHI. ROLE OF INTERLEUKIN-19 AND ALLOGRAFT INFLAMMATORY FACTOR-1 IN ENDOTHELIAL CELL PROLIFERATION, ACTIVATION, MIGRATION AND ANGIOGENIC POTENTIAL. [Doctoral Dissertation]. Temple University; 2009. Available from: http://digital.library.temple.edu/u?/p245801coll10,25799

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


Temple University

5. Ray, Mitali. Genetic Deletion of Interleukin-19 Exacerbates Atherogenesis in Double Knockout Mice by Modulation of mRNA Stability Protein HuR.

Degree: PhD, 2018, Temple University

Biomedical Sciences

Objective: To test the hypothesis that loss of IL-19 exacerbates atherosclerosis. Approach and Results: Il19-/- mice were crossed into Ldlr-/- mice. Double knockout… (more)

Subjects/Keywords: Biology;

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APA (6th Edition):

Ray, M. (2018). Genetic Deletion of Interleukin-19 Exacerbates Atherogenesis in Double Knockout Mice by Modulation of mRNA Stability Protein HuR. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,491387

Chicago Manual of Style (16th Edition):

Ray, Mitali. “Genetic Deletion of Interleukin-19 Exacerbates Atherogenesis in Double Knockout Mice by Modulation of mRNA Stability Protein HuR.” 2018. Doctoral Dissertation, Temple University. Accessed October 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,491387.

MLA Handbook (7th Edition):

Ray, Mitali. “Genetic Deletion of Interleukin-19 Exacerbates Atherogenesis in Double Knockout Mice by Modulation of mRNA Stability Protein HuR.” 2018. Web. 22 Oct 2019.

Vancouver:

Ray M. Genetic Deletion of Interleukin-19 Exacerbates Atherogenesis in Double Knockout Mice by Modulation of mRNA Stability Protein HuR. [Internet] [Doctoral dissertation]. Temple University; 2018. [cited 2019 Oct 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,491387.

Council of Science Editors:

Ray M. Genetic Deletion of Interleukin-19 Exacerbates Atherogenesis in Double Knockout Mice by Modulation of mRNA Stability Protein HuR. [Doctoral Dissertation]. Temple University; 2018. Available from: http://digital.library.temple.edu/u?/p245801coll10,491387


Temple University

6. Brennan, Tracy A. Abrogation of Cbl-PI3K Interaction Increases Bone Volume and Osteoblast Proliferation.

Degree: PhD, 2011, Temple University

Cell Biology

Cbl is a multivalent protein that interacts with a number of signaling molecules that affect cell proliferation, migration and apoptosis. Although it is… (more)

Subjects/Keywords: Cellular Biology; Biology; Bone; Cbl; Osteoblasts; PI3K

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APA (6th Edition):

Brennan, T. A. (2011). Abrogation of Cbl-PI3K Interaction Increases Bone Volume and Osteoblast Proliferation. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,107475

Chicago Manual of Style (16th Edition):

Brennan, Tracy A. “Abrogation of Cbl-PI3K Interaction Increases Bone Volume and Osteoblast Proliferation.” 2011. Doctoral Dissertation, Temple University. Accessed October 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,107475.

MLA Handbook (7th Edition):

Brennan, Tracy A. “Abrogation of Cbl-PI3K Interaction Increases Bone Volume and Osteoblast Proliferation.” 2011. Web. 22 Oct 2019.

Vancouver:

Brennan TA. Abrogation of Cbl-PI3K Interaction Increases Bone Volume and Osteoblast Proliferation. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2019 Oct 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,107475.

Council of Science Editors:

Brennan TA. Abrogation of Cbl-PI3K Interaction Increases Bone Volume and Osteoblast Proliferation. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,107475


Temple University

7. Feairheller, Deborah Lynn. Race-Dependent Modulation of Endothelial Cell Responses to Shear Stress: Implications for Vascular Health in African Americans.

Degree: PhD, 2011, Temple University

Kinesiology

It is known that African American ethnicity is an independent risk factor for exaggerated oxidative stress which is intricately intertwined with inflammation, hypertension (HT),… (more)

Subjects/Keywords: Cellular Biology; Exercise; HUVEC; NADPH Oxidase; Oxidative Stress; Shear Stress

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APA (6th Edition):

Feairheller, D. L. (2011). Race-Dependent Modulation of Endothelial Cell Responses to Shear Stress: Implications for Vascular Health in African Americans. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,121935

Chicago Manual of Style (16th Edition):

Feairheller, Deborah Lynn. “Race-Dependent Modulation of Endothelial Cell Responses to Shear Stress: Implications for Vascular Health in African Americans.” 2011. Doctoral Dissertation, Temple University. Accessed October 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,121935.

MLA Handbook (7th Edition):

Feairheller, Deborah Lynn. “Race-Dependent Modulation of Endothelial Cell Responses to Shear Stress: Implications for Vascular Health in African Americans.” 2011. Web. 22 Oct 2019.

Vancouver:

Feairheller DL. Race-Dependent Modulation of Endothelial Cell Responses to Shear Stress: Implications for Vascular Health in African Americans. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2019 Oct 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,121935.

Council of Science Editors:

Feairheller DL. Race-Dependent Modulation of Endothelial Cell Responses to Shear Stress: Implications for Vascular Health in African Americans. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,121935


Temple University

8. Sturgeon, Katie. In Vivo and In Vitro Interactions of Oxidative Stress and Laminar Shear Stress on Vascular Endothelial Growth Factor-Mediated Endothelial Nitric Oxide Synthase Activity.

Degree: PhD, 2011, Temple University

Kinesiology

Purpose: Translational research characterizing endothelial dysfunction and the progression of cardiovascular disease (CVD) is necessary for understanding the complex nature of multi-factorial diseases. Perhaps… (more)

Subjects/Keywords: Kinesiology; Molecular biology; Physiology; eNOS; Exercise; Hydrogen Peroxide; Hypertension; Oxidative Stress; VEGF

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APA (6th Edition):

Sturgeon, K. (2011). In Vivo and In Vitro Interactions of Oxidative Stress and Laminar Shear Stress on Vascular Endothelial Growth Factor-Mediated Endothelial Nitric Oxide Synthase Activity. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,202937

Chicago Manual of Style (16th Edition):

Sturgeon, Katie. “In Vivo and In Vitro Interactions of Oxidative Stress and Laminar Shear Stress on Vascular Endothelial Growth Factor-Mediated Endothelial Nitric Oxide Synthase Activity.” 2011. Doctoral Dissertation, Temple University. Accessed October 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,202937.

MLA Handbook (7th Edition):

Sturgeon, Katie. “In Vivo and In Vitro Interactions of Oxidative Stress and Laminar Shear Stress on Vascular Endothelial Growth Factor-Mediated Endothelial Nitric Oxide Synthase Activity.” 2011. Web. 22 Oct 2019.

Vancouver:

Sturgeon K. In Vivo and In Vitro Interactions of Oxidative Stress and Laminar Shear Stress on Vascular Endothelial Growth Factor-Mediated Endothelial Nitric Oxide Synthase Activity. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2019 Oct 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,202937.

Council of Science Editors:

Sturgeon K. In Vivo and In Vitro Interactions of Oxidative Stress and Laminar Shear Stress on Vascular Endothelial Growth Factor-Mediated Endothelial Nitric Oxide Synthase Activity. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,202937


Temple University

9. Lambi, Alex G. The Intricate Role of Connective Tissue Growth Factor (CTGF/CCN2) in Prenatal Osteogenesis: A Heretofore Oversimplified Dogma of the CCN Field.

Degree: PhD, 2015, Temple University

Cell Biology

Connective tissue growth factor (CTGF/CCN2) is axiomatically necessary for proper skeletal development and function. We need not look further than the studies that… (more)

Subjects/Keywords: Cellular biology; Developmental biology;

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APA (6th Edition):

Lambi, A. G. (2015). The Intricate Role of Connective Tissue Growth Factor (CTGF/CCN2) in Prenatal Osteogenesis: A Heretofore Oversimplified Dogma of the CCN Field. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,233693

Chicago Manual of Style (16th Edition):

Lambi, Alex G. “The Intricate Role of Connective Tissue Growth Factor (CTGF/CCN2) in Prenatal Osteogenesis: A Heretofore Oversimplified Dogma of the CCN Field.” 2015. Doctoral Dissertation, Temple University. Accessed October 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,233693.

MLA Handbook (7th Edition):

Lambi, Alex G. “The Intricate Role of Connective Tissue Growth Factor (CTGF/CCN2) in Prenatal Osteogenesis: A Heretofore Oversimplified Dogma of the CCN Field.” 2015. Web. 22 Oct 2019.

Vancouver:

Lambi AG. The Intricate Role of Connective Tissue Growth Factor (CTGF/CCN2) in Prenatal Osteogenesis: A Heretofore Oversimplified Dogma of the CCN Field. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Oct 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,233693.

Council of Science Editors:

Lambi AG. The Intricate Role of Connective Tissue Growth Factor (CTGF/CCN2) in Prenatal Osteogenesis: A Heretofore Oversimplified Dogma of the CCN Field. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,233693


Temple University

10. Reichenbach, Zachary Wilmer. Modulation of the Endogenous Cannabinoid System to Attenuate Inflammation in Central Nervous System Injury.

Degree: PhD, 2015, Temple University

Physiology

In non-pathological states the central nervous system maintains a degree of immunological privilege. When illness or injury occur, this privilege can be lost and… (more)

Subjects/Keywords: Neurosciences; Immunology; Pharmacology;

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APA (6th Edition):

Reichenbach, Z. W. (2015). Modulation of the Endogenous Cannabinoid System to Attenuate Inflammation in Central Nervous System Injury. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,253514

Chicago Manual of Style (16th Edition):

Reichenbach, Zachary Wilmer. “Modulation of the Endogenous Cannabinoid System to Attenuate Inflammation in Central Nervous System Injury.” 2015. Doctoral Dissertation, Temple University. Accessed October 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,253514.

MLA Handbook (7th Edition):

Reichenbach, Zachary Wilmer. “Modulation of the Endogenous Cannabinoid System to Attenuate Inflammation in Central Nervous System Injury.” 2015. Web. 22 Oct 2019.

Vancouver:

Reichenbach ZW. Modulation of the Endogenous Cannabinoid System to Attenuate Inflammation in Central Nervous System Injury. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Oct 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,253514.

Council of Science Editors:

Reichenbach ZW. Modulation of the Endogenous Cannabinoid System to Attenuate Inflammation in Central Nervous System Injury. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,253514


Temple University

11. Barr, Larry A. The Role of Calcium in the Regulation of Pathological Hypertrophy.

Degree: PhD, 2014, Temple University

Physiology

Pathological hypertrophy leads to cardiac dysfunction and heart failure. It is not clearly defined how this process occurs in the cardiomyocyte, or how the… (more)

Subjects/Keywords: Physiology; Molecular biology; Pharmacology;

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APA (6th Edition):

Barr, L. A. (2014). The Role of Calcium in the Regulation of Pathological Hypertrophy. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,254617

Chicago Manual of Style (16th Edition):

Barr, Larry A. “The Role of Calcium in the Regulation of Pathological Hypertrophy.” 2014. Doctoral Dissertation, Temple University. Accessed October 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,254617.

MLA Handbook (7th Edition):

Barr, Larry A. “The Role of Calcium in the Regulation of Pathological Hypertrophy.” 2014. Web. 22 Oct 2019.

Vancouver:

Barr LA. The Role of Calcium in the Regulation of Pathological Hypertrophy. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Oct 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,254617.

Council of Science Editors:

Barr LA. The Role of Calcium in the Regulation of Pathological Hypertrophy. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,254617


Temple University

12. Hendesi, Honey. CONNECTIVE TISSUE GROWTH FACTOR (CTGF/CCN2) REGULATES OSTEOBLAST CYTOSKELETAL REORGANIZATION AND MOTILITY AND ENHANCES DIFFERENTIATION VIA BINDING TO INTEGRIN RECEPTORS AND ACTIVATION OF DOWNSTREAM SIGNALINGS.

Degree: PhD, 2014, Temple University

Cell Biology

Connective Tissue Growth Factor (CTGF) is a matricellular protein that has been shown to mediate cell adhesion, and as a consequence, it regulates… (more)

Subjects/Keywords: Cellular biology;

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APA (6th Edition):

Hendesi, H. (2014). CONNECTIVE TISSUE GROWTH FACTOR (CTGF/CCN2) REGULATES OSTEOBLAST CYTOSKELETAL REORGANIZATION AND MOTILITY AND ENHANCES DIFFERENTIATION VIA BINDING TO INTEGRIN RECEPTORS AND ACTIVATION OF DOWNSTREAM SIGNALINGS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,263674

Chicago Manual of Style (16th Edition):

Hendesi, Honey. “CONNECTIVE TISSUE GROWTH FACTOR (CTGF/CCN2) REGULATES OSTEOBLAST CYTOSKELETAL REORGANIZATION AND MOTILITY AND ENHANCES DIFFERENTIATION VIA BINDING TO INTEGRIN RECEPTORS AND ACTIVATION OF DOWNSTREAM SIGNALINGS.” 2014. Doctoral Dissertation, Temple University. Accessed October 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,263674.

MLA Handbook (7th Edition):

Hendesi, Honey. “CONNECTIVE TISSUE GROWTH FACTOR (CTGF/CCN2) REGULATES OSTEOBLAST CYTOSKELETAL REORGANIZATION AND MOTILITY AND ENHANCES DIFFERENTIATION VIA BINDING TO INTEGRIN RECEPTORS AND ACTIVATION OF DOWNSTREAM SIGNALINGS.” 2014. Web. 22 Oct 2019.

Vancouver:

Hendesi H. CONNECTIVE TISSUE GROWTH FACTOR (CTGF/CCN2) REGULATES OSTEOBLAST CYTOSKELETAL REORGANIZATION AND MOTILITY AND ENHANCES DIFFERENTIATION VIA BINDING TO INTEGRIN RECEPTORS AND ACTIVATION OF DOWNSTREAM SIGNALINGS. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Oct 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,263674.

Council of Science Editors:

Hendesi H. CONNECTIVE TISSUE GROWTH FACTOR (CTGF/CCN2) REGULATES OSTEOBLAST CYTOSKELETAL REORGANIZATION AND MOTILITY AND ENHANCES DIFFERENTIATION VIA BINDING TO INTEGRIN RECEPTORS AND ACTIVATION OF DOWNSTREAM SIGNALINGS. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,263674


Temple University

13. Heayn, Michelle Diane. The Role of Caveolae in PECAM-1 Mechanotransduction.

Degree: PhD, 2014, Temple University

Physiology

Altered fluid flow, which is found in branches and curvatures of arteries, results in abnormal forces on the endothelial cells (EC). These forces have… (more)

Subjects/Keywords: Cellular biology; Molecular biology; Biomechanics;

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APA (6th Edition):

Heayn, M. D. (2014). The Role of Caveolae in PECAM-1 Mechanotransduction. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,263778

Chicago Manual of Style (16th Edition):

Heayn, Michelle Diane. “The Role of Caveolae in PECAM-1 Mechanotransduction.” 2014. Doctoral Dissertation, Temple University. Accessed October 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,263778.

MLA Handbook (7th Edition):

Heayn, Michelle Diane. “The Role of Caveolae in PECAM-1 Mechanotransduction.” 2014. Web. 22 Oct 2019.

Vancouver:

Heayn MD. The Role of Caveolae in PECAM-1 Mechanotransduction. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Oct 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,263778.

Council of Science Editors:

Heayn MD. The Role of Caveolae in PECAM-1 Mechanotransduction. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,263778


Temple University

14. Mai, Jietang. ROLE OF INTERLEUKIN-17 IN ENDOTHELIAL CELL ACTIVATION AND VASCULAR FUNCTION.

Degree: PhD, 2014, Temple University

Pharmacology

Endothelial cell (EC) activation is a change of the endothelium from a quiescent state to one that is involved in immune reactions. Activation of… (more)

Subjects/Keywords: Immunology; Physiology; Cellular biology;

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APA (6th Edition):

Mai, J. (2014). ROLE OF INTERLEUKIN-17 IN ENDOTHELIAL CELL ACTIVATION AND VASCULAR FUNCTION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,266288

Chicago Manual of Style (16th Edition):

Mai, Jietang. “ROLE OF INTERLEUKIN-17 IN ENDOTHELIAL CELL ACTIVATION AND VASCULAR FUNCTION.” 2014. Doctoral Dissertation, Temple University. Accessed October 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,266288.

MLA Handbook (7th Edition):

Mai, Jietang. “ROLE OF INTERLEUKIN-17 IN ENDOTHELIAL CELL ACTIVATION AND VASCULAR FUNCTION.” 2014. Web. 22 Oct 2019.

Vancouver:

Mai J. ROLE OF INTERLEUKIN-17 IN ENDOTHELIAL CELL ACTIVATION AND VASCULAR FUNCTION. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Oct 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,266288.

Council of Science Editors:

Mai J. ROLE OF INTERLEUKIN-17 IN ENDOTHELIAL CELL ACTIVATION AND VASCULAR FUNCTION. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,266288


Temple University

15. Makarewich, Catherine Anne. MICRODOMAIN BASED CALCIUM INFLUX PATHWAYS THAT REGULATE PATHOLOGICAL CARDIAC HYPERTROPHY AND CONTRACTILITY.

Degree: PhD, 2014, Temple University

Molecular and Cellular Physiology

Pathological cardiac stressors, including persistent hypertension or damage from ischemic heart disease, induce a chronic demand for enhanced contractile performance of… (more)

Subjects/Keywords: Physiology; Molecular biology; Cellular biology;

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APA (6th Edition):

Makarewich, C. A. (2014). MICRODOMAIN BASED CALCIUM INFLUX PATHWAYS THAT REGULATE PATHOLOGICAL CARDIAC HYPERTROPHY AND CONTRACTILITY. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,266828

Chicago Manual of Style (16th Edition):

Makarewich, Catherine Anne. “MICRODOMAIN BASED CALCIUM INFLUX PATHWAYS THAT REGULATE PATHOLOGICAL CARDIAC HYPERTROPHY AND CONTRACTILITY.” 2014. Doctoral Dissertation, Temple University. Accessed October 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,266828.

MLA Handbook (7th Edition):

Makarewich, Catherine Anne. “MICRODOMAIN BASED CALCIUM INFLUX PATHWAYS THAT REGULATE PATHOLOGICAL CARDIAC HYPERTROPHY AND CONTRACTILITY.” 2014. Web. 22 Oct 2019.

Vancouver:

Makarewich CA. MICRODOMAIN BASED CALCIUM INFLUX PATHWAYS THAT REGULATE PATHOLOGICAL CARDIAC HYPERTROPHY AND CONTRACTILITY. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Oct 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,266828.

Council of Science Editors:

Makarewich CA. MICRODOMAIN BASED CALCIUM INFLUX PATHWAYS THAT REGULATE PATHOLOGICAL CARDIAC HYPERTROPHY AND CONTRACTILITY. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,266828


Temple University

16. Crawford, Kevin John. THE ROLE OF CAVEOLAE IN THE FORMATION OF ABDOMINAL AORTIC ANEURYSMS.

Degree: PhD, 2015, Temple University

Cell Biology

Abdominal aortic aneurysm (AAA) is a major cardiovascular disease and involves enhancement of renin-angiotensin system and recruitment/activation of inflammatory factors such as matrix… (more)

Subjects/Keywords: Cellular biology;

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APA (6th Edition):

Crawford, K. J. (2015). THE ROLE OF CAVEOLAE IN THE FORMATION OF ABDOMINAL AORTIC ANEURYSMS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,312992

Chicago Manual of Style (16th Edition):

Crawford, Kevin John. “THE ROLE OF CAVEOLAE IN THE FORMATION OF ABDOMINAL AORTIC ANEURYSMS.” 2015. Doctoral Dissertation, Temple University. Accessed October 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,312992.

MLA Handbook (7th Edition):

Crawford, Kevin John. “THE ROLE OF CAVEOLAE IN THE FORMATION OF ABDOMINAL AORTIC ANEURYSMS.” 2015. Web. 22 Oct 2019.

Vancouver:

Crawford KJ. THE ROLE OF CAVEOLAE IN THE FORMATION OF ABDOMINAL AORTIC ANEURYSMS. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Oct 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,312992.

Council of Science Editors:

Crawford KJ. THE ROLE OF CAVEOLAE IN THE FORMATION OF ABDOMINAL AORTIC ANEURYSMS. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,312992


Temple University

17. Fisher, Paul William. THE ROLE OF CYTOKINES AND SUBSTANCE P IN REPETITIVE LOADING-INDUCED BEHAVIORAL DECLINES AND TISSUE FIBROSIS.

Degree: PhD, 2015, Temple University

Cell Biology

Key clinical features of cumulative trauma disorders include pain, muscle weakness, and tissue fibrosis, although the etiology is still under investigation. Therefore, we… (more)

Subjects/Keywords: Cellular biology; Behavioral sciences; Biomechanics;

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APA (6th Edition):

Fisher, P. W. (2015). THE ROLE OF CYTOKINES AND SUBSTANCE P IN REPETITIVE LOADING-INDUCED BEHAVIORAL DECLINES AND TISSUE FIBROSIS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,336057

Chicago Manual of Style (16th Edition):

Fisher, Paul William. “THE ROLE OF CYTOKINES AND SUBSTANCE P IN REPETITIVE LOADING-INDUCED BEHAVIORAL DECLINES AND TISSUE FIBROSIS.” 2015. Doctoral Dissertation, Temple University. Accessed October 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,336057.

MLA Handbook (7th Edition):

Fisher, Paul William. “THE ROLE OF CYTOKINES AND SUBSTANCE P IN REPETITIVE LOADING-INDUCED BEHAVIORAL DECLINES AND TISSUE FIBROSIS.” 2015. Web. 22 Oct 2019.

Vancouver:

Fisher PW. THE ROLE OF CYTOKINES AND SUBSTANCE P IN REPETITIVE LOADING-INDUCED BEHAVIORAL DECLINES AND TISSUE FIBROSIS. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Oct 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,336057.

Council of Science Editors:

Fisher PW. THE ROLE OF CYTOKINES AND SUBSTANCE P IN REPETITIVE LOADING-INDUCED BEHAVIORAL DECLINES AND TISSUE FIBROSIS. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,336057


Temple University

18. Frara, Nagat. THE ROLE OF OSTEOACTIVIN IN MUSCULOSKELETAL TISSUES AS A REPAIR AND ANABOLIC FACTOR.

Degree: PhD, 2015, Temple University

Cell Biology

Osteoactivin (OA) is a novel osteogenic and repair factor. It has the ability to regulate cell proliferation, adhesion, differentiation, and synthesis and regulation… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Frara, N. (2015). THE ROLE OF OSTEOACTIVIN IN MUSCULOSKELETAL TISSUES AS A REPAIR AND ANABOLIC FACTOR. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,336629

Chicago Manual of Style (16th Edition):

Frara, Nagat. “THE ROLE OF OSTEOACTIVIN IN MUSCULOSKELETAL TISSUES AS A REPAIR AND ANABOLIC FACTOR.” 2015. Doctoral Dissertation, Temple University. Accessed October 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,336629.

MLA Handbook (7th Edition):

Frara, Nagat. “THE ROLE OF OSTEOACTIVIN IN MUSCULOSKELETAL TISSUES AS A REPAIR AND ANABOLIC FACTOR.” 2015. Web. 22 Oct 2019.

Vancouver:

Frara N. THE ROLE OF OSTEOACTIVIN IN MUSCULOSKELETAL TISSUES AS A REPAIR AND ANABOLIC FACTOR. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Oct 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,336629.

Council of Science Editors:

Frara N. THE ROLE OF OSTEOACTIVIN IN MUSCULOSKELETAL TISSUES AS A REPAIR AND ANABOLIC FACTOR. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,336629


Temple University

19. Richards, Jamie Madison. The Potential of IL-19 As a Therapeutic Anti-inflammatory and Angiogenic Cytokine.

Degree: PhD, 2015, Temple University

Physiology

Our lab has recently shown that IL-19 is expressed in angiogenic ECs, opening the possibility for its use as a medicine to increase perfusion… (more)

Subjects/Keywords: Physiology; Health sciences; Medicine;

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APA (6th Edition):

Richards, J. M. (2015). The Potential of IL-19 As a Therapeutic Anti-inflammatory and Angiogenic Cytokine. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,345114

Chicago Manual of Style (16th Edition):

Richards, Jamie Madison. “The Potential of IL-19 As a Therapeutic Anti-inflammatory and Angiogenic Cytokine.” 2015. Doctoral Dissertation, Temple University. Accessed October 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,345114.

MLA Handbook (7th Edition):

Richards, Jamie Madison. “The Potential of IL-19 As a Therapeutic Anti-inflammatory and Angiogenic Cytokine.” 2015. Web. 22 Oct 2019.

Vancouver:

Richards JM. The Potential of IL-19 As a Therapeutic Anti-inflammatory and Angiogenic Cytokine. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Oct 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,345114.

Council of Science Editors:

Richards JM. The Potential of IL-19 As a Therapeutic Anti-inflammatory and Angiogenic Cytokine. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,345114


Temple University

20. Kim, Ji-Seok. Shear Stress-Induced SIRT1 Activation: Effects on Mitochondrial Biogenesis and Endothelial Senescence.

Degree: PhD, 2015, Temple University

Kinesiology

Vascular aging has been implicated in the etiology of many vascular diseases. A common process linking the vascular aging to the development of vascular… (more)

Subjects/Keywords: Kinesiology

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APA (6th Edition):

Kim, J. (2015). Shear Stress-Induced SIRT1 Activation: Effects on Mitochondrial Biogenesis and Endothelial Senescence. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,356508

Chicago Manual of Style (16th Edition):

Kim, Ji-Seok. “Shear Stress-Induced SIRT1 Activation: Effects on Mitochondrial Biogenesis and Endothelial Senescence.” 2015. Doctoral Dissertation, Temple University. Accessed October 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,356508.

MLA Handbook (7th Edition):

Kim, Ji-Seok. “Shear Stress-Induced SIRT1 Activation: Effects on Mitochondrial Biogenesis and Endothelial Senescence.” 2015. Web. 22 Oct 2019.

Vancouver:

Kim J. Shear Stress-Induced SIRT1 Activation: Effects on Mitochondrial Biogenesis and Endothelial Senescence. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Oct 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,356508.

Council of Science Editors:

Kim J. Shear Stress-Induced SIRT1 Activation: Effects on Mitochondrial Biogenesis and Endothelial Senescence. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,356508


Temple University

21. Michael, James. Regulation of Ras signaling and oncogenesis by plasma membrane microdomains.

Degree: PhD, 2016, Temple University

Cell Biology

In this study, we assessed the contributions of plasma membrane (PM) microdomain targeting to the functions of H-Ras and R-Ras. These paralogues have… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Michael, J. (2016). Regulation of Ras signaling and oncogenesis by plasma membrane microdomains. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,377230

Chicago Manual of Style (16th Edition):

Michael, James. “Regulation of Ras signaling and oncogenesis by plasma membrane microdomains.” 2016. Doctoral Dissertation, Temple University. Accessed October 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,377230.

MLA Handbook (7th Edition):

Michael, James. “Regulation of Ras signaling and oncogenesis by plasma membrane microdomains.” 2016. Web. 22 Oct 2019.

Vancouver:

Michael J. Regulation of Ras signaling and oncogenesis by plasma membrane microdomains. [Internet] [Doctoral dissertation]. Temple University; 2016. [cited 2019 Oct 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,377230.

Council of Science Editors:

Michael J. Regulation of Ras signaling and oncogenesis by plasma membrane microdomains. [Doctoral Dissertation]. Temple University; 2016. Available from: http://digital.library.temple.edu/u?/p245801coll10,377230


Temple University

22. Back, Steven. TULA-2: A Novel Protein Tyrosine Phosphatase That Regulates Osteoclast Differentiation and Function.

Degree: PhD, 2014, Temple University

Cell Biology

The human skeleton is a dynamic organ that serves multiple functions to maintain normal physiology and health. It protects vital organs, provides support… (more)

Subjects/Keywords: Cellular biology; Biochemistry; Molecular biology;

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APA (6th Edition):

Back, S. (2014). TULA-2: A Novel Protein Tyrosine Phosphatase That Regulates Osteoclast Differentiation and Function. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,254886

Chicago Manual of Style (16th Edition):

Back, Steven. “TULA-2: A Novel Protein Tyrosine Phosphatase That Regulates Osteoclast Differentiation and Function.” 2014. Doctoral Dissertation, Temple University. Accessed October 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,254886.

MLA Handbook (7th Edition):

Back, Steven. “TULA-2: A Novel Protein Tyrosine Phosphatase That Regulates Osteoclast Differentiation and Function.” 2014. Web. 22 Oct 2019.

Vancouver:

Back S. TULA-2: A Novel Protein Tyrosine Phosphatase That Regulates Osteoclast Differentiation and Function. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Oct 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,254886.

Council of Science Editors:

Back S. TULA-2: A Novel Protein Tyrosine Phosphatase That Regulates Osteoclast Differentiation and Function. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,254886

.