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You searched for +publisher:"Temple University" +contributor:("Potula, Raghava;"). Showing records 1 – 4 of 4 total matches.

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Temple University

1. Fernandes, Nicole Carmel. THE ROLE OF P2X7R IN METHAMPHETAMINE-INDUCED BEHAVIORAL CHANGES AND MICROGLIAL EFFECTOR FUNCTIONS.

Degree: PhD, 2017, Temple University

Biomedical Sciences

Methamphetamine (METH) is a powerful psychostimulant with a high abuse liability. Due to its potent and long lasting effects in the central nervous… (more)

Subjects/Keywords: Immunology; Neurosciences;

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APA (6th Edition):

Fernandes, N. C. (2017). THE ROLE OF P2X7R IN METHAMPHETAMINE-INDUCED BEHAVIORAL CHANGES AND MICROGLIAL EFFECTOR FUNCTIONS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,458640

Chicago Manual of Style (16th Edition):

Fernandes, Nicole Carmel. “THE ROLE OF P2X7R IN METHAMPHETAMINE-INDUCED BEHAVIORAL CHANGES AND MICROGLIAL EFFECTOR FUNCTIONS.” 2017. Doctoral Dissertation, Temple University. Accessed October 31, 2020. http://digital.library.temple.edu/u?/p245801coll10,458640.

MLA Handbook (7th Edition):

Fernandes, Nicole Carmel. “THE ROLE OF P2X7R IN METHAMPHETAMINE-INDUCED BEHAVIORAL CHANGES AND MICROGLIAL EFFECTOR FUNCTIONS.” 2017. Web. 31 Oct 2020.

Vancouver:

Fernandes NC. THE ROLE OF P2X7R IN METHAMPHETAMINE-INDUCED BEHAVIORAL CHANGES AND MICROGLIAL EFFECTOR FUNCTIONS. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2020 Oct 31]. Available from: http://digital.library.temple.edu/u?/p245801coll10,458640.

Council of Science Editors:

Fernandes NC. THE ROLE OF P2X7R IN METHAMPHETAMINE-INDUCED BEHAVIORAL CHANGES AND MICROGLIAL EFFECTOR FUNCTIONS. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,458640


Temple University

2. Pallai, Rajash. The CCAAT-box binding transcription factor, nuclear factor-Y (NF-Y) regulates transcription of human aldo-keto reductase 1C1 (AKR1C1) gene.

Degree: PhD, 2010, Temple University

Pathology

Dihydrodiol dehydrogenases are a family of aldo-keto reductases (AKR1Cs) involved in the metabolism of steroid hormones and xenobiotics. Whilst, several phase II drugs as… (more)

Subjects/Keywords: Biology, Molecular; Dihydrodiol dehydrogenase; Hydroxysteroid dehydrogenase; Liver hepatoblastoma; Lung adenocarcinoma; Ovarian carcinoma; Promoter regulation

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APA (6th Edition):

Pallai, R. (2010). The CCAAT-box binding transcription factor, nuclear factor-Y (NF-Y) regulates transcription of human aldo-keto reductase 1C1 (AKR1C1) gene. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,78307

Chicago Manual of Style (16th Edition):

Pallai, Rajash. “The CCAAT-box binding transcription factor, nuclear factor-Y (NF-Y) regulates transcription of human aldo-keto reductase 1C1 (AKR1C1) gene.” 2010. Doctoral Dissertation, Temple University. Accessed October 31, 2020. http://digital.library.temple.edu/u?/p245801coll10,78307.

MLA Handbook (7th Edition):

Pallai, Rajash. “The CCAAT-box binding transcription factor, nuclear factor-Y (NF-Y) regulates transcription of human aldo-keto reductase 1C1 (AKR1C1) gene.” 2010. Web. 31 Oct 2020.

Vancouver:

Pallai R. The CCAAT-box binding transcription factor, nuclear factor-Y (NF-Y) regulates transcription of human aldo-keto reductase 1C1 (AKR1C1) gene. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Oct 31]. Available from: http://digital.library.temple.edu/u?/p245801coll10,78307.

Council of Science Editors:

Pallai R. The CCAAT-box binding transcription factor, nuclear factor-Y (NF-Y) regulates transcription of human aldo-keto reductase 1C1 (AKR1C1) gene. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,78307


Temple University

3. Gofman, Larisa. Role of Purinergic Receptor (P2X4) in EtOH-Mediated Microglial Immune Responses.

Degree: PhD, 2015, Temple University

Pathology

Ethanol (EtOH) abuse is the third leading cause of preventable death in the United States. Mounting evidence indicates that EtOH-induced neuropathology may result from… (more)

Subjects/Keywords: Neurosciences;

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Gofman, L. (2015). Role of Purinergic Receptor (P2X4) in EtOH-Mediated Microglial Immune Responses. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,315641

Chicago Manual of Style (16th Edition):

Gofman, Larisa. “Role of Purinergic Receptor (P2X4) in EtOH-Mediated Microglial Immune Responses.” 2015. Doctoral Dissertation, Temple University. Accessed October 31, 2020. http://digital.library.temple.edu/u?/p245801coll10,315641.

MLA Handbook (7th Edition):

Gofman, Larisa. “Role of Purinergic Receptor (P2X4) in EtOH-Mediated Microglial Immune Responses.” 2015. Web. 31 Oct 2020.

Vancouver:

Gofman L. Role of Purinergic Receptor (P2X4) in EtOH-Mediated Microglial Immune Responses. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Oct 31]. Available from: http://digital.library.temple.edu/u?/p245801coll10,315641.

Council of Science Editors:

Gofman L. Role of Purinergic Receptor (P2X4) in EtOH-Mediated Microglial Immune Responses. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,315641


Temple University

4. Oppong, Gertrude Odamea. The Role of Bacterial Amyloids In Regulating Gastrointestinal Homeostasis.

Degree: PhD, 2015, Temple University

Microbiology and Immunology

Many bacterial species exist in nature as part of highly structured multicellular communities known as biofilms. Amyloids, proteins with a conserved β-sheet… (more)

Subjects/Keywords: Microbiology;

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Oppong, G. O. (2015). The Role of Bacterial Amyloids In Regulating Gastrointestinal Homeostasis. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,323094

Chicago Manual of Style (16th Edition):

Oppong, Gertrude Odamea. “The Role of Bacterial Amyloids In Regulating Gastrointestinal Homeostasis.” 2015. Doctoral Dissertation, Temple University. Accessed October 31, 2020. http://digital.library.temple.edu/u?/p245801coll10,323094.

MLA Handbook (7th Edition):

Oppong, Gertrude Odamea. “The Role of Bacterial Amyloids In Regulating Gastrointestinal Homeostasis.” 2015. Web. 31 Oct 2020.

Vancouver:

Oppong GO. The Role of Bacterial Amyloids In Regulating Gastrointestinal Homeostasis. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Oct 31]. Available from: http://digital.library.temple.edu/u?/p245801coll10,323094.

Council of Science Editors:

Oppong GO. The Role of Bacterial Amyloids In Regulating Gastrointestinal Homeostasis. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,323094

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