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You searched for +publisher:"Temple University" +contributor:("Muniswamy, Madesh"). Showing records 1 – 8 of 8 total matches.

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Temple University

1. Doonan, Patrick John. Mitochondrial Calcium Uptake: LETM1 and MICU1 Are Mitochondrial Proteins That Regulate Mitochondrial Calcium Homeostasis and Cellular Bioenergetics.

Degree: PhD, 2012, Temple University

Biochemistry

Mitochondrial calcium (Ca2+) uptake has been studied for over five decades, with crucial insights into its underlying mechanisms enabled by development of the chemi-osmotic… (more)

Subjects/Keywords: Biochemistry; Molecular biology; Calcium; LETM1; MICU1; Mitochondria; Mitochondria Calcium Uniporter; Uniporter

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APA (6th Edition):

Doonan, P. J. (2012). Mitochondrial Calcium Uptake: LETM1 and MICU1 Are Mitochondrial Proteins That Regulate Mitochondrial Calcium Homeostasis and Cellular Bioenergetics. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,214818

Chicago Manual of Style (16th Edition):

Doonan, Patrick John. “Mitochondrial Calcium Uptake: LETM1 and MICU1 Are Mitochondrial Proteins That Regulate Mitochondrial Calcium Homeostasis and Cellular Bioenergetics.” 2012. Doctoral Dissertation, Temple University. Accessed August 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,214818.

MLA Handbook (7th Edition):

Doonan, Patrick John. “Mitochondrial Calcium Uptake: LETM1 and MICU1 Are Mitochondrial Proteins That Regulate Mitochondrial Calcium Homeostasis and Cellular Bioenergetics.” 2012. Web. 22 Aug 2019.

Vancouver:

Doonan PJ. Mitochondrial Calcium Uptake: LETM1 and MICU1 Are Mitochondrial Proteins That Regulate Mitochondrial Calcium Homeostasis and Cellular Bioenergetics. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2019 Aug 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,214818.

Council of Science Editors:

Doonan PJ. Mitochondrial Calcium Uptake: LETM1 and MICU1 Are Mitochondrial Proteins That Regulate Mitochondrial Calcium Homeostasis and Cellular Bioenergetics. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,214818


Temple University

2. Wang, Xizhuo. STIM Protein Coupling Domains and The Activation of Orai Channels.

Degree: PhD, 2015, Temple University

Biochemistry

STIM1 and STIM2 are widely expressed endoplasmic reticulum (ER) Ca2+ sensor proteins able to translocate within the ER membrane to physically couple with and… (more)

Subjects/Keywords: Biochemistry

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APA (6th Edition):

Wang, X. (2015). STIM Protein Coupling Domains and The Activation of Orai Channels. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,366037

Chicago Manual of Style (16th Edition):

Wang, Xizhuo. “STIM Protein Coupling Domains and The Activation of Orai Channels.” 2015. Doctoral Dissertation, Temple University. Accessed August 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,366037.

MLA Handbook (7th Edition):

Wang, Xizhuo. “STIM Protein Coupling Domains and The Activation of Orai Channels.” 2015. Web. 22 Aug 2019.

Vancouver:

Wang X. STIM Protein Coupling Domains and The Activation of Orai Channels. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Aug 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,366037.

Council of Science Editors:

Wang X. STIM Protein Coupling Domains and The Activation of Orai Channels. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,366037


Temple University

3. Corradetti, Chelsea. THE ROLES OF RIPK3-MEDIATED NECROSIS AND ESTROGEN RECEPTOR-ALPHA IN THE PATHOGENESIS OF IMMUNE-MEDIATED NEPHROPATHY.

Degree: PhD, 2017, Temple University

Biomedical Sciences

Systemic lupus erythematosus (SLE) is an autoimmune disease characterized by loss of immune tolerance and the production of auto-antibodies which target various nuclear… (more)

Subjects/Keywords: Immunology;

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APA (6th Edition):

Corradetti, C. (2017). THE ROLES OF RIPK3-MEDIATED NECROSIS AND ESTROGEN RECEPTOR-ALPHA IN THE PATHOGENESIS OF IMMUNE-MEDIATED NEPHROPATHY. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,472051

Chicago Manual of Style (16th Edition):

Corradetti, Chelsea. “THE ROLES OF RIPK3-MEDIATED NECROSIS AND ESTROGEN RECEPTOR-ALPHA IN THE PATHOGENESIS OF IMMUNE-MEDIATED NEPHROPATHY.” 2017. Doctoral Dissertation, Temple University. Accessed August 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,472051.

MLA Handbook (7th Edition):

Corradetti, Chelsea. “THE ROLES OF RIPK3-MEDIATED NECROSIS AND ESTROGEN RECEPTOR-ALPHA IN THE PATHOGENESIS OF IMMUNE-MEDIATED NEPHROPATHY.” 2017. Web. 22 Aug 2019.

Vancouver:

Corradetti C. THE ROLES OF RIPK3-MEDIATED NECROSIS AND ESTROGEN RECEPTOR-ALPHA IN THE PATHOGENESIS OF IMMUNE-MEDIATED NEPHROPATHY. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2019 Aug 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,472051.

Council of Science Editors:

Corradetti C. THE ROLES OF RIPK3-MEDIATED NECROSIS AND ESTROGEN RECEPTOR-ALPHA IN THE PATHOGENESIS OF IMMUNE-MEDIATED NEPHROPATHY. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,472051


Temple University

4. Nemani, Neeharika. Molecular Determinant of Mitochondrial Shape Change.

Degree: PhD, 2018, Temple University

Biomedical Sciences

Mitochondria shape cytosolic Ca2+ (cCa2+) transients. Ca2+ entry into the mitochondria is driven by the highly negative mitochondrial membrane potential and through a… (more)

Subjects/Keywords: Molecular biology; Biochemistry;

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APA (6th Edition):

Nemani, N. (2018). Molecular Determinant of Mitochondrial Shape Change. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,511170

Chicago Manual of Style (16th Edition):

Nemani, Neeharika. “Molecular Determinant of Mitochondrial Shape Change.” 2018. Doctoral Dissertation, Temple University. Accessed August 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,511170.

MLA Handbook (7th Edition):

Nemani, Neeharika. “Molecular Determinant of Mitochondrial Shape Change.” 2018. Web. 22 Aug 2019.

Vancouver:

Nemani N. Molecular Determinant of Mitochondrial Shape Change. [Internet] [Doctoral dissertation]. Temple University; 2018. [cited 2019 Aug 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,511170.

Council of Science Editors:

Nemani N. Molecular Determinant of Mitochondrial Shape Change. [Doctoral Dissertation]. Temple University; 2018. Available from: http://digital.library.temple.edu/u?/p245801coll10,511170


Temple University

5. Hubert, Terrence L. Effect of rhCC10 on the Pro/Anti-Inflammatory Profile of the Immature Lung.

Degree: PhD, 2014, Temple University

Physiology

There is a gap in the treatment of preterm infants with respiratory distress syndrome. Despite addressing surfactant insufficiency and mechanical instability, currently available exogenous… (more)

Subjects/Keywords: Physiology;

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APA (6th Edition):

Hubert, T. L. (2014). Effect of rhCC10 on the Pro/Anti-Inflammatory Profile of the Immature Lung. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,240379

Chicago Manual of Style (16th Edition):

Hubert, Terrence L. “Effect of rhCC10 on the Pro/Anti-Inflammatory Profile of the Immature Lung.” 2014. Doctoral Dissertation, Temple University. Accessed August 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,240379.

MLA Handbook (7th Edition):

Hubert, Terrence L. “Effect of rhCC10 on the Pro/Anti-Inflammatory Profile of the Immature Lung.” 2014. Web. 22 Aug 2019.

Vancouver:

Hubert TL. Effect of rhCC10 on the Pro/Anti-Inflammatory Profile of the Immature Lung. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Aug 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,240379.

Council of Science Editors:

Hubert TL. Effect of rhCC10 on the Pro/Anti-Inflammatory Profile of the Immature Lung. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,240379


Temple University

6. Yin, Ying. CASPASE-1 ACTIVATION IS CRITICAL FOR ENDOTHELIAL CELL ACTIVATION, MONOCYTE MIGRATION, AND EARLY ATHEROGENESIS.

Degree: PhD, 2013, Temple University

Pharmacology

Atherosclerosis, considered a chronic inflammatory disease, is the underlying mechanism for several cardiovascular diseases. Hyperlipidemia is the number one risk factor for atherogenesis. Caspase-1… (more)

Subjects/Keywords: Pharmacology; atherosclerosis, caspase-1, vascular inflammation

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APA (6th Edition):

Yin, Y. (2013). CASPASE-1 ACTIVATION IS CRITICAL FOR ENDOTHELIAL CELL ACTIVATION, MONOCYTE MIGRATION, AND EARLY ATHEROGENESIS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,252725

Chicago Manual of Style (16th Edition):

Yin, Ying. “CASPASE-1 ACTIVATION IS CRITICAL FOR ENDOTHELIAL CELL ACTIVATION, MONOCYTE MIGRATION, AND EARLY ATHEROGENESIS.” 2013. Doctoral Dissertation, Temple University. Accessed August 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,252725.

MLA Handbook (7th Edition):

Yin, Ying. “CASPASE-1 ACTIVATION IS CRITICAL FOR ENDOTHELIAL CELL ACTIVATION, MONOCYTE MIGRATION, AND EARLY ATHEROGENESIS.” 2013. Web. 22 Aug 2019.

Vancouver:

Yin Y. CASPASE-1 ACTIVATION IS CRITICAL FOR ENDOTHELIAL CELL ACTIVATION, MONOCYTE MIGRATION, AND EARLY ATHEROGENESIS. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2019 Aug 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,252725.

Council of Science Editors:

Yin Y. CASPASE-1 ACTIVATION IS CRITICAL FOR ENDOTHELIAL CELL ACTIVATION, MONOCYTE MIGRATION, AND EARLY ATHEROGENESIS. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,252725


Temple University

7. Virtue, Anthony Thomas. The Contributions of miR-155 in Obesity, Metabolic Syndrome, and Atherosclerosis Development.

Degree: PhD, 2014, Temple University

Pharmacology

The global incidence of overweight and obese individuals has skyrocketed in the past few decades resulting in a new health epidemic. In 1980, 5%… (more)

Subjects/Keywords: Biology; Molecular biology;

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APA (6th Edition):

Virtue, A. T. (2014). The Contributions of miR-155 in Obesity, Metabolic Syndrome, and Atherosclerosis Development. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,276607

Chicago Manual of Style (16th Edition):

Virtue, Anthony Thomas. “The Contributions of miR-155 in Obesity, Metabolic Syndrome, and Atherosclerosis Development.” 2014. Doctoral Dissertation, Temple University. Accessed August 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,276607.

MLA Handbook (7th Edition):

Virtue, Anthony Thomas. “The Contributions of miR-155 in Obesity, Metabolic Syndrome, and Atherosclerosis Development.” 2014. Web. 22 Aug 2019.

Vancouver:

Virtue AT. The Contributions of miR-155 in Obesity, Metabolic Syndrome, and Atherosclerosis Development. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Aug 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,276607.

Council of Science Editors:

Virtue AT. The Contributions of miR-155 in Obesity, Metabolic Syndrome, and Atherosclerosis Development. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,276607


Temple University

8. Hoffman, Nicholas. Mitochondrial Calcium Influx is Determined by Multiple Protein Components Including SLC25A23 and MICU1.

Degree: PhD, 2014, Temple University

Biochemistry

Ca2+ control mechanisms employed by the cell at the plasma membrane include receptor operated, voltage-sensitive, and store operated channels for Ca2+ import. Upon entry… (more)

Subjects/Keywords: Biochemistry

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APA (6th Edition):

Hoffman, N. (2014). Mitochondrial Calcium Influx is Determined by Multiple Protein Components Including SLC25A23 and MICU1. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,287159

Chicago Manual of Style (16th Edition):

Hoffman, Nicholas. “Mitochondrial Calcium Influx is Determined by Multiple Protein Components Including SLC25A23 and MICU1.” 2014. Doctoral Dissertation, Temple University. Accessed August 22, 2019. http://digital.library.temple.edu/u?/p245801coll10,287159.

MLA Handbook (7th Edition):

Hoffman, Nicholas. “Mitochondrial Calcium Influx is Determined by Multiple Protein Components Including SLC25A23 and MICU1.” 2014. Web. 22 Aug 2019.

Vancouver:

Hoffman N. Mitochondrial Calcium Influx is Determined by Multiple Protein Components Including SLC25A23 and MICU1. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Aug 22]. Available from: http://digital.library.temple.edu/u?/p245801coll10,287159.

Council of Science Editors:

Hoffman N. Mitochondrial Calcium Influx is Determined by Multiple Protein Components Including SLC25A23 and MICU1. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,287159

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