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You searched for +publisher:"Temple University" +contributor:("Monestier, Marc"). Showing records 1 – 26 of 26 total matches.

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Temple University

1. Fernandes, Nicole Carmel. THE ROLE OF P2X7R IN METHAMPHETAMINE-INDUCED BEHAVIORAL CHANGES AND MICROGLIAL EFFECTOR FUNCTIONS.

Degree: PhD, 2017, Temple University

Biomedical Sciences

Methamphetamine (METH) is a powerful psychostimulant with a high abuse liability. Due to its potent and long lasting effects in the central nervous… (more)

Subjects/Keywords: Immunology; Neurosciences;

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APA (6th Edition):

Fernandes, N. C. (2017). THE ROLE OF P2X7R IN METHAMPHETAMINE-INDUCED BEHAVIORAL CHANGES AND MICROGLIAL EFFECTOR FUNCTIONS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,458640

Chicago Manual of Style (16th Edition):

Fernandes, Nicole Carmel. “THE ROLE OF P2X7R IN METHAMPHETAMINE-INDUCED BEHAVIORAL CHANGES AND MICROGLIAL EFFECTOR FUNCTIONS.” 2017. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,458640.

MLA Handbook (7th Edition):

Fernandes, Nicole Carmel. “THE ROLE OF P2X7R IN METHAMPHETAMINE-INDUCED BEHAVIORAL CHANGES AND MICROGLIAL EFFECTOR FUNCTIONS.” 2017. Web. 02 Apr 2020.

Vancouver:

Fernandes NC. THE ROLE OF P2X7R IN METHAMPHETAMINE-INDUCED BEHAVIORAL CHANGES AND MICROGLIAL EFFECTOR FUNCTIONS. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,458640.

Council of Science Editors:

Fernandes NC. THE ROLE OF P2X7R IN METHAMPHETAMINE-INDUCED BEHAVIORAL CHANGES AND MICROGLIAL EFFECTOR FUNCTIONS. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,458640


Temple University

2. Kocieda, Virginia Polonia. Prostaglandin E2-induced IL-23p19 is regulated by CREB and C/EBP beta in bone marrow derived dendritic cells.

Degree: PhD, 2013, Temple University

Microbiology and Immunology

We reported previously that prostaglandin E2 (PGE2) upregulates IL-23 in vitro in bone marrow-derived dendritic cells (DC), and in vivo in models… (more)

Subjects/Keywords: Immunology; Molecular biology; C/EBP; CREB; dendritic cell; IL-23; PGE2

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APA (6th Edition):

Kocieda, V. P. (2013). Prostaglandin E2-induced IL-23p19 is regulated by CREB and C/EBP beta in bone marrow derived dendritic cells. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,214805

Chicago Manual of Style (16th Edition):

Kocieda, Virginia Polonia. “Prostaglandin E2-induced IL-23p19 is regulated by CREB and C/EBP beta in bone marrow derived dendritic cells.” 2013. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,214805.

MLA Handbook (7th Edition):

Kocieda, Virginia Polonia. “Prostaglandin E2-induced IL-23p19 is regulated by CREB and C/EBP beta in bone marrow derived dendritic cells.” 2013. Web. 02 Apr 2020.

Vancouver:

Kocieda VP. Prostaglandin E2-induced IL-23p19 is regulated by CREB and C/EBP beta in bone marrow derived dendritic cells. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,214805.

Council of Science Editors:

Kocieda VP. Prostaglandin E2-induced IL-23p19 is regulated by CREB and C/EBP beta in bone marrow derived dendritic cells. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,214805


Temple University

3. Cutrera, Jason Lewis. Insights into the Structure and Function of PrgW and its Conserved Cysteines.

Degree: PhD, 2014, Temple University

Microbiology and Immunology

Enterococcus faecalis is a Gram-positive bacterial species that is typically a member of the human gastrointestinal tract microbiota. However, E. faecalis is… (more)

Subjects/Keywords: Microbiology;

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APA (6th Edition):

Cutrera, J. L. (2014). Insights into the Structure and Function of PrgW and its Conserved Cysteines. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,299645

Chicago Manual of Style (16th Edition):

Cutrera, Jason Lewis. “Insights into the Structure and Function of PrgW and its Conserved Cysteines.” 2014. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,299645.

MLA Handbook (7th Edition):

Cutrera, Jason Lewis. “Insights into the Structure and Function of PrgW and its Conserved Cysteines.” 2014. Web. 02 Apr 2020.

Vancouver:

Cutrera JL. Insights into the Structure and Function of PrgW and its Conserved Cysteines. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,299645.

Council of Science Editors:

Cutrera JL. Insights into the Structure and Function of PrgW and its Conserved Cysteines. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,299645


Temple University

4. Xu, Jun. Regulation of type I interferons in murine dendritic cells.

Degree: PhD, 2014, Temple University

Microbiology and Immunology

Conventional Dendritic cells (cDCs), a specialized group of immunological sentinels with tree-like or dendritic shapes, are critical for recognition of danger signals,… (more)

Subjects/Keywords: Immunology; Microbiology;

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APA (6th Edition):

Xu, J. (2014). Regulation of type I interferons in murine dendritic cells. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,308946

Chicago Manual of Style (16th Edition):

Xu, Jun. “Regulation of type I interferons in murine dendritic cells.” 2014. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,308946.

MLA Handbook (7th Edition):

Xu, Jun. “Regulation of type I interferons in murine dendritic cells.” 2014. Web. 02 Apr 2020.

Vancouver:

Xu J. Regulation of type I interferons in murine dendritic cells. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,308946.

Council of Science Editors:

Xu J. Regulation of type I interferons in murine dendritic cells. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,308946


Temple University

5. Di Donato, Francis Anthony. Determinants of compartmentalization of gene expression during sporulation in Bacillus subtilis.

Degree: 2008, Temple University

Microbiology and Immunology

Ph.D.;

Bacillus subtilis, a benign gram-positive bacterium, utilizes the strategy of sporulation, which enables it to survive stresses such as starvation, desiccation,… (more)

Subjects/Keywords: Biology; Microbiology

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APA (6th Edition):

Di Donato, F. A. (2008). Determinants of compartmentalization of gene expression during sporulation in Bacillus subtilis. (Thesis). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,4102

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Di Donato, Francis Anthony. “Determinants of compartmentalization of gene expression during sporulation in Bacillus subtilis.” 2008. Thesis, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,4102.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Di Donato, Francis Anthony. “Determinants of compartmentalization of gene expression during sporulation in Bacillus subtilis.” 2008. Web. 02 Apr 2020.

Vancouver:

Di Donato FA. Determinants of compartmentalization of gene expression during sporulation in Bacillus subtilis. [Internet] [Thesis]. Temple University; 2008. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,4102.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Di Donato FA. Determinants of compartmentalization of gene expression during sporulation in Bacillus subtilis. [Thesis]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,4102

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Temple University

6. JAIN,SURBHI. ROLE OF INTERLEUKIN-19 AND ALLOGRAFT INFLAMMATORY FACTOR-1 IN ENDOTHELIAL CELL PROLIFERATION, ACTIVATION, MIGRATION AND ANGIOGENIC POTENTIAL.

Degree: PhD, 2009, Temple University

Microbiology and Immunology

Angiogenesis is an important process in maintaining normal physiology as well as in the pathology of many diseases. Angiogenesis based therapies have… (more)

Subjects/Keywords: Biology, Microbiology; Health Sciences, Immunology; Allograft Inflammatory Factor-1; Angiogenesis; Endothelial Cell; Interleukin-19; Migration; Proliferation

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APA (6th Edition):

JAIN,SURBHI. (2009). ROLE OF INTERLEUKIN-19 AND ALLOGRAFT INFLAMMATORY FACTOR-1 IN ENDOTHELIAL CELL PROLIFERATION, ACTIVATION, MIGRATION AND ANGIOGENIC POTENTIAL. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,25799

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Chicago Manual of Style (16th Edition):

JAIN,SURBHI. “ROLE OF INTERLEUKIN-19 AND ALLOGRAFT INFLAMMATORY FACTOR-1 IN ENDOTHELIAL CELL PROLIFERATION, ACTIVATION, MIGRATION AND ANGIOGENIC POTENTIAL.” 2009. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,25799.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

MLA Handbook (7th Edition):

JAIN,SURBHI. “ROLE OF INTERLEUKIN-19 AND ALLOGRAFT INFLAMMATORY FACTOR-1 IN ENDOTHELIAL CELL PROLIFERATION, ACTIVATION, MIGRATION AND ANGIOGENIC POTENTIAL.” 2009. Web. 02 Apr 2020.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Vancouver:

JAIN,SURBHI. ROLE OF INTERLEUKIN-19 AND ALLOGRAFT INFLAMMATORY FACTOR-1 IN ENDOTHELIAL CELL PROLIFERATION, ACTIVATION, MIGRATION AND ANGIOGENIC POTENTIAL. [Internet] [Doctoral dissertation]. Temple University; 2009. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,25799.

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete

Council of Science Editors:

JAIN,SURBHI. ROLE OF INTERLEUKIN-19 AND ALLOGRAFT INFLAMMATORY FACTOR-1 IN ENDOTHELIAL CELL PROLIFERATION, ACTIVATION, MIGRATION AND ANGIOGENIC POTENTIAL. [Doctoral Dissertation]. Temple University; 2009. Available from: http://digital.library.temple.edu/u?/p245801coll10,25799

Note: this citation may be lacking information needed for this citation format:
Author name may be incomplete


Temple University

7. Purushe, Janaki. MLL4-Menin Complex Inhibition Promotes Central Memory In CD8 CAR-T Cells.

Degree: PhD, 2018, Temple University

Infectious Disease & Immunity

CAR-T cell immunotherapy is a highly efficacious treatment for CD19-positive hematological malignancies, however, some patients are non-responsive for reasons that are… (more)

Subjects/Keywords: Immunology;

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APA (6th Edition):

Purushe, J. (2018). MLL4-Menin Complex Inhibition Promotes Central Memory In CD8 CAR-T Cells. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,489872

Chicago Manual of Style (16th Edition):

Purushe, Janaki. “MLL4-Menin Complex Inhibition Promotes Central Memory In CD8 CAR-T Cells.” 2018. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,489872.

MLA Handbook (7th Edition):

Purushe, Janaki. “MLL4-Menin Complex Inhibition Promotes Central Memory In CD8 CAR-T Cells.” 2018. Web. 02 Apr 2020.

Vancouver:

Purushe J. MLL4-Menin Complex Inhibition Promotes Central Memory In CD8 CAR-T Cells. [Internet] [Doctoral dissertation]. Temple University; 2018. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,489872.

Council of Science Editors:

Purushe J. MLL4-Menin Complex Inhibition Promotes Central Memory In CD8 CAR-T Cells. [Doctoral Dissertation]. Temple University; 2018. Available from: http://digital.library.temple.edu/u?/p245801coll10,489872


Temple University

8. Corradetti, Chelsea. THE ROLES OF RIPK3-MEDIATED NECROSIS AND ESTROGEN RECEPTOR-ALPHA IN THE PATHOGENESIS OF IMMUNE-MEDIATED NEPHROPATHY.

Degree: PhD, 2017, Temple University

Biomedical Sciences

Systemic lupus erythematosus (SLE) is an autoimmune disease characterized by loss of immune tolerance and the production of auto-antibodies which target various nuclear… (more)

Subjects/Keywords: Immunology;

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APA (6th Edition):

Corradetti, C. (2017). THE ROLES OF RIPK3-MEDIATED NECROSIS AND ESTROGEN RECEPTOR-ALPHA IN THE PATHOGENESIS OF IMMUNE-MEDIATED NEPHROPATHY. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,472051

Chicago Manual of Style (16th Edition):

Corradetti, Chelsea. “THE ROLES OF RIPK3-MEDIATED NECROSIS AND ESTROGEN RECEPTOR-ALPHA IN THE PATHOGENESIS OF IMMUNE-MEDIATED NEPHROPATHY.” 2017. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,472051.

MLA Handbook (7th Edition):

Corradetti, Chelsea. “THE ROLES OF RIPK3-MEDIATED NECROSIS AND ESTROGEN RECEPTOR-ALPHA IN THE PATHOGENESIS OF IMMUNE-MEDIATED NEPHROPATHY.” 2017. Web. 02 Apr 2020.

Vancouver:

Corradetti C. THE ROLES OF RIPK3-MEDIATED NECROSIS AND ESTROGEN RECEPTOR-ALPHA IN THE PATHOGENESIS OF IMMUNE-MEDIATED NEPHROPATHY. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,472051.

Council of Science Editors:

Corradetti C. THE ROLES OF RIPK3-MEDIATED NECROSIS AND ESTROGEN RECEPTOR-ALPHA IN THE PATHOGENESIS OF IMMUNE-MEDIATED NEPHROPATHY. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,472051


Temple University

9. Moore, Andrea Rossi. COX-2 inhibition impaired resolution of chronic inflammation in a murine model of autoimmune arthritis.

Degree: PhD, 2010, Temple University

Microbiology and Immunology

Rheumatoid arthritis (RA) is a chronic disease characterized by cycles of inflammation and resolution. Previously, it was believed that the resolution of… (more)

Subjects/Keywords: Health Sciences, Immunology; autoimmunity; inflammation; lipid mediators; rheumatoid arthritis; rodent

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APA (6th Edition):

Moore, A. R. (2010). COX-2 inhibition impaired resolution of chronic inflammation in a murine model of autoimmune arthritis. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,81890

Chicago Manual of Style (16th Edition):

Moore, Andrea Rossi. “COX-2 inhibition impaired resolution of chronic inflammation in a murine model of autoimmune arthritis.” 2010. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,81890.

MLA Handbook (7th Edition):

Moore, Andrea Rossi. “COX-2 inhibition impaired resolution of chronic inflammation in a murine model of autoimmune arthritis.” 2010. Web. 02 Apr 2020.

Vancouver:

Moore AR. COX-2 inhibition impaired resolution of chronic inflammation in a murine model of autoimmune arthritis. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,81890.

Council of Science Editors:

Moore AR. COX-2 inhibition impaired resolution of chronic inflammation in a murine model of autoimmune arthritis. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,81890


Temple University

10. Busuioc, Monica. Survival Strategies of Streptococcus mutans during Carbohydrate Starvation.

Degree: PhD, 2010, Temple University

Microbiology and Immunology

Streptococcus mutans is a facultative member of the oral plaque and is associated with dental caries. It is able to survive long… (more)

Subjects/Keywords: Biology, Microbiology; Biology, Molecular; Biology, Genetics; gene regulation; IPS; PDH; starvation; Streptococcocus mutans; survival

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APA (6th Edition):

Busuioc, M. (2010). Survival Strategies of Streptococcus mutans during Carbohydrate Starvation. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,88876

Chicago Manual of Style (16th Edition):

Busuioc, Monica. “Survival Strategies of Streptococcus mutans during Carbohydrate Starvation.” 2010. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,88876.

MLA Handbook (7th Edition):

Busuioc, Monica. “Survival Strategies of Streptococcus mutans during Carbohydrate Starvation.” 2010. Web. 02 Apr 2020.

Vancouver:

Busuioc M. Survival Strategies of Streptococcus mutans during Carbohydrate Starvation. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,88876.

Council of Science Editors:

Busuioc M. Survival Strategies of Streptococcus mutans during Carbohydrate Starvation. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,88876


Temple University

11. Kong, Weimin. THE ANTI-INFLAMMATORY EFFECTS OF DOCOSAHEXAENOIC ACID ON DENDRITIC CELLS.

Degree: PhD, 2010, Temple University

Physiology

Dendritic cells (DC) are professional antigen presenting cells which link innate and adaptive immunity through recognition and processing of pathogens, migration to secondary lymph… (more)

Subjects/Keywords: Immunology

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APA (6th Edition):

Kong, W. (2010). THE ANTI-INFLAMMATORY EFFECTS OF DOCOSAHEXAENOIC ACID ON DENDRITIC CELLS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,119523

Chicago Manual of Style (16th Edition):

Kong, Weimin. “THE ANTI-INFLAMMATORY EFFECTS OF DOCOSAHEXAENOIC ACID ON DENDRITIC CELLS.” 2010. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,119523.

MLA Handbook (7th Edition):

Kong, Weimin. “THE ANTI-INFLAMMATORY EFFECTS OF DOCOSAHEXAENOIC ACID ON DENDRITIC CELLS.” 2010. Web. 02 Apr 2020.

Vancouver:

Kong W. THE ANTI-INFLAMMATORY EFFECTS OF DOCOSAHEXAENOIC ACID ON DENDRITIC CELLS. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,119523.

Council of Science Editors:

Kong W. THE ANTI-INFLAMMATORY EFFECTS OF DOCOSAHEXAENOIC ACID ON DENDRITIC CELLS. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,119523


Temple University

12. Schiraldi, Michael. Tim-3 and Cell Death in Murine Mercury Induced Autoimmunity.

Degree: PhD, 2011, Temple University

Microbiology and Immunology

There is a role for environmental factors in the pathogenesis of autoimmune diseases in humans and animals. Correlations have been made between… (more)

Subjects/Keywords: Immunology; autoimmunity; mercury; murine

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APA (6th Edition):

Schiraldi, M. (2011). Tim-3 and Cell Death in Murine Mercury Induced Autoimmunity. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,136653

Chicago Manual of Style (16th Edition):

Schiraldi, Michael. “Tim-3 and Cell Death in Murine Mercury Induced Autoimmunity.” 2011. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,136653.

MLA Handbook (7th Edition):

Schiraldi, Michael. “Tim-3 and Cell Death in Murine Mercury Induced Autoimmunity.” 2011. Web. 02 Apr 2020.

Vancouver:

Schiraldi M. Tim-3 and Cell Death in Murine Mercury Induced Autoimmunity. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,136653.

Council of Science Editors:

Schiraldi M. Tim-3 and Cell Death in Murine Mercury Induced Autoimmunity. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,136653


Temple University

13. Chatila, Wissam M. F. MicroRNA Expression in Regulatory T Cells in Chronic Obstructive Pulmonary Disease.

Degree: PhD, 2015, Temple University

Microbiology and Immunology

COPD is characterized by an abnormal regulatory T cell (Treg) response with a shift towards a Th1 and Th17 cell responses. However,… (more)

Subjects/Keywords: Immunology; Cellular biology; Molecular biology;

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APA (6th Edition):

Chatila, W. M. F. (2015). MicroRNA Expression in Regulatory T Cells in Chronic Obstructive Pulmonary Disease. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,333572

Chicago Manual of Style (16th Edition):

Chatila, Wissam M F. “MicroRNA Expression in Regulatory T Cells in Chronic Obstructive Pulmonary Disease.” 2015. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,333572.

MLA Handbook (7th Edition):

Chatila, Wissam M F. “MicroRNA Expression in Regulatory T Cells in Chronic Obstructive Pulmonary Disease.” 2015. Web. 02 Apr 2020.

Vancouver:

Chatila WMF. MicroRNA Expression in Regulatory T Cells in Chronic Obstructive Pulmonary Disease. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,333572.

Council of Science Editors:

Chatila WMF. MicroRNA Expression in Regulatory T Cells in Chronic Obstructive Pulmonary Disease. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,333572


Temple University

14. Firrman, Jenni Ann. ENHANCEMENT OF hFVIII ACTIVITY THROUGH LC MODIFICATIONS FOR GENE THERAPY OF HEMOPHILIA A.

Degree: PhD, 2015, Temple University

Microbiology and Immunology

Gene therapy for Hemophilia A (HA) using the recombinant Adeno-associated virus (rAAV) offers an alternative to classic treatment, which consists of FVIII… (more)

Subjects/Keywords: Microbiology; Immunology; Genetics;

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APA (6th Edition):

Firrman, J. A. (2015). ENHANCEMENT OF hFVIII ACTIVITY THROUGH LC MODIFICATIONS FOR GENE THERAPY OF HEMOPHILIA A. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,335863

Chicago Manual of Style (16th Edition):

Firrman, Jenni Ann. “ENHANCEMENT OF hFVIII ACTIVITY THROUGH LC MODIFICATIONS FOR GENE THERAPY OF HEMOPHILIA A.” 2015. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,335863.

MLA Handbook (7th Edition):

Firrman, Jenni Ann. “ENHANCEMENT OF hFVIII ACTIVITY THROUGH LC MODIFICATIONS FOR GENE THERAPY OF HEMOPHILIA A.” 2015. Web. 02 Apr 2020.

Vancouver:

Firrman JA. ENHANCEMENT OF hFVIII ACTIVITY THROUGH LC MODIFICATIONS FOR GENE THERAPY OF HEMOPHILIA A. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,335863.

Council of Science Editors:

Firrman JA. ENHANCEMENT OF hFVIII ACTIVITY THROUGH LC MODIFICATIONS FOR GENE THERAPY OF HEMOPHILIA A. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,335863


Temple University

15. Chakhtoura, Marita. Novel protocols to induce tolerance to solid organ transplants.

Degree: PhD, 2016, Temple University

Microbiology and Immunology

Dendritic cells (DCs) are the sentinels of the immune system. They mature at the encounter of the appropriate stimuli or danger signals,… (more)

Subjects/Keywords: Immunology; Medicine;

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APA (6th Edition):

Chakhtoura, M. (2016). Novel protocols to induce tolerance to solid organ transplants. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,383997

Chicago Manual of Style (16th Edition):

Chakhtoura, Marita. “Novel protocols to induce tolerance to solid organ transplants.” 2016. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,383997.

MLA Handbook (7th Edition):

Chakhtoura, Marita. “Novel protocols to induce tolerance to solid organ transplants.” 2016. Web. 02 Apr 2020.

Vancouver:

Chakhtoura M. Novel protocols to induce tolerance to solid organ transplants. [Internet] [Doctoral dissertation]. Temple University; 2016. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,383997.

Council of Science Editors:

Chakhtoura M. Novel protocols to induce tolerance to solid organ transplants. [Doctoral Dissertation]. Temple University; 2016. Available from: http://digital.library.temple.edu/u?/p245801coll10,383997


Temple University

16. Kang, Sun-ah. Apoptotic Cells, Anti-Phospholipid Antibodies, and Anti-Chromatin Antibodies in Autoimmunity.

Degree: PhD, 2008, Temple University

Microbiology and Immunology

Antiphospholipid antibodies (APAs) are detected in various autoimmune diseases, such as antiphospholipid syndrome (APS) and systemic lupus erythematosus. In addition to their… (more)

Subjects/Keywords: Health Sciences, Immunology

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APA (6th Edition):

Kang, S. (2008). Apoptotic Cells, Anti-Phospholipid Antibodies, and Anti-Chromatin Antibodies in Autoimmunity. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,15651

Chicago Manual of Style (16th Edition):

Kang, Sun-ah. “Apoptotic Cells, Anti-Phospholipid Antibodies, and Anti-Chromatin Antibodies in Autoimmunity.” 2008. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,15651.

MLA Handbook (7th Edition):

Kang, Sun-ah. “Apoptotic Cells, Anti-Phospholipid Antibodies, and Anti-Chromatin Antibodies in Autoimmunity.” 2008. Web. 02 Apr 2020.

Vancouver:

Kang S. Apoptotic Cells, Anti-Phospholipid Antibodies, and Anti-Chromatin Antibodies in Autoimmunity. [Internet] [Doctoral dissertation]. Temple University; 2008. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,15651.

Council of Science Editors:

Kang S. Apoptotic Cells, Anti-Phospholipid Antibodies, and Anti-Chromatin Antibodies in Autoimmunity. [Doctoral Dissertation]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,15651


Temple University

17. Vas, Jaya. REGULATORY ROLES FOR NATURAL KILLER T CELLS AND TOLL-LIKE RECEPTORS IN MERCURY-INDUCED AUTOIMMUNITY.

Degree: PhD, 2008, Temple University

Microbiology and Immunology

The development of autoimmune diseases is frequently linked to exposure to environmental factors such as chemicals, drugs or infections. In the experimental… (more)

Subjects/Keywords: Health Sciences; Immunology

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APA (6th Edition):

Vas, J. (2008). REGULATORY ROLES FOR NATURAL KILLER T CELLS AND TOLL-LIKE RECEPTORS IN MERCURY-INDUCED AUTOIMMUNITY. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,20283

Chicago Manual of Style (16th Edition):

Vas, Jaya. “REGULATORY ROLES FOR NATURAL KILLER T CELLS AND TOLL-LIKE RECEPTORS IN MERCURY-INDUCED AUTOIMMUNITY.” 2008. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,20283.

MLA Handbook (7th Edition):

Vas, Jaya. “REGULATORY ROLES FOR NATURAL KILLER T CELLS AND TOLL-LIKE RECEPTORS IN MERCURY-INDUCED AUTOIMMUNITY.” 2008. Web. 02 Apr 2020.

Vancouver:

Vas J. REGULATORY ROLES FOR NATURAL KILLER T CELLS AND TOLL-LIKE RECEPTORS IN MERCURY-INDUCED AUTOIMMUNITY. [Internet] [Doctoral dissertation]. Temple University; 2008. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,20283.

Council of Science Editors:

Vas J. REGULATORY ROLES FOR NATURAL KILLER T CELLS AND TOLL-LIKE RECEPTORS IN MERCURY-INDUCED AUTOIMMUNITY. [Doctoral Dissertation]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,20283


Temple University

18. Piaggio, Eduardo. The Role of PD-1 and Its Ligands in Mercury(Hg)-induced Autoimmunity.

Degree: PhD, 2009, Temple University

Microbiology and Immunology

The development of autoimmune diseases is frequently linked to exposure to environmental factors such as chemicals, drugs or infections. In the experimental… (more)

Subjects/Keywords: Health Sciences, Immunology; Autoimmunity; Mercury; PD-1

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APA (6th Edition):

Piaggio, E. (2009). The Role of PD-1 and Its Ligands in Mercury(Hg)-induced Autoimmunity. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,52042

Chicago Manual of Style (16th Edition):

Piaggio, Eduardo. “The Role of PD-1 and Its Ligands in Mercury(Hg)-induced Autoimmunity.” 2009. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,52042.

MLA Handbook (7th Edition):

Piaggio, Eduardo. “The Role of PD-1 and Its Ligands in Mercury(Hg)-induced Autoimmunity.” 2009. Web. 02 Apr 2020.

Vancouver:

Piaggio E. The Role of PD-1 and Its Ligands in Mercury(Hg)-induced Autoimmunity. [Internet] [Doctoral dissertation]. Temple University; 2009. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,52042.

Council of Science Editors:

Piaggio E. The Role of PD-1 and Its Ligands in Mercury(Hg)-induced Autoimmunity. [Doctoral Dissertation]. Temple University; 2009. Available from: http://digital.library.temple.edu/u?/p245801coll10,52042


Temple University

19. Li, Liping. The role of ICOS and retinoic acid in Mercury-Induced Autoimmunity.

Degree: PhD, 2009, Temple University

Microbiology and Immunology

Metal-induced autoimmunity is an experimental model of environmentally induced autoimmune syndrome. Subtoxic doses of heavy metals administered to genetically susceptible mice resulted… (more)

Subjects/Keywords: Health Sciences, Immunology; ICOS; IL-10; Mercury; Retinoic acid; tolerance; Treg

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APA (6th Edition):

Li, L. (2009). The role of ICOS and retinoic acid in Mercury-Induced Autoimmunity. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,55125

Chicago Manual of Style (16th Edition):

Li, Liping. “The role of ICOS and retinoic acid in Mercury-Induced Autoimmunity.” 2009. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,55125.

MLA Handbook (7th Edition):

Li, Liping. “The role of ICOS and retinoic acid in Mercury-Induced Autoimmunity.” 2009. Web. 02 Apr 2020.

Vancouver:

Li L. The role of ICOS and retinoic acid in Mercury-Induced Autoimmunity. [Internet] [Doctoral dissertation]. Temple University; 2009. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,55125.

Council of Science Editors:

Li L. The role of ICOS and retinoic acid in Mercury-Induced Autoimmunity. [Doctoral Dissertation]. Temple University; 2009. Available from: http://digital.library.temple.edu/u?/p245801coll10,55125


Temple University

20. Jha, Vibha. Cellular regulation of mercury-induced autoimmunity.

Degree: PhD, 2009, Temple University

Microbiology and Immunology

Etiological agents causing autoimmune diseases largely remain unknown. However, several lines of evidence suggest that environmental factors such as heavy metals (arsenic,… (more)

Subjects/Keywords: Health Sciences, Immunology; Anti-CD3; Autoimmunity; LAG-3; Mercury; Murine; Regulatory T cells

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APA (6th Edition):

Jha, V. (2009). Cellular regulation of mercury-induced autoimmunity. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,60597

Chicago Manual of Style (16th Edition):

Jha, Vibha. “Cellular regulation of mercury-induced autoimmunity.” 2009. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,60597.

MLA Handbook (7th Edition):

Jha, Vibha. “Cellular regulation of mercury-induced autoimmunity.” 2009. Web. 02 Apr 2020.

Vancouver:

Jha V. Cellular regulation of mercury-induced autoimmunity. [Internet] [Doctoral dissertation]. Temple University; 2009. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,60597.

Council of Science Editors:

Jha V. Cellular regulation of mercury-induced autoimmunity. [Doctoral Dissertation]. Temple University; 2009. Available from: http://digital.library.temple.edu/u?/p245801coll10,60597


Temple University

21. Hooper, Kirsten Mary. PGE2 AND IL-27: NOVEL PROINFLAMMATORY MECHANISMS INVOLVING DENDRITIC CELLS AND TYPE 1 REGULATORY T CELLS.

Degree: PhD, 2017, Temple University

Microbiology and Immunology

Interleukin-27 (p28/EBI3) is an immunomodulatory cytokine expressed by activated antigen presenting cells. Although first discovered to be involved in Th1 cell differentiation,… (more)

Subjects/Keywords: Immunology;

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APA (6th Edition):

Hooper, K. M. (2017). PGE2 AND IL-27: NOVEL PROINFLAMMATORY MECHANISMS INVOLVING DENDRITIC CELLS AND TYPE 1 REGULATORY T CELLS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,432693

Chicago Manual of Style (16th Edition):

Hooper, Kirsten Mary. “PGE2 AND IL-27: NOVEL PROINFLAMMATORY MECHANISMS INVOLVING DENDRITIC CELLS AND TYPE 1 REGULATORY T CELLS.” 2017. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,432693.

MLA Handbook (7th Edition):

Hooper, Kirsten Mary. “PGE2 AND IL-27: NOVEL PROINFLAMMATORY MECHANISMS INVOLVING DENDRITIC CELLS AND TYPE 1 REGULATORY T CELLS.” 2017. Web. 02 Apr 2020.

Vancouver:

Hooper KM. PGE2 AND IL-27: NOVEL PROINFLAMMATORY MECHANISMS INVOLVING DENDRITIC CELLS AND TYPE 1 REGULATORY T CELLS. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,432693.

Council of Science Editors:

Hooper KM. PGE2 AND IL-27: NOVEL PROINFLAMMATORY MECHANISMS INVOLVING DENDRITIC CELLS AND TYPE 1 REGULATORY T CELLS. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,432693


Temple University

22. Gallo, Paul Matthew. The Dendritic Cell Response to Exogenous and Endogenous Danger Signals.

Degree: PhD, 2017, Temple University

Microbiology and Immunology

Systemic lupus erythematosus (SLE) is complex autoimmune disease in which autoantibodies form against double stranded DNA (dsDNA) and nuclear antigens. Autoantigen immune… (more)

Subjects/Keywords: Biomechanics; Accounting;

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APA (6th Edition):

Gallo, P. M. (2017). The Dendritic Cell Response to Exogenous and Endogenous Danger Signals. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,452916

Chicago Manual of Style (16th Edition):

Gallo, Paul Matthew. “The Dendritic Cell Response to Exogenous and Endogenous Danger Signals.” 2017. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,452916.

MLA Handbook (7th Edition):

Gallo, Paul Matthew. “The Dendritic Cell Response to Exogenous and Endogenous Danger Signals.” 2017. Web. 02 Apr 2020.

Vancouver:

Gallo PM. The Dendritic Cell Response to Exogenous and Endogenous Danger Signals. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,452916.

Council of Science Editors:

Gallo PM. The Dendritic Cell Response to Exogenous and Endogenous Danger Signals. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,452916


Temple University

23. Milora, Katelynn Ann. Characterization of IL-1 and IL-36 Cytokines in Health and Disease.

Degree: PhD, 2017, Temple University

Microbiology and Immunology

Epithelial cells are the first line of defense against invading pathogens and external threats in the environment. Keratinocytes, often not perceived of… (more)

Subjects/Keywords: Immunology;

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APA (6th Edition):

Milora, K. A. (2017). Characterization of IL-1 and IL-36 Cytokines in Health and Disease. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,451599

Chicago Manual of Style (16th Edition):

Milora, Katelynn Ann. “Characterization of IL-1 and IL-36 Cytokines in Health and Disease.” 2017. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,451599.

MLA Handbook (7th Edition):

Milora, Katelynn Ann. “Characterization of IL-1 and IL-36 Cytokines in Health and Disease.” 2017. Web. 02 Apr 2020.

Vancouver:

Milora KA. Characterization of IL-1 and IL-36 Cytokines in Health and Disease. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,451599.

Council of Science Editors:

Milora KA. Characterization of IL-1 and IL-36 Cytokines in Health and Disease. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,451599


Temple University

24. Breslow, Jessica. Effect of Morphine on Immune Responses and Infection.

Degree: PhD, 2010, Temple University

Microbiology and Immunology

Opioids have been shown to modulate immune function in a variety of assays and animal models. In a more limited number of… (more)

Subjects/Keywords: Microbiology; Acinetobacter baumannii; Immunity; Infection; Morphine; Opioids; Salmonella enterica serovar Typhimurium

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APA (6th Edition):

Breslow, J. (2010). Effect of Morphine on Immune Responses and Infection. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,103429

Chicago Manual of Style (16th Edition):

Breslow, Jessica. “Effect of Morphine on Immune Responses and Infection.” 2010. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,103429.

MLA Handbook (7th Edition):

Breslow, Jessica. “Effect of Morphine on Immune Responses and Infection.” 2010. Web. 02 Apr 2020.

Vancouver:

Breslow J. Effect of Morphine on Immune Responses and Infection. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,103429.

Council of Science Editors:

Breslow J. Effect of Morphine on Immune Responses and Infection. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,103429


Temple University

25. Ingram, Justin Phillip. The Role of the Innate Immune System in Programmed Cell Death.

Degree: PhD, 2018, Temple University

Biomedical Sciences

Infectious diseases are the leading cause of illness worldwide, leading to over 20 million hospitalizations each year in the United States alone. Although… (more)

Subjects/Keywords: Immunology;

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APA (6th Edition):

Ingram, J. P. (2018). The Role of the Innate Immune System in Programmed Cell Death. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,516594

Chicago Manual of Style (16th Edition):

Ingram, Justin Phillip. “The Role of the Innate Immune System in Programmed Cell Death.” 2018. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,516594.

MLA Handbook (7th Edition):

Ingram, Justin Phillip. “The Role of the Innate Immune System in Programmed Cell Death.” 2018. Web. 02 Apr 2020.

Vancouver:

Ingram JP. The Role of the Innate Immune System in Programmed Cell Death. [Internet] [Doctoral dissertation]. Temple University; 2018. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,516594.

Council of Science Editors:

Ingram JP. The Role of the Innate Immune System in Programmed Cell Death. [Doctoral Dissertation]. Temple University; 2018. Available from: http://digital.library.temple.edu/u?/p245801coll10,516594


Temple University

26. Nwaneshiudu, Adaobi I. The Role of Gamma-Delta TCR+ T-cells in the Pathogenesis of Systemic Sclerosis.

Degree: PhD, 2008, Temple University

Microbiology and Immunology

The human gamma-delta (gd) TCR+ T-cell subset may undergo specific antigen-driven activation and clonal expansion, in the context of systemic sclerosis (SSc)… (more)

Subjects/Keywords: Health Sciences, Immunology; Gamma-delta TCR+ T-cells; Putative antigens; Clonal expansion; Systemic Sclerosis; Autoimmune disease; Fibrosis

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APA (6th Edition):

Nwaneshiudu, A. I. (2008). The Role of Gamma-Delta TCR+ T-cells in the Pathogenesis of Systemic Sclerosis. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,11843

Chicago Manual of Style (16th Edition):

Nwaneshiudu, Adaobi I. “The Role of Gamma-Delta TCR+ T-cells in the Pathogenesis of Systemic Sclerosis.” 2008. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,11843.

MLA Handbook (7th Edition):

Nwaneshiudu, Adaobi I. “The Role of Gamma-Delta TCR+ T-cells in the Pathogenesis of Systemic Sclerosis.” 2008. Web. 02 Apr 2020.

Vancouver:

Nwaneshiudu AI. The Role of Gamma-Delta TCR+ T-cells in the Pathogenesis of Systemic Sclerosis. [Internet] [Doctoral dissertation]. Temple University; 2008. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,11843.

Council of Science Editors:

Nwaneshiudu AI. The Role of Gamma-Delta TCR+ T-cells in the Pathogenesis of Systemic Sclerosis. [Doctoral Dissertation]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,11843

.