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You searched for +publisher:"Temple University" +contributor:("Kunapuli, Satya P.;"). Showing records 1 – 15 of 15 total matches.

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Temple University

1. Bhavaraju, Kamala. MOLECULAR PHYSIOLOGY OF THROMBOXANE A2 GENERATION IN PLATELETS.

Degree: PhD, 2010, Temple University

 <p>Molecular and Cellular Physiology p><p>Cardiovascular diseases are a major cause of mortality and morbidity in the developed countries. Anti-platelet therapy is a cornerstone treatment for… (more)

Subjects/Keywords: Biology, Physiology; ADP Receptors; Anti-platelet Therapy; G12/13 Pathways; Platelets; Serum Thromboxane; Thrombin

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APA (6th Edition):

Bhavaraju, K. (2010). MOLECULAR PHYSIOLOGY OF THROMBOXANE A2 GENERATION IN PLATELETS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,92746

Chicago Manual of Style (16th Edition):

Bhavaraju, Kamala. “MOLECULAR PHYSIOLOGY OF THROMBOXANE A2 GENERATION IN PLATELETS.” 2010. Doctoral Dissertation, Temple University. Accessed September 19, 2019. http://digital.library.temple.edu/u?/p245801coll10,92746.

MLA Handbook (7th Edition):

Bhavaraju, Kamala. “MOLECULAR PHYSIOLOGY OF THROMBOXANE A2 GENERATION IN PLATELETS.” 2010. Web. 19 Sep 2019.

Vancouver:

Bhavaraju K. MOLECULAR PHYSIOLOGY OF THROMBOXANE A2 GENERATION IN PLATELETS. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2019 Sep 19]. Available from: http://digital.library.temple.edu/u?/p245801coll10,92746.

Council of Science Editors:

Bhavaraju K. MOLECULAR PHYSIOLOGY OF THROMBOXANE A2 GENERATION IN PLATELETS. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,92746


Temple University

2. Buitrago Murcia, Claudia Lorena. Cbl proteins in platelet functional responses.

Degree: PhD, 2012, Temple University

 <p>Physiology p><p>c-Cbl protein functions as an E3 ligase and scaffolding protein, where three residues, Y700, Y731, and Y774, upon phosphorylation, have been shown to initiate… (more)

Subjects/Keywords: Physiology; Cellular biology; Cbl; hemostasis; kinases; platelets; signaling

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APA (6th Edition):

Buitrago Murcia, C. L. (2012). Cbl proteins in platelet functional responses. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,198139

Chicago Manual of Style (16th Edition):

Buitrago Murcia, Claudia Lorena. “Cbl proteins in platelet functional responses.” 2012. Doctoral Dissertation, Temple University. Accessed September 19, 2019. http://digital.library.temple.edu/u?/p245801coll10,198139.

MLA Handbook (7th Edition):

Buitrago Murcia, Claudia Lorena. “Cbl proteins in platelet functional responses.” 2012. Web. 19 Sep 2019.

Vancouver:

Buitrago Murcia CL. Cbl proteins in platelet functional responses. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2019 Sep 19]. Available from: http://digital.library.temple.edu/u?/p245801coll10,198139.

Council of Science Editors:

Buitrago Murcia CL. Cbl proteins in platelet functional responses. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,198139


Temple University

3. Bhavanasi, Dheeraj. Role of Protein Kinase C delta in regulating platelet functional responses.

Degree: PhD, 2014, Temple University

 <p>Physiology p><p>Platelets upon activation change their shape, aggregate and secrete alpha and dense granule contents among which ADP acts as a feedback activator. Different Protein… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Bhavanasi, D. (2014). Role of Protein Kinase C delta in regulating platelet functional responses. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,283099

Chicago Manual of Style (16th Edition):

Bhavanasi, Dheeraj. “Role of Protein Kinase C delta in regulating platelet functional responses.” 2014. Doctoral Dissertation, Temple University. Accessed September 19, 2019. http://digital.library.temple.edu/u?/p245801coll10,283099.

MLA Handbook (7th Edition):

Bhavanasi, Dheeraj. “Role of Protein Kinase C delta in regulating platelet functional responses.” 2014. Web. 19 Sep 2019.

Vancouver:

Bhavanasi D. Role of Protein Kinase C delta in regulating platelet functional responses. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Sep 19]. Available from: http://digital.library.temple.edu/u?/p245801coll10,283099.

Council of Science Editors:

Bhavanasi D. Role of Protein Kinase C delta in regulating platelet functional responses. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,283099


Temple University

4. BADOLIA, RACHIT. MOLECULAR PHYSIOLOGY OF p21-ACTIVATED KINASES.

Degree: PhD, 2015, Temple University

 <p>Organ Systems & Translational Medicine p><p>Platelets are involved in many processes ranging from fighting microbial infections and triggering inflammation to promoting tumor angiogenesis and metastasis.… (more)

Subjects/Keywords: Physiology;

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APA (6th Edition):

BADOLIA, R. (2015). MOLECULAR PHYSIOLOGY OF p21-ACTIVATED KINASES. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,368893

Chicago Manual of Style (16th Edition):

BADOLIA, RACHIT. “MOLECULAR PHYSIOLOGY OF p21-ACTIVATED KINASES.” 2015. Doctoral Dissertation, Temple University. Accessed September 19, 2019. http://digital.library.temple.edu/u?/p245801coll10,368893.

MLA Handbook (7th Edition):

BADOLIA, RACHIT. “MOLECULAR PHYSIOLOGY OF p21-ACTIVATED KINASES.” 2015. Web. 19 Sep 2019.

Vancouver:

BADOLIA R. MOLECULAR PHYSIOLOGY OF p21-ACTIVATED KINASES. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Sep 19]. Available from: http://digital.library.temple.edu/u?/p245801coll10,368893.

Council of Science Editors:

BADOLIA R. MOLECULAR PHYSIOLOGY OF p21-ACTIVATED KINASES. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,368893


Temple University

5. Soroush, Fariborz. A Novel Microfluidic System for Screening Anti-inflammatory Therapeutics.

Degree: PhD, 2018, Temple University

 <p>Mechanical Engineering p><p>Inflammation is a crucial physiological protective response of body to infection or injury. However, in pathological conditions such as sepsis or radiation damage,… (more)

Subjects/Keywords: Bioengineering; Fluid mechanics; Immunology

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APA (6th Edition):

Soroush, F. (2018). A Novel Microfluidic System for Screening Anti-inflammatory Therapeutics. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,500666

Chicago Manual of Style (16th Edition):

Soroush, Fariborz. “A Novel Microfluidic System for Screening Anti-inflammatory Therapeutics.” 2018. Doctoral Dissertation, Temple University. Accessed September 19, 2019. http://digital.library.temple.edu/u?/p245801coll10,500666.

MLA Handbook (7th Edition):

Soroush, Fariborz. “A Novel Microfluidic System for Screening Anti-inflammatory Therapeutics.” 2018. Web. 19 Sep 2019.

Vancouver:

Soroush F. A Novel Microfluidic System for Screening Anti-inflammatory Therapeutics. [Internet] [Doctoral dissertation]. Temple University; 2018. [cited 2019 Sep 19]. Available from: http://digital.library.temple.edu/u?/p245801coll10,500666.

Council of Science Editors:

Soroush F. A Novel Microfluidic System for Screening Anti-inflammatory Therapeutics. [Doctoral Dissertation]. Temple University; 2018. Available from: http://digital.library.temple.edu/u?/p245801coll10,500666


Temple University

6. Chari, Ramya. Molecular Mechanisms Underlying Differential Regulation of Platelet Dense Granule Secretion by Protein Kinase C delta.

Degree: PhD, 2010, Temple University

 <p>Physiology p><p>Protein Kinase C delta (PKCδ) is expressed in platelets and activated downstream of protease-activated receptors (PAR)s and glycoprotein VI (GPVI) receptors. We evaluated the… (more)

Subjects/Keywords: Biology, Physiology; Biology, Cell; Biology, Molecular; Dense Granule Secretion; Phosphorylation; Platelets; Protein Kinase C; Signaling; Thrombosis

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APA (6th Edition):

Chari, R. (2010). Molecular Mechanisms Underlying Differential Regulation of Platelet Dense Granule Secretion by Protein Kinase C delta. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,77283

Chicago Manual of Style (16th Edition):

Chari, Ramya. “Molecular Mechanisms Underlying Differential Regulation of Platelet Dense Granule Secretion by Protein Kinase C delta.” 2010. Doctoral Dissertation, Temple University. Accessed September 19, 2019. http://digital.library.temple.edu/u?/p245801coll10,77283.

MLA Handbook (7th Edition):

Chari, Ramya. “Molecular Mechanisms Underlying Differential Regulation of Platelet Dense Granule Secretion by Protein Kinase C delta.” 2010. Web. 19 Sep 2019.

Vancouver:

Chari R. Molecular Mechanisms Underlying Differential Regulation of Platelet Dense Granule Secretion by Protein Kinase C delta. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2019 Sep 19]. Available from: http://digital.library.temple.edu/u?/p245801coll10,77283.

Council of Science Editors:

Chari R. Molecular Mechanisms Underlying Differential Regulation of Platelet Dense Granule Secretion by Protein Kinase C delta. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,77283


Temple University

7. Mayanglambam, Azad. Regulation of Protein Kinases (Syk and PKC zeta) in platelets.

Degree: PhD, 2010, Temple University

 <p>Physiology p><p>Platelets are crucial components of the hemostatic machinery of the body. When the endothelial continuity is disrupted due to injury or atherosclerotic plaque rupture,… (more)

Subjects/Keywords: Biology, Physiology; Biology, Molecular; Hemostasis; Platelets; Protein Kinase C; Signaling; Syk; Thrombosis

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APA (6th Edition):

Mayanglambam, A. (2010). Regulation of Protein Kinases (Syk and PKC zeta) in platelets. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,91635

Chicago Manual of Style (16th Edition):

Mayanglambam, Azad. “Regulation of Protein Kinases (Syk and PKC zeta) in platelets.” 2010. Doctoral Dissertation, Temple University. Accessed September 19, 2019. http://digital.library.temple.edu/u?/p245801coll10,91635.

MLA Handbook (7th Edition):

Mayanglambam, Azad. “Regulation of Protein Kinases (Syk and PKC zeta) in platelets.” 2010. Web. 19 Sep 2019.

Vancouver:

Mayanglambam A. Regulation of Protein Kinases (Syk and PKC zeta) in platelets. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2019 Sep 19]. Available from: http://digital.library.temple.edu/u?/p245801coll10,91635.

Council of Science Editors:

Mayanglambam A. Regulation of Protein Kinases (Syk and PKC zeta) in platelets. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,91635


Temple University

8. Thomas, Dafydd Huw. Regulation of Syk activity in GPVI-mediated platelet activation.

Degree: PhD, 2010, Temple University

 <p>Pharmacology p><p>Activation of platelets is essential for hemostasis. Following damage to the vascular endothelium collagen is exposed, to which platelets stably adhere. After adhesion on… (more)

Subjects/Keywords: Health Sciences, Pharmacology; Collagen; GPVI; Phosphatase; Platelets; Syk; TULA

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APA (6th Edition):

Thomas, D. H. (2010). Regulation of Syk activity in GPVI-mediated platelet activation. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,92028

Chicago Manual of Style (16th Edition):

Thomas, Dafydd Huw. “Regulation of Syk activity in GPVI-mediated platelet activation.” 2010. Doctoral Dissertation, Temple University. Accessed September 19, 2019. http://digital.library.temple.edu/u?/p245801coll10,92028.

MLA Handbook (7th Edition):

Thomas, Dafydd Huw. “Regulation of Syk activity in GPVI-mediated platelet activation.” 2010. Web. 19 Sep 2019.

Vancouver:

Thomas DH. Regulation of Syk activity in GPVI-mediated platelet activation. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2019 Sep 19]. Available from: http://digital.library.temple.edu/u?/p245801coll10,92028.

Council of Science Editors:

Thomas DH. Regulation of Syk activity in GPVI-mediated platelet activation. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,92028


Temple University

9. Bynagari, Yamini Saraswathy. Molecular Physiology of Novel Class of Protein Kinase C isoforms in Platelets.

Degree: PhD, 2010, Temple University

 <p>Molecular and Cellular Physiology p><p>Platelets are primary components of hemostasis. However, incongruous activation of platelets lead to thrombosis, which result in multiple cardio-vascular and cerebrovascular… (more)

Subjects/Keywords: Biology, Cell; Biology, Physiology; ADP receptors; Phosphatases; Platelet signaling; Protein Kinase C; thromboxane

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APA (6th Edition):

Bynagari, Y. S. (2010). Molecular Physiology of Novel Class of Protein Kinase C isoforms in Platelets. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,103230

Chicago Manual of Style (16th Edition):

Bynagari, Yamini Saraswathy. “Molecular Physiology of Novel Class of Protein Kinase C isoforms in Platelets.” 2010. Doctoral Dissertation, Temple University. Accessed September 19, 2019. http://digital.library.temple.edu/u?/p245801coll10,103230.

MLA Handbook (7th Edition):

Bynagari, Yamini Saraswathy. “Molecular Physiology of Novel Class of Protein Kinase C isoforms in Platelets.” 2010. Web. 19 Sep 2019.

Vancouver:

Bynagari YS. Molecular Physiology of Novel Class of Protein Kinase C isoforms in Platelets. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2019 Sep 19]. Available from: http://digital.library.temple.edu/u?/p245801coll10,103230.

Council of Science Editors:

Bynagari YS. Molecular Physiology of Novel Class of Protein Kinase C isoforms in Platelets. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,103230


Temple University

10. Getz, Todd Michael. The Regulation of Phosphorylation Events in Platelets.

Degree: PhD, 2012, Temple University

 <p>Physiology p><p>Platelets play a vital role in processes of hemostasis and thrombosis under physiological and pathological conditions. Following vascular damage, platelets will accumulate and stably… (more)

Subjects/Keywords: Physiology; Biology; Biochemistry; Dextran sulfate; Myosin light chain; Phosphorylation; Platelets; signal transduction; Syk kinase

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APA (6th Edition):

Getz, T. M. (2012). The Regulation of Phosphorylation Events in Platelets. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,213114

Chicago Manual of Style (16th Edition):

Getz, Todd Michael. “The Regulation of Phosphorylation Events in Platelets.” 2012. Doctoral Dissertation, Temple University. Accessed September 19, 2019. http://digital.library.temple.edu/u?/p245801coll10,213114.

MLA Handbook (7th Edition):

Getz, Todd Michael. “The Regulation of Phosphorylation Events in Platelets.” 2012. Web. 19 Sep 2019.

Vancouver:

Getz TM. The Regulation of Phosphorylation Events in Platelets. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2019 Sep 19]. Available from: http://digital.library.temple.edu/u?/p245801coll10,213114.

Council of Science Editors:

Getz TM. The Regulation of Phosphorylation Events in Platelets. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,213114


Temple University

11. Manne, Bhanu Kanth. CLEC-2 SIGNAL TRANSDUCTION IN PLATELET ACTIVATION.

Degree: PhD, 2015, Temple University

 <p>Physiology p><p>Platelets are involved in many processes ranging from fighting microbial infections and triggering inflammation to promoting tumor angiogenesis and metastasis. Nevertheless, the primary physiological… (more)

Subjects/Keywords: Physiology; Cellular biology;

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APA (6th Edition):

Manne, B. K. (2015). CLEC-2 SIGNAL TRANSDUCTION IN PLATELET ACTIVATION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,340495

Chicago Manual of Style (16th Edition):

Manne, Bhanu Kanth. “CLEC-2 SIGNAL TRANSDUCTION IN PLATELET ACTIVATION.” 2015. Doctoral Dissertation, Temple University. Accessed September 19, 2019. http://digital.library.temple.edu/u?/p245801coll10,340495.

MLA Handbook (7th Edition):

Manne, Bhanu Kanth. “CLEC-2 SIGNAL TRANSDUCTION IN PLATELET ACTIVATION.” 2015. Web. 19 Sep 2019.

Vancouver:

Manne BK. CLEC-2 SIGNAL TRANSDUCTION IN PLATELET ACTIVATION. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Sep 19]. Available from: http://digital.library.temple.edu/u?/p245801coll10,340495.

Council of Science Editors:

Manne BK. CLEC-2 SIGNAL TRANSDUCTION IN PLATELET ACTIVATION. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,340495


Temple University

12. Inamdar, Vaishali Vijay. FUNCTIONAL PROTEIN TYROSINE PHOSPHATASES IN PLATELETS.

Degree: PhD, 2017, Temple University

 <p>Biomedical Sciences p><p>Platelets are small anucleate cells in blood that are derived from megakaryocytes and their primary function is to prevent bleeding. Upon vascular injury,… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Inamdar, V. V. (2017). FUNCTIONAL PROTEIN TYROSINE PHOSPHATASES IN PLATELETS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,473257

Chicago Manual of Style (16th Edition):

Inamdar, Vaishali Vijay. “FUNCTIONAL PROTEIN TYROSINE PHOSPHATASES IN PLATELETS.” 2017. Doctoral Dissertation, Temple University. Accessed September 19, 2019. http://digital.library.temple.edu/u?/p245801coll10,473257.

MLA Handbook (7th Edition):

Inamdar, Vaishali Vijay. “FUNCTIONAL PROTEIN TYROSINE PHOSPHATASES IN PLATELETS.” 2017. Web. 19 Sep 2019.

Vancouver:

Inamdar VV. FUNCTIONAL PROTEIN TYROSINE PHOSPHATASES IN PLATELETS. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2019 Sep 19]. Available from: http://digital.library.temple.edu/u?/p245801coll10,473257.

Council of Science Editors:

Inamdar VV. FUNCTIONAL PROTEIN TYROSINE PHOSPHATASES IN PLATELETS. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,473257


Temple University

13. Patel, Akruti. NOVEL REGULATORS OF GPVI-MEDIATED PLATELET ACTIVATION.

Degree: PhD, 2017, Temple University

 <p>Biomedical Sciences p><p>Platelets are anucleate cells that are crucial mediators of hemostasis and thrombosis. Under physiological conditions, platelets are maintained in a quiescent state within… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Patel, A. (2017). NOVEL REGULATORS OF GPVI-MEDIATED PLATELET ACTIVATION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,473397

Chicago Manual of Style (16th Edition):

Patel, Akruti. “NOVEL REGULATORS OF GPVI-MEDIATED PLATELET ACTIVATION.” 2017. Doctoral Dissertation, Temple University. Accessed September 19, 2019. http://digital.library.temple.edu/u?/p245801coll10,473397.

MLA Handbook (7th Edition):

Patel, Akruti. “NOVEL REGULATORS OF GPVI-MEDIATED PLATELET ACTIVATION.” 2017. Web. 19 Sep 2019.

Vancouver:

Patel A. NOVEL REGULATORS OF GPVI-MEDIATED PLATELET ACTIVATION. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2019 Sep 19]. Available from: http://digital.library.temple.edu/u?/p245801coll10,473397.

Council of Science Editors:

Patel A. NOVEL REGULATORS OF GPVI-MEDIATED PLATELET ACTIVATION. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,473397


Temple University

14. Meng, Shu. HYPERHOMOCYSTEINEMIA ACCELERATES THROMBOSIS THROUGH ICAM-1 DEPENDENT ENDOTHELIAL ACTIVATION AND DNA HYPOMETHYLATION.

Degree: PhD, 2013, Temple University

 <p>Pharmacology p><p>Background: Hyperhomocysteinemia (HHcy) is an established risk factor for thrombotic diseases yet the underlying mechanism remain unclear. In this study we investigated the effect… (more)

Subjects/Keywords: Pharmacology;

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APA (6th Edition):

Meng, S. (2013). HYPERHOMOCYSTEINEMIA ACCELERATES THROMBOSIS THROUGH ICAM-1 DEPENDENT ENDOTHELIAL ACTIVATION AND DNA HYPOMETHYLATION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,234268

Chicago Manual of Style (16th Edition):

Meng, Shu. “HYPERHOMOCYSTEINEMIA ACCELERATES THROMBOSIS THROUGH ICAM-1 DEPENDENT ENDOTHELIAL ACTIVATION AND DNA HYPOMETHYLATION.” 2013. Doctoral Dissertation, Temple University. Accessed September 19, 2019. http://digital.library.temple.edu/u?/p245801coll10,234268.

MLA Handbook (7th Edition):

Meng, Shu. “HYPERHOMOCYSTEINEMIA ACCELERATES THROMBOSIS THROUGH ICAM-1 DEPENDENT ENDOTHELIAL ACTIVATION AND DNA HYPOMETHYLATION.” 2013. Web. 19 Sep 2019.

Vancouver:

Meng S. HYPERHOMOCYSTEINEMIA ACCELERATES THROMBOSIS THROUGH ICAM-1 DEPENDENT ENDOTHELIAL ACTIVATION AND DNA HYPOMETHYLATION. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2019 Sep 19]. Available from: http://digital.library.temple.edu/u?/p245801coll10,234268.

Council of Science Editors:

Meng S. HYPERHOMOCYSTEINEMIA ACCELERATES THROMBOSIS THROUGH ICAM-1 DEPENDENT ENDOTHELIAL ACTIVATION AND DNA HYPOMETHYLATION. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,234268


Temple University

15. Mao, Yingying. ROLE OF PROTEASE-ACTIVATED RECEPTORS IN PLATELET ACTIVATION.

Degree: PhD, 2009, Temple University

 <p>Physiology p><p>Platelets act as a fundamental component of the hemostatic process and their activation leads to the formation of a stable clot at the injured… (more)

Subjects/Keywords: Biology, Physiology; ischemic injury; PAR1 agonist; plasmin; platelet; protease-activated receptor

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APA (6th Edition):

Mao, Y. (2009). ROLE OF PROTEASE-ACTIVATED RECEPTORS IN PLATELET ACTIVATION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,47279

Chicago Manual of Style (16th Edition):

Mao, Yingying. “ROLE OF PROTEASE-ACTIVATED RECEPTORS IN PLATELET ACTIVATION.” 2009. Doctoral Dissertation, Temple University. Accessed September 19, 2019. http://digital.library.temple.edu/u?/p245801coll10,47279.

MLA Handbook (7th Edition):

Mao, Yingying. “ROLE OF PROTEASE-ACTIVATED RECEPTORS IN PLATELET ACTIVATION.” 2009. Web. 19 Sep 2019.

Vancouver:

Mao Y. ROLE OF PROTEASE-ACTIVATED RECEPTORS IN PLATELET ACTIVATION. [Internet] [Doctoral dissertation]. Temple University; 2009. [cited 2019 Sep 19]. Available from: http://digital.library.temple.edu/u?/p245801coll10,47279.

Council of Science Editors:

Mao Y. ROLE OF PROTEASE-ACTIVATED RECEPTORS IN PLATELET ACTIVATION. [Doctoral Dissertation]. Temple University; 2009. Available from: http://digital.library.temple.edu/u?/p245801coll10,47279

.