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You searched for +publisher:"Temple University" +contributor:("Koch, Walter J.;"). Showing records 1 – 10 of 10 total matches.

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Temple University

1. Myers, Valerie. The Role of BAG3 in the Failing Heart.

Degree: PhD, 2018, Temple University

Biomedical Sciences

Heart disease has been the leading cause of death in the United States for more than 90 years. The leading cause of death… (more)

Subjects/Keywords: Cellular biology;

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APA (6th Edition):

Myers, V. (2018). The Role of BAG3 in the Failing Heart. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,490584

Chicago Manual of Style (16th Edition):

Myers, Valerie. “The Role of BAG3 in the Failing Heart.” 2018. Doctoral Dissertation, Temple University. Accessed July 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,490584.

MLA Handbook (7th Edition):

Myers, Valerie. “The Role of BAG3 in the Failing Heart.” 2018. Web. 02 Jul 2020.

Vancouver:

Myers V. The Role of BAG3 in the Failing Heart. [Internet] [Doctoral dissertation]. Temple University; 2018. [cited 2020 Jul 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,490584.

Council of Science Editors:

Myers V. The Role of BAG3 in the Failing Heart. [Doctoral Dissertation]. Temple University; 2018. Available from: http://digital.library.temple.edu/u?/p245801coll10,490584


Temple University

2. Harper, Shavonn Christine. The Effects of Growth Differentiation Factor 11 on Pathological Cardiac Hypertrophy.

Degree: PhD, 2018, Temple University

Biomedical Sciences

Pathological cardiac hypertrophy (PCH) occurs in response to pathological stimuli affecting the heart such as coronary artery disease, myocardial infarction, or hypertension. PCH… (more)

Subjects/Keywords: Physiology;

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APA (6th Edition):

Harper, S. C. (2018). The Effects of Growth Differentiation Factor 11 on Pathological Cardiac Hypertrophy. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,498061

Chicago Manual of Style (16th Edition):

Harper, Shavonn Christine. “The Effects of Growth Differentiation Factor 11 on Pathological Cardiac Hypertrophy.” 2018. Doctoral Dissertation, Temple University. Accessed July 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,498061.

MLA Handbook (7th Edition):

Harper, Shavonn Christine. “The Effects of Growth Differentiation Factor 11 on Pathological Cardiac Hypertrophy.” 2018. Web. 02 Jul 2020.

Vancouver:

Harper SC. The Effects of Growth Differentiation Factor 11 on Pathological Cardiac Hypertrophy. [Internet] [Doctoral dissertation]. Temple University; 2018. [cited 2020 Jul 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,498061.

Council of Science Editors:

Harper SC. The Effects of Growth Differentiation Factor 11 on Pathological Cardiac Hypertrophy. [Doctoral Dissertation]. Temple University; 2018. Available from: http://digital.library.temple.edu/u?/p245801coll10,498061


Temple University

3. Wasilewski, Melissa Anne. ABSENCE OF V1AR ALTERS CARDIAC FUNCTION AT BASELINE AND FOLLOWING ACUTE CARDIAC INJURY.

Degree: PhD, 2018, Temple University

Biomedical Sciences

Heart failure (HF) is a prevalent disease affecting over 5.1 million people in the United States and over 23 million people worldwide. Many… (more)

Subjects/Keywords: Molecular biology; Cellular biology

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APA (6th Edition):

Wasilewski, M. A. (2018). ABSENCE OF V1AR ALTERS CARDIAC FUNCTION AT BASELINE AND FOLLOWING ACUTE CARDIAC INJURY. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,525634

Chicago Manual of Style (16th Edition):

Wasilewski, Melissa Anne. “ABSENCE OF V1AR ALTERS CARDIAC FUNCTION AT BASELINE AND FOLLOWING ACUTE CARDIAC INJURY.” 2018. Doctoral Dissertation, Temple University. Accessed July 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,525634.

MLA Handbook (7th Edition):

Wasilewski, Melissa Anne. “ABSENCE OF V1AR ALTERS CARDIAC FUNCTION AT BASELINE AND FOLLOWING ACUTE CARDIAC INJURY.” 2018. Web. 02 Jul 2020.

Vancouver:

Wasilewski MA. ABSENCE OF V1AR ALTERS CARDIAC FUNCTION AT BASELINE AND FOLLOWING ACUTE CARDIAC INJURY. [Internet] [Doctoral dissertation]. Temple University; 2018. [cited 2020 Jul 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,525634.

Council of Science Editors:

Wasilewski MA. ABSENCE OF V1AR ALTERS CARDIAC FUNCTION AT BASELINE AND FOLLOWING ACUTE CARDIAC INJURY. [Doctoral Dissertation]. Temple University; 2018. Available from: http://digital.library.temple.edu/u?/p245801coll10,525634


Temple University

4. Luongo, Timothy Scott. The Role of Mitochondrial Calcium Exchange in Cardiac Physiology and Disease.

Degree: PhD, 2017, Temple University

Biomedical Sciences

The high metabolic demand of the heart makes it essential that an efficient and tightly controlled system be in place to regulate energy… (more)

Subjects/Keywords: Molecular biology; Cellular biology; Physiology;

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APA (6th Edition):

Luongo, T. S. (2017). The Role of Mitochondrial Calcium Exchange in Cardiac Physiology and Disease. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,437718

Chicago Manual of Style (16th Edition):

Luongo, Timothy Scott. “The Role of Mitochondrial Calcium Exchange in Cardiac Physiology and Disease.” 2017. Doctoral Dissertation, Temple University. Accessed July 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,437718.

MLA Handbook (7th Edition):

Luongo, Timothy Scott. “The Role of Mitochondrial Calcium Exchange in Cardiac Physiology and Disease.” 2017. Web. 02 Jul 2020.

Vancouver:

Luongo TS. The Role of Mitochondrial Calcium Exchange in Cardiac Physiology and Disease. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2020 Jul 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,437718.

Council of Science Editors:

Luongo TS. The Role of Mitochondrial Calcium Exchange in Cardiac Physiology and Disease. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,437718


Temple University

5. Sharp III, Thomas E. DRUG AND CELL–BASED THERAPIES TO REDUCE PATHOLOGICAL REMODELING AND CARDIAC DYSFUNCTION AFTER ACUTE MYOCARDIAL INFARCTION.

Degree: PhD, 2017, Temple University

Physiology

Remarkable advances have been made in the treatment of cardiovascular diseases (CVD), however, CVD still accounts for the most deaths in industrialized nations. Ischemic… (more)

Subjects/Keywords: Physiology;

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APA (6th Edition):

Sharp III, T. E. (2017). DRUG AND CELL–BASED THERAPIES TO REDUCE PATHOLOGICAL REMODELING AND CARDIAC DYSFUNCTION AFTER ACUTE MYOCARDIAL INFARCTION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,445275

Chicago Manual of Style (16th Edition):

Sharp III, Thomas E. “DRUG AND CELL–BASED THERAPIES TO REDUCE PATHOLOGICAL REMODELING AND CARDIAC DYSFUNCTION AFTER ACUTE MYOCARDIAL INFARCTION.” 2017. Doctoral Dissertation, Temple University. Accessed July 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,445275.

MLA Handbook (7th Edition):

Sharp III, Thomas E. “DRUG AND CELL–BASED THERAPIES TO REDUCE PATHOLOGICAL REMODELING AND CARDIAC DYSFUNCTION AFTER ACUTE MYOCARDIAL INFARCTION.” 2017. Web. 02 Jul 2020.

Vancouver:

Sharp III TE. DRUG AND CELL–BASED THERAPIES TO REDUCE PATHOLOGICAL REMODELING AND CARDIAC DYSFUNCTION AFTER ACUTE MYOCARDIAL INFARCTION. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2020 Jul 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,445275.

Council of Science Editors:

Sharp III TE. DRUG AND CELL–BASED THERAPIES TO REDUCE PATHOLOGICAL REMODELING AND CARDIAC DYSFUNCTION AFTER ACUTE MYOCARDIAL INFARCTION. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,445275


Temple University

6. Chakhtoura, Marita. Novel protocols to induce tolerance to solid organ transplants.

Degree: PhD, 2016, Temple University

Microbiology and Immunology

Dendritic cells (DCs) are the sentinels of the immune system. They mature at the encounter of the appropriate stimuli or danger signals,… (more)

Subjects/Keywords: Immunology; Medicine;

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APA (6th Edition):

Chakhtoura, M. (2016). Novel protocols to induce tolerance to solid organ transplants. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,383997

Chicago Manual of Style (16th Edition):

Chakhtoura, Marita. “Novel protocols to induce tolerance to solid organ transplants.” 2016. Doctoral Dissertation, Temple University. Accessed July 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,383997.

MLA Handbook (7th Edition):

Chakhtoura, Marita. “Novel protocols to induce tolerance to solid organ transplants.” 2016. Web. 02 Jul 2020.

Vancouver:

Chakhtoura M. Novel protocols to induce tolerance to solid organ transplants. [Internet] [Doctoral dissertation]. Temple University; 2016. [cited 2020 Jul 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,383997.

Council of Science Editors:

Chakhtoura M. Novel protocols to induce tolerance to solid organ transplants. [Doctoral Dissertation]. Temple University; 2016. Available from: http://digital.library.temple.edu/u?/p245801coll10,383997


Temple University

7. Woodall, Benjamin Philip. NOVEL ROLES FOR GRK2 IN METABOLIC HOMEOSTASIS AND SKELETAL MUSCLE PHYSIOLOGY.

Degree: PhD, 2016, Temple University

Biomedical Sciences

Over the past two decades, a vast body of research has demonstrated the importance of G protein-coupled receptor kinase 2 (GRK2) in the… (more)

Subjects/Keywords: Molecular biology;

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APA (6th Edition):

Woodall, B. P. (2016). NOVEL ROLES FOR GRK2 IN METABOLIC HOMEOSTASIS AND SKELETAL MUSCLE PHYSIOLOGY. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,391153

Chicago Manual of Style (16th Edition):

Woodall, Benjamin Philip. “NOVEL ROLES FOR GRK2 IN METABOLIC HOMEOSTASIS AND SKELETAL MUSCLE PHYSIOLOGY.” 2016. Doctoral Dissertation, Temple University. Accessed July 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,391153.

MLA Handbook (7th Edition):

Woodall, Benjamin Philip. “NOVEL ROLES FOR GRK2 IN METABOLIC HOMEOSTASIS AND SKELETAL MUSCLE PHYSIOLOGY.” 2016. Web. 02 Jul 2020.

Vancouver:

Woodall BP. NOVEL ROLES FOR GRK2 IN METABOLIC HOMEOSTASIS AND SKELETAL MUSCLE PHYSIOLOGY. [Internet] [Doctoral dissertation]. Temple University; 2016. [cited 2020 Jul 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,391153.

Council of Science Editors:

Woodall BP. NOVEL ROLES FOR GRK2 IN METABOLIC HOMEOSTASIS AND SKELETAL MUSCLE PHYSIOLOGY. [Doctoral Dissertation]. Temple University; 2016. Available from: http://digital.library.temple.edu/u?/p245801coll10,391153


Temple University

8. Makarewich, Catherine Anne. MICRODOMAIN BASED CALCIUM INFLUX PATHWAYS THAT REGULATE PATHOLOGICAL CARDIAC HYPERTROPHY AND CONTRACTILITY.

Degree: PhD, 2014, Temple University

Molecular and Cellular Physiology

Pathological cardiac stressors, including persistent hypertension or damage from ischemic heart disease, induce a chronic demand for enhanced contractile performance of… (more)

Subjects/Keywords: Physiology; Molecular biology; Cellular biology;

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APA (6th Edition):

Makarewich, C. A. (2014). MICRODOMAIN BASED CALCIUM INFLUX PATHWAYS THAT REGULATE PATHOLOGICAL CARDIAC HYPERTROPHY AND CONTRACTILITY. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,266828

Chicago Manual of Style (16th Edition):

Makarewich, Catherine Anne. “MICRODOMAIN BASED CALCIUM INFLUX PATHWAYS THAT REGULATE PATHOLOGICAL CARDIAC HYPERTROPHY AND CONTRACTILITY.” 2014. Doctoral Dissertation, Temple University. Accessed July 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,266828.

MLA Handbook (7th Edition):

Makarewich, Catherine Anne. “MICRODOMAIN BASED CALCIUM INFLUX PATHWAYS THAT REGULATE PATHOLOGICAL CARDIAC HYPERTROPHY AND CONTRACTILITY.” 2014. Web. 02 Jul 2020.

Vancouver:

Makarewich CA. MICRODOMAIN BASED CALCIUM INFLUX PATHWAYS THAT REGULATE PATHOLOGICAL CARDIAC HYPERTROPHY AND CONTRACTILITY. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2020 Jul 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,266828.

Council of Science Editors:

Makarewich CA. MICRODOMAIN BASED CALCIUM INFLUX PATHWAYS THAT REGULATE PATHOLOGICAL CARDIAC HYPERTROPHY AND CONTRACTILITY. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,266828


Temple University

9. Li, Yingxin. THE ROLES OF Cav3.1/a1G T-TYPE CALCIUM CHANNEL IN HEART RATE GENERATION, REGULATION AND CARDIAC ARRHYTHMIAS.

Degree: PhD, 2011, Temple University

Physiology

T-type Ca²+ channels (TTCCs) are expressed in cardiac pacemaker cells and conduction system of mammals. However, the roles of TTCCs in heart rate (HR)… (more)

Subjects/Keywords: Physiology; arrhythmia; Cav3.1/a1G; heart rate

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APA (6th Edition):

Li, Y. (2011). THE ROLES OF Cav3.1/a1G T-TYPE CALCIUM CHANNEL IN HEART RATE GENERATION, REGULATION AND CARDIAC ARRHYTHMIAS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,151806

Chicago Manual of Style (16th Edition):

Li, Yingxin. “THE ROLES OF Cav3.1/a1G T-TYPE CALCIUM CHANNEL IN HEART RATE GENERATION, REGULATION AND CARDIAC ARRHYTHMIAS.” 2011. Doctoral Dissertation, Temple University. Accessed July 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,151806.

MLA Handbook (7th Edition):

Li, Yingxin. “THE ROLES OF Cav3.1/a1G T-TYPE CALCIUM CHANNEL IN HEART RATE GENERATION, REGULATION AND CARDIAC ARRHYTHMIAS.” 2011. Web. 02 Jul 2020.

Vancouver:

Li Y. THE ROLES OF Cav3.1/a1G T-TYPE CALCIUM CHANNEL IN HEART RATE GENERATION, REGULATION AND CARDIAC ARRHYTHMIAS. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2020 Jul 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,151806.

Council of Science Editors:

Li Y. THE ROLES OF Cav3.1/a1G T-TYPE CALCIUM CHANNEL IN HEART RATE GENERATION, REGULATION AND CARDIAC ARRHYTHMIAS. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,151806


Temple University

10. Zhang, Hongyu. INCREASING MYOCYTE CONTRACTILITY EXACERBATES CARDIAC INJURY AND PUMP DYSFUNCTION AND ABLATION OF PHOSPHORYLATION.

Degree: PhD, 2010, Temple University

Physiology

Myocardial infarction (MI) leads to heart failure (HF) and premature death. The respective roles of myocyte death and depressed myocyte contractility in the induction… (more)

Subjects/Keywords: Biology, Physiology; cardiac function; cell death; heart failure; myocardial infarction; myocyte contractility; ryanodine receptor

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APA (6th Edition):

Zhang, H. (2010). INCREASING MYOCYTE CONTRACTILITY EXACERBATES CARDIAC INJURY AND PUMP DYSFUNCTION AND ABLATION OF PHOSPHORYLATION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,90859

Chicago Manual of Style (16th Edition):

Zhang, Hongyu. “INCREASING MYOCYTE CONTRACTILITY EXACERBATES CARDIAC INJURY AND PUMP DYSFUNCTION AND ABLATION OF PHOSPHORYLATION.” 2010. Doctoral Dissertation, Temple University. Accessed July 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,90859.

MLA Handbook (7th Edition):

Zhang, Hongyu. “INCREASING MYOCYTE CONTRACTILITY EXACERBATES CARDIAC INJURY AND PUMP DYSFUNCTION AND ABLATION OF PHOSPHORYLATION.” 2010. Web. 02 Jul 2020.

Vancouver:

Zhang H. INCREASING MYOCYTE CONTRACTILITY EXACERBATES CARDIAC INJURY AND PUMP DYSFUNCTION AND ABLATION OF PHOSPHORYLATION. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Jul 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,90859.

Council of Science Editors:

Zhang H. INCREASING MYOCYTE CONTRACTILITY EXACERBATES CARDIAC INJURY AND PUMP DYSFUNCTION AND ABLATION OF PHOSPHORYLATION. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,90859

.