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You searched for +publisher:"Temple University" +contributor:("Kirby, Lynn"). Showing records 1 – 17 of 17 total matches.

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Temple University

1. Xin, Dong. SERUM CYTOKINES INDUCED BY PERFORMANCE OF REPETITIVE TASKS AND THEIR RELATIONSHIP TO SICKNESS RESPONSES.

Degree: PhD, 2013, Temple University

Physical Therapy

Work-related repetitive strain injury (RSI), one of the work-related musculoskeletal disorders, is the most commonly reported occupational illness, yet the pathophysiological mechanisms are… (more)

Subjects/Keywords: Neurosciences; Behavioral sciences;

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APA (6th Edition):

Xin, D. (2013). SERUM CYTOKINES INDUCED BY PERFORMANCE OF REPETITIVE TASKS AND THEIR RELATIONSHIP TO SICKNESS RESPONSES. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,237253

Chicago Manual of Style (16th Edition):

Xin, Dong. “SERUM CYTOKINES INDUCED BY PERFORMANCE OF REPETITIVE TASKS AND THEIR RELATIONSHIP TO SICKNESS RESPONSES.” 2013. Doctoral Dissertation, Temple University. Accessed September 24, 2020. http://digital.library.temple.edu/u?/p245801coll10,237253.

MLA Handbook (7th Edition):

Xin, Dong. “SERUM CYTOKINES INDUCED BY PERFORMANCE OF REPETITIVE TASKS AND THEIR RELATIONSHIP TO SICKNESS RESPONSES.” 2013. Web. 24 Sep 2020.

Vancouver:

Xin D. SERUM CYTOKINES INDUCED BY PERFORMANCE OF REPETITIVE TASKS AND THEIR RELATIONSHIP TO SICKNESS RESPONSES. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2020 Sep 24]. Available from: http://digital.library.temple.edu/u?/p245801coll10,237253.

Council of Science Editors:

Xin D. SERUM CYTOKINES INDUCED BY PERFORMANCE OF REPETITIVE TASKS AND THEIR RELATIONSHIP TO SICKNESS RESPONSES. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,237253


Temple University

2. Giannopoulos, Phillip Fotis. THE ROLE OF 5-LIPOXYGENASE IN THE DEVELOPMENT OF TAU NEUROPATHOLOGY AND BEHAVIORAL PHENOTYPE.

Degree: PhD, 2015, Temple University

Pharmacology

5-Lipoxygenase (5LO) is a lipid-peroxidizing enzyme which inserts molecular oxygen into fatty acids leading to the biosynthesis of leukotrienes. This protein is widely expressed… (more)

Subjects/Keywords: Neurosciences;

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APA (6th Edition):

Giannopoulos, P. F. (2015). THE ROLE OF 5-LIPOXYGENASE IN THE DEVELOPMENT OF TAU NEUROPATHOLOGY AND BEHAVIORAL PHENOTYPE. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,315193

Chicago Manual of Style (16th Edition):

Giannopoulos, Phillip Fotis. “THE ROLE OF 5-LIPOXYGENASE IN THE DEVELOPMENT OF TAU NEUROPATHOLOGY AND BEHAVIORAL PHENOTYPE.” 2015. Doctoral Dissertation, Temple University. Accessed September 24, 2020. http://digital.library.temple.edu/u?/p245801coll10,315193.

MLA Handbook (7th Edition):

Giannopoulos, Phillip Fotis. “THE ROLE OF 5-LIPOXYGENASE IN THE DEVELOPMENT OF TAU NEUROPATHOLOGY AND BEHAVIORAL PHENOTYPE.” 2015. Web. 24 Sep 2020.

Vancouver:

Giannopoulos PF. THE ROLE OF 5-LIPOXYGENASE IN THE DEVELOPMENT OF TAU NEUROPATHOLOGY AND BEHAVIORAL PHENOTYPE. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Sep 24]. Available from: http://digital.library.temple.edu/u?/p245801coll10,315193.

Council of Science Editors:

Giannopoulos PF. THE ROLE OF 5-LIPOXYGENASE IN THE DEVELOPMENT OF TAU NEUROPATHOLOGY AND BEHAVIORAL PHENOTYPE. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,315193


Temple University

3. Lunden, Jason Wesley. The serotonergic dorsal raphe nucleus in opiate dependence and stress-induced relapse.

Degree: PhD, 2013, Temple University

Cell Biology

Opioids are used for the clinical treatment of pain, but can lead to tolerance and addiction. In this project we examined the role… (more)

Subjects/Keywords: Neurosciences; Animal behavior; Cellular biology;

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APA (6th Edition):

Lunden, J. W. (2013). The serotonergic dorsal raphe nucleus in opiate dependence and stress-induced relapse. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,228054

Chicago Manual of Style (16th Edition):

Lunden, Jason Wesley. “The serotonergic dorsal raphe nucleus in opiate dependence and stress-induced relapse.” 2013. Doctoral Dissertation, Temple University. Accessed September 24, 2020. http://digital.library.temple.edu/u?/p245801coll10,228054.

MLA Handbook (7th Edition):

Lunden, Jason Wesley. “The serotonergic dorsal raphe nucleus in opiate dependence and stress-induced relapse.” 2013. Web. 24 Sep 2020.

Vancouver:

Lunden JW. The serotonergic dorsal raphe nucleus in opiate dependence and stress-induced relapse. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2020 Sep 24]. Available from: http://digital.library.temple.edu/u?/p245801coll10,228054.

Council of Science Editors:

Lunden JW. The serotonergic dorsal raphe nucleus in opiate dependence and stress-induced relapse. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,228054


Temple University

4. Marcu, Jahan Phillip. Novel Insights into CB1 Receptor Signaling and the Anabolic Role of Cannabinoid Receptors in Bone.

Degree: PhD, 2013, Temple University

Cell Biology

Activation of the CB1 receptor is modulated by aspartate residue D2.63176 in transmembrane helix (TMH) II. Interestingly, D2.63 does not affect the affinity… (more)

Subjects/Keywords: Molecular biology; Pharmacology; Biochemistry;

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APA (6th Edition):

Marcu, J. P. (2013). Novel Insights into CB1 Receptor Signaling and the Anabolic Role of Cannabinoid Receptors in Bone. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,233543

Chicago Manual of Style (16th Edition):

Marcu, Jahan Phillip. “Novel Insights into CB1 Receptor Signaling and the Anabolic Role of Cannabinoid Receptors in Bone.” 2013. Doctoral Dissertation, Temple University. Accessed September 24, 2020. http://digital.library.temple.edu/u?/p245801coll10,233543.

MLA Handbook (7th Edition):

Marcu, Jahan Phillip. “Novel Insights into CB1 Receptor Signaling and the Anabolic Role of Cannabinoid Receptors in Bone.” 2013. Web. 24 Sep 2020.

Vancouver:

Marcu JP. Novel Insights into CB1 Receptor Signaling and the Anabolic Role of Cannabinoid Receptors in Bone. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2020 Sep 24]. Available from: http://digital.library.temple.edu/u?/p245801coll10,233543.

Council of Science Editors:

Marcu JP. Novel Insights into CB1 Receptor Signaling and the Anabolic Role of Cannabinoid Receptors in Bone. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,233543


Temple University

5. Craige, Caryne. Regulation of Cocaine-induced Behaviors and Anxiety Produced by Cocaine Withdrawal through the Serotonin(2C) Receptor.

Degree: PhD, 2013, Temple University

Pharmacology

Cocaine is a powerfully active psychostimulant which exerts its effects through blockade of dopamine, serotonin and norepinephrine transporters and resultant increases in extracellular levels… (more)

Subjects/Keywords: Neurosciences; Pharmacology;

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APA (6th Edition):

Craige, C. (2013). Regulation of Cocaine-induced Behaviors and Anxiety Produced by Cocaine Withdrawal through the Serotonin(2C) Receptor. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,239587

Chicago Manual of Style (16th Edition):

Craige, Caryne. “Regulation of Cocaine-induced Behaviors and Anxiety Produced by Cocaine Withdrawal through the Serotonin(2C) Receptor.” 2013. Doctoral Dissertation, Temple University. Accessed September 24, 2020. http://digital.library.temple.edu/u?/p245801coll10,239587.

MLA Handbook (7th Edition):

Craige, Caryne. “Regulation of Cocaine-induced Behaviors and Anxiety Produced by Cocaine Withdrawal through the Serotonin(2C) Receptor.” 2013. Web. 24 Sep 2020.

Vancouver:

Craige C. Regulation of Cocaine-induced Behaviors and Anxiety Produced by Cocaine Withdrawal through the Serotonin(2C) Receptor. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2020 Sep 24]. Available from: http://digital.library.temple.edu/u?/p245801coll10,239587.

Council of Science Editors:

Craige C. Regulation of Cocaine-induced Behaviors and Anxiety Produced by Cocaine Withdrawal through the Serotonin(2C) Receptor. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,239587


Temple University

6. Bagashev, Asen. MOLECULAR MECHANISM OF HIV-1 TAT INDUCED NEURONAL DYSFUNCTION.

Degree: PhD, 2014, Temple University

Cell Biology

In the early years of the AIDS epidemic, being infected with the virus that causes the disease was considered a virtual death sentence.… (more)

Subjects/Keywords: Neurosciences; Molecular biology; Virology

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APA (6th Edition):

Bagashev, A. (2014). MOLECULAR MECHANISM OF HIV-1 TAT INDUCED NEURONAL DYSFUNCTION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,255791

Chicago Manual of Style (16th Edition):

Bagashev, Asen. “MOLECULAR MECHANISM OF HIV-1 TAT INDUCED NEURONAL DYSFUNCTION.” 2014. Doctoral Dissertation, Temple University. Accessed September 24, 2020. http://digital.library.temple.edu/u?/p245801coll10,255791.

MLA Handbook (7th Edition):

Bagashev, Asen. “MOLECULAR MECHANISM OF HIV-1 TAT INDUCED NEURONAL DYSFUNCTION.” 2014. Web. 24 Sep 2020.

Vancouver:

Bagashev A. MOLECULAR MECHANISM OF HIV-1 TAT INDUCED NEURONAL DYSFUNCTION. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2020 Sep 24]. Available from: http://digital.library.temple.edu/u?/p245801coll10,255791.

Council of Science Editors:

Bagashev A. MOLECULAR MECHANISM OF HIV-1 TAT INDUCED NEURONAL DYSFUNCTION. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,255791


Temple University

7. Enman, Nicole Marie. Dopamine reward dysfunction and cocaine-seeking in a rat model of PTSD.

Degree: PhD, 2014, Temple University

Pharmacology

Posttraumatic stress disorder (PTSD) co-occurs with substance use disorders at high rates, but the neurobiological basis of this relationship remains largely unknown. Identifying mechanisms… (more)

Subjects/Keywords: Neurosciences; Animal behavior; Pharmacology;

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APA (6th Edition):

Enman, N. M. (2014). Dopamine reward dysfunction and cocaine-seeking in a rat model of PTSD. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,259963

Chicago Manual of Style (16th Edition):

Enman, Nicole Marie. “Dopamine reward dysfunction and cocaine-seeking in a rat model of PTSD.” 2014. Doctoral Dissertation, Temple University. Accessed September 24, 2020. http://digital.library.temple.edu/u?/p245801coll10,259963.

MLA Handbook (7th Edition):

Enman, Nicole Marie. “Dopamine reward dysfunction and cocaine-seeking in a rat model of PTSD.” 2014. Web. 24 Sep 2020.

Vancouver:

Enman NM. Dopamine reward dysfunction and cocaine-seeking in a rat model of PTSD. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2020 Sep 24]. Available from: http://digital.library.temple.edu/u?/p245801coll10,259963.

Council of Science Editors:

Enman NM. Dopamine reward dysfunction and cocaine-seeking in a rat model of PTSD. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,259963


Temple University

8. Joshi, Yash. The Role of 5-Lipoxygenase in the Stress-Mediated Exacerbation of the Alzheimer's Disease Phenotype.

Degree: PhD, 2015, Temple University

Pharmacology

BACKGROUND: Alzheimer's disease (AD) is the most common aging-associated neurodegenerative dementia. Current epidemiological trends indicate that a rapidly aging population, in conjunction with the… (more)

Subjects/Keywords: Neurosciences; Biology; Biochemistry;

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APA (6th Edition):

Joshi, Y. (2015). The Role of 5-Lipoxygenase in the Stress-Mediated Exacerbation of the Alzheimer's Disease Phenotype. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,318405

Chicago Manual of Style (16th Edition):

Joshi, Yash. “The Role of 5-Lipoxygenase in the Stress-Mediated Exacerbation of the Alzheimer's Disease Phenotype.” 2015. Doctoral Dissertation, Temple University. Accessed September 24, 2020. http://digital.library.temple.edu/u?/p245801coll10,318405.

MLA Handbook (7th Edition):

Joshi, Yash. “The Role of 5-Lipoxygenase in the Stress-Mediated Exacerbation of the Alzheimer's Disease Phenotype.” 2015. Web. 24 Sep 2020.

Vancouver:

Joshi Y. The Role of 5-Lipoxygenase in the Stress-Mediated Exacerbation of the Alzheimer's Disease Phenotype. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Sep 24]. Available from: http://digital.library.temple.edu/u?/p245801coll10,318405.

Council of Science Editors:

Joshi Y. The Role of 5-Lipoxygenase in the Stress-Mediated Exacerbation of the Alzheimer's Disease Phenotype. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,318405


Temple University

9. Frara, Nagat. THE ROLE OF OSTEOACTIVIN IN MUSCULOSKELETAL TISSUES AS A REPAIR AND ANABOLIC FACTOR.

Degree: PhD, 2015, Temple University

Cell Biology

Osteoactivin (OA) is a novel osteogenic and repair factor. It has the ability to regulate cell proliferation, adhesion, differentiation, and synthesis and regulation… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Frara, N. (2015). THE ROLE OF OSTEOACTIVIN IN MUSCULOSKELETAL TISSUES AS A REPAIR AND ANABOLIC FACTOR. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,336629

Chicago Manual of Style (16th Edition):

Frara, Nagat. “THE ROLE OF OSTEOACTIVIN IN MUSCULOSKELETAL TISSUES AS A REPAIR AND ANABOLIC FACTOR.” 2015. Doctoral Dissertation, Temple University. Accessed September 24, 2020. http://digital.library.temple.edu/u?/p245801coll10,336629.

MLA Handbook (7th Edition):

Frara, Nagat. “THE ROLE OF OSTEOACTIVIN IN MUSCULOSKELETAL TISSUES AS A REPAIR AND ANABOLIC FACTOR.” 2015. Web. 24 Sep 2020.

Vancouver:

Frara N. THE ROLE OF OSTEOACTIVIN IN MUSCULOSKELETAL TISSUES AS A REPAIR AND ANABOLIC FACTOR. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Sep 24]. Available from: http://digital.library.temple.edu/u?/p245801coll10,336629.

Council of Science Editors:

Frara N. THE ROLE OF OSTEOACTIVIN IN MUSCULOSKELETAL TISSUES AS A REPAIR AND ANABOLIC FACTOR. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,336629


Temple University

10. Gregg, Ryan Alexander. INVESTIGATIONS INTO THE STEREOCHEMICAL AND GLUTAMATERGIC MECHANISMS OF THE "BATH SALTS" SYNTHETIC CATHINONES MEPHEDRONE AND MDPV IN RATS.

Degree: PhD, 2015, Temple University

Pharmacology

Synthetic cathinones, commonly referred to as “bath salts”, are a subgroup of novel psychoactive substances that have seen a dramatic rise in abuse worldwide… (more)

Subjects/Keywords: Pharmacology; Neurosciences; Medicine;

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APA (6th Edition):

Gregg, R. A. (2015). INVESTIGATIONS INTO THE STEREOCHEMICAL AND GLUTAMATERGIC MECHANISMS OF THE "BATH SALTS" SYNTHETIC CATHINONES MEPHEDRONE AND MDPV IN RATS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,354278

Chicago Manual of Style (16th Edition):

Gregg, Ryan Alexander. “INVESTIGATIONS INTO THE STEREOCHEMICAL AND GLUTAMATERGIC MECHANISMS OF THE "BATH SALTS" SYNTHETIC CATHINONES MEPHEDRONE AND MDPV IN RATS.” 2015. Doctoral Dissertation, Temple University. Accessed September 24, 2020. http://digital.library.temple.edu/u?/p245801coll10,354278.

MLA Handbook (7th Edition):

Gregg, Ryan Alexander. “INVESTIGATIONS INTO THE STEREOCHEMICAL AND GLUTAMATERGIC MECHANISMS OF THE "BATH SALTS" SYNTHETIC CATHINONES MEPHEDRONE AND MDPV IN RATS.” 2015. Web. 24 Sep 2020.

Vancouver:

Gregg RA. INVESTIGATIONS INTO THE STEREOCHEMICAL AND GLUTAMATERGIC MECHANISMS OF THE "BATH SALTS" SYNTHETIC CATHINONES MEPHEDRONE AND MDPV IN RATS. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Sep 24]. Available from: http://digital.library.temple.edu/u?/p245801coll10,354278.

Council of Science Editors:

Gregg RA. INVESTIGATIONS INTO THE STEREOCHEMICAL AND GLUTAMATERGIC MECHANISMS OF THE "BATH SALTS" SYNTHETIC CATHINONES MEPHEDRONE AND MDPV IN RATS. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,354278


Temple University

11. Heinisch, Silke. Chemokine interactions with the serotonin and opioid systems: anatomical and electrophysiological studies in the rat brain.

Degree: PhD, 2008, Temple University

Anatomy

Chemokines, immune proteins that induce chemotaxis and adhesion, and their G-protein coupled receptors distribute throughout the central nervous system (CNS), regulate neuronal patterning, and… (more)

Subjects/Keywords: Biology, Anatomy; Biology, Neuroscience; Biology, Cell; Chemokines; Serotonin; Mu-opioid receptor; Co-localization; Electrophysiology; Heterologous desensitization

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APA (6th Edition):

Heinisch, S. (2008). Chemokine interactions with the serotonin and opioid systems: anatomical and electrophysiological studies in the rat brain. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,9181

Chicago Manual of Style (16th Edition):

Heinisch, Silke. “Chemokine interactions with the serotonin and opioid systems: anatomical and electrophysiological studies in the rat brain.” 2008. Doctoral Dissertation, Temple University. Accessed September 24, 2020. http://digital.library.temple.edu/u?/p245801coll10,9181.

MLA Handbook (7th Edition):

Heinisch, Silke. “Chemokine interactions with the serotonin and opioid systems: anatomical and electrophysiological studies in the rat brain.” 2008. Web. 24 Sep 2020.

Vancouver:

Heinisch S. Chemokine interactions with the serotonin and opioid systems: anatomical and electrophysiological studies in the rat brain. [Internet] [Doctoral dissertation]. Temple University; 2008. [cited 2020 Sep 24]. Available from: http://digital.library.temple.edu/u?/p245801coll10,9181.

Council of Science Editors:

Heinisch S. Chemokine interactions with the serotonin and opioid systems: anatomical and electrophysiological studies in the rat brain. [Doctoral Dissertation]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,9181


Temple University

12. Miller, Jonathan S. GSK3: A Neuromodulator of Cocaine-Induced Behavioral Responses.

Degree: PhD, 2009, Temple University

Pharmacology

Cocaine is a highly abused psychostimulant with repeated use potential culminating in addiction, a disease associated with compulsive drug seeking, use and high rates… (more)

Subjects/Keywords: Health Sciences, Pharmacology; Brain; Cocaine; Dopamine; Glutamate; Glycogen Synthase Kinase-3; Mouse

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APA (6th Edition):

Miller, J. S. (2009). GSK3: A Neuromodulator of Cocaine-Induced Behavioral Responses. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,40192

Chicago Manual of Style (16th Edition):

Miller, Jonathan S. “GSK3: A Neuromodulator of Cocaine-Induced Behavioral Responses.” 2009. Doctoral Dissertation, Temple University. Accessed September 24, 2020. http://digital.library.temple.edu/u?/p245801coll10,40192.

MLA Handbook (7th Edition):

Miller, Jonathan S. “GSK3: A Neuromodulator of Cocaine-Induced Behavioral Responses.” 2009. Web. 24 Sep 2020.

Vancouver:

Miller JS. GSK3: A Neuromodulator of Cocaine-Induced Behavioral Responses. [Internet] [Doctoral dissertation]. Temple University; 2009. [cited 2020 Sep 24]. Available from: http://digital.library.temple.edu/u?/p245801coll10,40192.

Council of Science Editors:

Miller JS. GSK3: A Neuromodulator of Cocaine-Induced Behavioral Responses. [Doctoral Dissertation]. Temple University; 2009. Available from: http://digital.library.temple.edu/u?/p245801coll10,40192


Temple University

13. Khalid, Helene. S-Mephedrone: preclinical investigation of a synthetic cathinone against behavioral and neurochemical effects of cocaine and MDPV.

Degree: PhD, 2017, Temple University

Pharmacology

Synthetic cathinones are an emerging class of novel psychoactive substances whose rising rates of abuse have made them a significant public health issue. Recently,… (more)

Subjects/Keywords: Pharmacology; Neurosciences; Behavioral sciences;

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APA (6th Edition):

Khalid, H. (2017). S-Mephedrone: preclinical investigation of a synthetic cathinone against behavioral and neurochemical effects of cocaine and MDPV. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,434922

Chicago Manual of Style (16th Edition):

Khalid, Helene. “S-Mephedrone: preclinical investigation of a synthetic cathinone against behavioral and neurochemical effects of cocaine and MDPV.” 2017. Doctoral Dissertation, Temple University. Accessed September 24, 2020. http://digital.library.temple.edu/u?/p245801coll10,434922.

MLA Handbook (7th Edition):

Khalid, Helene. “S-Mephedrone: preclinical investigation of a synthetic cathinone against behavioral and neurochemical effects of cocaine and MDPV.” 2017. Web. 24 Sep 2020.

Vancouver:

Khalid H. S-Mephedrone: preclinical investigation of a synthetic cathinone against behavioral and neurochemical effects of cocaine and MDPV. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2020 Sep 24]. Available from: http://digital.library.temple.edu/u?/p245801coll10,434922.

Council of Science Editors:

Khalid H. S-Mephedrone: preclinical investigation of a synthetic cathinone against behavioral and neurochemical effects of cocaine and MDPV. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,434922


Temple University

14. Simmons, Steven James. HYPOCRETIN/OREXIN AND THE VENTRAL MIDBRAIN: TOPOGRAPHY AND FUNCTION ASSOCIATED WITH PSYCHOSTIMULANT-TAKING AND AFFECT.

Degree: PhD, 2018, Temple University

Biomedical Sciences

Abuse of psychostimulants including cocaine and new synthetic formulations remains an international public health problem and economic burden. Addiction develops consequential to positive… (more)

Subjects/Keywords: Neurosciences; Pharmacology; Behavioral sciences;

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APA (6th Edition):

Simmons, S. J. (2018). HYPOCRETIN/OREXIN AND THE VENTRAL MIDBRAIN: TOPOGRAPHY AND FUNCTION ASSOCIATED WITH PSYCHOSTIMULANT-TAKING AND AFFECT. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,507835

Chicago Manual of Style (16th Edition):

Simmons, Steven James. “HYPOCRETIN/OREXIN AND THE VENTRAL MIDBRAIN: TOPOGRAPHY AND FUNCTION ASSOCIATED WITH PSYCHOSTIMULANT-TAKING AND AFFECT.” 2018. Doctoral Dissertation, Temple University. Accessed September 24, 2020. http://digital.library.temple.edu/u?/p245801coll10,507835.

MLA Handbook (7th Edition):

Simmons, Steven James. “HYPOCRETIN/OREXIN AND THE VENTRAL MIDBRAIN: TOPOGRAPHY AND FUNCTION ASSOCIATED WITH PSYCHOSTIMULANT-TAKING AND AFFECT.” 2018. Web. 24 Sep 2020.

Vancouver:

Simmons SJ. HYPOCRETIN/OREXIN AND THE VENTRAL MIDBRAIN: TOPOGRAPHY AND FUNCTION ASSOCIATED WITH PSYCHOSTIMULANT-TAKING AND AFFECT. [Internet] [Doctoral dissertation]. Temple University; 2018. [cited 2020 Sep 24]. Available from: http://digital.library.temple.edu/u?/p245801coll10,507835.

Council of Science Editors:

Simmons SJ. HYPOCRETIN/OREXIN AND THE VENTRAL MIDBRAIN: TOPOGRAPHY AND FUNCTION ASSOCIATED WITH PSYCHOSTIMULANT-TAKING AND AFFECT. [Doctoral Dissertation]. Temple University; 2018. Available from: http://digital.library.temple.edu/u?/p245801coll10,507835


Temple University

15. Shrestha, Jenny. HIV-1 gp120 Mediated Neuronal Deregulation: Unraveling the Molecular Mechanisms Involved.

Degree: PhD, 2016, Temple University

Molecular Biology and Genetics

The advancement in combinatory antiretroviral therapy (cART) has granted people with HIV-1 an improved lifespan by decreasing the likelihood of AIDS-defining… (more)

Subjects/Keywords: Molecular biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shrestha, J. (2016). HIV-1 gp120 Mediated Neuronal Deregulation: Unraveling the Molecular Mechanisms Involved. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,402666

Chicago Manual of Style (16th Edition):

Shrestha, Jenny. “HIV-1 gp120 Mediated Neuronal Deregulation: Unraveling the Molecular Mechanisms Involved.” 2016. Doctoral Dissertation, Temple University. Accessed September 24, 2020. http://digital.library.temple.edu/u?/p245801coll10,402666.

MLA Handbook (7th Edition):

Shrestha, Jenny. “HIV-1 gp120 Mediated Neuronal Deregulation: Unraveling the Molecular Mechanisms Involved.” 2016. Web. 24 Sep 2020.

Vancouver:

Shrestha J. HIV-1 gp120 Mediated Neuronal Deregulation: Unraveling the Molecular Mechanisms Involved. [Internet] [Doctoral dissertation]. Temple University; 2016. [cited 2020 Sep 24]. Available from: http://digital.library.temple.edu/u?/p245801coll10,402666.

Council of Science Editors:

Shrestha J. HIV-1 gp120 Mediated Neuronal Deregulation: Unraveling the Molecular Mechanisms Involved. [Doctoral Dissertation]. Temple University; 2016. Available from: http://digital.library.temple.edu/u?/p245801coll10,402666


Temple University

16. Soderman, Avery Rune. The Role of Mu Opioid Receptors in the Behavioral Effects of Cocaine.

Degree: PhD, 2008, Temple University

Pharmacology

Animal models have proven to be useful tools for modeling human neurochemical and behavioral responses to drugs of abuse, including cocaine. Cocaine is a… (more)

Subjects/Keywords: Health Sciences, Pharmacology; Biology, Neuroscience; cocaine; hyperlocomotion; Mu-opioid r; Nucleus Accumbens; reward

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Soderman, A. R. (2008). The Role of Mu Opioid Receptors in the Behavioral Effects of Cocaine. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,22825

Chicago Manual of Style (16th Edition):

Soderman, Avery Rune. “The Role of Mu Opioid Receptors in the Behavioral Effects of Cocaine.” 2008. Doctoral Dissertation, Temple University. Accessed September 24, 2020. http://digital.library.temple.edu/u?/p245801coll10,22825.

MLA Handbook (7th Edition):

Soderman, Avery Rune. “The Role of Mu Opioid Receptors in the Behavioral Effects of Cocaine.” 2008. Web. 24 Sep 2020.

Vancouver:

Soderman AR. The Role of Mu Opioid Receptors in the Behavioral Effects of Cocaine. [Internet] [Doctoral dissertation]. Temple University; 2008. [cited 2020 Sep 24]. Available from: http://digital.library.temple.edu/u?/p245801coll10,22825.

Council of Science Editors:

Soderman AR. The Role of Mu Opioid Receptors in the Behavioral Effects of Cocaine. [Doctoral Dissertation]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,22825


Temple University

17. Trecki, Jordan. THE MODULATION OF THE MESOLIMBIC AND NIGROSTRIATAL DOPAMINE PATHWAYS BY CXCL12 AND CXCR4.

Degree: PhD, 2009, Temple University

Pharmacology

The role of chemokines in immune function is clearly established. Recent evidence suggests that these molecules also play an important role in the CNS… (more)

Subjects/Keywords: Health Sciences, Pharmacology; Biology, Neuroscience; behavior; cocaine; immunohistochemistry; intracerebral; locomotion; microdialysis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Trecki, J. (2009). THE MODULATION OF THE MESOLIMBIC AND NIGROSTRIATAL DOPAMINE PATHWAYS BY CXCL12 AND CXCR4. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,57720

Chicago Manual of Style (16th Edition):

Trecki, Jordan. “THE MODULATION OF THE MESOLIMBIC AND NIGROSTRIATAL DOPAMINE PATHWAYS BY CXCL12 AND CXCR4.” 2009. Doctoral Dissertation, Temple University. Accessed September 24, 2020. http://digital.library.temple.edu/u?/p245801coll10,57720.

MLA Handbook (7th Edition):

Trecki, Jordan. “THE MODULATION OF THE MESOLIMBIC AND NIGROSTRIATAL DOPAMINE PATHWAYS BY CXCL12 AND CXCR4.” 2009. Web. 24 Sep 2020.

Vancouver:

Trecki J. THE MODULATION OF THE MESOLIMBIC AND NIGROSTRIATAL DOPAMINE PATHWAYS BY CXCL12 AND CXCR4. [Internet] [Doctoral dissertation]. Temple University; 2009. [cited 2020 Sep 24]. Available from: http://digital.library.temple.edu/u?/p245801coll10,57720.

Council of Science Editors:

Trecki J. THE MODULATION OF THE MESOLIMBIC AND NIGROSTRIATAL DOPAMINE PATHWAYS BY CXCL12 AND CXCR4. [Doctoral Dissertation]. Temple University; 2009. Available from: http://digital.library.temple.edu/u?/p245801coll10,57720

.