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You searched for +publisher:"Temple University" +contributor:("Kilpatrick, Laurie"). Showing records 1 – 12 of 12 total matches.

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Temple University

1. Hooshdaran, Bahman. DUAL INHIBITION OF CATHEPSIN G AND CHYMASE AFTER ISCHEMIA REPERFUSION: THE ROLE OF INFLAMMATORY SERINE PROTEASES IN ISCHEMIA REPERFUSION INJURY.

Degree: PhD, 2017, Temple University

Bioengineering

Acute myocardial infarction (AMI) is a leading cause of morbidity and mortality in the world (4). Restoration of coronary flow to the ischemic myocardium… (more)

Subjects/Keywords: Engineering; Bioengineering;

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APA (6th Edition):

Hooshdaran, B. (2017). DUAL INHIBITION OF CATHEPSIN G AND CHYMASE AFTER ISCHEMIA REPERFUSION: THE ROLE OF INFLAMMATORY SERINE PROTEASES IN ISCHEMIA REPERFUSION INJURY. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,475423

Chicago Manual of Style (16th Edition):

Hooshdaran, Bahman. “DUAL INHIBITION OF CATHEPSIN G AND CHYMASE AFTER ISCHEMIA REPERFUSION: THE ROLE OF INFLAMMATORY SERINE PROTEASES IN ISCHEMIA REPERFUSION INJURY.” 2017. Doctoral Dissertation, Temple University. Accessed September 18, 2019. http://digital.library.temple.edu/u?/p245801coll10,475423.

MLA Handbook (7th Edition):

Hooshdaran, Bahman. “DUAL INHIBITION OF CATHEPSIN G AND CHYMASE AFTER ISCHEMIA REPERFUSION: THE ROLE OF INFLAMMATORY SERINE PROTEASES IN ISCHEMIA REPERFUSION INJURY.” 2017. Web. 18 Sep 2019.

Vancouver:

Hooshdaran B. DUAL INHIBITION OF CATHEPSIN G AND CHYMASE AFTER ISCHEMIA REPERFUSION: THE ROLE OF INFLAMMATORY SERINE PROTEASES IN ISCHEMIA REPERFUSION INJURY. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2019 Sep 18]. Available from: http://digital.library.temple.edu/u?/p245801coll10,475423.

Council of Science Editors:

Hooshdaran B. DUAL INHIBITION OF CATHEPSIN G AND CHYMASE AFTER ISCHEMIA REPERFUSION: THE ROLE OF INFLAMMATORY SERINE PROTEASES IN ISCHEMIA REPERFUSION INJURY. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,475423


Temple University

2. Bhavanasi, Dheeraj. Role of Protein Kinase C delta in regulating platelet functional responses.

Degree: PhD, 2014, Temple University

Physiology

Platelets upon activation change their shape, aggregate and secrete alpha and dense granule contents among which ADP acts as a feedback activator. Different Protein… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Bhavanasi, D. (2014). Role of Protein Kinase C delta in regulating platelet functional responses. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,283099

Chicago Manual of Style (16th Edition):

Bhavanasi, Dheeraj. “Role of Protein Kinase C delta in regulating platelet functional responses.” 2014. Doctoral Dissertation, Temple University. Accessed September 18, 2019. http://digital.library.temple.edu/u?/p245801coll10,283099.

MLA Handbook (7th Edition):

Bhavanasi, Dheeraj. “Role of Protein Kinase C delta in regulating platelet functional responses.” 2014. Web. 18 Sep 2019.

Vancouver:

Bhavanasi D. Role of Protein Kinase C delta in regulating platelet functional responses. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Sep 18]. Available from: http://digital.library.temple.edu/u?/p245801coll10,283099.

Council of Science Editors:

Bhavanasi D. Role of Protein Kinase C delta in regulating platelet functional responses. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,283099


Temple University

3. Ray, Mitali. Genetic Deletion of Interleukin-19 Exacerbates Atherogenesis in Double Knockout Mice by Modulation of mRNA Stability Protein HuR.

Degree: PhD, 2018, Temple University

Biomedical Sciences

Objective: To test the hypothesis that loss of IL-19 exacerbates atherosclerosis. Approach and Results: Il19-/- mice were crossed into Ldlr-/- mice. Double knockout… (more)

Subjects/Keywords: Biology;

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APA (6th Edition):

Ray, M. (2018). Genetic Deletion of Interleukin-19 Exacerbates Atherogenesis in Double Knockout Mice by Modulation of mRNA Stability Protein HuR. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,491387

Chicago Manual of Style (16th Edition):

Ray, Mitali. “Genetic Deletion of Interleukin-19 Exacerbates Atherogenesis in Double Knockout Mice by Modulation of mRNA Stability Protein HuR.” 2018. Doctoral Dissertation, Temple University. Accessed September 18, 2019. http://digital.library.temple.edu/u?/p245801coll10,491387.

MLA Handbook (7th Edition):

Ray, Mitali. “Genetic Deletion of Interleukin-19 Exacerbates Atherogenesis in Double Knockout Mice by Modulation of mRNA Stability Protein HuR.” 2018. Web. 18 Sep 2019.

Vancouver:

Ray M. Genetic Deletion of Interleukin-19 Exacerbates Atherogenesis in Double Knockout Mice by Modulation of mRNA Stability Protein HuR. [Internet] [Doctoral dissertation]. Temple University; 2018. [cited 2019 Sep 18]. Available from: http://digital.library.temple.edu/u?/p245801coll10,491387.

Council of Science Editors:

Ray M. Genetic Deletion of Interleukin-19 Exacerbates Atherogenesis in Double Knockout Mice by Modulation of mRNA Stability Protein HuR. [Doctoral Dissertation]. Temple University; 2018. Available from: http://digital.library.temple.edu/u?/p245801coll10,491387


Temple University

4. Soroush, Fariborz. A Novel Microfluidic System for Screening Anti-inflammatory Therapeutics.

Degree: PhD, 2018, Temple University

Mechanical Engineering

Inflammation is a crucial physiological protective response of body to infection or injury. However, in pathological conditions such as sepsis or radiation damage,… (more)

Subjects/Keywords: Bioengineering; Fluid mechanics; Immunology

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APA (6th Edition):

Soroush, F. (2018). A Novel Microfluidic System for Screening Anti-inflammatory Therapeutics. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,500666

Chicago Manual of Style (16th Edition):

Soroush, Fariborz. “A Novel Microfluidic System for Screening Anti-inflammatory Therapeutics.” 2018. Doctoral Dissertation, Temple University. Accessed September 18, 2019. http://digital.library.temple.edu/u?/p245801coll10,500666.

MLA Handbook (7th Edition):

Soroush, Fariborz. “A Novel Microfluidic System for Screening Anti-inflammatory Therapeutics.” 2018. Web. 18 Sep 2019.

Vancouver:

Soroush F. A Novel Microfluidic System for Screening Anti-inflammatory Therapeutics. [Internet] [Doctoral dissertation]. Temple University; 2018. [cited 2019 Sep 18]. Available from: http://digital.library.temple.edu/u?/p245801coll10,500666.

Council of Science Editors:

Soroush F. A Novel Microfluidic System for Screening Anti-inflammatory Therapeutics. [Doctoral Dissertation]. Temple University; 2018. Available from: http://digital.library.temple.edu/u?/p245801coll10,500666


Temple University

5. Adhikary, Sabina. CANNABINOID RECEPTOR 2 AGONIST REDUCES IMMUNE CELL MIGRATION IN NEUROINFLAMMATION VIA INHIBITION OF MATRIX METALLOPROTEINASE-9.

Degree: PhD, 2013, Temple University

Physiology

Several studies have reported that administration of cannabinoid receptor agonists in inflammatory/autoimmune and CNS injury models resulted in significant attenuation of clinical disease. The… (more)

Subjects/Keywords: Immunology; Cannabinoid Receptor 2; Chemokines; Dendritic cell migration; Matrix Metalloproteinase 9; Multiple sclerosis; Spinal cord injury

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APA (6th Edition):

Adhikary, S. (2013). CANNABINOID RECEPTOR 2 AGONIST REDUCES IMMUNE CELL MIGRATION IN NEUROINFLAMMATION VIA INHIBITION OF MATRIX METALLOPROTEINASE-9. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,214799

Chicago Manual of Style (16th Edition):

Adhikary, Sabina. “CANNABINOID RECEPTOR 2 AGONIST REDUCES IMMUNE CELL MIGRATION IN NEUROINFLAMMATION VIA INHIBITION OF MATRIX METALLOPROTEINASE-9.” 2013. Doctoral Dissertation, Temple University. Accessed September 18, 2019. http://digital.library.temple.edu/u?/p245801coll10,214799.

MLA Handbook (7th Edition):

Adhikary, Sabina. “CANNABINOID RECEPTOR 2 AGONIST REDUCES IMMUNE CELL MIGRATION IN NEUROINFLAMMATION VIA INHIBITION OF MATRIX METALLOPROTEINASE-9.” 2013. Web. 18 Sep 2019.

Vancouver:

Adhikary S. CANNABINOID RECEPTOR 2 AGONIST REDUCES IMMUNE CELL MIGRATION IN NEUROINFLAMMATION VIA INHIBITION OF MATRIX METALLOPROTEINASE-9. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2019 Sep 18]. Available from: http://digital.library.temple.edu/u?/p245801coll10,214799.

Council of Science Editors:

Adhikary S. CANNABINOID RECEPTOR 2 AGONIST REDUCES IMMUNE CELL MIGRATION IN NEUROINFLAMMATION VIA INHIBITION OF MATRIX METALLOPROTEINASE-9. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,214799


Temple University

6. Reichenbach, Zachary Wilmer. Modulation of the Endogenous Cannabinoid System to Attenuate Inflammation in Central Nervous System Injury.

Degree: PhD, 2015, Temple University

Physiology

In non-pathological states the central nervous system maintains a degree of immunological privilege. When illness or injury occur, this privilege can be lost and… (more)

Subjects/Keywords: Neurosciences; Immunology; Pharmacology;

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APA (6th Edition):

Reichenbach, Z. W. (2015). Modulation of the Endogenous Cannabinoid System to Attenuate Inflammation in Central Nervous System Injury. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,253514

Chicago Manual of Style (16th Edition):

Reichenbach, Zachary Wilmer. “Modulation of the Endogenous Cannabinoid System to Attenuate Inflammation in Central Nervous System Injury.” 2015. Doctoral Dissertation, Temple University. Accessed September 18, 2019. http://digital.library.temple.edu/u?/p245801coll10,253514.

MLA Handbook (7th Edition):

Reichenbach, Zachary Wilmer. “Modulation of the Endogenous Cannabinoid System to Attenuate Inflammation in Central Nervous System Injury.” 2015. Web. 18 Sep 2019.

Vancouver:

Reichenbach ZW. Modulation of the Endogenous Cannabinoid System to Attenuate Inflammation in Central Nervous System Injury. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Sep 18]. Available from: http://digital.library.temple.edu/u?/p245801coll10,253514.

Council of Science Editors:

Reichenbach ZW. Modulation of the Endogenous Cannabinoid System to Attenuate Inflammation in Central Nervous System Injury. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,253514


Temple University

7. Heayn, Michelle Diane. The Role of Caveolae in PECAM-1 Mechanotransduction.

Degree: PhD, 2014, Temple University

Physiology

Altered fluid flow, which is found in branches and curvatures of arteries, results in abnormal forces on the endothelial cells (EC). These forces have… (more)

Subjects/Keywords: Cellular biology; Molecular biology; Biomechanics;

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APA (6th Edition):

Heayn, M. D. (2014). The Role of Caveolae in PECAM-1 Mechanotransduction. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,263778

Chicago Manual of Style (16th Edition):

Heayn, Michelle Diane. “The Role of Caveolae in PECAM-1 Mechanotransduction.” 2014. Doctoral Dissertation, Temple University. Accessed September 18, 2019. http://digital.library.temple.edu/u?/p245801coll10,263778.

MLA Handbook (7th Edition):

Heayn, Michelle Diane. “The Role of Caveolae in PECAM-1 Mechanotransduction.” 2014. Web. 18 Sep 2019.

Vancouver:

Heayn MD. The Role of Caveolae in PECAM-1 Mechanotransduction. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Sep 18]. Available from: http://digital.library.temple.edu/u?/p245801coll10,263778.

Council of Science Editors:

Heayn MD. The Role of Caveolae in PECAM-1 Mechanotransduction. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,263778


Temple University

8. Richards, Jamie Madison. The Potential of IL-19 As a Therapeutic Anti-inflammatory and Angiogenic Cytokine.

Degree: PhD, 2015, Temple University

Physiology

Our lab has recently shown that IL-19 is expressed in angiogenic ECs, opening the possibility for its use as a medicine to increase perfusion… (more)

Subjects/Keywords: Physiology; Health sciences; Medicine;

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APA (6th Edition):

Richards, J. M. (2015). The Potential of IL-19 As a Therapeutic Anti-inflammatory and Angiogenic Cytokine. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,345114

Chicago Manual of Style (16th Edition):

Richards, Jamie Madison. “The Potential of IL-19 As a Therapeutic Anti-inflammatory and Angiogenic Cytokine.” 2015. Doctoral Dissertation, Temple University. Accessed September 18, 2019. http://digital.library.temple.edu/u?/p245801coll10,345114.

MLA Handbook (7th Edition):

Richards, Jamie Madison. “The Potential of IL-19 As a Therapeutic Anti-inflammatory and Angiogenic Cytokine.” 2015. Web. 18 Sep 2019.

Vancouver:

Richards JM. The Potential of IL-19 As a Therapeutic Anti-inflammatory and Angiogenic Cytokine. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Sep 18]. Available from: http://digital.library.temple.edu/u?/p245801coll10,345114.

Council of Science Editors:

Richards JM. The Potential of IL-19 As a Therapeutic Anti-inflammatory and Angiogenic Cytokine. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,345114


Temple University

9. Preston, Kyle J. Macronutrient Activation of Endothelium Dependent Leukocyte Trafficking: Metabolic Implications.

Degree: PhD, 2015, Temple University

Physiology

Obesity and insulin resistance are characterized by elevated pro-inflammatory proteins in the blood and immune cell accumulation in the visceral adipose tissue. Resident leukocytes… (more)

Subjects/Keywords: Physiology;

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APA (6th Edition):

Preston, K. J. (2015). Macronutrient Activation of Endothelium Dependent Leukocyte Trafficking: Metabolic Implications. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,361365

Chicago Manual of Style (16th Edition):

Preston, Kyle J. “Macronutrient Activation of Endothelium Dependent Leukocyte Trafficking: Metabolic Implications.” 2015. Doctoral Dissertation, Temple University. Accessed September 18, 2019. http://digital.library.temple.edu/u?/p245801coll10,361365.

MLA Handbook (7th Edition):

Preston, Kyle J. “Macronutrient Activation of Endothelium Dependent Leukocyte Trafficking: Metabolic Implications.” 2015. Web. 18 Sep 2019.

Vancouver:

Preston KJ. Macronutrient Activation of Endothelium Dependent Leukocyte Trafficking: Metabolic Implications. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Sep 18]. Available from: http://digital.library.temple.edu/u?/p245801coll10,361365.

Council of Science Editors:

Preston KJ. Macronutrient Activation of Endothelium Dependent Leukocyte Trafficking: Metabolic Implications. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,361365


Temple University

10. Inamdar, Vaishali Vijay. FUNCTIONAL PROTEIN TYROSINE PHOSPHATASES IN PLATELETS.

Degree: PhD, 2017, Temple University

Biomedical Sciences

Platelets are small anucleate cells in blood that are derived from megakaryocytes and their primary function is to prevent bleeding. Upon vascular injury,… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Inamdar, V. V. (2017). FUNCTIONAL PROTEIN TYROSINE PHOSPHATASES IN PLATELETS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,473257

Chicago Manual of Style (16th Edition):

Inamdar, Vaishali Vijay. “FUNCTIONAL PROTEIN TYROSINE PHOSPHATASES IN PLATELETS.” 2017. Doctoral Dissertation, Temple University. Accessed September 18, 2019. http://digital.library.temple.edu/u?/p245801coll10,473257.

MLA Handbook (7th Edition):

Inamdar, Vaishali Vijay. “FUNCTIONAL PROTEIN TYROSINE PHOSPHATASES IN PLATELETS.” 2017. Web. 18 Sep 2019.

Vancouver:

Inamdar VV. FUNCTIONAL PROTEIN TYROSINE PHOSPHATASES IN PLATELETS. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2019 Sep 18]. Available from: http://digital.library.temple.edu/u?/p245801coll10,473257.

Council of Science Editors:

Inamdar VV. FUNCTIONAL PROTEIN TYROSINE PHOSPHATASES IN PLATELETS. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,473257


Temple University

11. Patel, Akruti. NOVEL REGULATORS OF GPVI-MEDIATED PLATELET ACTIVATION.

Degree: PhD, 2017, Temple University

Biomedical Sciences

Platelets are anucleate cells that are crucial mediators of hemostasis and thrombosis. Under physiological conditions, platelets are maintained in a quiescent state within… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Patel, A. (2017). NOVEL REGULATORS OF GPVI-MEDIATED PLATELET ACTIVATION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,473397

Chicago Manual of Style (16th Edition):

Patel, Akruti. “NOVEL REGULATORS OF GPVI-MEDIATED PLATELET ACTIVATION.” 2017. Doctoral Dissertation, Temple University. Accessed September 18, 2019. http://digital.library.temple.edu/u?/p245801coll10,473397.

MLA Handbook (7th Edition):

Patel, Akruti. “NOVEL REGULATORS OF GPVI-MEDIATED PLATELET ACTIVATION.” 2017. Web. 18 Sep 2019.

Vancouver:

Patel A. NOVEL REGULATORS OF GPVI-MEDIATED PLATELET ACTIVATION. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2019 Sep 18]. Available from: http://digital.library.temple.edu/u?/p245801coll10,473397.

Council of Science Editors:

Patel A. NOVEL REGULATORS OF GPVI-MEDIATED PLATELET ACTIVATION. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,473397


Temple University

12. Tursi, Sarah Anne. Curli-Extracellular DNA Complexes: Pathogenicity and Role in Enteric Biofilms.

Degree: PhD, 2018, Temple University

Microbiology and Immunology

The first recorded observation of bacterial biofilms dates back to the 17th century by Antoine Van Leeuwenhoek. Today, biofilms are known as… (more)

Subjects/Keywords: Microbiology;

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tursi, S. A. (2018). Curli-Extracellular DNA Complexes: Pathogenicity and Role in Enteric Biofilms. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,511656

Chicago Manual of Style (16th Edition):

Tursi, Sarah Anne. “Curli-Extracellular DNA Complexes: Pathogenicity and Role in Enteric Biofilms.” 2018. Doctoral Dissertation, Temple University. Accessed September 18, 2019. http://digital.library.temple.edu/u?/p245801coll10,511656.

MLA Handbook (7th Edition):

Tursi, Sarah Anne. “Curli-Extracellular DNA Complexes: Pathogenicity and Role in Enteric Biofilms.” 2018. Web. 18 Sep 2019.

Vancouver:

Tursi SA. Curli-Extracellular DNA Complexes: Pathogenicity and Role in Enteric Biofilms. [Internet] [Doctoral dissertation]. Temple University; 2018. [cited 2019 Sep 18]. Available from: http://digital.library.temple.edu/u?/p245801coll10,511656.

Council of Science Editors:

Tursi SA. Curli-Extracellular DNA Complexes: Pathogenicity and Role in Enteric Biofilms. [Doctoral Dissertation]. Temple University; 2018. Available from: http://digital.library.temple.edu/u?/p245801coll10,511656

.