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You searched for +publisher:"Temple University" +contributor:("Hu, Wenhui;"). Showing records 1 – 9 of 9 total matches.

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Temple University

1. Noch, Evan K. The Role of Cellular and Viral Oncogenes in the Regulation of Hypoxia and Glucose Metabolism in Malignant Brain Tumors.

Degree: PhD, 2011, Temple University

Biomedical Neuroscience

Glioblastomas continue to carry poor prognoses for patients despite advances in surgical, chemotherapeutic, and radiation regimens. One feature of glioblastoma associated with poor… (more)

Subjects/Keywords: Neurosciences; Oncology; Virology; AEG-1; Glioblastoma; Glucose metabolism; Glycolysis; Hypoxia; JC Virus

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APA (6th Edition):

Noch, E. K. (2011). The Role of Cellular and Viral Oncogenes in the Regulation of Hypoxia and Glucose Metabolism in Malignant Brain Tumors. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,153816

Chicago Manual of Style (16th Edition):

Noch, Evan K. “The Role of Cellular and Viral Oncogenes in the Regulation of Hypoxia and Glucose Metabolism in Malignant Brain Tumors.” 2011. Doctoral Dissertation, Temple University. Accessed October 28, 2020. http://digital.library.temple.edu/u?/p245801coll10,153816.

MLA Handbook (7th Edition):

Noch, Evan K. “The Role of Cellular and Viral Oncogenes in the Regulation of Hypoxia and Glucose Metabolism in Malignant Brain Tumors.” 2011. Web. 28 Oct 2020.

Vancouver:

Noch EK. The Role of Cellular and Viral Oncogenes in the Regulation of Hypoxia and Glucose Metabolism in Malignant Brain Tumors. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2020 Oct 28]. Available from: http://digital.library.temple.edu/u?/p245801coll10,153816.

Council of Science Editors:

Noch EK. The Role of Cellular and Viral Oncogenes in the Regulation of Hypoxia and Glucose Metabolism in Malignant Brain Tumors. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,153816


Temple University

2. Kogan, Michael. Myeloid specific regulation of NF-kB and M-CSF signaling in HIV-1 and AML.

Degree: PhD, 2013, Temple University

Biomedical Neuroscience

The HIV protein, Vpr, is a multifunctional accessory protein critical for efficient viral infection of target CD4+ T cells and macrophages. Vpr is… (more)

Subjects/Keywords: Neurosciences; Molecular biology; Biology; AML; HIV; Macrophage; M-CSF; NF-kappa B; Vpr

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APA (6th Edition):

Kogan, M. (2013). Myeloid specific regulation of NF-kB and M-CSF signaling in HIV-1 and AML. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,183721

Chicago Manual of Style (16th Edition):

Kogan, Michael. “Myeloid specific regulation of NF-kB and M-CSF signaling in HIV-1 and AML.” 2013. Doctoral Dissertation, Temple University. Accessed October 28, 2020. http://digital.library.temple.edu/u?/p245801coll10,183721.

MLA Handbook (7th Edition):

Kogan, Michael. “Myeloid specific regulation of NF-kB and M-CSF signaling in HIV-1 and AML.” 2013. Web. 28 Oct 2020.

Vancouver:

Kogan M. Myeloid specific regulation of NF-kB and M-CSF signaling in HIV-1 and AML. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2020 Oct 28]. Available from: http://digital.library.temple.edu/u?/p245801coll10,183721.

Council of Science Editors:

Kogan M. Myeloid specific regulation of NF-kB and M-CSF signaling in HIV-1 and AML. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,183721


Temple University

3. Zheng, Xiaoyi. GASP-1, a New Tumor Biomarker, Contributes to Tumorigenesis in Breast Cancer.

Degree: PhD, 2013, Temple University

Biology

Breast cancer is the second leading cause of death in United States. Using 2D-HPLE, a novel separation technology, G-protein coupled receptor-associated sorting protein 1(GASP-1)… (more)

Subjects/Keywords: Cellular biology; Biochemistry; Biology; biomarker; cancer; competitive ELISA; G-protein coupled receptor associated sorting protein 1; signaling pathway; tumorigenesis

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APA (6th Edition):

Zheng, X. (2013). GASP-1, a New Tumor Biomarker, Contributes to Tumorigenesis in Breast Cancer. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,214816

Chicago Manual of Style (16th Edition):

Zheng, Xiaoyi. “GASP-1, a New Tumor Biomarker, Contributes to Tumorigenesis in Breast Cancer.” 2013. Doctoral Dissertation, Temple University. Accessed October 28, 2020. http://digital.library.temple.edu/u?/p245801coll10,214816.

MLA Handbook (7th Edition):

Zheng, Xiaoyi. “GASP-1, a New Tumor Biomarker, Contributes to Tumorigenesis in Breast Cancer.” 2013. Web. 28 Oct 2020.

Vancouver:

Zheng X. GASP-1, a New Tumor Biomarker, Contributes to Tumorigenesis in Breast Cancer. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2020 Oct 28]. Available from: http://digital.library.temple.edu/u?/p245801coll10,214816.

Council of Science Editors:

Zheng X. GASP-1, a New Tumor Biomarker, Contributes to Tumorigenesis in Breast Cancer. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,214816


Temple University

4. Beltrami, Sarah. NEUROFIBROMATOSIS TYPE 2 PROTEIN (NF2) AS A REGULATOR OF TUMOR SUPPRESSORS AND VIRAL ONCOPROTEINS IN HUMAN GLIOBLASTOMA.

Degree: PhD, 2014, Temple University

Biomedical Neuroscience

Glioblastomas are the most common brain malignancy occurring in adults with the worst prognosis. Several obstacles have prevented the development of efficacious treatment… (more)

Subjects/Keywords: Neurosciences; Cellular biology; Molecular biology;

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APA (6th Edition):

Beltrami, S. (2014). NEUROFIBROMATOSIS TYPE 2 PROTEIN (NF2) AS A REGULATOR OF TUMOR SUPPRESSORS AND VIRAL ONCOPROTEINS IN HUMAN GLIOBLASTOMA. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,239188

Chicago Manual of Style (16th Edition):

Beltrami, Sarah. “NEUROFIBROMATOSIS TYPE 2 PROTEIN (NF2) AS A REGULATOR OF TUMOR SUPPRESSORS AND VIRAL ONCOPROTEINS IN HUMAN GLIOBLASTOMA.” 2014. Doctoral Dissertation, Temple University. Accessed October 28, 2020. http://digital.library.temple.edu/u?/p245801coll10,239188.

MLA Handbook (7th Edition):

Beltrami, Sarah. “NEUROFIBROMATOSIS TYPE 2 PROTEIN (NF2) AS A REGULATOR OF TUMOR SUPPRESSORS AND VIRAL ONCOPROTEINS IN HUMAN GLIOBLASTOMA.” 2014. Web. 28 Oct 2020.

Vancouver:

Beltrami S. NEUROFIBROMATOSIS TYPE 2 PROTEIN (NF2) AS A REGULATOR OF TUMOR SUPPRESSORS AND VIRAL ONCOPROTEINS IN HUMAN GLIOBLASTOMA. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2020 Oct 28]. Available from: http://digital.library.temple.edu/u?/p245801coll10,239188.

Council of Science Editors:

Beltrami S. NEUROFIBROMATOSIS TYPE 2 PROTEIN (NF2) AS A REGULATOR OF TUMOR SUPPRESSORS AND VIRAL ONCOPROTEINS IN HUMAN GLIOBLASTOMA. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,239188


Temple University

5. Kovalevich, Jane. Cocaine-Mediated Disruption of RXR-gamma Signaling: The Role of TNF-alpha.

Degree: PhD, 2014, Temple University

Biomedical Neuroscience

Cocaine abuse poses a substantial health and economic burden for which no effective treatment currently exists. Exposure to cocaine results in altered signaling… (more)

Subjects/Keywords: Neurosciences; Cellular biology; Molecular biology;

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APA (6th Edition):

Kovalevich, J. (2014). Cocaine-Mediated Disruption of RXR-gamma Signaling: The Role of TNF-alpha. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,242911

Chicago Manual of Style (16th Edition):

Kovalevich, Jane. “Cocaine-Mediated Disruption of RXR-gamma Signaling: The Role of TNF-alpha.” 2014. Doctoral Dissertation, Temple University. Accessed October 28, 2020. http://digital.library.temple.edu/u?/p245801coll10,242911.

MLA Handbook (7th Edition):

Kovalevich, Jane. “Cocaine-Mediated Disruption of RXR-gamma Signaling: The Role of TNF-alpha.” 2014. Web. 28 Oct 2020.

Vancouver:

Kovalevich J. Cocaine-Mediated Disruption of RXR-gamma Signaling: The Role of TNF-alpha. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2020 Oct 28]. Available from: http://digital.library.temple.edu/u?/p245801coll10,242911.

Council of Science Editors:

Kovalevich J. Cocaine-Mediated Disruption of RXR-gamma Signaling: The Role of TNF-alpha. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,242911


Temple University

6. Regan, Patrick M. Regulation and Functional Impact of Opioid Receptor Splicing in Response to Morphine.

Degree: PhD, 2015, Temple University

Biomedical Neuroscience

Multiple classes of pharmaceuticals, including acetaminophen, aspirin, and other nonsteroidal anti-inflammatory drugs (NSAIDs), are used to relieve mild to moderate pain; however, one… (more)

Subjects/Keywords: Neurosciences; Pharmacology; Virology;

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APA (6th Edition):

Regan, P. M. (2015). Regulation and Functional Impact of Opioid Receptor Splicing in Response to Morphine. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,344730

Chicago Manual of Style (16th Edition):

Regan, Patrick M. “Regulation and Functional Impact of Opioid Receptor Splicing in Response to Morphine.” 2015. Doctoral Dissertation, Temple University. Accessed October 28, 2020. http://digital.library.temple.edu/u?/p245801coll10,344730.

MLA Handbook (7th Edition):

Regan, Patrick M. “Regulation and Functional Impact of Opioid Receptor Splicing in Response to Morphine.” 2015. Web. 28 Oct 2020.

Vancouver:

Regan PM. Regulation and Functional Impact of Opioid Receptor Splicing in Response to Morphine. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Oct 28]. Available from: http://digital.library.temple.edu/u?/p245801coll10,344730.

Council of Science Editors:

Regan PM. Regulation and Functional Impact of Opioid Receptor Splicing in Response to Morphine. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,344730


Temple University

7. Pozniak, Paul Daniel. TNF-alpha-Induced Neuroregeneration through an NF-kappaB-dependent Pathway: A New Mechanism Involving EphB2 in the Context of HIV-1 Neuroinflammation.

Degree: PhD, 2016, Temple University

Biomedical Neuroscience

The use of highly active antiretroviral therapy (HAART) has significantly decreased the mortality rate of HIV-1 patients, however the increased survival has led… (more)

Subjects/Keywords: Neurosciences;

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APA (6th Edition):

Pozniak, P. D. (2016). TNF-alpha-Induced Neuroregeneration through an NF-kappaB-dependent Pathway: A New Mechanism Involving EphB2 in the Context of HIV-1 Neuroinflammation. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,386002

Chicago Manual of Style (16th Edition):

Pozniak, Paul Daniel. “TNF-alpha-Induced Neuroregeneration through an NF-kappaB-dependent Pathway: A New Mechanism Involving EphB2 in the Context of HIV-1 Neuroinflammation.” 2016. Doctoral Dissertation, Temple University. Accessed October 28, 2020. http://digital.library.temple.edu/u?/p245801coll10,386002.

MLA Handbook (7th Edition):

Pozniak, Paul Daniel. “TNF-alpha-Induced Neuroregeneration through an NF-kappaB-dependent Pathway: A New Mechanism Involving EphB2 in the Context of HIV-1 Neuroinflammation.” 2016. Web. 28 Oct 2020.

Vancouver:

Pozniak PD. TNF-alpha-Induced Neuroregeneration through an NF-kappaB-dependent Pathway: A New Mechanism Involving EphB2 in the Context of HIV-1 Neuroinflammation. [Internet] [Doctoral dissertation]. Temple University; 2016. [cited 2020 Oct 28]. Available from: http://digital.library.temple.edu/u?/p245801coll10,386002.

Council of Science Editors:

Pozniak PD. TNF-alpha-Induced Neuroregeneration through an NF-kappaB-dependent Pathway: A New Mechanism Involving EphB2 in the Context of HIV-1 Neuroinflammation. [Doctoral Dissertation]. Temple University; 2016. Available from: http://digital.library.temple.edu/u?/p245801coll10,386002


Temple University

8. Putatunda, Raj. HIV-1 INFECTION OF NEURAL STEM CELLS RESULTS IN COGNITIVE DEFICITS THROUGH ADULT NEUROGENIC MODULATION.

Degree: PhD, 2018, Temple University

Biomedical Sciences

While antiretroviral therapy (ART) regimens have significantly decreased the mortality rate in patients with HIV-1 infection and subsequent opportunistic infections, the co-morbidities continue… (more)

Subjects/Keywords: Neurosciences; Virology; Cellular biology;

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APA (6th Edition):

Putatunda, R. (2018). HIV-1 INFECTION OF NEURAL STEM CELLS RESULTS IN COGNITIVE DEFICITS THROUGH ADULT NEUROGENIC MODULATION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,514815

Chicago Manual of Style (16th Edition):

Putatunda, Raj. “HIV-1 INFECTION OF NEURAL STEM CELLS RESULTS IN COGNITIVE DEFICITS THROUGH ADULT NEUROGENIC MODULATION.” 2018. Doctoral Dissertation, Temple University. Accessed October 28, 2020. http://digital.library.temple.edu/u?/p245801coll10,514815.

MLA Handbook (7th Edition):

Putatunda, Raj. “HIV-1 INFECTION OF NEURAL STEM CELLS RESULTS IN COGNITIVE DEFICITS THROUGH ADULT NEUROGENIC MODULATION.” 2018. Web. 28 Oct 2020.

Vancouver:

Putatunda R. HIV-1 INFECTION OF NEURAL STEM CELLS RESULTS IN COGNITIVE DEFICITS THROUGH ADULT NEUROGENIC MODULATION. [Internet] [Doctoral dissertation]. Temple University; 2018. [cited 2020 Oct 28]. Available from: http://digital.library.temple.edu/u?/p245801coll10,514815.

Council of Science Editors:

Putatunda R. HIV-1 INFECTION OF NEURAL STEM CELLS RESULTS IN COGNITIVE DEFICITS THROUGH ADULT NEUROGENIC MODULATION. [Doctoral Dissertation]. Temple University; 2018. Available from: http://digital.library.temple.edu/u?/p245801coll10,514815


Temple University

9. Keefe, Kathleen Mary. In Vivo Visualization of Neural Pathways in the Rat Spinal Cord Using Viral Tracing.

Degree: PhD, 2018, Temple University

Neuroscience

Much of our understanding of the fascinating complexity of neuronal circuits comes from anatomical tracing studies that use dyes or fluorescent markers to highlight… (more)

Subjects/Keywords: Neurosciences; Health sciences; Cellular biology;

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APA (6th Edition):

Keefe, K. M. (2018). In Vivo Visualization of Neural Pathways in the Rat Spinal Cord Using Viral Tracing. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,521830

Chicago Manual of Style (16th Edition):

Keefe, Kathleen Mary. “In Vivo Visualization of Neural Pathways in the Rat Spinal Cord Using Viral Tracing.” 2018. Doctoral Dissertation, Temple University. Accessed October 28, 2020. http://digital.library.temple.edu/u?/p245801coll10,521830.

MLA Handbook (7th Edition):

Keefe, Kathleen Mary. “In Vivo Visualization of Neural Pathways in the Rat Spinal Cord Using Viral Tracing.” 2018. Web. 28 Oct 2020.

Vancouver:

Keefe KM. In Vivo Visualization of Neural Pathways in the Rat Spinal Cord Using Viral Tracing. [Internet] [Doctoral dissertation]. Temple University; 2018. [cited 2020 Oct 28]. Available from: http://digital.library.temple.edu/u?/p245801coll10,521830.

Council of Science Editors:

Keefe KM. In Vivo Visualization of Neural Pathways in the Rat Spinal Cord Using Viral Tracing. [Doctoral Dissertation]. Temple University; 2018. Available from: http://digital.library.temple.edu/u?/p245801coll10,521830

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