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You searched for +publisher:"Temple University" +contributor:("Hoffman, Barbara"). Showing records 1 – 7 of 7 total matches.

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Temple University

1. Mukherjee, Kaushiki. Role Of Gadd45a In BCR-ABL and NRASD12 Driven Leukemia.

Degree: PhD, 2015, Temple University

Biochemistry

BCR-ABL is known as the most common translocation in the myeloproliferative (MPD) disorder chronic myelogenous leukemia (CML); it is the first leukemia to be… (more)

Subjects/Keywords: Oncology; Molecular biology; Cellular biology;

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APA (6th Edition):

Mukherjee, K. (2015). Role Of Gadd45a In BCR-ABL and NRASD12 Driven Leukemia. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,322334

Chicago Manual of Style (16th Edition):

Mukherjee, Kaushiki. “Role Of Gadd45a In BCR-ABL and NRASD12 Driven Leukemia.” 2015. Doctoral Dissertation, Temple University. Accessed September 25, 2020. http://digital.library.temple.edu/u?/p245801coll10,322334.

MLA Handbook (7th Edition):

Mukherjee, Kaushiki. “Role Of Gadd45a In BCR-ABL and NRASD12 Driven Leukemia.” 2015. Web. 25 Sep 2020.

Vancouver:

Mukherjee K. Role Of Gadd45a In BCR-ABL and NRASD12 Driven Leukemia. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Sep 25]. Available from: http://digital.library.temple.edu/u?/p245801coll10,322334.

Council of Science Editors:

Mukherjee K. Role Of Gadd45a In BCR-ABL and NRASD12 Driven Leukemia. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,322334


Temple University

2. Mohamed-Hadley, Alisha. THE ROLE OF THE STRESS RESPONSE GENE GADD45A IN MODULATING MYC MEDIATED APOPTOSIS AND DIFFERENTIATION.

Degree: PhD, 2011, Temple University

Molecular Biology and Genetics

The Gadd45 family of proteins is known to play a central role as cellular stress sensors that modulate the response of… (more)

Subjects/Keywords: Molecular Biology; Apoptosis; Cytokine; Gadd45a; Myc; Myeloid cells

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APA (6th Edition):

Mohamed-Hadley, A. (2011). THE ROLE OF THE STRESS RESPONSE GENE GADD45A IN MODULATING MYC MEDIATED APOPTOSIS AND DIFFERENTIATION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,126332

Chicago Manual of Style (16th Edition):

Mohamed-Hadley, Alisha. “THE ROLE OF THE STRESS RESPONSE GENE GADD45A IN MODULATING MYC MEDIATED APOPTOSIS AND DIFFERENTIATION.” 2011. Doctoral Dissertation, Temple University. Accessed September 25, 2020. http://digital.library.temple.edu/u?/p245801coll10,126332.

MLA Handbook (7th Edition):

Mohamed-Hadley, Alisha. “THE ROLE OF THE STRESS RESPONSE GENE GADD45A IN MODULATING MYC MEDIATED APOPTOSIS AND DIFFERENTIATION.” 2011. Web. 25 Sep 2020.

Vancouver:

Mohamed-Hadley A. THE ROLE OF THE STRESS RESPONSE GENE GADD45A IN MODULATING MYC MEDIATED APOPTOSIS AND DIFFERENTIATION. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2020 Sep 25]. Available from: http://digital.library.temple.edu/u?/p245801coll10,126332.

Council of Science Editors:

Mohamed-Hadley A. THE ROLE OF THE STRESS RESPONSE GENE GADD45A IN MODULATING MYC MEDIATED APOPTOSIS AND DIFFERENTIATION. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,126332


Temple University

3. Maifrede, Silvia. EGR-1 TUMOR SUPPRESSOR IN BCR-ABL DRIVEN LEUKEMIA.

Degree: PhD, 2015, Temple University

Molecular Biology and Genetics

Chronic Myelogenous Leukemia (CML) is a hematological disease originated with a chromosomal translocation t(9;22)(q34;q11) in a pluripotent hematopoietic stem cell. CML… (more)

Subjects/Keywords: Molecular biology;

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APA (6th Edition):

Maifrede, S. (2015). EGR-1 TUMOR SUPPRESSOR IN BCR-ABL DRIVEN LEUKEMIA. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,321048

Chicago Manual of Style (16th Edition):

Maifrede, Silvia. “EGR-1 TUMOR SUPPRESSOR IN BCR-ABL DRIVEN LEUKEMIA.” 2015. Doctoral Dissertation, Temple University. Accessed September 25, 2020. http://digital.library.temple.edu/u?/p245801coll10,321048.

MLA Handbook (7th Edition):

Maifrede, Silvia. “EGR-1 TUMOR SUPPRESSOR IN BCR-ABL DRIVEN LEUKEMIA.” 2015. Web. 25 Sep 2020.

Vancouver:

Maifrede S. EGR-1 TUMOR SUPPRESSOR IN BCR-ABL DRIVEN LEUKEMIA. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Sep 25]. Available from: http://digital.library.temple.edu/u?/p245801coll10,321048.

Council of Science Editors:

Maifrede S. EGR-1 TUMOR SUPPRESSOR IN BCR-ABL DRIVEN LEUKEMIA. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,321048


Temple University

4. Chang, Jen-Kuan. The Biochemical Basis of The miR-21 Expression by The Mu-Opioid Receptor.

Degree: PhD, 2015, Temple University

Molecular Biology and Genetics

Opioid receptors are members of the superfamily of seven transmembrane G protein-coupled receptors (GPCRs) which share several structural and functional characteristics.… (more)

Subjects/Keywords: Molecular biology; Genetics; Immunology;

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APA (6th Edition):

Chang, J. (2015). The Biochemical Basis of The miR-21 Expression by The Mu-Opioid Receptor. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,352105

Chicago Manual of Style (16th Edition):

Chang, Jen-Kuan. “The Biochemical Basis of The miR-21 Expression by The Mu-Opioid Receptor.” 2015. Doctoral Dissertation, Temple University. Accessed September 25, 2020. http://digital.library.temple.edu/u?/p245801coll10,352105.

MLA Handbook (7th Edition):

Chang, Jen-Kuan. “The Biochemical Basis of The miR-21 Expression by The Mu-Opioid Receptor.” 2015. Web. 25 Sep 2020.

Vancouver:

Chang J. The Biochemical Basis of The miR-21 Expression by The Mu-Opioid Receptor. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Sep 25]. Available from: http://digital.library.temple.edu/u?/p245801coll10,352105.

Council of Science Editors:

Chang J. The Biochemical Basis of The miR-21 Expression by The Mu-Opioid Receptor. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,352105


Temple University

5. Kotredes, Kevin Patrick. Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression.

Degree: PhD, 2015, Temple University

Molecular Biology and Genetics

The isocitrate dehydrogenase (IDH) family of enzymes is central to cellular metabolism, catalyzing the oxidative decarboxylation of isocitrate to α-ketoglutarate (α-KG)… (more)

Subjects/Keywords: Molecular biology; Genetics; Biochemistry;

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APA (6th Edition):

Kotredes, K. P. (2015). Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,356693

Chicago Manual of Style (16th Edition):

Kotredes, Kevin Patrick. “Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression.” 2015. Doctoral Dissertation, Temple University. Accessed September 25, 2020. http://digital.library.temple.edu/u?/p245801coll10,356693.

MLA Handbook (7th Edition):

Kotredes, Kevin Patrick. “Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression.” 2015. Web. 25 Sep 2020.

Vancouver:

Kotredes KP. Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Sep 25]. Available from: http://digital.library.temple.edu/u?/p245801coll10,356693.

Council of Science Editors:

Kotredes KP. Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,356693


Temple University

6. Zumbrun, Steven David. Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b.

Degree: PhD, 2008, Temple University

Molecular Biology and Genetics

The GADD45 family of proteins consists of three small nuclear proteins, GADD45A, GADD45B, and GADD45G, which are implicated in modulating the… (more)

Subjects/Keywords: Biology, Molecular; gadd45b; MMS; post-transcriptional regulation; Sorbitol; stress response; transcriptional regulation

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zumbrun, S. D. (2008). Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,23355

Chicago Manual of Style (16th Edition):

Zumbrun, Steven David. “Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b.” 2008. Doctoral Dissertation, Temple University. Accessed September 25, 2020. http://digital.library.temple.edu/u?/p245801coll10,23355.

MLA Handbook (7th Edition):

Zumbrun, Steven David. “Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b.” 2008. Web. 25 Sep 2020.

Vancouver:

Zumbrun SD. Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b. [Internet] [Doctoral dissertation]. Temple University; 2008. [cited 2020 Sep 25]. Available from: http://digital.library.temple.edu/u?/p245801coll10,23355.

Council of Science Editors:

Zumbrun SD. Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b. [Doctoral Dissertation]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,23355


Temple University

7. Finley, Matthew James. Molecular Basis for Kappa-Opioid Regulation of Chemokine Receptor Function.

Degree: PhD, 2009, Temple University

Molecular Biology and Genetics

Opioid receptor-mediated regulation of chemokine receptors is vital for the host immune response, development, and neurological function. Previous studies have demonstrated… (more)

Subjects/Keywords: Biology, Molecular; Biology, Genetics; Biology, Neuroscience; Chemokine; Expression; Interferon Regulatory Factor; Opioid, Regulation; STAT3

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Finley, M. J. (2009). Molecular Basis for Kappa-Opioid Regulation of Chemokine Receptor Function. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,62878

Chicago Manual of Style (16th Edition):

Finley, Matthew James. “Molecular Basis for Kappa-Opioid Regulation of Chemokine Receptor Function.” 2009. Doctoral Dissertation, Temple University. Accessed September 25, 2020. http://digital.library.temple.edu/u?/p245801coll10,62878.

MLA Handbook (7th Edition):

Finley, Matthew James. “Molecular Basis for Kappa-Opioid Regulation of Chemokine Receptor Function.” 2009. Web. 25 Sep 2020.

Vancouver:

Finley MJ. Molecular Basis for Kappa-Opioid Regulation of Chemokine Receptor Function. [Internet] [Doctoral dissertation]. Temple University; 2009. [cited 2020 Sep 25]. Available from: http://digital.library.temple.edu/u?/p245801coll10,62878.

Council of Science Editors:

Finley MJ. Molecular Basis for Kappa-Opioid Regulation of Chemokine Receptor Function. [Doctoral Dissertation]. Temple University; 2009. Available from: http://digital.library.temple.edu/u?/p245801coll10,62878

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