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You searched for +publisher:"Temple University" +contributor:("Haines, Dale"). Showing records 1 – 11 of 11 total matches.

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Temple University

1. Ramkumar, Poornima. Interaction of JLP with PLK1 recruits FoxK1 to form a ternary complex during mitosis.

Degree: PhD, 2015, Temple University

Molecular Biology and Genetics

JLP (JNK associated Leucine zipper protein) is a scaffolding protein that has been shown to interact with and activate the JNK/p38MAPK… (more)

Subjects/Keywords: Molecular biology; Biochemistry; Cellular biology;

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APA (6th Edition):

Ramkumar, P. (2015). Interaction of JLP with PLK1 recruits FoxK1 to form a ternary complex during mitosis. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,323815

Chicago Manual of Style (16th Edition):

Ramkumar, Poornima. “Interaction of JLP with PLK1 recruits FoxK1 to form a ternary complex during mitosis.” 2015. Doctoral Dissertation, Temple University. Accessed August 14, 2020. http://digital.library.temple.edu/u?/p245801coll10,323815.

MLA Handbook (7th Edition):

Ramkumar, Poornima. “Interaction of JLP with PLK1 recruits FoxK1 to form a ternary complex during mitosis.” 2015. Web. 14 Aug 2020.

Vancouver:

Ramkumar P. Interaction of JLP with PLK1 recruits FoxK1 to form a ternary complex during mitosis. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Aug 14]. Available from: http://digital.library.temple.edu/u?/p245801coll10,323815.

Council of Science Editors:

Ramkumar P. Interaction of JLP with PLK1 recruits FoxK1 to form a ternary complex during mitosis. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,323815


Temple University

2. Gardner, Jacob Andrew. GPCR Signaling in the Genesis and Progression of Pancreatic Cancer.

Degree: PhD, 2009, Temple University

Molecular Biology and Genetics

Ductal adenocarcinomas of the pancreas are the 4th most common cause of cancer death. The 1 and 5 year survival rates… (more)

Subjects/Keywords: Biology, Molecular; G alpha 13; GPCR; LPA; lysophosphatidic acid; Migration; Pancreatic Cancer

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APA (6th Edition):

Gardner, J. A. (2009). GPCR Signaling in the Genesis and Progression of Pancreatic Cancer. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,35453

Chicago Manual of Style (16th Edition):

Gardner, Jacob Andrew. “GPCR Signaling in the Genesis and Progression of Pancreatic Cancer.” 2009. Doctoral Dissertation, Temple University. Accessed August 14, 2020. http://digital.library.temple.edu/u?/p245801coll10,35453.

MLA Handbook (7th Edition):

Gardner, Jacob Andrew. “GPCR Signaling in the Genesis and Progression of Pancreatic Cancer.” 2009. Web. 14 Aug 2020.

Vancouver:

Gardner JA. GPCR Signaling in the Genesis and Progression of Pancreatic Cancer. [Internet] [Doctoral dissertation]. Temple University; 2009. [cited 2020 Aug 14]. Available from: http://digital.library.temple.edu/u?/p245801coll10,35453.

Council of Science Editors:

Gardner JA. GPCR Signaling in the Genesis and Progression of Pancreatic Cancer. [Doctoral Dissertation]. Temple University; 2009. Available from: http://digital.library.temple.edu/u?/p245801coll10,35453


Temple University

3. Magimaidas, Andrew. The Role of the Stress Response Gene Gadd45b in Senescence.

Degree: PhD, 2015, Temple University

Molecular Biology and Genetics

The Gadd45 family of proteins (Gadd45a, Gadd45b, and Gadd45g) has been shown to act as stress sensors in response to various… (more)

Subjects/Keywords: Molecular biology; Genetics;

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APA (6th Edition):

Magimaidas, A. (2015). The Role of the Stress Response Gene Gadd45b in Senescence. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,376546

Chicago Manual of Style (16th Edition):

Magimaidas, Andrew. “The Role of the Stress Response Gene Gadd45b in Senescence.” 2015. Doctoral Dissertation, Temple University. Accessed August 14, 2020. http://digital.library.temple.edu/u?/p245801coll10,376546.

MLA Handbook (7th Edition):

Magimaidas, Andrew. “The Role of the Stress Response Gene Gadd45b in Senescence.” 2015. Web. 14 Aug 2020.

Vancouver:

Magimaidas A. The Role of the Stress Response Gene Gadd45b in Senescence. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Aug 14]. Available from: http://digital.library.temple.edu/u?/p245801coll10,376546.

Council of Science Editors:

Magimaidas A. The Role of the Stress Response Gene Gadd45b in Senescence. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,376546


Temple University

4. Cuneo, Anthony. THERAPEUTIC MECHANISMS OF INTERLEUKIN-19 FOR VASCULAR PROLIFERATIVE DISEASES.

Degree: PhD, 2012, Temple University

Molecular and Cellular Physiology

Cardiovascular disease is the leading cause of mortality in the western world. The pro-inflammatory and pro-proliferative etiology of vascular proliferative diseases… (more)

Subjects/Keywords: Biology, Molecular; Biology, General; Biology, Cell; Atherosclerosis; Human antigen R (HuR); Interleukin-19 (IL-19); oxidized LDL (ox-LDL); Vascular Proliferative Diseases; Vascular Smooth Muscle Cells (VSMC)

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APA (6th Edition):

Cuneo, A. (2012). THERAPEUTIC MECHANISMS OF INTERLEUKIN-19 FOR VASCULAR PROLIFERATIVE DISEASES. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,78032

Chicago Manual of Style (16th Edition):

Cuneo, Anthony. “THERAPEUTIC MECHANISMS OF INTERLEUKIN-19 FOR VASCULAR PROLIFERATIVE DISEASES.” 2012. Doctoral Dissertation, Temple University. Accessed August 14, 2020. http://digital.library.temple.edu/u?/p245801coll10,78032.

MLA Handbook (7th Edition):

Cuneo, Anthony. “THERAPEUTIC MECHANISMS OF INTERLEUKIN-19 FOR VASCULAR PROLIFERATIVE DISEASES.” 2012. Web. 14 Aug 2020.

Vancouver:

Cuneo A. THERAPEUTIC MECHANISMS OF INTERLEUKIN-19 FOR VASCULAR PROLIFERATIVE DISEASES. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Aug 14]. Available from: http://digital.library.temple.edu/u?/p245801coll10,78032.

Council of Science Editors:

Cuneo A. THERAPEUTIC MECHANISMS OF INTERLEUKIN-19 FOR VASCULAR PROLIFERATIVE DISEASES. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,78032


Temple University

5. Mohamed-Hadley, Alisha. THE ROLE OF THE STRESS RESPONSE GENE GADD45A IN MODULATING MYC MEDIATED APOPTOSIS AND DIFFERENTIATION.

Degree: PhD, 2011, Temple University

Molecular Biology and Genetics

The Gadd45 family of proteins is known to play a central role as cellular stress sensors that modulate the response of… (more)

Subjects/Keywords: Molecular Biology; Apoptosis; Cytokine; Gadd45a; Myc; Myeloid cells

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APA (6th Edition):

Mohamed-Hadley, A. (2011). THE ROLE OF THE STRESS RESPONSE GENE GADD45A IN MODULATING MYC MEDIATED APOPTOSIS AND DIFFERENTIATION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,126332

Chicago Manual of Style (16th Edition):

Mohamed-Hadley, Alisha. “THE ROLE OF THE STRESS RESPONSE GENE GADD45A IN MODULATING MYC MEDIATED APOPTOSIS AND DIFFERENTIATION.” 2011. Doctoral Dissertation, Temple University. Accessed August 14, 2020. http://digital.library.temple.edu/u?/p245801coll10,126332.

MLA Handbook (7th Edition):

Mohamed-Hadley, Alisha. “THE ROLE OF THE STRESS RESPONSE GENE GADD45A IN MODULATING MYC MEDIATED APOPTOSIS AND DIFFERENTIATION.” 2011. Web. 14 Aug 2020.

Vancouver:

Mohamed-Hadley A. THE ROLE OF THE STRESS RESPONSE GENE GADD45A IN MODULATING MYC MEDIATED APOPTOSIS AND DIFFERENTIATION. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2020 Aug 14]. Available from: http://digital.library.temple.edu/u?/p245801coll10,126332.

Council of Science Editors:

Mohamed-Hadley A. THE ROLE OF THE STRESS RESPONSE GENE GADD45A IN MODULATING MYC MEDIATED APOPTOSIS AND DIFFERENTIATION. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,126332


Temple University

6. Hoffman, Nicholas. Mitochondrial Calcium Influx is Determined by Multiple Protein Components Including SLC25A23 and MICU1.

Degree: PhD, 2014, Temple University

Biochemistry

Ca2+ control mechanisms employed by the cell at the plasma membrane include receptor operated, voltage-sensitive, and store operated channels for Ca2+ import. Upon entry… (more)

Subjects/Keywords: Biochemistry

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APA (6th Edition):

Hoffman, N. (2014). Mitochondrial Calcium Influx is Determined by Multiple Protein Components Including SLC25A23 and MICU1. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,287159

Chicago Manual of Style (16th Edition):

Hoffman, Nicholas. “Mitochondrial Calcium Influx is Determined by Multiple Protein Components Including SLC25A23 and MICU1.” 2014. Doctoral Dissertation, Temple University. Accessed August 14, 2020. http://digital.library.temple.edu/u?/p245801coll10,287159.

MLA Handbook (7th Edition):

Hoffman, Nicholas. “Mitochondrial Calcium Influx is Determined by Multiple Protein Components Including SLC25A23 and MICU1.” 2014. Web. 14 Aug 2020.

Vancouver:

Hoffman N. Mitochondrial Calcium Influx is Determined by Multiple Protein Components Including SLC25A23 and MICU1. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2020 Aug 14]. Available from: http://digital.library.temple.edu/u?/p245801coll10,287159.

Council of Science Editors:

Hoffman N. Mitochondrial Calcium Influx is Determined by Multiple Protein Components Including SLC25A23 and MICU1. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,287159


Temple University

7. Kurimchak, Alison. The B55α/PP2A Holoenzyme in Cell Cycle Exit, Maturation/Differentiation, and Cancer.

Degree: PhD, 2014, Temple University

Molecular Biology and Genetics

The cell cycle is negatively regulated by members of the pocket protein family, which consists of the tumor suppressor pRB and… (more)

Subjects/Keywords: Molecular biology;

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APA (6th Edition):

Kurimchak, A. (2014). The B55α/PP2A Holoenzyme in Cell Cycle Exit, Maturation/Differentiation, and Cancer. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,288430

Chicago Manual of Style (16th Edition):

Kurimchak, Alison. “The B55α/PP2A Holoenzyme in Cell Cycle Exit, Maturation/Differentiation, and Cancer.” 2014. Doctoral Dissertation, Temple University. Accessed August 14, 2020. http://digital.library.temple.edu/u?/p245801coll10,288430.

MLA Handbook (7th Edition):

Kurimchak, Alison. “The B55α/PP2A Holoenzyme in Cell Cycle Exit, Maturation/Differentiation, and Cancer.” 2014. Web. 14 Aug 2020.

Vancouver:

Kurimchak A. The B55α/PP2A Holoenzyme in Cell Cycle Exit, Maturation/Differentiation, and Cancer. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2020 Aug 14]. Available from: http://digital.library.temple.edu/u?/p245801coll10,288430.

Council of Science Editors:

Kurimchak A. The B55α/PP2A Holoenzyme in Cell Cycle Exit, Maturation/Differentiation, and Cancer. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,288430


Temple University

8. Firrman, Jenni Ann. ENHANCEMENT OF hFVIII ACTIVITY THROUGH LC MODIFICATIONS FOR GENE THERAPY OF HEMOPHILIA A.

Degree: PhD, 2015, Temple University

Microbiology and Immunology

Gene therapy for Hemophilia A (HA) using the recombinant Adeno-associated virus (rAAV) offers an alternative to classic treatment, which consists of FVIII… (more)

Subjects/Keywords: Microbiology; Immunology; Genetics;

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APA (6th Edition):

Firrman, J. A. (2015). ENHANCEMENT OF hFVIII ACTIVITY THROUGH LC MODIFICATIONS FOR GENE THERAPY OF HEMOPHILIA A. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,335863

Chicago Manual of Style (16th Edition):

Firrman, Jenni Ann. “ENHANCEMENT OF hFVIII ACTIVITY THROUGH LC MODIFICATIONS FOR GENE THERAPY OF HEMOPHILIA A.” 2015. Doctoral Dissertation, Temple University. Accessed August 14, 2020. http://digital.library.temple.edu/u?/p245801coll10,335863.

MLA Handbook (7th Edition):

Firrman, Jenni Ann. “ENHANCEMENT OF hFVIII ACTIVITY THROUGH LC MODIFICATIONS FOR GENE THERAPY OF HEMOPHILIA A.” 2015. Web. 14 Aug 2020.

Vancouver:

Firrman JA. ENHANCEMENT OF hFVIII ACTIVITY THROUGH LC MODIFICATIONS FOR GENE THERAPY OF HEMOPHILIA A. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Aug 14]. Available from: http://digital.library.temple.edu/u?/p245801coll10,335863.

Council of Science Editors:

Firrman JA. ENHANCEMENT OF hFVIII ACTIVITY THROUGH LC MODIFICATIONS FOR GENE THERAPY OF HEMOPHILIA A. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,335863


Temple University

9. Zumbrun, Steven David. Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b.

Degree: PhD, 2008, Temple University

Molecular Biology and Genetics

The GADD45 family of proteins consists of three small nuclear proteins, GADD45A, GADD45B, and GADD45G, which are implicated in modulating the… (more)

Subjects/Keywords: Biology, Molecular; gadd45b; MMS; post-transcriptional regulation; Sorbitol; stress response; transcriptional regulation

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APA (6th Edition):

Zumbrun, S. D. (2008). Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,23355

Chicago Manual of Style (16th Edition):

Zumbrun, Steven David. “Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b.” 2008. Doctoral Dissertation, Temple University. Accessed August 14, 2020. http://digital.library.temple.edu/u?/p245801coll10,23355.

MLA Handbook (7th Edition):

Zumbrun, Steven David. “Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b.” 2008. Web. 14 Aug 2020.

Vancouver:

Zumbrun SD. Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b. [Internet] [Doctoral dissertation]. Temple University; 2008. [cited 2020 Aug 14]. Available from: http://digital.library.temple.edu/u?/p245801coll10,23355.

Council of Science Editors:

Zumbrun SD. Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b. [Doctoral Dissertation]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,23355


Temple University

10. Jayadeva, Girish. B55alpha modulates the phosphorylation status of the pRb-related p107 and p130 proteins.

Degree: PhD, 2010, Temple University

Molecular Biology and Genetics

The retinoblastoma family of phosphoproteins consisting of the retinoblastoma protein (pRB) and the two structurally related proteins p130 and p107 play… (more)

Subjects/Keywords: Biology, Molecular; B55; p107; p130; pocket; PP2A; pRb

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APA (6th Edition):

Jayadeva, G. (2010). B55alpha modulates the phosphorylation status of the pRb-related p107 and p130 proteins. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,64785

Chicago Manual of Style (16th Edition):

Jayadeva, Girish. “B55alpha modulates the phosphorylation status of the pRb-related p107 and p130 proteins.” 2010. Doctoral Dissertation, Temple University. Accessed August 14, 2020. http://digital.library.temple.edu/u?/p245801coll10,64785.

MLA Handbook (7th Edition):

Jayadeva, Girish. “B55alpha modulates the phosphorylation status of the pRb-related p107 and p130 proteins.” 2010. Web. 14 Aug 2020.

Vancouver:

Jayadeva G. B55alpha modulates the phosphorylation status of the pRb-related p107 and p130 proteins. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Aug 14]. Available from: http://digital.library.temple.edu/u?/p245801coll10,64785.

Council of Science Editors:

Jayadeva G. B55alpha modulates the phosphorylation status of the pRb-related p107 and p130 proteins. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,64785


Temple University

11. Wagner, Jessica Michelle. Investigation of a novel small molecule TRAIL inducer, ONC201: pre-clinical anti-cancer efficacy, anti-metastasis effects, tumor immunity; and the structure-activity relationships (SAR) and mechanism of action of potential analogues.

Degree: PhD, 2018, Temple University

Cancer Biology & Genetics

ONC201 is a novel compound that upregulates endogenous TNF-Related Apoptosis-Inducing Ligand (TRAIL), in tumor and normal cells, restoring autocrine and paracrine… (more)

Subjects/Keywords: Biology; Pharmacology;

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APA (6th Edition):

Wagner, J. M. (2018). Investigation of a novel small molecule TRAIL inducer, ONC201: pre-clinical anti-cancer efficacy, anti-metastasis effects, tumor immunity; and the structure-activity relationships (SAR) and mechanism of action of potential analogues. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,499420

Chicago Manual of Style (16th Edition):

Wagner, Jessica Michelle. “Investigation of a novel small molecule TRAIL inducer, ONC201: pre-clinical anti-cancer efficacy, anti-metastasis effects, tumor immunity; and the structure-activity relationships (SAR) and mechanism of action of potential analogues.” 2018. Doctoral Dissertation, Temple University. Accessed August 14, 2020. http://digital.library.temple.edu/u?/p245801coll10,499420.

MLA Handbook (7th Edition):

Wagner, Jessica Michelle. “Investigation of a novel small molecule TRAIL inducer, ONC201: pre-clinical anti-cancer efficacy, anti-metastasis effects, tumor immunity; and the structure-activity relationships (SAR) and mechanism of action of potential analogues.” 2018. Web. 14 Aug 2020.

Vancouver:

Wagner JM. Investigation of a novel small molecule TRAIL inducer, ONC201: pre-clinical anti-cancer efficacy, anti-metastasis effects, tumor immunity; and the structure-activity relationships (SAR) and mechanism of action of potential analogues. [Internet] [Doctoral dissertation]. Temple University; 2018. [cited 2020 Aug 14]. Available from: http://digital.library.temple.edu/u?/p245801coll10,499420.

Council of Science Editors:

Wagner JM. Investigation of a novel small molecule TRAIL inducer, ONC201: pre-clinical anti-cancer efficacy, anti-metastasis effects, tumor immunity; and the structure-activity relationships (SAR) and mechanism of action of potential analogues. [Doctoral Dissertation]. Temple University; 2018. Available from: http://digital.library.temple.edu/u?/p245801coll10,499420

.