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You searched for +publisher:"Temple University" +contributor:("Grana-Amat, Xavier"). Showing records 1 – 11 of 11 total matches.

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Temple University

1. Beauparlant, Stephen Lewis. Functional characterization of the p97 adaptor protein UBXD1.

Degree: PhD, 2011, Temple University

Molecular Biology and Genetics

p97 is a member of the AAA family of proteins (ATPase Associated with various cellular Activities). It is a highly conserved… (more)

Subjects/Keywords: Molecular biology; Autophagy; ERGIC-53; p97; UBXD1

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APA (6th Edition):

Beauparlant, S. L. (2011). Functional characterization of the p97 adaptor protein UBXD1. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,213118

Chicago Manual of Style (16th Edition):

Beauparlant, Stephen Lewis. “Functional characterization of the p97 adaptor protein UBXD1.” 2011. Doctoral Dissertation, Temple University. Accessed July 20, 2019. http://digital.library.temple.edu/u?/p245801coll10,213118.

MLA Handbook (7th Edition):

Beauparlant, Stephen Lewis. “Functional characterization of the p97 adaptor protein UBXD1.” 2011. Web. 20 Jul 2019.

Vancouver:

Beauparlant SL. Functional characterization of the p97 adaptor protein UBXD1. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2019 Jul 20]. Available from: http://digital.library.temple.edu/u?/p245801coll10,213118.

Council of Science Editors:

Beauparlant SL. Functional characterization of the p97 adaptor protein UBXD1. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,213118


Temple University

2. Magimaidas, Andrew. The Role of the Stress Response Gene Gadd45b in Senescence.

Degree: PhD, 2015, Temple University

Molecular Biology and Genetics

The Gadd45 family of proteins (Gadd45a, Gadd45b, and Gadd45g) has been shown to act as stress sensors in response to various… (more)

Subjects/Keywords: Molecular biology; Genetics;

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APA (6th Edition):

Magimaidas, A. (2015). The Role of the Stress Response Gene Gadd45b in Senescence. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,376546

Chicago Manual of Style (16th Edition):

Magimaidas, Andrew. “The Role of the Stress Response Gene Gadd45b in Senescence.” 2015. Doctoral Dissertation, Temple University. Accessed July 20, 2019. http://digital.library.temple.edu/u?/p245801coll10,376546.

MLA Handbook (7th Edition):

Magimaidas, Andrew. “The Role of the Stress Response Gene Gadd45b in Senescence.” 2015. Web. 20 Jul 2019.

Vancouver:

Magimaidas A. The Role of the Stress Response Gene Gadd45b in Senescence. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Jul 20]. Available from: http://digital.library.temple.edu/u?/p245801coll10,376546.

Council of Science Editors:

Magimaidas A. The Role of the Stress Response Gene Gadd45b in Senescence. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,376546


Temple University

3. Padgaonkar, Amol. Discovery, Biological and Structural Characterization of ON108600, a Novel Kinase Inhibitor in Triple Negative Breast Cancer.

Degree: PhD, 2014, Temple University

Molecular Biology and Genetics

Selective killing of tumor cells requires the identification of drug targets critical to pathways that drive or support cancer progression. Protein… (more)

Subjects/Keywords: Molecular biology; Cellular biology; Oncology;

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APA (6th Edition):

Padgaonkar, A. (2014). Discovery, Biological and Structural Characterization of ON108600, a Novel Kinase Inhibitor in Triple Negative Breast Cancer. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,269327

Chicago Manual of Style (16th Edition):

Padgaonkar, Amol. “Discovery, Biological and Structural Characterization of ON108600, a Novel Kinase Inhibitor in Triple Negative Breast Cancer.” 2014. Doctoral Dissertation, Temple University. Accessed July 20, 2019. http://digital.library.temple.edu/u?/p245801coll10,269327.

MLA Handbook (7th Edition):

Padgaonkar, Amol. “Discovery, Biological and Structural Characterization of ON108600, a Novel Kinase Inhibitor in Triple Negative Breast Cancer.” 2014. Web. 20 Jul 2019.

Vancouver:

Padgaonkar A. Discovery, Biological and Structural Characterization of ON108600, a Novel Kinase Inhibitor in Triple Negative Breast Cancer. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Jul 20]. Available from: http://digital.library.temple.edu/u?/p245801coll10,269327.

Council of Science Editors:

Padgaonkar A. Discovery, Biological and Structural Characterization of ON108600, a Novel Kinase Inhibitor in Triple Negative Breast Cancer. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,269327


Temple University

4. Maifrede, Silvia. EGR-1 TUMOR SUPPRESSOR IN BCR-ABL DRIVEN LEUKEMIA.

Degree: PhD, 2015, Temple University

Molecular Biology and Genetics

Chronic Myelogenous Leukemia (CML) is a hematological disease originated with a chromosomal translocation t(9;22)(q34;q11) in a pluripotent hematopoietic stem cell. CML… (more)

Subjects/Keywords: Molecular biology;

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APA (6th Edition):

Maifrede, S. (2015). EGR-1 TUMOR SUPPRESSOR IN BCR-ABL DRIVEN LEUKEMIA. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,321048

Chicago Manual of Style (16th Edition):

Maifrede, Silvia. “EGR-1 TUMOR SUPPRESSOR IN BCR-ABL DRIVEN LEUKEMIA.” 2015. Doctoral Dissertation, Temple University. Accessed July 20, 2019. http://digital.library.temple.edu/u?/p245801coll10,321048.

MLA Handbook (7th Edition):

Maifrede, Silvia. “EGR-1 TUMOR SUPPRESSOR IN BCR-ABL DRIVEN LEUKEMIA.” 2015. Web. 20 Jul 2019.

Vancouver:

Maifrede S. EGR-1 TUMOR SUPPRESSOR IN BCR-ABL DRIVEN LEUKEMIA. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Jul 20]. Available from: http://digital.library.temple.edu/u?/p245801coll10,321048.

Council of Science Editors:

Maifrede S. EGR-1 TUMOR SUPPRESSOR IN BCR-ABL DRIVEN LEUKEMIA. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,321048


Temple University

5. Chawla, Rachna. Role of CDK4 in Development and Cancer.

Degree: PhD, 2008, Temple University

Molecular Biology and Genetics

The mammalian cell cycle is divided into four distinct phases: G1, S, G2, and M. The transition from G1 to S… (more)

Subjects/Keywords: Biology; Molecular

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APA (6th Edition):

Chawla, R. (2008). Role of CDK4 in Development and Cancer. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,7450

Chicago Manual of Style (16th Edition):

Chawla, Rachna. “Role of CDK4 in Development and Cancer.” 2008. Doctoral Dissertation, Temple University. Accessed July 20, 2019. http://digital.library.temple.edu/u?/p245801coll10,7450.

MLA Handbook (7th Edition):

Chawla, Rachna. “Role of CDK4 in Development and Cancer.” 2008. Web. 20 Jul 2019.

Vancouver:

Chawla R. Role of CDK4 in Development and Cancer. [Internet] [Doctoral dissertation]. Temple University; 2008. [cited 2019 Jul 20]. Available from: http://digital.library.temple.edu/u?/p245801coll10,7450.

Council of Science Editors:

Chawla R. Role of CDK4 in Development and Cancer. [Doctoral Dissertation]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,7450


Temple University

6. Zumbrun, Steven David. Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b.

Degree: PhD, 2008, Temple University

Molecular Biology and Genetics

The GADD45 family of proteins consists of three small nuclear proteins, GADD45A, GADD45B, and GADD45G, which are implicated in modulating the… (more)

Subjects/Keywords: Biology, Molecular; gadd45b; MMS; post-transcriptional regulation; Sorbitol; stress response; transcriptional regulation

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APA (6th Edition):

Zumbrun, S. D. (2008). Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,23355

Chicago Manual of Style (16th Edition):

Zumbrun, Steven David. “Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b.” 2008. Doctoral Dissertation, Temple University. Accessed July 20, 2019. http://digital.library.temple.edu/u?/p245801coll10,23355.

MLA Handbook (7th Edition):

Zumbrun, Steven David. “Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b.” 2008. Web. 20 Jul 2019.

Vancouver:

Zumbrun SD. Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b. [Internet] [Doctoral dissertation]. Temple University; 2008. [cited 2019 Jul 20]. Available from: http://digital.library.temple.edu/u?/p245801coll10,23355.

Council of Science Editors:

Zumbrun SD. Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b. [Doctoral Dissertation]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,23355


Temple University

7. Jayadeva, Girish. B55alpha modulates the phosphorylation status of the pRb-related p107 and p130 proteins.

Degree: PhD, 2010, Temple University

Molecular Biology and Genetics

The retinoblastoma family of phosphoproteins consisting of the retinoblastoma protein (pRB) and the two structurally related proteins p130 and p107 play… (more)

Subjects/Keywords: Biology, Molecular; B55; p107; p130; pocket; PP2A; pRb

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APA (6th Edition):

Jayadeva, G. (2010). B55alpha modulates the phosphorylation status of the pRb-related p107 and p130 proteins. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,64785

Chicago Manual of Style (16th Edition):

Jayadeva, Girish. “B55alpha modulates the phosphorylation status of the pRb-related p107 and p130 proteins.” 2010. Doctoral Dissertation, Temple University. Accessed July 20, 2019. http://digital.library.temple.edu/u?/p245801coll10,64785.

MLA Handbook (7th Edition):

Jayadeva, Girish. “B55alpha modulates the phosphorylation status of the pRb-related p107 and p130 proteins.” 2010. Web. 20 Jul 2019.

Vancouver:

Jayadeva G. B55alpha modulates the phosphorylation status of the pRb-related p107 and p130 proteins. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2019 Jul 20]. Available from: http://digital.library.temple.edu/u?/p245801coll10,64785.

Council of Science Editors:

Jayadeva G. B55alpha modulates the phosphorylation status of the pRb-related p107 and p130 proteins. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,64785


Temple University

8. Wagner, Jessica Michelle. Investigation of a novel small molecule TRAIL inducer, ONC201: pre-clinical anti-cancer efficacy, anti-metastasis effects, tumor immunity; and the structure-activity relationships (SAR) and mechanism of action of potential analogues.

Degree: PhD, 2018, Temple University

Cancer Biology & Genetics

ONC201 is a novel compound that upregulates endogenous TNF-Related Apoptosis-Inducing Ligand (TRAIL), in tumor and normal cells, restoring autocrine and paracrine… (more)

Subjects/Keywords: Biology; Pharmacology;

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APA (6th Edition):

Wagner, J. M. (2018). Investigation of a novel small molecule TRAIL inducer, ONC201: pre-clinical anti-cancer efficacy, anti-metastasis effects, tumor immunity; and the structure-activity relationships (SAR) and mechanism of action of potential analogues. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,499420

Chicago Manual of Style (16th Edition):

Wagner, Jessica Michelle. “Investigation of a novel small molecule TRAIL inducer, ONC201: pre-clinical anti-cancer efficacy, anti-metastasis effects, tumor immunity; and the structure-activity relationships (SAR) and mechanism of action of potential analogues.” 2018. Doctoral Dissertation, Temple University. Accessed July 20, 2019. http://digital.library.temple.edu/u?/p245801coll10,499420.

MLA Handbook (7th Edition):

Wagner, Jessica Michelle. “Investigation of a novel small molecule TRAIL inducer, ONC201: pre-clinical anti-cancer efficacy, anti-metastasis effects, tumor immunity; and the structure-activity relationships (SAR) and mechanism of action of potential analogues.” 2018. Web. 20 Jul 2019.

Vancouver:

Wagner JM. Investigation of a novel small molecule TRAIL inducer, ONC201: pre-clinical anti-cancer efficacy, anti-metastasis effects, tumor immunity; and the structure-activity relationships (SAR) and mechanism of action of potential analogues. [Internet] [Doctoral dissertation]. Temple University; 2018. [cited 2019 Jul 20]. Available from: http://digital.library.temple.edu/u?/p245801coll10,499420.

Council of Science Editors:

Wagner JM. Investigation of a novel small molecule TRAIL inducer, ONC201: pre-clinical anti-cancer efficacy, anti-metastasis effects, tumor immunity; and the structure-activity relationships (SAR) and mechanism of action of potential analogues. [Doctoral Dissertation]. Temple University; 2018. Available from: http://digital.library.temple.edu/u?/p245801coll10,499420


Temple University

9. Michael, James. Regulation of Ras signaling and oncogenesis by plasma membrane microdomains.

Degree: PhD, 2016, Temple University

Cell Biology

In this study, we assessed the contributions of plasma membrane (PM) microdomain targeting to the functions of H-Ras and R-Ras. These paralogues have… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Michael, J. (2016). Regulation of Ras signaling and oncogenesis by plasma membrane microdomains. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,377230

Chicago Manual of Style (16th Edition):

Michael, James. “Regulation of Ras signaling and oncogenesis by plasma membrane microdomains.” 2016. Doctoral Dissertation, Temple University. Accessed July 20, 2019. http://digital.library.temple.edu/u?/p245801coll10,377230.

MLA Handbook (7th Edition):

Michael, James. “Regulation of Ras signaling and oncogenesis by plasma membrane microdomains.” 2016. Web. 20 Jul 2019.

Vancouver:

Michael J. Regulation of Ras signaling and oncogenesis by plasma membrane microdomains. [Internet] [Doctoral dissertation]. Temple University; 2016. [cited 2019 Jul 20]. Available from: http://digital.library.temple.edu/u?/p245801coll10,377230.

Council of Science Editors:

Michael J. Regulation of Ras signaling and oncogenesis by plasma membrane microdomains. [Doctoral Dissertation]. Temple University; 2016. Available from: http://digital.library.temple.edu/u?/p245801coll10,377230


Temple University

10. Samakai, Elsie. TRANSCRIPTIONAL CONTROL OF Ca2+ SIGNALING IN T CELLS.

Degree: PhD, 2017, Temple University

Biochemistry

Antigen presentation to T cells results in their activation through T Cell Receptor (TCR) stimulation, resulting in sustained elevation of cytosolic Ca2+ concentration critical… (more)

Subjects/Keywords: Biochemistry; Immunology;

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APA (6th Edition):

Samakai, E. (2017). TRANSCRIPTIONAL CONTROL OF Ca2+ SIGNALING IN T CELLS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,466164

Chicago Manual of Style (16th Edition):

Samakai, Elsie. “TRANSCRIPTIONAL CONTROL OF Ca2+ SIGNALING IN T CELLS.” 2017. Doctoral Dissertation, Temple University. Accessed July 20, 2019. http://digital.library.temple.edu/u?/p245801coll10,466164.

MLA Handbook (7th Edition):

Samakai, Elsie. “TRANSCRIPTIONAL CONTROL OF Ca2+ SIGNALING IN T CELLS.” 2017. Web. 20 Jul 2019.

Vancouver:

Samakai E. TRANSCRIPTIONAL CONTROL OF Ca2+ SIGNALING IN T CELLS. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2019 Jul 20]. Available from: http://digital.library.temple.edu/u?/p245801coll10,466164.

Council of Science Editors:

Samakai E. TRANSCRIPTIONAL CONTROL OF Ca2+ SIGNALING IN T CELLS. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,466164


Temple University

11. Reed, Katherine Sullivan. Simultaneous Targeting of PARP1 and RAD52 Triggers Dual Synthetic Lethality in BRCA-Deficient Cancers.

Degree: PhD, 2018, Temple University

Biomedical Sciences

PARP inhibitors (PARPi) have been used to induce synthetic lethality in BRCA-deficient tumors in clinical trials with limited success due to the development… (more)

Subjects/Keywords: Oncology; Cellular biology;

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APA (6th Edition):

Reed, K. S. (2018). Simultaneous Targeting of PARP1 and RAD52 Triggers Dual Synthetic Lethality in BRCA-Deficient Cancers. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,507327

Chicago Manual of Style (16th Edition):

Reed, Katherine Sullivan. “Simultaneous Targeting of PARP1 and RAD52 Triggers Dual Synthetic Lethality in BRCA-Deficient Cancers.” 2018. Doctoral Dissertation, Temple University. Accessed July 20, 2019. http://digital.library.temple.edu/u?/p245801coll10,507327.

MLA Handbook (7th Edition):

Reed, Katherine Sullivan. “Simultaneous Targeting of PARP1 and RAD52 Triggers Dual Synthetic Lethality in BRCA-Deficient Cancers.” 2018. Web. 20 Jul 2019.

Vancouver:

Reed KS. Simultaneous Targeting of PARP1 and RAD52 Triggers Dual Synthetic Lethality in BRCA-Deficient Cancers. [Internet] [Doctoral dissertation]. Temple University; 2018. [cited 2019 Jul 20]. Available from: http://digital.library.temple.edu/u?/p245801coll10,507327.

Council of Science Editors:

Reed KS. Simultaneous Targeting of PARP1 and RAD52 Triggers Dual Synthetic Lethality in BRCA-Deficient Cancers. [Doctoral Dissertation]. Temple University; 2018. Available from: http://digital.library.temple.edu/u?/p245801coll10,507327

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