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You searched for +publisher:"Temple University" +contributor:("Goldfinger, Lawrence"). Showing records 1 – 11 of 11 total matches.

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Temple University

1. Singh, Harinder. CAVEOLAE AS SPATIO-TEMPORAL COMPARTMENTS FOR ROS/RNS GENERATION AND NITROXIDATIVE STRESS SIGNALING.

Degree: PhD, 2014, Temple University

Cell Biology

During inflammatory conditions excessive production of reactive oxygen (ROS) and nitrogen species (RNS), peroxynitrite, is implicated in the development of vascular pathologies. Our… (more)

Subjects/Keywords: Biology; Cellular biology

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APA (6th Edition):

Singh, H. (2014). CAVEOLAE AS SPATIO-TEMPORAL COMPARTMENTS FOR ROS/RNS GENERATION AND NITROXIDATIVE STRESS SIGNALING. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,275561

Chicago Manual of Style (16th Edition):

Singh, Harinder. “CAVEOLAE AS SPATIO-TEMPORAL COMPARTMENTS FOR ROS/RNS GENERATION AND NITROXIDATIVE STRESS SIGNALING.” 2014. Doctoral Dissertation, Temple University. Accessed September 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,275561.

MLA Handbook (7th Edition):

Singh, Harinder. “CAVEOLAE AS SPATIO-TEMPORAL COMPARTMENTS FOR ROS/RNS GENERATION AND NITROXIDATIVE STRESS SIGNALING.” 2014. Web. 21 Sep 2019.

Vancouver:

Singh H. CAVEOLAE AS SPATIO-TEMPORAL COMPARTMENTS FOR ROS/RNS GENERATION AND NITROXIDATIVE STRESS SIGNALING. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,275561.

Council of Science Editors:

Singh H. CAVEOLAE AS SPATIO-TEMPORAL COMPARTMENTS FOR ROS/RNS GENERATION AND NITROXIDATIVE STRESS SIGNALING. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,275561


Temple University

2. Bhavanasi, Dheeraj. Role of Protein Kinase C delta in regulating platelet functional responses.

Degree: PhD, 2014, Temple University

Physiology

Platelets upon activation change their shape, aggregate and secrete alpha and dense granule contents among which ADP acts as a feedback activator. Different Protein… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Bhavanasi, D. (2014). Role of Protein Kinase C delta in regulating platelet functional responses. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,283099

Chicago Manual of Style (16th Edition):

Bhavanasi, Dheeraj. “Role of Protein Kinase C delta in regulating platelet functional responses.” 2014. Doctoral Dissertation, Temple University. Accessed September 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,283099.

MLA Handbook (7th Edition):

Bhavanasi, Dheeraj. “Role of Protein Kinase C delta in regulating platelet functional responses.” 2014. Web. 21 Sep 2019.

Vancouver:

Bhavanasi D. Role of Protein Kinase C delta in regulating platelet functional responses. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,283099.

Council of Science Editors:

Bhavanasi D. Role of Protein Kinase C delta in regulating platelet functional responses. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,283099


Temple University

3. BADOLIA, RACHIT. MOLECULAR PHYSIOLOGY OF p21-ACTIVATED KINASES.

Degree: PhD, 2015, Temple University

Organ Systems & Translational Medicine

Platelets are involved in many processes ranging from fighting microbial infections and triggering inflammation to promoting tumor angiogenesis and metastasis.… (more)

Subjects/Keywords: Physiology;

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APA (6th Edition):

BADOLIA, R. (2015). MOLECULAR PHYSIOLOGY OF p21-ACTIVATED KINASES. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,368893

Chicago Manual of Style (16th Edition):

BADOLIA, RACHIT. “MOLECULAR PHYSIOLOGY OF p21-ACTIVATED KINASES.” 2015. Doctoral Dissertation, Temple University. Accessed September 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,368893.

MLA Handbook (7th Edition):

BADOLIA, RACHIT. “MOLECULAR PHYSIOLOGY OF p21-ACTIVATED KINASES.” 2015. Web. 21 Sep 2019.

Vancouver:

BADOLIA R. MOLECULAR PHYSIOLOGY OF p21-ACTIVATED KINASES. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,368893.

Council of Science Editors:

BADOLIA R. MOLECULAR PHYSIOLOGY OF p21-ACTIVATED KINASES. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,368893


Temple University

4. Nemani, Neeharika. Molecular Determinant of Mitochondrial Shape Change.

Degree: PhD, 2018, Temple University

Biomedical Sciences

Mitochondria shape cytosolic Ca2+ (cCa2+) transients. Ca2+ entry into the mitochondria is driven by the highly negative mitochondrial membrane potential and through a… (more)

Subjects/Keywords: Molecular biology; Biochemistry;

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APA (6th Edition):

Nemani, N. (2018). Molecular Determinant of Mitochondrial Shape Change. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,511170

Chicago Manual of Style (16th Edition):

Nemani, Neeharika. “Molecular Determinant of Mitochondrial Shape Change.” 2018. Doctoral Dissertation, Temple University. Accessed September 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,511170.

MLA Handbook (7th Edition):

Nemani, Neeharika. “Molecular Determinant of Mitochondrial Shape Change.” 2018. Web. 21 Sep 2019.

Vancouver:

Nemani N. Molecular Determinant of Mitochondrial Shape Change. [Internet] [Doctoral dissertation]. Temple University; 2018. [cited 2019 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,511170.

Council of Science Editors:

Nemani N. Molecular Determinant of Mitochondrial Shape Change. [Doctoral Dissertation]. Temple University; 2018. Available from: http://digital.library.temple.edu/u?/p245801coll10,511170


Temple University

5. Hendesi, Honey. CONNECTIVE TISSUE GROWTH FACTOR (CTGF/CCN2) REGULATES OSTEOBLAST CYTOSKELETAL REORGANIZATION AND MOTILITY AND ENHANCES DIFFERENTIATION VIA BINDING TO INTEGRIN RECEPTORS AND ACTIVATION OF DOWNSTREAM SIGNALINGS.

Degree: PhD, 2014, Temple University

Cell Biology

Connective Tissue Growth Factor (CTGF) is a matricellular protein that has been shown to mediate cell adhesion, and as a consequence, it regulates… (more)

Subjects/Keywords: Cellular biology;

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APA (6th Edition):

Hendesi, H. (2014). CONNECTIVE TISSUE GROWTH FACTOR (CTGF/CCN2) REGULATES OSTEOBLAST CYTOSKELETAL REORGANIZATION AND MOTILITY AND ENHANCES DIFFERENTIATION VIA BINDING TO INTEGRIN RECEPTORS AND ACTIVATION OF DOWNSTREAM SIGNALINGS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,263674

Chicago Manual of Style (16th Edition):

Hendesi, Honey. “CONNECTIVE TISSUE GROWTH FACTOR (CTGF/CCN2) REGULATES OSTEOBLAST CYTOSKELETAL REORGANIZATION AND MOTILITY AND ENHANCES DIFFERENTIATION VIA BINDING TO INTEGRIN RECEPTORS AND ACTIVATION OF DOWNSTREAM SIGNALINGS.” 2014. Doctoral Dissertation, Temple University. Accessed September 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,263674.

MLA Handbook (7th Edition):

Hendesi, Honey. “CONNECTIVE TISSUE GROWTH FACTOR (CTGF/CCN2) REGULATES OSTEOBLAST CYTOSKELETAL REORGANIZATION AND MOTILITY AND ENHANCES DIFFERENTIATION VIA BINDING TO INTEGRIN RECEPTORS AND ACTIVATION OF DOWNSTREAM SIGNALINGS.” 2014. Web. 21 Sep 2019.

Vancouver:

Hendesi H. CONNECTIVE TISSUE GROWTH FACTOR (CTGF/CCN2) REGULATES OSTEOBLAST CYTOSKELETAL REORGANIZATION AND MOTILITY AND ENHANCES DIFFERENTIATION VIA BINDING TO INTEGRIN RECEPTORS AND ACTIVATION OF DOWNSTREAM SIGNALINGS. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,263674.

Council of Science Editors:

Hendesi H. CONNECTIVE TISSUE GROWTH FACTOR (CTGF/CCN2) REGULATES OSTEOBLAST CYTOSKELETAL REORGANIZATION AND MOTILITY AND ENHANCES DIFFERENTIATION VIA BINDING TO INTEGRIN RECEPTORS AND ACTIVATION OF DOWNSTREAM SIGNALINGS. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,263674


Temple University

6. Mundy, Christina Maria. The Interaction Between Connective Tissue Growth Factor and Bone Morphogenetic Protein-2 During Osteoblast Differentiation and Function.

Degree: PhD, 2014, Temple University

Cell Biology

Connective tissue growth factor (CTGF/CCN2) and bone morphogenetic protein (BMP)-2 are both produced and secreted by osteoblasts. Both proteins have been shown to… (more)

Subjects/Keywords: Cellular biology;

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APA (6th Edition):

Mundy, C. M. (2014). The Interaction Between Connective Tissue Growth Factor and Bone Morphogenetic Protein-2 During Osteoblast Differentiation and Function. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,269581

Chicago Manual of Style (16th Edition):

Mundy, Christina Maria. “The Interaction Between Connective Tissue Growth Factor and Bone Morphogenetic Protein-2 During Osteoblast Differentiation and Function.” 2014. Doctoral Dissertation, Temple University. Accessed September 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,269581.

MLA Handbook (7th Edition):

Mundy, Christina Maria. “The Interaction Between Connective Tissue Growth Factor and Bone Morphogenetic Protein-2 During Osteoblast Differentiation and Function.” 2014. Web. 21 Sep 2019.

Vancouver:

Mundy CM. The Interaction Between Connective Tissue Growth Factor and Bone Morphogenetic Protein-2 During Osteoblast Differentiation and Function. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,269581.

Council of Science Editors:

Mundy CM. The Interaction Between Connective Tissue Growth Factor and Bone Morphogenetic Protein-2 During Osteoblast Differentiation and Function. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,269581


Temple University

7. Crawford, Kevin John. THE ROLE OF CAVEOLAE IN THE FORMATION OF ABDOMINAL AORTIC ANEURYSMS.

Degree: PhD, 2015, Temple University

Cell Biology

Abdominal aortic aneurysm (AAA) is a major cardiovascular disease and involves enhancement of renin-angiotensin system and recruitment/activation of inflammatory factors such as matrix… (more)

Subjects/Keywords: Cellular biology;

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APA (6th Edition):

Crawford, K. J. (2015). THE ROLE OF CAVEOLAE IN THE FORMATION OF ABDOMINAL AORTIC ANEURYSMS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,312992

Chicago Manual of Style (16th Edition):

Crawford, Kevin John. “THE ROLE OF CAVEOLAE IN THE FORMATION OF ABDOMINAL AORTIC ANEURYSMS.” 2015. Doctoral Dissertation, Temple University. Accessed September 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,312992.

MLA Handbook (7th Edition):

Crawford, Kevin John. “THE ROLE OF CAVEOLAE IN THE FORMATION OF ABDOMINAL AORTIC ANEURYSMS.” 2015. Web. 21 Sep 2019.

Vancouver:

Crawford KJ. THE ROLE OF CAVEOLAE IN THE FORMATION OF ABDOMINAL AORTIC ANEURYSMS. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,312992.

Council of Science Editors:

Crawford KJ. THE ROLE OF CAVEOLAE IN THE FORMATION OF ABDOMINAL AORTIC ANEURYSMS. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,312992


Temple University

8. Firrman, Jenni Ann. ENHANCEMENT OF hFVIII ACTIVITY THROUGH LC MODIFICATIONS FOR GENE THERAPY OF HEMOPHILIA A.

Degree: PhD, 2015, Temple University

Microbiology and Immunology

Gene therapy for Hemophilia A (HA) using the recombinant Adeno-associated virus (rAAV) offers an alternative to classic treatment, which consists of FVIII… (more)

Subjects/Keywords: Microbiology; Immunology; Genetics;

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APA (6th Edition):

Firrman, J. A. (2015). ENHANCEMENT OF hFVIII ACTIVITY THROUGH LC MODIFICATIONS FOR GENE THERAPY OF HEMOPHILIA A. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,335863

Chicago Manual of Style (16th Edition):

Firrman, Jenni Ann. “ENHANCEMENT OF hFVIII ACTIVITY THROUGH LC MODIFICATIONS FOR GENE THERAPY OF HEMOPHILIA A.” 2015. Doctoral Dissertation, Temple University. Accessed September 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,335863.

MLA Handbook (7th Edition):

Firrman, Jenni Ann. “ENHANCEMENT OF hFVIII ACTIVITY THROUGH LC MODIFICATIONS FOR GENE THERAPY OF HEMOPHILIA A.” 2015. Web. 21 Sep 2019.

Vancouver:

Firrman JA. ENHANCEMENT OF hFVIII ACTIVITY THROUGH LC MODIFICATIONS FOR GENE THERAPY OF HEMOPHILIA A. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,335863.

Council of Science Editors:

Firrman JA. ENHANCEMENT OF hFVIII ACTIVITY THROUGH LC MODIFICATIONS FOR GENE THERAPY OF HEMOPHILIA A. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,335863


Temple University

9. Kotredes, Kevin Patrick. Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression.

Degree: PhD, 2015, Temple University

Molecular Biology and Genetics

The isocitrate dehydrogenase (IDH) family of enzymes is central to cellular metabolism, catalyzing the oxidative decarboxylation of isocitrate to α-ketoglutarate (α-KG)… (more)

Subjects/Keywords: Molecular biology; Genetics; Biochemistry;

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APA (6th Edition):

Kotredes, K. P. (2015). Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,356693

Chicago Manual of Style (16th Edition):

Kotredes, Kevin Patrick. “Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression.” 2015. Doctoral Dissertation, Temple University. Accessed September 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,356693.

MLA Handbook (7th Edition):

Kotredes, Kevin Patrick. “Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression.” 2015. Web. 21 Sep 2019.

Vancouver:

Kotredes KP. Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,356693.

Council of Science Editors:

Kotredes KP. Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,356693


Temple University

10. Etwebi, Zienab. MYELOPEROXIDASE INDUCES ENDOTHELIAL DYSFUNCTION VIA ACTIVATION OF THE CALCIUM DEPENDENT PROTEASE CALPAIN.

Degree: PhD, 2018, Temple University

Biomedical Sciences

Cardiovascular disease and the associated endothelial dysfunction are characterized by leukocyte activation, decrease endothelial nitric oxide synthase (eNOS) activity, and increased endothelial cell… (more)

Subjects/Keywords: Physiology; Cellular biology;

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APA (6th Edition):

Etwebi, Z. (2018). MYELOPEROXIDASE INDUCES ENDOTHELIAL DYSFUNCTION VIA ACTIVATION OF THE CALCIUM DEPENDENT PROTEASE CALPAIN. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,508536

Chicago Manual of Style (16th Edition):

Etwebi, Zienab. “MYELOPEROXIDASE INDUCES ENDOTHELIAL DYSFUNCTION VIA ACTIVATION OF THE CALCIUM DEPENDENT PROTEASE CALPAIN.” 2018. Doctoral Dissertation, Temple University. Accessed September 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,508536.

MLA Handbook (7th Edition):

Etwebi, Zienab. “MYELOPEROXIDASE INDUCES ENDOTHELIAL DYSFUNCTION VIA ACTIVATION OF THE CALCIUM DEPENDENT PROTEASE CALPAIN.” 2018. Web. 21 Sep 2019.

Vancouver:

Etwebi Z. MYELOPEROXIDASE INDUCES ENDOTHELIAL DYSFUNCTION VIA ACTIVATION OF THE CALCIUM DEPENDENT PROTEASE CALPAIN. [Internet] [Doctoral dissertation]. Temple University; 2018. [cited 2019 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,508536.

Council of Science Editors:

Etwebi Z. MYELOPEROXIDASE INDUCES ENDOTHELIAL DYSFUNCTION VIA ACTIVATION OF THE CALCIUM DEPENDENT PROTEASE CALPAIN. [Doctoral Dissertation]. Temple University; 2018. Available from: http://digital.library.temple.edu/u?/p245801coll10,508536


Temple University

11. Michael, James. Regulation of Ras signaling and oncogenesis by plasma membrane microdomains.

Degree: PhD, 2016, Temple University

Cell Biology

In this study, we assessed the contributions of plasma membrane (PM) microdomain targeting to the functions of H-Ras and R-Ras. These paralogues have… (more)

Subjects/Keywords: Cellular biology

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Michael, J. (2016). Regulation of Ras signaling and oncogenesis by plasma membrane microdomains. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,377230

Chicago Manual of Style (16th Edition):

Michael, James. “Regulation of Ras signaling and oncogenesis by plasma membrane microdomains.” 2016. Doctoral Dissertation, Temple University. Accessed September 21, 2019. http://digital.library.temple.edu/u?/p245801coll10,377230.

MLA Handbook (7th Edition):

Michael, James. “Regulation of Ras signaling and oncogenesis by plasma membrane microdomains.” 2016. Web. 21 Sep 2019.

Vancouver:

Michael J. Regulation of Ras signaling and oncogenesis by plasma membrane microdomains. [Internet] [Doctoral dissertation]. Temple University; 2016. [cited 2019 Sep 21]. Available from: http://digital.library.temple.edu/u?/p245801coll10,377230.

Council of Science Editors:

Michael J. Regulation of Ras signaling and oncogenesis by plasma membrane microdomains. [Doctoral Dissertation]. Temple University; 2016. Available from: http://digital.library.temple.edu/u?/p245801coll10,377230

.