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You searched for +publisher:"Temple University" +contributor:("Gamero, Ana;"). Showing records 1 – 17 of 17 total matches.

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Temple University

1. Teets, Bryan Wilson. SF-1, BUT NOT DAX-1, PREVENTS P19 CELLS FROM DIFFERENTIATING TO EITHER TROMA-1 OR TUJ1 POSITIVE CELLS UPON RA-TREATMENT.

Degree: PhD, 2011, Temple University

Biochemistry

Retinoic acid (RA) is critical for embryonic development and cell differentiation. Previous work in our laboratory has shown that blocking the RA-dependent increase in… (more)

Subjects/Keywords: Biochemistry; adrenal; Differentiation; neuronal; P19; PBX; Vitamin A

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APA (6th Edition):

Teets, B. W. (2011). SF-1, BUT NOT DAX-1, PREVENTS P19 CELLS FROM DIFFERENTIATING TO EITHER TROMA-1 OR TUJ1 POSITIVE CELLS UPON RA-TREATMENT. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,124708

Chicago Manual of Style (16th Edition):

Teets, Bryan Wilson. “SF-1, BUT NOT DAX-1, PREVENTS P19 CELLS FROM DIFFERENTIATING TO EITHER TROMA-1 OR TUJ1 POSITIVE CELLS UPON RA-TREATMENT.” 2011. Doctoral Dissertation, Temple University. Accessed October 16, 2019. http://digital.library.temple.edu/u?/p245801coll10,124708.

MLA Handbook (7th Edition):

Teets, Bryan Wilson. “SF-1, BUT NOT DAX-1, PREVENTS P19 CELLS FROM DIFFERENTIATING TO EITHER TROMA-1 OR TUJ1 POSITIVE CELLS UPON RA-TREATMENT.” 2011. Web. 16 Oct 2019.

Vancouver:

Teets BW. SF-1, BUT NOT DAX-1, PREVENTS P19 CELLS FROM DIFFERENTIATING TO EITHER TROMA-1 OR TUJ1 POSITIVE CELLS UPON RA-TREATMENT. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2019 Oct 16]. Available from: http://digital.library.temple.edu/u?/p245801coll10,124708.

Council of Science Editors:

Teets BW. SF-1, BUT NOT DAX-1, PREVENTS P19 CELLS FROM DIFFERENTIATING TO EITHER TROMA-1 OR TUJ1 POSITIVE CELLS UPON RA-TREATMENT. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,124708


Temple University

2. Kocieda, Virginia Polonia. Prostaglandin E2-induced IL-23p19 is regulated by CREB and C/EBP beta in bone marrow derived dendritic cells.

Degree: PhD, 2013, Temple University

Microbiology and Immunology

We reported previously that prostaglandin E2 (PGE2) upregulates IL-23 in vitro in bone marrow-derived dendritic cells (DC), and in vivo in models… (more)

Subjects/Keywords: Immunology; Molecular biology; C/EBP; CREB; dendritic cell; IL-23; PGE2

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APA (6th Edition):

Kocieda, V. P. (2013). Prostaglandin E2-induced IL-23p19 is regulated by CREB and C/EBP beta in bone marrow derived dendritic cells. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,214805

Chicago Manual of Style (16th Edition):

Kocieda, Virginia Polonia. “Prostaglandin E2-induced IL-23p19 is regulated by CREB and C/EBP beta in bone marrow derived dendritic cells.” 2013. Doctoral Dissertation, Temple University. Accessed October 16, 2019. http://digital.library.temple.edu/u?/p245801coll10,214805.

MLA Handbook (7th Edition):

Kocieda, Virginia Polonia. “Prostaglandin E2-induced IL-23p19 is regulated by CREB and C/EBP beta in bone marrow derived dendritic cells.” 2013. Web. 16 Oct 2019.

Vancouver:

Kocieda VP. Prostaglandin E2-induced IL-23p19 is regulated by CREB and C/EBP beta in bone marrow derived dendritic cells. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2019 Oct 16]. Available from: http://digital.library.temple.edu/u?/p245801coll10,214805.

Council of Science Editors:

Kocieda VP. Prostaglandin E2-induced IL-23p19 is regulated by CREB and C/EBP beta in bone marrow derived dendritic cells. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,214805


Temple University

3. Doonan, Patrick John. Mitochondrial Calcium Uptake: LETM1 and MICU1 Are Mitochondrial Proteins That Regulate Mitochondrial Calcium Homeostasis and Cellular Bioenergetics.

Degree: PhD, 2012, Temple University

Biochemistry

Mitochondrial calcium (Ca2+) uptake has been studied for over five decades, with crucial insights into its underlying mechanisms enabled by development of the chemi-osmotic… (more)

Subjects/Keywords: Biochemistry; Molecular biology; Calcium; LETM1; MICU1; Mitochondria; Mitochondria Calcium Uniporter; Uniporter

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APA (6th Edition):

Doonan, P. J. (2012). Mitochondrial Calcium Uptake: LETM1 and MICU1 Are Mitochondrial Proteins That Regulate Mitochondrial Calcium Homeostasis and Cellular Bioenergetics. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,214818

Chicago Manual of Style (16th Edition):

Doonan, Patrick John. “Mitochondrial Calcium Uptake: LETM1 and MICU1 Are Mitochondrial Proteins That Regulate Mitochondrial Calcium Homeostasis and Cellular Bioenergetics.” 2012. Doctoral Dissertation, Temple University. Accessed October 16, 2019. http://digital.library.temple.edu/u?/p245801coll10,214818.

MLA Handbook (7th Edition):

Doonan, Patrick John. “Mitochondrial Calcium Uptake: LETM1 and MICU1 Are Mitochondrial Proteins That Regulate Mitochondrial Calcium Homeostasis and Cellular Bioenergetics.” 2012. Web. 16 Oct 2019.

Vancouver:

Doonan PJ. Mitochondrial Calcium Uptake: LETM1 and MICU1 Are Mitochondrial Proteins That Regulate Mitochondrial Calcium Homeostasis and Cellular Bioenergetics. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2019 Oct 16]. Available from: http://digital.library.temple.edu/u?/p245801coll10,214818.

Council of Science Editors:

Doonan PJ. Mitochondrial Calcium Uptake: LETM1 and MICU1 Are Mitochondrial Proteins That Regulate Mitochondrial Calcium Homeostasis and Cellular Bioenergetics. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,214818


Temple University

4. Xu, Jun. Regulation of type I interferons in murine dendritic cells.

Degree: PhD, 2014, Temple University

Microbiology and Immunology

Conventional Dendritic cells (cDCs), a specialized group of immunological sentinels with tree-like or dendritic shapes, are critical for recognition of danger signals,… (more)

Subjects/Keywords: Immunology; Microbiology;

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APA (6th Edition):

Xu, J. (2014). Regulation of type I interferons in murine dendritic cells. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,308946

Chicago Manual of Style (16th Edition):

Xu, Jun. “Regulation of type I interferons in murine dendritic cells.” 2014. Doctoral Dissertation, Temple University. Accessed October 16, 2019. http://digital.library.temple.edu/u?/p245801coll10,308946.

MLA Handbook (7th Edition):

Xu, Jun. “Regulation of type I interferons in murine dendritic cells.” 2014. Web. 16 Oct 2019.

Vancouver:

Xu J. Regulation of type I interferons in murine dendritic cells. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Oct 16]. Available from: http://digital.library.temple.edu/u?/p245801coll10,308946.

Council of Science Editors:

Xu J. Regulation of type I interferons in murine dendritic cells. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,308946


Temple University

5. Mukherjee, Kaushiki. Role Of Gadd45a In BCR-ABL and NRASD12 Driven Leukemia.

Degree: PhD, 2015, Temple University

Biochemistry

BCR-ABL is known as the most common translocation in the myeloproliferative (MPD) disorder chronic myelogenous leukemia (CML); it is the first leukemia to be… (more)

Subjects/Keywords: Oncology; Molecular biology; Cellular biology;

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APA (6th Edition):

Mukherjee, K. (2015). Role Of Gadd45a In BCR-ABL and NRASD12 Driven Leukemia. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,322334

Chicago Manual of Style (16th Edition):

Mukherjee, Kaushiki. “Role Of Gadd45a In BCR-ABL and NRASD12 Driven Leukemia.” 2015. Doctoral Dissertation, Temple University. Accessed October 16, 2019. http://digital.library.temple.edu/u?/p245801coll10,322334.

MLA Handbook (7th Edition):

Mukherjee, Kaushiki. “Role Of Gadd45a In BCR-ABL and NRASD12 Driven Leukemia.” 2015. Web. 16 Oct 2019.

Vancouver:

Mukherjee K. Role Of Gadd45a In BCR-ABL and NRASD12 Driven Leukemia. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Oct 16]. Available from: http://digital.library.temple.edu/u?/p245801coll10,322334.

Council of Science Editors:

Mukherjee K. Role Of Gadd45a In BCR-ABL and NRASD12 Driven Leukemia. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,322334


Temple University

6. Yue, Chanyu. STAT2 in the regulation of tumor growth and antitumor effects of type I interferons in a mouse model of melanoma.

Degree: PhD, 2013, Temple University

Biochemistry

Signal Transducer and Activator of Transcription (STAT) 2 is one of seven members of the STAT family of transcription factors with dual roles in… (more)

Subjects/Keywords: Biochemistry; Oncology;

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APA (6th Edition):

Yue, C. (2013). STAT2 in the regulation of tumor growth and antitumor effects of type I interferons in a mouse model of melanoma. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,233175

Chicago Manual of Style (16th Edition):

Yue, Chanyu. “STAT2 in the regulation of tumor growth and antitumor effects of type I interferons in a mouse model of melanoma.” 2013. Doctoral Dissertation, Temple University. Accessed October 16, 2019. http://digital.library.temple.edu/u?/p245801coll10,233175.

MLA Handbook (7th Edition):

Yue, Chanyu. “STAT2 in the regulation of tumor growth and antitumor effects of type I interferons in a mouse model of melanoma.” 2013. Web. 16 Oct 2019.

Vancouver:

Yue C. STAT2 in the regulation of tumor growth and antitumor effects of type I interferons in a mouse model of melanoma. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2019 Oct 16]. Available from: http://digital.library.temple.edu/u?/p245801coll10,233175.

Council of Science Editors:

Yue C. STAT2 in the regulation of tumor growth and antitumor effects of type I interferons in a mouse model of melanoma. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,233175


Temple University

7. Wang, Fang. The Role of Acinus in Retinoic Acid Signaling Pathway.

Degree: PhD, 2014, Temple University

Biochemistry

Retinoic acid receptor (RAR), a member of the steroid/thyroid hormone nuclear receptor superfamily, functions as a RA-dependent transcription activator bound to the RA response… (more)

Subjects/Keywords: Biochemistry;

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APA (6th Edition):

Wang, F. (2014). The Role of Acinus in Retinoic Acid Signaling Pathway. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,277479

Chicago Manual of Style (16th Edition):

Wang, Fang. “The Role of Acinus in Retinoic Acid Signaling Pathway.” 2014. Doctoral Dissertation, Temple University. Accessed October 16, 2019. http://digital.library.temple.edu/u?/p245801coll10,277479.

MLA Handbook (7th Edition):

Wang, Fang. “The Role of Acinus in Retinoic Acid Signaling Pathway.” 2014. Web. 16 Oct 2019.

Vancouver:

Wang F. The Role of Acinus in Retinoic Acid Signaling Pathway. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Oct 16]. Available from: http://digital.library.temple.edu/u?/p245801coll10,277479.

Council of Science Editors:

Wang F. The Role of Acinus in Retinoic Acid Signaling Pathway. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,277479


Temple University

8. Kurimchak, Alison. The B55α/PP2A Holoenzyme in Cell Cycle Exit, Maturation/Differentiation, and Cancer.

Degree: PhD, 2014, Temple University

Molecular Biology and Genetics

The cell cycle is negatively regulated by members of the pocket protein family, which consists of the tumor suppressor pRB and… (more)

Subjects/Keywords: Molecular biology;

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APA (6th Edition):

Kurimchak, A. (2014). The B55α/PP2A Holoenzyme in Cell Cycle Exit, Maturation/Differentiation, and Cancer. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,288430

Chicago Manual of Style (16th Edition):

Kurimchak, Alison. “The B55α/PP2A Holoenzyme in Cell Cycle Exit, Maturation/Differentiation, and Cancer.” 2014. Doctoral Dissertation, Temple University. Accessed October 16, 2019. http://digital.library.temple.edu/u?/p245801coll10,288430.

MLA Handbook (7th Edition):

Kurimchak, Alison. “The B55α/PP2A Holoenzyme in Cell Cycle Exit, Maturation/Differentiation, and Cancer.” 2014. Web. 16 Oct 2019.

Vancouver:

Kurimchak A. The B55α/PP2A Holoenzyme in Cell Cycle Exit, Maturation/Differentiation, and Cancer. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2019 Oct 16]. Available from: http://digital.library.temple.edu/u?/p245801coll10,288430.

Council of Science Editors:

Kurimchak A. The B55α/PP2A Holoenzyme in Cell Cycle Exit, Maturation/Differentiation, and Cancer. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,288430


Temple University

9. Oppong, Gertrude Odamea. The Role of Bacterial Amyloids In Regulating Gastrointestinal Homeostasis.

Degree: PhD, 2015, Temple University

Microbiology and Immunology

Many bacterial species exist in nature as part of highly structured multicellular communities known as biofilms. Amyloids, proteins with a conserved β-sheet… (more)

Subjects/Keywords: Microbiology;

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APA (6th Edition):

Oppong, G. O. (2015). The Role of Bacterial Amyloids In Regulating Gastrointestinal Homeostasis. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,323094

Chicago Manual of Style (16th Edition):

Oppong, Gertrude Odamea. “The Role of Bacterial Amyloids In Regulating Gastrointestinal Homeostasis.” 2015. Doctoral Dissertation, Temple University. Accessed October 16, 2019. http://digital.library.temple.edu/u?/p245801coll10,323094.

MLA Handbook (7th Edition):

Oppong, Gertrude Odamea. “The Role of Bacterial Amyloids In Regulating Gastrointestinal Homeostasis.” 2015. Web. 16 Oct 2019.

Vancouver:

Oppong GO. The Role of Bacterial Amyloids In Regulating Gastrointestinal Homeostasis. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Oct 16]. Available from: http://digital.library.temple.edu/u?/p245801coll10,323094.

Council of Science Editors:

Oppong GO. The Role of Bacterial Amyloids In Regulating Gastrointestinal Homeostasis. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,323094


Temple University

10. Daswani, Varsha. Fluorescence and aggregation properties of the anti-cancer drug, CA4P, in archaeal liposomes.

Degree: PhD, 2015, Temple University

Biomedical Sciences

Combretastatin A4 phosphate (CA4P) is a potent vascular disrupting agent utilized in the treatment of cancer. The observed rapid vascular shutdown post administration… (more)

Subjects/Keywords: Biochemistry; Biophysics; Chemistry;

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APA (6th Edition):

Daswani, V. (2015). Fluorescence and aggregation properties of the anti-cancer drug, CA4P, in archaeal liposomes. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,334013

Chicago Manual of Style (16th Edition):

Daswani, Varsha. “Fluorescence and aggregation properties of the anti-cancer drug, CA4P, in archaeal liposomes.” 2015. Doctoral Dissertation, Temple University. Accessed October 16, 2019. http://digital.library.temple.edu/u?/p245801coll10,334013.

MLA Handbook (7th Edition):

Daswani, Varsha. “Fluorescence and aggregation properties of the anti-cancer drug, CA4P, in archaeal liposomes.” 2015. Web. 16 Oct 2019.

Vancouver:

Daswani V. Fluorescence and aggregation properties of the anti-cancer drug, CA4P, in archaeal liposomes. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Oct 16]. Available from: http://digital.library.temple.edu/u?/p245801coll10,334013.

Council of Science Editors:

Daswani V. Fluorescence and aggregation properties of the anti-cancer drug, CA4P, in archaeal liposomes. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,334013


Temple University

11. Kotredes, Kevin Patrick. Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression.

Degree: PhD, 2015, Temple University

Molecular Biology and Genetics

The isocitrate dehydrogenase (IDH) family of enzymes is central to cellular metabolism, catalyzing the oxidative decarboxylation of isocitrate to α-ketoglutarate (α-KG)… (more)

Subjects/Keywords: Molecular biology; Genetics; Biochemistry;

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APA (6th Edition):

Kotredes, K. P. (2015). Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,356693

Chicago Manual of Style (16th Edition):

Kotredes, Kevin Patrick. “Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression.” 2015. Doctoral Dissertation, Temple University. Accessed October 16, 2019. http://digital.library.temple.edu/u?/p245801coll10,356693.

MLA Handbook (7th Edition):

Kotredes, Kevin Patrick. “Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression.” 2015. Web. 16 Oct 2019.

Vancouver:

Kotredes KP. Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2019 Oct 16]. Available from: http://digital.library.temple.edu/u?/p245801coll10,356693.

Council of Science Editors:

Kotredes KP. Characterization of the effects of IDH2 mutations and (R)-2-HG in cancer progression. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,356693


Temple University

12. Ayesa, Umme. CHARACTERIZATION OF THERMOSENSITIVE HYBRID ARCHAEOSOMES AND DPA-CY3[22,22]/POPC LIPOSOMES AND IN VITRO EVALUATION OF THEIR POTENTIAL USEFULNESS IN TARGETED DELIVERY AND CONTROLLED RELEASE.

Degree: PhD, 2016, Temple University

Biochemistry

One of earlier challenges in treating cancer was utilizing drugs that are powerful yet do not cause severe toxicity to patients. Although the use… (more)

Subjects/Keywords: Biophysics

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APA (6th Edition):

Ayesa, U. (2016). CHARACTERIZATION OF THERMOSENSITIVE HYBRID ARCHAEOSOMES AND DPA-CY3[22,22]/POPC LIPOSOMES AND IN VITRO EVALUATION OF THEIR POTENTIAL USEFULNESS IN TARGETED DELIVERY AND CONTROLLED RELEASE. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,368614

Chicago Manual of Style (16th Edition):

Ayesa, Umme. “CHARACTERIZATION OF THERMOSENSITIVE HYBRID ARCHAEOSOMES AND DPA-CY3[22,22]/POPC LIPOSOMES AND IN VITRO EVALUATION OF THEIR POTENTIAL USEFULNESS IN TARGETED DELIVERY AND CONTROLLED RELEASE.” 2016. Doctoral Dissertation, Temple University. Accessed October 16, 2019. http://digital.library.temple.edu/u?/p245801coll10,368614.

MLA Handbook (7th Edition):

Ayesa, Umme. “CHARACTERIZATION OF THERMOSENSITIVE HYBRID ARCHAEOSOMES AND DPA-CY3[22,22]/POPC LIPOSOMES AND IN VITRO EVALUATION OF THEIR POTENTIAL USEFULNESS IN TARGETED DELIVERY AND CONTROLLED RELEASE.” 2016. Web. 16 Oct 2019.

Vancouver:

Ayesa U. CHARACTERIZATION OF THERMOSENSITIVE HYBRID ARCHAEOSOMES AND DPA-CY3[22,22]/POPC LIPOSOMES AND IN VITRO EVALUATION OF THEIR POTENTIAL USEFULNESS IN TARGETED DELIVERY AND CONTROLLED RELEASE. [Internet] [Doctoral dissertation]. Temple University; 2016. [cited 2019 Oct 16]. Available from: http://digital.library.temple.edu/u?/p245801coll10,368614.

Council of Science Editors:

Ayesa U. CHARACTERIZATION OF THERMOSENSITIVE HYBRID ARCHAEOSOMES AND DPA-CY3[22,22]/POPC LIPOSOMES AND IN VITRO EVALUATION OF THEIR POTENTIAL USEFULNESS IN TARGETED DELIVERY AND CONTROLLED RELEASE. [Doctoral Dissertation]. Temple University; 2016. Available from: http://digital.library.temple.edu/u?/p245801coll10,368614


Temple University

13. Zumbrun, Steven David. Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b.

Degree: PhD, 2008, Temple University

Molecular Biology and Genetics

The GADD45 family of proteins consists of three small nuclear proteins, GADD45A, GADD45B, and GADD45G, which are implicated in modulating the… (more)

Subjects/Keywords: Biology, Molecular; gadd45b; MMS; post-transcriptional regulation; Sorbitol; stress response; transcriptional regulation

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APA (6th Edition):

Zumbrun, S. D. (2008). Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,23355

Chicago Manual of Style (16th Edition):

Zumbrun, Steven David. “Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b.” 2008. Doctoral Dissertation, Temple University. Accessed October 16, 2019. http://digital.library.temple.edu/u?/p245801coll10,23355.

MLA Handbook (7th Edition):

Zumbrun, Steven David. “Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b.” 2008. Web. 16 Oct 2019.

Vancouver:

Zumbrun SD. Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b. [Internet] [Doctoral dissertation]. Temple University; 2008. [cited 2019 Oct 16]. Available from: http://digital.library.temple.edu/u?/p245801coll10,23355.

Council of Science Editors:

Zumbrun SD. Distinct Mechanisms Regulate Induction of Stress Effector, gadd45b. [Doctoral Dissertation]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,23355


Temple University

14. Hooper, Kirsten Mary. PGE2 AND IL-27: NOVEL PROINFLAMMATORY MECHANISMS INVOLVING DENDRITIC CELLS AND TYPE 1 REGULATORY T CELLS.

Degree: PhD, 2017, Temple University

Microbiology and Immunology

Interleukin-27 (p28/EBI3) is an immunomodulatory cytokine expressed by activated antigen presenting cells. Although first discovered to be involved in Th1 cell differentiation,… (more)

Subjects/Keywords: Immunology;

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APA (6th Edition):

Hooper, K. M. (2017). PGE2 AND IL-27: NOVEL PROINFLAMMATORY MECHANISMS INVOLVING DENDRITIC CELLS AND TYPE 1 REGULATORY T CELLS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,432693

Chicago Manual of Style (16th Edition):

Hooper, Kirsten Mary. “PGE2 AND IL-27: NOVEL PROINFLAMMATORY MECHANISMS INVOLVING DENDRITIC CELLS AND TYPE 1 REGULATORY T CELLS.” 2017. Doctoral Dissertation, Temple University. Accessed October 16, 2019. http://digital.library.temple.edu/u?/p245801coll10,432693.

MLA Handbook (7th Edition):

Hooper, Kirsten Mary. “PGE2 AND IL-27: NOVEL PROINFLAMMATORY MECHANISMS INVOLVING DENDRITIC CELLS AND TYPE 1 REGULATORY T CELLS.” 2017. Web. 16 Oct 2019.

Vancouver:

Hooper KM. PGE2 AND IL-27: NOVEL PROINFLAMMATORY MECHANISMS INVOLVING DENDRITIC CELLS AND TYPE 1 REGULATORY T CELLS. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2019 Oct 16]. Available from: http://digital.library.temple.edu/u?/p245801coll10,432693.

Council of Science Editors:

Hooper KM. PGE2 AND IL-27: NOVEL PROINFLAMMATORY MECHANISMS INVOLVING DENDRITIC CELLS AND TYPE 1 REGULATORY T CELLS. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,432693


Temple University

15. Tursi, Sarah Anne. Curli-Extracellular DNA Complexes: Pathogenicity and Role in Enteric Biofilms.

Degree: PhD, 2018, Temple University

Microbiology and Immunology

The first recorded observation of bacterial biofilms dates back to the 17th century by Antoine Van Leeuwenhoek. Today, biofilms are known as… (more)

Subjects/Keywords: Microbiology;

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tursi, S. A. (2018). Curli-Extracellular DNA Complexes: Pathogenicity and Role in Enteric Biofilms. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,511656

Chicago Manual of Style (16th Edition):

Tursi, Sarah Anne. “Curli-Extracellular DNA Complexes: Pathogenicity and Role in Enteric Biofilms.” 2018. Doctoral Dissertation, Temple University. Accessed October 16, 2019. http://digital.library.temple.edu/u?/p245801coll10,511656.

MLA Handbook (7th Edition):

Tursi, Sarah Anne. “Curli-Extracellular DNA Complexes: Pathogenicity and Role in Enteric Biofilms.” 2018. Web. 16 Oct 2019.

Vancouver:

Tursi SA. Curli-Extracellular DNA Complexes: Pathogenicity and Role in Enteric Biofilms. [Internet] [Doctoral dissertation]. Temple University; 2018. [cited 2019 Oct 16]. Available from: http://digital.library.temple.edu/u?/p245801coll10,511656.

Council of Science Editors:

Tursi SA. Curli-Extracellular DNA Complexes: Pathogenicity and Role in Enteric Biofilms. [Doctoral Dissertation]. Temple University; 2018. Available from: http://digital.library.temple.edu/u?/p245801coll10,511656


Temple University

16. Barton, Maria. Lnc-EPCAM AND Lnc-BHLHE41 AS RNA REGULATORS OF BREAST CANCER AND BREAST CANCER PREVENTION.

Degree: PhD, 2017, Temple University

Biochemistry

The objective of this study was to unveil a novel area of gene regulation in breast cancer and breast cancer prevention through the study… (more)

Subjects/Keywords: Cellular biology; Molecular biology; Biochemistry;

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Barton, M. (2017). Lnc-EPCAM AND Lnc-BHLHE41 AS RNA REGULATORS OF BREAST CANCER AND BREAST CANCER PREVENTION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,424232

Chicago Manual of Style (16th Edition):

Barton, Maria. “Lnc-EPCAM AND Lnc-BHLHE41 AS RNA REGULATORS OF BREAST CANCER AND BREAST CANCER PREVENTION.” 2017. Doctoral Dissertation, Temple University. Accessed October 16, 2019. http://digital.library.temple.edu/u?/p245801coll10,424232.

MLA Handbook (7th Edition):

Barton, Maria. “Lnc-EPCAM AND Lnc-BHLHE41 AS RNA REGULATORS OF BREAST CANCER AND BREAST CANCER PREVENTION.” 2017. Web. 16 Oct 2019.

Vancouver:

Barton M. Lnc-EPCAM AND Lnc-BHLHE41 AS RNA REGULATORS OF BREAST CANCER AND BREAST CANCER PREVENTION. [Internet] [Doctoral dissertation]. Temple University; 2017. [cited 2019 Oct 16]. Available from: http://digital.library.temple.edu/u?/p245801coll10,424232.

Council of Science Editors:

Barton M. Lnc-EPCAM AND Lnc-BHLHE41 AS RNA REGULATORS OF BREAST CANCER AND BREAST CANCER PREVENTION. [Doctoral Dissertation]. Temple University; 2017. Available from: http://digital.library.temple.edu/u?/p245801coll10,424232


Temple University

17. Pickens, Brandy S. THE ROLE OF COUP-TFI DURING RETINOIC ACID INDUCED ENDODERMAL DIFFERENTIATION OF P19 CELLS.

Degree: PhD, 2012, Temple University

Biochemistry

ABSTRACT Retinoic acid (RA) is a positive regulator of P19 EC cell differentiation. Pre-B cell leukemia transcription factors (PBXs) act in conjunction with homeobox… (more)

Subjects/Keywords: Biochemistry; COUP-TFI; Endodermal Differentiation; P19 Cells; Retinoic Acid

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Pickens, B. S. (2012). THE ROLE OF COUP-TFI DURING RETINOIC ACID INDUCED ENDODERMAL DIFFERENTIATION OF P19 CELLS. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,196883

Chicago Manual of Style (16th Edition):

Pickens, Brandy S. “THE ROLE OF COUP-TFI DURING RETINOIC ACID INDUCED ENDODERMAL DIFFERENTIATION OF P19 CELLS.” 2012. Doctoral Dissertation, Temple University. Accessed October 16, 2019. http://digital.library.temple.edu/u?/p245801coll10,196883.

MLA Handbook (7th Edition):

Pickens, Brandy S. “THE ROLE OF COUP-TFI DURING RETINOIC ACID INDUCED ENDODERMAL DIFFERENTIATION OF P19 CELLS.” 2012. Web. 16 Oct 2019.

Vancouver:

Pickens BS. THE ROLE OF COUP-TFI DURING RETINOIC ACID INDUCED ENDODERMAL DIFFERENTIATION OF P19 CELLS. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2019 Oct 16]. Available from: http://digital.library.temple.edu/u?/p245801coll10,196883.

Council of Science Editors:

Pickens BS. THE ROLE OF COUP-TFI DURING RETINOIC ACID INDUCED ENDODERMAL DIFFERENTIATION OF P19 CELLS. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,196883

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