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You searched for +publisher:"Temple University" +contributor:("Dun, Nae J."). Showing records 1 – 11 of 11 total matches.

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Temple University

1. Moore, Andrea Rossi. COX-2 inhibition impaired resolution of chronic inflammation in a murine model of autoimmune arthritis.

Degree: PhD, 2010, Temple University

Microbiology and Immunology

Rheumatoid arthritis (RA) is a chronic disease characterized by cycles of inflammation and resolution. Previously, it was believed that the resolution of… (more)

Subjects/Keywords: Health Sciences, Immunology; autoimmunity; inflammation; lipid mediators; rheumatoid arthritis; rodent

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APA (6th Edition):

Moore, A. R. (2010). COX-2 inhibition impaired resolution of chronic inflammation in a murine model of autoimmune arthritis. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,81890

Chicago Manual of Style (16th Edition):

Moore, Andrea Rossi. “COX-2 inhibition impaired resolution of chronic inflammation in a murine model of autoimmune arthritis.” 2010. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,81890.

MLA Handbook (7th Edition):

Moore, Andrea Rossi. “COX-2 inhibition impaired resolution of chronic inflammation in a murine model of autoimmune arthritis.” 2010. Web. 02 Apr 2020.

Vancouver:

Moore AR. COX-2 inhibition impaired resolution of chronic inflammation in a murine model of autoimmune arthritis. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,81890.

Council of Science Editors:

Moore AR. COX-2 inhibition impaired resolution of chronic inflammation in a murine model of autoimmune arthritis. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,81890


Temple University

2. Deliu, Elena. GPER/GPR30 Estrogen Receptor: A Target for Pain Modulation.

Degree: PhD, 2012, Temple University

Pharmacology

The G protein-coupled estrogen receptor GPER/GPER1, also known as GPR30, was originally cloned as an orphan receptor and later shown to be specifically activated… (more)

Subjects/Keywords: Pharmacology; Biochemistry; calcium imaging; estrogen; gpr30; pain; reactive oxygen species

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APA (6th Edition):

Deliu, E. (2012). GPER/GPR30 Estrogen Receptor: A Target for Pain Modulation. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,194862

Chicago Manual of Style (16th Edition):

Deliu, Elena. “GPER/GPR30 Estrogen Receptor: A Target for Pain Modulation.” 2012. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,194862.

MLA Handbook (7th Edition):

Deliu, Elena. “GPER/GPR30 Estrogen Receptor: A Target for Pain Modulation.” 2012. Web. 02 Apr 2020.

Vancouver:

Deliu E. GPER/GPR30 Estrogen Receptor: A Target for Pain Modulation. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,194862.

Council of Science Editors:

Deliu E. GPER/GPR30 Estrogen Receptor: A Target for Pain Modulation. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,194862


Temple University

3. Craige, Caryne. Regulation of Cocaine-induced Behaviors and Anxiety Produced by Cocaine Withdrawal through the Serotonin(2C) Receptor.

Degree: PhD, 2013, Temple University

Pharmacology

Cocaine is a powerfully active psychostimulant which exerts its effects through blockade of dopamine, serotonin and norepinephrine transporters and resultant increases in extracellular levels… (more)

Subjects/Keywords: Neurosciences; Pharmacology;

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APA (6th Edition):

Craige, C. (2013). Regulation of Cocaine-induced Behaviors and Anxiety Produced by Cocaine Withdrawal through the Serotonin(2C) Receptor. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,239587

Chicago Manual of Style (16th Edition):

Craige, Caryne. “Regulation of Cocaine-induced Behaviors and Anxiety Produced by Cocaine Withdrawal through the Serotonin(2C) Receptor.” 2013. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,239587.

MLA Handbook (7th Edition):

Craige, Caryne. “Regulation of Cocaine-induced Behaviors and Anxiety Produced by Cocaine Withdrawal through the Serotonin(2C) Receptor.” 2013. Web. 02 Apr 2020.

Vancouver:

Craige C. Regulation of Cocaine-induced Behaviors and Anxiety Produced by Cocaine Withdrawal through the Serotonin(2C) Receptor. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,239587.

Council of Science Editors:

Craige C. Regulation of Cocaine-induced Behaviors and Anxiety Produced by Cocaine Withdrawal through the Serotonin(2C) Receptor. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,239587


Temple University

4. Joshi, Yash. The Role of 5-Lipoxygenase in the Stress-Mediated Exacerbation of the Alzheimer's Disease Phenotype.

Degree: PhD, 2015, Temple University

Pharmacology

BACKGROUND: Alzheimer's disease (AD) is the most common aging-associated neurodegenerative dementia. Current epidemiological trends indicate that a rapidly aging population, in conjunction with the… (more)

Subjects/Keywords: Neurosciences; Biology; Biochemistry;

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APA (6th Edition):

Joshi, Y. (2015). The Role of 5-Lipoxygenase in the Stress-Mediated Exacerbation of the Alzheimer's Disease Phenotype. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,318405

Chicago Manual of Style (16th Edition):

Joshi, Yash. “The Role of 5-Lipoxygenase in the Stress-Mediated Exacerbation of the Alzheimer's Disease Phenotype.” 2015. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,318405.

MLA Handbook (7th Edition):

Joshi, Yash. “The Role of 5-Lipoxygenase in the Stress-Mediated Exacerbation of the Alzheimer's Disease Phenotype.” 2015. Web. 02 Apr 2020.

Vancouver:

Joshi Y. The Role of 5-Lipoxygenase in the Stress-Mediated Exacerbation of the Alzheimer's Disease Phenotype. [Internet] [Doctoral dissertation]. Temple University; 2015. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,318405.

Council of Science Editors:

Joshi Y. The Role of 5-Lipoxygenase in the Stress-Mediated Exacerbation of the Alzheimer's Disease Phenotype. [Doctoral Dissertation]. Temple University; 2015. Available from: http://digital.library.temple.edu/u?/p245801coll10,318405


Temple University

5. Nwaneshiudu, Chinwe A. Characterization of a functional role of the neurokinin-3 receptor in behavioral effects of cocaine.

Degree: PhD, 2011, Temple University

Pharmacology

The tachykinin NK-3 receptor is a G-protein coupled receptor activated by mammalian tachykinin neuropeptides, which can modulate dopaminergic neurotransmission, and alter dopamine-mediated behaviors. The… (more)

Subjects/Keywords: Health Sciences, Pharmacology; Behavioral Sciences; Biology, Neuroscience; CD-1; cocaine; dopamine; GSK3; NK-3; striatum

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APA (6th Edition):

Nwaneshiudu, C. A. (2011). Characterization of a functional role of the neurokinin-3 receptor in behavioral effects of cocaine. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,62614

Chicago Manual of Style (16th Edition):

Nwaneshiudu, Chinwe A. “Characterization of a functional role of the neurokinin-3 receptor in behavioral effects of cocaine.” 2011. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,62614.

MLA Handbook (7th Edition):

Nwaneshiudu, Chinwe A. “Characterization of a functional role of the neurokinin-3 receptor in behavioral effects of cocaine.” 2011. Web. 02 Apr 2020.

Vancouver:

Nwaneshiudu CA. Characterization of a functional role of the neurokinin-3 receptor in behavioral effects of cocaine. [Internet] [Doctoral dissertation]. Temple University; 2011. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,62614.

Council of Science Editors:

Nwaneshiudu CA. Characterization of a functional role of the neurokinin-3 receptor in behavioral effects of cocaine. [Doctoral Dissertation]. Temple University; 2011. Available from: http://digital.library.temple.edu/u?/p245801coll10,62614


Temple University

6. Inan, Saadet. Pharmacological and Neuroanatomical Analysis of GNTI-Induced Repetitive Behavior in Mice.

Degree: PhD, 2010, Temple University

Pharmacology

This thesis is comprised of two parts. In the first part, we investigated a) the pharmacology of GNTI, a selective kappa opioid receptor antagonist,… (more)

Subjects/Keywords: Health Sciences, Pharmacology; Neurobiology; GNTI; Itch; Kappa opioid receptor; Muscarinic receptor; Nalfurafine; Pain

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APA (6th Edition):

Inan, S. (2010). Pharmacological and Neuroanatomical Analysis of GNTI-Induced Repetitive Behavior in Mice. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,75332

Chicago Manual of Style (16th Edition):

Inan, Saadet. “Pharmacological and Neuroanatomical Analysis of GNTI-Induced Repetitive Behavior in Mice.” 2010. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,75332.

MLA Handbook (7th Edition):

Inan, Saadet. “Pharmacological and Neuroanatomical Analysis of GNTI-Induced Repetitive Behavior in Mice.” 2010. Web. 02 Apr 2020.

Vancouver:

Inan S. Pharmacological and Neuroanatomical Analysis of GNTI-Induced Repetitive Behavior in Mice. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,75332.

Council of Science Editors:

Inan S. Pharmacological and Neuroanatomical Analysis of GNTI-Induced Repetitive Behavior in Mice. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,75332


Temple University

7. Huang, Xiaofang. Functional study of amylin and regulation of amylin receptor.

Degree: PhD, 2010, Temple University

Pharmacology

Amylin, a 37 amino acid peptide secreted from pancreatic beta cells upon stimulation by meal/glucose, belongs to the family of the calcitonin or calcitonin… (more)

Subjects/Keywords: Pharmacology; amylin; antinociceptive; calcitonin receptor; RAMP; spinal cord

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APA (6th Edition):

Huang, X. (2010). Functional study of amylin and regulation of amylin receptor. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,114036

Chicago Manual of Style (16th Edition):

Huang, Xiaofang. “Functional study of amylin and regulation of amylin receptor.” 2010. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,114036.

MLA Handbook (7th Edition):

Huang, Xiaofang. “Functional study of amylin and regulation of amylin receptor.” 2010. Web. 02 Apr 2020.

Vancouver:

Huang X. Functional study of amylin and regulation of amylin receptor. [Internet] [Doctoral dissertation]. Temple University; 2010. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,114036.

Council of Science Editors:

Huang X. Functional study of amylin and regulation of amylin receptor. [Doctoral Dissertation]. Temple University; 2010. Available from: http://digital.library.temple.edu/u?/p245801coll10,114036


Temple University

8. Yang, Fan. Amylin mediates brainstem control of heart rate in the diving reflex.

Degree: PhD, 2012, Temple University

Pharmacology

Amylin, or islet amyloid polypeptide is a 37-amino acid member of the calcitonin peptide family. Amylin role in the brainstem and its function in… (more)

Subjects/Keywords: Pharmacology; Neurosciences; amylin; bradycardia; diving reflex; neuropeptide; nucleus ambiguus; trigeminal ganglion

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APA (6th Edition):

Yang, F. (2012). Amylin mediates brainstem control of heart rate in the diving reflex. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,193415

Chicago Manual of Style (16th Edition):

Yang, Fan. “Amylin mediates brainstem control of heart rate in the diving reflex.” 2012. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,193415.

MLA Handbook (7th Edition):

Yang, Fan. “Amylin mediates brainstem control of heart rate in the diving reflex.” 2012. Web. 02 Apr 2020.

Vancouver:

Yang F. Amylin mediates brainstem control of heart rate in the diving reflex. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,193415.

Council of Science Editors:

Yang F. Amylin mediates brainstem control of heart rate in the diving reflex. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,193415


Temple University

9. Albano, Jennifer Nicole. Localization of Human Prostaglandin E2 Receptors in Polarized Epithelial Cells.

Degree: PhD, 2008, Temple University

Pharmacology

The underlying mechanisms of protein sorting in polarized epithelial cells are poorly understood. Several studies have determined membrane targeting of G protein-coupled receptors (GPCRs)… (more)

Subjects/Keywords: Health Sciences, Pharmacology; Biology, Molecular

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APA (6th Edition):

Albano, J. N. (2008). Localization of Human Prostaglandin E2 Receptors in Polarized Epithelial Cells. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,17898

Chicago Manual of Style (16th Edition):

Albano, Jennifer Nicole. “Localization of Human Prostaglandin E2 Receptors in Polarized Epithelial Cells.” 2008. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,17898.

MLA Handbook (7th Edition):

Albano, Jennifer Nicole. “Localization of Human Prostaglandin E2 Receptors in Polarized Epithelial Cells.” 2008. Web. 02 Apr 2020.

Vancouver:

Albano JN. Localization of Human Prostaglandin E2 Receptors in Polarized Epithelial Cells. [Internet] [Doctoral dissertation]. Temple University; 2008. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,17898.

Council of Science Editors:

Albano JN. Localization of Human Prostaglandin E2 Receptors in Polarized Epithelial Cells. [Doctoral Dissertation]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,17898


Temple University

10. Soderman, Avery Rune. The Role of Mu Opioid Receptors in the Behavioral Effects of Cocaine.

Degree: PhD, 2008, Temple University

Pharmacology

Animal models have proven to be useful tools for modeling human neurochemical and behavioral responses to drugs of abuse, including cocaine. Cocaine is a… (more)

Subjects/Keywords: Health Sciences, Pharmacology; Biology, Neuroscience; cocaine; hyperlocomotion; Mu-opioid r; Nucleus Accumbens; reward

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APA (6th Edition):

Soderman, A. R. (2008). The Role of Mu Opioid Receptors in the Behavioral Effects of Cocaine. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,22825

Chicago Manual of Style (16th Edition):

Soderman, Avery Rune. “The Role of Mu Opioid Receptors in the Behavioral Effects of Cocaine.” 2008. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,22825.

MLA Handbook (7th Edition):

Soderman, Avery Rune. “The Role of Mu Opioid Receptors in the Behavioral Effects of Cocaine.” 2008. Web. 02 Apr 2020.

Vancouver:

Soderman AR. The Role of Mu Opioid Receptors in the Behavioral Effects of Cocaine. [Internet] [Doctoral dissertation]. Temple University; 2008. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,22825.

Council of Science Editors:

Soderman AR. The Role of Mu Opioid Receptors in the Behavioral Effects of Cocaine. [Doctoral Dissertation]. Temple University; 2008. Available from: http://digital.library.temple.edu/u?/p245801coll10,22825


Temple University

11. Trecki, Jordan. THE MODULATION OF THE MESOLIMBIC AND NIGROSTRIATAL DOPAMINE PATHWAYS BY CXCL12 AND CXCR4.

Degree: PhD, 2009, Temple University

Pharmacology

The role of chemokines in immune function is clearly established. Recent evidence suggests that these molecules also play an important role in the CNS… (more)

Subjects/Keywords: Health Sciences, Pharmacology; Biology, Neuroscience; behavior; cocaine; immunohistochemistry; intracerebral; locomotion; microdialysis

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APA (6th Edition):

Trecki, J. (2009). THE MODULATION OF THE MESOLIMBIC AND NIGROSTRIATAL DOPAMINE PATHWAYS BY CXCL12 AND CXCR4. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,57720

Chicago Manual of Style (16th Edition):

Trecki, Jordan. “THE MODULATION OF THE MESOLIMBIC AND NIGROSTRIATAL DOPAMINE PATHWAYS BY CXCL12 AND CXCR4.” 2009. Doctoral Dissertation, Temple University. Accessed April 02, 2020. http://digital.library.temple.edu/u?/p245801coll10,57720.

MLA Handbook (7th Edition):

Trecki, Jordan. “THE MODULATION OF THE MESOLIMBIC AND NIGROSTRIATAL DOPAMINE PATHWAYS BY CXCL12 AND CXCR4.” 2009. Web. 02 Apr 2020.

Vancouver:

Trecki J. THE MODULATION OF THE MESOLIMBIC AND NIGROSTRIATAL DOPAMINE PATHWAYS BY CXCL12 AND CXCR4. [Internet] [Doctoral dissertation]. Temple University; 2009. [cited 2020 Apr 02]. Available from: http://digital.library.temple.edu/u?/p245801coll10,57720.

Council of Science Editors:

Trecki J. THE MODULATION OF THE MESOLIMBIC AND NIGROSTRIATAL DOPAMINE PATHWAYS BY CXCL12 AND CXCR4. [Doctoral Dissertation]. Temple University; 2009. Available from: http://digital.library.temple.edu/u?/p245801coll10,57720

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