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You searched for +publisher:"Temple University" +contributor:("Abood, Mary Ellen"). Showing records 1 – 9 of 9 total matches.

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Temple University

1. Bagashev, Asen. MOLECULAR MECHANISM OF HIV-1 TAT INDUCED NEURONAL DYSFUNCTION.

Degree: PhD, 2014, Temple University

Cell Biology

In the early years of the AIDS epidemic, being infected with the virus that causes the disease was considered a virtual death sentence.… (more)

Subjects/Keywords: Neurosciences; Molecular biology; Virology

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APA (6th Edition):

Bagashev, A. (2014). MOLECULAR MECHANISM OF HIV-1 TAT INDUCED NEURONAL DYSFUNCTION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,255791

Chicago Manual of Style (16th Edition):

Bagashev, Asen. “MOLECULAR MECHANISM OF HIV-1 TAT INDUCED NEURONAL DYSFUNCTION.” 2014. Doctoral Dissertation, Temple University. Accessed October 26, 2020. http://digital.library.temple.edu/u?/p245801coll10,255791.

MLA Handbook (7th Edition):

Bagashev, Asen. “MOLECULAR MECHANISM OF HIV-1 TAT INDUCED NEURONAL DYSFUNCTION.” 2014. Web. 26 Oct 2020.

Vancouver:

Bagashev A. MOLECULAR MECHANISM OF HIV-1 TAT INDUCED NEURONAL DYSFUNCTION. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2020 Oct 26]. Available from: http://digital.library.temple.edu/u?/p245801coll10,255791.

Council of Science Editors:

Bagashev A. MOLECULAR MECHANISM OF HIV-1 TAT INDUCED NEURONAL DYSFUNCTION. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,255791


Temple University

2. Chiu, Yi-Ting. STUDIES ON NEURITE OUTGROWTH AND RECEPTOR PHOSPHORYLATION FOLLOWING KAPPA OPIOID RECEPTOR ACTIVATION.

Degree: PhD, 2016, Temple University

Pharmacology

Kappa opioid receptor (KOPR) is involved in many physiological functions and pharmacological responses such as analgesia, anti-pruritic effect, sedation, motor incoordination and aversion (Simonin… (more)

Subjects/Keywords: Pharmacology;

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APA (6th Edition):

Chiu, Y. (2016). STUDIES ON NEURITE OUTGROWTH AND RECEPTOR PHOSPHORYLATION FOLLOWING KAPPA OPIOID RECEPTOR ACTIVATION. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,384383

Chicago Manual of Style (16th Edition):

Chiu, Yi-Ting. “STUDIES ON NEURITE OUTGROWTH AND RECEPTOR PHOSPHORYLATION FOLLOWING KAPPA OPIOID RECEPTOR ACTIVATION.” 2016. Doctoral Dissertation, Temple University. Accessed October 26, 2020. http://digital.library.temple.edu/u?/p245801coll10,384383.

MLA Handbook (7th Edition):

Chiu, Yi-Ting. “STUDIES ON NEURITE OUTGROWTH AND RECEPTOR PHOSPHORYLATION FOLLOWING KAPPA OPIOID RECEPTOR ACTIVATION.” 2016. Web. 26 Oct 2020.

Vancouver:

Chiu Y. STUDIES ON NEURITE OUTGROWTH AND RECEPTOR PHOSPHORYLATION FOLLOWING KAPPA OPIOID RECEPTOR ACTIVATION. [Internet] [Doctoral dissertation]. Temple University; 2016. [cited 2020 Oct 26]. Available from: http://digital.library.temple.edu/u?/p245801coll10,384383.

Council of Science Editors:

Chiu Y. STUDIES ON NEURITE OUTGROWTH AND RECEPTOR PHOSPHORYLATION FOLLOWING KAPPA OPIOID RECEPTOR ACTIVATION. [Doctoral Dissertation]. Temple University; 2016. Available from: http://digital.library.temple.edu/u?/p245801coll10,384383


Temple University

3. Deliu, Elena. GPER/GPR30 Estrogen Receptor: A Target for Pain Modulation.

Degree: PhD, 2012, Temple University

Pharmacology

The G protein-coupled estrogen receptor GPER/GPER1, also known as GPR30, was originally cloned as an orphan receptor and later shown to be specifically activated… (more)

Subjects/Keywords: Pharmacology; Biochemistry; calcium imaging; estrogen; gpr30; pain; reactive oxygen species

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APA (6th Edition):

Deliu, E. (2012). GPER/GPR30 Estrogen Receptor: A Target for Pain Modulation. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,194862

Chicago Manual of Style (16th Edition):

Deliu, Elena. “GPER/GPR30 Estrogen Receptor: A Target for Pain Modulation.” 2012. Doctoral Dissertation, Temple University. Accessed October 26, 2020. http://digital.library.temple.edu/u?/p245801coll10,194862.

MLA Handbook (7th Edition):

Deliu, Elena. “GPER/GPR30 Estrogen Receptor: A Target for Pain Modulation.” 2012. Web. 26 Oct 2020.

Vancouver:

Deliu E. GPER/GPR30 Estrogen Receptor: A Target for Pain Modulation. [Internet] [Doctoral dissertation]. Temple University; 2012. [cited 2020 Oct 26]. Available from: http://digital.library.temple.edu/u?/p245801coll10,194862.

Council of Science Editors:

Deliu E. GPER/GPR30 Estrogen Receptor: A Target for Pain Modulation. [Doctoral Dissertation]. Temple University; 2012. Available from: http://digital.library.temple.edu/u?/p245801coll10,194862


Temple University

4. DiMattio, Kelly Marie. Studies on Ligands of the Kappa Opioid Receptor.

Degree: PhD, 2016, Temple University

Pharmacology

This thesis is comprised of three parts. In the first part, we investigated zyklophin, a novel selective short-acting kappa opioid receptor (KOPR) antagonist, and… (more)

Subjects/Keywords: Pharmacology; Biogeochemistry; Behavioral sciences;

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APA (6th Edition):

DiMattio, K. M. (2016). Studies on Ligands of the Kappa Opioid Receptor. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,334919

Chicago Manual of Style (16th Edition):

DiMattio, Kelly Marie. “Studies on Ligands of the Kappa Opioid Receptor.” 2016. Doctoral Dissertation, Temple University. Accessed October 26, 2020. http://digital.library.temple.edu/u?/p245801coll10,334919.

MLA Handbook (7th Edition):

DiMattio, Kelly Marie. “Studies on Ligands of the Kappa Opioid Receptor.” 2016. Web. 26 Oct 2020.

Vancouver:

DiMattio KM. Studies on Ligands of the Kappa Opioid Receptor. [Internet] [Doctoral dissertation]. Temple University; 2016. [cited 2020 Oct 26]. Available from: http://digital.library.temple.edu/u?/p245801coll10,334919.

Council of Science Editors:

DiMattio KM. Studies on Ligands of the Kappa Opioid Receptor. [Doctoral Dissertation]. Temple University; 2016. Available from: http://digital.library.temple.edu/u?/p245801coll10,334919


Temple University

5. Lunden, Jason Wesley. The serotonergic dorsal raphe nucleus in opiate dependence and stress-induced relapse.

Degree: PhD, 2013, Temple University

Cell Biology

Opioids are used for the clinical treatment of pain, but can lead to tolerance and addiction. In this project we examined the role… (more)

Subjects/Keywords: Neurosciences; Animal behavior; Cellular biology;

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APA (6th Edition):

Lunden, J. W. (2013). The serotonergic dorsal raphe nucleus in opiate dependence and stress-induced relapse. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,228054

Chicago Manual of Style (16th Edition):

Lunden, Jason Wesley. “The serotonergic dorsal raphe nucleus in opiate dependence and stress-induced relapse.” 2013. Doctoral Dissertation, Temple University. Accessed October 26, 2020. http://digital.library.temple.edu/u?/p245801coll10,228054.

MLA Handbook (7th Edition):

Lunden, Jason Wesley. “The serotonergic dorsal raphe nucleus in opiate dependence and stress-induced relapse.” 2013. Web. 26 Oct 2020.

Vancouver:

Lunden JW. The serotonergic dorsal raphe nucleus in opiate dependence and stress-induced relapse. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2020 Oct 26]. Available from: http://digital.library.temple.edu/u?/p245801coll10,228054.

Council of Science Editors:

Lunden JW. The serotonergic dorsal raphe nucleus in opiate dependence and stress-induced relapse. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,228054


Temple University

6. Marcu, Jahan Phillip. Novel Insights into CB1 Receptor Signaling and the Anabolic Role of Cannabinoid Receptors in Bone.

Degree: PhD, 2013, Temple University

Cell Biology

Activation of the CB1 receptor is modulated by aspartate residue D2.63176 in transmembrane helix (TMH) II. Interestingly, D2.63 does not affect the affinity… (more)

Subjects/Keywords: Molecular biology; Pharmacology; Biochemistry;

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APA (6th Edition):

Marcu, J. P. (2013). Novel Insights into CB1 Receptor Signaling and the Anabolic Role of Cannabinoid Receptors in Bone. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,233543

Chicago Manual of Style (16th Edition):

Marcu, Jahan Phillip. “Novel Insights into CB1 Receptor Signaling and the Anabolic Role of Cannabinoid Receptors in Bone.” 2013. Doctoral Dissertation, Temple University. Accessed October 26, 2020. http://digital.library.temple.edu/u?/p245801coll10,233543.

MLA Handbook (7th Edition):

Marcu, Jahan Phillip. “Novel Insights into CB1 Receptor Signaling and the Anabolic Role of Cannabinoid Receptors in Bone.” 2013. Web. 26 Oct 2020.

Vancouver:

Marcu JP. Novel Insights into CB1 Receptor Signaling and the Anabolic Role of Cannabinoid Receptors in Bone. [Internet] [Doctoral dissertation]. Temple University; 2013. [cited 2020 Oct 26]. Available from: http://digital.library.temple.edu/u?/p245801coll10,233543.

Council of Science Editors:

Marcu JP. Novel Insights into CB1 Receptor Signaling and the Anabolic Role of Cannabinoid Receptors in Bone. [Doctoral Dissertation]. Temple University; 2013. Available from: http://digital.library.temple.edu/u?/p245801coll10,233543


Temple University

7. Massicotte, Vicky S. THE EFFECTS OF OVERUSE ON CELLULAR, MOLECULAR AND MORPHOMETRIC BONE HOMEOSTASIS IN A VOLUNTARY REPETITIVE STRAIN INJURY RAT MODEL.

Degree: PhD, 2014, Temple University

Cell Biology

Injuries of the hands and wrist are prevalent in many occupations requiring repetitive tasks and may be further aggravated by advancing age; these… (more)

Subjects/Keywords: Cellular biology; Occupational health;

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APA (6th Edition):

Massicotte, V. S. (2014). THE EFFECTS OF OVERUSE ON CELLULAR, MOLECULAR AND MORPHOMETRIC BONE HOMEOSTASIS IN A VOLUNTARY REPETITIVE STRAIN INJURY RAT MODEL. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,305196

Chicago Manual of Style (16th Edition):

Massicotte, Vicky S. “THE EFFECTS OF OVERUSE ON CELLULAR, MOLECULAR AND MORPHOMETRIC BONE HOMEOSTASIS IN A VOLUNTARY REPETITIVE STRAIN INJURY RAT MODEL.” 2014. Doctoral Dissertation, Temple University. Accessed October 26, 2020. http://digital.library.temple.edu/u?/p245801coll10,305196.

MLA Handbook (7th Edition):

Massicotte, Vicky S. “THE EFFECTS OF OVERUSE ON CELLULAR, MOLECULAR AND MORPHOMETRIC BONE HOMEOSTASIS IN A VOLUNTARY REPETITIVE STRAIN INJURY RAT MODEL.” 2014. Web. 26 Oct 2020.

Vancouver:

Massicotte VS. THE EFFECTS OF OVERUSE ON CELLULAR, MOLECULAR AND MORPHOMETRIC BONE HOMEOSTASIS IN A VOLUNTARY REPETITIVE STRAIN INJURY RAT MODEL. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2020 Oct 26]. Available from: http://digital.library.temple.edu/u?/p245801coll10,305196.

Council of Science Editors:

Massicotte VS. THE EFFECTS OF OVERUSE ON CELLULAR, MOLECULAR AND MORPHOMETRIC BONE HOMEOSTASIS IN A VOLUNTARY REPETITIVE STRAIN INJURY RAT MODEL. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,305196


Temple University

8. Michael, James. Regulation of Ras signaling and oncogenesis by plasma membrane microdomains.

Degree: PhD, 2016, Temple University

Cell Biology

In this study, we assessed the contributions of plasma membrane (PM) microdomain targeting to the functions of H-Ras and R-Ras. These paralogues have… (more)

Subjects/Keywords: Cellular biology

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APA (6th Edition):

Michael, J. (2016). Regulation of Ras signaling and oncogenesis by plasma membrane microdomains. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,377230

Chicago Manual of Style (16th Edition):

Michael, James. “Regulation of Ras signaling and oncogenesis by plasma membrane microdomains.” 2016. Doctoral Dissertation, Temple University. Accessed October 26, 2020. http://digital.library.temple.edu/u?/p245801coll10,377230.

MLA Handbook (7th Edition):

Michael, James. “Regulation of Ras signaling and oncogenesis by plasma membrane microdomains.” 2016. Web. 26 Oct 2020.

Vancouver:

Michael J. Regulation of Ras signaling and oncogenesis by plasma membrane microdomains. [Internet] [Doctoral dissertation]. Temple University; 2016. [cited 2020 Oct 26]. Available from: http://digital.library.temple.edu/u?/p245801coll10,377230.

Council of Science Editors:

Michael J. Regulation of Ras signaling and oncogenesis by plasma membrane microdomains. [Doctoral Dissertation]. Temple University; 2016. Available from: http://digital.library.temple.edu/u?/p245801coll10,377230


Temple University

9. Mundy, Christina Maria. The Interaction Between Connective Tissue Growth Factor and Bone Morphogenetic Protein-2 During Osteoblast Differentiation and Function.

Degree: PhD, 2014, Temple University

Cell Biology

Connective tissue growth factor (CTGF/CCN2) and bone morphogenetic protein (BMP)-2 are both produced and secreted by osteoblasts. Both proteins have been shown to… (more)

Subjects/Keywords: Cellular biology;

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APA (6th Edition):

Mundy, C. M. (2014). The Interaction Between Connective Tissue Growth Factor and Bone Morphogenetic Protein-2 During Osteoblast Differentiation and Function. (Doctoral Dissertation). Temple University. Retrieved from http://digital.library.temple.edu/u?/p245801coll10,269581

Chicago Manual of Style (16th Edition):

Mundy, Christina Maria. “The Interaction Between Connective Tissue Growth Factor and Bone Morphogenetic Protein-2 During Osteoblast Differentiation and Function.” 2014. Doctoral Dissertation, Temple University. Accessed October 26, 2020. http://digital.library.temple.edu/u?/p245801coll10,269581.

MLA Handbook (7th Edition):

Mundy, Christina Maria. “The Interaction Between Connective Tissue Growth Factor and Bone Morphogenetic Protein-2 During Osteoblast Differentiation and Function.” 2014. Web. 26 Oct 2020.

Vancouver:

Mundy CM. The Interaction Between Connective Tissue Growth Factor and Bone Morphogenetic Protein-2 During Osteoblast Differentiation and Function. [Internet] [Doctoral dissertation]. Temple University; 2014. [cited 2020 Oct 26]. Available from: http://digital.library.temple.edu/u?/p245801coll10,269581.

Council of Science Editors:

Mundy CM. The Interaction Between Connective Tissue Growth Factor and Bone Morphogenetic Protein-2 During Osteoblast Differentiation and Function. [Doctoral Dissertation]. Temple University; 2014. Available from: http://digital.library.temple.edu/u?/p245801coll10,269581

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