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You searched for +publisher:"Rutgers University" +contributor:("Zhou, Renping"). Showing records 1 – 30 of 32 total matches.

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Rutgers University

1. Lalani, Almin Iqbalali, 1986-. In vivo functions of myeloid cell TRAF3.

Degree: PhD, Pharmacology, Cellular and Molecular, 2017, Rutgers University

 Tumor necrosis factor receptor-associated factor 3 (TRAF3) is a member of the TRAF family of cytoplasmic adaptor proteins that is ubiquitously expressed in various cell… (more)

Subjects/Keywords: Natural immunity; Immune response

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APA (6th Edition):

Lalani, Almin Iqbalali, 1. (2017). In vivo functions of myeloid cell TRAF3. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/52215/

Chicago Manual of Style (16th Edition):

Lalani, Almin Iqbalali, 1986-. “In vivo functions of myeloid cell TRAF3.” 2017. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/52215/.

MLA Handbook (7th Edition):

Lalani, Almin Iqbalali, 1986-. “In vivo functions of myeloid cell TRAF3.” 2017. Web. 09 May 2021.

Vancouver:

Lalani, Almin Iqbalali 1. In vivo functions of myeloid cell TRAF3. [Internet] [Doctoral dissertation]. Rutgers University; 2017. [cited 2021 May 09]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/52215/.

Council of Science Editors:

Lalani, Almin Iqbalali 1. In vivo functions of myeloid cell TRAF3. [Doctoral Dissertation]. Rutgers University; 2017. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/52215/


Rutgers University

2. Zhang, Le, 1987-. Alleviation of metabolic syndrome by green tea polyphenol EGCG: molecular mechanism involving amp-activated protein kinase.

Degree: MS, Pharmaceutical Science, 2016, Rutgers University

 Metabolic syndrome (MetS) is a clustering of several cardiometabolic risk factors, including abdominal obesity, hyperglycemia, dyslipidemia and elevated blood pressure. More and more attention has… (more)

Subjects/Keywords: Metabolism – Disorders; Green tea – Health aspects

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APA (6th Edition):

Zhang, Le, 1. (2016). Alleviation of metabolic syndrome by green tea polyphenol EGCG: molecular mechanism involving amp-activated protein kinase. (Masters Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/50498/

Chicago Manual of Style (16th Edition):

Zhang, Le, 1987-. “Alleviation of metabolic syndrome by green tea polyphenol EGCG: molecular mechanism involving amp-activated protein kinase.” 2016. Masters Thesis, Rutgers University. Accessed May 09, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/50498/.

MLA Handbook (7th Edition):

Zhang, Le, 1987-. “Alleviation of metabolic syndrome by green tea polyphenol EGCG: molecular mechanism involving amp-activated protein kinase.” 2016. Web. 09 May 2021.

Vancouver:

Zhang, Le 1. Alleviation of metabolic syndrome by green tea polyphenol EGCG: molecular mechanism involving amp-activated protein kinase. [Internet] [Masters thesis]. Rutgers University; 2016. [cited 2021 May 09]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/50498/.

Council of Science Editors:

Zhang, Le 1. Alleviation of metabolic syndrome by green tea polyphenol EGCG: molecular mechanism involving amp-activated protein kinase. [Masters Thesis]. Rutgers University; 2016. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/50498/


Rutgers University

3. Weig, Blair Christopher, 1962-. Response of neural stem cells to trimethyltin-induced hippocampal injury.

Degree: PhD, Toxicology, 2016, Rutgers University

 The mammalian brain retains the ability to produce new neurons and glia throughout life. The retention of stem cells in the adult brain is phylogenetically… (more)

Subjects/Keywords: Developmental neurobiology; Nervous system

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APA (6th Edition):

Weig, Blair Christopher, 1. (2016). Response of neural stem cells to trimethyltin-induced hippocampal injury. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/51497/

Chicago Manual of Style (16th Edition):

Weig, Blair Christopher, 1962-. “Response of neural stem cells to trimethyltin-induced hippocampal injury.” 2016. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/51497/.

MLA Handbook (7th Edition):

Weig, Blair Christopher, 1962-. “Response of neural stem cells to trimethyltin-induced hippocampal injury.” 2016. Web. 09 May 2021.

Vancouver:

Weig, Blair Christopher 1. Response of neural stem cells to trimethyltin-induced hippocampal injury. [Internet] [Doctoral dissertation]. Rutgers University; 2016. [cited 2021 May 09]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/51497/.

Council of Science Editors:

Weig, Blair Christopher 1. Response of neural stem cells to trimethyltin-induced hippocampal injury. [Doctoral Dissertation]. Rutgers University; 2016. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/51497/


Rutgers University

4. Hader, Anna T., 1980-. EphA/ephrin-A signaling in excitatory synapse development.

Degree: PhD, Neuroscience, 2016, Rutgers University

Interactions between EphA receptors and ephrin-A ligands have been implicated in the regulation of postsynaptic spine density, astrocytic glutamate transport, and synaptic plasticity. Nonetheless, many… (more)

Subjects/Keywords: Protein-tyrosine kinase – Receptors; Synapses; Excitory amino acids

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APA (6th Edition):

Hader, Anna T., 1. (2016). EphA/ephrin-A signaling in excitatory synapse development. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/49974/

Chicago Manual of Style (16th Edition):

Hader, Anna T., 1980-. “EphA/ephrin-A signaling in excitatory synapse development.” 2016. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/49974/.

MLA Handbook (7th Edition):

Hader, Anna T., 1980-. “EphA/ephrin-A signaling in excitatory synapse development.” 2016. Web. 09 May 2021.

Vancouver:

Hader, Anna T. 1. EphA/ephrin-A signaling in excitatory synapse development. [Internet] [Doctoral dissertation]. Rutgers University; 2016. [cited 2021 May 09]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/49974/.

Council of Science Editors:

Hader, Anna T. 1. EphA/ephrin-A signaling in excitatory synapse development. [Doctoral Dissertation]. Rutgers University; 2016. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/49974/


Rutgers University

5. Sheleg, Michal, 1983-. Regulation of mouse behavior by ephrin-a5.

Degree: PhD, Toxicology, 2015, Rutgers University

Social behaviors in mammals are regulated by core neural circuits that respond to stimuli from the environment. These circuits are formed by guidance molecules and… (more)

Subjects/Keywords: Receptor-ligand complexes; Protein-tyrosine kinase

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APA (6th Edition):

Sheleg, Michal, 1. (2015). Regulation of mouse behavior by ephrin-a5. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/46436/

Chicago Manual of Style (16th Edition):

Sheleg, Michal, 1983-. “Regulation of mouse behavior by ephrin-a5.” 2015. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/46436/.

MLA Handbook (7th Edition):

Sheleg, Michal, 1983-. “Regulation of mouse behavior by ephrin-a5.” 2015. Web. 09 May 2021.

Vancouver:

Sheleg, Michal 1. Regulation of mouse behavior by ephrin-a5. [Internet] [Doctoral dissertation]. Rutgers University; 2015. [cited 2021 May 09]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/46436/.

Council of Science Editors:

Sheleg, Michal 1. Regulation of mouse behavior by ephrin-a5. [Doctoral Dissertation]. Rutgers University; 2015. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/46436/


Rutgers University

6. Vail, Gwyndolin M., 1991-. The neuroendocrine and physiological impact of organophosphate flame retardants on energy homeostasis in adult mice.

Degree: PhD, Oraganophosphate flame retardants, 2020, Rutgers University

The maintenance of energy homeostasis is an integral endocrine-mediated function and is centrally coordinated through hypothalamic melanocortin circuitry. Two nuclear receptors that regulate energy homeostasis… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Vail, Gwyndolin M., 1. (2020). The neuroendocrine and physiological impact of organophosphate flame retardants on energy homeostasis in adult mice. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/64892/

Chicago Manual of Style (16th Edition):

Vail, Gwyndolin M., 1991-. “The neuroendocrine and physiological impact of organophosphate flame retardants on energy homeostasis in adult mice.” 2020. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/64892/.

MLA Handbook (7th Edition):

Vail, Gwyndolin M., 1991-. “The neuroendocrine and physiological impact of organophosphate flame retardants on energy homeostasis in adult mice.” 2020. Web. 09 May 2021.

Vancouver:

Vail, Gwyndolin M. 1. The neuroendocrine and physiological impact of organophosphate flame retardants on energy homeostasis in adult mice. [Internet] [Doctoral dissertation]. Rutgers University; 2020. [cited 2021 May 09]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/64892/.

Council of Science Editors:

Vail, Gwyndolin M. 1. The neuroendocrine and physiological impact of organophosphate flame retardants on energy homeostasis in adult mice. [Doctoral Dissertation]. Rutgers University; 2020. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/64892/


Rutgers University

7. Omelchenko, Anton. Development of traumatic brain injury therapeutics.

Degree: PhD, Traumatic brain injury, 2021, Rutgers University

Traumatic brain injury (TBI) is one of the leading causes of death and disability in the world, and there are currently no available therapies to… (more)

Subjects/Keywords: Brain  – Wounds and injuries; Neuroscience

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APA (6th Edition):

Omelchenko, A. (2021). Development of traumatic brain injury therapeutics. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/65302/

Chicago Manual of Style (16th Edition):

Omelchenko, Anton. “Development of traumatic brain injury therapeutics.” 2021. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/65302/.

MLA Handbook (7th Edition):

Omelchenko, Anton. “Development of traumatic brain injury therapeutics.” 2021. Web. 09 May 2021.

Vancouver:

Omelchenko A. Development of traumatic brain injury therapeutics. [Internet] [Doctoral dissertation]. Rutgers University; 2021. [cited 2021 May 09]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/65302/.

Council of Science Editors:

Omelchenko A. Development of traumatic brain injury therapeutics. [Doctoral Dissertation]. Rutgers University; 2021. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/65302/


Rutgers University

8. Prem, Smrithi. Autism human neural precursor cells exhibit common defects in neurite outgrowth, cell migration, and mTOR signaling.

Degree: PhD, Neuroscience, 2018, Rutgers University

 Autism spectrum disorders (ASD) are a group of developmental disorders characterized by deficits in social interaction and communication and the presence of repetitive and restrictive… (more)

Subjects/Keywords: Autism spectrum disorders; Neurons

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APA (6th Edition):

Prem, S. (2018). Autism human neural precursor cells exhibit common defects in neurite outgrowth, cell migration, and mTOR signaling. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/59193/

Chicago Manual of Style (16th Edition):

Prem, Smrithi. “Autism human neural precursor cells exhibit common defects in neurite outgrowth, cell migration, and mTOR signaling.” 2018. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/59193/.

MLA Handbook (7th Edition):

Prem, Smrithi. “Autism human neural precursor cells exhibit common defects in neurite outgrowth, cell migration, and mTOR signaling.” 2018. Web. 09 May 2021.

Vancouver:

Prem S. Autism human neural precursor cells exhibit common defects in neurite outgrowth, cell migration, and mTOR signaling. [Internet] [Doctoral dissertation]. Rutgers University; 2018. [cited 2021 May 09]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/59193/.

Council of Science Editors:

Prem S. Autism human neural precursor cells exhibit common defects in neurite outgrowth, cell migration, and mTOR signaling. [Doctoral Dissertation]. Rutgers University; 2018. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/59193/


Rutgers University

9. Saitta, Kyle S., 1989-. The role of the group I metabotropic glutamate receptor agonist, CHPG, in oligodendrocyte regeneration and repair following a cuprizone-induced lesion.

Degree: PhD, BDNF, 2020, Rutgers University

Demyelinating diseases or demyelination in response to toxic agents can be debilitating to patients and can greatly impair the quality of life. Therefore, new therapeutic… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Saitta, Kyle S., 1. (2020). The role of the group I metabotropic glutamate receptor agonist, CHPG, in oligodendrocyte regeneration and repair following a cuprizone-induced lesion. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/64381/

Chicago Manual of Style (16th Edition):

Saitta, Kyle S., 1989-. “The role of the group I metabotropic glutamate receptor agonist, CHPG, in oligodendrocyte regeneration and repair following a cuprizone-induced lesion.” 2020. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/64381/.

MLA Handbook (7th Edition):

Saitta, Kyle S., 1989-. “The role of the group I metabotropic glutamate receptor agonist, CHPG, in oligodendrocyte regeneration and repair following a cuprizone-induced lesion.” 2020. Web. 09 May 2021.

Vancouver:

Saitta, Kyle S. 1. The role of the group I metabotropic glutamate receptor agonist, CHPG, in oligodendrocyte regeneration and repair following a cuprizone-induced lesion. [Internet] [Doctoral dissertation]. Rutgers University; 2020. [cited 2021 May 09]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/64381/.

Council of Science Editors:

Saitta, Kyle S. 1. The role of the group I metabotropic glutamate receptor agonist, CHPG, in oligodendrocyte regeneration and repair following a cuprizone-induced lesion. [Doctoral Dissertation]. Rutgers University; 2020. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/64381/


Rutgers University

10. Shah, Raj D. Role of GRM1 in altering glutamate metabolism and bioavailability.

Degree: PhD, Glutamate, 2020, Rutgers University

 Altered metabolic activity has been implicated in several types of cancer including malignant melanoma. Previously, we have illustrated the role of a neuronal receptor, metabotropic… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Shah, R. D. (2020). Role of GRM1 in altering glutamate metabolism and bioavailability. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/64382/

Chicago Manual of Style (16th Edition):

Shah, Raj D. “Role of GRM1 in altering glutamate metabolism and bioavailability.” 2020. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/64382/.

MLA Handbook (7th Edition):

Shah, Raj D. “Role of GRM1 in altering glutamate metabolism and bioavailability.” 2020. Web. 09 May 2021.

Vancouver:

Shah RD. Role of GRM1 in altering glutamate metabolism and bioavailability. [Internet] [Doctoral dissertation]. Rutgers University; 2020. [cited 2021 May 09]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/64382/.

Council of Science Editors:

Shah RD. Role of GRM1 in altering glutamate metabolism and bioavailability. [Doctoral Dissertation]. Rutgers University; 2020. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/64382/

11. Qin, Mei. The actions of IGFBPs on growth and glucose metabolism in transgenic mouse models.

Degree: MS, Neuroscience, 2010, Rutgers University

 The roles of insulin/IGF signaling in growth control and glucose metabolism have been extensively studied. To date, the growth inhibitory effects of IGFBPs and impaired… (more)

Subjects/Keywords: Glucose – Metabolism; Insulin – Receptors; Repressors, Genetic; Mice – Growth; Mice as laboratory animals

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APA (6th Edition):

Qin, M. (2010). The actions of IGFBPs on growth and glucose metabolism in transgenic mouse models. (Masters Thesis). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000056728

Chicago Manual of Style (16th Edition):

Qin, Mei. “The actions of IGFBPs on growth and glucose metabolism in transgenic mouse models.” 2010. Masters Thesis, Rutgers University. Accessed May 09, 2021. http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000056728.

MLA Handbook (7th Edition):

Qin, Mei. “The actions of IGFBPs on growth and glucose metabolism in transgenic mouse models.” 2010. Web. 09 May 2021.

Vancouver:

Qin M. The actions of IGFBPs on growth and glucose metabolism in transgenic mouse models. [Internet] [Masters thesis]. Rutgers University; 2010. [cited 2021 May 09]. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000056728.

Council of Science Editors:

Qin M. The actions of IGFBPs on growth and glucose metabolism in transgenic mouse models. [Masters Thesis]. Rutgers University; 2010. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000056728

12. Barve, Avantika. Prostate cancer chemoprevention by dietary phytochemicals:.

Degree: PhD, Pharmaceutical Science, 2009, Rutgers University

Prostate cancer is one of the most frequent diagnosed forms of cancer among men in the United States. It displays considerable clinical, morphological and biological… (more)

Subjects/Keywords: Prostate – Cancer; Cancer – Chemoprevention; Cancer – Diet therapy

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APA (6th Edition):

Barve, A. (2009). Prostate cancer chemoprevention by dietary phytochemicals:. (Doctoral Dissertation). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000050491

Chicago Manual of Style (16th Edition):

Barve, Avantika. “Prostate cancer chemoprevention by dietary phytochemicals:.” 2009. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000050491.

MLA Handbook (7th Edition):

Barve, Avantika. “Prostate cancer chemoprevention by dietary phytochemicals:.” 2009. Web. 09 May 2021.

Vancouver:

Barve A. Prostate cancer chemoprevention by dietary phytochemicals:. [Internet] [Doctoral dissertation]. Rutgers University; 2009. [cited 2021 May 09]. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000050491.

Council of Science Editors:

Barve A. Prostate cancer chemoprevention by dietary phytochemicals:. [Doctoral Dissertation]. Rutgers University; 2009. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000050491

13. Tzatzalos, Evangeline, 1983-. A regulatory element for interneuron progenitors in the developing vertebrate central nervous system.

Degree: Biomedical Engineering, 2012, Rutgers University

Subjects/Keywords: Nerve tissue proteins; Nerve growth factor; Nervous system – Growth

…Neuroscience at Rutgers University. Heterozygous mutants reeler+/- were crossed with Notch1CR2-βGP… 

Page 1 Page 2 Page 3 Page 4 Page 5 Page 6 Page 7 Sample image

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APA (6th Edition):

Tzatzalos, Evangeline, 1. (2012). A regulatory element for interneuron progenitors in the developing vertebrate central nervous system. (Thesis). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000065283

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tzatzalos, Evangeline, 1983-. “A regulatory element for interneuron progenitors in the developing vertebrate central nervous system.” 2012. Thesis, Rutgers University. Accessed May 09, 2021. http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000065283.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tzatzalos, Evangeline, 1983-. “A regulatory element for interneuron progenitors in the developing vertebrate central nervous system.” 2012. Web. 09 May 2021.

Vancouver:

Tzatzalos, Evangeline 1. A regulatory element for interneuron progenitors in the developing vertebrate central nervous system. [Internet] [Thesis]. Rutgers University; 2012. [cited 2021 May 09]. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000065283.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tzatzalos, Evangeline 1. A regulatory element for interneuron progenitors in the developing vertebrate central nervous system. [Thesis]. Rutgers University; 2012. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000065283

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

14. Rathod, Trushar, 1979-. Generation of anti-tumorigenic MET receptor variants by splicing interference.

Degree: PhD, Pharmacology, Cellular and Molecular, 2019, Rutgers University

Aberrant activation of MET, a receptor tyrosine kinase (RTK), leads to tumor growth, invasion and metastasis and is implicated in multiple types of cancers. Hence,… (more)

Subjects/Keywords: Hepatocyte growth factor  – Receptors

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APA (6th Edition):

Rathod, Trushar, 1. (2019). Generation of anti-tumorigenic MET receptor variants by splicing interference. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/60961/

Chicago Manual of Style (16th Edition):

Rathod, Trushar, 1979-. “Generation of anti-tumorigenic MET receptor variants by splicing interference.” 2019. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/60961/.

MLA Handbook (7th Edition):

Rathod, Trushar, 1979-. “Generation of anti-tumorigenic MET receptor variants by splicing interference.” 2019. Web. 09 May 2021.

Vancouver:

Rathod, Trushar 1. Generation of anti-tumorigenic MET receptor variants by splicing interference. [Internet] [Doctoral dissertation]. Rutgers University; 2019. [cited 2021 May 09]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/60961/.

Council of Science Editors:

Rathod, Trushar 1. Generation of anti-tumorigenic MET receptor variants by splicing interference. [Doctoral Dissertation]. Rutgers University; 2019. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/60961/

15. Voyiadjis, Andrewv George, 1981-. An improved model for neurite guidance validated to a novel micropatterned In vitro surrogate.

Degree: PhD, Biomedical Engineering, 2011, Rutgers University

Recent research on repair mechanisms in injured spinal cords focuses on the regeneration of neurites across the injury site. While integral to developing a therapy,… (more)

Subjects/Keywords: Nerves, Peripheral—Wounds and injuries; Spinal nerves; Spinal cord—Regeneration

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APA (6th Edition):

Voyiadjis, Andrewv George, 1. (2011). An improved model for neurite guidance validated to a novel micropatterned In vitro surrogate. (Doctoral Dissertation). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000061533

Chicago Manual of Style (16th Edition):

Voyiadjis, Andrewv George, 1981-. “An improved model for neurite guidance validated to a novel micropatterned In vitro surrogate.” 2011. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000061533.

MLA Handbook (7th Edition):

Voyiadjis, Andrewv George, 1981-. “An improved model for neurite guidance validated to a novel micropatterned In vitro surrogate.” 2011. Web. 09 May 2021.

Vancouver:

Voyiadjis, Andrewv George 1. An improved model for neurite guidance validated to a novel micropatterned In vitro surrogate. [Internet] [Doctoral dissertation]. Rutgers University; 2011. [cited 2021 May 09]. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000061533.

Council of Science Editors:

Voyiadjis, Andrewv George 1. An improved model for neurite guidance validated to a novel micropatterned In vitro surrogate. [Doctoral Dissertation]. Rutgers University; 2011. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000061533

16. Tseng, Chia-Yi, 1980-. The roles of postsynaptic density-95 protein (PSD-95) and its interactor, cypin, in the regulation of microtubule organization, dendrite morphology, and neuroprotection.

Degree: Neuroscience, 2011, Rutgers University

Subjects/Keywords: Dendrites; Nerve tissue proteins; Synapses

Rutgers University Electron Imaging Facility, Division of Life Sciences. Imaging and analysis… …microscope (Rutgers University Electron Imaging Facility, Division of Life Sciences)… 

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APA (6th Edition):

Tseng, Chia-Yi, 1. (2011). The roles of postsynaptic density-95 protein (PSD-95) and its interactor, cypin, in the regulation of microtubule organization, dendrite morphology, and neuroprotection. (Thesis). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000063681

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tseng, Chia-Yi, 1980-. “The roles of postsynaptic density-95 protein (PSD-95) and its interactor, cypin, in the regulation of microtubule organization, dendrite morphology, and neuroprotection.” 2011. Thesis, Rutgers University. Accessed May 09, 2021. http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000063681.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tseng, Chia-Yi, 1980-. “The roles of postsynaptic density-95 protein (PSD-95) and its interactor, cypin, in the regulation of microtubule organization, dendrite morphology, and neuroprotection.” 2011. Web. 09 May 2021.

Vancouver:

Tseng, Chia-Yi 1. The roles of postsynaptic density-95 protein (PSD-95) and its interactor, cypin, in the regulation of microtubule organization, dendrite morphology, and neuroprotection. [Internet] [Thesis]. Rutgers University; 2011. [cited 2021 May 09]. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000063681.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tseng, Chia-Yi 1. The roles of postsynaptic density-95 protein (PSD-95) and its interactor, cypin, in the regulation of microtubule organization, dendrite morphology, and neuroprotection. [Thesis]. Rutgers University; 2011. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000063681

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

17. Kim, Hyun Jong, 1972. The role of SEPT2 on neuronal development:.

Degree: PhD, Cell and Developmental Biology, 2009, Rutgers University

Septins constitute a family of evolutionarily conserved proteins that were first discovered for their involvement in cell division. Recently, they are also found to be… (more)

Subjects/Keywords: Septins; Developmental neurobiology

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APA (6th Edition):

Kim, Hyun Jong, 1. (2009). The role of SEPT2 on neuronal development:. (Doctoral Dissertation). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000051363

Chicago Manual of Style (16th Edition):

Kim, Hyun Jong, 1972. “The role of SEPT2 on neuronal development:.” 2009. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000051363.

MLA Handbook (7th Edition):

Kim, Hyun Jong, 1972. “The role of SEPT2 on neuronal development:.” 2009. Web. 09 May 2021.

Vancouver:

Kim, Hyun Jong 1. The role of SEPT2 on neuronal development:. [Internet] [Doctoral dissertation]. Rutgers University; 2009. [cited 2021 May 09]. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000051363.

Council of Science Editors:

Kim, Hyun Jong 1. The role of SEPT2 on neuronal development:. [Doctoral Dissertation]. Rutgers University; 2009. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000051363

18. Swiatkowski, Przemyslaw, 1987-. Traumatic brain injury: novel targets for potential therapies.

Degree: PhD, Pharmacology, Cellular and Molecular, 2018, Rutgers University

Traumatic brain injury (TBI) induces severe neurological damage leading to significant deficits in learning and memory and poor quality of life. It is a pathology… (more)

Subjects/Keywords: Brain – Wounds and injuries – Treatment

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APA (6th Edition):

Swiatkowski, Przemyslaw, 1. (2018). Traumatic brain injury: novel targets for potential therapies. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/56146/

Chicago Manual of Style (16th Edition):

Swiatkowski, Przemyslaw, 1987-. “Traumatic brain injury: novel targets for potential therapies.” 2018. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/56146/.

MLA Handbook (7th Edition):

Swiatkowski, Przemyslaw, 1987-. “Traumatic brain injury: novel targets for potential therapies.” 2018. Web. 09 May 2021.

Vancouver:

Swiatkowski, Przemyslaw 1. Traumatic brain injury: novel targets for potential therapies. [Internet] [Doctoral dissertation]. Rutgers University; 2018. [cited 2021 May 09]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/56146/.

Council of Science Editors:

Swiatkowski, Przemyslaw 1. Traumatic brain injury: novel targets for potential therapies. [Doctoral Dissertation]. Rutgers University; 2018. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/56146/

19. Yu, Qili. Genetic factors influencing oligodendrocyte demyelination.

Degree: PhD, Neuroscience, 2018, Rutgers University

Oligodendrocytes are an important cell type in the central nervous system (CNS). Their most prominent function in the brain is to provide axonal processes of… (more)

Subjects/Keywords: Multiple sclerosis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yu, Q. (2018). Genetic factors influencing oligodendrocyte demyelination. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/56157/

Chicago Manual of Style (16th Edition):

Yu, Qili. “Genetic factors influencing oligodendrocyte demyelination.” 2018. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/56157/.

MLA Handbook (7th Edition):

Yu, Qili. “Genetic factors influencing oligodendrocyte demyelination.” 2018. Web. 09 May 2021.

Vancouver:

Yu Q. Genetic factors influencing oligodendrocyte demyelination. [Internet] [Doctoral dissertation]. Rutgers University; 2018. [cited 2021 May 09]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/56157/.

Council of Science Editors:

Yu Q. Genetic factors influencing oligodendrocyte demyelination. [Doctoral Dissertation]. Rutgers University; 2018. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/56157/

20. Son, Alexander I., 1983-. The role of ephrin-A5 in lens maintenance and vitreous humor development.

Degree: Neuroscience, 2013, Rutgers University

Subjects/Keywords: Protein-tyrosine kinase – Receptors; Vitreous body – Diseases; Vitreous humor

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APA (6th Edition):

Son, Alexander I., 1. (2013). The role of ephrin-A5 in lens maintenance and vitreous humor development. (Thesis). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000067846

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Son, Alexander I., 1983-. “The role of ephrin-A5 in lens maintenance and vitreous humor development.” 2013. Thesis, Rutgers University. Accessed May 09, 2021. http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000067846.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Son, Alexander I., 1983-. “The role of ephrin-A5 in lens maintenance and vitreous humor development.” 2013. Web. 09 May 2021.

Vancouver:

Son, Alexander I. 1. The role of ephrin-A5 in lens maintenance and vitreous humor development. [Internet] [Thesis]. Rutgers University; 2013. [cited 2021 May 09]. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000067846.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Son, Alexander I. 1. The role of ephrin-A5 in lens maintenance and vitreous humor development. [Thesis]. Rutgers University; 2013. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000067846

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

21. Komlos, Daniel, 1979-. Expression and function of SIRT1, SIRT4, and glutamate dehydrogenase 1 within the central nervous system.

Degree: Neuroscience, 2011, Rutgers University

Subjects/Keywords: Glutamate dehydrogenase; Central nervous system—Metabolism

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APA (6th Edition):

Komlos, Daniel, 1. (2011). Expression and function of SIRT1, SIRT4, and glutamate dehydrogenase 1 within the central nervous system. (Thesis). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000063504

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Komlos, Daniel, 1979-. “Expression and function of SIRT1, SIRT4, and glutamate dehydrogenase 1 within the central nervous system.” 2011. Thesis, Rutgers University. Accessed May 09, 2021. http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000063504.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Komlos, Daniel, 1979-. “Expression and function of SIRT1, SIRT4, and glutamate dehydrogenase 1 within the central nervous system.” 2011. Web. 09 May 2021.

Vancouver:

Komlos, Daniel 1. Expression and function of SIRT1, SIRT4, and glutamate dehydrogenase 1 within the central nervous system. [Internet] [Thesis]. Rutgers University; 2011. [cited 2021 May 09]. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000063504.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Komlos, Daniel 1. Expression and function of SIRT1, SIRT4, and glutamate dehydrogenase 1 within the central nervous system. [Thesis]. Rutgers University; 2011. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000063504

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rutgers University

22. Namkoong, Jin. Metabotropic glutamate receptor 1 and glutamate signaling in human melanoma.

Degree: PhD, Microbiology and Molecular Genetics, 2007, Rutgers University

Melanoma is the most malignant form of skin cancers that originated from melanocytes, the pigment cells in the skin. Early detection is the key for… (more)

Subjects/Keywords: Melanoma; Cancer; Skin

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APA (6th Edition):

Namkoong, J. (2007). Metabotropic glutamate receptor 1 and glutamate signaling in human melanoma. (Doctoral Dissertation). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.13495

Chicago Manual of Style (16th Edition):

Namkoong, Jin. “Metabotropic glutamate receptor 1 and glutamate signaling in human melanoma.” 2007. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.13495.

MLA Handbook (7th Edition):

Namkoong, Jin. “Metabotropic glutamate receptor 1 and glutamate signaling in human melanoma.” 2007. Web. 09 May 2021.

Vancouver:

Namkoong J. Metabotropic glutamate receptor 1 and glutamate signaling in human melanoma. [Internet] [Doctoral dissertation]. Rutgers University; 2007. [cited 2021 May 09]. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.13495.

Council of Science Editors:

Namkoong J. Metabotropic glutamate receptor 1 and glutamate signaling in human melanoma. [Doctoral Dissertation]. Rutgers University; 2007. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.13495


Rutgers University

23. Loy, Shea, 1986-. The development of a model of PhIP-induced colon carcinogenesis in CYP1A2-humanized mice.

Degree: MS, Cell and Developmental Biology, 2010, Rutgers University

 The dietary carcinogen 2-amino-1-methyl-6-phenylimidazo [4,5-b] pyridine (PhIP) is considered one of the most abundant heterocyclic amines formed during the cooking of meats and fish. PhIP… (more)

Subjects/Keywords: Transgenic mice – Diseases; Colon (Anatomy) – Cancer – Pathophysiology; Colon (Anatomy) – Cancer – Prevention; Heterocyclic chemistry; Cytochrome P-450 – Physiological effect

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Loy, Shea, 1. (2010). The development of a model of PhIP-induced colon carcinogenesis in CYP1A2-humanized mice. (Masters Thesis). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000053614

Chicago Manual of Style (16th Edition):

Loy, Shea, 1986-. “The development of a model of PhIP-induced colon carcinogenesis in CYP1A2-humanized mice.” 2010. Masters Thesis, Rutgers University. Accessed May 09, 2021. http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000053614.

MLA Handbook (7th Edition):

Loy, Shea, 1986-. “The development of a model of PhIP-induced colon carcinogenesis in CYP1A2-humanized mice.” 2010. Web. 09 May 2021.

Vancouver:

Loy, Shea 1. The development of a model of PhIP-induced colon carcinogenesis in CYP1A2-humanized mice. [Internet] [Masters thesis]. Rutgers University; 2010. [cited 2021 May 09]. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000053614.

Council of Science Editors:

Loy, Shea 1. The development of a model of PhIP-induced colon carcinogenesis in CYP1A2-humanized mice. [Masters Thesis]. Rutgers University; 2010. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000053614


Rutgers University

24. Hu, Tianjing. Regulation of process retraction and cell migration by Epha3 is mediated by the by the adaptor protein Nck1.

Degree: PhD, Neuroscience, 2007, Rutgers University

Members of the largest RTK (receptor tyrosine kinase) family, Eph receptors, and their ligands play pivotal roles in embryonic development. They have been shown to… (more)

Subjects/Keywords: Receptor-ligand complexes; Protein-tyrosine kinase; Central nervous system; Hippocampus (Brain)

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hu, T. (2007). Regulation of process retraction and cell migration by Epha3 is mediated by the by the adaptor protein Nck1. (Doctoral Dissertation). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.16096

Chicago Manual of Style (16th Edition):

Hu, Tianjing. “Regulation of process retraction and cell migration by Epha3 is mediated by the by the adaptor protein Nck1.” 2007. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.16096.

MLA Handbook (7th Edition):

Hu, Tianjing. “Regulation of process retraction and cell migration by Epha3 is mediated by the by the adaptor protein Nck1.” 2007. Web. 09 May 2021.

Vancouver:

Hu T. Regulation of process retraction and cell migration by Epha3 is mediated by the by the adaptor protein Nck1. [Internet] [Doctoral dissertation]. Rutgers University; 2007. [cited 2021 May 09]. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.16096.

Council of Science Editors:

Hu T. Regulation of process retraction and cell migration by Epha3 is mediated by the by the adaptor protein Nck1. [Doctoral Dissertation]. Rutgers University; 2007. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.16096


Rutgers University

25. Bi, Caixia. Interaction between ephrin/Eph and BDNF in modulating hippocampal synaptic transmission and synapse formation.

Degree: PhD, Neuroscience, 2008, Rutgers University

The hippocampus is a brain structure known to be critical for learning and memory consolidation. Abnormal development or damage to this structure is known to… (more)

Subjects/Keywords: Hippocampus (Brain); Neural transmission

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APA (6th Edition):

Bi, C. (2008). Interaction between ephrin/Eph and BDNF in modulating hippocampal synaptic transmission and synapse formation. (Doctoral Dissertation). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17436

Chicago Manual of Style (16th Edition):

Bi, Caixia. “Interaction between ephrin/Eph and BDNF in modulating hippocampal synaptic transmission and synapse formation.” 2008. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17436.

MLA Handbook (7th Edition):

Bi, Caixia. “Interaction between ephrin/Eph and BDNF in modulating hippocampal synaptic transmission and synapse formation.” 2008. Web. 09 May 2021.

Vancouver:

Bi C. Interaction between ephrin/Eph and BDNF in modulating hippocampal synaptic transmission and synapse formation. [Internet] [Doctoral dissertation]. Rutgers University; 2008. [cited 2021 May 09]. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17436.

Council of Science Editors:

Bi C. Interaction between ephrin/Eph and BDNF in modulating hippocampal synaptic transmission and synapse formation. [Doctoral Dissertation]. Rutgers University; 2008. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17436


Rutgers University

26. Wu, Rachel Tsai-Han. Pharmacokinetics of chemopreventive compounds:.

Degree: MS, Pharmaceutical Science, 2008, Rutgers University

 Cancer claims millions of lives per year. In an effort to find cures to cancer, scientists have turned to chemopreventive compounds that are ingested by… (more)

Subjects/Keywords: Antioxidants – Physiological effect; Pharmacokinetics

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APA (6th Edition):

Wu, R. T. (2008). Pharmacokinetics of chemopreventive compounds:. (Masters Thesis). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000050477

Chicago Manual of Style (16th Edition):

Wu, Rachel Tsai-Han. “Pharmacokinetics of chemopreventive compounds:.” 2008. Masters Thesis, Rutgers University. Accessed May 09, 2021. http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000050477.

MLA Handbook (7th Edition):

Wu, Rachel Tsai-Han. “Pharmacokinetics of chemopreventive compounds:.” 2008. Web. 09 May 2021.

Vancouver:

Wu RT. Pharmacokinetics of chemopreventive compounds:. [Internet] [Masters thesis]. Rutgers University; 2008. [cited 2021 May 09]. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000050477.

Council of Science Editors:

Wu RT. Pharmacokinetics of chemopreventive compounds:. [Masters Thesis]. Rutgers University; 2008. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000050477


Rutgers University

27. Liu, Yingying. The role of Pak4 in tumorigenesis in vivo.

Degree: PhD, Pharmacology, Cellular and Molecular, 2010, Rutgers University

The Pak4 (P21 activated kinase) serine/threonine kinase is an important effector of Rho GTPases and has been implicated in the regulation of cell morphology and… (more)

Subjects/Keywords: Carcinogenesis; Cells – Morphology; Nude mouse – Research

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APA (6th Edition):

Liu, Y. (2010). The role of Pak4 in tumorigenesis in vivo. (Doctoral Dissertation). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000056516

Chicago Manual of Style (16th Edition):

Liu, Yingying. “The role of Pak4 in tumorigenesis in vivo.” 2010. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000056516.

MLA Handbook (7th Edition):

Liu, Yingying. “The role of Pak4 in tumorigenesis in vivo.” 2010. Web. 09 May 2021.

Vancouver:

Liu Y. The role of Pak4 in tumorigenesis in vivo. [Internet] [Doctoral dissertation]. Rutgers University; 2010. [cited 2021 May 09]. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000056516.

Council of Science Editors:

Liu Y. The role of Pak4 in tumorigenesis in vivo. [Doctoral Dissertation]. Rutgers University; 2010. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000056516


Rutgers University

28. DeBoer, Erik Michael, 1980-. The role of HuD, a post-transcriptional regulator, in the development and function of the murine neocortex.

Degree: Neuroscience, 2014, Rutgers University

Subjects/Keywords: Neocortex; RNA-protein interactions

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APA (6th Edition):

DeBoer, Erik Michael, 1. (2014). The role of HuD, a post-transcriptional regulator, in the development and function of the murine neocortex. (Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/42374/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

DeBoer, Erik Michael, 1980-. “The role of HuD, a post-transcriptional regulator, in the development and function of the murine neocortex.” 2014. Thesis, Rutgers University. Accessed May 09, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/42374/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

DeBoer, Erik Michael, 1980-. “The role of HuD, a post-transcriptional regulator, in the development and function of the murine neocortex.” 2014. Web. 09 May 2021.

Vancouver:

DeBoer, Erik Michael 1. The role of HuD, a post-transcriptional regulator, in the development and function of the murine neocortex. [Internet] [Thesis]. Rutgers University; 2014. [cited 2021 May 09]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/42374/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

DeBoer, Erik Michael 1. The role of HuD, a post-transcriptional regulator, in the development and function of the murine neocortex. [Thesis]. Rutgers University; 2014. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/42374/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rutgers University

29. Cooper, Joel F. The effects of methylmercuric chloride exposure on immediate early gene induction in the murine brain.

Degree: PhD, Toxicology, 2008, Rutgers University

The present set of experiments was designed to examine the effects of dose, route of administration, and frequency of MeHg exposure on activation of neural… (more)

Subjects/Keywords: Methylmercury – Physiological effect; Mice; Mice – Effects of chemicals on

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APA (6th Edition):

Cooper, J. F. (2008). The effects of methylmercuric chloride exposure on immediate early gene induction in the murine brain. (Doctoral Dissertation). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17292

Chicago Manual of Style (16th Edition):

Cooper, Joel F. “The effects of methylmercuric chloride exposure on immediate early gene induction in the murine brain.” 2008. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17292.

MLA Handbook (7th Edition):

Cooper, Joel F. “The effects of methylmercuric chloride exposure on immediate early gene induction in the murine brain.” 2008. Web. 09 May 2021.

Vancouver:

Cooper JF. The effects of methylmercuric chloride exposure on immediate early gene induction in the murine brain. [Internet] [Doctoral dissertation]. Rutgers University; 2008. [cited 2021 May 09]. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17292.

Council of Science Editors:

Cooper JF. The effects of methylmercuric chloride exposure on immediate early gene induction in the murine brain. [Doctoral Dissertation]. Rutgers University; 2008. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17292


Rutgers University

30. Cooper, Margaret Ann. Novel developmental roles of EphA receptors.

Degree: PhD, Neuroscience, 2008, Rutgers University

The EphA tyrosine kinase receptors mediate diverse cellular functions including axon guidance, cell migration, cell adhesion, and differentiation. A member of this family, EphA5, is… (more)

Subjects/Keywords: Receptor-ligand complexes; Protein-tyrosine kinase; Central nervous system; Hippocampus (Brain)

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APA (6th Edition):

Cooper, M. A. (2008). Novel developmental roles of EphA receptors. (Doctoral Dissertation). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17454

Chicago Manual of Style (16th Edition):

Cooper, Margaret Ann. “Novel developmental roles of EphA receptors.” 2008. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17454.

MLA Handbook (7th Edition):

Cooper, Margaret Ann. “Novel developmental roles of EphA receptors.” 2008. Web. 09 May 2021.

Vancouver:

Cooper MA. Novel developmental roles of EphA receptors. [Internet] [Doctoral dissertation]. Rutgers University; 2008. [cited 2021 May 09]. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17454.

Council of Science Editors:

Cooper MA. Novel developmental roles of EphA receptors. [Doctoral Dissertation]. Rutgers University; 2008. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17454

[1] [2]

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