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You searched for +publisher:"Rutgers University" +contributor:("Xia, Bing"). Showing records 1 – 8 of 8 total matches.

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Rutgers University

1. Rodriguez-Colon, Lizahira, 1987-. Role of G9a methyltransferase in the dna damage response signal.

Degree: PhD, Pharmacology, Cellular and Molecular, 2018, Rutgers University

DNA damage induces a choreographed set of local changes in histone modifications which leads to efficient recruitment of DNA repair factors. The regulation of these… (more)

Subjects/Keywords: DNA repair

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APA (6th Edition):

Rodriguez-Colon, Lizahira, 1. (2018). Role of G9a methyltransferase in the dna damage response signal. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/57687/

Chicago Manual of Style (16th Edition):

Rodriguez-Colon, Lizahira, 1987-. “Role of G9a methyltransferase in the dna damage response signal.” 2018. Doctoral Dissertation, Rutgers University. Accessed April 12, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/57687/.

MLA Handbook (7th Edition):

Rodriguez-Colon, Lizahira, 1987-. “Role of G9a methyltransferase in the dna damage response signal.” 2018. Web. 12 Apr 2021.

Vancouver:

Rodriguez-Colon, Lizahira 1. Role of G9a methyltransferase in the dna damage response signal. [Internet] [Doctoral dissertation]. Rutgers University; 2018. [cited 2021 Apr 12]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/57687/.

Council of Science Editors:

Rodriguez-Colon, Lizahira 1. Role of G9a methyltransferase in the dna damage response signal. [Doctoral Dissertation]. Rutgers University; 2018. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/57687/


Rutgers University

2. Zheng, Jiaying. Voltage-gated proton channel contributes to secondary damage following spinal cord injury.

Degree: MS, Microbiology and Molecular Genetics, 2018, Rutgers University

 Traumatic injury to the spinal cord initiates a series of destructive cellular processes that exacerbate tissue damage at and beyond the original site of injury.… (more)

Subjects/Keywords: Spinal cord – Wounds and injuries; Microglia

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APA (6th Edition):

Zheng, J. (2018). Voltage-gated proton channel contributes to secondary damage following spinal cord injury. (Masters Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/57764/

Chicago Manual of Style (16th Edition):

Zheng, Jiaying. “Voltage-gated proton channel contributes to secondary damage following spinal cord injury.” 2018. Masters Thesis, Rutgers University. Accessed April 12, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/57764/.

MLA Handbook (7th Edition):

Zheng, Jiaying. “Voltage-gated proton channel contributes to secondary damage following spinal cord injury.” 2018. Web. 12 Apr 2021.

Vancouver:

Zheng J. Voltage-gated proton channel contributes to secondary damage following spinal cord injury. [Internet] [Masters thesis]. Rutgers University; 2018. [cited 2021 Apr 12]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/57764/.

Council of Science Editors:

Zheng J. Voltage-gated proton channel contributes to secondary damage following spinal cord injury. [Masters Thesis]. Rutgers University; 2018. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/57764/


Rutgers University

3. Foo, Tzeh Keong, 1989-. BRCA1-PALB2 interaction and its roles in maintenance of genome stability and suppression of cancer development.

Degree: PhD, Pharmacology, Cellular and Molecular, 2019, Rutgers University

 DDR is often considered as a critical barrier during tumor initiation with most pre-malignant cells accumulate endogenous DNA damage before acquiring additional genetic alterations that… (more)

Subjects/Keywords: BRCA genes

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APA (6th Edition):

Foo, Tzeh Keong, 1. (2019). BRCA1-PALB2 interaction and its roles in maintenance of genome stability and suppression of cancer development. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/60154/

Chicago Manual of Style (16th Edition):

Foo, Tzeh Keong, 1989-. “BRCA1-PALB2 interaction and its roles in maintenance of genome stability and suppression of cancer development.” 2019. Doctoral Dissertation, Rutgers University. Accessed April 12, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/60154/.

MLA Handbook (7th Edition):

Foo, Tzeh Keong, 1989-. “BRCA1-PALB2 interaction and its roles in maintenance of genome stability and suppression of cancer development.” 2019. Web. 12 Apr 2021.

Vancouver:

Foo, Tzeh Keong 1. BRCA1-PALB2 interaction and its roles in maintenance of genome stability and suppression of cancer development. [Internet] [Doctoral dissertation]. Rutgers University; 2019. [cited 2021 Apr 12]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/60154/.

Council of Science Editors:

Foo, Tzeh Keong 1. BRCA1-PALB2 interaction and its roles in maintenance of genome stability and suppression of cancer development. [Doctoral Dissertation]. Rutgers University; 2019. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/60154/


Rutgers University

4. Karsli Uzunbas, Gizem, 1984-. Autophagy in tissue homeostasis and cancer.

Degree: PhD, Homeostasis, 2015, Rutgers University

Macroautophagy (autophagy hereafter) is a protective process that recycles cellular components to maintain homeostasis and survival. By removing damaged protein and organelles, autophagy ensures protein… (more)

Subjects/Keywords: Tumors; Cancer – Treatment; Biochemistry

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APA (6th Edition):

Karsli Uzunbas, Gizem, 1. (2015). Autophagy in tissue homeostasis and cancer. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/46502/

Chicago Manual of Style (16th Edition):

Karsli Uzunbas, Gizem, 1984-. “Autophagy in tissue homeostasis and cancer.” 2015. Doctoral Dissertation, Rutgers University. Accessed April 12, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/46502/.

MLA Handbook (7th Edition):

Karsli Uzunbas, Gizem, 1984-. “Autophagy in tissue homeostasis and cancer.” 2015. Web. 12 Apr 2021.

Vancouver:

Karsli Uzunbas, Gizem 1. Autophagy in tissue homeostasis and cancer. [Internet] [Doctoral dissertation]. Rutgers University; 2015. [cited 2021 Apr 12]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/46502/.

Council of Science Editors:

Karsli Uzunbas, Gizem 1. Autophagy in tissue homeostasis and cancer. [Doctoral Dissertation]. Rutgers University; 2015. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/46502/


Rutgers University

5. Yang, Yang, 1989-. Autophagy suppresses oxidative stress and p53 for adult mice survival and is essential for telocytes survival and intestinal homeostasis.

Degree: PhD, Biochemistry, 2020, Rutgers University

Autophagy captures intracellular components and delivers them to lysosomes for degradation and recycling. Conditional autophagy deficiency in adult mice causes liver damage, and shortens lifespan… (more)

Subjects/Keywords: Cellular control mechanisms

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APA (6th Edition):

Yang, Yang, 1. (2020). Autophagy suppresses oxidative stress and p53 for adult mice survival and is essential for telocytes survival and intestinal homeostasis. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/62539/

Chicago Manual of Style (16th Edition):

Yang, Yang, 1989-. “Autophagy suppresses oxidative stress and p53 for adult mice survival and is essential for telocytes survival and intestinal homeostasis.” 2020. Doctoral Dissertation, Rutgers University. Accessed April 12, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/62539/.

MLA Handbook (7th Edition):

Yang, Yang, 1989-. “Autophagy suppresses oxidative stress and p53 for adult mice survival and is essential for telocytes survival and intestinal homeostasis.” 2020. Web. 12 Apr 2021.

Vancouver:

Yang, Yang 1. Autophagy suppresses oxidative stress and p53 for adult mice survival and is essential for telocytes survival and intestinal homeostasis. [Internet] [Doctoral dissertation]. Rutgers University; 2020. [cited 2021 Apr 12]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/62539/.

Council of Science Editors:

Yang, Yang 1. Autophagy suppresses oxidative stress and p53 for adult mice survival and is essential for telocytes survival and intestinal homeostasis. [Doctoral Dissertation]. Rutgers University; 2020. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/62539/

6. Mahdi, Amar Hekmat, 1977-. Understanding the role of the PALB2-BRCA1 interaction in tumor suppression.

Degree: PhD, Physiology and Integrative Biology, 2017, Rutgers University

 Homologous recombination (HR) is the only error-free pathway for the repair of DNA double strand breaks (DSBs). BRCA1 and BRCA2, the two major breast cancer… (more)

Subjects/Keywords: Cancer – Prevention; Breast – Cancer

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APA (6th Edition):

Mahdi, Amar Hekmat, 1. (2017). Understanding the role of the PALB2-BRCA1 interaction in tumor suppression. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/53735/

Chicago Manual of Style (16th Edition):

Mahdi, Amar Hekmat, 1977-. “Understanding the role of the PALB2-BRCA1 interaction in tumor suppression.” 2017. Doctoral Dissertation, Rutgers University. Accessed April 12, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/53735/.

MLA Handbook (7th Edition):

Mahdi, Amar Hekmat, 1977-. “Understanding the role of the PALB2-BRCA1 interaction in tumor suppression.” 2017. Web. 12 Apr 2021.

Vancouver:

Mahdi, Amar Hekmat 1. Understanding the role of the PALB2-BRCA1 interaction in tumor suppression. [Internet] [Doctoral dissertation]. Rutgers University; 2017. [cited 2021 Apr 12]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/53735/.

Council of Science Editors:

Mahdi, Amar Hekmat 1. Understanding the role of the PALB2-BRCA1 interaction in tumor suppression. [Doctoral Dissertation]. Rutgers University; 2017. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/53735/

7. Olayanju, Julia Bolanle, 1976-. Understanding the anti-cancer activities of Moringa isothiocyanates in breast cancer cells.

Degree: PhD, Microbiology and Molecular Genetics, 2018, Rutgers University

Isothiocyanates (ITCs) are a class of naturally occurring compounds shown to have anti-cancer activities and promising chemopreventive prospects. Moringa oleifera (MO), a cruciferous vegetable widely… (more)

Subjects/Keywords: Moringa oleifera; Breast – Cancer

…Aldrich, ST Louis, MO, USA while MIC-1 was a generous gift from Dr. Ilya Raskin’s lab at Rutgers… …University New Brunswick. Cells were treated with different concentrations of PEITC (0.01 µM… 

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APA (6th Edition):

Olayanju, Julia Bolanle, 1. (2018). Understanding the anti-cancer activities of Moringa isothiocyanates in breast cancer cells. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/57669/

Chicago Manual of Style (16th Edition):

Olayanju, Julia Bolanle, 1976-. “Understanding the anti-cancer activities of Moringa isothiocyanates in breast cancer cells.” 2018. Doctoral Dissertation, Rutgers University. Accessed April 12, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/57669/.

MLA Handbook (7th Edition):

Olayanju, Julia Bolanle, 1976-. “Understanding the anti-cancer activities of Moringa isothiocyanates in breast cancer cells.” 2018. Web. 12 Apr 2021.

Vancouver:

Olayanju, Julia Bolanle 1. Understanding the anti-cancer activities of Moringa isothiocyanates in breast cancer cells. [Internet] [Doctoral dissertation]. Rutgers University; 2018. [cited 2021 Apr 12]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/57669/.

Council of Science Editors:

Olayanju, Julia Bolanle 1. Understanding the anti-cancer activities of Moringa isothiocyanates in breast cancer cells. [Doctoral Dissertation]. Rutgers University; 2018. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/57669/


Rutgers University

8. Cheung, Ka Lung. NRF2 and chemoprevention: signaling, epigenetics and role in intestinal carcinogensis.

Degree: PhD, Pharmaceutical Science, 2011, Rutgers University

Prevention is better than cure. The carcinogenesis could take as long as 20 to 30 years to develop from initiated cells to malignant tumor, therefore… (more)

Subjects/Keywords: Cancer – Chemoprevention

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APA (6th Edition):

Cheung, K. L. (2011). NRF2 and chemoprevention: signaling, epigenetics and role in intestinal carcinogensis. (Doctoral Dissertation). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000061171

Chicago Manual of Style (16th Edition):

Cheung, Ka Lung. “NRF2 and chemoprevention: signaling, epigenetics and role in intestinal carcinogensis.” 2011. Doctoral Dissertation, Rutgers University. Accessed April 12, 2021. http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000061171.

MLA Handbook (7th Edition):

Cheung, Ka Lung. “NRF2 and chemoprevention: signaling, epigenetics and role in intestinal carcinogensis.” 2011. Web. 12 Apr 2021.

Vancouver:

Cheung KL. NRF2 and chemoprevention: signaling, epigenetics and role in intestinal carcinogensis. [Internet] [Doctoral dissertation]. Rutgers University; 2011. [cited 2021 Apr 12]. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000061171.

Council of Science Editors:

Cheung KL. NRF2 and chemoprevention: signaling, epigenetics and role in intestinal carcinogensis. [Doctoral Dissertation]. Rutgers University; 2011. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000061171

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