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You searched for +publisher:"Rutgers University" +contributor:("Sarkar, Dipak"). Showing records 1 – 12 of 12 total matches.

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Rutgers University

1. Al-Yasari, Ali Mosa Rashid, 1981-. Does preconception alcohol abuse make the offspring vulnerable to developing type II diabetes?.

Degree: PhD, Endocrinology and Animal Biosciences, 2017, Rutgers University

 Recently, it was reported in our laboratory that binge-like alcohol drinking for three weeks prior to conception by female rats produces poor birth outcome and… (more)

Subjects/Keywords: Diabetes; Alcoholism

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APA (6th Edition):

Al-Yasari, Ali Mosa Rashid, 1. (2017). Does preconception alcohol abuse make the offspring vulnerable to developing type II diabetes?. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/55304/

Chicago Manual of Style (16th Edition):

Al-Yasari, Ali Mosa Rashid, 1981-. “Does preconception alcohol abuse make the offspring vulnerable to developing type II diabetes?.” 2017. Doctoral Dissertation, Rutgers University. Accessed January 18, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/55304/.

MLA Handbook (7th Edition):

Al-Yasari, Ali Mosa Rashid, 1981-. “Does preconception alcohol abuse make the offspring vulnerable to developing type II diabetes?.” 2017. Web. 18 Jan 2021.

Vancouver:

Al-Yasari, Ali Mosa Rashid 1. Does preconception alcohol abuse make the offspring vulnerable to developing type II diabetes?. [Internet] [Doctoral dissertation]. Rutgers University; 2017. [cited 2021 Jan 18]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/55304/.

Council of Science Editors:

Al-Yasari, Ali Mosa Rashid 1. Does preconception alcohol abuse make the offspring vulnerable to developing type II diabetes?. [Doctoral Dissertation]. Rutgers University; 2017. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/55304/


Rutgers University

2. Jabbar, Shaima, 1978-. Fetal alcohol exposure increases susceptibility to tumorigenesis in the pituitary.

Degree: PhD, Animal Sciences, 2017, Rutgers University

Alcohol exposure during gestation increases susceptibility to the development of pituitary tumors in response to estrogen in rat offspring. While the mechanism underlie this effect… (more)

Subjects/Keywords: Fetal alcohol spectrum disorders; Pituitary gland

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APA (6th Edition):

Jabbar, Shaima, 1. (2017). Fetal alcohol exposure increases susceptibility to tumorigenesis in the pituitary. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/55511/

Chicago Manual of Style (16th Edition):

Jabbar, Shaima, 1978-. “Fetal alcohol exposure increases susceptibility to tumorigenesis in the pituitary.” 2017. Doctoral Dissertation, Rutgers University. Accessed January 18, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/55511/.

MLA Handbook (7th Edition):

Jabbar, Shaima, 1978-. “Fetal alcohol exposure increases susceptibility to tumorigenesis in the pituitary.” 2017. Web. 18 Jan 2021.

Vancouver:

Jabbar, Shaima 1. Fetal alcohol exposure increases susceptibility to tumorigenesis in the pituitary. [Internet] [Doctoral dissertation]. Rutgers University; 2017. [cited 2021 Jan 18]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/55511/.

Council of Science Editors:

Jabbar, Shaima 1. Fetal alcohol exposure increases susceptibility to tumorigenesis in the pituitary. [Doctoral Dissertation]. Rutgers University; 2017. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/55511/


Rutgers University

3. Cabrera, Miguel Alexander. Fetal alcohol exposure induces neurotoxic effects on β-endorphin neurons through microglial activation.

Degree: PhD, Endocrinology and Animal Biosciences, 2018, Rutgers University

Fetal alcohol exposure has many detrimental effects on the developing brain and can cause fetal alcohol spectrum disorders (FASDs). Many FASDs patients show lifelong stress… (more)

Subjects/Keywords: Fetal alcohol spectrum disorders

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APA (6th Edition):

Cabrera, M. A. (2018). Fetal alcohol exposure induces neurotoxic effects on β-endorphin neurons through microglial activation. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/58931/

Chicago Manual of Style (16th Edition):

Cabrera, Miguel Alexander. “Fetal alcohol exposure induces neurotoxic effects on β-endorphin neurons through microglial activation.” 2018. Doctoral Dissertation, Rutgers University. Accessed January 18, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/58931/.

MLA Handbook (7th Edition):

Cabrera, Miguel Alexander. “Fetal alcohol exposure induces neurotoxic effects on β-endorphin neurons through microglial activation.” 2018. Web. 18 Jan 2021.

Vancouver:

Cabrera MA. Fetal alcohol exposure induces neurotoxic effects on β-endorphin neurons through microglial activation. [Internet] [Doctoral dissertation]. Rutgers University; 2018. [cited 2021 Jan 18]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/58931/.

Council of Science Editors:

Cabrera MA. Fetal alcohol exposure induces neurotoxic effects on β-endorphin neurons through microglial activation. [Doctoral Dissertation]. Rutgers University; 2018. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/58931/


Rutgers University

4. Polanco, Tiffany, 1982-. Effect of fetal alcohol on mammary carcinogenesis: the role of the estrogen/IGF axis.

Degree: PhD, Animal Sciences, 2011, Rutgers University

Altered fetal programming as a result of a suboptimal in utero environment can increase disease susceptibility in adulthood. Alcohol exposure in utero increases mammary cancer… (more)

Subjects/Keywords: Children of prenatal alcohol abuse; Breast – Cancer – Etiology; Alcohol – Physiological effect

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APA (6th Edition):

Polanco, Tiffany, 1. (2011). Effect of fetal alcohol on mammary carcinogenesis: the role of the estrogen/IGF axis. (Doctoral Dissertation). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000061439

Chicago Manual of Style (16th Edition):

Polanco, Tiffany, 1982-. “Effect of fetal alcohol on mammary carcinogenesis: the role of the estrogen/IGF axis.” 2011. Doctoral Dissertation, Rutgers University. Accessed January 18, 2021. http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000061439.

MLA Handbook (7th Edition):

Polanco, Tiffany, 1982-. “Effect of fetal alcohol on mammary carcinogenesis: the role of the estrogen/IGF axis.” 2011. Web. 18 Jan 2021.

Vancouver:

Polanco, Tiffany 1. Effect of fetal alcohol on mammary carcinogenesis: the role of the estrogen/IGF axis. [Internet] [Doctoral dissertation]. Rutgers University; 2011. [cited 2021 Jan 18]. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000061439.

Council of Science Editors:

Polanco, Tiffany 1. Effect of fetal alcohol on mammary carcinogenesis: the role of the estrogen/IGF axis. [Doctoral Dissertation]. Rutgers University; 2011. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000061439


Rutgers University

5. Zhu, Qiaoqiao, 1991-. Crosstalk between long non-coding RNAs and circadian chromatin.

Degree: PhD, Circadian rhythms, 2020, Rutgers University

The circadian rhythm is governed by transcriptional negative feedback facilitated by oscillating histone modifications and chromatin remodeling. The circadian rhythm is entrained by external zeitgebers… (more)

Subjects/Keywords: Ribonucleases; Endocrinology and Animal Biosciences

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APA (6th Edition):

Zhu, Qiaoqiao, 1. (2020). Crosstalk between long non-coding RNAs and circadian chromatin. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/64311/

Chicago Manual of Style (16th Edition):

Zhu, Qiaoqiao, 1991-. “Crosstalk between long non-coding RNAs and circadian chromatin.” 2020. Doctoral Dissertation, Rutgers University. Accessed January 18, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/64311/.

MLA Handbook (7th Edition):

Zhu, Qiaoqiao, 1991-. “Crosstalk between long non-coding RNAs and circadian chromatin.” 2020. Web. 18 Jan 2021.

Vancouver:

Zhu, Qiaoqiao 1. Crosstalk between long non-coding RNAs and circadian chromatin. [Internet] [Doctoral dissertation]. Rutgers University; 2020. [cited 2021 Jan 18]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/64311/.

Council of Science Editors:

Zhu, Qiaoqiao 1. Crosstalk between long non-coding RNAs and circadian chromatin. [Doctoral Dissertation]. Rutgers University; 2020. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/64311/


Rutgers University

6. Ramos, Brigette, 1993-. The effect of alcohol on genetically modified exosome deposition in the hypothalamus.

Degree: MS, Exosome, 2020, Rutgers University

 Fetal Alcohol Syndrome (FAS) is a condition, particularly found in children, characterized by damage to the central nervous system as a result of alcohol exposure… (more)

Subjects/Keywords: Fetal alcohol spectrum disorders; Physiology and Integrative Biology

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APA (6th Edition):

Ramos, Brigette, 1. (2020). The effect of alcohol on genetically modified exosome deposition in the hypothalamus. (Masters Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/64354/

Chicago Manual of Style (16th Edition):

Ramos, Brigette, 1993-. “The effect of alcohol on genetically modified exosome deposition in the hypothalamus.” 2020. Masters Thesis, Rutgers University. Accessed January 18, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/64354/.

MLA Handbook (7th Edition):

Ramos, Brigette, 1993-. “The effect of alcohol on genetically modified exosome deposition in the hypothalamus.” 2020. Web. 18 Jan 2021.

Vancouver:

Ramos, Brigette 1. The effect of alcohol on genetically modified exosome deposition in the hypothalamus. [Internet] [Masters thesis]. Rutgers University; 2020. [cited 2021 Jan 18]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/64354/.

Council of Science Editors:

Ramos, Brigette 1. The effect of alcohol on genetically modified exosome deposition in the hypothalamus. [Masters Thesis]. Rutgers University; 2020. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/64354/

7. Agapito, Maria. The role of Period 2 gene in mediating alcohol effects on metabolic signals in proopiomelanocortin producing neurons.

Degree: Neuroscience, 2013, Rutgers University

Subjects/Keywords: Proopiomelanocortin; Fetus – Metabolism; Fetal alcohol syndrome

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APA (6th Edition):

Agapito, M. (2013). The role of Period 2 gene in mediating alcohol effects on metabolic signals in proopiomelanocortin producing neurons. (Thesis). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000068807

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Agapito, Maria. “The role of Period 2 gene in mediating alcohol effects on metabolic signals in proopiomelanocortin producing neurons.” 2013. Thesis, Rutgers University. Accessed January 18, 2021. http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000068807.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Agapito, Maria. “The role of Period 2 gene in mediating alcohol effects on metabolic signals in proopiomelanocortin producing neurons.” 2013. Web. 18 Jan 2021.

Vancouver:

Agapito M. The role of Period 2 gene in mediating alcohol effects on metabolic signals in proopiomelanocortin producing neurons. [Internet] [Thesis]. Rutgers University; 2013. [cited 2021 Jan 18]. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000068807.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Agapito M. The role of Period 2 gene in mediating alcohol effects on metabolic signals in proopiomelanocortin producing neurons. [Thesis]. Rutgers University; 2013. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000068807

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

8. Bekdash, Rola Aldana, 1971-. Epigenetic effects of fetal alcohol exposure on hypothalamic proopiomelanocortin gene.

Degree: Neuroscience, 2012, Rutgers University

Subjects/Keywords: Fetal alcohol syndrome; Fetus—Development; Gene expression

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APA (6th Edition):

Bekdash, Rola Aldana, 1. (2012). Epigenetic effects of fetal alcohol exposure on hypothalamic proopiomelanocortin gene. (Thesis). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000063984

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bekdash, Rola Aldana, 1971-. “Epigenetic effects of fetal alcohol exposure on hypothalamic proopiomelanocortin gene.” 2012. Thesis, Rutgers University. Accessed January 18, 2021. http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000063984.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bekdash, Rola Aldana, 1971-. “Epigenetic effects of fetal alcohol exposure on hypothalamic proopiomelanocortin gene.” 2012. Web. 18 Jan 2021.

Vancouver:

Bekdash, Rola Aldana 1. Epigenetic effects of fetal alcohol exposure on hypothalamic proopiomelanocortin gene. [Internet] [Thesis]. Rutgers University; 2012. [cited 2021 Jan 18]. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000063984.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bekdash, Rola Aldana 1. Epigenetic effects of fetal alcohol exposure on hypothalamic proopiomelanocortin gene. [Thesis]. Rutgers University; 2012. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000063984

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rutgers University

9. Maeng, Lisa Y., 1983-. The stressed female brain: dissociating the prelimbic and infralimbic regions of the medial prefrontal cortex in the suppression of learning after acute stress.

Degree: Psychology, 2013, Rutgers University

Subjects/Keywords: Stress management for women; Women – Psychology; Women – Health and hygiene; Stress (Psychology) – Sex differences

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APA (6th Edition):

Maeng, Lisa Y., 1. (2013). The stressed female brain: dissociating the prelimbic and infralimbic regions of the medial prefrontal cortex in the suppression of learning after acute stress. (Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/41858/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Maeng, Lisa Y., 1983-. “The stressed female brain: dissociating the prelimbic and infralimbic regions of the medial prefrontal cortex in the suppression of learning after acute stress.” 2013. Thesis, Rutgers University. Accessed January 18, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/41858/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Maeng, Lisa Y., 1983-. “The stressed female brain: dissociating the prelimbic and infralimbic regions of the medial prefrontal cortex in the suppression of learning after acute stress.” 2013. Web. 18 Jan 2021.

Vancouver:

Maeng, Lisa Y. 1. The stressed female brain: dissociating the prelimbic and infralimbic regions of the medial prefrontal cortex in the suppression of learning after acute stress. [Internet] [Thesis]. Rutgers University; 2013. [cited 2021 Jan 18]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/41858/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Maeng, Lisa Y. 1. The stressed female brain: dissociating the prelimbic and infralimbic regions of the medial prefrontal cortex in the suppression of learning after acute stress. [Thesis]. Rutgers University; 2013. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/41858/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rutgers University

10. Franklin, Tina Carla, 1985-. The immediate and long term effects of microglial cell activation on stress response in fetal alcohol exposure model.

Degree: Cell and Developmental Biology, 2013, Rutgers University

Subjects/Keywords: Fetal alcohol syndrome; Microglia; Stress (Physiology) – Endocrine aspects

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APA (6th Edition):

Franklin, Tina Carla, 1. (2013). The immediate and long term effects of microglial cell activation on stress response in fetal alcohol exposure model. (Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/41769/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Franklin, Tina Carla, 1985-. “The immediate and long term effects of microglial cell activation on stress response in fetal alcohol exposure model.” 2013. Thesis, Rutgers University. Accessed January 18, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/41769/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Franklin, Tina Carla, 1985-. “The immediate and long term effects of microglial cell activation on stress response in fetal alcohol exposure model.” 2013. Web. 18 Jan 2021.

Vancouver:

Franklin, Tina Carla 1. The immediate and long term effects of microglial cell activation on stress response in fetal alcohol exposure model. [Internet] [Thesis]. Rutgers University; 2013. [cited 2021 Jan 18]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/41769/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Franklin, Tina Carla 1. The immediate and long term effects of microglial cell activation on stress response in fetal alcohol exposure model. [Thesis]. Rutgers University; 2013. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/41769/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rutgers University

11. Zhang, Changqing, 1984-. Role of beta-endorphin in control of stress and cancer progression in fetal alcohol exposed rats.

Degree: Animal Sciences, 2013, Rutgers University

Subjects/Keywords: Endorphins; Rats – Effect of stress on; Alcohol – Physiological effect; Cancer – Etiology; Cancer in animals

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Zhang, Changqing, 1. (2013). Role of beta-endorphin in control of stress and cancer progression in fetal alcohol exposed rats. (Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/42092/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhang, Changqing, 1984-. “Role of beta-endorphin in control of stress and cancer progression in fetal alcohol exposed rats.” 2013. Thesis, Rutgers University. Accessed January 18, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/42092/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhang, Changqing, 1984-. “Role of beta-endorphin in control of stress and cancer progression in fetal alcohol exposed rats.” 2013. Web. 18 Jan 2021.

Vancouver:

Zhang, Changqing 1. Role of beta-endorphin in control of stress and cancer progression in fetal alcohol exposed rats. [Internet] [Thesis]. Rutgers University; 2013. [cited 2021 Jan 18]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/42092/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhang, Changqing 1. Role of beta-endorphin in control of stress and cancer progression in fetal alcohol exposed rats. [Thesis]. Rutgers University; 2013. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/42092/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rutgers University

12. Spiegler, Elizabeth Kuhn, 1986-. The role of beta-carotene and its cleavage enzymes during vitamin A deficiency and mammalian embryonic development.

Degree: Nutritional Sciences, 2014, Rutgers University

Subjects/Keywords: Beta carotene; Vitamin A deficiency; Vitamin A in human nutrition; Embryology, Human

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Spiegler, Elizabeth Kuhn, 1. (2014). The role of beta-carotene and its cleavage enzymes during vitamin A deficiency and mammalian embryonic development. (Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/44219/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Spiegler, Elizabeth Kuhn, 1986-. “The role of beta-carotene and its cleavage enzymes during vitamin A deficiency and mammalian embryonic development.” 2014. Thesis, Rutgers University. Accessed January 18, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/44219/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Spiegler, Elizabeth Kuhn, 1986-. “The role of beta-carotene and its cleavage enzymes during vitamin A deficiency and mammalian embryonic development.” 2014. Web. 18 Jan 2021.

Vancouver:

Spiegler, Elizabeth Kuhn 1. The role of beta-carotene and its cleavage enzymes during vitamin A deficiency and mammalian embryonic development. [Internet] [Thesis]. Rutgers University; 2014. [cited 2021 Jan 18]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/44219/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Spiegler, Elizabeth Kuhn 1. The role of beta-carotene and its cleavage enzymes during vitamin A deficiency and mammalian embryonic development. [Thesis]. Rutgers University; 2014. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/44219/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.