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You searched for +publisher:"Rutgers University" +contributor:("Ma, Jianjie"). Showing records 1 – 4 of 4 total matches.

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Rutgers University

1. Thornton, Angela M. Regulation of store-operated calcium channel by Mitsugumin29 in skeletal muscle aging:.

Degree: PhD, Physiology and Integrative Biology, 2009, Rutgers University

The study of store-operated Ca2+ channel entry (SOCE) and its role in muscle contractility in young and aged skeletal muscle necessitates a thorough knowledge of… (more)

Subjects/Keywords: Calcium channels; Calcium – Physiological effect

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APA (6th Edition):

Thornton, A. M. (2009). Regulation of store-operated calcium channel by Mitsugumin29 in skeletal muscle aging:. (Doctoral Dissertation). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000051074

Chicago Manual of Style (16th Edition):

Thornton, Angela M. “Regulation of store-operated calcium channel by Mitsugumin29 in skeletal muscle aging:.” 2009. Doctoral Dissertation, Rutgers University. Accessed May 20, 2019. http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000051074.

MLA Handbook (7th Edition):

Thornton, Angela M. “Regulation of store-operated calcium channel by Mitsugumin29 in skeletal muscle aging:.” 2009. Web. 20 May 2019.

Vancouver:

Thornton AM. Regulation of store-operated calcium channel by Mitsugumin29 in skeletal muscle aging:. [Internet] [Doctoral dissertation]. Rutgers University; 2009. [cited 2019 May 20]. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000051074.

Council of Science Editors:

Thornton AM. Regulation of store-operated calcium channel by Mitsugumin29 in skeletal muscle aging:. [Doctoral Dissertation]. Rutgers University; 2009. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000051074


Rutgers University

2. Jaques, Kathryn Michelle. Oxidant-induced injury in the mammalian myocardium: functional and molecular effects of acetaminophen.

Degree: PhD, Physiology and Integrative Biology, 2010, Rutgers University

For over 100 years, acetaminophen (APAP) has been exclusively used as an efficacious antipyretic and analgesic. Here we report our ongoing investigation of the cardiovascular… (more)

Subjects/Keywords: Acetaminophen; Myocardium – Diseases; Arrhythmia – Treatment

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jaques, K. M. (2010). Oxidant-induced injury in the mammalian myocardium: functional and molecular effects of acetaminophen. (Doctoral Dissertation). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000053099

Chicago Manual of Style (16th Edition):

Jaques, Kathryn Michelle. “Oxidant-induced injury in the mammalian myocardium: functional and molecular effects of acetaminophen.” 2010. Doctoral Dissertation, Rutgers University. Accessed May 20, 2019. http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000053099.

MLA Handbook (7th Edition):

Jaques, Kathryn Michelle. “Oxidant-induced injury in the mammalian myocardium: functional and molecular effects of acetaminophen.” 2010. Web. 20 May 2019.

Vancouver:

Jaques KM. Oxidant-induced injury in the mammalian myocardium: functional and molecular effects of acetaminophen. [Internet] [Doctoral dissertation]. Rutgers University; 2010. [cited 2019 May 20]. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000053099.

Council of Science Editors:

Jaques KM. Oxidant-induced injury in the mammalian myocardium: functional and molecular effects of acetaminophen. [Doctoral Dissertation]. Rutgers University; 2010. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000053099


Rutgers University

3. Baliga, Sunanda S., 1982. Acetaminophen confers neuroprotection during early cerebral ischemia-reperfusion.

Degree: PhD, Physiology and Integrative Biology, 2009, Rutgers University

Stroke is the leading cause of disability in the United States. It usually occurs from a clot blocking a cerebral artery, resulting in cerebral ischemia… (more)

Subjects/Keywords: Cerebral ischemia – Treatment; Cerebrovascular disease – Treatment; Acetaminophen

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Baliga, Sunanda S., 1. (2009). Acetaminophen confers neuroprotection during early cerebral ischemia-reperfusion. (Doctoral Dissertation). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000051174

Chicago Manual of Style (16th Edition):

Baliga, Sunanda S., 1982. “Acetaminophen confers neuroprotection during early cerebral ischemia-reperfusion.” 2009. Doctoral Dissertation, Rutgers University. Accessed May 20, 2019. http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000051174.

MLA Handbook (7th Edition):

Baliga, Sunanda S., 1982. “Acetaminophen confers neuroprotection during early cerebral ischemia-reperfusion.” 2009. Web. 20 May 2019.

Vancouver:

Baliga, Sunanda S. 1. Acetaminophen confers neuroprotection during early cerebral ischemia-reperfusion. [Internet] [Doctoral dissertation]. Rutgers University; 2009. [cited 2019 May 20]. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000051174.

Council of Science Editors:

Baliga, Sunanda S. 1. Acetaminophen confers neuroprotection during early cerebral ischemia-reperfusion. [Doctoral Dissertation]. Rutgers University; 2009. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000051174


Rutgers University

4. Hadzimichalis, Norell Melissa. Acetaminophen-mediated cardioprotection via inhibition of the mitochondrial permeability transition pore-induced apoptotic pathway.

Degree: PhD, Physiology and Integrative Biology, 2008, Rutgers University

Historically, acetaminophen has been employed as a safe and effective analgesic and antipyretic agent. However, our laboratory has recently reported that acetaminophen also confers functional… (more)

Subjects/Keywords: Acetaminophen; Mitochondria; Heart – Effect of drugs on

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Hadzimichalis, N. M. (2008). Acetaminophen-mediated cardioprotection via inhibition of the mitochondrial permeability transition pore-induced apoptotic pathway. (Doctoral Dissertation). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17322

Chicago Manual of Style (16th Edition):

Hadzimichalis, Norell Melissa. “Acetaminophen-mediated cardioprotection via inhibition of the mitochondrial permeability transition pore-induced apoptotic pathway.” 2008. Doctoral Dissertation, Rutgers University. Accessed May 20, 2019. http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17322.

MLA Handbook (7th Edition):

Hadzimichalis, Norell Melissa. “Acetaminophen-mediated cardioprotection via inhibition of the mitochondrial permeability transition pore-induced apoptotic pathway.” 2008. Web. 20 May 2019.

Vancouver:

Hadzimichalis NM. Acetaminophen-mediated cardioprotection via inhibition of the mitochondrial permeability transition pore-induced apoptotic pathway. [Internet] [Doctoral dissertation]. Rutgers University; 2008. [cited 2019 May 20]. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17322.

Council of Science Editors:

Hadzimichalis NM. Acetaminophen-mediated cardioprotection via inhibition of the mitochondrial permeability transition pore-induced apoptotic pathway. [Doctoral Dissertation]. Rutgers University; 2008. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17322

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