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You searched for +publisher:"Rutgers University" +contributor:("Jin, Shengkan"). Showing records 1 – 9 of 9 total matches.

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Rutgers University

1. Al-Asadi, Amer, 1980-. Preventing and treating hepatic metastases of colon and pancreatic cancers by targeting cancer cell metabolism.

Degree: PhD, Physiology and Integrative Biology, 2018, Rutgers University

Alteration of glucose metabolism is a unique feature for a majority of cancers. Cancer cells exhibit aerobic glycolysis also known as the Warburg effect even… (more)

Subjects/Keywords: Cancer cells; Pancreas – Cancer

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APA (6th Edition):

Al-Asadi, Amer, 1. (2018). Preventing and treating hepatic metastases of colon and pancreatic cancers by targeting cancer cell metabolism. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/57463/

Chicago Manual of Style (16th Edition):

Al-Asadi, Amer, 1980-. “Preventing and treating hepatic metastases of colon and pancreatic cancers by targeting cancer cell metabolism.” 2018. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/57463/.

MLA Handbook (7th Edition):

Al-Asadi, Amer, 1980-. “Preventing and treating hepatic metastases of colon and pancreatic cancers by targeting cancer cell metabolism.” 2018. Web. 09 May 2021.

Vancouver:

Al-Asadi, Amer 1. Preventing and treating hepatic metastases of colon and pancreatic cancers by targeting cancer cell metabolism. [Internet] [Doctoral dissertation]. Rutgers University; 2018. [cited 2021 May 09]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/57463/.

Council of Science Editors:

Al-Asadi, Amer 1. Preventing and treating hepatic metastases of colon and pancreatic cancers by targeting cancer cell metabolism. [Doctoral Dissertation]. Rutgers University; 2018. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/57463/


Rutgers University

2. Bin Umer, Mohamed Anwar. Characterizing the role of mitochondria in the toxicity of trichothecenes produced by Fusarium graminearum.

Degree: Microbiology and Molecular Genetics, 2014, Rutgers University

Subjects/Keywords: Mitochondrial pathology; Fusarium; Yeast fungi – Biotechnology

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APA (6th Edition):

Bin Umer, M. A. (2014). Characterizing the role of mitochondria in the toxicity of trichothecenes produced by Fusarium graminearum. (Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/42366/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Bin Umer, Mohamed Anwar. “Characterizing the role of mitochondria in the toxicity of trichothecenes produced by Fusarium graminearum.” 2014. Thesis, Rutgers University. Accessed May 09, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/42366/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Bin Umer, Mohamed Anwar. “Characterizing the role of mitochondria in the toxicity of trichothecenes produced by Fusarium graminearum.” 2014. Web. 09 May 2021.

Vancouver:

Bin Umer MA. Characterizing the role of mitochondria in the toxicity of trichothecenes produced by Fusarium graminearum. [Internet] [Thesis]. Rutgers University; 2014. [cited 2021 May 09]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/42366/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Bin Umer MA. Characterizing the role of mitochondria in the toxicity of trichothecenes produced by Fusarium graminearum. [Thesis]. Rutgers University; 2014. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/42366/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rutgers University

3. Cai, Na, 1988-. The functional interplay between TPRM7 channel-kinase autophosphorylation and its cellular regulation.

Degree: PhD, Pharmacology, Cellular and Molecular, 2018, Rutgers University

As a member of the transient receptor potential ion channel subfamily, TRPM7 is a remarkable ion channel in possession of its own functional kinase domain.… (more)

Subjects/Keywords: TRP channels

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APA (6th Edition):

Cai, Na, 1. (2018). The functional interplay between TPRM7 channel-kinase autophosphorylation and its cellular regulation. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/57513/

Chicago Manual of Style (16th Edition):

Cai, Na, 1988-. “The functional interplay between TPRM7 channel-kinase autophosphorylation and its cellular regulation.” 2018. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/57513/.

MLA Handbook (7th Edition):

Cai, Na, 1988-. “The functional interplay between TPRM7 channel-kinase autophosphorylation and its cellular regulation.” 2018. Web. 09 May 2021.

Vancouver:

Cai, Na 1. The functional interplay between TPRM7 channel-kinase autophosphorylation and its cellular regulation. [Internet] [Doctoral dissertation]. Rutgers University; 2018. [cited 2021 May 09]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/57513/.

Council of Science Editors:

Cai, Na 1. The functional interplay between TPRM7 channel-kinase autophosphorylation and its cellular regulation. [Doctoral Dissertation]. Rutgers University; 2018. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/57513/

4. Chen, Hsin-Yi, 1977-. Role of autophagy in cancer.

Degree: PhD, Biochemistry, 2011, Rutgers University

(Macro)autophagy is a catabolic process whereby intracellular components are enclosed into autophagosomes and delivered to lysosomes for degradation. Constitutive activity of autophagy contributes to turnover… (more)

Subjects/Keywords: Cancer cells – Growth – Regulation; Cancer – Treatment

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APA (6th Edition):

Chen, Hsin-Yi, 1. (2011). Role of autophagy in cancer. (Doctoral Dissertation). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000057531

Chicago Manual of Style (16th Edition):

Chen, Hsin-Yi, 1977-. “Role of autophagy in cancer.” 2011. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000057531.

MLA Handbook (7th Edition):

Chen, Hsin-Yi, 1977-. “Role of autophagy in cancer.” 2011. Web. 09 May 2021.

Vancouver:

Chen, Hsin-Yi 1. Role of autophagy in cancer. [Internet] [Doctoral dissertation]. Rutgers University; 2011. [cited 2021 May 09]. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000057531.

Council of Science Editors:

Chen, Hsin-Yi 1. Role of autophagy in cancer. [Doctoral Dissertation]. Rutgers University; 2011. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000057531


Rutgers University

5. Collantes, Juan Carlos, 1981-. A novel CRISPR/RNA-aptamer-mediated base editing system with potential therapeutic value.

Degree: PhD, Pharmacology, Cellular and Molecular, 2019, Rutgers University

Nuclease-dependent precise genome editing such as correction of point mutations requires introduction of targeted DNA double strand breaks (DSB) and activation of homology dependent repair… (more)

Subjects/Keywords: Gene editing  – Therapeutic use; CRISPR-associated protein 9

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APA (6th Edition):

Collantes, Juan Carlos, 1. (2019). A novel CRISPR/RNA-aptamer-mediated base editing system with potential therapeutic value. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/60101/

Chicago Manual of Style (16th Edition):

Collantes, Juan Carlos, 1981-. “A novel CRISPR/RNA-aptamer-mediated base editing system with potential therapeutic value.” 2019. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/60101/.

MLA Handbook (7th Edition):

Collantes, Juan Carlos, 1981-. “A novel CRISPR/RNA-aptamer-mediated base editing system with potential therapeutic value.” 2019. Web. 09 May 2021.

Vancouver:

Collantes, Juan Carlos 1. A novel CRISPR/RNA-aptamer-mediated base editing system with potential therapeutic value. [Internet] [Doctoral dissertation]. Rutgers University; 2019. [cited 2021 May 09]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/60101/.

Council of Science Editors:

Collantes, Juan Carlos 1. A novel CRISPR/RNA-aptamer-mediated base editing system with potential therapeutic value. [Doctoral Dissertation]. Rutgers University; 2019. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/60101/


Rutgers University

6. Guo, Jingjing, 1990-. Application of 5-oh polymethoxyflavones and safe mitochondrial uncouplers in preventing and treating type 2 diabetes and fatty liver diseases.

Degree: PhD, Food Science, 2019, Rutgers University

 In modern society, obesity-related metabolic diseases have become a serious public health concern globally. The increasing prevalence, disease progression, diverse complications and subsequent morbidity and… (more)

Subjects/Keywords: Non-insulin-dependent diabetes  – Prevention; Lipids; Fatty liver  – Prevention

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APA (6th Edition):

Guo, Jingjing, 1. (2019). Application of 5-oh polymethoxyflavones and safe mitochondrial uncouplers in preventing and treating type 2 diabetes and fatty liver diseases. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/60162/

Chicago Manual of Style (16th Edition):

Guo, Jingjing, 1990-. “Application of 5-oh polymethoxyflavones and safe mitochondrial uncouplers in preventing and treating type 2 diabetes and fatty liver diseases.” 2019. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/60162/.

MLA Handbook (7th Edition):

Guo, Jingjing, 1990-. “Application of 5-oh polymethoxyflavones and safe mitochondrial uncouplers in preventing and treating type 2 diabetes and fatty liver diseases.” 2019. Web. 09 May 2021.

Vancouver:

Guo, Jingjing 1. Application of 5-oh polymethoxyflavones and safe mitochondrial uncouplers in preventing and treating type 2 diabetes and fatty liver diseases. [Internet] [Doctoral dissertation]. Rutgers University; 2019. [cited 2021 May 09]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/60162/.

Council of Science Editors:

Guo, Jingjing 1. Application of 5-oh polymethoxyflavones and safe mitochondrial uncouplers in preventing and treating type 2 diabetes and fatty liver diseases. [Doctoral Dissertation]. Rutgers University; 2019. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/60162/


Rutgers University

7. Karsli Uzunbas, Gizem, 1984-. Autophagy in tissue homeostasis and cancer.

Degree: PhD, Homeostasis, 2015, Rutgers University

Macroautophagy (autophagy hereafter) is a protective process that recycles cellular components to maintain homeostasis and survival. By removing damaged protein and organelles, autophagy ensures protein… (more)

Subjects/Keywords: Tumors; Cancer – Treatment; Biochemistry

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APA (6th Edition):

Karsli Uzunbas, Gizem, 1. (2015). Autophagy in tissue homeostasis and cancer. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/46502/

Chicago Manual of Style (16th Edition):

Karsli Uzunbas, Gizem, 1984-. “Autophagy in tissue homeostasis and cancer.” 2015. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/46502/.

MLA Handbook (7th Edition):

Karsli Uzunbas, Gizem, 1984-. “Autophagy in tissue homeostasis and cancer.” 2015. Web. 09 May 2021.

Vancouver:

Karsli Uzunbas, Gizem 1. Autophagy in tissue homeostasis and cancer. [Internet] [Doctoral dissertation]. Rutgers University; 2015. [cited 2021 May 09]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/46502/.

Council of Science Editors:

Karsli Uzunbas, Gizem 1. Autophagy in tissue homeostasis and cancer. [Doctoral Dissertation]. Rutgers University; 2015. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/46502/


Rutgers University

8. Shah, Raj D. Role of GRM1 in altering glutamate metabolism and bioavailability.

Degree: PhD, Glutamate, 2020, Rutgers University

 Altered metabolic activity has been implicated in several types of cancer including malignant melanoma. Previously, we have illustrated the role of a neuronal receptor, metabotropic… (more)

Subjects/Keywords: Toxicology

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APA (6th Edition):

Shah, R. D. (2020). Role of GRM1 in altering glutamate metabolism and bioavailability. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/64382/

Chicago Manual of Style (16th Edition):

Shah, Raj D. “Role of GRM1 in altering glutamate metabolism and bioavailability.” 2020. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/64382/.

MLA Handbook (7th Edition):

Shah, Raj D. “Role of GRM1 in altering glutamate metabolism and bioavailability.” 2020. Web. 09 May 2021.

Vancouver:

Shah RD. Role of GRM1 in altering glutamate metabolism and bioavailability. [Internet] [Doctoral dissertation]. Rutgers University; 2020. [cited 2021 May 09]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/64382/.

Council of Science Editors:

Shah RD. Role of GRM1 in altering glutamate metabolism and bioavailability. [Doctoral Dissertation]. Rutgers University; 2020. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/64382/


Rutgers University

9. Wang, Guannan. The Smad3 linker region: transcriptional activity and phosphorylation-mediated regulation.

Degree: PhD, Biochemistry, 2008, Rutgers University

TGF-β regulates cell proliferation, differentiation, apoptosis, and extracellular matrix production. Smad proteins are central mediators of TGF-β signaling. Upon ligand binding, Smad2 and Smad3 are… (more)

Subjects/Keywords: Smad proteins; Transforming growth factors-beta; Cellular signal transduction; Cancer cells – Growth – Regulation

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APA (6th Edition):

Wang, G. (2008). The Smad3 linker region: transcriptional activity and phosphorylation-mediated regulation. (Doctoral Dissertation). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17061

Chicago Manual of Style (16th Edition):

Wang, Guannan. “The Smad3 linker region: transcriptional activity and phosphorylation-mediated regulation.” 2008. Doctoral Dissertation, Rutgers University. Accessed May 09, 2021. http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17061.

MLA Handbook (7th Edition):

Wang, Guannan. “The Smad3 linker region: transcriptional activity and phosphorylation-mediated regulation.” 2008. Web. 09 May 2021.

Vancouver:

Wang G. The Smad3 linker region: transcriptional activity and phosphorylation-mediated regulation. [Internet] [Doctoral dissertation]. Rutgers University; 2008. [cited 2021 May 09]. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17061.

Council of Science Editors:

Wang G. The Smad3 linker region: transcriptional activity and phosphorylation-mediated regulation. [Doctoral Dissertation]. Rutgers University; 2008. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17061

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