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You searched for +publisher:"Rutgers University" +contributor:("Cohick, Wendie S"). Showing records 1 – 14 of 14 total matches.

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Rutgers University

1. Chiou, Jiachi, 1979-. The role of the ribosomal stalk in the activity of ricin, Shiga-like toxin 1 and Shiga-like toxin 2 in Saccharomyces cerevisiae.

Degree: PhD, Food Science, 2011, Rutgers University

Ricin, Shiga-like toxin 1 (Stx1) and Shiga-like toxin 2 (Stx2) are ribosome inactivating proteins (RIPs) that catalytically remove an adenine residue from the highly conserved… (more)

Subjects/Keywords: Ribosomes – Research; Ricin; Saccharomyces cerevisiae

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APA (6th Edition):

Chiou, Jiachi, 1. (2011). The role of the ribosomal stalk in the activity of ricin, Shiga-like toxin 1 and Shiga-like toxin 2 in Saccharomyces cerevisiae. (Doctoral Dissertation). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000057535

Chicago Manual of Style (16th Edition):

Chiou, Jiachi, 1979-. “The role of the ribosomal stalk in the activity of ricin, Shiga-like toxin 1 and Shiga-like toxin 2 in Saccharomyces cerevisiae.” 2011. Doctoral Dissertation, Rutgers University. Accessed January 18, 2021. http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000057535.

MLA Handbook (7th Edition):

Chiou, Jiachi, 1979-. “The role of the ribosomal stalk in the activity of ricin, Shiga-like toxin 1 and Shiga-like toxin 2 in Saccharomyces cerevisiae.” 2011. Web. 18 Jan 2021.

Vancouver:

Chiou, Jiachi 1. The role of the ribosomal stalk in the activity of ricin, Shiga-like toxin 1 and Shiga-like toxin 2 in Saccharomyces cerevisiae. [Internet] [Doctoral dissertation]. Rutgers University; 2011. [cited 2021 Jan 18]. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000057535.

Council of Science Editors:

Chiou, Jiachi 1. The role of the ribosomal stalk in the activity of ricin, Shiga-like toxin 1 and Shiga-like toxin 2 in Saccharomyces cerevisiae. [Doctoral Dissertation]. Rutgers University; 2011. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000057535


Rutgers University

2. Skorupa, Jennifer A., 1990-. IGFBP-3 induced by ribotoxic stress is not secreted prior to nuclear localization in mammary epithelial cells.

Degree: MS, Endocrinology and Animal Biosciences, 2018, Rutgers University

 Ribotoxic stressors such as anisomycin (ANS) and deoxynivalenol (DON) induce apoptosis in MAC-T cells. These agents also increase IGFBP-3 expression and knockdown of IGFBP-3 mitigates… (more)

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APA (6th Edition):

Skorupa, Jennifer A., 1. (2018). IGFBP-3 induced by ribotoxic stress is not secreted prior to nuclear localization in mammary epithelial cells. (Masters Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/56124/

Chicago Manual of Style (16th Edition):

Skorupa, Jennifer A., 1990-. “IGFBP-3 induced by ribotoxic stress is not secreted prior to nuclear localization in mammary epithelial cells.” 2018. Masters Thesis, Rutgers University. Accessed January 18, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/56124/.

MLA Handbook (7th Edition):

Skorupa, Jennifer A., 1990-. “IGFBP-3 induced by ribotoxic stress is not secreted prior to nuclear localization in mammary epithelial cells.” 2018. Web. 18 Jan 2021.

Vancouver:

Skorupa, Jennifer A. 1. IGFBP-3 induced by ribotoxic stress is not secreted prior to nuclear localization in mammary epithelial cells. [Internet] [Masters thesis]. Rutgers University; 2018. [cited 2021 Jan 18]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/56124/.

Council of Science Editors:

Skorupa, Jennifer A. 1. IGFBP-3 induced by ribotoxic stress is not secreted prior to nuclear localization in mammary epithelial cells. [Masters Thesis]. Rutgers University; 2018. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/56124/


Rutgers University

3. Polanco, Tiffany, 1982-. Effect of fetal alcohol on mammary carcinogenesis: the role of the estrogen/IGF axis.

Degree: PhD, Animal Sciences, 2011, Rutgers University

Altered fetal programming as a result of a suboptimal in utero environment can increase disease susceptibility in adulthood. Alcohol exposure in utero increases mammary cancer… (more)

Subjects/Keywords: Children of prenatal alcohol abuse; Breast – Cancer – Etiology; Alcohol – Physiological effect

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APA (6th Edition):

Polanco, Tiffany, 1. (2011). Effect of fetal alcohol on mammary carcinogenesis: the role of the estrogen/IGF axis. (Doctoral Dissertation). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000061439

Chicago Manual of Style (16th Edition):

Polanco, Tiffany, 1982-. “Effect of fetal alcohol on mammary carcinogenesis: the role of the estrogen/IGF axis.” 2011. Doctoral Dissertation, Rutgers University. Accessed January 18, 2021. http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000061439.

MLA Handbook (7th Edition):

Polanco, Tiffany, 1982-. “Effect of fetal alcohol on mammary carcinogenesis: the role of the estrogen/IGF axis.” 2011. Web. 18 Jan 2021.

Vancouver:

Polanco, Tiffany 1. Effect of fetal alcohol on mammary carcinogenesis: the role of the estrogen/IGF axis. [Internet] [Doctoral dissertation]. Rutgers University; 2011. [cited 2021 Jan 18]. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000061439.

Council of Science Editors:

Polanco, Tiffany 1. Effect of fetal alcohol on mammary carcinogenesis: the role of the estrogen/IGF axis. [Doctoral Dissertation]. Rutgers University; 2011. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000061439


Rutgers University

4. Stires, Hillary. Elucidating mechanisms that drive increased tumorigenesis in rats exposed to alcohol in utero.

Degree: PhD, Endocrinology and Animal Biosciences, 2016, Rutgers University

Alcohol exposure during gestation increases breast cancer risk in offspring. While the mechanisms that underlie this effect are not fully understood, serum estradiol (E2) is… (more)

Subjects/Keywords: Alcoholism in pregnancy; Fetus – Effects of drugs on

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APA (6th Edition):

Stires, H. (2016). Elucidating mechanisms that drive increased tumorigenesis in rats exposed to alcohol in utero. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/50188/

Chicago Manual of Style (16th Edition):

Stires, Hillary. “Elucidating mechanisms that drive increased tumorigenesis in rats exposed to alcohol in utero.” 2016. Doctoral Dissertation, Rutgers University. Accessed January 18, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/50188/.

MLA Handbook (7th Edition):

Stires, Hillary. “Elucidating mechanisms that drive increased tumorigenesis in rats exposed to alcohol in utero.” 2016. Web. 18 Jan 2021.

Vancouver:

Stires H. Elucidating mechanisms that drive increased tumorigenesis in rats exposed to alcohol in utero. [Internet] [Doctoral dissertation]. Rutgers University; 2016. [cited 2021 Jan 18]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/50188/.

Council of Science Editors:

Stires H. Elucidating mechanisms that drive increased tumorigenesis in rats exposed to alcohol in utero. [Doctoral Dissertation]. Rutgers University; 2016. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/50188/


Rutgers University

5. Camp, Meredith E., 1985-. Lactocrine signaling and neonatal porcine reproductive tract development.

Degree: PhD, Animal Sciences, 2015, Rutgers University

Nursing supports neonatal porcine reproductive tract development by delivering milk-borne bioactive factors (MbFs) from mother to offspring as proposed in the lactocrine hypothesis. In pigs,… (more)

Subjects/Keywords: Colostrum; Milk – Composition; Swine – Fetuses

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APA (6th Edition):

Camp, Meredith E., 1. (2015). Lactocrine signaling and neonatal porcine reproductive tract development. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/46315/

Chicago Manual of Style (16th Edition):

Camp, Meredith E., 1985-. “Lactocrine signaling and neonatal porcine reproductive tract development.” 2015. Doctoral Dissertation, Rutgers University. Accessed January 18, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/46315/.

MLA Handbook (7th Edition):

Camp, Meredith E., 1985-. “Lactocrine signaling and neonatal porcine reproductive tract development.” 2015. Web. 18 Jan 2021.

Vancouver:

Camp, Meredith E. 1. Lactocrine signaling and neonatal porcine reproductive tract development. [Internet] [Doctoral dissertation]. Rutgers University; 2015. [cited 2021 Jan 18]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/46315/.

Council of Science Editors:

Camp, Meredith E. 1. Lactocrine signaling and neonatal porcine reproductive tract development. [Doctoral Dissertation]. Rutgers University; 2015. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/46315/


Rutgers University

6. Datar, Ketaki Rajendra, 1994-. The effect of alcohol consumption on mammary epithelial cell composition and mammary tumorigenesis.

Degree: MS, Microbiology and Molecular Genetics, 2019, Rutgers University

 Breast cancer is the most common cancer in women worldwide and in 2019 it is estimated that approximately 41,000 women will die from the disease.… (more)

Subjects/Keywords: Mammary; Breast  – Cancer; Alcohol  – Physiological effect

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APA (6th Edition):

Datar, Ketaki Rajendra, 1. (2019). The effect of alcohol consumption on mammary epithelial cell composition and mammary tumorigenesis. (Masters Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/61726/

Chicago Manual of Style (16th Edition):

Datar, Ketaki Rajendra, 1994-. “The effect of alcohol consumption on mammary epithelial cell composition and mammary tumorigenesis.” 2019. Masters Thesis, Rutgers University. Accessed January 18, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/61726/.

MLA Handbook (7th Edition):

Datar, Ketaki Rajendra, 1994-. “The effect of alcohol consumption on mammary epithelial cell composition and mammary tumorigenesis.” 2019. Web. 18 Jan 2021.

Vancouver:

Datar, Ketaki Rajendra 1. The effect of alcohol consumption on mammary epithelial cell composition and mammary tumorigenesis. [Internet] [Masters thesis]. Rutgers University; 2019. [cited 2021 Jan 18]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/61726/.

Council of Science Editors:

Datar, Ketaki Rajendra 1. The effect of alcohol consumption on mammary epithelial cell composition and mammary tumorigenesis. [Masters Thesis]. Rutgers University; 2019. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/61726/


Rutgers University

7. Duncan, Christine, 1981-. Nutritional and hormonal modulation of insulin-like growth factor-1 with respect to growth in sexually dimorphic lizards.

Degree: PhD, Animal Sciences, 2011, Rutgers University

In many species of animals, adults of one sex grow faster or for a longer period of time to become larger than the other. Sex… (more)

Subjects/Keywords: Lizards – Growth; Sexual dimorphism (Animals); Molecular cloning

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APA (6th Edition):

Duncan, Christine, 1. (2011). Nutritional and hormonal modulation of insulin-like growth factor-1 with respect to growth in sexually dimorphic lizards. (Doctoral Dissertation). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000061190

Chicago Manual of Style (16th Edition):

Duncan, Christine, 1981-. “Nutritional and hormonal modulation of insulin-like growth factor-1 with respect to growth in sexually dimorphic lizards.” 2011. Doctoral Dissertation, Rutgers University. Accessed January 18, 2021. http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000061190.

MLA Handbook (7th Edition):

Duncan, Christine, 1981-. “Nutritional and hormonal modulation of insulin-like growth factor-1 with respect to growth in sexually dimorphic lizards.” 2011. Web. 18 Jan 2021.

Vancouver:

Duncan, Christine 1. Nutritional and hormonal modulation of insulin-like growth factor-1 with respect to growth in sexually dimorphic lizards. [Internet] [Doctoral dissertation]. Rutgers University; 2011. [cited 2021 Jan 18]. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000061190.

Council of Science Editors:

Duncan, Christine 1. Nutritional and hormonal modulation of insulin-like growth factor-1 with respect to growth in sexually dimorphic lizards. [Doctoral Dissertation]. Rutgers University; 2011. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000061190


Rutgers University

8. Saboya, Mariana de Andrade, 1990-. The effects of fetal alcohol exposure on mammary epithelial cell subpopulations and tumorigenesis.

Degree: PhD, Endocrinology and Animal Biosciences, 2019, Rutgers University

Previous work from our laboratory found that alcohol exposure in utero using the Lieber-DeCarli diet increases the risk of carcinogen-induced mammary tumorigenesis in adult rat… (more)

Subjects/Keywords: Breast  – Cancer; Rats  – Fetuses  – Effect of drugs on; Alcoholism in pregnancy  – Complications

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APA (6th Edition):

Saboya, Mariana de Andrade, 1. (2019). The effects of fetal alcohol exposure on mammary epithelial cell subpopulations and tumorigenesis. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/61929/

Chicago Manual of Style (16th Edition):

Saboya, Mariana de Andrade, 1990-. “The effects of fetal alcohol exposure on mammary epithelial cell subpopulations and tumorigenesis.” 2019. Doctoral Dissertation, Rutgers University. Accessed January 18, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/61929/.

MLA Handbook (7th Edition):

Saboya, Mariana de Andrade, 1990-. “The effects of fetal alcohol exposure on mammary epithelial cell subpopulations and tumorigenesis.” 2019. Web. 18 Jan 2021.

Vancouver:

Saboya, Mariana de Andrade 1. The effects of fetal alcohol exposure on mammary epithelial cell subpopulations and tumorigenesis. [Internet] [Doctoral dissertation]. Rutgers University; 2019. [cited 2021 Jan 18]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/61929/.

Council of Science Editors:

Saboya, Mariana de Andrade 1. The effects of fetal alcohol exposure on mammary epithelial cell subpopulations and tumorigenesis. [Doctoral Dissertation]. Rutgers University; 2019. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/61929/

9. Hanke, Jennifer, 1988-. Role of NFkB and Bcl-2 in IGFBP-3 mediated intrinsic apoptosis.

Degree: MS, Endocrinology and Animal Biosciences, 2017, Rutgers University

 The overall goal of this work was to investigate the mechanisms by which IGFBP-3 mediates ribotoxin-induced apoptosis in the non-transformed bovine mammary epithelial cell line… (more)

Subjects/Keywords: Insulin-like growth factor-binding proteins; Apoptosis

…provided by Dr. Rong Di, Rutgers University. Cell Culture The bovine mammary epithelial cell… 

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APA (6th Edition):

Hanke, Jennifer, 1. (2017). Role of NFkB and Bcl-2 in IGFBP-3 mediated intrinsic apoptosis. (Masters Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/53632/

Chicago Manual of Style (16th Edition):

Hanke, Jennifer, 1988-. “Role of NFkB and Bcl-2 in IGFBP-3 mediated intrinsic apoptosis.” 2017. Masters Thesis, Rutgers University. Accessed January 18, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/53632/.

MLA Handbook (7th Edition):

Hanke, Jennifer, 1988-. “Role of NFkB and Bcl-2 in IGFBP-3 mediated intrinsic apoptosis.” 2017. Web. 18 Jan 2021.

Vancouver:

Hanke, Jennifer 1. Role of NFkB and Bcl-2 in IGFBP-3 mediated intrinsic apoptosis. [Internet] [Masters thesis]. Rutgers University; 2017. [cited 2021 Jan 18]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/53632/.

Council of Science Editors:

Hanke, Jennifer 1. Role of NFkB and Bcl-2 in IGFBP-3 mediated intrinsic apoptosis. [Masters Thesis]. Rutgers University; 2017. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/53632/

10. Cheng, Ju-Shun, 1978-. Molecular mechanisms of cytotoxicity induced by ribosome-inactivating proteins in mammalian cells.

Degree: MS, Animal Sciences, 2010, Rutgers University

 Ribosome-inactivating proteins (RIP) represent a family of plant and bacterial toxins that inhibit protein synthesis and are cytotoxic to mammalian cells. Ricin is isolated from… (more)

Subjects/Keywords: Ribosomes; Cell-mediated cytotoxicity; Ricin; Apoptosis; Verocytotoxins

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APA (6th Edition):

Cheng, Ju-Shun, 1. (2010). Molecular mechanisms of cytotoxicity induced by ribosome-inactivating proteins in mammalian cells. (Masters Thesis). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000056279

Chicago Manual of Style (16th Edition):

Cheng, Ju-Shun, 1978-. “Molecular mechanisms of cytotoxicity induced by ribosome-inactivating proteins in mammalian cells.” 2010. Masters Thesis, Rutgers University. Accessed January 18, 2021. http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000056279.

MLA Handbook (7th Edition):

Cheng, Ju-Shun, 1978-. “Molecular mechanisms of cytotoxicity induced by ribosome-inactivating proteins in mammalian cells.” 2010. Web. 18 Jan 2021.

Vancouver:

Cheng, Ju-Shun 1. Molecular mechanisms of cytotoxicity induced by ribosome-inactivating proteins in mammalian cells. [Internet] [Masters thesis]. Rutgers University; 2010. [cited 2021 Jan 18]. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000056279.

Council of Science Editors:

Cheng, Ju-Shun 1. Molecular mechanisms of cytotoxicity induced by ribosome-inactivating proteins in mammalian cells. [Masters Thesis]. Rutgers University; 2010. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000056279


Rutgers University

11. Wang, Chao-ting, 1982-. Ricin-A-chain (RTA) inhibits the unfolded protein response (UPR) in mammalian cells.

Degree: MS, Nutritional Sciences, 2010, Rutgers University

 Ricin, a type II ribosome-inactivating protein, has been used as a biochemical weapon due to its ability to inhibit protein synthesis and induce cytotoxicity. The… (more)

Subjects/Keywords: Ricin – Analysis; Ricin – Denaturation; Cell-mediated cytotoxicity

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APA (6th Edition):

Wang, Chao-ting, 1. (2010). Ricin-A-chain (RTA) inhibits the unfolded protein response (UPR) in mammalian cells. (Masters Thesis). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000056855

Chicago Manual of Style (16th Edition):

Wang, Chao-ting, 1982-. “Ricin-A-chain (RTA) inhibits the unfolded protein response (UPR) in mammalian cells.” 2010. Masters Thesis, Rutgers University. Accessed January 18, 2021. http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000056855.

MLA Handbook (7th Edition):

Wang, Chao-ting, 1982-. “Ricin-A-chain (RTA) inhibits the unfolded protein response (UPR) in mammalian cells.” 2010. Web. 18 Jan 2021.

Vancouver:

Wang, Chao-ting 1. Ricin-A-chain (RTA) inhibits the unfolded protein response (UPR) in mammalian cells. [Internet] [Masters thesis]. Rutgers University; 2010. [cited 2021 Jan 18]. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000056855.

Council of Science Editors:

Wang, Chao-ting 1. Ricin-A-chain (RTA) inhibits the unfolded protein response (UPR) in mammalian cells. [Masters Thesis]. Rutgers University; 2010. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000056855


Rutgers University

12. Zhang, Changqing, 1984-. Role of beta-endorphin in control of stress and cancer progression in fetal alcohol exposed rats.

Degree: Animal Sciences, 2013, Rutgers University

Subjects/Keywords: Endorphins; Rats – Effect of stress on; Alcohol – Physiological effect; Cancer – Etiology; Cancer in animals

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APA (6th Edition):

Zhang, Changqing, 1. (2013). Role of beta-endorphin in control of stress and cancer progression in fetal alcohol exposed rats. (Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/42092/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Zhang, Changqing, 1984-. “Role of beta-endorphin in control of stress and cancer progression in fetal alcohol exposed rats.” 2013. Thesis, Rutgers University. Accessed January 18, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/42092/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Zhang, Changqing, 1984-. “Role of beta-endorphin in control of stress and cancer progression in fetal alcohol exposed rats.” 2013. Web. 18 Jan 2021.

Vancouver:

Zhang, Changqing 1. Role of beta-endorphin in control of stress and cancer progression in fetal alcohol exposed rats. [Internet] [Thesis]. Rutgers University; 2013. [cited 2021 Jan 18]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/42092/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Zhang, Changqing 1. Role of beta-endorphin in control of stress and cancer progression in fetal alcohol exposed rats. [Thesis]. Rutgers University; 2013. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/42092/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rutgers University

13. Crismale-Gann, Catina, 1982-. Effect of fetal alcohol exposure on mammary gland development and tumorigenesis in offspring.

Degree: Cell and Developmental Biology, 2014, Rutgers University

Subjects/Keywords: Fetal alcohol syndrome; Carcinogenesis – Animal models

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APA (6th Edition):

Crismale-Gann, Catina, 1. (2014). Effect of fetal alcohol exposure on mammary gland development and tumorigenesis in offspring. (Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/44065/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Crismale-Gann, Catina, 1982-. “Effect of fetal alcohol exposure on mammary gland development and tumorigenesis in offspring.” 2014. Thesis, Rutgers University. Accessed January 18, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/44065/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Crismale-Gann, Catina, 1982-. “Effect of fetal alcohol exposure on mammary gland development and tumorigenesis in offspring.” 2014. Web. 18 Jan 2021.

Vancouver:

Crismale-Gann, Catina 1. Effect of fetal alcohol exposure on mammary gland development and tumorigenesis in offspring. [Internet] [Thesis]. Rutgers University; 2014. [cited 2021 Jan 18]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/44065/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Crismale-Gann, Catina 1. Effect of fetal alcohol exposure on mammary gland development and tumorigenesis in offspring. [Thesis]. Rutgers University; 2014. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/44065/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rutgers University

14. Agostini-Dreyer, Allyson M., 1986-. Role of endogenous IGFBP-3 in mammary epithelial cell apoptosis.

Degree: Nutritional Sciences, 2014, Rutgers University

Subjects/Keywords: Somatomedin; Epithelial cells

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APA (6th Edition):

Agostini-Dreyer, Allyson M., 1. (2014). Role of endogenous IGFBP-3 in mammary epithelial cell apoptosis. (Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/43942/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Agostini-Dreyer, Allyson M., 1986-. “Role of endogenous IGFBP-3 in mammary epithelial cell apoptosis.” 2014. Thesis, Rutgers University. Accessed January 18, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/43942/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Agostini-Dreyer, Allyson M., 1986-. “Role of endogenous IGFBP-3 in mammary epithelial cell apoptosis.” 2014. Web. 18 Jan 2021.

Vancouver:

Agostini-Dreyer, Allyson M. 1. Role of endogenous IGFBP-3 in mammary epithelial cell apoptosis. [Internet] [Thesis]. Rutgers University; 2014. [cited 2021 Jan 18]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/43942/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Agostini-Dreyer, Allyson M. 1. Role of endogenous IGFBP-3 in mammary epithelial cell apoptosis. [Thesis]. Rutgers University; 2014. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/43942/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.