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You searched for +publisher:"Rutgers University" +contributor:("Banerjee, Debabrata"). Showing records 1 – 9 of 9 total matches.

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Rutgers University

1. Aijaz, Ayesha, 1988-. Coencapsulation of insulin-producing cells and mesenchymal stromal cells in PEGDA hydrogels to enhance chronic wound healing.

Degree: PhD, Biomedical Engineering, 2017, Rutgers University

 Wound healing is a hierarchical process of intracellular and intercellular signaling. Insulin is a potent chemoattractant and mitogen for cells involved in wound healing. Insulin’s… (more)

Subjects/Keywords: Wound healing; Wounds and injuries

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APA (6th Edition):

Aijaz, Ayesha, 1. (2017). Coencapsulation of insulin-producing cells and mesenchymal stromal cells in PEGDA hydrogels to enhance chronic wound healing. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/53063/

Chicago Manual of Style (16th Edition):

Aijaz, Ayesha, 1988-. “Coencapsulation of insulin-producing cells and mesenchymal stromal cells in PEGDA hydrogels to enhance chronic wound healing.” 2017. Doctoral Dissertation, Rutgers University. Accessed April 12, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/53063/.

MLA Handbook (7th Edition):

Aijaz, Ayesha, 1988-. “Coencapsulation of insulin-producing cells and mesenchymal stromal cells in PEGDA hydrogels to enhance chronic wound healing.” 2017. Web. 12 Apr 2021.

Vancouver:

Aijaz, Ayesha 1. Coencapsulation of insulin-producing cells and mesenchymal stromal cells in PEGDA hydrogels to enhance chronic wound healing. [Internet] [Doctoral dissertation]. Rutgers University; 2017. [cited 2021 Apr 12]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/53063/.

Council of Science Editors:

Aijaz, Ayesha 1. Coencapsulation of insulin-producing cells and mesenchymal stromal cells in PEGDA hydrogels to enhance chronic wound healing. [Doctoral Dissertation]. Rutgers University; 2017. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/53063/


Rutgers University

2. Gray, Andrea, 1987-. Approaches to improving the therapeutic potential of Mesenchymal Stromal Cells.

Degree: PhD, Biomedical Engineering, 2015, Rutgers University

Mesenchymal stromal cells (MSCs) hold great potential as a cellular therapy due in part to their tissue protective and regenerative abilities, achieved via the secretion… (more)

Subjects/Keywords: Immune response – Regulation; Cellular therapy

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APA (6th Edition):

Gray, Andrea, 1. (2015). Approaches to improving the therapeutic potential of Mesenchymal Stromal Cells. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/48498/

Chicago Manual of Style (16th Edition):

Gray, Andrea, 1987-. “Approaches to improving the therapeutic potential of Mesenchymal Stromal Cells.” 2015. Doctoral Dissertation, Rutgers University. Accessed April 12, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/48498/.

MLA Handbook (7th Edition):

Gray, Andrea, 1987-. “Approaches to improving the therapeutic potential of Mesenchymal Stromal Cells.” 2015. Web. 12 Apr 2021.

Vancouver:

Gray, Andrea 1. Approaches to improving the therapeutic potential of Mesenchymal Stromal Cells. [Internet] [Doctoral dissertation]. Rutgers University; 2015. [cited 2021 Apr 12]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/48498/.

Council of Science Editors:

Gray, Andrea 1. Approaches to improving the therapeutic potential of Mesenchymal Stromal Cells. [Doctoral Dissertation]. Rutgers University; 2015. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/48498/


Rutgers University

3. Al-Asadi, Amer, 1980-. Preventing and treating hepatic metastases of colon and pancreatic cancers by targeting cancer cell metabolism.

Degree: PhD, Physiology and Integrative Biology, 2018, Rutgers University

Alteration of glucose metabolism is a unique feature for a majority of cancers. Cancer cells exhibit aerobic glycolysis also known as the Warburg effect even… (more)

Subjects/Keywords: Cancer cells; Pancreas – Cancer

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APA (6th Edition):

Al-Asadi, Amer, 1. (2018). Preventing and treating hepatic metastases of colon and pancreatic cancers by targeting cancer cell metabolism. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/57463/

Chicago Manual of Style (16th Edition):

Al-Asadi, Amer, 1980-. “Preventing and treating hepatic metastases of colon and pancreatic cancers by targeting cancer cell metabolism.” 2018. Doctoral Dissertation, Rutgers University. Accessed April 12, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/57463/.

MLA Handbook (7th Edition):

Al-Asadi, Amer, 1980-. “Preventing and treating hepatic metastases of colon and pancreatic cancers by targeting cancer cell metabolism.” 2018. Web. 12 Apr 2021.

Vancouver:

Al-Asadi, Amer 1. Preventing and treating hepatic metastases of colon and pancreatic cancers by targeting cancer cell metabolism. [Internet] [Doctoral dissertation]. Rutgers University; 2018. [cited 2021 Apr 12]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/57463/.

Council of Science Editors:

Al-Asadi, Amer 1. Preventing and treating hepatic metastases of colon and pancreatic cancers by targeting cancer cell metabolism. [Doctoral Dissertation]. Rutgers University; 2018. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/57463/


Rutgers University

4. Collantes, Juan Carlos, 1981-. A novel CRISPR/RNA-aptamer-mediated base editing system with potential therapeutic value.

Degree: PhD, Pharmacology, Cellular and Molecular, 2019, Rutgers University

Nuclease-dependent precise genome editing such as correction of point mutations requires introduction of targeted DNA double strand breaks (DSB) and activation of homology dependent repair… (more)

Subjects/Keywords: Gene editing  – Therapeutic use; CRISPR-associated protein 9

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APA (6th Edition):

Collantes, Juan Carlos, 1. (2019). A novel CRISPR/RNA-aptamer-mediated base editing system with potential therapeutic value. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/60101/

Chicago Manual of Style (16th Edition):

Collantes, Juan Carlos, 1981-. “A novel CRISPR/RNA-aptamer-mediated base editing system with potential therapeutic value.” 2019. Doctoral Dissertation, Rutgers University. Accessed April 12, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/60101/.

MLA Handbook (7th Edition):

Collantes, Juan Carlos, 1981-. “A novel CRISPR/RNA-aptamer-mediated base editing system with potential therapeutic value.” 2019. Web. 12 Apr 2021.

Vancouver:

Collantes, Juan Carlos 1. A novel CRISPR/RNA-aptamer-mediated base editing system with potential therapeutic value. [Internet] [Doctoral dissertation]. Rutgers University; 2019. [cited 2021 Apr 12]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/60101/.

Council of Science Editors:

Collantes, Juan Carlos 1. A novel CRISPR/RNA-aptamer-mediated base editing system with potential therapeutic value. [Doctoral Dissertation]. Rutgers University; 2019. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/60101/


Rutgers University

5. Ekwueme, Emmanuel C., 1987-. Non-surgical tissue engineering approaches for sub-failure ligament and tendon injury repair.

Degree: PhD, Biomedical Engineering, 2015, Rutgers University

 Ligaments and tendons are dense collagenous tissues that assist the body during locomotion and mechanically stabilize joints by operating primarily in tension. During sports and… (more)

Subjects/Keywords: Tissue engineering; Ligaments; Tendons

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APA (6th Edition):

Ekwueme, Emmanuel C., 1. (2015). Non-surgical tissue engineering approaches for sub-failure ligament and tendon injury repair. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/47360/

Chicago Manual of Style (16th Edition):

Ekwueme, Emmanuel C., 1987-. “Non-surgical tissue engineering approaches for sub-failure ligament and tendon injury repair.” 2015. Doctoral Dissertation, Rutgers University. Accessed April 12, 2021. https://rucore.libraries.rutgers.edu/rutgers-lib/47360/.

MLA Handbook (7th Edition):

Ekwueme, Emmanuel C., 1987-. “Non-surgical tissue engineering approaches for sub-failure ligament and tendon injury repair.” 2015. Web. 12 Apr 2021.

Vancouver:

Ekwueme, Emmanuel C. 1. Non-surgical tissue engineering approaches for sub-failure ligament and tendon injury repair. [Internet] [Doctoral dissertation]. Rutgers University; 2015. [cited 2021 Apr 12]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/47360/.

Council of Science Editors:

Ekwueme, Emmanuel C. 1. Non-surgical tissue engineering approaches for sub-failure ligament and tendon injury repair. [Doctoral Dissertation]. Rutgers University; 2015. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/47360/

6. Hamed, Salaheldin. Mechanisms of chemotherapeutic resistance in glioma: mathematical modeling and gene expression profiling.

Degree: PhD, Biomedical Engineering, 2010, Rutgers University

Gliomas are refractory to chemotherapy because of acquired resistance, which is associated with changes in important cellular processes, such as cell cycle kinetics and cell… (more)

Subjects/Keywords: Gliomas – Chemotherapy; Drugs – Side effects; Gene expression

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APA (6th Edition):

Hamed, S. (2010). Mechanisms of chemotherapeutic resistance in glioma: mathematical modeling and gene expression profiling. (Doctoral Dissertation). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000053568

Chicago Manual of Style (16th Edition):

Hamed, Salaheldin. “Mechanisms of chemotherapeutic resistance in glioma: mathematical modeling and gene expression profiling.” 2010. Doctoral Dissertation, Rutgers University. Accessed April 12, 2021. http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000053568.

MLA Handbook (7th Edition):

Hamed, Salaheldin. “Mechanisms of chemotherapeutic resistance in glioma: mathematical modeling and gene expression profiling.” 2010. Web. 12 Apr 2021.

Vancouver:

Hamed S. Mechanisms of chemotherapeutic resistance in glioma: mathematical modeling and gene expression profiling. [Internet] [Doctoral dissertation]. Rutgers University; 2010. [cited 2021 Apr 12]. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000053568.

Council of Science Editors:

Hamed S. Mechanisms of chemotherapeutic resistance in glioma: mathematical modeling and gene expression profiling. [Doctoral Dissertation]. Rutgers University; 2010. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000053568

7. Barminko, Jeffrey Avi, 1985-. Encapsulated Mesenchymal stromal cells for spinal cord injury repair.

Degree: Biomedical Engineering, 2012, Rutgers University

Subjects/Keywords: Spinal cord – Wounds and injuries – Treatment; Mesenchymal stem cells – Transplantation

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APA (6th Edition):

Barminko, Jeffrey Avi, 1. (2012). Encapsulated Mesenchymal stromal cells for spinal cord injury repair. (Thesis). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000066610

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Barminko, Jeffrey Avi, 1985-. “Encapsulated Mesenchymal stromal cells for spinal cord injury repair.” 2012. Thesis, Rutgers University. Accessed April 12, 2021. http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000066610.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Barminko, Jeffrey Avi, 1985-. “Encapsulated Mesenchymal stromal cells for spinal cord injury repair.” 2012. Web. 12 Apr 2021.

Vancouver:

Barminko, Jeffrey Avi 1. Encapsulated Mesenchymal stromal cells for spinal cord injury repair. [Internet] [Thesis]. Rutgers University; 2012. [cited 2021 Apr 12]. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000066610.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Barminko, Jeffrey Avi 1. Encapsulated Mesenchymal stromal cells for spinal cord injury repair. [Thesis]. Rutgers University; 2012. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000066610

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rutgers University

8. Piso, Katherine. CBX2 and DNA damage repair: development of CBX2-specific reagents.

Degree: MS, Microbiology and Molecular Genetics, 2008, Rutgers University

 DNA damage is inevitable, however, methods of DNA repair exist which allows cells to recover, or die, depending on the severity of the damage. Homologous… (more)

Subjects/Keywords: DNA repair; DNA-protein interactions

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APA (6th Edition):

Piso, K. (2008). CBX2 and DNA damage repair: development of CBX2-specific reagents. (Masters Thesis). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17370

Chicago Manual of Style (16th Edition):

Piso, Katherine. “CBX2 and DNA damage repair: development of CBX2-specific reagents.” 2008. Masters Thesis, Rutgers University. Accessed April 12, 2021. http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17370.

MLA Handbook (7th Edition):

Piso, Katherine. “CBX2 and DNA damage repair: development of CBX2-specific reagents.” 2008. Web. 12 Apr 2021.

Vancouver:

Piso K. CBX2 and DNA damage repair: development of CBX2-specific reagents. [Internet] [Masters thesis]. Rutgers University; 2008. [cited 2021 Apr 12]. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17370.

Council of Science Editors:

Piso K. CBX2 and DNA damage repair: development of CBX2-specific reagents. [Masters Thesis]. Rutgers University; 2008. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.17370


Rutgers University

9. Sohail, Honeah, 1977. PARP inhibitor ABT-888 as potentiating agent for topoisomerase inhibitor SN-38.

Degree: MS, Microbiology and Molecular Genetics, 2009, Rutgers University

 BRCA1 is a tumor suppressor gene that has been implicated as being involved in DNA repair through a process known as homologous recombination. Mutations in… (more)

Subjects/Keywords: DNA topoisomerases; NAD-ADP-ribosyltransferase – Inhibitors

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APA (6th Edition):

Sohail, Honeah, 1. (2009). PARP inhibitor ABT-888 as potentiating agent for topoisomerase inhibitor SN-38. (Masters Thesis). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000051407

Chicago Manual of Style (16th Edition):

Sohail, Honeah, 1977. “PARP inhibitor ABT-888 as potentiating agent for topoisomerase inhibitor SN-38.” 2009. Masters Thesis, Rutgers University. Accessed April 12, 2021. http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000051407.

MLA Handbook (7th Edition):

Sohail, Honeah, 1977. “PARP inhibitor ABT-888 as potentiating agent for topoisomerase inhibitor SN-38.” 2009. Web. 12 Apr 2021.

Vancouver:

Sohail, Honeah 1. PARP inhibitor ABT-888 as potentiating agent for topoisomerase inhibitor SN-38. [Internet] [Masters thesis]. Rutgers University; 2009. [cited 2021 Apr 12]. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000051407.

Council of Science Editors:

Sohail, Honeah 1. PARP inhibitor ABT-888 as potentiating agent for topoisomerase inhibitor SN-38. [Masters Thesis]. Rutgers University; 2009. Available from: http://hdl.rutgers.edu/1782.2/rucore10001600001.ETD.000051407

.