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You searched for +publisher:"Rutgers University" +contributor:("Alder, Janet"). Showing records 1 – 9 of 9 total matches.

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Rutgers University

1. Nikolaeva, Ina. Targeting the PI3K/Akt/mTOR pathway in traumatic brain injury and developmental disease.

Degree: PhD, Microbiology and Molecular Genetics, 2016, Rutgers University

The PI3K/Akt/mTOR signaling pathway mediates many aspects of cell growth and regeneration. Dysregulation of the pathway during development or following injury can lead to severe… (more)

Subjects/Keywords: Biology, Molecular; Barin – Wounds and injuries

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APA (6th Edition):

Nikolaeva, I. (2016). Targeting the PI3K/Akt/mTOR pathway in traumatic brain injury and developmental disease. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/50108/

Chicago Manual of Style (16th Edition):

Nikolaeva, Ina. “Targeting the PI3K/Akt/mTOR pathway in traumatic brain injury and developmental disease.” 2016. Doctoral Dissertation, Rutgers University. Accessed July 11, 2020. https://rucore.libraries.rutgers.edu/rutgers-lib/50108/.

MLA Handbook (7th Edition):

Nikolaeva, Ina. “Targeting the PI3K/Akt/mTOR pathway in traumatic brain injury and developmental disease.” 2016. Web. 11 Jul 2020.

Vancouver:

Nikolaeva I. Targeting the PI3K/Akt/mTOR pathway in traumatic brain injury and developmental disease. [Internet] [Doctoral dissertation]. Rutgers University; 2016. [cited 2020 Jul 11]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/50108/.

Council of Science Editors:

Nikolaeva I. Targeting the PI3K/Akt/mTOR pathway in traumatic brain injury and developmental disease. [Doctoral Dissertation]. Rutgers University; 2016. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/50108/


Rutgers University

2. Dal Pozzo, Valentina. The role of Reelin after traumatic brain injury.

Degree: PhD, Neuroscience, 2019, Rutgers University

Traumatic brain injury (TBI) causes severe cognitive disability or death, resulting from common occurrences as car accidents, falls, high-impact sport, violence, or explosive blasts. Statistical… (more)

Subjects/Keywords: Traumatic brain injury; Brain  – Wounds and injuries  – Treatment

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APA (6th Edition):

Dal Pozzo, V. (2019). The role of Reelin after traumatic brain injury. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/61724/

Chicago Manual of Style (16th Edition):

Dal Pozzo, Valentina. “The role of Reelin after traumatic brain injury.” 2019. Doctoral Dissertation, Rutgers University. Accessed July 11, 2020. https://rucore.libraries.rutgers.edu/rutgers-lib/61724/.

MLA Handbook (7th Edition):

Dal Pozzo, Valentina. “The role of Reelin after traumatic brain injury.” 2019. Web. 11 Jul 2020.

Vancouver:

Dal Pozzo V. The role of Reelin after traumatic brain injury. [Internet] [Doctoral dissertation]. Rutgers University; 2019. [cited 2020 Jul 11]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/61724/.

Council of Science Editors:

Dal Pozzo V. The role of Reelin after traumatic brain injury. [Doctoral Dissertation]. Rutgers University; 2019. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/61724/


Rutgers University

3. Lowe, Christopher J. Sustained presentation of therapeutic factors.

Degree: PhD, Biomedical Engineering, 2018, Rutgers University

The continued development of complex protein therapeutics as life saving medicines has greatly improved outcomes and quality of life for countless patients. Unfortunately, these large… (more)

Subjects/Keywords: Therapeutics; Pharmacology

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APA (6th Edition):

Lowe, C. J. (2018). Sustained presentation of therapeutic factors. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/59161/

Chicago Manual of Style (16th Edition):

Lowe, Christopher J. “Sustained presentation of therapeutic factors.” 2018. Doctoral Dissertation, Rutgers University. Accessed July 11, 2020. https://rucore.libraries.rutgers.edu/rutgers-lib/59161/.

MLA Handbook (7th Edition):

Lowe, Christopher J. “Sustained presentation of therapeutic factors.” 2018. Web. 11 Jul 2020.

Vancouver:

Lowe CJ. Sustained presentation of therapeutic factors. [Internet] [Doctoral dissertation]. Rutgers University; 2018. [cited 2020 Jul 11]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/59161/.

Council of Science Editors:

Lowe CJ. Sustained presentation of therapeutic factors. [Doctoral Dissertation]. Rutgers University; 2018. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/59161/


Rutgers University

4. Anderson, Jeremy, 1993-. Role of notch signaling after traumatic brain injury in a transgenic mouse model.

Degree: PhD, Biomedical Engineering, 2019, Rutgers University

Traumatic brain injury (TBI) is a leading cause of disability and death in the United States and worldwide. Endogenous neural stem/progenitor cells (NSPCs) in the… (more)

Subjects/Keywords: Neural stem cells; Transgenic mice

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APA (6th Edition):

Anderson, Jeremy, 1. (2019). Role of notch signaling after traumatic brain injury in a transgenic mouse model. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/61683/

Chicago Manual of Style (16th Edition):

Anderson, Jeremy, 1993-. “Role of notch signaling after traumatic brain injury in a transgenic mouse model.” 2019. Doctoral Dissertation, Rutgers University. Accessed July 11, 2020. https://rucore.libraries.rutgers.edu/rutgers-lib/61683/.

MLA Handbook (7th Edition):

Anderson, Jeremy, 1993-. “Role of notch signaling after traumatic brain injury in a transgenic mouse model.” 2019. Web. 11 Jul 2020.

Vancouver:

Anderson, Jeremy 1. Role of notch signaling after traumatic brain injury in a transgenic mouse model. [Internet] [Doctoral dissertation]. Rutgers University; 2019. [cited 2020 Jul 11]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/61683/.

Council of Science Editors:

Anderson, Jeremy 1. Role of notch signaling after traumatic brain injury in a transgenic mouse model. [Doctoral Dissertation]. Rutgers University; 2019. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/61683/


Rutgers University

5. Patel, Mihir V., 1987-. The roles of postsynaptic density-95 interactors in dendrite development and recovery after traumatic brain injury.

Degree: PhD, Neuroscience, 2018, Rutgers University

Postsynaptic density 95 (PSD-95) is the major scaffolding protein at excitatory synapses, and it plays a major role in synaptic plasticity. Furthermore, PSD-95 and its… (more)

Subjects/Keywords: Dendrites; Brain damage

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APA (6th Edition):

Patel, Mihir V., 1. (2018). The roles of postsynaptic density-95 interactors in dendrite development and recovery after traumatic brain injury. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/59121/

Chicago Manual of Style (16th Edition):

Patel, Mihir V., 1987-. “The roles of postsynaptic density-95 interactors in dendrite development and recovery after traumatic brain injury.” 2018. Doctoral Dissertation, Rutgers University. Accessed July 11, 2020. https://rucore.libraries.rutgers.edu/rutgers-lib/59121/.

MLA Handbook (7th Edition):

Patel, Mihir V., 1987-. “The roles of postsynaptic density-95 interactors in dendrite development and recovery after traumatic brain injury.” 2018. Web. 11 Jul 2020.

Vancouver:

Patel, Mihir V. 1. The roles of postsynaptic density-95 interactors in dendrite development and recovery after traumatic brain injury. [Internet] [Doctoral dissertation]. Rutgers University; 2018. [cited 2020 Jul 11]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/59121/.

Council of Science Editors:

Patel, Mihir V. 1. The roles of postsynaptic density-95 interactors in dendrite development and recovery after traumatic brain injury. [Doctoral Dissertation]. Rutgers University; 2018. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/59121/


Rutgers University

6. Giarratana, Anna Obee, 1987-. The effects of genetic polymorphisms on recovery after repeated mild traumatic brain injury in a mouse model and personalized treatment approaches.

Degree: PhD, Traumatic brain injury, 2019, Rutgers University

Traumatic Brain Injury (TBI) is a serious and potentially life-threatening clinical problem. In 2013, there were 2.5 million TBIs in the United States, 50,000 of… (more)

Subjects/Keywords: Neuroscience; Brain  – Wounds and injuries; Single nucleotide polymorphisms

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APA (6th Edition):

Giarratana, Anna Obee, 1. (2019). The effects of genetic polymorphisms on recovery after repeated mild traumatic brain injury in a mouse model and personalized treatment approaches. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/60759/

Chicago Manual of Style (16th Edition):

Giarratana, Anna Obee, 1987-. “The effects of genetic polymorphisms on recovery after repeated mild traumatic brain injury in a mouse model and personalized treatment approaches.” 2019. Doctoral Dissertation, Rutgers University. Accessed July 11, 2020. https://rucore.libraries.rutgers.edu/rutgers-lib/60759/.

MLA Handbook (7th Edition):

Giarratana, Anna Obee, 1987-. “The effects of genetic polymorphisms on recovery after repeated mild traumatic brain injury in a mouse model and personalized treatment approaches.” 2019. Web. 11 Jul 2020.

Vancouver:

Giarratana, Anna Obee 1. The effects of genetic polymorphisms on recovery after repeated mild traumatic brain injury in a mouse model and personalized treatment approaches. [Internet] [Doctoral dissertation]. Rutgers University; 2019. [cited 2020 Jul 11]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/60759/.

Council of Science Editors:

Giarratana, Anna Obee 1. The effects of genetic polymorphisms on recovery after repeated mild traumatic brain injury in a mouse model and personalized treatment approaches. [Doctoral Dissertation]. Rutgers University; 2019. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/60759/

7. Swiatkowski, Przemyslaw, 1987-. Traumatic brain injury: novel targets for potential therapies.

Degree: PhD, Pharmacology, Cellular and Molecular, 2018, Rutgers University

Traumatic brain injury (TBI) induces severe neurological damage leading to significant deficits in learning and memory and poor quality of life. It is a pathology… (more)

Subjects/Keywords: Brain – Wounds and injuries – Treatment

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APA (6th Edition):

Swiatkowski, Przemyslaw, 1. (2018). Traumatic brain injury: novel targets for potential therapies. (Doctoral Dissertation). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/56146/

Chicago Manual of Style (16th Edition):

Swiatkowski, Przemyslaw, 1987-. “Traumatic brain injury: novel targets for potential therapies.” 2018. Doctoral Dissertation, Rutgers University. Accessed July 11, 2020. https://rucore.libraries.rutgers.edu/rutgers-lib/56146/.

MLA Handbook (7th Edition):

Swiatkowski, Przemyslaw, 1987-. “Traumatic brain injury: novel targets for potential therapies.” 2018. Web. 11 Jul 2020.

Vancouver:

Swiatkowski, Przemyslaw 1. Traumatic brain injury: novel targets for potential therapies. [Internet] [Doctoral dissertation]. Rutgers University; 2018. [cited 2020 Jul 11]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/56146/.

Council of Science Editors:

Swiatkowski, Przemyslaw 1. Traumatic brain injury: novel targets for potential therapies. [Doctoral Dissertation]. Rutgers University; 2018. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/56146/

8. Komlos, Daniel, 1979-. Expression and function of SIRT1, SIRT4, and glutamate dehydrogenase 1 within the central nervous system.

Degree: Neuroscience, 2011, Rutgers University

Subjects/Keywords: Glutamate dehydrogenase; Central nervous system—Metabolism

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APA (6th Edition):

Komlos, Daniel, 1. (2011). Expression and function of SIRT1, SIRT4, and glutamate dehydrogenase 1 within the central nervous system. (Thesis). Rutgers University. Retrieved from http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000063504

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Komlos, Daniel, 1979-. “Expression and function of SIRT1, SIRT4, and glutamate dehydrogenase 1 within the central nervous system.” 2011. Thesis, Rutgers University. Accessed July 11, 2020. http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000063504.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Komlos, Daniel, 1979-. “Expression and function of SIRT1, SIRT4, and glutamate dehydrogenase 1 within the central nervous system.” 2011. Web. 11 Jul 2020.

Vancouver:

Komlos, Daniel 1. Expression and function of SIRT1, SIRT4, and glutamate dehydrogenase 1 within the central nervous system. [Internet] [Thesis]. Rutgers University; 2011. [cited 2020 Jul 11]. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000063504.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Komlos, Daniel 1. Expression and function of SIRT1, SIRT4, and glutamate dehydrogenase 1 within the central nervous system. [Thesis]. Rutgers University; 2011. Available from: http://hdl.rutgers.edu/1782.1/rucore10001600001.ETD.000063504

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Rutgers University

9. Franklin, Tina Carla, 1985-. The immediate and long term effects of microglial cell activation on stress response in fetal alcohol exposure model.

Degree: Cell and Developmental Biology, 2013, Rutgers University

Subjects/Keywords: Fetal alcohol syndrome; Microglia; Stress (Physiology) – Endocrine aspects

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APA (6th Edition):

Franklin, Tina Carla, 1. (2013). The immediate and long term effects of microglial cell activation on stress response in fetal alcohol exposure model. (Thesis). Rutgers University. Retrieved from https://rucore.libraries.rutgers.edu/rutgers-lib/41769/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Franklin, Tina Carla, 1985-. “The immediate and long term effects of microglial cell activation on stress response in fetal alcohol exposure model.” 2013. Thesis, Rutgers University. Accessed July 11, 2020. https://rucore.libraries.rutgers.edu/rutgers-lib/41769/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Franklin, Tina Carla, 1985-. “The immediate and long term effects of microglial cell activation on stress response in fetal alcohol exposure model.” 2013. Web. 11 Jul 2020.

Vancouver:

Franklin, Tina Carla 1. The immediate and long term effects of microglial cell activation on stress response in fetal alcohol exposure model. [Internet] [Thesis]. Rutgers University; 2013. [cited 2020 Jul 11]. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/41769/.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Franklin, Tina Carla 1. The immediate and long term effects of microglial cell activation on stress response in fetal alcohol exposure model. [Thesis]. Rutgers University; 2013. Available from: https://rucore.libraries.rutgers.edu/rutgers-lib/41769/

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.