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You searched for +publisher:"Penn State University" +contributor:("Lisa M Shantz, Committee Member"). Showing records 1 – 29 of 29 total matches.

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Penn State University

1. Hernandez Borrero, Liz Janice. Anti-tumor effect and destabilization of mutant p53 by CB002, a p53-pathway restoring small molecule that stimulates autophagy.

Degree: 2016, Penn State University

 Tumor suppressor p53 mediates genotoxic and cellular stress signals by controlling cell fate through transcriptional activation of genes involved in DNA repair, cell cycle arrest,… (more)

Subjects/Keywords: mutant p53; apoptosis; autophagy; p53 pathway restoration; small molecules; NOXA

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APA (6th Edition):

Hernandez Borrero, L. J. (2016). Anti-tumor effect and destabilization of mutant p53 by CB002, a p53-pathway restoring small molecule that stimulates autophagy. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/13038ljh216

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Hernandez Borrero, Liz Janice. “Anti-tumor effect and destabilization of mutant p53 by CB002, a p53-pathway restoring small molecule that stimulates autophagy.” 2016. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/13038ljh216.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Hernandez Borrero, Liz Janice. “Anti-tumor effect and destabilization of mutant p53 by CB002, a p53-pathway restoring small molecule that stimulates autophagy.” 2016. Web. 09 Mar 2021.

Vancouver:

Hernandez Borrero LJ. Anti-tumor effect and destabilization of mutant p53 by CB002, a p53-pathway restoring small molecule that stimulates autophagy. [Internet] [Thesis]. Penn State University; 2016. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/13038ljh216.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hernandez Borrero LJ. Anti-tumor effect and destabilization of mutant p53 by CB002, a p53-pathway restoring small molecule that stimulates autophagy. [Thesis]. Penn State University; 2016. Available from: https://submit-etda.libraries.psu.edu/catalog/13038ljh216

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

2. Jones, Victoria M. THE ROLE OF SIRTUIN 3 IN OVARIAN CANCER METASTASIS.

Degree: 2016, Penn State University

 Epithelial ovarian cancer (EOC) is highly metastatic with late-onset symptoms, making it the leading cause of death in women with gynecologic malignancies. The proposed mechanism… (more)

Subjects/Keywords: SIRT3; Sirtuin 3; Ovarian Cancer; SOD2; Spheroids; Mitochondria

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APA (6th Edition):

Jones, V. M. (2016). THE ROLE OF SIRTUIN 3 IN OVARIAN CANCER METASTASIS. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/13078vmj2

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jones, Victoria M. “THE ROLE OF SIRTUIN 3 IN OVARIAN CANCER METASTASIS.” 2016. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/13078vmj2.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jones, Victoria M. “THE ROLE OF SIRTUIN 3 IN OVARIAN CANCER METASTASIS.” 2016. Web. 09 Mar 2021.

Vancouver:

Jones VM. THE ROLE OF SIRTUIN 3 IN OVARIAN CANCER METASTASIS. [Internet] [Thesis]. Penn State University; 2016. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/13078vmj2.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jones VM. THE ROLE OF SIRTUIN 3 IN OVARIAN CANCER METASTASIS. [Thesis]. Penn State University; 2016. Available from: https://submit-etda.libraries.psu.edu/catalog/13078vmj2

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

3. Walsh, Erin. Repetitive Dna Sequence Elements and Dna Polymerases Modeulate Instability in the Human Genome.

Degree: 2014, Penn State University

 Abstract Repetitive DNA sequences make up over 50% of the human genome and are located in both intra- and inter-genic regions. These sequences are prone… (more)

Subjects/Keywords: Specialized DNA Polymerase; Repetitive DNA Sequence; Replication Stalling; Microsatellite DNA; Common Fragile Sites

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APA (6th Edition):

Walsh, E. (2014). Repetitive Dna Sequence Elements and Dna Polymerases Modeulate Instability in the Human Genome. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/20639

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Walsh, Erin. “Repetitive Dna Sequence Elements and Dna Polymerases Modeulate Instability in the Human Genome.” 2014. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/20639.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Walsh, Erin. “Repetitive Dna Sequence Elements and Dna Polymerases Modeulate Instability in the Human Genome.” 2014. Web. 09 Mar 2021.

Vancouver:

Walsh E. Repetitive Dna Sequence Elements and Dna Polymerases Modeulate Instability in the Human Genome. [Internet] [Thesis]. Penn State University; 2014. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/20639.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Walsh E. Repetitive Dna Sequence Elements and Dna Polymerases Modeulate Instability in the Human Genome. [Thesis]. Penn State University; 2014. Available from: https://submit-etda.libraries.psu.edu/catalog/20639

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

4. Kelleher, Andrew Ryan. The regulation of mTORC1 signaling in immobilized rat hindlimb skeletal muscle.

Degree: 2014, Penn State University

 Limb immobilization, limb suspension, and bed rest cause substantial loss of skeletal muscle mass, a phenomenon termed disuse atrophy. Disuse atrophy is attributed to a… (more)

Subjects/Keywords: disuse atrophy; anabolic resistance; casting; REDD1; REDD2; Ddit4; Ddit4l; p70S6K1; muscle wasting; fixed muscle length

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APA (6th Edition):

Kelleher, A. R. (2014). The regulation of mTORC1 signaling in immobilized rat hindlimb skeletal muscle. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/22587

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kelleher, Andrew Ryan. “The regulation of mTORC1 signaling in immobilized rat hindlimb skeletal muscle.” 2014. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/22587.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kelleher, Andrew Ryan. “The regulation of mTORC1 signaling in immobilized rat hindlimb skeletal muscle.” 2014. Web. 09 Mar 2021.

Vancouver:

Kelleher AR. The regulation of mTORC1 signaling in immobilized rat hindlimb skeletal muscle. [Internet] [Thesis]. Penn State University; 2014. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/22587.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kelleher AR. The regulation of mTORC1 signaling in immobilized rat hindlimb skeletal muscle. [Thesis]. Penn State University; 2014. Available from: https://submit-etda.libraries.psu.edu/catalog/22587

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

5. Trivedi, Darshan V. Allosteric Communication and Force Generation in Myosin Motors.

Degree: 2014, Penn State University

 Muscle contraction, intracellular transport and a myriad of other mechanical functions in a cell are governed by myosin motors. Myosins utilize the chemical energy derived… (more)

Subjects/Keywords: Myosin; Actin; FRET; Motor Proteins

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APA (6th Edition):

Trivedi, D. V. (2014). Allosteric Communication and Force Generation in Myosin Motors. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/23282

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Trivedi, Darshan V. “Allosteric Communication and Force Generation in Myosin Motors.” 2014. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/23282.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Trivedi, Darshan V. “Allosteric Communication and Force Generation in Myosin Motors.” 2014. Web. 09 Mar 2021.

Vancouver:

Trivedi DV. Allosteric Communication and Force Generation in Myosin Motors. [Internet] [Thesis]. Penn State University; 2014. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/23282.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Trivedi DV. Allosteric Communication and Force Generation in Myosin Motors. [Thesis]. Penn State University; 2014. Available from: https://submit-etda.libraries.psu.edu/catalog/23282

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

6. Kren, Nancy Porterfield. Characterization of Functional Regions of OGFr.

Degree: 2015, Penn State University

 The Opioid Growth Factor (OGF)-OGF receptor (OGFr) axis is present and tonically active in animal and human cancer cell lines, as well as human tumors.… (more)

Subjects/Keywords: OGFr; NES; Tandem Repeats

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APA (6th Edition):

Kren, N. P. (2015). Characterization of Functional Regions of OGFr. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/26247

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kren, Nancy Porterfield. “Characterization of Functional Regions of OGFr.” 2015. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/26247.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kren, Nancy Porterfield. “Characterization of Functional Regions of OGFr.” 2015. Web. 09 Mar 2021.

Vancouver:

Kren NP. Characterization of Functional Regions of OGFr. [Internet] [Thesis]. Penn State University; 2015. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/26247.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kren NP. Characterization of Functional Regions of OGFr. [Thesis]. Penn State University; 2015. Available from: https://submit-etda.libraries.psu.edu/catalog/26247

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

7. Fino, Kristin Kelly. Role of the Cholecystokinin-B Receptor in Proliferation of Pancreatic Cancer.

Degree: 2012, Penn State University

 Pancreatic cancer ranks as the fourth most common cause of cancer-related mortality with a five-year survival rate of less than 1% and a median survival… (more)

Subjects/Keywords: pancreatic cancer; gastrin; CCK-BR; G-protein coupled receptor; RNAi

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APA (6th Edition):

Fino, K. K. (2012). Role of the Cholecystokinin-B Receptor in Proliferation of Pancreatic Cancer. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/15980

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Fino, Kristin Kelly. “Role of the Cholecystokinin-B Receptor in Proliferation of Pancreatic Cancer.” 2012. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/15980.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Fino, Kristin Kelly. “Role of the Cholecystokinin-B Receptor in Proliferation of Pancreatic Cancer.” 2012. Web. 09 Mar 2021.

Vancouver:

Fino KK. Role of the Cholecystokinin-B Receptor in Proliferation of Pancreatic Cancer. [Internet] [Thesis]. Penn State University; 2012. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/15980.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fino KK. Role of the Cholecystokinin-B Receptor in Proliferation of Pancreatic Cancer. [Thesis]. Penn State University; 2012. Available from: https://submit-etda.libraries.psu.edu/catalog/15980

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

8. Raval, Manmeet Harishbhai. MECHANISM OF CLASS III MYOSIN MEDIATED REGULATION OF ACTIN BUNDLE BASED PROTRUSIONS.

Degree: 2016, Penn State University

 Class III myosins (MYO3A and MYO3B) are actin based motors which are proposed to function as transporters in parallel actin-bundle based protrusions such as stereocilia… (more)

Subjects/Keywords: Myosins; Actin cytoskeleton; Filopodia; Stereocilia; Deafness

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APA (6th Edition):

Raval, M. H. (2016). MECHANISM OF CLASS III MYOSIN MEDIATED REGULATION OF ACTIN BUNDLE BASED PROTRUSIONS. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/13601mhr15

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Raval, Manmeet Harishbhai. “MECHANISM OF CLASS III MYOSIN MEDIATED REGULATION OF ACTIN BUNDLE BASED PROTRUSIONS.” 2016. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/13601mhr15.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Raval, Manmeet Harishbhai. “MECHANISM OF CLASS III MYOSIN MEDIATED REGULATION OF ACTIN BUNDLE BASED PROTRUSIONS.” 2016. Web. 09 Mar 2021.

Vancouver:

Raval MH. MECHANISM OF CLASS III MYOSIN MEDIATED REGULATION OF ACTIN BUNDLE BASED PROTRUSIONS. [Internet] [Thesis]. Penn State University; 2016. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/13601mhr15.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Raval MH. MECHANISM OF CLASS III MYOSIN MEDIATED REGULATION OF ACTIN BUNDLE BASED PROTRUSIONS. [Thesis]. Penn State University; 2016. Available from: https://submit-etda.libraries.psu.edu/catalog/13601mhr15

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

9. Watters, Rebecca Jean. Targeting Sphingolipid Metabolism in Natural Killer Cell Large Granular Lymphocyte Leukemia .

Degree: 2011, Penn State University

 Large granular lymphocyte (LGL) leukemia is a rare disorder characterized by clonal expansion of cytotoxic lymphocytes. LGL cells play an integral role in the immune… (more)

Subjects/Keywords: NK LGL Leukemia; nanoliposomes; ceramide; sphingolipid

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APA (6th Edition):

Watters, R. J. (2011). Targeting Sphingolipid Metabolism in Natural Killer Cell Large Granular Lymphocyte Leukemia . (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/12502

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Watters, Rebecca Jean. “Targeting Sphingolipid Metabolism in Natural Killer Cell Large Granular Lymphocyte Leukemia .” 2011. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/12502.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Watters, Rebecca Jean. “Targeting Sphingolipid Metabolism in Natural Killer Cell Large Granular Lymphocyte Leukemia .” 2011. Web. 09 Mar 2021.

Vancouver:

Watters RJ. Targeting Sphingolipid Metabolism in Natural Killer Cell Large Granular Lymphocyte Leukemia . [Internet] [Thesis]. Penn State University; 2011. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/12502.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Watters RJ. Targeting Sphingolipid Metabolism in Natural Killer Cell Large Granular Lymphocyte Leukemia . [Thesis]. Penn State University; 2011. Available from: https://submit-etda.libraries.psu.edu/catalog/12502

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

10. Kazi, Abid A. ROLE OF PRAS40 AND DEPTOR – TWO mTOR BINDING PROTEINS IN C2C12 MYOCYTES .

Degree: 2011, Penn State University

 PRAS40 and DEPTOR are mTOR binding proteins that affect cell metabolism. Under catabolic conditions such as sepsis and glucocorticoid excess, there is an increase in… (more)

Subjects/Keywords: protein synthesis; mTOR; knockdown; lentivirus; shRNA; sepsis; disuse atrophy

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APA (6th Edition):

Kazi, A. A. (2011). ROLE OF PRAS40 AND DEPTOR – TWO mTOR BINDING PROTEINS IN C2C12 MYOCYTES . (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/11847

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kazi, Abid A. “ROLE OF PRAS40 AND DEPTOR – TWO mTOR BINDING PROTEINS IN C2C12 MYOCYTES .” 2011. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/11847.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kazi, Abid A. “ROLE OF PRAS40 AND DEPTOR – TWO mTOR BINDING PROTEINS IN C2C12 MYOCYTES .” 2011. Web. 09 Mar 2021.

Vancouver:

Kazi AA. ROLE OF PRAS40 AND DEPTOR – TWO mTOR BINDING PROTEINS IN C2C12 MYOCYTES . [Internet] [Thesis]. Penn State University; 2011. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/11847.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kazi AA. ROLE OF PRAS40 AND DEPTOR – TWO mTOR BINDING PROTEINS IN C2C12 MYOCYTES . [Thesis]. Penn State University; 2011. Available from: https://submit-etda.libraries.psu.edu/catalog/11847

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

11. Rennoll, Sherri Ann. Transcriptional regulation of the c-MYC (MYC) proto-oncogene by oncogenic Wnt/β-catenin signaling in colorectal carcinomas.

Degree: 2015, Penn State University

 Over 90% of sporadic colorectal cancers (CRCs) are associated with initiating mutations in components of the Wnt/β-catenin signaling pathway. These mutations result in elevated nuclear… (more)

Subjects/Keywords: MYC; beta-catenin; AXIN2; colorectal cancer

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APA (6th Edition):

Rennoll, S. A. (2015). Transcriptional regulation of the c-MYC (MYC) proto-oncogene by oncogenic Wnt/β-catenin signaling in colorectal carcinomas. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/26902

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Rennoll, Sherri Ann. “Transcriptional regulation of the c-MYC (MYC) proto-oncogene by oncogenic Wnt/β-catenin signaling in colorectal carcinomas.” 2015. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/26902.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Rennoll, Sherri Ann. “Transcriptional regulation of the c-MYC (MYC) proto-oncogene by oncogenic Wnt/β-catenin signaling in colorectal carcinomas.” 2015. Web. 09 Mar 2021.

Vancouver:

Rennoll SA. Transcriptional regulation of the c-MYC (MYC) proto-oncogene by oncogenic Wnt/β-catenin signaling in colorectal carcinomas. [Internet] [Thesis]. Penn State University; 2015. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/26902.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Rennoll SA. Transcriptional regulation of the c-MYC (MYC) proto-oncogene by oncogenic Wnt/β-catenin signaling in colorectal carcinomas. [Thesis]. Penn State University; 2015. Available from: https://submit-etda.libraries.psu.edu/catalog/26902

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

12. Young, Megan Marie. The Role Of Sphingosine Kinase In The Crosstalk Between Apoptosis, Autophagy And Endocytosis.

Degree: 2015, Penn State University

 Autophagy is a catabolic process in which cytoplasmic components are sequestered within double-membrane vesicles called autophagosomes and delivered to lysosomes for degradation and recycling. Autophagy… (more)

Subjects/Keywords: autophagy; apoptosis; endocytosis; iDISC; sphingosine kinase; sphingosine; sphingolipid; SK1i; SKI-I; late endosome

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Young, M. M. (2015). The Role Of Sphingosine Kinase In The Crosstalk Between Apoptosis, Autophagy And Endocytosis. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/27408

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Young, Megan Marie. “The Role Of Sphingosine Kinase In The Crosstalk Between Apoptosis, Autophagy And Endocytosis.” 2015. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/27408.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Young, Megan Marie. “The Role Of Sphingosine Kinase In The Crosstalk Between Apoptosis, Autophagy And Endocytosis.” 2015. Web. 09 Mar 2021.

Vancouver:

Young MM. The Role Of Sphingosine Kinase In The Crosstalk Between Apoptosis, Autophagy And Endocytosis. [Internet] [Thesis]. Penn State University; 2015. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/27408.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Young MM. The Role Of Sphingosine Kinase In The Crosstalk Between Apoptosis, Autophagy And Endocytosis. [Thesis]. Penn State University; 2015. Available from: https://submit-etda.libraries.psu.edu/catalog/27408

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

13. Black, Adam James. Characterization of the high-fat diet-induced effects on pre-mRNA alternative splicing in skeletal muscle: a focus on fatty acids and troponin T3.

Degree: 2018, Penn State University

 Fat-enriched diets produce metabolic changes in skeletal muscle, that in turn mediate changes in gene regulation. The alternative splicing of pre-mRNA is an important part… (more)

Subjects/Keywords: alternative splicing; fatty acids; Tnnt3; high-fat diet; fast skeletal muscle troponin T; skeletal muscle; ceramide

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APA (6th Edition):

Black, A. J. (2018). Characterization of the high-fat diet-induced effects on pre-mRNA alternative splicing in skeletal muscle: a focus on fatty acids and troponin T3. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/14934ajb31

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Black, Adam James. “Characterization of the high-fat diet-induced effects on pre-mRNA alternative splicing in skeletal muscle: a focus on fatty acids and troponin T3.” 2018. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/14934ajb31.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Black, Adam James. “Characterization of the high-fat diet-induced effects on pre-mRNA alternative splicing in skeletal muscle: a focus on fatty acids and troponin T3.” 2018. Web. 09 Mar 2021.

Vancouver:

Black AJ. Characterization of the high-fat diet-induced effects on pre-mRNA alternative splicing in skeletal muscle: a focus on fatty acids and troponin T3. [Internet] [Thesis]. Penn State University; 2018. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/14934ajb31.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Black AJ. Characterization of the high-fat diet-induced effects on pre-mRNA alternative splicing in skeletal muscle: a focus on fatty acids and troponin T3. [Thesis]. Penn State University; 2018. Available from: https://submit-etda.libraries.psu.edu/catalog/14934ajb31

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

14. Keller, Ross Richard. Elucidating Evolutionary Constraints of Mouse Mammary Cancer Using Adenomatous Polyposis Coli Mutations.

Degree: 2018, Penn State University

 Cancer is a disease of evolution. Species evolve because mutations are passed to progeny through the germline, but somatic cells acquire mutations as well, and… (more)

Subjects/Keywords: Cancer; Evolution; Genetics

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APA (6th Edition):

Keller, R. R. (2018). Elucidating Evolutionary Constraints of Mouse Mammary Cancer Using Adenomatous Polyposis Coli Mutations. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/15024rrk145

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Keller, Ross Richard. “Elucidating Evolutionary Constraints of Mouse Mammary Cancer Using Adenomatous Polyposis Coli Mutations.” 2018. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/15024rrk145.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Keller, Ross Richard. “Elucidating Evolutionary Constraints of Mouse Mammary Cancer Using Adenomatous Polyposis Coli Mutations.” 2018. Web. 09 Mar 2021.

Vancouver:

Keller RR. Elucidating Evolutionary Constraints of Mouse Mammary Cancer Using Adenomatous Polyposis Coli Mutations. [Internet] [Thesis]. Penn State University; 2018. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/15024rrk145.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Keller RR. Elucidating Evolutionary Constraints of Mouse Mammary Cancer Using Adenomatous Polyposis Coli Mutations. [Thesis]. Penn State University; 2018. Available from: https://submit-etda.libraries.psu.edu/catalog/15024rrk145

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

15. Mikse, Oliver. CHARACTERIZATION OF FOXO3A AS A SUPPRESSOR OF LUNG ADENOCARCINOMA .

Degree: 2011, Penn State University

 Lung tumor development is believed to occur through a step-wise series of molecular changes that influence cell growth and survival. This process is facilitated by… (more)

Subjects/Keywords: CDC14A; Lung Cancer; FOXO3A; Tumor Suppressor

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APA (6th Edition):

Mikse, O. (2011). CHARACTERIZATION OF FOXO3A AS A SUPPRESSOR OF LUNG ADENOCARCINOMA . (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/11586

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Mikse, Oliver. “CHARACTERIZATION OF FOXO3A AS A SUPPRESSOR OF LUNG ADENOCARCINOMA .” 2011. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/11586.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Mikse, Oliver. “CHARACTERIZATION OF FOXO3A AS A SUPPRESSOR OF LUNG ADENOCARCINOMA .” 2011. Web. 09 Mar 2021.

Vancouver:

Mikse O. CHARACTERIZATION OF FOXO3A AS A SUPPRESSOR OF LUNG ADENOCARCINOMA . [Internet] [Thesis]. Penn State University; 2011. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/11586.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Mikse O. CHARACTERIZATION OF FOXO3A AS A SUPPRESSOR OF LUNG ADENOCARCINOMA . [Thesis]. Penn State University; 2011. Available from: https://submit-etda.libraries.psu.edu/catalog/11586

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

16. Chin-quee, Karis P. Breast cancer cell secretions promote a pre-metastatic niche in bone; an in vitro look .

Degree: 2013, Penn State University

 ABSTRACT Successful metastasis requires some degree of hospitality from the host organ that is metastasized. In recent years, evidence has been mounting that the primary… (more)

Subjects/Keywords: breast cancer metastasis; pre-metastatic niche; bone; collagen fragments; collagenase

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APA (6th Edition):

Chin-quee, K. P. (2013). Breast cancer cell secretions promote a pre-metastatic niche in bone; an in vitro look . (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/17496

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chin-quee, Karis P. “Breast cancer cell secretions promote a pre-metastatic niche in bone; an in vitro look .” 2013. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/17496.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chin-quee, Karis P. “Breast cancer cell secretions promote a pre-metastatic niche in bone; an in vitro look .” 2013. Web. 09 Mar 2021.

Vancouver:

Chin-quee KP. Breast cancer cell secretions promote a pre-metastatic niche in bone; an in vitro look . [Internet] [Thesis]. Penn State University; 2013. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/17496.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chin-quee KP. Breast cancer cell secretions promote a pre-metastatic niche in bone; an in vitro look . [Thesis]. Penn State University; 2013. Available from: https://submit-etda.libraries.psu.edu/catalog/17496

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

17. Shi, Chenxu. INVESTIGATIONS OF POLYAMINE FUNCTION USING TRANSGENIC MOUSE MODELS WITH SPERMIDINE SYNTHASE AND S-ADENOSYLMETHIONINE DECARBOXYLASE OVEREXPRESSION .

Degree: 2011, Penn State University

 Polyamines, including putrescine, spermidine, and spermine, are ubiquitous polycationic compounds that are essential for cell growth and differentiation. To better understand cellular function of polyamines,… (more)

Subjects/Keywords: Polyamines; S-adenosylmethionine decarboxylase; Spermidine synthase; Transgenic mice; Nonmelanoma skin cancer

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APA (6th Edition):

Shi, C. (2011). INVESTIGATIONS OF POLYAMINE FUNCTION USING TRANSGENIC MOUSE MODELS WITH SPERMIDINE SYNTHASE AND S-ADENOSYLMETHIONINE DECARBOXYLASE OVEREXPRESSION . (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/12128

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shi, Chenxu. “INVESTIGATIONS OF POLYAMINE FUNCTION USING TRANSGENIC MOUSE MODELS WITH SPERMIDINE SYNTHASE AND S-ADENOSYLMETHIONINE DECARBOXYLASE OVEREXPRESSION .” 2011. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/12128.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shi, Chenxu. “INVESTIGATIONS OF POLYAMINE FUNCTION USING TRANSGENIC MOUSE MODELS WITH SPERMIDINE SYNTHASE AND S-ADENOSYLMETHIONINE DECARBOXYLASE OVEREXPRESSION .” 2011. Web. 09 Mar 2021.

Vancouver:

Shi C. INVESTIGATIONS OF POLYAMINE FUNCTION USING TRANSGENIC MOUSE MODELS WITH SPERMIDINE SYNTHASE AND S-ADENOSYLMETHIONINE DECARBOXYLASE OVEREXPRESSION . [Internet] [Thesis]. Penn State University; 2011. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/12128.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shi C. INVESTIGATIONS OF POLYAMINE FUNCTION USING TRANSGENIC MOUSE MODELS WITH SPERMIDINE SYNTHASE AND S-ADENOSYLMETHIONINE DECARBOXYLASE OVEREXPRESSION . [Thesis]. Penn State University; 2011. Available from: https://submit-etda.libraries.psu.edu/catalog/12128

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

18. Runkle, Edwin Aaron. The Tight Junction Protein Occludin Contributes to Centrosome Separation, Cell Cycle Progression, and Barrier Properties .

Degree: 2011, Penn State University

 The transmembrane tight junction (TJ) protein occludin has been implicated in a variety of cell processes including barrier properties particularly towards small cations, regulation of… (more)

Subjects/Keywords: Mitosis; Centrosomes; Tight Junctions; Occludin; Barrier Function

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APA (6th Edition):

Runkle, E. A. (2011). The Tight Junction Protein Occludin Contributes to Centrosome Separation, Cell Cycle Progression, and Barrier Properties . (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/11595

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Runkle, Edwin Aaron. “The Tight Junction Protein Occludin Contributes to Centrosome Separation, Cell Cycle Progression, and Barrier Properties .” 2011. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/11595.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Runkle, Edwin Aaron. “The Tight Junction Protein Occludin Contributes to Centrosome Separation, Cell Cycle Progression, and Barrier Properties .” 2011. Web. 09 Mar 2021.

Vancouver:

Runkle EA. The Tight Junction Protein Occludin Contributes to Centrosome Separation, Cell Cycle Progression, and Barrier Properties . [Internet] [Thesis]. Penn State University; 2011. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/11595.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Runkle EA. The Tight Junction Protein Occludin Contributes to Centrosome Separation, Cell Cycle Progression, and Barrier Properties . [Thesis]. Penn State University; 2011. Available from: https://submit-etda.libraries.psu.edu/catalog/11595

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

19. Kalapila, Aley George. O6-ALKYLGUANINE-DNA ALKYLTRANSFERASE - MEDIATED TOXICITY OF BIS-ELECTROPHILES: BIOCHEMICAL MECHANISMS .

Degree: 2009, Penn State University

 Bis-electrophiles are compounds that are capable of forming cross-links between two nucleophilic sites. This chemical attribute has contributed to increased genotoxicity of such compounds in… (more)

Subjects/Keywords: dibromoalkanes; dibromomethane; dibromoethane; AGT; O6-alkylguanine-DNA alkyltransferase; DNA repair; Bis-electrophiles; butadiene diepoxide; epibromohydrin; epichlorohydrin; epoxides

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APA (6th Edition):

Kalapila, A. G. (2009). O6-ALKYLGUANINE-DNA ALKYLTRANSFERASE - MEDIATED TOXICITY OF BIS-ELECTROPHILES: BIOCHEMICAL MECHANISMS . (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/7914

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kalapila, Aley George. “O6-ALKYLGUANINE-DNA ALKYLTRANSFERASE - MEDIATED TOXICITY OF BIS-ELECTROPHILES: BIOCHEMICAL MECHANISMS .” 2009. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/7914.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kalapila, Aley George. “O6-ALKYLGUANINE-DNA ALKYLTRANSFERASE - MEDIATED TOXICITY OF BIS-ELECTROPHILES: BIOCHEMICAL MECHANISMS .” 2009. Web. 09 Mar 2021.

Vancouver:

Kalapila AG. O6-ALKYLGUANINE-DNA ALKYLTRANSFERASE - MEDIATED TOXICITY OF BIS-ELECTROPHILES: BIOCHEMICAL MECHANISMS . [Internet] [Thesis]. Penn State University; 2009. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/7914.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kalapila AG. O6-ALKYLGUANINE-DNA ALKYLTRANSFERASE - MEDIATED TOXICITY OF BIS-ELECTROPHILES: BIOCHEMICAL MECHANISMS . [Thesis]. Penn State University; 2009. Available from: https://submit-etda.libraries.psu.edu/catalog/7914

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

20. Balliet, Renee Marie. CHARACTERIZATION OF UGTS ACTIVE AGAINST SUBEROYLANILIDE HYDROXAMIC ACID (SAHA): VARIATIONS IN SAHA GLUCURONIDATION ASSOCIATED WITH UGT GENETIC VARIANTS .

Degree: 2009, Penn State University

 Suberoylanilide Hydroxamic Acid (SAHA) is a histone deacetylase inhibitor used in the treatment of cutaneous T-cell lymphoma and in clinical trials for treatment of multiple… (more)

Subjects/Keywords: SAHA; pharmacogenetic; UGT

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APA (6th Edition):

Balliet, R. M. (2009). CHARACTERIZATION OF UGTS ACTIVE AGAINST SUBEROYLANILIDE HYDROXAMIC ACID (SAHA): VARIATIONS IN SAHA GLUCURONIDATION ASSOCIATED WITH UGT GENETIC VARIANTS . (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/9519

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Balliet, Renee Marie. “CHARACTERIZATION OF UGTS ACTIVE AGAINST SUBEROYLANILIDE HYDROXAMIC ACID (SAHA): VARIATIONS IN SAHA GLUCURONIDATION ASSOCIATED WITH UGT GENETIC VARIANTS .” 2009. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/9519.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Balliet, Renee Marie. “CHARACTERIZATION OF UGTS ACTIVE AGAINST SUBEROYLANILIDE HYDROXAMIC ACID (SAHA): VARIATIONS IN SAHA GLUCURONIDATION ASSOCIATED WITH UGT GENETIC VARIANTS .” 2009. Web. 09 Mar 2021.

Vancouver:

Balliet RM. CHARACTERIZATION OF UGTS ACTIVE AGAINST SUBEROYLANILIDE HYDROXAMIC ACID (SAHA): VARIATIONS IN SAHA GLUCURONIDATION ASSOCIATED WITH UGT GENETIC VARIANTS . [Internet] [Thesis]. Penn State University; 2009. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/9519.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Balliet RM. CHARACTERIZATION OF UGTS ACTIVE AGAINST SUBEROYLANILIDE HYDROXAMIC ACID (SAHA): VARIATIONS IN SAHA GLUCURONIDATION ASSOCIATED WITH UGT GENETIC VARIANTS . [Thesis]. Penn State University; 2009. Available from: https://submit-etda.libraries.psu.edu/catalog/9519

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

21. Plichta, Kristin Ann. Mammary Epithelial Cell Subtype-Specific Analysis of Ras Pathway Activation.

Degree: 2010, Penn State University

 Breast cancers can be divided into subtypes based in part on how closely the tumor cells resemble mammary epithelial cell (MEC) subtypes resident in normal… (more)

Subjects/Keywords: Ras; mammary organoids; breast cancer; transgenic mice; mammary epithelial cells

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APA (6th Edition):

Plichta, K. A. (2010). Mammary Epithelial Cell Subtype-Specific Analysis of Ras Pathway Activation. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/11251

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Plichta, Kristin Ann. “Mammary Epithelial Cell Subtype-Specific Analysis of Ras Pathway Activation.” 2010. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/11251.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Plichta, Kristin Ann. “Mammary Epithelial Cell Subtype-Specific Analysis of Ras Pathway Activation.” 2010. Web. 09 Mar 2021.

Vancouver:

Plichta KA. Mammary Epithelial Cell Subtype-Specific Analysis of Ras Pathway Activation. [Internet] [Thesis]. Penn State University; 2010. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/11251.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Plichta KA. Mammary Epithelial Cell Subtype-Specific Analysis of Ras Pathway Activation. [Thesis]. Penn State University; 2010. Available from: https://submit-etda.libraries.psu.edu/catalog/11251

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

22. Luu, Kieu. Positional and directional repair of O6-alkylguanine lesions by human O6-alkylguanine-DNA alkyltransferase.

Degree: 2008, Penn State University

 Human O6-alkylguanine-DNA alkyltranserase (AGT) is a widely expressed DNA repair protein present in prokaryotes and eukaryotes. AGT repairs O6-alkylguanine adducts in DNA via a stoichiometric… (more)

Subjects/Keywords: DNA repair; AGT; alkyltransferase; alkylation; chemotherapy resistance; O6-alkylguanine-DNA alkyltransferase

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APA (6th Edition):

Luu, K. (2008). Positional and directional repair of O6-alkylguanine lesions by human O6-alkylguanine-DNA alkyltransferase. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/6576

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Luu, Kieu. “Positional and directional repair of O6-alkylguanine lesions by human O6-alkylguanine-DNA alkyltransferase.” 2008. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/6576.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Luu, Kieu. “Positional and directional repair of O6-alkylguanine lesions by human O6-alkylguanine-DNA alkyltransferase.” 2008. Web. 09 Mar 2021.

Vancouver:

Luu K. Positional and directional repair of O6-alkylguanine lesions by human O6-alkylguanine-DNA alkyltransferase. [Internet] [Thesis]. Penn State University; 2008. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/6576.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Luu K. Positional and directional repair of O6-alkylguanine lesions by human O6-alkylguanine-DNA alkyltransferase. [Thesis]. Penn State University; 2008. Available from: https://submit-etda.libraries.psu.edu/catalog/6576

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

23. Nisenberg, Oleg. TARGETED OVEREXPRESSION OF S-ADENOSYLMETHIONINE DECARBOXYLASE IN MURINE HEARTS.

Degree: 2008, Penn State University

 Myocardial disease is the most common cause of death in industrialized nations. The underlying causes of cardiac malfunction are diverse, but often relate to thromboembolic… (more)

Subjects/Keywords: AdoMetDC; SAMDC; beta-adrenergic hypertrophy; isoproterenol; polyamine biosynthesis; transgenic mice

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APA (6th Edition):

Nisenberg, O. (2008). TARGETED OVEREXPRESSION OF S-ADENOSYLMETHIONINE DECARBOXYLASE IN MURINE HEARTS. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/6593

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Nisenberg, Oleg. “TARGETED OVEREXPRESSION OF S-ADENOSYLMETHIONINE DECARBOXYLASE IN MURINE HEARTS.” 2008. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/6593.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Nisenberg, Oleg. “TARGETED OVEREXPRESSION OF S-ADENOSYLMETHIONINE DECARBOXYLASE IN MURINE HEARTS.” 2008. Web. 09 Mar 2021.

Vancouver:

Nisenberg O. TARGETED OVEREXPRESSION OF S-ADENOSYLMETHIONINE DECARBOXYLASE IN MURINE HEARTS. [Internet] [Thesis]. Penn State University; 2008. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/6593.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Nisenberg O. TARGETED OVEREXPRESSION OF S-ADENOSYLMETHIONINE DECARBOXYLASE IN MURINE HEARTS. [Thesis]. Penn State University; 2008. Available from: https://submit-etda.libraries.psu.edu/catalog/6593

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

24. Davidshofer, Kristine C. ALLOSTERIC ACTIVATION OF UBIQUITIN CONJUGATING ENZYMES BY RING DOMAINS.

Degree: 2008, Penn State University

 Ubiquitin is a highly conserved eukaryotic protein of 76 amino acids that is added post-translationally to other proteins or to itself by a hierarchical cascade… (more)

Subjects/Keywords: RING domain; ubiquitin; ubiquitin conjugating enzyme

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APA (6th Edition):

Davidshofer, K. C. (2008). ALLOSTERIC ACTIVATION OF UBIQUITIN CONJUGATING ENZYMES BY RING DOMAINS. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/8500

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Davidshofer, Kristine C. “ALLOSTERIC ACTIVATION OF UBIQUITIN CONJUGATING ENZYMES BY RING DOMAINS.” 2008. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/8500.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Davidshofer, Kristine C. “ALLOSTERIC ACTIVATION OF UBIQUITIN CONJUGATING ENZYMES BY RING DOMAINS.” 2008. Web. 09 Mar 2021.

Vancouver:

Davidshofer KC. ALLOSTERIC ACTIVATION OF UBIQUITIN CONJUGATING ENZYMES BY RING DOMAINS. [Internet] [Thesis]. Penn State University; 2008. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/8500.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Davidshofer KC. ALLOSTERIC ACTIVATION OF UBIQUITIN CONJUGATING ENZYMES BY RING DOMAINS. [Thesis]. Penn State University; 2008. Available from: https://submit-etda.libraries.psu.edu/catalog/8500

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

25. Jacob, Kimberly Dawn. The Role of Non-Replicative DNA Polymerases in Microsatellite Mutagenesis.

Degree: 2009, Penn State University

 Microsatellites, or short tandem repeats (STR), are sequences of 1-6 base pairs per unit repeat and are found throughout the human genome. Mutations in these… (more)

Subjects/Keywords: mutagenesis; poymerae beta; polymerase IV; DNA replication

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Jacob, K. D. (2009). The Role of Non-Replicative DNA Polymerases in Microsatellite Mutagenesis. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/10186

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Jacob, Kimberly Dawn. “The Role of Non-Replicative DNA Polymerases in Microsatellite Mutagenesis.” 2009. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/10186.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Jacob, Kimberly Dawn. “The Role of Non-Replicative DNA Polymerases in Microsatellite Mutagenesis.” 2009. Web. 09 Mar 2021.

Vancouver:

Jacob KD. The Role of Non-Replicative DNA Polymerases in Microsatellite Mutagenesis. [Internet] [Thesis]. Penn State University; 2009. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/10186.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jacob KD. The Role of Non-Replicative DNA Polymerases in Microsatellite Mutagenesis. [Thesis]. Penn State University; 2009. Available from: https://submit-etda.libraries.psu.edu/catalog/10186

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

26. Heakal, Yasser M. IDENTIFICATION OF NOVEL PHARMACOLOGICAL APPROACHES TO INHIBIT NEUROTENSIN RECEPTOR-1 MITOGENIC SIGNALING IN BREAST CANCER CELLS .

Degree: 2009, Penn State University

 G protein coupled receptors (GPCRs) represent the largest family of cell surface receptors and serve as the primary pharmacological targets for more than thirty percent… (more)

Subjects/Keywords: Breast Cancer; Neurotensin; Ceramide; GPCR

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Heakal, Y. M. (2009). IDENTIFICATION OF NOVEL PHARMACOLOGICAL APPROACHES TO INHIBIT NEUROTENSIN RECEPTOR-1 MITOGENIC SIGNALING IN BREAST CANCER CELLS . (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/10222

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Heakal, Yasser M. “IDENTIFICATION OF NOVEL PHARMACOLOGICAL APPROACHES TO INHIBIT NEUROTENSIN RECEPTOR-1 MITOGENIC SIGNALING IN BREAST CANCER CELLS .” 2009. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/10222.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Heakal, Yasser M. “IDENTIFICATION OF NOVEL PHARMACOLOGICAL APPROACHES TO INHIBIT NEUROTENSIN RECEPTOR-1 MITOGENIC SIGNALING IN BREAST CANCER CELLS .” 2009. Web. 09 Mar 2021.

Vancouver:

Heakal YM. IDENTIFICATION OF NOVEL PHARMACOLOGICAL APPROACHES TO INHIBIT NEUROTENSIN RECEPTOR-1 MITOGENIC SIGNALING IN BREAST CANCER CELLS . [Internet] [Thesis]. Penn State University; 2009. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/10222.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Heakal YM. IDENTIFICATION OF NOVEL PHARMACOLOGICAL APPROACHES TO INHIBIT NEUROTENSIN RECEPTOR-1 MITOGENIC SIGNALING IN BREAST CANCER CELLS . [Thesis]. Penn State University; 2009. Available from: https://submit-etda.libraries.psu.edu/catalog/10222

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

27. Shah, Sandeep. Microsatellites as inducers of genome instability: Studies into the mechanisms of replication and repair of repetitive DNA .

Degree: 2010, Penn State University

 Genomic instability is a hallmark of tumor initiation and progression. A tumor genome can contain both gross chromosomal alterations as well as multiple submicroscopic nucleotide… (more)

Subjects/Keywords: primer extension; polymerase; WRN; replication; FRA16D; PMS2; Mismatch Repair; Fragile sites; HNPCC; mutation bias

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Shah, S. (2010). Microsatellites as inducers of genome instability: Studies into the mechanisms of replication and repair of repetitive DNA . (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/10468

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Shah, Sandeep. “Microsatellites as inducers of genome instability: Studies into the mechanisms of replication and repair of repetitive DNA .” 2010. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/10468.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Shah, Sandeep. “Microsatellites as inducers of genome instability: Studies into the mechanisms of replication and repair of repetitive DNA .” 2010. Web. 09 Mar 2021.

Vancouver:

Shah S. Microsatellites as inducers of genome instability: Studies into the mechanisms of replication and repair of repetitive DNA . [Internet] [Thesis]. Penn State University; 2010. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/10468.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shah S. Microsatellites as inducers of genome instability: Studies into the mechanisms of replication and repair of repetitive DNA . [Thesis]. Penn State University; 2010. Available from: https://submit-etda.libraries.psu.edu/catalog/10468

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

28. Ackermann, Joseph Michael. NOVEL APPROACHES TO MODULATE ORNITHINE DECARBOXYLASE ACTIVITY AND TO DETERMINE A ROLE FOR ORNITHINE DECARBOXYLASE IN CELL TRANSFORMATION.

Degree: 2008, Penn State University

 Ornithine decarboxylase (ODC) is known to have an important role in cell transformation, but that role is not well understood. The scientific and clinical value… (more)

Subjects/Keywords: polyamines; ornithine decarboxylase; gene silencing; DNazyme; ribozyme; antisense oligodeoxynucleotides; difluoromethylornithine; DFMO; resonance energy transfer; RET; FRET; catalytic nuleic acids

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Ackermann, J. M. (2008). NOVEL APPROACHES TO MODULATE ORNITHINE DECARBOXYLASE ACTIVITY AND TO DETERMINE A ROLE FOR ORNITHINE DECARBOXYLASE IN CELL TRANSFORMATION. (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/6533

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ackermann, Joseph Michael. “NOVEL APPROACHES TO MODULATE ORNITHINE DECARBOXYLASE ACTIVITY AND TO DETERMINE A ROLE FOR ORNITHINE DECARBOXYLASE IN CELL TRANSFORMATION.” 2008. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/6533.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ackermann, Joseph Michael. “NOVEL APPROACHES TO MODULATE ORNITHINE DECARBOXYLASE ACTIVITY AND TO DETERMINE A ROLE FOR ORNITHINE DECARBOXYLASE IN CELL TRANSFORMATION.” 2008. Web. 09 Mar 2021.

Vancouver:

Ackermann JM. NOVEL APPROACHES TO MODULATE ORNITHINE DECARBOXYLASE ACTIVITY AND TO DETERMINE A ROLE FOR ORNITHINE DECARBOXYLASE IN CELL TRANSFORMATION. [Internet] [Thesis]. Penn State University; 2008. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/6533.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ackermann JM. NOVEL APPROACHES TO MODULATE ORNITHINE DECARBOXYLASE ACTIVITY AND TO DETERMINE A ROLE FOR ORNITHINE DECARBOXYLASE IN CELL TRANSFORMATION. [Thesis]. Penn State University; 2008. Available from: https://submit-etda.libraries.psu.edu/catalog/6533

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


Penn State University

29. Tuckow, Alexander Paul. CONTROL OF SKELETAL MUSCLE PROTEIN SYNTHESIS: FUNCTION AND REGULATION OF EUKARYOTIC INITIATION FACTOR 2B EPSILON .

Degree: 2010, Penn State University

 Eukaryotic initiation factor 2B (eIF2B) is a heteropentameric complex that functions as a guanine nucleotide exchange factor (GEF) toward its substrate, eIF2. The activity of… (more)

Subjects/Keywords: translation initiation; protein synthesis; skeletal muscle

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Tuckow, A. P. (2010). CONTROL OF SKELETAL MUSCLE PROTEIN SYNTHESIS: FUNCTION AND REGULATION OF EUKARYOTIC INITIATION FACTOR 2B EPSILON . (Thesis). Penn State University. Retrieved from https://submit-etda.libraries.psu.edu/catalog/11619

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tuckow, Alexander Paul. “CONTROL OF SKELETAL MUSCLE PROTEIN SYNTHESIS: FUNCTION AND REGULATION OF EUKARYOTIC INITIATION FACTOR 2B EPSILON .” 2010. Thesis, Penn State University. Accessed March 09, 2021. https://submit-etda.libraries.psu.edu/catalog/11619.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tuckow, Alexander Paul. “CONTROL OF SKELETAL MUSCLE PROTEIN SYNTHESIS: FUNCTION AND REGULATION OF EUKARYOTIC INITIATION FACTOR 2B EPSILON .” 2010. Web. 09 Mar 2021.

Vancouver:

Tuckow AP. CONTROL OF SKELETAL MUSCLE PROTEIN SYNTHESIS: FUNCTION AND REGULATION OF EUKARYOTIC INITIATION FACTOR 2B EPSILON . [Internet] [Thesis]. Penn State University; 2010. [cited 2021 Mar 09]. Available from: https://submit-etda.libraries.psu.edu/catalog/11619.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tuckow AP. CONTROL OF SKELETAL MUSCLE PROTEIN SYNTHESIS: FUNCTION AND REGULATION OF EUKARYOTIC INITIATION FACTOR 2B EPSILON . [Thesis]. Penn State University; 2010. Available from: https://submit-etda.libraries.psu.edu/catalog/11619

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.