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You searched for +publisher:"NSYSU" +contributor:("Wen-Chun Hung"). Showing records 1 – 30 of 39 total matches.

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NSYSU

1. Tsao, Yu-chen. A Prospective Small Volume Albumin Therapy in Cirrhosis and Spontaneous Bacterial Peritonitis Treatment.

Degree: Master, Institute of Biomedical Sciences, 2008, NSYSU

 In clinical findings, complications are the major cause of death in cirrhotic patients. Among all the complications, ascites is most frequent type. All cirrhotic patients… (more)

Subjects/Keywords: cirrhosis; spontaneous bacterial peritonitis; albumin

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APA (6th Edition):

Tsao, Y. (2008). A Prospective Small Volume Albumin Therapy in Cirrhosis and Spontaneous Bacterial Peritonitis Treatment. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0730108-134947

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Tsao, Yu-chen. “A Prospective Small Volume Albumin Therapy in Cirrhosis and Spontaneous Bacterial Peritonitis Treatment.” 2008. Thesis, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0730108-134947.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Tsao, Yu-chen. “A Prospective Small Volume Albumin Therapy in Cirrhosis and Spontaneous Bacterial Peritonitis Treatment.” 2008. Web. 19 Jan 2021.

Vancouver:

Tsao Y. A Prospective Small Volume Albumin Therapy in Cirrhosis and Spontaneous Bacterial Peritonitis Treatment. [Internet] [Thesis]. NSYSU; 2008. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0730108-134947.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tsao Y. A Prospective Small Volume Albumin Therapy in Cirrhosis and Spontaneous Bacterial Peritonitis Treatment. [Thesis]. NSYSU; 2008. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0730108-134947

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

2. Cheng, Yun-Ching. Evolutionary relationship and cytotoxic mechanism of Taiwan banded krait β-Bungarotoxin's B chains and homologous proteins.

Degree: PhD, Institute of Biomedical Sciences, 2008, NSYSU

 β-Bungarotoxin (β-Bgt), a presynaptic phospholipase A2 (PLA2) neurotoxin isolated from the venom of Bungarus multicinctus (Taiwan banded krait), consists of A chain and B chain,… (more)

Subjects/Keywords: β-Bungarotoxin

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APA (6th Edition):

Cheng, Y. (2008). Evolutionary relationship and cytotoxic mechanism of Taiwan banded krait β-Bungarotoxin's B chains and homologous proteins. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0124108-173924

Chicago Manual of Style (16th Edition):

Cheng, Yun-Ching. “Evolutionary relationship and cytotoxic mechanism of Taiwan banded krait β-Bungarotoxin's B chains and homologous proteins.” 2008. Doctoral Dissertation, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0124108-173924.

MLA Handbook (7th Edition):

Cheng, Yun-Ching. “Evolutionary relationship and cytotoxic mechanism of Taiwan banded krait β-Bungarotoxin's B chains and homologous proteins.” 2008. Web. 19 Jan 2021.

Vancouver:

Cheng Y. Evolutionary relationship and cytotoxic mechanism of Taiwan banded krait β-Bungarotoxin's B chains and homologous proteins. [Internet] [Doctoral dissertation]. NSYSU; 2008. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0124108-173924.

Council of Science Editors:

Cheng Y. Evolutionary relationship and cytotoxic mechanism of Taiwan banded krait β-Bungarotoxin's B chains and homologous proteins. [Doctoral Dissertation]. NSYSU; 2008. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0124108-173924


NSYSU

3. Liao, Yen-Shun. Study the functional region involves in targeting of KChIP1 to membrane.

Degree: Master, Institute of Biomedical Sciences, 2008, NSYSU

 Potassium channel-interacting protein 1 (KChIP1), a Ca2+ sensor protein, regulates the function of A-type Kv4 potassium channels and increases their cell surface expression. Myristoylation at… (more)

Subjects/Keywords: NCS protein; KChIP1; phosphatidylserine

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APA (6th Edition):

Liao, Y. (2008). Study the functional region involves in targeting of KChIP1 to membrane. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0715108-124010

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liao, Yen-Shun. “Study the functional region involves in targeting of KChIP1 to membrane.” 2008. Thesis, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0715108-124010.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liao, Yen-Shun. “Study the functional region involves in targeting of KChIP1 to membrane.” 2008. Web. 19 Jan 2021.

Vancouver:

Liao Y. Study the functional region involves in targeting of KChIP1 to membrane. [Internet] [Thesis]. NSYSU; 2008. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0715108-124010.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liao Y. Study the functional region involves in targeting of KChIP1 to membrane. [Thesis]. NSYSU; 2008. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0715108-124010

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

4. Kuo, Lai-Hsin. HDGF Up-regulation Enhances the Invasive Capability and Metastatic Potential of Melanoma Cells.

Degree: Master, Institute of Biomedical Sciences, 2008, NSYSU

 Cutaneous malignant melanoma is the fastest increasing malignancy in humans. Hepatoma-derived growth factor (HDGF) is a novel growth factor identified from human hepatoma cell line.… (more)

Subjects/Keywords: tumorigenesis; angiogenesis; Melanoma; hepatoma-derived growth factor; HDGF

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APA (6th Edition):

Kuo, L. (2008). HDGF Up-regulation Enhances the Invasive Capability and Metastatic Potential of Melanoma Cells. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0814108-003244

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Kuo, Lai-Hsin. “HDGF Up-regulation Enhances the Invasive Capability and Metastatic Potential of Melanoma Cells.” 2008. Thesis, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0814108-003244.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Kuo, Lai-Hsin. “HDGF Up-regulation Enhances the Invasive Capability and Metastatic Potential of Melanoma Cells.” 2008. Web. 19 Jan 2021.

Vancouver:

Kuo L. HDGF Up-regulation Enhances the Invasive Capability and Metastatic Potential of Melanoma Cells. [Internet] [Thesis]. NSYSU; 2008. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0814108-003244.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Kuo L. HDGF Up-regulation Enhances the Invasive Capability and Metastatic Potential of Melanoma Cells. [Thesis]. NSYSU; 2008. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0814108-003244

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

5. Liu, Yi-Jia. Study of Kazal motifs of RECK protein on MMP-2 and MMP-9 activity and metastasis of lung adenocarcinoma.

Degree: Master, Institute of Biomedical Sciences, 2009, NSYSU

 RECK stands for âreversion-inducing cysteine-rich protein with Kazal motifsâ. This gene was initially discovered by screening a human fibroblast cDNA library for genes giving rise… (more)

Subjects/Keywords: RECK; MMP-9

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APA (6th Edition):

Liu, Y. (2009). Study of Kazal motifs of RECK protein on MMP-2 and MMP-9 activity and metastasis of lung adenocarcinoma. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0806109-130653

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Liu, Yi-Jia. “Study of Kazal motifs of RECK protein on MMP-2 and MMP-9 activity and metastasis of lung adenocarcinoma.” 2009. Thesis, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0806109-130653.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Liu, Yi-Jia. “Study of Kazal motifs of RECK protein on MMP-2 and MMP-9 activity and metastasis of lung adenocarcinoma.” 2009. Web. 19 Jan 2021.

Vancouver:

Liu Y. Study of Kazal motifs of RECK protein on MMP-2 and MMP-9 activity and metastasis of lung adenocarcinoma. [Internet] [Thesis]. NSYSU; 2009. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0806109-130653.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liu Y. Study of Kazal motifs of RECK protein on MMP-2 and MMP-9 activity and metastasis of lung adenocarcinoma. [Thesis]. NSYSU; 2009. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0806109-130653

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

6. Chiang, Chi-hsiang. Inhibition of sialylation of beta1 integrin and CXCR4 by a lithocholic acid-based sialyltransferase inhibitor suppresses cancer metastasis.

Degree: Master, Institute of Biomedical Sciences, 2009, NSYSU

 Sialyltransferases (STs), which catalyze the sialylation reaction by adding sialic acids to the terminal positions of oligosaccharide of glycoproteins and glycolipids, are over-expressed in cancer… (more)

Subjects/Keywords: chemokine receptor; sialylation; sialyltransferase; sialic acid

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APA (6th Edition):

Chiang, C. (2009). Inhibition of sialylation of beta1 integrin and CXCR4 by a lithocholic acid-based sialyltransferase inhibitor suppresses cancer metastasis. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0811109-180108

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chiang, Chi-hsiang. “Inhibition of sialylation of beta1 integrin and CXCR4 by a lithocholic acid-based sialyltransferase inhibitor suppresses cancer metastasis.” 2009. Thesis, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0811109-180108.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chiang, Chi-hsiang. “Inhibition of sialylation of beta1 integrin and CXCR4 by a lithocholic acid-based sialyltransferase inhibitor suppresses cancer metastasis.” 2009. Web. 19 Jan 2021.

Vancouver:

Chiang C. Inhibition of sialylation of beta1 integrin and CXCR4 by a lithocholic acid-based sialyltransferase inhibitor suppresses cancer metastasis. [Internet] [Thesis]. NSYSU; 2009. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0811109-180108.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chiang C. Inhibition of sialylation of beta1 integrin and CXCR4 by a lithocholic acid-based sialyltransferase inhibitor suppresses cancer metastasis. [Thesis]. NSYSU; 2009. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0811109-180108

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

7. Chuang, Chun-Wei. Study of the molecular mechanism by which COX-2 regulates CCR7 expression.

Degree: Master, Institute of Biomedical Sciences, 2010, NSYSU

 The metastatic spread of tumor cells is the major lethal aspect of cancer, and lymphatic metastasis is one of the most important routes. Recent studies… (more)

Subjects/Keywords: Sp1; CCR7; AKT; COX-2

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APA (6th Edition):

Chuang, C. (2010). Study of the molecular mechanism by which COX-2 regulates CCR7 expression. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823110-172725

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chuang, Chun-Wei. “Study of the molecular mechanism by which COX-2 regulates CCR7 expression.” 2010. Thesis, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823110-172725.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chuang, Chun-Wei. “Study of the molecular mechanism by which COX-2 regulates CCR7 expression.” 2010. Web. 19 Jan 2021.

Vancouver:

Chuang C. Study of the molecular mechanism by which COX-2 regulates CCR7 expression. [Internet] [Thesis]. NSYSU; 2010. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823110-172725.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chuang C. Study of the molecular mechanism by which COX-2 regulates CCR7 expression. [Thesis]. NSYSU; 2010. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823110-172725

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

8. Chen, Chien-wei. Down-regulation of Jab1 by ER stress in Hep3B hepatocellular carcinoma cell line.

Degree: Master, Institute of Biomedical Sciences, 2009, NSYSU

 Endoplasmic reticulum (ER) stress is the condition that unfolded or misfolded proteins accumulated in the ER which leads to the solubility stress. ER can activate… (more)

Subjects/Keywords: jab1; er stress

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APA (6th Edition):

Chen, C. (2009). Down-regulation of Jab1 by ER stress in Hep3B hepatocellular carcinoma cell line. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0727109-113910

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Chien-wei. “Down-regulation of Jab1 by ER stress in Hep3B hepatocellular carcinoma cell line.” 2009. Thesis, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0727109-113910.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Chien-wei. “Down-regulation of Jab1 by ER stress in Hep3B hepatocellular carcinoma cell line.” 2009. Web. 19 Jan 2021.

Vancouver:

Chen C. Down-regulation of Jab1 by ER stress in Hep3B hepatocellular carcinoma cell line. [Internet] [Thesis]. NSYSU; 2009. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0727109-113910.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen C. Down-regulation of Jab1 by ER stress in Hep3B hepatocellular carcinoma cell line. [Thesis]. NSYSU; 2009. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0727109-113910

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

9. Su, Huei-Ting. The Stem Cell Marker Nestin is Critical for TGF beta1- Mediated Tumor Progression in Pancreatic Cancer.

Degree: Master, Institute of Biomedical Sciences, 2012, NSYSU

 Stem cell marker Nestin is an intermediate filament protein that plays an important role in cell integrity, migration and differentiation. Nestin expression occurs in approximately… (more)

Subjects/Keywords: EMT; Intermediate filament proteins; Nestin; PDAC

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APA (6th Edition):

Su, H. (2012). The Stem Cell Marker Nestin is Critical for TGF beta1- Mediated Tumor Progression in Pancreatic Cancer. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0625112-152043

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Su, Huei-Ting. “The Stem Cell Marker Nestin is Critical for TGF beta1- Mediated Tumor Progression in Pancreatic Cancer.” 2012. Thesis, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0625112-152043.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Su, Huei-Ting. “The Stem Cell Marker Nestin is Critical for TGF beta1- Mediated Tumor Progression in Pancreatic Cancer.” 2012. Web. 19 Jan 2021.

Vancouver:

Su H. The Stem Cell Marker Nestin is Critical for TGF beta1- Mediated Tumor Progression in Pancreatic Cancer. [Internet] [Thesis]. NSYSU; 2012. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0625112-152043.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Su H. The Stem Cell Marker Nestin is Critical for TGF beta1- Mediated Tumor Progression in Pancreatic Cancer. [Thesis]. NSYSU; 2012. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0625112-152043

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

10. Lin, Hsin-Ying. STAT3-upregulated miR-92a in the control RECK expression in lung cancer cells.

Degree: Master, Institute of Biomedical Sciences, 2012, NSYSU

 Lung cancer is the common cause of cancer death. STAT3 (signal transducer and activator of transcription 3) has been reported to be an oncogenic transcription… (more)

Subjects/Keywords: RECK; miR-92a; STAT3

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APA (6th Edition):

Lin, H. (2012). STAT3-upregulated miR-92a in the control RECK expression in lung cancer cells. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0706112-173625

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lin, Hsin-Ying. “STAT3-upregulated miR-92a in the control RECK expression in lung cancer cells.” 2012. Thesis, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0706112-173625.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lin, Hsin-Ying. “STAT3-upregulated miR-92a in the control RECK expression in lung cancer cells.” 2012. Web. 19 Jan 2021.

Vancouver:

Lin H. STAT3-upregulated miR-92a in the control RECK expression in lung cancer cells. [Internet] [Thesis]. NSYSU; 2012. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0706112-173625.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lin H. STAT3-upregulated miR-92a in the control RECK expression in lung cancer cells. [Thesis]. NSYSU; 2012. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0706112-173625

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

11. Ye, Min-Yi. Anti-cancer mechanism of a novel tyrosine kinase inhibitor on human lung cancer cells.

Degree: Master, Institute of Biomedical Sciences, 2012, NSYSU

 Tyrosine kinases regulate fundamental signal pathways in cells including cell proliferation, motility, and differentiation. The kinase activity is tightly controlled in normal cells but is… (more)

Subjects/Keywords: p53; migration; CDK1; lung cancer; tyrosine kinase inhibitor

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APA (6th Edition):

Ye, M. (2012). Anti-cancer mechanism of a novel tyrosine kinase inhibitor on human lung cancer cells. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0706112-184416

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Ye, Min-Yi. “Anti-cancer mechanism of a novel tyrosine kinase inhibitor on human lung cancer cells.” 2012. Thesis, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0706112-184416.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Ye, Min-Yi. “Anti-cancer mechanism of a novel tyrosine kinase inhibitor on human lung cancer cells.” 2012. Web. 19 Jan 2021.

Vancouver:

Ye M. Anti-cancer mechanism of a novel tyrosine kinase inhibitor on human lung cancer cells. [Internet] [Thesis]. NSYSU; 2012. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0706112-184416.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Ye M. Anti-cancer mechanism of a novel tyrosine kinase inhibitor on human lung cancer cells. [Thesis]. NSYSU; 2012. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0706112-184416

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

12. Su, Mei-lin. Sialylation of CCR7 is critical for CCL19-stimulated proliferation, invasion and anti-anoikis in breast cancer cells.

Degree: Master, Institute of Biomedical Sciences, 2012, NSYSU

 Sialylation is catalyzed by sialyltransferases (STs) that adding sialic acids to the terminal positions of oligosaccharide of glycoproteins and glycolipids. This process is frequently enhanced… (more)

Subjects/Keywords: sialylation; CCR7; metastasis; AL10; breast cancer

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Su, M. (2012). Sialylation of CCR7 is critical for CCL19-stimulated proliferation, invasion and anti-anoikis in breast cancer cells. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0716112-124147

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Su, Mei-lin. “Sialylation of CCR7 is critical for CCL19-stimulated proliferation, invasion and anti-anoikis in breast cancer cells.” 2012. Thesis, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0716112-124147.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Su, Mei-lin. “Sialylation of CCR7 is critical for CCL19-stimulated proliferation, invasion and anti-anoikis in breast cancer cells.” 2012. Web. 19 Jan 2021.

Vancouver:

Su M. Sialylation of CCR7 is critical for CCL19-stimulated proliferation, invasion and anti-anoikis in breast cancer cells. [Internet] [Thesis]. NSYSU; 2012. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0716112-124147.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Su M. Sialylation of CCR7 is critical for CCL19-stimulated proliferation, invasion and anti-anoikis in breast cancer cells. [Thesis]. NSYSU; 2012. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0716112-124147

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

13. Weng, Ching-Chieh. The Role of CD133 to Bind to EGFR and Modulate Its Activation in Pancreatic Cancer.

Degree: Master, Institute of Biomedical Sciences, 2012, NSYSU

 Most of tumor consists of a heterogeneous population of tumor cells among a tumor initiating and chemo or radiation resistant subpopulation, called cancer stem cells… (more)

Subjects/Keywords: Cancer stem cells; CD133; Epidermal Growth Factor Receptor; MAPK/ ERK pathways

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APA (6th Edition):

Weng, C. (2012). The Role of CD133 to Bind to EGFR and Modulate Its Activation in Pancreatic Cancer. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823112-145009

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Weng, Ching-Chieh. “The Role of CD133 to Bind to EGFR and Modulate Its Activation in Pancreatic Cancer.” 2012. Thesis, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823112-145009.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Weng, Ching-Chieh. “The Role of CD133 to Bind to EGFR and Modulate Its Activation in Pancreatic Cancer.” 2012. Web. 19 Jan 2021.

Vancouver:

Weng C. The Role of CD133 to Bind to EGFR and Modulate Its Activation in Pancreatic Cancer. [Internet] [Thesis]. NSYSU; 2012. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823112-145009.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Weng C. The Role of CD133 to Bind to EGFR and Modulate Its Activation in Pancreatic Cancer. [Thesis]. NSYSU; 2012. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823112-145009

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

14. Wu, Nein-chi. Single Nucleotide Polymorphism Analysis of the Metastasis Supressor RECK Gene Promoter and Itâs Clinical Significance.

Degree: Master, Institute of Biomedical Sciences, 2011, NSYSU

 Reversion-inducing cysteine-rich with Kazal motif (RECK) is a cell surface anchoring protein, which known for the ability to inhibit matrix metalloproteinases (MMPs) and participate in… (more)

Subjects/Keywords: RECK promoter; Reversion-inducing cysteine-rich with Kazal motif; RECK; RECK SNP; SNP; single nucleotide polymorphism

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APA (6th Edition):

Wu, N. (2011). Single Nucleotide Polymorphism Analysis of the Metastasis Supressor RECK Gene Promoter and Itâs Clinical Significance. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0809111-202826

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Wu, Nein-chi. “Single Nucleotide Polymorphism Analysis of the Metastasis Supressor RECK Gene Promoter and Itâs Clinical Significance.” 2011. Thesis, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0809111-202826.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Wu, Nein-chi. “Single Nucleotide Polymorphism Analysis of the Metastasis Supressor RECK Gene Promoter and Itâs Clinical Significance.” 2011. Web. 19 Jan 2021.

Vancouver:

Wu N. Single Nucleotide Polymorphism Analysis of the Metastasis Supressor RECK Gene Promoter and Itâs Clinical Significance. [Internet] [Thesis]. NSYSU; 2011. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0809111-202826.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wu N. Single Nucleotide Polymorphism Analysis of the Metastasis Supressor RECK Gene Promoter and Itâs Clinical Significance. [Thesis]. NSYSU; 2011. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0809111-202826

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

15. Chen, Ku-chung. Cytotoxic mechanisms of Taiwan cobra phospholipase A2.

Degree: PhD, Institute of Biomedical Sciences, 2009, NSYSU

 The enzyme phospholipase A2 (PLA2) specifically hydrolyzes the 2-acyl ester bond of 1,2-diacyl-3-sn-phosphoglycerides releasing fatty acids and lysophospholipids in the presence of Ca2+. Both products… (more)

Subjects/Keywords: arachidonic acid; PLA2; phospholipase A2; FasL; Fas; JNK; ROS; Ca; p38 MAPK; SK-N-SH; lysophosphatidylcholine; K562

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APA (6th Edition):

Chen, K. (2009). Cytotoxic mechanisms of Taiwan cobra phospholipase A2. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0903109-184305

Chicago Manual of Style (16th Edition):

Chen, Ku-chung. “Cytotoxic mechanisms of Taiwan cobra phospholipase A2.” 2009. Doctoral Dissertation, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0903109-184305.

MLA Handbook (7th Edition):

Chen, Ku-chung. “Cytotoxic mechanisms of Taiwan cobra phospholipase A2.” 2009. Web. 19 Jan 2021.

Vancouver:

Chen K. Cytotoxic mechanisms of Taiwan cobra phospholipase A2. [Internet] [Doctoral dissertation]. NSYSU; 2009. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0903109-184305.

Council of Science Editors:

Chen K. Cytotoxic mechanisms of Taiwan cobra phospholipase A2. [Doctoral Dissertation]. NSYSU; 2009. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0903109-184305


NSYSU

16. Chen, Jing-yi. Regulation of Skp2 by Bcr-ABL oncogene in chronic meyloid leukemia cells and its therapeutic significance.

Degree: PhD, Institute of Biomedical Sciences, 2010, NSYSU

 Part I BCR-ABL fusion oncogene results fromt(9;22)(q34;q11) translocation of chromosome is the most common genetic abnormality found in chronic myeloid leukemia (CML) cells . The… (more)

Subjects/Keywords: miR-92a; SAHA; withaferin A; Skp2; CML

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APA (6th Edition):

Chen, J. (2010). Regulation of Skp2 by Bcr-ABL oncogene in chronic meyloid leukemia cells and its therapeutic significance. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0802110-181153

Chicago Manual of Style (16th Edition):

Chen, Jing-yi. “Regulation of Skp2 by Bcr-ABL oncogene in chronic meyloid leukemia cells and its therapeutic significance.” 2010. Doctoral Dissertation, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0802110-181153.

MLA Handbook (7th Edition):

Chen, Jing-yi. “Regulation of Skp2 by Bcr-ABL oncogene in chronic meyloid leukemia cells and its therapeutic significance.” 2010. Web. 19 Jan 2021.

Vancouver:

Chen J. Regulation of Skp2 by Bcr-ABL oncogene in chronic meyloid leukemia cells and its therapeutic significance. [Internet] [Doctoral dissertation]. NSYSU; 2010. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0802110-181153.

Council of Science Editors:

Chen J. Regulation of Skp2 by Bcr-ABL oncogene in chronic meyloid leukemia cells and its therapeutic significance. [Doctoral Dissertation]. NSYSU; 2010. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0802110-181153


NSYSU

17. Wang, Chi-Hung. Effects and mechanisms of novel indole compound on the regulation of breast cancer cell growth, invasion and the reversion of epithelial-to-mesenchymal transition.

Degree: PhD, Institute of Biomedical Sciences, 2013, NSYSU

 Breast cancer is the leading cause of death among Taiwanese women as well as women all over the world. Epidemiological and dietary studies have shown… (more)

Subjects/Keywords: epithelial-mesenchymal transition (EMT); miRNA-200; Histone deacetylase (HDAC); E-cadherin; ZEB1; apoptosis; 3,3â-diindolylmethane; Indole-3-carbinol (I3C)

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APA (6th Edition):

Wang, C. (2013). Effects and mechanisms of novel indole compound on the regulation of breast cancer cell growth, invasion and the reversion of epithelial-to-mesenchymal transition. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0712113-113112

Chicago Manual of Style (16th Edition):

Wang, Chi-Hung. “Effects and mechanisms of novel indole compound on the regulation of breast cancer cell growth, invasion and the reversion of epithelial-to-mesenchymal transition.” 2013. Doctoral Dissertation, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0712113-113112.

MLA Handbook (7th Edition):

Wang, Chi-Hung. “Effects and mechanisms of novel indole compound on the regulation of breast cancer cell growth, invasion and the reversion of epithelial-to-mesenchymal transition.” 2013. Web. 19 Jan 2021.

Vancouver:

Wang C. Effects and mechanisms of novel indole compound on the regulation of breast cancer cell growth, invasion and the reversion of epithelial-to-mesenchymal transition. [Internet] [Doctoral dissertation]. NSYSU; 2013. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0712113-113112.

Council of Science Editors:

Wang C. Effects and mechanisms of novel indole compound on the regulation of breast cancer cell growth, invasion and the reversion of epithelial-to-mesenchymal transition. [Doctoral Dissertation]. NSYSU; 2013. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0712113-113112


NSYSU

18. Chu, Tian-huei. Therapeutic Efficacy and Mechanism of Celecoxib for Hepatocellular Carcinoma.

Degree: PhD, Institute of Biomedical Sciences, 2014, NSYSU

 Hepatocellular carcinoma (HCC) is the one of most common malignancies in Taiwan. Current HCC therapies include surgery, chemotherapy, radiofrequency ablation and target therapy. However, the… (more)

Subjects/Keywords: cyclooxygenase 2; sorafenib; cancer stem cells; celecoxib; hepatocellular carcinoma

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APA (6th Edition):

Chu, T. (2014). Therapeutic Efficacy and Mechanism of Celecoxib for Hepatocellular Carcinoma. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0809114-182128

Chicago Manual of Style (16th Edition):

Chu, Tian-huei. “Therapeutic Efficacy and Mechanism of Celecoxib for Hepatocellular Carcinoma.” 2014. Doctoral Dissertation, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0809114-182128.

MLA Handbook (7th Edition):

Chu, Tian-huei. “Therapeutic Efficacy and Mechanism of Celecoxib for Hepatocellular Carcinoma.” 2014. Web. 19 Jan 2021.

Vancouver:

Chu T. Therapeutic Efficacy and Mechanism of Celecoxib for Hepatocellular Carcinoma. [Internet] [Doctoral dissertation]. NSYSU; 2014. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0809114-182128.

Council of Science Editors:

Chu T. Therapeutic Efficacy and Mechanism of Celecoxib for Hepatocellular Carcinoma. [Doctoral Dissertation]. NSYSU; 2014. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0809114-182128


NSYSU

19. Chiang, Chi-Hsiang. Regulation of microRNA-182 and its functional role in breast cancer.

Degree: PhD, Institute of Biomedical Sciences, 2013, NSYSU

 MicroRNAs (MiRNAs) are endogenous small non-coding RNAs which negatively regulate gene expression by inducing translation repression or mRNA cleavage. MiR-182 is a member of the… (more)

Subjects/Keywords: RECK; β-catenin; microRNA; matrix metalloproteinase

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APA (6th Edition):

Chiang, C. (2013). Regulation of microRNA-182 and its functional role in breast cancer. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0624113-191725

Chicago Manual of Style (16th Edition):

Chiang, Chi-Hsiang. “Regulation of microRNA-182 and its functional role in breast cancer.” 2013. Doctoral Dissertation, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0624113-191725.

MLA Handbook (7th Edition):

Chiang, Chi-Hsiang. “Regulation of microRNA-182 and its functional role in breast cancer.” 2013. Web. 19 Jan 2021.

Vancouver:

Chiang C. Regulation of microRNA-182 and its functional role in breast cancer. [Internet] [Doctoral dissertation]. NSYSU; 2013. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0624113-191725.

Council of Science Editors:

Chiang C. Regulation of microRNA-182 and its functional role in breast cancer. [Doctoral Dissertation]. NSYSU; 2013. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0624113-191725


NSYSU

20. Hong, Kun-jing. Study of the molecular mechanism by which RECK suppresses drug resistance and cancer stemness by regulating Her2/Neu and Notch oncogenic pathways.

Degree: PhD, Institute of Biomedical Sciences, 2016, NSYSU

 Reversion-inducing cysteine rich protein with Kazal motifs (RECK) is an endogenous metastatic suppressor gene, which can inhibit matrix metalloproteinase MMP-2, MMP-9 and MT1-MMP to reduce… (more)

Subjects/Keywords: RECK; glycoprotein; matrix metalloproteinase; metastasis; cancer stem cell; dimerization; autophosphorylation

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APA (6th Edition):

Hong, K. (2016). Study of the molecular mechanism by which RECK suppresses drug resistance and cancer stemness by regulating Her2/Neu and Notch oncogenic pathways. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0028116-155048

Chicago Manual of Style (16th Edition):

Hong, Kun-jing. “Study of the molecular mechanism by which RECK suppresses drug resistance and cancer stemness by regulating Her2/Neu and Notch oncogenic pathways.” 2016. Doctoral Dissertation, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0028116-155048.

MLA Handbook (7th Edition):

Hong, Kun-jing. “Study of the molecular mechanism by which RECK suppresses drug resistance and cancer stemness by regulating Her2/Neu and Notch oncogenic pathways.” 2016. Web. 19 Jan 2021.

Vancouver:

Hong K. Study of the molecular mechanism by which RECK suppresses drug resistance and cancer stemness by regulating Her2/Neu and Notch oncogenic pathways. [Internet] [Doctoral dissertation]. NSYSU; 2016. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0028116-155048.

Council of Science Editors:

Hong K. Study of the molecular mechanism by which RECK suppresses drug resistance and cancer stemness by regulating Her2/Neu and Notch oncogenic pathways. [Doctoral Dissertation]. NSYSU; 2016. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0028116-155048


NSYSU

21. Kuo, Tzu-Lei. Investigation of APC gene functional roles in pancreatic cancer initiation and progression.

Degree: PhD, Institute of Biomedical Sciences, 2016, NSYSU

 Chapter â  Adenomatous polyposis coli (APC), a tumor suppressor gene critically involved in familial adenomatous polyposis, is integral in Wnt/β-catenin signaling and is implicated in… (more)

Subjects/Keywords: Haploinsufficiency; Mucinous cystic neoplasms; p53; APC; Pancreatic cancer; metastasis; mice model; tumor organoid

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APA (6th Edition):

Kuo, T. (2016). Investigation of APC gene functional roles in pancreatic cancer initiation and progression. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0501116-194326

Chicago Manual of Style (16th Edition):

Kuo, Tzu-Lei. “Investigation of APC gene functional roles in pancreatic cancer initiation and progression.” 2016. Doctoral Dissertation, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0501116-194326.

MLA Handbook (7th Edition):

Kuo, Tzu-Lei. “Investigation of APC gene functional roles in pancreatic cancer initiation and progression.” 2016. Web. 19 Jan 2021.

Vancouver:

Kuo T. Investigation of APC gene functional roles in pancreatic cancer initiation and progression. [Internet] [Doctoral dissertation]. NSYSU; 2016. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0501116-194326.

Council of Science Editors:

Kuo T. Investigation of APC gene functional roles in pancreatic cancer initiation and progression. [Doctoral Dissertation]. NSYSU; 2016. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0501116-194326


NSYSU

22. Chang, Tsung-ming. Prox1 suppresses growth and metastasis of hepatocellular carcinoma by downregulating Twist1.

Degree: PhD, Institute of Biomedical Sciences, 2013, NSYSU

 Prospero-related homeobox 1 (Prox1) was cloned as homeobox gene which homologous to the Drosophila prospero gene. As a transcription factor, Prox1 is important for liver… (more)

Subjects/Keywords: AKT2; p53; Prox1; Twist1

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APA (6th Edition):

Chang, T. (2013). Prox1 suppresses growth and metastasis of hepatocellular carcinoma by downregulating Twist1. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0110113-155844

Chicago Manual of Style (16th Edition):

Chang, Tsung-ming. “Prox1 suppresses growth and metastasis of hepatocellular carcinoma by downregulating Twist1.” 2013. Doctoral Dissertation, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0110113-155844.

MLA Handbook (7th Edition):

Chang, Tsung-ming. “Prox1 suppresses growth and metastasis of hepatocellular carcinoma by downregulating Twist1.” 2013. Web. 19 Jan 2021.

Vancouver:

Chang T. Prox1 suppresses growth and metastasis of hepatocellular carcinoma by downregulating Twist1. [Internet] [Doctoral dissertation]. NSYSU; 2013. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0110113-155844.

Council of Science Editors:

Chang T. Prox1 suppresses growth and metastasis of hepatocellular carcinoma by downregulating Twist1. [Doctoral Dissertation]. NSYSU; 2013. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0110113-155844


NSYSU

23. Cheng, Hsueh-Tsen. Anti-lymphangiogenic action of SAHA on breast cancer.

Degree: PhD, Institute of Biomedical Sciences, 2013, NSYSU

 HDAC inhibitor suberoylanilide hydroxamic acid (SAHA) exhibits anti-tumor effects on various types of human cancers and is now approved by U.S FDA for clinical cancer… (more)

Subjects/Keywords: HDAC inhibitor SAHA; lymphangiogenesis; breast cancer; VEGF-C; c-Cbl

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APA (6th Edition):

Cheng, H. (2013). Anti-lymphangiogenic action of SAHA on breast cancer. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0403113-151923

Chicago Manual of Style (16th Edition):

Cheng, Hsueh-Tsen. “Anti-lymphangiogenic action of SAHA on breast cancer.” 2013. Doctoral Dissertation, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0403113-151923.

MLA Handbook (7th Edition):

Cheng, Hsueh-Tsen. “Anti-lymphangiogenic action of SAHA on breast cancer.” 2013. Web. 19 Jan 2021.

Vancouver:

Cheng H. Anti-lymphangiogenic action of SAHA on breast cancer. [Internet] [Doctoral dissertation]. NSYSU; 2013. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0403113-151923.

Council of Science Editors:

Cheng H. Anti-lymphangiogenic action of SAHA on breast cancer. [Doctoral Dissertation]. NSYSU; 2013. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0403113-151923


NSYSU

24. Tsai, Han-en. The Therapeutic Potential and Mechanism of POMC stress Hormone for Metastatic Cancer.

Degree: PhD, Institute of Biomedical Sciences, 2012, NSYSU

 Despite the development of novel target therapy drugs in recent years, metastatic cancer remains refractory to current cancer therapies and accounts for the majority of… (more)

Subjects/Keywords: Gene Therapy; Immune system; POMC; Metastasis; Melanoma; Epithelial-mesenchymal transition

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APA (6th Edition):

Tsai, H. (2012). The Therapeutic Potential and Mechanism of POMC stress Hormone for Metastatic Cancer. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823112-163353

Chicago Manual of Style (16th Edition):

Tsai, Han-en. “The Therapeutic Potential and Mechanism of POMC stress Hormone for Metastatic Cancer.” 2012. Doctoral Dissertation, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823112-163353.

MLA Handbook (7th Edition):

Tsai, Han-en. “The Therapeutic Potential and Mechanism of POMC stress Hormone for Metastatic Cancer.” 2012. Web. 19 Jan 2021.

Vancouver:

Tsai H. The Therapeutic Potential and Mechanism of POMC stress Hormone for Metastatic Cancer. [Internet] [Doctoral dissertation]. NSYSU; 2012. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823112-163353.

Council of Science Editors:

Tsai H. The Therapeutic Potential and Mechanism of POMC stress Hormone for Metastatic Cancer. [Doctoral Dissertation]. NSYSU; 2012. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823112-163353


NSYSU

25. Weng, Ching-Chieh. Investigation of TGF-β repressors inactivation enhances metastasis in Pancreatic cancer.

Degree: PhD, Institute of Biomedical Sciences, 2018, NSYSU

 The overall five-year survival rate for pancreatic cancer reported as low as 5% a decade ago, even now still less than 10%. The main issue… (more)

Subjects/Keywords: KLF10; Pancreatic cancer; PDAC mouse model; TGIF1; Metastasis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Weng, C. (2018). Investigation of TGF-β repressors inactivation enhances metastasis in Pancreatic cancer. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0624118-181253

Chicago Manual of Style (16th Edition):

Weng, Ching-Chieh. “Investigation of TGF-β repressors inactivation enhances metastasis in Pancreatic cancer.” 2018. Doctoral Dissertation, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0624118-181253.

MLA Handbook (7th Edition):

Weng, Ching-Chieh. “Investigation of TGF-β repressors inactivation enhances metastasis in Pancreatic cancer.” 2018. Web. 19 Jan 2021.

Vancouver:

Weng C. Investigation of TGF-β repressors inactivation enhances metastasis in Pancreatic cancer. [Internet] [Doctoral dissertation]. NSYSU; 2018. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0624118-181253.

Council of Science Editors:

Weng C. Investigation of TGF-β repressors inactivation enhances metastasis in Pancreatic cancer. [Doctoral Dissertation]. NSYSU; 2018. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0624118-181253


NSYSU

26. Yang, Kuei-Ting. Regulation of Jab1 by Bcr-Abl Oncogene.

Degree: Master, Institute of Biomedical Sciences, 2007, NSYSU

 The COP9 signalosome (CSN) contains eight-subunits and is a highly conserved protein complex implicated in ubiquitin-mediated proteolysis. Jun activation domain-binding protein 1 (Jab1) is the… (more)

Subjects/Keywords: Bcr-Abl; CML; Jab1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yang, K. (2007). Regulation of Jab1 by Bcr-Abl Oncogene. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0816107-112047

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yang, Kuei-Ting. “Regulation of Jab1 by Bcr-Abl Oncogene.” 2007. Thesis, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0816107-112047.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yang, Kuei-Ting. “Regulation of Jab1 by Bcr-Abl Oncogene.” 2007. Web. 19 Jan 2021.

Vancouver:

Yang K. Regulation of Jab1 by Bcr-Abl Oncogene. [Internet] [Thesis]. NSYSU; 2007. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0816107-112047.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yang K. Regulation of Jab1 by Bcr-Abl Oncogene. [Thesis]. NSYSU; 2007. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0816107-112047

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

27. Chen, Bo-rong. Role of insulin resistance in nucleus tractus solitarii on central cardiovascular regulation in rats.

Degree: Master, Institute of Biomedical Sciences, 2007, NSYSU

 Insulin resistance was thought as the major etiology of hypertension of the metabolic syndrome. Both human and animal studies revealed sympathetic overactivity were present in… (more)

Subjects/Keywords: hypertension; nucleus tractus solitarii; insulin resistance

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chen, B. (2007). Role of insulin resistance in nucleus tractus solitarii on central cardiovascular regulation in rats. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0723107-143624

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Bo-rong. “Role of insulin resistance in nucleus tractus solitarii on central cardiovascular regulation in rats.” 2007. Thesis, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0723107-143624.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Bo-rong. “Role of insulin resistance in nucleus tractus solitarii on central cardiovascular regulation in rats.” 2007. Web. 19 Jan 2021.

Vancouver:

Chen B. Role of insulin resistance in nucleus tractus solitarii on central cardiovascular regulation in rats. [Internet] [Thesis]. NSYSU; 2007. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0723107-143624.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen B. Role of insulin resistance in nucleus tractus solitarii on central cardiovascular regulation in rats. [Thesis]. NSYSU; 2007. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0723107-143624

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

28. Yen, Yi-Chen. Molecular Interaction of Tau and Microtubule.

Degree: Master, Institute of Biomedical Sciences, 2002, NSYSU

 Tau protein is one of the microtubule-associated proteins (MAPs) and mainly expressed in neuronal cells. It hasbeen demonstrated that Tau may play an important role… (more)

Subjects/Keywords: phosphorylation; tau protein; microtubule

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Yen, Y. (2002). Molecular Interaction of Tau and Microtubule. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0821102-151102

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Yen, Yi-Chen. “Molecular Interaction of Tau and Microtubule.” 2002. Thesis, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0821102-151102.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Yen, Yi-Chen. “Molecular Interaction of Tau and Microtubule.” 2002. Web. 19 Jan 2021.

Vancouver:

Yen Y. Molecular Interaction of Tau and Microtubule. [Internet] [Thesis]. NSYSU; 2002. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0821102-151102.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yen Y. Molecular Interaction of Tau and Microtubule. [Thesis]. NSYSU; 2002. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0821102-151102

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

29. Chen, Chia-Yi. Divalent cation-induced conformational changes and oligomerization of KChIP1.

Degree: Master, Institute of Biomedical Sciences, 2003, NSYSU

Abstract KChIPs are Kv channel-interacting proteins that bind to the cytoplasmic N-terminus of Kv4 Advisors/Committee Members: Chun-Chang Chang (chair), Long-Sen Chang (committee member), Wen-Chun Hung (chair).

Subjects/Keywords: KChIP; conformational change; oligomerization

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chen, C. (2003). Divalent cation-induced conformational changes and oligomerization of KChIP1. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0619103-133648

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Chia-Yi. “Divalent cation-induced conformational changes and oligomerization of KChIP1.” 2003. Thesis, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0619103-133648.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Chia-Yi. “Divalent cation-induced conformational changes and oligomerization of KChIP1.” 2003. Web. 19 Jan 2021.

Vancouver:

Chen C. Divalent cation-induced conformational changes and oligomerization of KChIP1. [Internet] [Thesis]. NSYSU; 2003. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0619103-133648.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen C. Divalent cation-induced conformational changes and oligomerization of KChIP1. [Thesis]. NSYSU; 2003. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0619103-133648

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

30. Cheng, Yun-Ching. Cloning and functional expression of Taiwan cobra chymotrypsin inhibitor.

Degree: Master, Institute of Biomedical Sciences, 2003, NSYSU

 Previous studies showed that dendrotoxins and B chain of b-Bungarotoxin shared sequence and structural homology with Kunitz-type protease inhibitors. In the present study, the cDNA… (more)

Subjects/Keywords: taiwan cobra; chymotrypsin inhibitor

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Cheng, Y. (2003). Cloning and functional expression of Taiwan cobra chymotrypsin inhibitor. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0620103-142902

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Cheng, Yun-Ching. “Cloning and functional expression of Taiwan cobra chymotrypsin inhibitor.” 2003. Thesis, NSYSU. Accessed January 19, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0620103-142902.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Cheng, Yun-Ching. “Cloning and functional expression of Taiwan cobra chymotrypsin inhibitor.” 2003. Web. 19 Jan 2021.

Vancouver:

Cheng Y. Cloning and functional expression of Taiwan cobra chymotrypsin inhibitor. [Internet] [Thesis]. NSYSU; 2003. [cited 2021 Jan 19]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0620103-142902.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Cheng Y. Cloning and functional expression of Taiwan cobra chymotrypsin inhibitor. [Thesis]. NSYSU; 2003. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0620103-142902

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

[1] [2]

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