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You searched for +publisher:"NSYSU" +contributor:("Michael Hsiao"). Showing records 1 – 5 of 5 total matches.

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NSYSU

1. Yeh, Tung-Chen. Caffeine intake improves fructose-induced hypertension and insulin resistance by enhancing central insulin signaling.

Degree: PhD, Biological Sciences, 2014, NSYSU

 Recent clinical studies found that fructose intake leads to insulin resistance and hypertension. Fructose consumption promotes protein fructosylation and the formation of superoxide. In a… (more)

Subjects/Keywords: hypertension; nucleus tractus solitarii; caffeine; nitric oxide; fructose

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APA (6th Edition):

Yeh, T. (2014). Caffeine intake improves fructose-induced hypertension and insulin resistance by enhancing central insulin signaling. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0212114-135518

Chicago Manual of Style (16th Edition):

Yeh, Tung-Chen. “Caffeine intake improves fructose-induced hypertension and insulin resistance by enhancing central insulin signaling.” 2014. Doctoral Dissertation, NSYSU. Accessed March 08, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0212114-135518.

MLA Handbook (7th Edition):

Yeh, Tung-Chen. “Caffeine intake improves fructose-induced hypertension and insulin resistance by enhancing central insulin signaling.” 2014. Web. 08 Mar 2021.

Vancouver:

Yeh T. Caffeine intake improves fructose-induced hypertension and insulin resistance by enhancing central insulin signaling. [Internet] [Doctoral dissertation]. NSYSU; 2014. [cited 2021 Mar 08]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0212114-135518.

Council of Science Editors:

Yeh T. Caffeine intake improves fructose-induced hypertension and insulin resistance by enhancing central insulin signaling. [Doctoral Dissertation]. NSYSU; 2014. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0212114-135518


NSYSU

2. Yang, Ming-Chang. Investigation of the functional role of Bcl2L12 and its variant in glioma and breast cancer.

Degree: PhD, Biological Sciences, 2017, NSYSU

 Bcl2L12 regulates apoptosis, but the different roles of Bcl2L12 in different cancer types remains controversial. In glioma multiforme (GBM), Bcl2L12 is involved in post-mitochondrial apoptosis… (more)

Subjects/Keywords: Triple-Negative Breast Cancer; Glioblastoma multiform; Breast cancer; Bcl2L12A; Bcl2L12; mRNA biomarker; microRNA

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APA (6th Edition):

Yang, M. (2017). Investigation of the functional role of Bcl2L12 and its variant in glioma and breast cancer. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0813117-095448

Chicago Manual of Style (16th Edition):

Yang, Ming-Chang. “Investigation of the functional role of Bcl2L12 and its variant in glioma and breast cancer.” 2017. Doctoral Dissertation, NSYSU. Accessed March 08, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0813117-095448.

MLA Handbook (7th Edition):

Yang, Ming-Chang. “Investigation of the functional role of Bcl2L12 and its variant in glioma and breast cancer.” 2017. Web. 08 Mar 2021.

Vancouver:

Yang M. Investigation of the functional role of Bcl2L12 and its variant in glioma and breast cancer. [Internet] [Doctoral dissertation]. NSYSU; 2017. [cited 2021 Mar 08]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0813117-095448.

Council of Science Editors:

Yang M. Investigation of the functional role of Bcl2L12 and its variant in glioma and breast cancer. [Doctoral Dissertation]. NSYSU; 2017. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0813117-095448


NSYSU

3. Chen, Yi-Li. Role of Annexin-7 in the Molecular Pathogenesis of Malignant Tumor.

Degree: Master, Institute of Biomedical Sciences, 2003, NSYSU

 The annexin-7 (ANX7) gene is located on human chromosome 10q21, a site long been hypothesized to harbor a tumor suppressor gene (TSG) associated with brain,… (more)

Subjects/Keywords: ANX7 astrocytoma PCR WB

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Chen, Y. (2003). Role of Annexin-7 in the Molecular Pathogenesis of Malignant Tumor. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0901103-095317

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Chen, Yi-Li. “Role of Annexin-7 in the Molecular Pathogenesis of Malignant Tumor.” 2003. Thesis, NSYSU. Accessed March 08, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0901103-095317.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Chen, Yi-Li. “Role of Annexin-7 in the Molecular Pathogenesis of Malignant Tumor.” 2003. Web. 08 Mar 2021.

Vancouver:

Chen Y. Role of Annexin-7 in the Molecular Pathogenesis of Malignant Tumor. [Internet] [Thesis]. NSYSU; 2003. [cited 2021 Mar 08]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0901103-095317.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen Y. Role of Annexin-7 in the Molecular Pathogenesis of Malignant Tumor. [Thesis]. NSYSU; 2003. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0901103-095317

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

4. Huang, Kai-Lieh. Androgen Promotes Osteoblast Proliferation through Activation of Phosphatidylinositol-3-OH Kinase /Akt Signaling Pathway.

Degree: Master, Biological Sciences, 2003, NSYSU

 Androgen has been shown to stimulate proliferation of osteoblast-like MC3T3-E1 cells. However, the molecular mechanism responsible for this effect remains to be elucidated. In the… (more)

Subjects/Keywords: Akt; PI(3)K; proliferation; androgen; osteoblast

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Huang, K. (2003). Androgen Promotes Osteoblast Proliferation through Activation of Phosphatidylinositol-3-OH Kinase /Akt Signaling Pathway. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0708103-151701

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Huang, Kai-Lieh. “Androgen Promotes Osteoblast Proliferation through Activation of Phosphatidylinositol-3-OH Kinase /Akt Signaling Pathway.” 2003. Thesis, NSYSU. Accessed March 08, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0708103-151701.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Huang, Kai-Lieh. “Androgen Promotes Osteoblast Proliferation through Activation of Phosphatidylinositol-3-OH Kinase /Akt Signaling Pathway.” 2003. Web. 08 Mar 2021.

Vancouver:

Huang K. Androgen Promotes Osteoblast Proliferation through Activation of Phosphatidylinositol-3-OH Kinase /Akt Signaling Pathway. [Internet] [Thesis]. NSYSU; 2003. [cited 2021 Mar 08]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0708103-151701.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huang K. Androgen Promotes Osteoblast Proliferation through Activation of Phosphatidylinositol-3-OH Kinase /Akt Signaling Pathway. [Thesis]. NSYSU; 2003. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0708103-151701

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation


NSYSU

5. Lin, James. The Differential Expression of Bcl10 in the Tumor Cell Lines.

Degree: Master, Biological Sciences, 2004, NSYSU

 Bcl10 is one of the apoptosis regulatory protein. It is located at 1p22,one site harbor tumor suppressor tumor gene. We screen Bcl10 expression in different… (more)

Subjects/Keywords: tumor suppressor tumor gene

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6th Edition):

Lin, J. (2004). The Differential Expression of Bcl10 in the Tumor Cell Lines. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0816104-155835

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16th Edition):

Lin, James. “The Differential Expression of Bcl10 in the Tumor Cell Lines.” 2004. Thesis, NSYSU. Accessed March 08, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0816104-155835.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7th Edition):

Lin, James. “The Differential Expression of Bcl10 in the Tumor Cell Lines.” 2004. Web. 08 Mar 2021.

Vancouver:

Lin J. The Differential Expression of Bcl10 in the Tumor Cell Lines. [Internet] [Thesis]. NSYSU; 2004. [cited 2021 Mar 08]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0816104-155835.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lin J. The Differential Expression of Bcl10 in the Tumor Cell Lines. [Thesis]. NSYSU; 2004. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0816104-155835

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

.