+publisher:"NSYSU" +contributor:("Hung-Wen Huamg")

NSYSU

1. Chen, Hsiu-Ping. Genetic susceptibilities for COX-1, CYP2C9, CYP2C19 and UGT1A6 on NSAIDs-related peptic ulcer.

Degree: Master, Institute of Biomedical Sciences, 2013, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0521113-113657

► Most cases of peptic ulcer disease result from gastric infection with Helicobacter pylori or from chronic use of aspirin or nonsteroidal antiinflammatory drugs (NSAIDs). Aspirin… (more)

Subjects/Keywords: COX-1; polymorphism; CYP2C9; UGT1A6; CYP2C19; peptic ulcer; aspirin

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Chen, H. (2013). Genetic susceptibilities for COX-1, CYP2C9, CYP2C19 and UGT1A6 on NSAIDs-related peptic ulcer. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0521113-113657

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Chen, Hsiu-Ping. “Genetic susceptibilities for COX-1, CYP2C9, CYP2C19 and UGT1A6 on NSAIDs-related peptic ulcer.” 2013. Thesis, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0521113-113657.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Chen, Hsiu-Ping. “Genetic susceptibilities for COX-1, CYP2C9, CYP2C19 and UGT1A6 on NSAIDs-related peptic ulcer.” 2013. Web. 25 Jan 2021.

Vancouver:

Chen H. Genetic susceptibilities for COX-1, CYP2C9, CYP2C19 and UGT1A6 on NSAIDs-related peptic ulcer. [Internet] [Thesis]. NSYSU; 2013. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0521113-113657.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen H. Genetic susceptibilities for COX-1, CYP2C9, CYP2C19 and UGT1A6 on NSAIDs-related peptic ulcer. [Thesis]. NSYSU; 2013. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0521113-113657

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

2. Chen, Te-Chung. The Roles of p38 MAPK and SUMOs in Arsenic Trioxide-induced Apoptosis.

Degree: Master, Institute of Biomedical Sciences, 2013, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0521113-113828

► Arsenic trioxide (ATO), an anti-cancer drug for acute promyelocytic leukemia (APL), was able to induce apoptosis in several cancer cell lines including solid tumors, myeloma… (more)

Subjects/Keywords: ATO; Arsenic trioxide; Helicobacter pylori (Hp); Apoptosis; SUMO; p38

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Chen, T. (2013). The Roles of p38 MAPK and SUMOs in Arsenic Trioxide-induced Apoptosis. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0521113-113828

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Chen, Te-Chung. “The Roles of p38 MAPK and SUMOs in Arsenic Trioxide-induced Apoptosis.” 2013. Thesis, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0521113-113828.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Chen, Te-Chung. “The Roles of p38 MAPK and SUMOs in Arsenic Trioxide-induced Apoptosis.” 2013. Web. 25 Jan 2021.

Vancouver:

Chen T. The Roles of p38 MAPK and SUMOs in Arsenic Trioxide-induced Apoptosis. [Internet] [Thesis]. NSYSU; 2013. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0521113-113828.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen T. The Roles of p38 MAPK and SUMOs in Arsenic Trioxide-induced Apoptosis. [Thesis]. NSYSU; 2013. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0521113-113828

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

3. Fan, Hsiao-Li. Macrophage uptake of platelets-H. pylori is mainly IgG-mediated.

Degree: Master, Institute of Biomedical Sciences, 2015, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0719115-101356

► One of the most important physiological functions of platelets (PLTs) is clotting in the wound. Recent studies have revealed that PLTs also play important roles… (more)

▼ One of the most important physiological functions of platelets (PLTs) is clotting in the wound. Recent studies have revealed that PLTs also play important roles in innate immunity. Weâre interested in the association between H. pylori and PLTs due to patients with thrombocytopenia often accompanied with H. pylori infection. There is growing evidence showed that the PLTs count recovered after H. pylori eradication therapy. Itâs known that adhesion of some strains of H. pylori to PLT leaded to PLT activation, aggregation and apoptosis. However, the role of PLT in the innate immunity of macrophage eliminating H. pylori, and in the association between phagocytosis and H. pylori-induced thrombocytopenia remains unclear. We therefore investigated the association between the H. pylori infected PLTs and the uptake of PLTs by macrophage in this studies. Firstly, itâs observed that the expression of phosphatidylserine (PS), a molecular marker for macrophage clearance, increased on washed PLTs in response to H. pylori and lipopolysaccharide (LPS) of H. pylori stimulation. Secondly, the uptake of H. pylori infected washed PLTs by macrophage was slightly enhances, but it was slightly decreased when LPS was used instead of H. pylori. Finally, we investigated the role of H. pylori antibody. Results showed that H. pylori positive sera significantly enhanced the phagocytosis of washed PLTs in response to some strains of H. pylori infection. Our results suggest the increased uptake of PLTs-H. pylori by macrophage was mainly IgG-mediated. It would lead to decrease in platelet counts. Our previous data on H. pylori-induced PLTs aggregation and apoptosis and the results of this study will provide further understandings on the pathogenesis of H. pylori-induced thrombocytopenia. It will further assist physicians in diagnosis and making therapeutic decisions on the H. pylori infected patients with unknown reason thrombocytopenia. Advisors/Committee Members: Wen%20Huamg%22%29&pagesize-30">Hung-Wen Huamg (chair), Angela Chen (committee member), Ping-I Hsu (chair).

Subjects/Keywords: Helicobacter pylori (H. pylori); Platelet; Immunoglobulin G (IgG); Macrophage; Thrombocytopenia

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Fan, H. (2015). Macrophage uptake of platelets-H. pylori is mainly IgG-mediated. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0719115-101356

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Fan, Hsiao-Li. “Macrophage uptake of platelets-H. pylori is mainly IgG-mediated.” 2015. Thesis, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0719115-101356.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Fan, Hsiao-Li. “Macrophage uptake of platelets-H. pylori is mainly IgG-mediated.” 2015. Web. 25 Jan 2021.

Vancouver:

Fan H. Macrophage uptake of platelets-H. pylori is mainly IgG-mediated. [Internet] [Thesis]. NSYSU; 2015. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0719115-101356.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Fan H. Macrophage uptake of platelets-H. pylori is mainly IgG-mediated. [Thesis]. NSYSU; 2015. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0719115-101356

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

4. Hsu, Chih-wei. Investigation of the SUMOylation sites of Gfi1.

Degree: Master, Institute of Biomedical Sciences, 2015, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0421115-100738

► The transcriptional repressor Growth Factor Independence 1 (Gfi1) regulates many biological functions, such as development of granulocytes, T-cell differentiation, macrophage-dependent cytokine production and the development… (more)

Subjects/Keywords: SIM; Colocalization; In vitro SUMOylation; SUMO; Gfi1

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Hsu, C. (2015). Investigation of the SUMOylation sites of Gfi1. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0421115-100738

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Hsu, Chih-wei. “Investigation of the SUMOylation sites of Gfi1.” 2015. Thesis, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0421115-100738.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Hsu, Chih-wei. “Investigation of the SUMOylation sites of Gfi1.” 2015. Web. 25 Jan 2021.

Vancouver:

Hsu C. Investigation of the SUMOylation sites of Gfi1. [Internet] [Thesis]. NSYSU; 2015. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0421115-100738.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hsu C. Investigation of the SUMOylation sites of Gfi1. [Thesis]. NSYSU; 2015. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0421115-100738

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

5. Lin, Tse-yu. Investigation of the SUMO-interacting motif of p38.

Degree: Master, Institute of Biomedical Sciences, 2015, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0721115-145645

► SUMOylation is a posttranslational modification by the small ubiquitin-related modifier (SUMO), SUMOylation plays important roles in the regulation of diverse cellular processes. SUMO can interact… (more)

Subjects/Keywords: p38; SUMO-interacting motif; non-covalent interaction; SUMO-1; SUMO-2

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Lin, T. (2015). Investigation of the SUMO-interacting motif of p38. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0721115-145645

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Lin, Tse-yu. “Investigation of the SUMO-interacting motif of p38.” 2015. Thesis, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0721115-145645.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Lin, Tse-yu. “Investigation of the SUMO-interacting motif of p38.” 2015. Web. 25 Jan 2021.

Vancouver:

Lin T. Investigation of the SUMO-interacting motif of p38. [Internet] [Thesis]. NSYSU; 2015. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0721115-145645.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lin T. Investigation of the SUMO-interacting motif of p38. [Thesis]. NSYSU; 2015. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0721115-145645

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

6. Hsu, Cheng-Chieh. Study of CYP26B1 and associated proteins in betel quid induced oral cancers.

Degree: Master, Institute of Biomedical Sciences, 2013, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0615113-102942

► Approximately 600 million people in the world chew betel quid (BQ). In 2004, the International Agency for Research on Cancer (IARC) declared BQ chewing without… (more)

Subjects/Keywords: Betel Quid; Arecoline; CYP26B1; Areca nut; Oral cancer; Proteomic

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Hsu, C. (2013). Study of CYP26B1 and associated proteins in betel quid induced oral cancers. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0615113-102942

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Hsu, Cheng-Chieh. “Study of CYP26B1 and associated proteins in betel quid induced oral cancers.” 2013. Thesis, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0615113-102942.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Hsu, Cheng-Chieh. “Study of CYP26B1 and associated proteins in betel quid induced oral cancers.” 2013. Web. 25 Jan 2021.

Vancouver:

Hsu C. Study of CYP26B1 and associated proteins in betel quid induced oral cancers. [Internet] [Thesis]. NSYSU; 2013. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0615113-102942.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hsu C. Study of CYP26B1 and associated proteins in betel quid induced oral cancers. [Thesis]. NSYSU; 2013. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0615113-102942

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

7. Yeh, Chung-Yu. Studies on the activity of Lhb proximal promoter region and its peroxisome proliferator-activated receptors-responsive elements in rat pituitary cells.

Degree: Master, Institute of Biomedical Sciences, 2013, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0727113-205307

► In our previous studies, treatment of peroxisome proliferator activated receptor (PPARs) agonists and antagonists, respectively, induced and suppressed endogenous luteinizing hormone subunit beta (LHB) gene… (more)

Subjects/Keywords: luteinizing hormone subunit beta; peroxisome proliferator-activated receptors; hematopoietic prostaglandin D synthase; rat pituitary cells; peroxisome proliferator response element

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Yeh, C. (2013). Studies on the activity of Lhb proximal promoter region and its peroxisome proliferator-activated receptors-responsive elements in rat pituitary cells. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0727113-205307

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Yeh, Chung-Yu. “Studies on the activity of Lhb proximal promoter region and its peroxisome proliferator-activated receptors-responsive elements in rat pituitary cells.” 2013. Thesis, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0727113-205307.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Yeh, Chung-Yu. “Studies on the activity of Lhb proximal promoter region and its peroxisome proliferator-activated receptors-responsive elements in rat pituitary cells.” 2013. Web. 25 Jan 2021.

Vancouver:

Yeh C. Studies on the activity of Lhb proximal promoter region and its peroxisome proliferator-activated receptors-responsive elements in rat pituitary cells. [Internet] [Thesis]. NSYSU; 2013. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0727113-205307.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yeh C. Studies on the activity of Lhb proximal promoter region and its peroxisome proliferator-activated receptors-responsive elements in rat pituitary cells. [Thesis]. NSYSU; 2013. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0727113-205307

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

8. Li, Yi-Jin. The Molecular Study of Fluoxetine Induces Cell Cycle Arrest and Apoptosis in Pancreatic Cancer Cells.

Degree: Master, Institute of Biomedical Sciences, 2014, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0513114-143723

► Fluoxetine (FLX) is a commonly antidepressant for oral administration and belonging to the selective serotonin reuptake inhibitors (SSRI), which is used for the treatment of… (more)

Subjects/Keywords: migration; proliferation; Pancreatic cancer; apoptosis; cell cycle

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Li, Y. (2014). The Molecular Study of Fluoxetine Induces Cell Cycle Arrest and Apoptosis in Pancreatic Cancer Cells. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0513114-143723

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Li, Yi-Jin. “The Molecular Study of Fluoxetine Induces Cell Cycle Arrest and Apoptosis in Pancreatic Cancer Cells.” 2014. Thesis, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0513114-143723.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Li, Yi-Jin. “The Molecular Study of Fluoxetine Induces Cell Cycle Arrest and Apoptosis in Pancreatic Cancer Cells.” 2014. Web. 25 Jan 2021.

Vancouver:

Li Y. The Molecular Study of Fluoxetine Induces Cell Cycle Arrest and Apoptosis in Pancreatic Cancer Cells. [Internet] [Thesis]. NSYSU; 2014. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0513114-143723.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Li Y. The Molecular Study of Fluoxetine Induces Cell Cycle Arrest and Apoptosis in Pancreatic Cancer Cells. [Thesis]. NSYSU; 2014. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0513114-143723

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

9. Tai , Po-han. Therapeutic potential and mechanism of LECT2 for liver cancer.

Degree: Master, Institute of Biomedical Sciences, 2014, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0621114-111413

► Hepatocellular carcinoma (HCC) is one of the most common cancers in the world. However, current therapeutic modalities for HCC, including surgery, transendothelial embolization (TAE) and… (more)

▼ Hepatocellular carcinoma (HCC) is one of the most common cancers in the world. However, current therapeutic modalities for HCC, including surgery, transendothelial embolization (TAE) and radiofrequency ablation (RFA), remain largely ineffectively. One major mechanism underlying the failure for cancer therapy is the presence of cancer stem cells (CSCs), which possess the capability of drug efflux and self renewal. The emergence of CSCs results in resistance and metastasis, which ultimately leads to tumor recurrence. Wnt/Î²-catenin signaling pathway participates in CSCs genesis and targeting Wnt/Î²-catenin may hold promise for CSCs control. Leukocyte cell-derived chemotaxin 2 (LECT2) was first isolated as a 16-kDa secreted protein from cultured media of phytohemagglutinin-activated human T-cell leukemia SKW-3 cells. Recent studies suggest that LECT2 may act as a tumor suppressor gene in liver through antagonizing Wnt signaling pathway. However, the detailed mechanism of LECT2 in liver cancer remains unclear. In this study, we first observed that intra-tumor LECT2 gene delivery effectively suppressed the progression of orthotopic Novikoff hepatoma in rats through apoptosis induction, angiogenesis blockade and cancer stemness inhibition. Subsequently, we investigated the role of LECT2 on oncogenic behaviors and stemness of hepatoma cells. Despite of marginal effect on proliferation, recombinant LECT2 dose-dependently inhibited the migration, invasion and anchorage-independent growth of HCC cells. Besides, LECT2 application attenuated the epithelial-mesenchymal transition (EMT), drug-efflux and self-renewal capabilities of HCC cells. This was correlated with the downregulation of EMT marker genes, Wnt/Î²-catenin signaling pathway and c-Met activation. In summary, LECT2 suppresses HCC progression via attenuating the hepatic cancer stemness. Advisors/Committee Members: Wen%20Huamg%22%29&pagesize-30">Hung-Wen Huamg (chair), Deng-chyang Wu (committee member), Po-Lin Kuo (chair).

Subjects/Keywords: Î²-catenin; Hepatocellular carcinoma; Wnt signaling pathway; Cancer stem cell; LECT2

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Tai , P. (2014). Therapeutic potential and mechanism of LECT2 for liver cancer. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0621114-111413

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Tai , Po-han. “Therapeutic potential and mechanism of LECT2 for liver cancer.” 2014. Thesis, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0621114-111413.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Tai , Po-han. “Therapeutic potential and mechanism of LECT2 for liver cancer.” 2014. Web. 25 Jan 2021.

Vancouver:

Tai P. Therapeutic potential and mechanism of LECT2 for liver cancer. [Internet] [Thesis]. NSYSU; 2014. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0621114-111413.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tai P. Therapeutic potential and mechanism of LECT2 for liver cancer. [Thesis]. NSYSU; 2014. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0621114-111413

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

10. Chung, Yi-An. Bioactive evaluation of anti-oral cancer effects in methanol crude extracts of several Lauraceae plants.

Degree: Master, Institute of Biomedical Sciences, 2014, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0628114-101929

► Cryptocarya-derived natural products were reported to have many biological effects such antiproliferation of some cancers. However, the possible antioral cancer effect of Cryptocarya-derived natural products… (more)

Subjects/Keywords: apoptosis; antiproliferation; oral cancer; Cryptocarya; ROS

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Chung, Y. (2014). Bioactive evaluation of anti-oral cancer effects in methanol crude extracts of several Lauraceae plants. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0628114-101929

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Chung, Yi-An. “Bioactive evaluation of anti-oral cancer effects in methanol crude extracts of several Lauraceae plants.” 2014. Thesis, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0628114-101929.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Chung, Yi-An. “Bioactive evaluation of anti-oral cancer effects in methanol crude extracts of several Lauraceae plants.” 2014. Web. 25 Jan 2021.

Vancouver:

Chung Y. Bioactive evaluation of anti-oral cancer effects in methanol crude extracts of several Lauraceae plants. [Internet] [Thesis]. NSYSU; 2014. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0628114-101929.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chung Y. Bioactive evaluation of anti-oral cancer effects in methanol crude extracts of several Lauraceae plants. [Thesis]. NSYSU; 2014. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0628114-101929

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

11. Hou, You-syuan. Using proteomic approach to discover novel biomarkers involved in chemoresistance to B-RAF inhibitor in thyroid cancer.

Degree: Master, Institute of Biomedical Sciences, 2014, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0631114-003207

► Thyroid carcinoma (TC) is the most common endocrine malignancy and is one of the most rapidly increasing human cancers worldwide. Anaplastic thyroid carcinoma (ATC) is… (more)

Subjects/Keywords: thyroid cancer; BRAF inhibitor; proteomic; apoptosis; resistance

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Hou, Y. (2014). Using proteomic approach to discover novel biomarkers involved in chemoresistance to B-RAF inhibitor in thyroid cancer. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0631114-003207

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Hou, You-syuan. “Using proteomic approach to discover novel biomarkers involved in chemoresistance to B-RAF inhibitor in thyroid cancer.” 2014. Thesis, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0631114-003207.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Hou, You-syuan. “Using proteomic approach to discover novel biomarkers involved in chemoresistance to B-RAF inhibitor in thyroid cancer.” 2014. Web. 25 Jan 2021.

Vancouver:

Hou Y. Using proteomic approach to discover novel biomarkers involved in chemoresistance to B-RAF inhibitor in thyroid cancer. [Internet] [Thesis]. NSYSU; 2014. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0631114-003207.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hou Y. Using proteomic approach to discover novel biomarkers involved in chemoresistance to B-RAF inhibitor in thyroid cancer. [Thesis]. NSYSU; 2014. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0631114-003207

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

12. SU, HUEI-YU. Inflammatory cytokine IL6 promotes in vitro cell proliferation and cell migration in SMAD4 null pancreatic cancer cells.

Degree: Master, Institute of Biomedical Sciences, 2014, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0631114-021652

► Early cancers of the pancreas often cause no symptoms leading to pancreatic cancer often can't be detected until the cancer already reach to advanced stage.… (more)

▼ Early cancers of the pancreas often cause no symptoms leading to pancreatic cancer often can't be detected until the cancer already reach to advanced stage. The prognosis of patients with pancreatic ductal adenocarcinoma (PDAC) is extremely poor and 5 year survival rate only 4%. Many studies indicated that advanced or metastatic pancreatic cancer patientscan be detected higher levels of Interleukin-6 (IL-6) in their blood. IL-6 is a multifunctional cytokine which plays a central role in host defense due to its wide range of immune and biologic activities in different types of cells including tumor cells. These effects of IL-6 can be transduced by multiple signaling pathways, in particular signal transducer and activator of transcription 3 (STAT3). Hence we want to figure out whether IL-6 plays an important role in mediating EMT in Smad4-null pancreatic cancers. We assessed the overall effect of IL-6 on TGF-Î²1 signaling in human PDAC cells and found that Smad4 null PDAC cells could increase the TGF-Î² protein expression level after IL-6 treatment in vitro. In our laboratoryâs previous study, Nestin was involved in TGF-Î²1/Smad4 signaling pathway to induce EMT in PDAC. Transfection of IL-6 siRNA markedly decreased Nestin mRNA and protein levels compared to control cells, which lead to reduce various EMT-related marker SMA, E-cadherin and vimentin expression. Thereby, the treatment of IL-6 may regulate Nestin protein expression in Smad4 null PDAC cells. In Western blot and Wound healing analysis, we alos observed that IL-6 could induce EMT and promoted cell migration in Smad4-deficient PDAC cells. Moreover, Western blots revealed that overexpression or knockdown of SMAD4 had an impact on the regulation of IL- 6 expression in pancreatic cancer cell lines. Our research suggest that IL-6 could induce P-Stat3 to promote EMT may through TGF-Î² signaling pathway with ablation of SMAD4 gene in pancreatic cancer. Furthermore, MTT assays showed IL-6 increased the ability of cell proliferation in Smad4-deficient PDAC cells. Silimar results were obtained when we, transfected with IL-6 siRNA to silence IL-6 protein expression to led to reduce the ability of cell growth in Smad4-deficient PDAC cells. Keywords: PDAC, Interleukin-6, Nestin ,TGF-Î²1/Smad4 ,EMT Advisors/Committee Members: Wen%20Huamg%22%29&pagesize-30">Hung-Wen Huamg (chair), Chang-Chun Hsiao (chair), hung%20Cheng%22%29&pagesize-30">Kuang-hung Cheng (committee member).

Subjects/Keywords: EMT; TGF-Î²1/Smad4; Nestin; Interleukin-6; PDAC

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

SU, H. (2014). Inflammatory cytokine IL6 promotes in vitro cell proliferation and cell migration in SMAD4 null pancreatic cancer cells. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0631114-021652

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

SU, HUEI-YU. “Inflammatory cytokine IL6 promotes in vitro cell proliferation and cell migration in SMAD4 null pancreatic cancer cells.” 2014. Thesis, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0631114-021652.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

SU, HUEI-YU. “Inflammatory cytokine IL6 promotes in vitro cell proliferation and cell migration in SMAD4 null pancreatic cancer cells.” 2014. Web. 25 Jan 2021.

Vancouver:

SU H. Inflammatory cytokine IL6 promotes in vitro cell proliferation and cell migration in SMAD4 null pancreatic cancer cells. [Internet] [Thesis]. NSYSU; 2014. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0631114-021652.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

SU H. Inflammatory cytokine IL6 promotes in vitro cell proliferation and cell migration in SMAD4 null pancreatic cancer cells. [Thesis]. NSYSU; 2014. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0631114-021652

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

13. Peng, Yu-Ting. Studies on the regulatory mechanisms of GTN-induced protein kinase inhibitors in hepatocellular carcinoma-derived cells.

Degree: Master, Institute of Biomedical Sciences, 2014, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0720114-004147

► The study was to investigate the regulatory mechanisms of goniothalamin (GTN)-induced protein kinase inhibitors, which means cyclin-dependent kinase inhibitors (CKIs), in two hepatocellular carcinoma (HCC)-derived… (more)

Subjects/Keywords: E3-ligase inhibitor; HDAC inhibitor; Goniothalamin (GTN); CDKN1B; Cyclin-dependent kinase inhibitors (CKIs); CDKN1C

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Peng, Y. (2014). Studies on the regulatory mechanisms of GTN-induced protein kinase inhibitors in hepatocellular carcinoma-derived cells. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0720114-004147

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Peng, Yu-Ting. “Studies on the regulatory mechanisms of GTN-induced protein kinase inhibitors in hepatocellular carcinoma-derived cells.” 2014. Thesis, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0720114-004147.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Peng, Yu-Ting. “Studies on the regulatory mechanisms of GTN-induced protein kinase inhibitors in hepatocellular carcinoma-derived cells.” 2014. Web. 25 Jan 2021.

Vancouver:

Peng Y. Studies on the regulatory mechanisms of GTN-induced protein kinase inhibitors in hepatocellular carcinoma-derived cells. [Internet] [Thesis]. NSYSU; 2014. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0720114-004147.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Peng Y. Studies on the regulatory mechanisms of GTN-induced protein kinase inhibitors in hepatocellular carcinoma-derived cells. [Thesis]. NSYSU; 2014. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0720114-004147

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

14. Jheng, Huei-Ying. Studies on the relationship between ABT-751-induced apoptosis and autophagy in the hepatocellular carcinoma Hep-3B cells.

Degree: Master, Institute of Biomedical Sciences, 2014, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0720114-115444

► The study was to evaluate the effect of ABT-751 on apoptosis and autophagy in Hep-3B cells, a human hepatocellular carcinoma (HCC)-derived cell line. ABT-751 is… (more)

Subjects/Keywords: Microtubules; apoptosis; apoptosis-inducing factor; autophagy; caspase-independent pathway; ABT-751

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Jheng, H. (2014). Studies on the relationship between ABT-751-induced apoptosis and autophagy in the hepatocellular carcinoma Hep-3B cells. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0720114-115444

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Jheng, Huei-Ying. “Studies on the relationship between ABT-751-induced apoptosis and autophagy in the hepatocellular carcinoma Hep-3B cells.” 2014. Thesis, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0720114-115444.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Jheng, Huei-Ying. “Studies on the relationship between ABT-751-induced apoptosis and autophagy in the hepatocellular carcinoma Hep-3B cells.” 2014. Web. 25 Jan 2021.

Vancouver:

Jheng H. Studies on the relationship between ABT-751-induced apoptosis and autophagy in the hepatocellular carcinoma Hep-3B cells. [Internet] [Thesis]. NSYSU; 2014. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0720114-115444.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Jheng H. Studies on the relationship between ABT-751-induced apoptosis and autophagy in the hepatocellular carcinoma Hep-3B cells. [Thesis]. NSYSU; 2014. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0720114-115444

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

15. Chen, Ya-chun. Studies on the relationship between autophagy and apoptosis in hepatocellular carcinoma-derived cells Huh7 by treatment with ABT-751.

Degree: Master, Institute of Biomedical Sciences, 2014, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0725114-131202

► The objective was to study the effects of ABT-751 on apoptosis and autophagy in hepatocellular carcinoma-derived cells. Hepatocellular carcinoma (HCC) is a common malignant tumor… (more)

Subjects/Keywords: Hepatocellular carcinoma; Autophagy; Apoptosis; Microtubule; ABT-751

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Chen, Y. (2014). Studies on the relationship between autophagy and apoptosis in hepatocellular carcinoma-derived cells Huh7 by treatment with ABT-751. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0725114-131202

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Chen, Ya-chun. “Studies on the relationship between autophagy and apoptosis in hepatocellular carcinoma-derived cells Huh7 by treatment with ABT-751.” 2014. Thesis, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0725114-131202.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Chen, Ya-chun. “Studies on the relationship between autophagy and apoptosis in hepatocellular carcinoma-derived cells Huh7 by treatment with ABT-751.” 2014. Web. 25 Jan 2021.

Vancouver:

Chen Y. Studies on the relationship between autophagy and apoptosis in hepatocellular carcinoma-derived cells Huh7 by treatment with ABT-751. [Internet] [Thesis]. NSYSU; 2014. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0725114-131202.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen Y. Studies on the relationship between autophagy and apoptosis in hepatocellular carcinoma-derived cells Huh7 by treatment with ABT-751. [Thesis]. NSYSU; 2014. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0725114-131202

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

16. Su, Pei-xuan. CB1 Antisense in the Treatment of Liver Fibrosis.

Degree: Master, Institute of Biomedical Sciences, 2014, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0801114-161740

► Liver fibrosis results from chronic injury to the liver and inflammation activation of hepatic stellate cells produce excessive accumulation of type I collagen proteins, ultimately… (more)

▼ Liver fibrosis results from chronic injury to the liver and inflammation activation of hepatic stellate cells produce excessive accumulation of type I collagen proteins, ultimately leads to cirrhosis, a major public health in Taiwan. Cannabinoid receptor 1 (CB1) was highly induced in human cirrhotic samples. CB1 transgenic mice (CB1 overexpression) induced by carbon tetrachloride (CCl4 ) for 4 weeks with higher liver fibrosis level than the wild type mice. Collagen type I in liver tissue of CB1 transgenic mice is higher than wild type mice by Massonâs trichrome stain and immunohistochemistry(IHC) stain. Both CB1 and Î±-SMA Immunohistochemistry (liver fibrosis biomarker) expression in liver tissue of CB1 transgenic mice significantly increased compared with the wild type mice. Overexpression of CB1 promotes progression of fibrosis. This suggests that CB1 antisense oligonucleotide may have therapeutics effect for liver fibrosis. B6 mice with CCl4-induced liver fibrosis were randomized to receive CB1 antisense oligonucleotide, CB1 sense oligonucleotide, or the CB1 antagonist AM251 for 4 weeks. After B6 mice with liver fibrosis had 28 days CB1 antisense oligonucleotide treatment, type I collagen expression in liver tissue of CB1 antisense oligonucleotide therapy group returned to similar with normal mice. The CB1, Î±-SMA expression in liver tissue of CB1 antisense oligonucleotide therapy group were also significantly decreased compared with the liver fibrosis mice. The hepatic IL-6 expression in CB1 antisense oligonucleotide therapy group was significantly decreased than liver fibrosis group. For serum albumin expression respect, CB1 antisense oligonucleotide therapy group were higher than both liver fibrosis mice and normal mice. The CB1 antisense oligonucleotide therapy group liver function is restored to normal. Genetic and pharmacologic blockade of CB1 by CB1 antisense oligonucleotide and AM251 attenuated liver fibrosis level and can repair liver fibrosis mice hepatic function to normal. Modulation of CB1 signaling has therapeutic potential for liver fibrosis. Advisors/Committee Members: Chang-Chun Hsiao (committee member), Chun-lin Chen (chair), Wen%20Huamg%22%29&pagesize-30">Hung-Wen Huamg (chair), hung%20Cheng%22%29&pagesize-30">Kuang-hung Cheng (committee member).

Subjects/Keywords: AM251; Collagen type I; Liver fibrosis; CB1 antisense oligonucleotide; CB1 receptor

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Su, P. (2014). CB1 Antisense in the Treatment of Liver Fibrosis. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0801114-161740

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Su, Pei-xuan. “CB1 Antisense in the Treatment of Liver Fibrosis.” 2014. Thesis, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0801114-161740.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Su, Pei-xuan. “CB1 Antisense in the Treatment of Liver Fibrosis.” 2014. Web. 25 Jan 2021.

Vancouver:

Su P. CB1 Antisense in the Treatment of Liver Fibrosis. [Internet] [Thesis]. NSYSU; 2014. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0801114-161740.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Su P. CB1 Antisense in the Treatment of Liver Fibrosis. [Thesis]. NSYSU; 2014. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0801114-161740

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

17. Hsiao, Hsuan-Yi. Exercise protein irisin promotes angiogenesis in endothelial cells.

Degree: Master, Institute of Biomedical Sciences, 2014, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0805114-103028

► Myokinesis a critical cytokines in organism major secretes from muscle cells after doing exercise. Myokines involved in angiogenesis, tissue regenerate and repair, preserved normal functions… (more)

Subjects/Keywords: Angiogenesis; white adipose; brown adipose; FNDC5 (Fibronectin type III domain-containing protein 5); endothelial cells

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Hsiao, H. (2014). Exercise protein irisin promotes angiogenesis in endothelial cells. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0805114-103028

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Hsiao, Hsuan-Yi. “Exercise protein irisin promotes angiogenesis in endothelial cells.” 2014. Thesis, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0805114-103028.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Hsiao, Hsuan-Yi. “Exercise protein irisin promotes angiogenesis in endothelial cells.” 2014. Web. 25 Jan 2021.

Vancouver:

Hsiao H. Exercise protein irisin promotes angiogenesis in endothelial cells. [Internet] [Thesis]. NSYSU; 2014. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0805114-103028.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Hsiao H. Exercise protein irisin promotes angiogenesis in endothelial cells. [Thesis]. NSYSU; 2014. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0805114-103028

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

18. Wan, Sih-Wei. Evaluation of HeyL alteration and study its tumor suppressor function in hepatocellular carcinoma.

Degree: Master, Institute of Biomedical Sciences, 2013, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0609113-163231

► The Hairy/Enhancer of split-related with YRPW-like motif (HEY) family of proteins were first identified as transcriptional repressors and were downstream effectors of Notch and BMP… (more)

Subjects/Keywords: tumor cell proliferation; HeyL; tumor metastasis; Notch; HCC

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Wan, S. (2013). Evaluation of HeyL alteration and study its tumor suppressor function in hepatocellular carcinoma. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0609113-163231

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Wan, Sih-Wei. “Evaluation of HeyL alteration and study its tumor suppressor function in hepatocellular carcinoma.” 2013. Thesis, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0609113-163231.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Wan, Sih-Wei. “Evaluation of HeyL alteration and study its tumor suppressor function in hepatocellular carcinoma.” 2013. Web. 25 Jan 2021.

Vancouver:

Wan S. Evaluation of HeyL alteration and study its tumor suppressor function in hepatocellular carcinoma. [Internet] [Thesis]. NSYSU; 2013. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0609113-163231.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wan S. Evaluation of HeyL alteration and study its tumor suppressor function in hepatocellular carcinoma. [Thesis]. NSYSU; 2013. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0609113-163231

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

19. Huang, Wei-han. Extracorporeal shock wave therapy ameliorates kidney functions in diabetic nephropathy rat.

Degree: Master, Institute of Biomedical Sciences, 2015, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0801115-152657

► Diabetic nephropathy which is the most common cause of end stage renal disease continues to increase developed countries in association with the epidemic rise in… (more)

Subjects/Keywords: Extracorporeal shock wave therapy; Diabetic nephropathy; Microalbuminuria; Glomerular; Tissue regeneration; Anti-inflammatory

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Huang, W. (2015). Extracorporeal shock wave therapy ameliorates kidney functions in diabetic nephropathy rat. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0801115-152657

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Huang, Wei-han. “Extracorporeal shock wave therapy ameliorates kidney functions in diabetic nephropathy rat.” 2015. Thesis, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0801115-152657.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Huang, Wei-han. “Extracorporeal shock wave therapy ameliorates kidney functions in diabetic nephropathy rat.” 2015. Web. 25 Jan 2021.

Vancouver:

Huang W. Extracorporeal shock wave therapy ameliorates kidney functions in diabetic nephropathy rat. [Internet] [Thesis]. NSYSU; 2015. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0801115-152657.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huang W. Extracorporeal shock wave therapy ameliorates kidney functions in diabetic nephropathy rat. [Thesis]. NSYSU; 2015. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0801115-152657

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

20. Lin, Wan-jyun. Studies on ABT-751-induced Apoptosis and Autophagy via Reactive Oxygen Species in the Hepatocellular Carcinoma-derived Hep-3B cells.

Degree: Master, Biological Sciences, 2016, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0118116-092731

► Hepatocellular carcinoma (HCC) is one of the most frequently lethal malignant tumors in the world. Systemic therapies in hepatocellular carcinoma of solid tumors, one of… (more)

Subjects/Keywords: Caspase-dependent pathway; Autophagy; Apoptosis; DNA damage; ROS; ABT-751

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Lin, W. (2016). Studies on ABT-751-induced Apoptosis and Autophagy via Reactive Oxygen Species in the Hepatocellular Carcinoma-derived Hep-3B cells. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0118116-092731

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Lin, Wan-jyun. “Studies on ABT-751-induced Apoptosis and Autophagy via Reactive Oxygen Species in the Hepatocellular Carcinoma-derived Hep-3B cells.” 2016. Thesis, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0118116-092731.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Lin, Wan-jyun. “Studies on ABT-751-induced Apoptosis and Autophagy via Reactive Oxygen Species in the Hepatocellular Carcinoma-derived Hep-3B cells.” 2016. Web. 25 Jan 2021.

Vancouver:

Lin W. Studies on ABT-751-induced Apoptosis and Autophagy via Reactive Oxygen Species in the Hepatocellular Carcinoma-derived Hep-3B cells. [Internet] [Thesis]. NSYSU; 2016. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0118116-092731.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Lin W. Studies on ABT-751-induced Apoptosis and Autophagy via Reactive Oxygen Species in the Hepatocellular Carcinoma-derived Hep-3B cells. [Thesis]. NSYSU; 2016. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0118116-092731

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

21. Li, Yu-wei. The role of Mst1/Mst2 in early brain development and glioblastoma formation of the mice.

Degree: Master, Institute of Biomedical Sciences, 2016, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0628116-114955

► Glioblastoma multiforme (GBM) is the most aggressive and malignant primary brain tumor. Primary glioblastomas have a worse prognosis with a median survival of less than… (more)

Subjects/Keywords: Mouse model; Glioma; YAP1; MST1/2; Hippo pathway; Glioblastoma; Apoptosis; Verteporfin

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Li, Y. (2016). The role of Mst1/Mst2 in early brain development and glioblastoma formation of the mice. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0628116-114955

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Li, Yu-wei. “The role of Mst1/Mst2 in early brain development and glioblastoma formation of the mice.” 2016. Thesis, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0628116-114955.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Li, Yu-wei. “The role of Mst1/Mst2 in early brain development and glioblastoma formation of the mice.” 2016. Web. 25 Jan 2021.

Vancouver:

Li Y. The role of Mst1/Mst2 in early brain development and glioblastoma formation of the mice. [Internet] [Thesis]. NSYSU; 2016. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0628116-114955.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Li Y. The role of Mst1/Mst2 in early brain development and glioblastoma formation of the mice. [Thesis]. NSYSU; 2016. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0628116-114955

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

22. Huang, Jhen-hao. Arecoline and areca nut extracts induce differential expression of MAO-A gene in normal and oral cancer cells.

Degree: Master, Institute of Biomedical Sciences, 2015, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0623115-014311

► Globally, there are about 600 million betel quid chewers. In 2004, the International Agency for Research on Cancer had declared betel quid (BQ) chewing without… (more)

Subjects/Keywords: betel quid; oral cancer; arecoline; MAO-A; areca nut extracts; arecaidine

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Huang, J. (2015). Arecoline and areca nut extracts induce differential expression of MAO-A gene in normal and oral cancer cells. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0623115-014311

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Huang, Jhen-hao. “Arecoline and areca nut extracts induce differential expression of MAO-A gene in normal and oral cancer cells.” 2015. Thesis, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0623115-014311.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Huang, Jhen-hao. “Arecoline and areca nut extracts induce differential expression of MAO-A gene in normal and oral cancer cells.” 2015. Web. 25 Jan 2021.

Vancouver:

Huang J. Arecoline and areca nut extracts induce differential expression of MAO-A gene in normal and oral cancer cells. [Internet] [Thesis]. NSYSU; 2015. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0623115-014311.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Huang J. Arecoline and areca nut extracts induce differential expression of MAO-A gene in normal and oral cancer cells. [Thesis]. NSYSU; 2015. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0623115-014311

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

23. Yang, Chun-feng. The disturbance effect of phthalate DEHP on camptothecin-treated human breast cancer cells in vitro and in zebrafish xenograft model.

Degree: Master, Institute of Biomedical Sciences, 2016, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0115116-075905

► Breast cancer is a leading cause of death in women worldwide, and chemotherapy is one of the primary strategies for breast cancer treatment. However, the… (more)

Subjects/Keywords: DNA double-strand breaks; camptothecin; Phthalate; Bis(2-ethylhexyl) phthalate; DEHP; zebrafish; chemoresistance; breast cancer

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❌

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Yang, C. (2016). The disturbance effect of phthalate DEHP on camptothecin-treated human breast cancer cells in vitro and in zebrafish xenograft model. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0115116-075905

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Yang, Chun-feng. “The disturbance effect of phthalate DEHP on camptothecin-treated human breast cancer cells in vitro and in zebrafish xenograft model.” 2016. Thesis, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0115116-075905.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Yang, Chun-feng. “The disturbance effect of phthalate DEHP on camptothecin-treated human breast cancer cells in vitro and in zebrafish xenograft model.” 2016. Web. 25 Jan 2021.

Vancouver:

Yang C. The disturbance effect of phthalate DEHP on camptothecin-treated human breast cancer cells in vitro and in zebrafish xenograft model. [Internet] [Thesis]. NSYSU; 2016. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0115116-075905.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Yang C. The disturbance effect of phthalate DEHP on camptothecin-treated human breast cancer cells in vitro and in zebrafish xenograft model. [Thesis]. NSYSU; 2016. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0115116-075905

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

24. Chen, Tian-zong. Extracorporeal Shock Wave Therapy Attenuates CCl4 induced Liver Fibrosis in Rat.

Degree: Master, Institute of Biomedical Sciences, 2016, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0604116-132259

► Liver fibrosis is the excessive accumulation of extracellular matrix proteins including collagen that occurs in most types of chronic liver diseases. Several studies indicated liver… (more)

Subjects/Keywords: ECSW; Anti-oxidative stress; Liver fibrosis; Proliferation; Inflammatory

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Chen, T. (2016). Extracorporeal Shock Wave Therapy Attenuates CCl4 induced Liver Fibrosis in Rat. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0604116-132259

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Chen, Tian-zong. “Extracorporeal Shock Wave Therapy Attenuates CCl4 induced Liver Fibrosis in Rat.” 2016. Thesis, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0604116-132259.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Chen, Tian-zong. “Extracorporeal Shock Wave Therapy Attenuates CCl4 induced Liver Fibrosis in Rat.” 2016. Web. 25 Jan 2021.

Vancouver:

Chen T. Extracorporeal Shock Wave Therapy Attenuates CCl4 induced Liver Fibrosis in Rat. [Internet] [Thesis]. NSYSU; 2016. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0604116-132259.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Chen T. Extracorporeal Shock Wave Therapy Attenuates CCl4 induced Liver Fibrosis in Rat. [Thesis]. NSYSU; 2016. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0604116-132259

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

25. Lu, Chiu-chin. An extracellular short-chain peptide ABMOK1 from the benthic diatom Nitzschia sp. and its antimicrobial activity.

Degree: PhD, Department of Oceanography, 2015, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0020115-161552

► Most of antimicrobial peptides (AMPs) are less than 10 kDa in molecular weight, hydrophobic, membrane active, and have an overall net positive charge. To date,… (more)

Subjects/Keywords: LC-MS-MS; SEM; Nitzschia sp.; Antimicrobial peptides; Marine benthic diatoms

Record Details Similar Records Cite « Share

❌

APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Lu, C. (2015). An extracellular short-chain peptide ABMOK1 from the benthic diatom Nitzschia sp. and its antimicrobial activity. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0020115-161552

Chicago Manual of Style (16^th Edition):

Lu, Chiu-chin. “An extracellular short-chain peptide ABMOK1 from the benthic diatom Nitzschia sp. and its antimicrobial activity.” 2015. Doctoral Dissertation, NSYSU. Accessed January 25, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0020115-161552.

MLA Handbook (7^th Edition):

Lu, Chiu-chin. “An extracellular short-chain peptide ABMOK1 from the benthic diatom Nitzschia sp. and its antimicrobial activity.” 2015. Web. 25 Jan 2021.

Vancouver:

Lu C. An extracellular short-chain peptide ABMOK1 from the benthic diatom Nitzschia sp. and its antimicrobial activity. [Internet] [Doctoral dissertation]. NSYSU; 2015. [cited 2021 Jan 25]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0020115-161552.

Council of Science Editors:

Lu C. An extracellular short-chain peptide ABMOK1 from the benthic diatom Nitzschia sp. and its antimicrobial activity. [Doctoral Dissertation]. NSYSU; 2015. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0020115-161552

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Open Access Theses and Dissertations