+publisher:"NSYSU" +contributor:("Hsu, Ching-Mei")

NSYSU

1. Wang, Yi-hsuan. Effect of aluminum on peroxidase isozyme in two rice cultivars.

Degree: Master, Biological Sciences, 2008, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0114108-121227

► Aluminum-treated rice seeding (Oryza sativa L. cv. Taichung Native 1 and O. sativa L. cv. Tainung 67) showed significant inhibition in rice root growth, and… (more)

Subjects/Keywords: aluminum; hydrogen peroxide; lignin; Oryza sativa; peroxidase

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Wang, Y. (2008). Effect of aluminum on peroxidase isozyme in two rice cultivars. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0114108-121227

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Wang, Yi-hsuan. “Effect of aluminum on peroxidase isozyme in two rice cultivars.” 2008. Thesis, NSYSU. Accessed January 18, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0114108-121227.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Wang, Yi-hsuan. “Effect of aluminum on peroxidase isozyme in two rice cultivars.” 2008. Web. 18 Jan 2021.

Vancouver:

Wang Y. Effect of aluminum on peroxidase isozyme in two rice cultivars. [Internet] [Thesis]. NSYSU; 2008. [cited 2021 Jan 18]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0114108-121227.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Wang Y. Effect of aluminum on peroxidase isozyme in two rice cultivars. [Thesis]. NSYSU; 2008. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0114108-121227

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

2. Liu, Shih-ming. Study of heat-shock-induced cell death in Saccharomyces cerevisiae with a deficiency of YDL100c.

Degree: Master, Biological Sciences, 2008, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0719108-213007

► YDL100cp is the ArsA homologous protein found in Saccharomyces cerevisiae. Previous studies show that deletion of YDL100c was not lethal but unable to grow at… (more)

Subjects/Keywords: Saccharomyces cerevisiae; YDL100c; lethal heat shock

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APA (6^th Edition):

Liu, S. (2008). Study of heat-shock-induced cell death in Saccharomyces cerevisiae with a deficiency of YDL100c. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0719108-213007

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Liu, Shih-ming. “Study of heat-shock-induced cell death in Saccharomyces cerevisiae with a deficiency of YDL100c.” 2008. Thesis, NSYSU. Accessed January 18, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0719108-213007.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Liu, Shih-ming. “Study of heat-shock-induced cell death in Saccharomyces cerevisiae with a deficiency of YDL100c.” 2008. Web. 18 Jan 2021.

Vancouver:

Liu S. Study of heat-shock-induced cell death in Saccharomyces cerevisiae with a deficiency of YDL100c. [Internet] [Thesis]. NSYSU; 2008. [cited 2021 Jan 18]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0719108-213007.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liu S. Study of heat-shock-induced cell death in Saccharomyces cerevisiae with a deficiency of YDL100c. [Thesis]. NSYSU; 2008. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0719108-213007

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

3. Tseng, Hsiu- Ting. Peritonitis-induced Peroxynitrite Production of Hematopoietic Cells and Lung Damage Depends on the JNK Signaling Pathway.

Degree: Master, Biological Sciences, 2008, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0804108-215703

► Abdominal sepsis is a common, life-threatening condition in the critically ill patients. The c-Jun N-terminal kinase (JNK) is known as a stress-activated protein kinase, in… (more)

Subjects/Keywords: JNK; Peroxynitrite

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APA (6^th Edition):

Tseng, H. T. (2008). Peritonitis-induced Peroxynitrite Production of Hematopoietic Cells and Lung Damage Depends on the JNK Signaling Pathway. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0804108-215703

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Tseng, Hsiu- Ting. “Peritonitis-induced Peroxynitrite Production of Hematopoietic Cells and Lung Damage Depends on the JNK Signaling Pathway.” 2008. Thesis, NSYSU. Accessed January 18, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0804108-215703.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Tseng, Hsiu- Ting. “Peritonitis-induced Peroxynitrite Production of Hematopoietic Cells and Lung Damage Depends on the JNK Signaling Pathway.” 2008. Web. 18 Jan 2021.

Vancouver:

Tseng HT. Peritonitis-induced Peroxynitrite Production of Hematopoietic Cells and Lung Damage Depends on the JNK Signaling Pathway. [Internet] [Thesis]. NSYSU; 2008. [cited 2021 Jan 18]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0804108-215703.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Tseng HT. Peritonitis-induced Peroxynitrite Production of Hematopoietic Cells and Lung Damage Depends on the JNK Signaling Pathway. [Thesis]. NSYSU; 2008. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0804108-215703

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

4. Liu, Ye-Chong. Epigallocatechin gallate attenuate Cisplatin induced acute renal failure in rat.

Degree: Master, Biological Sciences, 2010, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0819110-105433

► Abstract Cisplatin is one of the most effective chemotherapeutic agents used in treatment of a variety of human solid tumors. The most common adverse side… (more)

▼ Abstract Cisplatin is one of the most effective chemotherapeutic agents used in treatment of a variety of human solid tumors. The most common adverse side effect limiting the use of Cisplatin is nephrotoxicity. Recent studies indicate that inflammatory and oxidative signaling play critical role in pathogenesis of Cisplatin related nephrotoxicity. Cisplatin-induced nephrotoxicity is associated with lipid peroxidation and the superoxide anion and hydroxyl radical scavenger could prevent acute renal failure through both attenuation of tubular damage and enhanced regenerative response of the damaged tubular cells. It has been shown that green tea polyphenols with antioxidant properties inhibit inflammatory and oxidative responses in mice. However, the evidence indicating the protective effect of epigallocatechin gallate (EGCG), the major polyphenol found in green tea, on Cisplatin-induced nephrotoxicity is lacking. The present study is to evaluate the effect of EGCG injection on Cisplatin-induced nephrotoxicity in rats. The male rats were divided into four groups (n = 6 each); control group, Cisplatin group, EGCG group and Cisplatin + EGCG group. The control group received only intraperitoneal normal saline injection. Cisplatin (6 mg/kg) was given single dose intraperitoneally at day 0, EGCG (10 mg/time) was given subcutaneously at day 4, day 2 and day 0 before Cisplatin challenge and day 2 and day 4 after Cisplatin injection. Nephrotoxicity was evaluated by biochemical analysis of blood and histopathological observations of rat kidney. Nuclear factor-kappa B (NF-ÎºB) activation, inducible nitric oxide synthase (iNOS) expression and malonyldialdehyde (MDA) content were also determined in rat kidney. Cisplatin injection induced an increase in serum blood urea nitrogen, creatinine and tubular necrosis, and upregulation of NF-ÎºB and iNOS expression and MDA content in kidney. All the increases were significantly inhibited by EGCG treatment. The results suggest that EGCG may attenuate Cisplatin induced nephrotoxicity through the anti-inflammatory/oxidative effects. Advisors/Committee Members: Chen, Hsien-Jung (chair), Mei%22%29&pagesize-30">Hsu, Ching-Mei (committee member), Liu, Jong-Kang (chair).

Subjects/Keywords: acute renal failure; Cisplatin; epigallocatechin gallate

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Liu, Y. (2010). Epigallocatechin gallate attenuate Cisplatin induced acute renal failure in rat. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0819110-105433

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Liu, Ye-Chong. “Epigallocatechin gallate attenuate Cisplatin induced acute renal failure in rat.” 2010. Thesis, NSYSU. Accessed January 18, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0819110-105433.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Liu, Ye-Chong. “Epigallocatechin gallate attenuate Cisplatin induced acute renal failure in rat.” 2010. Web. 18 Jan 2021.

Vancouver:

Liu Y. Epigallocatechin gallate attenuate Cisplatin induced acute renal failure in rat. [Internet] [Thesis]. NSYSU; 2010. [cited 2021 Jan 18]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0819110-105433.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Liu Y. Epigallocatechin gallate attenuate Cisplatin induced acute renal failure in rat. [Thesis]. NSYSU; 2010. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0819110-105433

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

5. CHUNG, HSIU-CHANG. Screening of Antimitotic Drug Combretastatin-A4 Derivatives That Are Capable of Inducing Autophagy in Neuroblastoma SHSY5Y Cell Line.

Degree: Master, Biological Sciences, 2013, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0726113-125155

► Autophagy is a process to clear unwanted protein aggregates and damaged organelles for recycling cellular resources. It is a critical mechanism to maintain cellular homeostasis… (more)

▼ Autophagy is a process to clear unwanted protein aggregates and damaged organelles for recycling cellular resources. It is a critical mechanism to maintain cellular homeostasis for cellular survival when facing environmental stress. Therefore, aberrant autophagy might lead to the diseases such as cancer, pathogen infection and neurodegenerative diseases. Autophagic proecess depends on the fusion of autophagosome with lysosome for the enzymatic degradation of its content. Previous reports indicated the involvement of microtubule in mediating the fusion of autophagosome with either lysosome or late endosome. Therefore, proper dynamic for microtubule formation is critical for autophagic process. Combretastatin A-4 (CA-4) is a useful nature product exhibits anticancer and anti-angiogenesis activity by targeting microtubule formation. However, its clinical use is limited as it exhibits drawbacks such as high toxicity and low water solubility. To improve its clinical use, efforts are ongoing in synthesizing better CA-4 derivatives as anticancer drug candidates. A series of Combretastatin A-4 (CA-4) enediyne derivatives were synthesized by Professor Ming-Jung Wu in the Department of Chemistry of National Sun Yat-Sen University. In this thesis, research was focusing on the analyzing their effects on the microtubule formation in neuroblastoma SHSY5Y and hepatoma Hep3B cell lines by confocal microscopic analysis. The results showed that like CA-4, among 9 enediyne derivatives analyzed, LO-OMeãLO-NH2ãLO-py and HYH10f could inhibit microtubule formation using dosage of their individual IC50. The results of flow cytometric cell cycle analysis showed that LO-OMeãLO-NH2 and HYH10f also had antimitotic effect in that they caused G2/M arrest of SHSY5Y cells. Whereas, the derivatives CPC14c, CPC20a, CPC15a, CPC19a and HYH10a had no effects on microtubule formation or cell cycle arrest. These results indicated the modification or replacement of different function groups might affect their activities differentially. We further investigated the effects of these CA-4 enediyne derivatives on the autophagic process by monitoring the level of two autophagic marker proteins LC3-II and p62 using western blot analysis. Our results showed that like CA-4, LO-OMe, LO-NH2 and CPC15a could lead to the increase of both LC3-II and p62. Compound HYH10f could increase LC3-II level, but not on the level of p62. The rest of the derivative compounds had no effects on the level of both LC3-II and p62. Our results again showed that functional modification might affects autophagic process differentially. The detail signaling mediating their effects on autophagy awaits further investigation. Understanding the molecular mechanism underlies might be helpful in evaluation of their use as therapeutic agents for cancer or neurodegenerative diseases. Advisors/Committee Members: Ming-Jung Wu (chair), Mei%22%29&pagesize-30">Hsu, Ching-Mei (chair), Jiin-Tsuey Cheng (committee member).

Subjects/Keywords: Combrestastatin A-4; autophagy; LC3-II; p62; mitosis

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

CHUNG, H. (2013). Screening of Antimitotic Drug Combretastatin-A4 Derivatives That Are Capable of Inducing Autophagy in Neuroblastoma SHSY5Y Cell Line. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0726113-125155

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

CHUNG, HSIU-CHANG. “Screening of Antimitotic Drug Combretastatin-A4 Derivatives That Are Capable of Inducing Autophagy in Neuroblastoma SHSY5Y Cell Line.” 2013. Thesis, NSYSU. Accessed January 18, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0726113-125155.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

CHUNG, HSIU-CHANG. “Screening of Antimitotic Drug Combretastatin-A4 Derivatives That Are Capable of Inducing Autophagy in Neuroblastoma SHSY5Y Cell Line.” 2013. Web. 18 Jan 2021.

Vancouver:

CHUNG H. Screening of Antimitotic Drug Combretastatin-A4 Derivatives That Are Capable of Inducing Autophagy in Neuroblastoma SHSY5Y Cell Line. [Internet] [Thesis]. NSYSU; 2013. [cited 2021 Jan 18]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0726113-125155.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

CHUNG H. Screening of Antimitotic Drug Combretastatin-A4 Derivatives That Are Capable of Inducing Autophagy in Neuroblastoma SHSY5Y Cell Line. [Thesis]. NSYSU; 2013. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0726113-125155

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

6. Shi, Jhih-Yin. Glial Cell LineâDerived Neurotrophic Factor Gene Transfer Exerts Protective Effect on Axons in Sciatic Nerve Following Constriction-Induced Peripheral Nerve Injury.

Degree: PhD, Biological Sciences, 2011, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823111-140424

► Damage to peripheral nerves following trauma or disease has a number of consequences including burning pain, muscle wasting, paralysis, or organ dysfunction. The most common… (more)

▼ Damage to peripheral nerves following trauma or disease has a number of consequences including burning pain, muscle wasting, paralysis, or organ dysfunction. The most common form of neuropathy is that associated with metabolic abnormality, notably diabetes. Many diabetics, especially those with poor blood sugar control, ultimately develop a distal symmetrical and painful neuropathy that initially affects the longest peripheral axons, but with time spreads proximally. Deficiency in neurotrophic support has been proposed to contribute to the development of diabetic neuropathy. Recently, peripheral gene delivery of vascular endothelial growth factor (VEGF), neurotrophin-3 (NT-3), NGF, BDNF or hepatocyte growth factor (HGF) has been shown to facilitate the continuous production of neurotrophic factors and alleviate the diabetic neuropathy. The role of glial cell-derived neurotrophic factor (GDNF) in the pathogenesis and therapeutics of diabetic neuropathy is not well defined. The main objectives of this research sought to inspect the protective effect of GDNF peripheral gene delivery during hyperglycemia- or constriction- induced sciatic nerve injury in rats. In present proposal, we propose to investigate the change in organization and expressions of GDNF signaling complex in the sciatic nerve following injury in the initial stage. Subsequently, the recombinant adenovirus was used gene delivery system for GDNF to evaluate the potential of intramuscular administration of gene delivery for prevent nerve degeneration, and the molecular mechanism of GDNF to ameliorate neuropathy will be clarified. The above study would enable us to test the hypothesis that the topical gene delivery might be a suitable strategy for the treatment of diabetic neuropathy and other disorders in peripheral nerve. Furthermore, the results of animal studies might be extrapolated for future clinical application. Advisors/Committee Members: Huang, Hung-Tu (chair), Chao, David (committee member), Chen, Lee-Wei (chair), Cheng, Jiin-Tsuey (chair), Tai, ming-hong (committee member), Mei%22%29&pagesize-30">Hsu, Ching-Mei (chair).

Subjects/Keywords: Angiogenesis; Diabetes; Diabetic neuropathy; GDNF; Gene delivery

10 more images…

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Shi, J. (2011). Glial Cell LineâDerived Neurotrophic Factor Gene Transfer Exerts Protective Effect on Axons in Sciatic Nerve Following Constriction-Induced Peripheral Nerve Injury. (Doctoral Dissertation). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823111-140424

Chicago Manual of Style (16^th Edition):

Shi, Jhih-Yin. “Glial Cell LineâDerived Neurotrophic Factor Gene Transfer Exerts Protective Effect on Axons in Sciatic Nerve Following Constriction-Induced Peripheral Nerve Injury.” 2011. Doctoral Dissertation, NSYSU. Accessed January 18, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823111-140424.

MLA Handbook (7^th Edition):

Shi, Jhih-Yin. “Glial Cell LineâDerived Neurotrophic Factor Gene Transfer Exerts Protective Effect on Axons in Sciatic Nerve Following Constriction-Induced Peripheral Nerve Injury.” 2011. Web. 18 Jan 2021.

Vancouver:

Shi J. Glial Cell LineâDerived Neurotrophic Factor Gene Transfer Exerts Protective Effect on Axons in Sciatic Nerve Following Constriction-Induced Peripheral Nerve Injury. [Internet] [Doctoral dissertation]. NSYSU; 2011. [cited 2021 Jan 18]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823111-140424.

Council of Science Editors:

Shi J. Glial Cell LineâDerived Neurotrophic Factor Gene Transfer Exerts Protective Effect on Axons in Sciatic Nerve Following Constriction-Induced Peripheral Nerve Injury. [Doctoral Dissertation]. NSYSU; 2011. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0823111-140424

NSYSU

7. Sy, Wei-Dih. Analysis of human Dynamin IV (Dymple) gene promoter.

Degree: Master, Biological Sciences, 2003, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0904103-145445

► We first identified the transcriptional regulatory element of the human dynamin IV gene (Hdyn IV; dymple). The Hdyn IV belongs to a large GTPase family.… (more)

▼ We first identified the transcriptional regulatory element of the human dynamin IV gene (Hdyn IV; dymple). The Hdyn IV belongs to a large GTPase family. This protein has a N-terminal highly conserved tripartite GTP-binding domain, coiled-coil (CC) region, but it lacks the pleckstrin homology (PH) domain and a modestly conserved C-terminal proline rich domain (PRD). Hdyn IV gene is enriched in subcellular membrane fractions of cytoplasmic vesicles and endoplasmic reticulum, and the function of Hdyn IV gene is considered to be associated with the functions of mitochondria. The Hdyn IV is expressed as four alternative splicing variants in all eukaryotic organisms. Our question concerning why expressions of four alternative splicing variants in brain tumor tissues? To elucidate the regulatory mechanism and the transcription factors involved, we firstly determined the transcriptional start site by 5â RACE. We next cloned the 5â-flanking region of the Hdyn IV gene and determined the nucleotide sequence of 999 bases upstream from the transcription start site. The promoter has several potential binding sites for AP2, Sp1 binding protein, but it lacks TATA and CAAT boxes. Transfection studies using a series of Hdyn IV promoter luciferase constructs in HeLa cell demonstrate that the 5âflanking region has a promoter activity. Functional promoter element of the Hdyn IV gene was located within the â140~ +29 region. Deletion analyses demonstrated that the minimal promoter activity for the transcriptional element of Hdyn IV was detected in the sequence between nucleotides â110 and â100. Electorphoretic mobility shift assay demonstrated that a putative transcriptional factor bound to the â119 to â90 region. Site-directed mutagenesis analysis of this region revealed that nucleotides at positions â108 to â100 were essential for transactivation mediated by this element. To summary, the data indicated that the ââCTCCCAGCAââ (-108~ -100) sequence is capable of regulating Hdyn IV gene expression. However, the protein involved in the binding of this novel sequence requires further study. Advisors/Committee Members: Mei%22%29&pagesize-30">Hsu, Ching-Mei (chair), Hong, Yi-Ren (committee member), Cheng, Jiin-Tsuey (chair), Cho, Chung-Lung (committee member).

Subjects/Keywords: transcription factor; promoter; DynIV

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APA · Chicago · MLA · Vancouver · CSE | Export to Zotero / EndNote / Reference Manager

APA (6^th Edition):

Sy, W. (2003). Analysis of human Dynamin IV (Dymple) gene promoter. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0904103-145445

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Sy, Wei-Dih. “Analysis of human Dynamin IV (Dymple) gene promoter.” 2003. Thesis, NSYSU. Accessed January 18, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0904103-145445.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Sy, Wei-Dih. “Analysis of human Dynamin IV (Dymple) gene promoter.” 2003. Web. 18 Jan 2021.

Vancouver:

Sy W. Analysis of human Dynamin IV (Dymple) gene promoter. [Internet] [Thesis]. NSYSU; 2003. [cited 2021 Jan 18]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0904103-145445.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Sy W. Analysis of human Dynamin IV (Dymple) gene promoter. [Thesis]. NSYSU; 2003. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0904103-145445

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

NSYSU

8. Shiue, Wen-Ming. Studies on the culture and mitochondrial rDNA sequence analysis of Antrodia camphorata mycelium.

Degree: Master, Biological Sciences, 2003, NSYSU

URL: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0816103-004317

► Antrodia camphorata is a Taiwan-unique fungus which grows on the walls inside the core of decayed Cinnamomum kanehirae. Antrodia camphorata provides many bioactive substance and… (more)

Subjects/Keywords: polysaccharides; mitochondrial DNA; antrodia camphorata; triterpenoids

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APA (6^th Edition):

Shiue, W. (2003). Studies on the culture and mitochondrial rDNA sequence analysis of Antrodia camphorata mycelium. (Thesis). NSYSU. Retrieved from http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0816103-004317

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Chicago Manual of Style (16^th Edition):

Shiue, Wen-Ming. “Studies on the culture and mitochondrial rDNA sequence analysis of Antrodia camphorata mycelium.” 2003. Thesis, NSYSU. Accessed January 18, 2021. http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0816103-004317.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

MLA Handbook (7^th Edition):

Shiue, Wen-Ming. “Studies on the culture and mitochondrial rDNA sequence analysis of Antrodia camphorata mycelium.” 2003. Web. 18 Jan 2021.

Vancouver:

Shiue W. Studies on the culture and mitochondrial rDNA sequence analysis of Antrodia camphorata mycelium. [Internet] [Thesis]. NSYSU; 2003. [cited 2021 Jan 18]. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0816103-004317.

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

Council of Science Editors:

Shiue W. Studies on the culture and mitochondrial rDNA sequence analysis of Antrodia camphorata mycelium. [Thesis]. NSYSU; 2003. Available from: http://etd.lib.nsysu.edu.tw/ETD-db/ETD-search/view_etd?URN=etd-0816103-004317

Note: this citation may be lacking information needed for this citation format:
Not specified: Masters Thesis or Doctoral Dissertation

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Open Access Theses and Dissertations