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You searched for +publisher:"McGill University" +contributor:("Andrey V E Cybulsky"). Showing records 1 – 10 of 10 total matches.

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1. Luhovy, Artem. The activation of protein kinase SLK.

Degree: MS, Department of Medicine, 2011, McGill University

The inappropriate expression, localization, or activation of kidney protein kinases may precipitate renal dysplasia or cystic diseases. The expression and activation of the Ste20-like kinase,… (more)

Subjects/Keywords: Health Sciences - Medicine and Surgery

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APA (6th Edition):

Luhovy, A. (2011). The activation of protein kinase SLK. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile97038.pdf

Chicago Manual of Style (16th Edition):

Luhovy, Artem. “The activation of protein kinase SLK.” 2011. Masters Thesis, McGill University. Accessed October 16, 2019. http://digitool.library.mcgill.ca/thesisfile97038.pdf.

MLA Handbook (7th Edition):

Luhovy, Artem. “The activation of protein kinase SLK.” 2011. Web. 16 Oct 2019.

Vancouver:

Luhovy A. The activation of protein kinase SLK. [Internet] [Masters thesis]. McGill University; 2011. [cited 2019 Oct 16]. Available from: http://digitool.library.mcgill.ca/thesisfile97038.pdf.

Council of Science Editors:

Luhovy A. The activation of protein kinase SLK. [Masters Thesis]. McGill University; 2011. Available from: http://digitool.library.mcgill.ca/thesisfile97038.pdf

2. Delarosa, Sierra. Regulation of the Ste20-like kinase, SLK.

Degree: MS, Department of Medicine, 2010, McGill University

Strict regulation of cell growth, apoptosis and differentiation is essential for normal kidney development. The expression and activation of the Ste20-like kinase, SLK, is increased… (more)

Subjects/Keywords: Health Sciences - Medicine and Surgery

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APA (6th Edition):

Delarosa, S. (2010). Regulation of the Ste20-like kinase, SLK. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile86512.pdf

Chicago Manual of Style (16th Edition):

Delarosa, Sierra. “Regulation of the Ste20-like kinase, SLK.” 2010. Masters Thesis, McGill University. Accessed October 16, 2019. http://digitool.library.mcgill.ca/thesisfile86512.pdf.

MLA Handbook (7th Edition):

Delarosa, Sierra. “Regulation of the Ste20-like kinase, SLK.” 2010. Web. 16 Oct 2019.

Vancouver:

Delarosa S. Regulation of the Ste20-like kinase, SLK. [Internet] [Masters thesis]. McGill University; 2010. [cited 2019 Oct 16]. Available from: http://digitool.library.mcgill.ca/thesisfile86512.pdf.

Council of Science Editors:

Delarosa S. Regulation of the Ste20-like kinase, SLK. [Masters Thesis]. McGill University; 2010. Available from: http://digitool.library.mcgill.ca/thesisfile86512.pdf

3. Drozdova, Tetyana. Nephrin missense mutations altez cellular trafficking and induce endoplasmic retioulum stress.

Degree: MS, Department of Medicine, 2012, McGill University

Nephrin, a key component of the filtration slit diaphragm, undergoes post-translational modifications in the endoplasmic reticulum (ER). Mutations in nephrin lead to proteinuria. We examined… (more)

Subjects/Keywords: Biology - Molecular

…of nephrin) was provided by Dr. Tomoko Takano (McGill University) [49… 

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APA (6th Edition):

Drozdova, T. (2012). Nephrin missense mutations altez cellular trafficking and induce endoplasmic retioulum stress. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile106541.pdf

Chicago Manual of Style (16th Edition):

Drozdova, Tetyana. “Nephrin missense mutations altez cellular trafficking and induce endoplasmic retioulum stress.” 2012. Masters Thesis, McGill University. Accessed October 16, 2019. http://digitool.library.mcgill.ca/thesisfile106541.pdf.

MLA Handbook (7th Edition):

Drozdova, Tetyana. “Nephrin missense mutations altez cellular trafficking and induce endoplasmic retioulum stress.” 2012. Web. 16 Oct 2019.

Vancouver:

Drozdova T. Nephrin missense mutations altez cellular trafficking and induce endoplasmic retioulum stress. [Internet] [Masters thesis]. McGill University; 2012. [cited 2019 Oct 16]. Available from: http://digitool.library.mcgill.ca/thesisfile106541.pdf.

Council of Science Editors:

Drozdova T. Nephrin missense mutations altez cellular trafficking and induce endoplasmic retioulum stress. [Masters Thesis]. McGill University; 2012. Available from: http://digitool.library.mcgill.ca/thesisfile106541.pdf

4. Nezvitsky, Lisa. Role of protein-tyrosine phosphatase 1B in complement- mediated glomerular injury.

Degree: MS, Department of Medicine, 2013, McGill University

Activation of endoplasmic reticulum (ER) stress, notably the unfolded protein response (UPR) and ER-associated degradation (ERAD), contributes to injury in certain renal glomerular diseases. Protein-tyrosine… (more)

Subjects/Keywords: Biology - Cell

…immortalized (generously donated by Dr. Michel Tremblay, McGill University), and GEC… …differential sieving technique [46]. All studies were approved by the McGill University… 

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APA (6th Edition):

Nezvitsky, L. (2013). Role of protein-tyrosine phosphatase 1B in complement- mediated glomerular injury. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile119531.pdf

Chicago Manual of Style (16th Edition):

Nezvitsky, Lisa. “Role of protein-tyrosine phosphatase 1B in complement- mediated glomerular injury.” 2013. Masters Thesis, McGill University. Accessed October 16, 2019. http://digitool.library.mcgill.ca/thesisfile119531.pdf.

MLA Handbook (7th Edition):

Nezvitsky, Lisa. “Role of protein-tyrosine phosphatase 1B in complement- mediated glomerular injury.” 2013. Web. 16 Oct 2019.

Vancouver:

Nezvitsky L. Role of protein-tyrosine phosphatase 1B in complement- mediated glomerular injury. [Internet] [Masters thesis]. McGill University; 2013. [cited 2019 Oct 16]. Available from: http://digitool.library.mcgill.ca/thesisfile119531.pdf.

Council of Science Editors:

Nezvitsky L. Role of protein-tyrosine phosphatase 1B in complement- mediated glomerular injury. [Masters Thesis]. McGill University; 2013. Available from: http://digitool.library.mcgill.ca/thesisfile119531.pdf

5. Kaufman, Daniel. Deletion of inositol-requiring enzyme-1α in podocytes disrupts glomerular capillary integrity and autophagy.

Degree: MS, Department of Physiology, 2016, McGill University

 Background: Inositol-requiring enzyme-1α (IRE1α) is an endoplasmic reticulum -transmembrane endoribonuclease-kinase that plays an essential function in extraembryonic tissues during normal development, and is activated during… (more)

Subjects/Keywords: Physiology

…in the Nephrology division of McGill University, for their collaboration and generous… …Ann Rasminsky, my sister Lauren Kaufman, and our late father Elliot Kaufman (McGill… …University BA'81, BCL'84, LLB'85). My mother was always there for me during… 

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APA (6th Edition):

Kaufman, D. (2016). Deletion of inositol-requiring enzyme-1α in podocytes disrupts glomerular capillary integrity and autophagy. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile143727.pdf

Chicago Manual of Style (16th Edition):

Kaufman, Daniel. “Deletion of inositol-requiring enzyme-1α in podocytes disrupts glomerular capillary integrity and autophagy.” 2016. Masters Thesis, McGill University. Accessed October 16, 2019. http://digitool.library.mcgill.ca/thesisfile143727.pdf.

MLA Handbook (7th Edition):

Kaufman, Daniel. “Deletion of inositol-requiring enzyme-1α in podocytes disrupts glomerular capillary integrity and autophagy.” 2016. Web. 16 Oct 2019.

Vancouver:

Kaufman D. Deletion of inositol-requiring enzyme-1α in podocytes disrupts glomerular capillary integrity and autophagy. [Internet] [Masters thesis]. McGill University; 2016. [cited 2019 Oct 16]. Available from: http://digitool.library.mcgill.ca/thesisfile143727.pdf.

Council of Science Editors:

Kaufman D. Deletion of inositol-requiring enzyme-1α in podocytes disrupts glomerular capillary integrity and autophagy. [Masters Thesis]. McGill University; 2016. Available from: http://digitool.library.mcgill.ca/thesisfile143727.pdf


McGill University

6. Yee, Albert. Mutant actinin-4 in the pathogenesis of focal segmental glomerulosclerosis.

Degree: MS, Department of Medicine, 2017, McGill University

Mutations in actinin-4 are associated with heritable focal segmental glomerulosclerosis (FSGS) in humans. We showed that actinin-4 K256E is a misfolded protein that undergoes aggregation… (more)

Subjects/Keywords: Medicine

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APA (6th Edition):

Yee, A. (2017). Mutant actinin-4 in the pathogenesis of focal segmental glomerulosclerosis. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile145436.pdf

Chicago Manual of Style (16th Edition):

Yee, Albert. “Mutant actinin-4 in the pathogenesis of focal segmental glomerulosclerosis.” 2017. Masters Thesis, McGill University. Accessed October 16, 2019. http://digitool.library.mcgill.ca/thesisfile145436.pdf.

MLA Handbook (7th Edition):

Yee, Albert. “Mutant actinin-4 in the pathogenesis of focal segmental glomerulosclerosis.” 2017. Web. 16 Oct 2019.

Vancouver:

Yee A. Mutant actinin-4 in the pathogenesis of focal segmental glomerulosclerosis. [Internet] [Masters thesis]. McGill University; 2017. [cited 2019 Oct 16]. Available from: http://digitool.library.mcgill.ca/thesisfile145436.pdf.

Council of Science Editors:

Yee A. Mutant actinin-4 in the pathogenesis of focal segmental glomerulosclerosis. [Masters Thesis]. McGill University; 2017. Available from: http://digitool.library.mcgill.ca/thesisfile145436.pdf


McGill University

7. Sareen-Khanna, Kapil. [The]] role of ubiquitin-specific protease 14 in glomerular injury.

Degree: MS, Department of Medicine, 2014, McGill University

Protein misfolding in the endoplasmic reticulum (ER) of glomerular epithelial cells (GEC)/podocytes is a feature of certain types of glomerulonephritis. In experimental membranous nephropathy, complement… (more)

Subjects/Keywords: Biology - Physiology

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APA (6th Edition):

Sareen-Khanna, K. (2014). [The]] role of ubiquitin-specific protease 14 in glomerular injury. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile126980.pdf

Chicago Manual of Style (16th Edition):

Sareen-Khanna, Kapil. “[The]] role of ubiquitin-specific protease 14 in glomerular injury.” 2014. Masters Thesis, McGill University. Accessed October 16, 2019. http://digitool.library.mcgill.ca/thesisfile126980.pdf.

MLA Handbook (7th Edition):

Sareen-Khanna, Kapil. “[The]] role of ubiquitin-specific protease 14 in glomerular injury.” 2014. Web. 16 Oct 2019.

Vancouver:

Sareen-Khanna K. [The]] role of ubiquitin-specific protease 14 in glomerular injury. [Internet] [Masters thesis]. McGill University; 2014. [cited 2019 Oct 16]. Available from: http://digitool.library.mcgill.ca/thesisfile126980.pdf.

Council of Science Editors:

Sareen-Khanna K. [The]] role of ubiquitin-specific protease 14 in glomerular injury. [Masters Thesis]. McGill University; 2014. Available from: http://digitool.library.mcgill.ca/thesisfile126980.pdf


McGill University

8. Woychyshyn, Boyan. SLK deletion induces cytoskeletal changes in podocytes.

Degree: MS, Department of Physiology, 2019, McGill University

 Introduction The Ste20-like Kinase, SLK is a serine/threonine protein kinase ubiquitously expressed in mammalian tissues, including glomerular epithelial cells (GECs; podocytes). SLK was shown to… (more)

Subjects/Keywords: Physiology

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APA (6th Edition):

Woychyshyn, B. (2019). SLK deletion induces cytoskeletal changes in podocytes. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile163592.pdf

Chicago Manual of Style (16th Edition):

Woychyshyn, Boyan. “SLK deletion induces cytoskeletal changes in podocytes.” 2019. Masters Thesis, McGill University. Accessed October 16, 2019. http://digitool.library.mcgill.ca/thesisfile163592.pdf.

MLA Handbook (7th Edition):

Woychyshyn, Boyan. “SLK deletion induces cytoskeletal changes in podocytes.” 2019. Web. 16 Oct 2019.

Vancouver:

Woychyshyn B. SLK deletion induces cytoskeletal changes in podocytes. [Internet] [Masters thesis]. McGill University; 2019. [cited 2019 Oct 16]. Available from: http://digitool.library.mcgill.ca/thesisfile163592.pdf.

Council of Science Editors:

Woychyshyn B. SLK deletion induces cytoskeletal changes in podocytes. [Masters Thesis]. McGill University; 2019. Available from: http://digitool.library.mcgill.ca/thesisfile163592.pdf


McGill University

9. Pessina, Katarina. The tumor necrosis factor alpha signalling pathway in glomerular injury.

Degree: MS, Department of Medicine, 2018, McGill University

Focal segmental glomerulosclerosis (FSGS) is among the leading causes of end stage renal failure in adults (requiring dialysis and/or kidney transplantation) and a cause of… (more)

Subjects/Keywords: Medicine

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APA (6th Edition):

Pessina, K. (2018). The tumor necrosis factor alpha signalling pathway in glomerular injury. (Masters Thesis). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile154693.pdf

Chicago Manual of Style (16th Edition):

Pessina, Katarina. “The tumor necrosis factor alpha signalling pathway in glomerular injury.” 2018. Masters Thesis, McGill University. Accessed October 16, 2019. http://digitool.library.mcgill.ca/thesisfile154693.pdf.

MLA Handbook (7th Edition):

Pessina, Katarina. “The tumor necrosis factor alpha signalling pathway in glomerular injury.” 2018. Web. 16 Oct 2019.

Vancouver:

Pessina K. The tumor necrosis factor alpha signalling pathway in glomerular injury. [Internet] [Masters thesis]. McGill University; 2018. [cited 2019 Oct 16]. Available from: http://digitool.library.mcgill.ca/thesisfile154693.pdf.

Council of Science Editors:

Pessina K. The tumor necrosis factor alpha signalling pathway in glomerular injury. [Masters Thesis]. McGill University; 2018. Available from: http://digitool.library.mcgill.ca/thesisfile154693.pdf

10. Elimam, Hanan Attia. Role of calcium-independent phospholipase A2 gamma (iPLA2y) in glomerular epithelial cell injury.

Degree: PhD, Department of Medicine, 2015, McGill University

 In experimental membranous nephropathy, complement C5b-9-induces glomerular epithelial cell (GEC)/podocyte injury and proteinuria. The effects of C5b-9 are mediated via signaling pathways, including calcium-independent phospholipase… (more)

Subjects/Keywords: Health Sciences - General

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APA (6th Edition):

Elimam, H. A. (2015). Role of calcium-independent phospholipase A2 gamma (iPLA2y) in glomerular epithelial cell injury. (Doctoral Dissertation). McGill University. Retrieved from http://digitool.library.mcgill.ca/thesisfile130273.pdf

Chicago Manual of Style (16th Edition):

Elimam, Hanan Attia. “Role of calcium-independent phospholipase A2 gamma (iPLA2y) in glomerular epithelial cell injury.” 2015. Doctoral Dissertation, McGill University. Accessed October 16, 2019. http://digitool.library.mcgill.ca/thesisfile130273.pdf.

MLA Handbook (7th Edition):

Elimam, Hanan Attia. “Role of calcium-independent phospholipase A2 gamma (iPLA2y) in glomerular epithelial cell injury.” 2015. Web. 16 Oct 2019.

Vancouver:

Elimam HA. Role of calcium-independent phospholipase A2 gamma (iPLA2y) in glomerular epithelial cell injury. [Internet] [Doctoral dissertation]. McGill University; 2015. [cited 2019 Oct 16]. Available from: http://digitool.library.mcgill.ca/thesisfile130273.pdf.

Council of Science Editors:

Elimam HA. Role of calcium-independent phospholipase A2 gamma (iPLA2y) in glomerular epithelial cell injury. [Doctoral Dissertation]. McGill University; 2015. Available from: http://digitool.library.mcgill.ca/thesisfile130273.pdf

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